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Books on the topic 'Hyperbaric oxygenation; Oxygen therapy'

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Published: 10 December 2022
Last updated: 28 January 2023

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1

Morton, Walker, ed. Hyperbaric oxygen therapy. Garden City Park, N.Y: Avery Publishing Group, 1998.

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2

Harch, Paul. The oxygen revolution. New York: Hatherleigh Press, 2007.

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3

Shinomiya, Nariyoshi, and Yasufumi Asai, eds. Hyperbaric Oxygenation Therapy. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-13-7836-2.

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4

Mitton, Craig. Hyperbaric oxygen treatment in Alberta. Edmonton, Alta: Alberta Heritage Foundation for Medical Research : University of Calgary, 1998.

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5

Fischer, Bernd, Kewal K. Jain, Erwin Braun, and Siegfried Lehrl. Handbook of Hyperbaric Oxygen Therapy. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-72990-4.

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6

Oxygenation. Thorofare, NJ: Slack, 2001.

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7

Sheldon, Lisa Kennedy. Oxygenation. 2nd ed. Sudbury, MA: Jones and Bartlett Publishers, 2007.

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8

AIDS under pressure: Hyperbaric medicine in the management of HIV disease. Seattle: Hogrefe & Huber, 1997.

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9

Guli͡ar, S. A. Transport respiratornykh gazov pri adaptat͡sii cheloveka k giperbarii. Kiev: Nauk. dumka, 1988.

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10

Rutherford, Basham Kimberley A., ed. Essentials of oxygenation: Implication for clinical practice. Boston: Jones and Bartlett Publishers, 1993.

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11

Saunders, Patrick. Hyperbaric oxygen therapy in the management of carbon monoxide poisoning, osteoradionecrosis, burns, skin grafts and crush injury. Birmingham: University of Birmingham, Department of Public Health and Epidemiology, 2000.

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12

1939-, Fischer Bernd, ed. Handbook of hyperbaric oxygen therapy. Berlin: Springer-Verlag, 1988.

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13

S, Neuman Tom, and Thom Stephen R, eds. Physiology and medicine of hyperbaric oxygen therapy. Philadelphia: Saunders/Elsevier, 2008.

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14

N, Yanagita, and Nakashima T, eds. Hyperbaric oxygen therapy in otorhinolaryngology. Basel: Karger, 1998.

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15

Maxfield, William S. The oxygen cure: A complete guide to hyperbaric oxygen therapy. 2017.

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16

author, McCullough Virginia, and Duncan, William A., author of foreword, eds. The oxygen revolution: Hyperbaric oxygen therapy : breakthrough gene therapy for traumatic brain injury & other disorders. Hatherleigh Press, 2016.

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17

The Oxygen Revolution Hyperbaric Oxygen Therapy The Groundbreaking Treatment For Diabetes. Hatherleigh Press, 2010.

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18

S, McDonagh Marian, United States. Agency for Healthcare Research and Quality., and Oregon Health & Science University. Evidence-based Practice Center., eds. Hyperbaric oxygen therapy for brain injury, cerebral palsy, and stroke. Rockville, MD: U.S. Dept. of Health and Human Services, Public Health Service Agency for Healthcare Research and Quality, 2003.

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19

1932-, Davis Jefferson C., and Hunt Thomas K. 1930-, eds. Problem wounds: The role of oxygen. New York: Elsevier, 1988.

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20

David, Hailey, and Canadian Agency for Drugs and Technologies in Health., eds. Adjunctive hyperbaric oxygen therapy for diabetic foot ulcer: An economic analysis. Ottawa: Canadian Agency for Drugs and Technologies in Health, 2007.

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21

Oxygen to the Rescue: Oxygen Therapies and How They Help Overcome Disease, Promote Repair, and Improve Overall Function. Basic Health Publications, 2003.

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22

Walker, Dpm Morton, and Richard A. Neubauer. Hyperbaric Oxygen Therapy (Neubauer and Walker - Dr. Morton Walker Health Book). Avery, 2001.

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23

Hailey, David. Hyperbaric oxygen therapy: Recent findings on evidence for its effectiveness (Information paper). Alberta Heritage Foundation for Medical Research, 2003.

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24

Rivera, Tanya. PTSD and Traumatic Brain Injury among Servicemembers: Reviews of Medication Practices and Research on Hyperbaric Oxygen Therapy. Nova Science Publishers, Incorporated, 2016.

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25

Harch, Paul G., and Virginia Mccullough. The Oxygen Revolution: Hyperbaric Oxygen Therapy: The Groundbreaking New Treatment for Stroke, Alzheimer's, Parkinson's, Arthritis, Autism, Learning Disabilities and More. Hatherleigh Press, 2007.

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26

Stroke: Recovery with Oxygen. Best Publishing Company, 2005.

