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1

Jariwala, Krishna B., and Prof Jaimeel Shah. "Survey of Detecting Heartbeats, Temperature and ECG of Human Body using IOT." International Journal of Trend in Scientific Research and Development Volume-2, Issue-5 (August 31, 2018): 2457–61. http://dx.doi.org/10.31142/ijtsrd17153.

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2

Nazarenko, V. I., V. G. Martirosova, I. M. Cherednichenko, N. S. Tikhonova, and O. Y. Beseda. "Combined effect of lighting and high air temperature on human visual performance." Ukrainian Journal of Occupational Health 2019, no. 2 (June 27, 2019): 102–9. http://dx.doi.org/10.33573/ujoh2019.02.102.

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3

Aysa, Noor Hadi. "Elastic Properties of Undegradable Nanocomposites at Human Body Temperature Using as Prosthetics." NeuroQuantology 18, no. 1 (January 30, 2020): 32–36. http://dx.doi.org/10.14704/nq.2020.18.1.nq20104.

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4

Husain, Muhammad Dawood, Shenela Naqvi, Ozgur Atalay, Syed Talha Ali Hamdani, and Richard Kennon. "Measuring Human Body Temperature through Temperature Sensing Fabric." AATCC Journal of Research 3, no. 4 (July 1, 2016): 1–12. http://dx.doi.org/10.14504/ajr.3.4.1.

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5

Kuzubasoglu, Burcu Arman, Ersin Sayar, Cedric Cochrane, Vladan Koncar, and Senem Kursun Bahadir. "Wearable temperature sensor for human body temperature detection." Journal of Materials Science: Materials in Electronics 32, no. 4 (January 11, 2021): 4784–97. http://dx.doi.org/10.1007/s10854-020-05217-2.

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6

Santariová, M., L. Pinc, L. Bartoš, P. Vyplelová, J. Gerneš, and V. Sekyrová. "Resistance of human odours to extremely high temperature as revealed by trained dogs." Czech Journal of Animal Science 61, No. 4 (July 15, 2016): 172–76. http://dx.doi.org/10.17221/8848-cjas.

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7

NAKATANI, Kaoru, Tatsuaki FURUMOTO, Takashi UEDA, Akira HOSOKAWA, and Ryutaro TANAKA. "3373 Study on Temperature Measurement of Human Enamel by Er:YAG Laser Irradiation : The Influence of Surface Temperature on the Dental Pulp." Proceedings of International Conference on Leading Edge Manufacturing in 21st century : LEM21 2011.6 (2011): _3373–1_—_3373–4_. http://dx.doi.org/10.1299/jsmelem.2011.6._3373-1_.

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8

Lakie, M., E. G. Walsh, L. A. Arblaster, F. Villagra, and R. C. Roberts. "Limb temperature and human tremors." Journal of Neurology, Neurosurgery & Psychiatry 57, no. 1 (January 1, 1994): 35–42. http://dx.doi.org/10.1136/jnnp.57.1.35.

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9

Anderson, G. S. "Human morphology and temperature regulation." International Journal of Biometeorology 43, no. 3 (November 29, 1999): 99–109. http://dx.doi.org/10.1007/s004840050123.

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10

Strigo, Irina A., Franco Carli, and M. Catherine Bushnell. "Effect of Ambient Temperature on Human Pain and Temperature Perception." Anesthesiology 92, no. 3 (March 1, 2000): 699–707. http://dx.doi.org/10.1097/00000542-200003000-00014.

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Background Animal studies show reduced nociceptive responses to noxious heat stimuli and increases in endogenous beta-endorphin levels in cold environments, suggesting that human pain perception may be dependent on ambient temperature. However, studies of changes in local skin temperature on human pain perception have yielded variable results. This study examines the effect of both warm and cool ambient temperature on the perception of noxious and innocuous mechanical and thermal stimuli. Methods Ten subjects (7 men and 3 women, aged 20-23 yr) used visual analog scales to rate the stimulus intensity, pain intensity, and unpleasantness of thermal (0-50 degrees C) and mechanical (1.2-28.9 g) stimuli applied on the volar forearm with a 1-cm2 contact thermode and von Frey filaments, respectively. Mean skin temperatures were measured throughout the experiment by infrared pyrometer. Each subject was tested in ambient temperatures of 15 degrees C (cool), 25 degrees C (neutral), and 35 degrees C (warm) on separate days, after a 30-min acclimation to the environment. Studies began in the morning after an 8-h fast. Results Mean skin temperature was altered by ambient temperature (cool room: 30.1 degrees C; neutral room: 33.4 degrees C; warm room: 34.5 degrees C; P < 0.0001). Ambient temperature affected both heat (44-50 degrees C) and cold (25-0 degrees C) perception (P < 0.01). Stimulus intensity ratings tended to be lower in the cool than in the neutral environment (P < 0.07) but were not different between the neutral and warm environments. Unpleasantness ratings revealed that cold stimuli were more unpleasant than hot stimuli in the cool room and that noxious heat stimuli were more unpleasant in a warm environment. Environmental temperature did not alter ratings of warm (37 and 40 degrees C) or mechanical stimuli. Conclusions These results indicate that, in humans, a decrease in skin temperature following exposure to cool environments reduces thermal pain. Suppression of Adelta primary afferent cold fiber activity has been shown to increase cold pain produced by skin cooling. Our current findings may represent the reverse phenomenon, i.e., a reduction in thermal nociceptive transmission by the activation of Adelta cutaneous cold fibers.
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11

