Relevant bibliographies by topics / Hypercholesteremia

Academic literature on the topic 'Hypercholesteremia'

Author: Grafiati
Published: 4 June 2021
Last updated: 28 February 2023

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Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters

Journal articles on the topic "Hypercholesteremia":

1

Visavadiya, Nishant P., and A. V. R. L. Narasimhacharya. "AsparagusRoot Regulates Cholesterol Metabolism and Improves Antioxidant Status in Hypercholesteremic Rats." Evidence-Based Complementary and Alternative Medicine 6, no. 2 (2009): 219–26. http://dx.doi.org/10.1093/ecam/nem091.

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Hyperlipidemia/hypercholesteremia are major risk factors for atherosclerosis and cardiovascular diseases. Root ofAsparagus racemosus(AR) is widely used in Ayurvedic system of medicine in India and is known for its steroidal saponin content. This study was designed to investigate the hypocholesteremic and antioxidant potential of AR root in both normo- and hypercholesteremic animals. Normal and hypercholesteremic male albino rats were administered with root powder of AR (5 and 10 g% dose levels) along with normal and hypercholesteremic diets, respectively, for a duration of 4 weeks. Plasma and hepatic lipid profiles, fecal sterol, bile acid excretion and hepatic antioxidant activity were assessed. Inclusion of AR root powder in diet, resulted in a dose-dependant reduction in plasma and hepatic lipid profiles, increased fecal excretion of cholesterol, neutral sterol and bile acid along with increases in hepatic HMG-CoA reductase activity and bile acid content in hypercholesteremic rats. Further, AR root also improved the hepatic antioxidant status (catalase, SOD and ascorbic acid levels). No significant changes in lipid and antioxidant profiles occurred in the normocholesteremic rats administered with AR root powder. AR root appeared to be useful as a dietary supplement that offers a protection against hyperlipidemia/hypercholesteremia in hypercholesteremic animals. The results of the present study indicate that the potent therapeutic phyto-components present in AR root i.e. phytosterols, saponins, polyphenols, flavonoids and ascorbic acid, could be responsible for increased bile acid production, elimination of excess cholesterol and elevation of hepatic antioxidant status in hypercholesteremic conditions.
2

Kamalova, Aelita A. "Diet Therapy in Children with Hypercholesteremia." Current Pediatrics 19, no. 4 (September 19, 2020): 309–15. http://dx.doi.org/10.15690/vsp.v19i4.2140.

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Dietrich, Hans H. "Apolipoprotein E in Hypercholesteremia and Beyond." Stroke 38, no. 7 (July 2007): 2036. http://dx.doi.org/10.1161/strokeaha.107.489856.

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LEWIS, S. "P514 Hypercholesteremia induces baroreceptro reflex dysfunction." European Heart Journal 24, no. 5 (March 2003): 81. http://dx.doi.org/10.1016/s0195-668x(03)93952-2.

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NAKAI, TSUGUHIKO. "Familial hypercholesteremia complicated by nephrotic syndrome." Nihon Naika Gakkai Zasshi 79, no. 8 (1990): 1079–80. http://dx.doi.org/10.2169/naika.79.1079.

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HURLEY, D. "Hypercholesteremia induces enhanced circulating inflammatory activity." American Journal of Hypertension 17, no. 5 (May 2004): S242. http://dx.doi.org/10.1016/j.amjhyper.2004.03.648.

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7

Li, Nan F., Hong M. Wang, Jin Yang, Ling Zhou, Xiao G. Yao, and Jing Hong. "Serum uric acid is associated with metabolic risk factors for cardiovascular disease in the Uygur population." Applied Physiology, Nutrition, and Metabolism 34, no. 6 (December 2009): 1032–39. http://dx.doi.org/10.1139/h09-101.

