Relevant bibliographies by topics / Hydrocephalus

Academic literature on the topic 'Hydrocephalus'

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Published: 4 June 2021
Last updated: 29 July 2024

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Journal articles on the topic "Hydrocephalus"

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Chaulagain, Dipak, Volodymyr Smolanka, and Andriy Smolanka. "Clinical Impact of the Patient Age Following Endoscopic Third Ventriculostomy: A Review." International Journal of Health Sciences and Research 11, no. 12 (December 20, 2021): 188–96. http://dx.doi.org/10.52403/ijhsr.20211225.

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Background: Clinically erratic cerebrospinal fluid (CSF) outflow within brain ventricles was described as non-communicating and communicating hydrocephalous. Neurosurgical patients are commonly seen with increased intracranial pressure due to increased CSF accumulation. (1) Initially scientists followed ventriculoperitoneal shunt an inexpensive and available technique to treat hydrocephalous disorders. Due to least successful outcomes, Scientists used other alternative endoscopic techniques. These endoscopic techniques include aqueductal endoscopic stenting, aqueductoplasty and third ventriculostomy (2). Defining success rate of Endoscopic third ventriculostomy clinical and radiographic analysis are considered gold standard. Objectives: Our review aims to assess comparative role of patient age, patient selection, hydrocephalus etiology, surgical advancement and epigenetic treatment strategies following endoscopic third ventriculostomy focusing hydrocephalous. The main objectives of current study, to critically analyze available literature till to date and a step forward for the development of standard surgical protocols. Methodology: Clinical studies from Level I-IV published in English language focusing human subject only were only considered by retrieving NCBI/PubMed, Medline databases. Studies purely focusing third ventriculostomy in subject of patient age, hydrocephalus etiology and age associated complications were processed further. Objective based data inspection approach was followed. From each included study focusing third ventriculostomy detailed information demographic information was collected. Further data analysis was done by using SAS and multiple tools of Microsoft Excel Version 2010. Outcomes: From total 11 included studies specifically focusing hydrocephaly treatment via endoscopic third ventriculostomy, 757 patients with equal 1:1 male and female gender ratio and 125 cases of unknown gender were considered. Current study highlighting efficacy of Endoscopic third ventriculostomy at the rate of 77% effective treating obstructive hydrocephalus. On basis of etiological concerns about 36.32 % cases hydrocephalous cases were linked with encephalitis, underweight birth and nonspecific etiology. However, 24 % cases of hydrocephalus were led by group of tumors including, Ependymoma, Medulloblastoma, Meningioma, Cerebella pontine angle Tumors and Pineal gland carcinomas as well. 154 (20%) cases of obstructive hydrocephalus were associated with aqueduct stenosis, 16% cases reporting hemorrhage and only 23 cyst cases. Conclusion and future recommendations: Endoscopic third ventriculostomy (ETV) is a safe and effective (77%) treatment option for the treatment of obstructive hydrocephalus among all age groups. However, specifically highest efficacy was noted among patients of 15-30-year age group. Understanding disease etiology and patient selection criteria both are considered potential components following successful endoscopic third ventriculostomy. We highly recommend further research following a universal age criteria and reporting results in distinct age category to standardize ETV treating hydrocephaly efficiently. Key words: Endoscopic third ventriculostomy, ETV, hydrocephalous, aqueductal endoscopic stenting, aqueductoplasty.
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Jones, H. C., H. K. Richards, R. M. Bucknall, and J. D. Pickard. "Local Cerebral Blood Flow in Rats with Congenital Hydrocephalus." Journal of Cerebral Blood Flow & Metabolism 13, no. 3 (May 1993): 531–34. http://dx.doi.org/10.1038/jcbfm.1993.69.

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Local cerebral blood flow (LCBF) has been measured by [14C]iodoantipyrine quantitative autoradiography in H-Tx rats with inherited congenital hydrocephalus at 10, 21, and 30 days after birth. LCBF at 10 days was uniformly low in all ten brain regions studied and not significantly different between hydrocephalic and age-matched control rats. By 21 days LCBF had increased significantly in control rats. LCBF was significantly lower (<69%) in the hydrocephalics in all cortical regions and in the inferior colliculus LCBF at both 21 and 30 days. The cerebellar cortex, pons, and caudate were not significantly affected. At 30 days LCBF ranged from 55 to 115 ml 100 g−1 min−1 for hydrocephalics and from 100 to 183 ml 100 g−1 min−1 for controls.
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Shapiro, Kenneth, Futoshi Takei, Arno Fried, and Ira Kohn. "Experimental feline hydrocephalus." Journal of Neurosurgery 63, no. 1 (July 1985): 82–87. http://dx.doi.org/10.3171/jns.1985.63.1.0082.