Wang, Lijuan, Minzhou Chen, and Zefeng Chen. "Local thermal discomfort in low temperature environments." Thermal Science 23, no. 4 (2019): 2211–18. http://dx.doi.org/10.2298/tsci1904211w.

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Human local parts have different thermal responses to low temperature environment. The objective of this paper is to find out the most sensitive parts which are extremely discomforting in low temperature environments. Based on previous experimental data, the relationship among skin temperature, air temperature, and clothing insulation was fitted, and the neutral skin temperatures were obtained. The local skin temperatures at different parts of the human body were compared with neutral skin temperatures in different air temperatures and clothes. The results showed that the local parts of foot, hand, upper arm, and calf deviated far from the neutral condition and were selected as the principal parts to be warmed. The findings are significant to improve human local thermal discomfort.
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12

Shiraki, K., S. Sagawa, F. Tajima, A. Yokota, M. Hashimoto, and G. L. Brengelmann. "Independence of brain and tympanic temperatures in an unanesthetized human." Journal of Applied Physiology 65, no. 1 (July 1, 1988): 482–86. http://dx.doi.org/10.1152/jappl.1988.65.1.482.

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Temperature within the brain and the esophagus and at the tympanum were obtained in a 12-yr-old male in a series of experiments that began 8 days after surgery for implantation of a drainage catheter. Fanning the face did reduce tympanic temperature but not temperature in the brain; brain temperatures followed esophageal temperatures. In long-term monitoring, temperature in the lateral ventricle was 0.5 degree C above esophageal temperature and 0.2 degree C below that in white matter 1 cm above, with the offsets fixed throughout the overnight cycle. All temperatures went through similar excursions when the face was excluded from fanning applied to the body. These observations highlight the fact that in humans the defense against hyperthermia takes advantage of cooling distributed over the entire skin surface.
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13

Qian, Cheng, and Xuebin Zhang. "Changes in Temperature Seasonality in China: Human Influences and Internal Variability." Journal of Climate 32, no. 19 (August 26, 2019): 6237–49. http://dx.doi.org/10.1175/jcli-d-19-0081.1.

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Abstract Temperature seasonality, the difference between summer and winter temperatures in mid–high latitudes, is an important component of the climate. Whether humans have had detectable influences on changing surface temperature seasonality at scales smaller than the subcontinental scale, where humans are directly impacted, is not clear. In this study, the first detection and attribution analysis of changes in temperature seasonality in China has been carried out. Detection and attribution of both summer and winter temperatures were also conducted, with careful consideration of observational uncertainty and the inconsistency between observation and model simulations induced by the long coastline and country border in China. The results show that the response to external forcings is robustly detectable in the spatiotemporal pattern of weakening seasonality and in that of warming winter temperature, although models may have underestimated the observed changes. The response to external forcings is detectable and consistent with the observed change in summer temperature averaged over China. Human influences are detectable in changes in seasonality and summer and winter temperatures, most robustly in winter, and these influences can be separated from those of natural forcing when averaged over China. The recent increase in summer temperature was found to be due to external forcings, and the warming hiatus in winter temperature from 1998 to 2013 was due to a statistically significant cooling trend induced by internal variability. These results will give insights into the understanding of the warming hiatus in China, as well as the hot summers and cold winters in recent years.
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14

Goncharevskyi, S., and V. Martynyuk. "Daily temperature dynamics of human skin representative areas." Bulletin of Taras Shevchenko National University of Kyiv. Series: Problems of Physiological Functions Regulation 21, no. 2 (2016): 86–91. http://dx.doi.org/10.17721/2616_6410.2016.21.86-91.