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The prevalence of hyperuricemia is low in Uygurs, who have a high prevalence of cardiovascular risk factors such as hypertension, overweight–obesity, dyslipidemia, hyperglycemia, and insulin resistance (IR). This study sought to investigate the relationships between serum uric acid (UA) and these risk factors in this population. A cross-sectional study was conducted in Uygurs (859 males, 1268 females) aged 20 to 70 years. Demographic data, systolic blood pressure (SBP), diastolic blood pressure (DBP), body mass index (BMI), and fasting and postprandial blood were obtained, and biological measurements were determined. The mean of BMI, SBP, DBP, total cholesterol, high-density lipoprotein cholesterol (HDL-c), low-density lipoprotein cholesterol (LDL-c), triglycerides, fasting blood glucose, fasting insulin, and homeostasis model assessment insulin resistance index (HOMA-IR), and the prevalence of hypertension, IR, hyperglycemia, overweight–obesity, hypercholesteremia, hyper-LDL-c, and hypertriglyceridemia increased with UA but the prevalence of hypo-HDL-c decreased (p < 0.05). Logistic regression analysis showed that the odds ratios for IR, overweight–obesity, hypercholesteremia, hyper-LDL-c, and hypertriglyceridemia against the lowest UA group increased but decreased for hypo-HDL-c (p < 0.05). The UA in the hypo-HDL-c group was lower than that of the controls; the prevalence of hypo-HDL-c in hyperuricemia subjects was lower than in those with normal UA (p < 0.05). But the opposite results were observed between overweight–obesity, hyperglycemia, IR, hypercholesteremia, hypertriglyceridemia, and hyper-LDL-c and correspondence controls, respectively (p < 0.05). In Uygur, elevated UA is associated with overweight–obesity, hypercholesteremia, hyper-LDL-c, hypertriglyceridemia, hyperglycemia, and IR. The HDL-c level significantly increases with UA, whereas the prevalence of hypo-HDL-c decreases. Further studies are needed to clarify why UA is positively correlated to HDL-c.
8

Nichols, E. H., and M. R. Jaff. "Hypercholesteremia, Hypertension, and Antiplatelet Therapy in PAD." MD Conference Express 13, no. 19 (December 1, 2013): 31. http://dx.doi.org/10.1177/155989771319015.

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9

Kumawat, Vinod Bihari, Surendra Kumar Sharma, Uttam Kumar Sharma, and Sudhir Sharma. "Effect of trikatu compound in hypercholesteremia- a clinical study." Environment Conservation Journal 16, no. 1&2 (June 12, 2015): 57–61. http://dx.doi.org/10.36953/ecj.2015.161209.

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Tokura, T. "Changes in Bruch's Membrane in Experimental Hypercholesteremia in Rats." Japanese Journal of Ophthalmology 43, no. 4 (July 8, 1999): 337–38. http://dx.doi.org/10.1016/s0021-5155(99)00055-6.

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You might also be interested in the extended bibliographies on the topic 'Hypercholesteremia' for particular source types:
Journal articles Dissertations / Theses Books

Dissertations / Theses on the topic "Hypercholesteremia":

1

Staples, Heidi. "The effect of lipid-lowering pharmacotherapy on concurrent diet and exercise behaviors /." Thesis, McGill University, 2000. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=31543.

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The National Cholesterol Education Program Adult Treatment Panel II (NCEP ATP II) unequivocally advocates an initial trial of dietary modification in both primary and secondary prevention prior to the institution of pharmacotherapy. Perhaps the rationale for this delay rests in the inherent, yet unsubstantiated, fear among clinicians that lifestyle change will be compromised in the presence of concurrent pharmacotherapy. However, the question of adherence to diet and exercise interventions following the initiation of lipid-lowering drug therapy has seemingly never been addressed scientifically.
It was therefore hypothesized that pharmacologically-treated patients with untreated hypercholesterolemia started on a program of lifestyle modification would achieve relatively less reduction in dietary fat intake and body weight, and participate less often in physical activity, if a pharmacologic agent was simultaneously prescribed. This was tested by a protocol in which these and related variables were assessed in participants who thought they were taking a lipid-lowering medication at diagnosis, compared to conventional initial treatment of diet and exercise alone. (Abstract shortened by UMI.)
2

Collins, Melissa. "Effects of plant sterols and exercise training on apolipoprotein A and B, adiponectin, growth hormone and ghrelin in hypercholesterolemic sedentary adults." Thesis, McGill University, 2006. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=99331.

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Plant sterols (PS) lower total cholesterol (TC), low density lipoprotein cholesterol (LDL-C), and inflammatory markers, and decrease risk of atherosclerotic cardiovascular disease (CVD). Exercise increases high density lipoprotein cholesterol (HDL-C) levels and decreases triglycerides (TG) and inflammation, also reducing the risk of CVD. The study objective was to investigate the combined effects of PS and exercise on apolipoproteins (apo) A and B, adiponectin, growth hormone (GH) and ghrelin, in context of previously obtained lipid data. In an 8-wk, placebo-controlled, parallel-arm clinical trial, 84 subjects were randomly assigned to: (1) combination of PS and exercise, (2) exercise, (3) PS, or (4) control group. PS increased (P=0.04) adiponectin values by 15%. ApoA was associated with HDL and apoB with LDL values at baseline. ApoA %change was correlated to HDL %change in the exercise group. ApoB, GH and ghrelin were unchanged. The capability of PS to increase adiponectin values reinforce their role in preventing inflammation, atherosclerosis, and CVD.
3

Vergotine, Joseph Vincent. "The MED-PED project : presymptomatic diagnosis in families with disease- related LDL receptor gene mutations." Thesis, Stellenbosch : Stellenbosch University, 2000. http://hdl.handle.net/10019.1/51997.