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27

Nakashima, T., and N. Yanagita, eds. Hyperbaric Oxygen Therapy in Otorhinolaryngology. S. Karger AG, 1998. http://dx.doi.org/10.1159/isbn.978-3-318-00248-5.

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28

Hyperbaric Oxygen Therapy Committee Report. Undersea & Hyperbaric Med Soc, 1996.

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29

Jain, K. K., E. Braun, B. Fischer, and S. Lehrl. Handbook of Hyperbaric Oxygen Therapy. Springer Berlin / Heidelberg, 1988.

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30

Shinomiya, Nariyoshi, and Yasufumi Asai. Hyperbaric Oxygenation Therapy: Molecular Mechanisms and Clinical Applications. Springer Singapore Pte. Limited, 2020.

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31

Shinomiya, Nariyoshi, and Yasufumi Asai. Hyperbaric Oxygenation Therapy: Molecular Mechanisms and Clinical Applications. Springer, 2019.

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32

Camporesi. Hyperbaric Oxygen Therapy: A Critical Review. Undersea & Hyperbaric Med Soc, 1991.

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33

Physiology and Medicine of Hyperbaric Oxygen Therapy. Elsevier, 2008. http://dx.doi.org/10.1016/b978-1-4160-3406-3.x5001-x.

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34

1908-, Petrovskiĭ B. V., Malinovskiĭ N. N, Akademii͡a︡ nauk SSSR. Sekt͡s︡ii͡a︡ khimiko-tekhnologicheskikh i biologicheskikh nauk., and Mezhvedomstvennyĭ nauchnyĭ sovet AN SSSR i AMN SSSR po fundamentalʹnym problemam medit͡s︡iny., eds. Giperbaricheskai͡a︡ oksigenat͡s︡ii͡a︡ i serdechno-sosudistai͡a︡ sistema. Moskva: "Nauka", 1987.

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35

Hyperbaric Oxygen Therapy: The Ultimate Beginner's Guide to Understanding the Hyperbaric Chamber. Createspace Independent Publishing Platform, 2014.

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36

Quick Look Nursing: Oxygenation (Quick Look Nursing) (Quick Look Nursing). 2nd ed. Jones and Bartlett Publishers, Inc., 2007.

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37

K, Reinhart, and Eyrich K, eds. Clinical aspects of O₂-transport and tissue oxygenation. Berlin: Springer-Verlag, 1989.

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38

Hyperbaric Oxygenation for Cerebral Palsy and the Brain Injured Child: A Promising Treatment. Best Publishing, 2003.

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39

The Proceedings of the 2nd International Symposium on Hyperbaric Oxygenation for Cerebral Palsy and the Brain-Injured Child. Best Publishing Company, 2002.

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40

Fullerton, James N., and Mervyn Singer. Oxygen in critical illness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0032.

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Oxygen therapy is primarily administered to alleviate arterial hypoxaemia and tissue hypoxia, and to facilitate aerobic cellular respiration. Hypoxaemia (PaO2 < 8 kPa [60 mmHg], SaO2 <92%) is associated with end-organ damage and adverse clinical outcomes, serving as a proxy measure for reduced intracellular PO2. Increasing the fraction of inspired oxygen should form part of an overall strategy to maximize tissue oxygen delivery. Permissive hypoxaemia represents a valid treatment strategy in a selected patient cohort. Oxygen is a drug and oxygen therapy is not benign, and oxygen administration at high, sustained doses (FiO2 >0.5, >12 hours) may cause oxygen toxicity. Observational studies in both mechanically-ventilated patients and survivors of non-traumatic cardiac arrest indicate an independent association between increasing hyperoxaemia and mortality. Oxygen therapy may additionally precipitate hypercapnic ventilatory failure in those at risk and oxygen should be administered to achieve a prescribed target SaO2 or PaO2 range, via adjustment of dose and delivery device. If no monitoring is available, hypoxaemia should be avoided by giving high-flow oxygen to achieve a FiO2 of near 1.0 with subsequent titration once oxygenation status is established.
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41

Wagner, Beth. Withdrawal of Respiratory Technology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190204709.003.0012.

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Respiratory failure can be defined as the inability of the lungs to provide adequate oxygenation or ventilation to sustain life. Respiratory failure can lead to abrupt clinical deterioration and is extremely distressing for patients and families. Advances in technology over the past decade have produced many life-sustaining therapies for patients with respiratory failure. Examples include high-flow oxygen therapy, invasive and noninvasive mechanically assisted breathing ventilation, prostacyclin therapy, and extracorporeal membrane oxygenation (ECMO). The care of these complex patients necessitates policies and procedures to assure quality care in withdrawal. Standardized protocols for withdrawal of life-sustaining respiratory therapies provide structured guidance, reduce variation in practice, and improve family and healthcare provider satisfaction.
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42

Annane, Djillali, and B. Jérôme Aboab. Management of carbon monoxide poisoning. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0328.