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✓ In a craniectomy-durectomy model of kaolin-induced feline hydrocephalus, the pressure-volume index (PVI) technique of bolus manipulations of cerebrospinal fluid (CSF) was used to study the biomechanical changes associated with hydrocephalus. Steady-state intracranial pressure (ICP), PVI, and the resistance to the absorption of CSF were determined acutely and 3 to 5 weeks later in hydrocephalic cats and time-matched control cats. Steady-state ICP was 11.0 ± 2.1 mm Hg (± standard deviation) in the hydrocephalic cats, compared to 10.8 ± 2.2 mm Hg in the chronic control group (p > 0.1). The ICP in both the chronic hydrocephalic and chronic control groups was significantly higher (p < 0.001) than after acute durectomy (mean ICP 8.5 ± 1.2 mm Hg). Immediately after dural opening, the mean PVI was 3.6 ± 0.2 ml (± standard error of the mean); over time, it decreased to 1.3 ± 0.1 ml in the chronic control group (p < 0.001), but remained elevated in the hydrocephalic group at 3.5 ± 0.4 ml (p < 0.001). Resistance to CSF absorption was 9.1 ± 1.4 mm Hg/ml/min immediately after dural opening and increased to 28.8 ± 4.5 mm Hg/ml/min (p < 0.001) in the hydrocephalic cats; it increased even further in the chronic measurements in control cats, to 82.3 ± 9.2 mm Hg/ml/min (p < 0.001). Ventricular size was moderate to severely enlarged in all hydrocephalic cats, and normal in the control group. These results indicate that the biomechanical profile of the altered brain container model of kaolin-induced feline hydrocephalus resembles that described in hydrocephalic infants. As shown in the control subjects, an absorptive defect alone is not sufficient to cause progressive ventricular enlargement. Increased volume-buffering capacity coupled with a moderate increase of CSF absorption resistance facilitates volume storage in the ventricles.
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da Silva, Marcia C., Shalom Michowicz, James M. Drake, Paul D. Chumas, and Ursula I. Tuor. "Reduced Local Cerebral Blood Flow in Periventricular White Matter in Experimental Neonatal Hydrocephalus—Restoration with CSF Shunting." Journal of Cerebral Blood Flow & Metabolism 15, no. 6 (November 1995): 1057–65. http://dx.doi.org/10.1038/jcbfm.1995.132.

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The extent to which the reduction in CBF occurring in hydrocephalus is a primary or secondary event in the pathogenesis of the brain injury that ensues has not been clearly established. This is particularly true in neonatal hydrocephalus, where the disorder is most common, and where timing of the treatment of the developing nervous system is so important. We investigated the changes in local CBF (lCBF) in an animal model of severe progressive neonatal hydrocephalus before and after CSF shunting. Hydrocephalus was induced in 27 1-week-old kittens by percutaneous injection of 0.05 ml of 25% kaolin into the cisterna magna. Fourteen littermates acted as controls. The lCBF was measured by 14C-iodoantipyrine quantitative autoradiography after 1 week in 15 animals (8 hydrocephalic, 7 controls) and after 3 weeks in 26 animals (19 hydrocephalic, 7 controls) following induction of hydrocephalus. Twelve of the 3-week hydrocephalic group received a ventriculoperitoneal shunt 10 days following kaolin injection. At 1 week following induction of hydrocephalus, lCBF was globally reduced in cortical gray matter and white matter as well as deep subcortical structures. The maximum reduction was in the parietal white matter, to 37% of control levels. At 3 weeks a significant reduction in lCBF persisted only in the white matter (parietal, occipital, and corpus callosum; average, 42% of control levels), whereas cortical gray and deep subcortical structures had returned to normal levels spontaneously. lCBF was normal in 3-week hydrocephalic shunted animals in all areas. CSF shunting restores the fall in lCBF in the periventricular white matter in this model. These findings are consistent with previous studies in the same model demonstrating derangement of high-energy phosphate metabolism and white matter anaerobic glycolysis with hydrocephalus and resolution with shunting.
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Garg, Aditi Jain, Rehana Najam, Ritika Agarwal, and Rajul Rastogi. "Evaluation of the incidence, risk factors, severity, and outcome of hydrocephalic fetuses in a tertiary healthcare center located in Northern India: a prospective observational study." International Journal of Reproduction, Contraception, Obstetrics and Gynecology 9, no. 2 (January 28, 2020): 595. http://dx.doi.org/10.18203/2320-1770.ijrcog20200343.

