Academic literature on the topic 'HYPERTENTION'
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Journal articles on the topic "HYPERTENTION"
Sumiatin, Titik, Yasin Wahyurianto, and Wahyu Tri Ningsih. "Relation Between Exercise and The Mortal of Hypertention Level At Poliklinik Jantung Rsud Dr. R Koesma Tuban." Jurnal Ners dan Kebidanan (Journal of Ners and Midwifery) 2, no. 2 (August 1, 2015): 164–68. http://dx.doi.org/10.26699/jnk.v2i2.art.p164-168.
Full textSalmiyati, Suri. "Pengaruh Self Help Group terhadap Pengetahuan tentang Hipertensi." Journal of Health Studies 2, no. 1 (April 4, 2018): 75–83. http://dx.doi.org/10.31101/jhes.428.
Full textKim, Seong Yeon. "Weight Gain and Hypertention." Journal of the Korean Medical Association 40, no. 2 (1997): 231. http://dx.doi.org/10.5124/jkma.1997.40.2.231.
Full textNakamura, Y., S. Tamaki, T. Okamura, T. Kadowaki, T. Hayakawa, Y. Kita, H. Kanda, A. Okayama, and H. Ueshima. "HYPERTENTION IN THE ELDERLY." Journal of Hypertension 22, Suppl. 1 (February 2004): S95. http://dx.doi.org/10.1097/00004872-200402001-00404.
Full textRahman, Z., KK Karmaker, M. Ahmed, M. Aziz, S. Chowdhury, and B. Datta. "New Hope for Resistant Hypertention." Cardiovascular Journal 5, no. 1 (October 19, 2012): 81–91. http://dx.doi.org/10.3329/cardio.v5i1.12278.
Full textGrossi, A., R. Corso, N. Tandurella, S. Moretti, G. Cavallaro, L. Robustelli Test, M. Agostinis, et al. "CARDIOVASCULAR REMODELING IN MILD HYPERTENTION." Journal of Hypertension 36, Supplement 1 (June 2018): e230-e231. http://dx.doi.org/10.1097/01.hjh.0000539648.36597.0d.
Full textTAKESHITA, Akira. "Altered vascular reactivity in hypertention." Nihon Naika Gakkai Zasshi 80, no. 9 (1991): 1505–7. http://dx.doi.org/10.2169/naika.80.1505.
Full textYoshino, Yasue, Shunsei Hirohata, Akiteru Takeuchi, and Takashi Hashimoto. "Systemic lupus erythematosus presenting with pulmonary hypertention and renovascular hypertention-association with anti-cardiolipin antibodies." Japanese Journal of Clinical Immunology 17, no. 5 (1994): 585–91. http://dx.doi.org/10.2177/jsci.17.585.
Full textGimaletdinova, Irina A., and SAyar R. Abdоulkhakov. "TO DIFFERENTIAL DIAGNOSIS OF PORTAL HYPERTENTION." Bulletin of Contemporary Clinical Medicine 1, no. 1 (2008): 125–27. http://dx.doi.org/10.20969/vskm.2008.1(1).125-127.
Full textChoi, Hye Sook, and Sang Do Lee. "Medeical Therapy For Pulmonary Arterial Hypertention." Tuberculosis and Respiratory Diseases 60, no. 2 (2006): 142. http://dx.doi.org/10.4046/trd.2006.60.2.142.
Full textDissertations / Theses on the topic "HYPERTENTION"
Kusch, Fabio. "Antihypertensive mechanism of amlodipine in essential hypertention /." [S.l : s.n.], 1994. http://www.ub.unibe.ch/content/bibliotheken_sammlungen/sondersammlungen/dissen_bestellformular/index_ger.html.
Full textMangos, George Jack St George Clinical School UNSW. "MECHANISMS OF STEROID-INDUCED HYPERTENSION IN MAN AND RAT." Awarded by:University of New South Wales. St. George Clinical School, 1999. http://handle.unsw.edu.au/1959.4/32666.
Full textLee, Jung-Ok. "Cardiovascular protective effects of Lindera obtusiloba." Thesis, Strasbourg, 2013. http://www.theses.fr/2013STRAJ005.