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Thesis (MSc)--Stellenbosch University, 2000.
ENGLISH ABSTRACT: Familial hypercholesterolaemia (FH) contributes significantly to the high death rate from cardiovascular disease worldwide. FH is a common autosomal co-dominant disease characterised by raised cholesterol levels and premature coronary heart disease (CHD). Whilst these features usually are very prominent in homozygotes the clinical diagnosis of heterozygotes is complicated by variable phenotypic expression. Specific founder genes in the low-density lipoprotein receptor (LDLR) gene have increased the prevalence of FH in South African Afrikaners, Indians, Jews and Coloureds, and screening for these known mutations allows unequivocal diagnosis of FH-affected individuals. The systematic molecular analysis of FH resulted in the identification of at least ten founder-type LDLR gene mutations among the 56 different gene defects described to date in the diverse South African population. DNA screening of 792 at-risk family members for the FH-related mutations identified in 379 index cases, allowed accurate disease diagnosis in an additional 340 relatives and exclusion of the relevant mutation in 452 individuals. This effort forms part of the MED PED FH initiative, a collaborative project to "Make Early Diagnosis and Prevent Early Deaths in MEDical PEDigrees with FH". Evaluation of clinical criteria versus DNA diagnosis of three founder-related mutations (D154N, D206E and V408M) in the South African population demonstrated that the sensitivity and specificity of diagnoses, based on total cholesterol values measured in family members of index cases recruited for this study, were 88% and 77%, respectively. A population-directed DNA diagnosis of FH is therefore justified in South Africa on a routine basis, since expression of the defective gene measured in biochemical tests does not allow accurate diagnosis of FH in all cases. The application of mutation detection was illustrated by prenatal diagnosis of FH performed for a couple who are both heterozygous for the most common Afrikaner mutation, D206E. The mutation was absent in the foetus and a normocholesterolaemic infant was born. Prenatal diagnosis of FH, aimed at the detection of homozygous cases, is particularly applicable in populations and families with molecularly defined LDLR gene mutations. The MED-PED approach resulted in accurate diagnosis and subsequent treatment of FH in more patients, and referral to lipid clinics where they could receive the intensive care their condition justifies. Molecularly diagnosed FH patients will be the first to benefit from future treatment approaches based on mutation type.
AFRIKAANSE OPSOMMING: Familiële hiprcholesterolemie dra grootliks by tot die wêreldwye hoë sterftesyfer van kardiovaskulêre siekte. FH is 'n algemene outosomale ko-dominante siekte wat gekenmerk word deur verhoogde cholesterolvlakke en vroeë koronêre hartsiekte. Terwyl hierdie kenmerke prominent is in homosigote, word die kliniese diagnose van heterosigote bemoeilik deur variasie in fenotipiese uitdrukking. Spesifieke stigtergene in die lae-digtheids lipoproteien reseptor (LDLR) geen het die voorkomssyfer van FH verhoog in Suid Afrikaanse Afrikaners, Indiërs, Jode en Kleurlinge. Sifting vir hierdie bekende mutasies maak akkurate diagnose van FH geaffekteerde individue moontlik. Die sistematiese molekulêre analise van FH het aangetoon dat ten minste tien van die 56 verskillende geen defekte wat tot dusver beskryf is in die Suid-Afrikaanse populasie stigtertipe LDLR geen mutasies is. DNA sifting van 792 familielede vir die FH-verwante mutasie in 379 indeksgevalle geïdentifiseer is, het akkurate diagnose moontlik gemaak in 340 addisionele familielede, en uitsluiting daarvan in 452 individue. Hierdie poging vorm deel van die MED-PED FH ("Make Early Diagnosis and Prevent Early Deaths in MEDical PEDigrees with FH) inisiatief. Evaluering van kliniese kriteria teenoor DNA diagnose van drie stigter verwante mutasies (D154N, D206E en V408M) in die Suid Afrikaanse populasie het getoon dat die sensitiwiteit en spesifisiteit van die diagnose, wat gebasseer is op totale cholesterol waardes in familielede van indeksgevalle, onderskeidelik 88% en 77% was. 'n Populasie gerigte DNA diagnose van FH is dus geregverdig in Suid-Afrika op "n roetine basis, omdat die defektiewe geen nie altyd in biochemiese toetse uitgedruk word nie. Die waarde van mutasie opsporing is geillustreer deur 'n voorgeboortelike diagnose van FH wat aangevra is vir ouers wat beide heterosigoties is vir die mees algemene Afrikaner mutasie, D206E. Die mutasie was afwesig in die fetus en 'n normocholesterolemiese baba is gebore. Voorgeboortelike diagnose van FH, wat gemik is op die opsporing van homosigotiese gevalle, is veral van toepassing in populasies en families met bekende LDLR geen mutasies. Die MED-PED benadering het gelei tot akkurate diagnose en daaropvolgende behandeling van FH in meer pasiënte, en verwysings na lipiedklinieke waar hulle intensiewe aandag kan geniet. Molekulêre gediagnoseerde FH pasiënte sal die eerste wees om baat te vind by toekomstige behandeling wat moontlik gebasseer sal word op mutasie status.
4