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Background: Hydrocephalus is a heterogeneous disease marked by abnormal dilatation of the cerebral ventricles secondary to varying etiologies. This study was aimed to determine the incidence, risk factors, severity, and outcome of hydrocephalic fetuses presenting to a tertiary healthcare hospital located in northern India.Methods: In this prospective observational study, pregnant women visiting the obstetrics outpatient department of our hospital from 01 July 2017 to 31 June 2018 were screened for hydrocephalic fetuses via ultrasonography along with a detailed history, and a comprehensive battery of diagnostic investigations. They were followed up for a minimum period of two months after delivery/termination of pregnancy.Results: A total of 3627 pregnant women were screened, of which 10 had hydrocephalic fetuses in the observed time period. The incidence of hydrocephalus was determined to be 2.75 per 1000 live births. Low socio-economic status was identified as a major risk factor. 50% of the hydrocephalic fetuses were severely afflicted and were discontinued. The remaining 50% were successfully delivered and were managed via a ventriculo-peritoneal shunt or are under close observation in the postnatal period without any adverse outcome.Conclusions: The burden of hydrocephalus is considerably high in India, as compared to western countries. In rural settings, low socioeconomic status and lack of folic acid supplementation have a major influence in the etiopathogenesis of hydrocephalus. Management of hydrocephalus requires a multidisciplinary approach and is tailored according to the severity of the presentation. Severe cases of hydrocephalus and cases with associated anomalies have a poor prognosis.
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Balafif, Fachriy, Muhammad Arifin Parenrengi, Wihasto Suryaningtyas, Dyah Fauziah, I. Ketut Sudiana, and Budi Utomo. "Aquaporin-4 expression related to hydrocephalus severity in hydrocephalus mice model." Bali Medical Journal 12, no. 3 (November 7, 2023): 3151–55. http://dx.doi.org/10.15562/bmj.v12i3.4742.

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Link of Video Abstract: https://youtu.be/h1rynQ_AAaY Background: Aquaporin-4 (AQP4), a water channel protein, is important in regulating brain water distribution. We hypothesized that increased expression of AQP4 in the kaolin-induced hydrocephalic mice brain is associated with the severity of hydrocephalus. This study aims to evaluate the AQP4 expression related to hydrocephalus severity in hydrocephalus mice model Methods: Hydrocephalus was induced in 8-10 weeks Sprague-Dawley mice by kaolin injection into cisterna magna. The mice were randomly divided into normal control and hydrocephalus groups and were sacrificed on days 7, 14, and 21 after kaolin induction. The brains were analyzed for AQP4 expression by histological and immunohistochemistry analysis. Data were analyzed using SPSS version 25.0 for Windows. Results: Histopathological analysis showed an increase in AQP4 expression in periventricular zone astrocytes with the duration of hydrocephalus (p < 0.001). A significant difference in AQP4 expression in this study was found in the hydrocephalus group between the 7th and 14th days (p = 0.023), 7th and 21st days (p < 0.001), and 14th and 21st days (p = 0.044) after kaolin induction. The highest expression of AQP4 was found in the hydrocephalus induction group on day 21. Conclusion: The results showed that the expression of AQP4 increased with the severity of hydrocephalus. Expression of AQP4 in the kaolin-induced hydrocephalic mice brain was significantly altered depending on the length of time after kaolin induction. Changes in AQP4 expression in periventricular zone astrocytes may be a compensatory mechanism resulting in drainage of CSF accumulation.
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McCarthy, David J., Dallas L. Sheinberg, Evan Luther, and Heather J. McCrea. "Myelomeningocele-associated hydrocephalus: nationwide analysis and systematic review." Neurosurgical Focus 47, no. 4 (October 2019): E5. http://dx.doi.org/10.3171/2019.7.focus19469.

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OBJECTIVEMyelomeningocele (MMC), the most severe form of spina bifida, is characterized by protrusion of the meninges and spinal cord through a defect in the vertebral arches. The management and prevention of MMC-associated hydrocephalus has evolved since its initial introduction with regard to treatment of MMC defect, MMC-associated hydrocephalus treatment modality, and timing of hydrocephalus treatment.METHODSThe Nationwide Inpatient Sample (NIS) database from the years 1998–2014 was reviewed and neonates with spina bifida and hydrocephalus status were identified. Timing of hydrocephalus treatment, delayed treatment (DT) versus simultaneous MMC repair with hydrocephalus treatment (ST), and treatment modality (ETV vs ventriculoperitoneal shunt [VPS]) were analyzed. Yearly trends were assessed with univariable logarithmic regression. Multivariable logistic regression identified correlates of inpatient shunt failure. A PRISMA systematic literature review was conducted that analyzed data from studies that investigated 1) MMC closure technique and hydrocephalus rate, 2) hydrocephalus treatment modality, and 3) timing of hydrocephalus treatment.RESULTSA weighted total of 10,627 inpatient MMC repairs were documented in the NIS, 8233 (77.5%) of which had documented hydrocephalus: 5876 (71.4%) were treated with VPS, 331 (4.0%) were treated with ETV, and 2026 (24.6%) remained untreated on initial inpatient stay. Treatment modality rates were stable over time; however, hydrocephalic patients in later years were less likely to receive hydrocephalus treatment during initial inpatient stay (odds ratio [OR] 0.974, p = 0.0331). The inpatient hydrocephalus treatment failure rate was higher for patients who received ETV treatment (17.5% ETV failure rate vs 7.9% VPS failure rate; p = 0.0028). Delayed hydrocephalus treatment was more prevalent in the later time period (77.9% vs 69.5%, p = 0.0287). Predictors of inpatient shunt failure included length of stay, shunt infection, jaundice, and delayed treatment. A longer time between operations increased the likelihood of inpatient shunt failure (OR 1.10, p < 0.0001). However, a meta-analysis of hydrocephalus timing studies revealed no difference between ST and DT with respect to shunt failure or infection rates.CONCLUSIONSFrom 1998 to 2014, hydrocephalus treatment has become more delayed and the number of hydrocephalic MMC patients not treated on initial inpatient stay has increased. Meta-analysis demonstrated that shunt malfunction and infection rates do not differ between delayed and simultaneous hydrocephalus treatment.
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Shokunbi, M. T., F. E. Olopade, O. M. Femi-Akinlosotu, and E. O. Ajiboye. "Pyramidal cell morphology and cell death in the hippocampus of adult mice with experimentally induced hydrocephalus." Nigerian Journal of Paediatrics 47, no. 4 (August 28, 2020): 298–304. http://dx.doi.org/10.4314/njp.v47i4.1.