Full textEndothelial dysfunction is a major worldwide topic because it is an important component and risk factor of a number of common human diseases. Therefore, endothelial dysfunction is considered a hallmark for vascular diseases, and has also been shown to be predictive of future adverse cardiovascular events. The main characteristic is a reduced NO production and bioavailability, and an increased vascular oxidative stress. The goal of the present research was to find a candidate for cardiovascular protective herbal medicine for the treatment of endothelial dysfunction. Through measurement of changes in isometric tension of porcine coronary artery rings, Lindera obtusiloba was selected amongst three hundred plants. Thereafter, the aim of our research was to determine whether an ethanolic extract of L. obtusiloba stems (LOE) improves endothelial dysfunction via activation of endothelial nitric oxide synthase and reduction of oxidative stress oxidase in vitro and in several animal models of cardiovascular diseases, and, if so, to elucidate the underlying mechanism. Altogether, the present findings indicate that LOE has vasoprotective effects both in vitro and in vivo including the Ang II-induced hypertention in rats, a type 2 diabetic mice model, and an atherosclerotic mice model, and a thromboembolism mice model, which involve its ability to stimulate the formation of NO, to reduce oxidative stress in the arterial wall, and to inhibit platelet aggregation. In conclusion, our studies reveal that LOE might be an interesting candidate as a cardiovascular protective herbal medicine in pathologies with endothelial dysfunction
Ngwenchi, Nkeh Benedicta. "Mode of Impact of Genetic Determinants of Hypertension in People of African Descent." Thesis, 2006. http://hdl.handle.net/10539/1620.
Full textBlood pressure (BP) is a heritable trait. However, the loci responsible and the mechanisms by which these genes determine BP are uncertain. Based on widely published data regarding frequent phenotypic characteristics that exemplify essential hypertension (EHT) in persons of African ancestry, in the present thesis I explored the role of gene candidates most likely to contribute to BP in this group. In this regard a high frequency of persons of African descent experience increases in BP in response to an enhanced salt intake (salt-sensitive hypertension). In addition, many patients of African origin with EHT fail to respond to inhibition of angiotensin-converting enzyme (ACE) with an appropriate decrease in BP, a factor that cannot be explained entirely on the basis of reduced plasma renin levels in this group. Thus, I evaluated the role of several gene variants that could influence either renal salt handling or the activity and effects of the renin-angiotensin system on BP in subjects of African ancestry. Although the angiotensinogen (AGT) gene has at least 3 variants in the promoter region that influence angiotensinogen expression and which occur with a remarkably high frequency in populations of African ancestry, their role in this group is still controversial. To-date, interactions between these variants have not been considered. Using a casecontrol study design in a sample of 1325 subjects, as well as association analysis with 24 hour ambulatory BP (ABP) values in 626 hypertensives, I confirmed that an independent effect of functional AGT gene variants on the risk for EHT or 24 hour ABP was weak at best. Importantly, however, interactions between the -20A C and -217G A variants were noted to strongly impact on the risk for EHT as well as ABP. Furthermore, interactions between the -20A C and -217G A variants played a major role in iii contributing toward the variability of ABP responses to ACE inhibitors, but not calcium channel blockers in this population group, with genotype determining whether or not ACE inhibitor responses occurred. Although the 825C T polymorphism of the guanosine triphosphate (G) protein 3 subunit (GNB3) gene influences the activity of a substance that modifies renal salt handling, namely the Na+/H+ exchanger, its impact in hypertensives of African descent is controversial. In the present thesis I confirmed in a large sample that the GNB3 variant was not associated with the risk for EHT or ABP values in subjects of African ancestry. However, because the activity of the exchanger is enhanced in obesity I hypothesised that the GNB3 gene variant could mediate a clinically relevant BP effect by modifying the impact of body size on BP (type I or II genetic effect). Indeed, GNB3 genotype proved to be a strong determinant of the impact of body size on systolic BP values, with genotype determining whether or not the effect occurred. The epithelial sodium channel (ENaC) and atrial natriuretic peptide (ANP) have an important influence on renal salt handling. The T594M polymorphism of the -subunit of the ENaC gene only exists with a relatively high frequency in subjects of African ancestry. Previous studies conducted in this population group in relatively small samples have indicated that the ENaC and ANP gene variants determine BP in subjects of African descent. In a larger sample of subjects of African descent I demonstrated that the T594M polymorphism of the ENaC gene has no impact on BP in this population group. However, my results suggest that the ANP gene may be a candidate worthy of further study. In conclusion, the results described in this thesis provide evidence that lends some clarity to the role of likely gene candidates for BP control in people of African descent. iv Importantly, data from this thesis suggest that interactions between functional variants of specific loci (e.g the AGT gene), and clinically relevant type I or II genetic effects (no independent actions, but modifier gene effects, e.g, GNB3) should be considered before excluding loci as playing an important role in BP control. Moreover, this thesis provides the first substantial data to indicate that gene variants determine the variability of BP responses to pharmacological agents in hypertension in this population group.