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CO poisoning is the commonest cause of toxic death. Carbon monoxide is colourless, odourless, and tasteless, and is produced under various conditions. When people inhale CO, the gas diffuses rapidly to the body and replaces oxygen at the level of haemoglobin, myoglobin, and other oxygen carriers. Subsequently, CO causes oxygen deprivation of all body tissues. CO also induces oxidative stress and systemic inflammation. After CO poisoning a broad variety of symptoms may occur. Survivors of CO poisoning often present with persistent neurological sequels or develop delayed neurological symptoms. There is poor correlation between carboxyhaemoglobin levels and clinical symptoms. The presence of coma, underlying co-morbid conditions and need for mechanical ventilation are the main prognostic factors. Management includes prompt extraction from the toxic environment and breathing 100% oxygen, although the role and practicalities of hyperbaric oxygen therapy remain controversial.
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43

Gattinon, Luciano, and Eleonora Carlesso. Acute respiratory failure and acute respiratory distress syndrome. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0064.

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Respiratory failure (RF) is defined as the acute or chronic impairment of respiratory system function to maintain normal oxygen and CO2 values when breathing room air. ‘Oxygenation failure’ occurs when O2 partial pressure (PaO2) value is lower than the normal predicted values for age and altitude and may be due to ventilation/perfusion mismatch or low oxygen concentration in the inspired air. In contrast, ‘ventilatory failure’ primarily involves CO2 elimination, with arterial CO2 partial pressure (PaCO2) higher than 45 mmHg. The most common causes are exacerbation of chronic obstructive pulmonary disease (COPD), asthma, and neuromuscular fatigue, leading to dyspnoea, tachypnoea, tachycardia, use of accessory muscles of respiration, and altered consciousness. History and arterial blood gas analysis is the easiest way to assess the nature of acute RF and treatment should solve the baseline pathology. In severe cases mechanical ventilation is necessary as a ‘buying time’ therapy. The acute hypoxemic RF arising from widespread diffuse injury to the alveolar-capillary membrane is termed Acute Respiratory Distress Syndrome (ARDS), which is the clinical and radiographic manifestation of acute pulmonary inflammatory states.
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44

Gattinon, Luciano, and Eleonora Carlesso. Acute respiratory failure and acute respiratory distress syndrome. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0064_update_001.

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Respiratory failure (RF) is defined as the acute or chronic impairment of respiratory system function to maintain normal oxygen and CO2 values when breathing room air. ‘Oxygenation failure’ occurs when O2 partial pressure (PaO2) value is lower than the normal predicted values for age and altitude and may be due to ventilation/perfusion mismatch or low oxygen concentration in the inspired air. In contrast, ‘ventilatory failure’ primarily involves CO2 elimination, with arterial CO2 partial pressure (PaCO2) higher than 45 mmHg. The most common causes are exacerbation of chronic obstructive pulmonary disease (COPD), asthma, and neuromuscular fatigue, leading to dyspnoea, tachypnoea, tachycardia, use of accessory muscles of respiration, and altered consciousness. History and arterial blood gas analysis is the easiest way to assess the nature of acute RF and treatment should solve the baseline pathology. In severe cases mechanical ventilation is necessary as a ‘buying time’ therapy. The acute hypoxemic RF arising from widespread diffuse injury to the alveolar-capillary membrane is termed Acute Respiratory Distress Syndrome (ARDS), which is the clinical and radiographic manifestation of acute pulmonary inflammatory states.
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45

Seyfried, Thomas N., and Laura M. Shelton. Metabolism-Based Treatments to Counter Cancer. Edited by Jong M. Rho. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0012.

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Accumulating evidence indicates that cancer is a type of mitochondrial metabolic disease. Chronic damage to mitochondria causes a gradual shift in cellular energy metabolism from respiration to fermentation. Consequently, fermentable metabolites become the drivers of cancer. Mitochondrial injury can explain the long-standing “oncogenic paradox,” and all major hallmarks of cancer including genomic instability. Restriction of fermentable fuels therefore becomes a viable therapeutic strategy for cancer management. The ketogenic diet (KD) is a metabolic therapy that lowers blood glucose and elevates blood ketone bodies. Ketone bodies are a “super fuel” for functional mitochondria, but cannot be metabolized efficiently by tumor mitochondria. The efficacy of KDs for cancer management can be enhanced when used together with drugs and procedures (such as hyperbaric oxygen therapy) (that further target fermentation. Therapeutic ketosis can represent an alternative, nontoxic strategy for managing and preventing a broad range of cancers while reducing healthcare costs.
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