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Background: Hippocampus is a neural structure in the temporal lobe that plays a crucial role in learning and memory. Cognitive impairment with learning disabilities is a common feature in hydrocephalus and is more prominent in adult-onset hydrocephalus. The aim of this study is to describethe morphological alterations in the pyramidal cells of the hippocampus of adult hydrocephalic mice. Method: Hydrocephalus was induced in adult albino mice by intra-cisternal injection of kaolin suspension (250 mg/ml in sterile water). They were sacrificed 7, 14 and 21 days post-induction. Morphological analysis was carried out on hematoxylin and eosinstained coronal sections of the hippocampus: the pyramidal neurons (normal and pyknotic) in the CA1 and CA3 subregions were counted and the pyknotic index (PI) was calculated. The somatic and dendritic features of Golgistained pyramidal neurons were examined by light microscopy in both hydrocephalic and control mice. Result: The PI was significantly greater in the CA1 region of the hippocampus in the hydrocephalic groups compared to the agematched controls. The dendritic processes of pyramidal neurons in the CA1 region were fewer with shorter terminal branches in the hydrocephalic mice than in controls; this was pronounced at 7 days post-induction. In the CA3 region, there was no difference in dendritic arborization between hydrocephalic and control mice. Conclusion: Acute adult-onset hydrocephalus was associated with increased pyknosis and reduced dendritic arborization in hippocampal pyramidal cells in the CA1 but not CA3 region. Keywords: Hippocampal pyramidal cell, Hydrocephalus, Pyknotic index, Golgi stain
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Italiya, Amit, Sunil Mhaske, and Liza Bulsara. "Hydrocephalus." Indian Journal of Trauma and Emergency Pediatrics 8, no. 2 (2016): 171–74. http://dx.doi.org/10.21088/ijtep.2348.9987.8216.26.

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Hwang, Yong Sup, Insop Shim, and Jin Woo Chang. "Anxiety responses and neurochemical changes in a kaolin-induced rat model of hydrocephalus." Journal of Neurosurgery: Pediatrics 7, no. 4 (April 2011): 401–7. http://dx.doi.org/10.3171/2011.1.peds10182.

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Object Hydrocephalus is a pathological enlargement of the ventricles of the brain, which can result from various diseases of the central nervous system. Patients with hydrocephalus frequently show motor abnormalities, such as abnormal gait and posture, as well as intellectual and emotional impairment. The present study was designed to investigate anxiety responses in rats with kaolin-induced hydrocephalus. Methods A total of 26 Sprague-Dawley rats were used for this study. Hydrocephalus was induced in 14 Sprague-Dawley rats by injecting 0.1 ml of 20% kaolin solution into the cisterna magna; 12 rats were administered the same volume of saline in the same fashion and served as controls. Seven of the rats that were injected with kaolin and 6 of the rats injected with saline were killed 3 days after injection (Group 1); the remaining rats were killed 4 weeks after injection (Group 2) to evaluate effects related to acute and chronic hydrocephalus. The rats were tested in an elevated plus maze after induction of hydrocephalus by kaolin injection. After the animals were killed, brain sections were immunostained for cholecystokinin and neuropeptide Y. In addition, tyrosine hydroxlyase immunoreactivity in the ventral tegmental area was evaluated by immunohistological staining. Results The rats with acute hydrocephalus showed decreased entry into and spent less time in the open arms of the elevated plus maze as compared with the control rats. The hydrocephalic rats had significantly more cholecystokinin-immunoreactive neurons and fewer neuropeptide Y–immunoreactive neurons in their brains. In addition, hydrocephalus progress in this model was positively correlated with the anxiety response. The numbers of tyrosine hydroxlyase–immunoreactive neurons were decreased significantly in the hydrocephalic rats as compared with the control rats. Conclusions These results suggest that the rat model of hydrocephalus is characterized by increased anxiety response and is associated with the functional impairment of the central dopamine system.
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Dissertations / Theses on the topic "Hydrocephalus"