Papadopoulos, Zisis. "ANALYSIS OF PHARMACOTHERAPY AND DRUG RELATED PROBLEMS IN PATIENT WITH ARTERIAL HYPERTENSION IN GREECE." Master's thesis, 2014. http://www.nusl.cz/ntk/nusl-333038.
Full textMostert, Karien. "The prevalencce of certain risk factors of non-communicable diseases in a rural community : a physiotherapeutic perspective." Diss., 2001. http://hdl.handle.net/2263/27267.
Full textDissertation (MSc (Physiotherapy Management))--University of Pretoria, 2005.
Physiotherapy
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CHEN, LI-JU, and 陳麗如. "A study of the intention of regular brisk walking of Hypertention, Hyperglycemia and Hyperlidemia cases in a District of Taipei city- The application of the Theory of Planned Behavior." Thesis, 2009. http://ndltd.ncl.edu.tw/handle/j9ta7f.
Full textBooks on the topic "HYPERTENTION"
Garbarz, Eric. Cuisine anti-choleste rol: Diabe te, hypertention, surpoids. Paris: ESI, 2009.
Find full textK, Hollenberg Norman, ed. Atlas of hypertention. 5th ed. Philadelphia, PA: Current Medicine, 2005.
Find full textClinical Management Hypertention Diabetes. Informa Healthcare, 2001.
Find full textKatritsis, Demosthenes G., Bernard J. Gersh, and A. John Camm. Primary (essential) hypertension. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199685288.003.0459_update_004.
Full textConference papers on the topic "HYPERTENTION"
Arofiati, Fitri, and Fitri Ramadhani. "The Quality of Life in Hypertention Patients’ Before and After E-Discharge Planning Intervention." In International Conference on Health and Medical Sciences (AHMS 2020). Paris, France: Atlantis Press, 2021. http://dx.doi.org/10.2991/ahsr.k.210127.041.
Full textWicaksono, Kurniawan Erman, Ristya Widi Endah Yani, and Elfian Zulkarnain. "Effectiveness Difference of Family Psychoeducation Model and Family Centered Empowerment Model on Knowledge and Attitude in The Poor Family of Preventing Hypertention on Families in Distric Jember." In The 9th International Nursing Conference: Nurses at The Forefront Transforming Care, Science and Research. SCITEPRESS - Science and Technology Publications, 2018. http://dx.doi.org/10.5220/0008320700730077.
Full textFerdianto, Angga, Didik Gunawan Tamtomo, and Endang Sutisna Sulaeman. "Factors Affecting Tertiary Preventive Behaviors among Patients with High Blood Pressure." In The 7th International Conference on Public Health 2020. Masters Program in Public Health, Universitas Sebelas Maret, 2020. http://dx.doi.org/10.26911/the7thicph.01.43.
Full textChin, Kelly, Gwyn D'Souza, Zhi-Cheng Jing, Gary Burgess, Lorraine Collings, John G. Teeter, Janice Lamb, and Diane Zwicke. "A Multinational, Randomized, Double-Blind Trial Of Sitaxentan Monotherapy Compared With Sitaxentan In Combination With Sildenafil In Patients With Pulmonary Arterial Hypertention: The Sitaxentan Randomized Prospective Assessment Adding Sildenafil (SR-PAAS)." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a4812.
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