Wong, Kwok-kit Sunny. "A study of DNA mutations in LDL receptor gene of Chinese patients with familial hypercholesterolaemia /." Hong Kong : University of Hong Kong, 1997. http://sunzi.lib.hku.hk/hkuto/record.jsp?B19109878.

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St-Onge, Marie-Pierre. "Effect of kefir supplementation on blood lipid parameters in free-living hypercholesterolemic men." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape9/PQDD_0024/MQ50885.pdf.

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Miller, Stephanie M. "The Relationship of Waist Size to Blood Pressure and Cholesterol Among College Students." Lynchburg, Va. : Liberty University, 2007. http://digitalcommons.liberty.edu.

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Yang, Tat-chi Teddy. "Effect of Chinese green tea on diet-induced hypercholesterolemia and arteriosclerosis in rats /." Hong Kong : University of Hong Kong, 1998. http://sunzi.lib.hku.hk/hkuto/record.jsp?B19657560.

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Neubauer, Tamara E. "Cholesterol reduction in men : an experimental investigation of intensive treatment with frequent feedback versus a simple educational treatment /." Thesis, This resource online, 1990. http://scholar.lib.vt.edu/theses/available/etd-03122009-040807/.

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Matthan, Nirupa Rachel. "Impact of hydrogenated fat consumption on in vivo lipid metabolism in moderately hypercholesterolemic women." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape4/PQDD_0033/NQ64617.pdf.

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Fourie, Anne Madeleine. "The expression and metabolism of low density lipoprotein receptors in familial hypercholesterolaemia." Doctoral thesis, University of Cape Town, 1989. http://hdl.handle.net/11427/27174.

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The expression of two phenotypically-contrasting LDL receptor mutations was characterized in cultured fibroblasts from the genetically-homozygous Afrikaner subjects, FH1a and lb, and FH3a and 3b, respectively. Surface receptor expression and functional activity were studied by ligand (¹²⁵I-LDL) and monoclonal antibody (¹²⁵I-IgG-C7) binding, and c35s]-methionine pulse-chase experiments were used to analyze biosynthesis, processing and degradation of IgG-C7- immunoprecipitable mutant receptors. Cells from the "receptor-negative" subjects, FH3a and 3b exhibited reduced, but significant (40-60% of normal) LDL receptor synthesis rates. Newly-synthesized precursors were processed slowly (t½ 1.5 hours versus normal t½ of approximately 15 minutes) to mature receptors which reached the cell-surface, but were rapidly degraded thereafter with a half-life of approximately 1.7 hours (normal value 12.6 hours) thus representing a new type of LDL receptor defect. Lysosomotropic weak bases such as ammonium chloride partially inhibited rapid degradation of the mutant receptors, suggesting the involvement of proteolysis in acidic compartments such as lysosomes or endosomes. Fibroblasts from FH1a and lb exhibited normal synthesis rates of LDL receptor precursors that were processed at a severely reduced rate (t½ approximately 5 hours) to functionally heterogeneous mature surface receptors. Onethird of the receptors (20% of normal levels) bound ¹²⁵I-LDL with normal affinity at 4°C and 37°C, whereas the majority were able to recognize only ¹²⁵I-IgG-C7, and apparently showed defective internalisation and subsequent degradation of the bound IgG-C7 at 37°C. The existence of the two receptor populations was further supported by selective intracellular trapping and degradation of only the active, LDL-binding population, in the presence of ammonium chloride and LOL. The abnormal form predominated even in newly-synthesized receptors and reached a maximum of 50-70% of normal levels after 48 hours of upregulation. Upregulation kinetics and degradation rates (t½ = 10-11 hours) of both functionally-active and abnormal receptor populations were similar to normal. A progressive increase in apparent molecular weight of the slowly-processed precursor receptors suggested a possible role for abnormal glycosylation in the formation of both "normal" and abnormal conformations of the same receptor molecule.
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Books on the topic "Hypercholesteremia":