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Noser-Isenschmid, Eva. "Hydrocephalus bei congenitaler Toxoplasmose /." [S.l : s.n.], 1987. http://www.ub.unibe.ch/content/bibliotheken_sammlungen/sondersammlungen/dissen_bestellformular/index_ger.html.

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Owler, Brian Kenneth. "Pathophysiology of normal pressure hydrocephalus." Thesis, The University of Sydney, 2004. http://hdl.handle.net/2123/685.

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Normal pressure hydrocephalus (NPH), a CSF circulation disorder, is important as a reversible cause of gait and cognitive disturbance in an aging population. The inconsistent response to CSF shunting is usually attributed to difficulties in differential diagnosis or co-morbidity. Improving outcome depends on an increased understanding of the pathophysiology of NPH. Specifically, this thesis examines the contribution of, and inter-relationship between, the brain parenchyma and CSF circulation in the pathophysiology of NPH. Of the four core studies of the thesis, the first quantifies the characteristics of the CSF circulation and parenchyma in NPH using CSF infusion studies to measure the resistance to CSF absorption and brain compliance. The second study assesses cerebral blood flow (CBF) was using O15-labelled positron emission tomography (PET) with MR co-registration. By performing CSF infusion studies in the PET scanner, CBF at baseline CSF pressure and at a higher equilibrium pressure is measured. Regional changes and autoregulatory capacity are assessed. The final study examines the microstructural integrity of the parenchyma using MR diffusion tensor imaging. These studies confirm the importance of the inter-relationship of the brain parenchyma and CSF circulation. NPH symptomatology and its relationship to the observed regional CBF reductions in the basal ganglia and thalamus are discussed. Regional CBF reductions with increased CSF pressure and the implications for autoregulatory capacity in NPH are considered. The reduction in CBF when CSF was increased was most striking in the periventricular regions. In addition, periventricular structures demonstrated increased diffusivity and decreased anisotropy. The relationship between these changes and mechanisms such as transependymal CSF passage are reviewed. The findings of this thesis support a role of both the CSF circulation and the brain parenchyma in the pathophysiology of NPH. The results have implications for the approach to the management of patients with NPH.
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Owler, Brian Kenneth. "Pathophysiology of normal pressure hydrocephalus." University of Sydney. Surgery, 2004. http://hdl.handle.net/2123/685.

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Normal pressure hydrocephalus (NPH), a CSF circulation disorder, is important as a reversible cause of gait and cognitive disturbance in an aging population. The inconsistent response to CSF shunting is usually attributed to difficulties in differential diagnosis or co-morbidity. Improving outcome depends on an increased understanding of the pathophysiology of NPH. Specifically, this thesis examines the contribution of, and inter-relationship between, the brain parenchyma and CSF circulation in the pathophysiology of NPH. Of the four core studies of the thesis, the first quantifies the characteristics of the CSF circulation and parenchyma in NPH using CSF infusion studies to measure the resistance to CSF absorption and brain compliance. The second study assesses cerebral blood flow (CBF) was using O15-labelled positron emission tomography (PET) with MR co-registration. By performing CSF infusion studies in the PET scanner, CBF at baseline CSF pressure and at a higher equilibrium pressure is measured. Regional changes and autoregulatory capacity are assessed. The final study examines the microstructural integrity of the parenchyma using MR diffusion tensor imaging. These studies confirm the importance of the inter-relationship of the brain parenchyma and CSF circulation. NPH symptomatology and its relationship to the observed regional CBF reductions in the basal ganglia and thalamus are discussed. Regional CBF reductions with increased CSF pressure and the implications for autoregulatory capacity in NPH are considered. The reduction in CBF when CSF was increased was most striking in the periventricular regions. In addition, periventricular structures demonstrated increased diffusivity and decreased anisotropy. The relationship between these changes and mechanisms such as transependymal CSF passage are reviewed. The findings of this thesis support a role of both the CSF circulation and the brain parenchyma in the pathophysiology of NPH. The results have implications for the approach to the management of patients with NPH.
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Lindquist, Barbro. "Hydrocephalus in children : cognition and behaviour /." Göteborg : Department of Pediatrics, Institute of Clinical Sciences, Sahlgrenska Academy at Göteborg University, 2007. http://hdl.handle.net/2077/2557.

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Persson, Eva-Karin. "Hydrocephalus in children : epidemiology and outcome /." Göteborg : Department of Pediatrics, Institute of Clinical Sciences, Sahlgrenska Academy at Göteborg University, 2007. http://hdl.handle.net/2077/2556.

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Rodgers, Jackie Michele. "The immunology of hydrocephalus shunt infections." Thesis, University College London (University of London), 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.246215.

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Manwaring, Preston K. "A provocative test to determine brain compliance in the management of patients with hydrocephalus /." Diss., CLICK HERE for online access, 2005. http://contentdm.lib.byu.edu/ETD/image/etd1103.pdf.

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Pauer, Anke. "Zytokine als prognostische Faktoren beim kindlichen Hydrocephalus." Doctoral thesis, Universitätsbibliothek Leipzig, 2013. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-110071.

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Wir untersuchten Liquor- und Serumproben von 40 an einem shuntversorgten Hydrocephalus erkrankten Kindern auf die Konzentration der Zytokine bFGF, TGF-β1, VEGF, IL-6, IGF-1 und Leptin sowie deren Korrelation mit dem Risiko von Shuntinsuffizienzen. Dabei konnten wir die Hypothese bestätigen, dass erhöhte Konzentration der fibrogenen Zytokine bFGF und TGF-β1 im Serum bzw. Liquor mit einem erhöhten Risiko für operationspflichtige Shuntinsuffizienzen durch Obstruktion des Schlauchsystems einhergehen, und dass diese Komplikationen mit steigenden Zytokinkonzentrationen umso eher eintreten. Außerdem war bFGF im Liquor von Kindern, die zum Abnahmezeitpunkt an einer Shuntdysfunktion durch Obstruktion oder Einwachsen des Shunts litten, signifikant höher als bei Kindern, die zum Zeitpunkt der Abnahme keine Shuntdysfunktion aus eben genannten Gründen hatten. Des Weiteren fanden wir Konzentrationsunterschiede für IL-6 im Liquor zwischen den einzelnen Ursachen der Erkrankung, wobei das Zytokin am höchsten bei Tumorpatienten war, gefolgt von posthämorrhagischem und postmeningitischem Hydrocephalus, und am niedrigsten bei Kindern mit kongenitaler ZNS-Fehlbildung.
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Jouet, Monique Marie Helene. "The molecular genetics of X-linked hydrocephalus." Thesis, Open University, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.295639.

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Gaston, Hannah. "Ophthalmic complications of spina bifida and hydrocephalus." Thesis, University of Southampton, 1986. https://eprints.soton.ac.uk/209759/.

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This thesis represents an attempt to further our knowledge of the ophthalmic complications of spina bifida and hydrocephalus by means of literature review and a long term clinical study, and to determine whether regular ophthalmic supervision can assist in the general management of affected children. The ophthalmic complications of spina bifida have often been reported in the literature and thought to merit regular supervision of affected children, yet few centres currently offer this service. In this study 322 children attending one regional centre were examined repeatedly over a six year period by one ophthalmologist. Ophthalmic complications were found to be very common. They frequently provided evidence of raised intracranial pressure due to shunt dysfunction even when other objective evidence was lacking. Every spina bifida and hydrocephalus clinic should have an ophthalmalogist in its medical team. Preservation of visual function and early diagnosis of raised intracranial pressure in these children should result from this arrangement.
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Books on the topic "Hydrocephalus"

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Aygok, Gunes A., and Harold L. Rekate, eds. Hydrocephalus. Vienna: Springer Vienna, 2012. http://dx.doi.org/10.1007/978-3-7091-0923-6.

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Matsumoto, Satoshi, and Norihiko Tamaki, eds. Hydrocephalus. Tokyo: Springer Japan, 1991. http://dx.doi.org/10.1007/978-4-431-68156-4.

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Ammar, Ahmed, ed. Hydrocephalus. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-61304-8.

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H, Schurr Peter, and Polkey C. E, eds. Hydrocephalus. Oxford: Oxford University Press, 1993.

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National Institutes of Health (U.S.), ed. Hydrocephalus. Bethesda, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1999.

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Michael, Scott R., and Congress of Neurological Surgeons, eds. Hydrocephalus. Baltimore: Williams & Wilkins, 1990.

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Cinalli, Giuseppe, M. Memet Ozek, and Christian Sainte-Rose, eds. Pediatric Hydrocephalus. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-31889-9.

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Rigamonti, Daniele, ed. Adult Hydrocephalus. Cambridge: Cambridge University Press, 2014. http://dx.doi.org/10.1017/cbo9781139382816.

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Cinalli, Giuseppe, Christian Sainte-Rose, and Wirginia June Maixner, eds. Pediatric Hydrocephalus. Milano: Springer Milan, 2005. http://dx.doi.org/10.1007/978-88-470-2121-1.

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Bayston, Roger. Hydrocephalus shunt infections. London: Chapman and Hall, 1989.