1

National Cholesterol Education Program (U.S.). Expert Panel on Detection, Evaluation, and Treatment of High Blood Cholesterol in Adults., ed. Report of the Expert Panel on Detection, Evaluation, and Treatment of High Blood Cholesterol in Adults. [Bethesda, Md.]: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1989.

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Huang, Zhaosheng. Fei pang bing zhi fang gan yu gao zhi xue zheng Zhong Xi yi zhen liao yu tiao yang =: Obesity fat liver and high-lipidemia. Xianggang: Tian heng wen hua chu ban gong si, 2001.

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National, Cholesterol Education Program (U S. ). Expert Panel on Detection Evaluation and Treatment of High Blood Cholesterol in Adults. Third report of the National Cholesterol Education Program (NCEP) Expert Panel on Detection, Evaluation, and Treatment of High Blood Cholesterol in Adults (adult treatment panel III): Final report. [Washington, D.C.]: The Program, 2002.

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McCord, Holly. Win the cholesterol war: 100 real-life secrets to trimming points (and pounds). New York: Berkley Books, 2003.

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McCord, Holly. Win the cholesterol war: 100 real-life secrets to trimming points (and pounds). New York: Berkley Books, 2003.

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McCord, Holly. Win the cholesterol war: 100 real-life secrets to trimming points (and pounds). New York: Berkley Books, 2003.

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Wetherill, Douglas. High cholesterol. Cincinnati, OH: Betterway Books, 2000.

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R, Gordon Bruce, and Gotto Antonio M, eds. The Treatment of severe hypercholesterolemia-- can we impact disease course?: Proceedings of a symposium, November 9, 1991, Anaheim, California. Princeton, NJ: Excerpta Medica, 1992.

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National Cholesterol Education Program (U.S.), ed. Second report of the expert panel on detection, evaluation, and treatment of high blood cholesterol in adults (adult treatment panel II). [Bethesda, Md.?]: National Cholesterol Education Program, National Institute of Health, National Heart, Lung, and Blood Institute, 1993.

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National Cholesterol Education Program (U.S.). Expert Panel on Detection, Evaluation, and Treatment of High Blood Cholesterol in Adults. Third report of the National Cholesterol Education Program (NCEP) Expert Panel on Detection, Evaluation, and Treatment of High Blood Cholesterol in Adults (adult treatment panel III): Executive summary. Bethesda, Md.?]: The Program, 2001.

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Book chapters on the topic "Hypercholesteremia":

1

Loria, Roger M. "Effect of Dietary Hypercholesteremia on Host Immune Response." In Nutrients, Stress, and Medical Disorders, 267–78. Totowa, NJ: Humana Press, 2006. http://dx.doi.org/10.1385/1-59259-952-4:267.

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Hunninghake, D. B., D. M. Hibbard, W. C. Duane, M. L. Freeman, W. F. Prigge, K. J. Graham, and R. L. Gebhard. "Metabolic Studies with Lovastatin in Patients with Primary Hypercholesteremia." In Drugs Affecting Lipid Metabolism, 150–54. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71702-4_27.

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Wisnu Murti, Tridjoko. "Fermentation of Bovine, Non-Bovine and Vegetable Milk." In Fermentation - Processes, Benefits and Risks. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.96699.

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Fermented milk has been developing alongside the history of human civilization. It is observed having positive effect on gastrointestinal health. It has reaches at the steps of explaining what happens in the process, despite some information is still unclear. Fermentation involves many organisms, technique, biochemical reactions, tool and apparatus as well as cultural diversity among people and regions due to differences and changes in climate. Fermented milks, using milk as the raw material from bovine and non-bovine dairy species, and in some regions, especially in Asia and Africa, they also use materials from vegetable extracts. Some progress in Fermented Milk Science, has explained the role of such fermented foods for human health. These benefits have been more and more progressing to select specific microbes, known as probiotic cultures, which combined with specific substances from vegetable extract (prebiotic) could improve lactose digestion, role anti-cancer, anti-hypercholesteremic and anti-pathogenic bacteria as well as anti-virus were discussed in this article.

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