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Book chapters on the topic "Hydrocephalus"

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Valk, Jacob, and Marjo S. van der Knaap. "Hydrocephalus." In Magnetic Resonance of Myelin, Myelination, and Myelin Disorders, 309–18. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-662-02568-0_48.

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Weis, Serge, Michael Sonnberger, Andreas Dunzinger, Eva Voglmayr, Martin Aichholzer, Raimund Kleiser, and Peter Strasser. "Hydrocephalus." In Imaging Brain Diseases, 443–52. Vienna: Springer Vienna, 2019. http://dx.doi.org/10.1007/978-3-7091-1544-2_16.

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Igbaseimokumo, Usiakimi. "Hydrocephalus." In Brain CT Scans in Clinical Practice, 73–88. London: Springer London, 2009. http://dx.doi.org/10.1007/b98343_4.

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Ellenbogen, Jonathan R., J. Kandasamy, and Conor L. Mallucci. "Hydrocephalus." In Pediatric Surgery, 1237–55. Berlin, Heidelberg: Springer Berlin Heidelberg, 2020. http://dx.doi.org/10.1007/978-3-662-43588-5_88.

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Tye, Gary, and John Brown. "Hydrocephalus." In Encyclopedia of Clinical Neuropsychology, 1749–52. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_324.

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Ellenbogen, J. R., J. Kandasamy, and C. Mallucci. "Hydrocephalus." In Pediatric Surgery, 1–19. Berlin, Heidelberg: Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-642-38482-0_88-1.

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Ellenbogen, J. R., J. Kandasamy, and C. Mallucci. "Hydrocephalus." In Pediatric Surgery, 1–19. Berlin, Heidelberg: Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-642-38482-0_88-2.

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Ross, Jerard, and Conor Mallucci. "Hydrocephalus." In Pediatric Surgery, 775–87. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-69560-8_80.

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Nielsen, Nadine, and Amanda Breedt. "Hydrocephalus." In Nursing Care of the Pediatric Neurosurgery Patient, 39–89. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-49319-0_2.

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Avellino, Anthony M. "Hydrocephalus." In Emergency Approaches to Neurosurgical Conditions, 71–78. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-10693-9_6.

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Conference papers on the topic "Hydrocephalus"

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Pattison, Adam J., S. Scott Lollis, Phillip R. Perrinez, John B. Weaver, and Keith D. Paulsen. "MR elastography of hydrocephalus." In SPIE Medical Imaging, edited by Xiaoping P. Hu and Anne V. Clough. SPIE, 2009. http://dx.doi.org/10.1117/12.811820.

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Lodhi, W., and S. Shepherd. "Coccidiodal Meningitis with Hydrocephalus." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a7003.

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Joswig, Jurgen, Jens Oswald, and Steffen Seifert. "Biodriven microsystem for treatment of hydrocephalus." In Micromachining and Microfabrication, edited by Ray M. Roop and Kevin H. Chau. SPIE, 1995. http://dx.doi.org/10.1117/12.221180.

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Kozodoy-Pins, Rebecca L., James A. Harrington, George A. Zazanis, Michael G. Nosko, and Richard M. Lehman. "Stereotactic CO2 laser therapy for hydrocephalus." In OE/LASE '94, edited by Leonard J. Cerullo, Kenneth S. Heiferman, Hong Liu, Halina Podbielska, Abund O. Wist, and Lucia J. Zamorano. SPIE, 1994. http://dx.doi.org/10.1117/12.176597.

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Smith, Joshua H., and Jose Jaime García. "A Nonlinear Biphasic Hyperelastic Model for Acute Hydrocephalus." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192865.

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The cerebrospinal fluid present in the central nervous system plays an important role in the physiological activities and protection of the brain. Disruptions of CSF flow lead to different forms of a disease known as hydrocephalus, characterized by a significant increment of the ventricular space. In acute hydrocephalus the Sylvius aqueduct is blocked and ventricular pressure is greatly increased.
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Guha, Subhajit, Indranil Ghosh, and Bibhukalyani Das. "Hydrocephalus Secondary to Cervical Spinal Cord Surgery." In Abstracts of 21st Annual Conference of the Indian Society of Neuroanaesthesiology and Critical Care (ISNACC 2020). Thieme Medical and Scientific Publishers Private Ltd., 2020. http://dx.doi.org/10.1055/s-0040-1709586.

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Wallmeier, Julia, Diana Frank, Oliver Schwarz, Barbara Fiedler, Angela Brentrup, and Heymut Omran. "FV 1177. Genetic Causes of Congenital Hydrocephalus." In Abstracts of the 44th Annual Meeting of the Society for Neuropediatrics. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1675916.

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Wijayanti, Erna, and Idam Arif. "Simulation of hydrocephalus condition in infant head." In 4TH INTERNATIONAL CONFERENCE ON MATHEMATICS AND NATURAL SCIENCES (ICMNS 2012): Science for Health, Food and Sustainable Energy. AIP Publishing LLC, 2014. http://dx.doi.org/10.1063/1.4868834.

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Narayanaswamy, A., M. Nourani, L. Tamil, and S. Bianco. "A wireless monitoring system for Hydrocephalus shunts." In 2015 37th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2015. http://dx.doi.org/10.1109/embc.2015.7318490.

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Jetzki, S., and S. Leonhardt. "An electronic implant for Hydrocephalus therapy assistance." In 2008 30th Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2008. http://dx.doi.org/10.1109/iembs.2008.4649252.

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Reports on the topic "Hydrocephalus"

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Li, Tao, Handong Wang, Haichen Sun, Chaochao Gao, Yong-qiang Li, and Meng-liang Zhou. Risk factors for posttraumatic hydrocephalus: A systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review Protocols, April 2020. http://dx.doi.org/10.37766/inplasy2020.4.0180.

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Sun, Tong, Jingguo Yang, Yikai Yuan, Chao You, and Junwen Guan. Shunting outcomes in posthemorrhagic hydrocephalus: A systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2020. http://dx.doi.org/10.37766/inplasy2020.6.0063.

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Xu, Dongsheng. The Assessment of Cognitive Function Determines the Time of Operation of Hydrocephalus Drainage in Severe TBI. Science Repository OÜ, January 2019. http://dx.doi.org/10.31487/j.scr.2019.01.001.

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liu, xinxin, xueyan han, yiguo wang, fengxing zhong, xiuping yin, and qiming zhang. Acupuncture as a complementary therapy for hydrocephalus in children: a protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2022. http://dx.doi.org/10.37766/inplasy2022.4.0169.

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Treadwell, Jonathan R., Mingche Wu, and Amy Y. Tsou. Management of Infantile Epilepsies. Agency for Healthcare Research and Quality (AHRQ), October 2022. http://dx.doi.org/10.23970/ahrqepccer252.

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Objectives. Uncontrolled seizures in children 1 to 36 months old have serious short-term health risks and may be associated with substantial developmental, behavioral, and psychological impairments. We evaluated the effectiveness, comparative effectiveness, and harms of pharmacologic, dietary, surgical, neuromodulation, and gene therapy treatments for infantile epilepsies. Data sources. We searched Embase®, MEDLINE®, PubMed®, the Cochrane Library, and gray literature for studies published from January 1, 1999, to August 19, 2021. Review methods. Using standard Evidence-based Practice Center methods, we refined the scope and applied a priori inclusion criteria to the >10,000 articles identified. We ordered full text of any pediatric epilepsy articles to determine if they reported any data on those age 1 month to <36 months. We extracted key information from each included study, rated risk of bias, and rated the strength of evidence. We summarized the studies and outcomes narratively. Results. Forty-one studies (44 articles) met inclusion criteria. For pharmacotherapy, levetiracetam may cause seizure freedom in some patients (strength of evidence [SOE]: low), but data on other medications (topiramate, lamotrigine, phenytoin, vigabatrin, rufinamide, stiripentol) were insufficient to permit conclusions. Both ketogenic diet and the modified Atkins diet may reduce seizure frequency (SOE: low for both). In addition, the ketogenic diet may cause seizure freedom in some infants (SOE: low) and may be more likely than the modified Atkins diet to reduce seizure frequency (SOE: low). Both hemispherectomy/hemispherotomy and non-hemispheric surgical procedures may cause seizure freedom in some infants (SOE: low for both), but the precise proportion is too variable to estimate. For three medications (levetiracetam, topiramate, and lamotrigine), adverse effects may rarely be severe enough to warrant discontinuation (SOE: low). For topiramate, non-severe adverse effects include loss of appetite and upper respiratory tract infection (SOE: moderate). Harms of diets were sparsely reported. For surgical interventions, surgical mortality is rare for functional hemispherectomy/hemispherotomy and non-hemispheric procedures (SOE: low), but evidence was insufficient to permit quantitative estimates of mortality or morbidity risk. Hydrocephalus requiring shunt placement after multilobar, lobar, or focal resection is uncommon (SOE: low). No studies assessed neuromodulation or gene therapy. Conclusions. Levetiracetam, ketogenic diet, modified Atkins diet, and surgery all appear to be effective for some infants. However, the strength of the evidence is low for all of these modalities due to lack of control groups, low patient enrollment, and inconsistent reporting. Future studies should compare different pharmacologic treatments and compare pharmacotherapy with dietary therapy. Critical outcomes underrepresented in the literature include quality of life, sleep outcomes, and long-term development.
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Fewer infections with antibiotic-impregnated shunts for hydrocephalus. National Institute for Health Research, January 2020. http://dx.doi.org/10.3310/signal-000863.

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