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The main aim of our research was to study the temperature variation of representative are a soft the cranial part of the autonomic nervous system of the human skin during the day. The temperature of representative are a soft the thoracic autonomic nervous system we measured by infrared thermometer (Medisana FTO D-53340, with anaccuracy of 0.1 degree Celsius). During the study identified minimums and maximums temperatures for representative are as during the day: the hypothalamus – 13 (maximum), 3 (minimum) an hour, midbrain – 15 (maximum), 5 (minimum) an hour, pons- not found, the medulla oblongata – 9, 15 (maximum), 3.21 (minimum) an hour, the vagus nerve (right side) – 15 (maximum), 5 (at least) an hour, the vagus nerve (left side) – 15 (maximum), 21 (minimum) an hour. The presence of minimums and maximums temperature in representative areas indicates different activity related to their brain structures.
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15

Silaban, Saronom, Murniaty Simorangkir, Shabarni Gaffar, Iman Permana Maksum, and Toto Subroto. "Temperature effect on expression of recombinant human prethrombin-2 in Escherichia coli BL21(DE3) ArcticExpress." Jurnal Pendidikan Kimia 11, no. 3 (December 7, 2019): 122–28. http://dx.doi.org/10.24114/jpkim.v11i3.15779.

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16

Nakamura, Masahiro, Norio Nonomura, Mikio Namiki, Akihiko Okuyama, Eitetsu Koh, Nobuyuki Kondoh, Masami Takeyama, Hisakazu Kiyohara, and Hideki Fujioka. "TEMPERATURE INFLUENCE ON HUMAN TESTICULAR FUNCTION." Japanese Journal of Urology 80, no. 9 (1989): 1362–66. http://dx.doi.org/10.5980/jpnjurol1989.80.1362.

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17

Guest, Steve, Fabian Grabenhorst, Greg Essick, Yasheng Chen, Mike Young, Francis McGlone, Ivan de Araujo, and Edmund T. Rolls. "Human cortical representation of oral temperature." Physiology & Behavior 92, no. 5 (December 2007): 975–84. http://dx.doi.org/10.1016/j.physbeh.2007.07.004.

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18

Savastano, David M., Alexander M. Gorbach, Henry S. Eden, Sheila M. Brady, James C. Reynolds, and Jack A. Yanovski. "Adiposity and human regional body temperature." American Journal of Clinical Nutrition 90, no. 5 (September 9, 2009): 1124–31. http://dx.doi.org/10.3945/ajcn.2009.27567.

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19

Garg, Teevrat, Maulik Jagnani, and Vis Taraz. "Temperature and Human Capital in India." Journal of the Association of Environmental and Resource Economists 7, no. 6 (November 1, 2020): 1113–50. http://dx.doi.org/10.1086/710066.

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20

Santer, B. D., J. F. Painter, C. A. Mears, C. Doutriaux, P. Caldwell, J. M. Arblaster, P. J. Cameron-Smith, et al. "Identifying human influences on atmospheric temperature." Proceedings of the National Academy of Sciences 110, no. 1 (November 29, 2012): 26–33. http://dx.doi.org/10.1073/pnas.1210514109.

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21

Zivin, Joshua Graff, and Jeffrey Shrader. "Temperature Extremes, Health, and Human Capital." Future of Children 26, no. 1 (2016): 31–50. http://dx.doi.org/10.1353/foc.2016.0002.

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22

Guslisty, A. A., N. P. Malomuzh, and A. I. Fisenko. "Optimal Temperature for Human Life Activity." Ukrainian Journal of Physics 63, no. 9 (September 24, 2018): 809. http://dx.doi.org/10.15407/ujpe63.9.809.

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The optimal temperature for the human life activity has been determined, by assuming that this parameter corresponds to the most intensive oxygen transport in arteries and the most intensive chemical reactions in the cells. The oxygen transport is found to be mainly governed by the blood saturation with oxygen and the blood plasma viscosity, with the both parameters depending on the temperature and the acid-base balance in blood. Additional parameters affecting the erythrocyte volume and, accordingly, the temperature of the most intensive oxygen transport are also taken into account. Erythrocytes are assumed to affect the shear viscosity of blood in the same way, as impurity particles change the suspension viscosity. It is shown that theoptimal temperature equals 36.6 ∘C under normal environmental conditions. The dependence of the optimal temperature for the human life activity on the acid-base index is discussed.
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23

Parmala, Mikael, Mikael Eriksson, Maria Rytioja, Jukka Tanttu, and Max Köhler. "Temperature measurement in human fat withT2imaging." Journal of Magnetic Resonance Imaging 43, no. 5 (October 5, 2015): 1171–78. http://dx.doi.org/10.1002/jmri.25064.

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24

Maurer-Spurej, Elisabeth, Gisela Pfeiler, Norbert Maurer, Helmut Lindner, Otto Glatter, and Dana V. Devine. "Room Temperature Activates Human Blood Platelets." Laboratory Investigation 81, no. 4 (April 2001): 581–92. http://dx.doi.org/10.1038/labinvest.3780267.

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25

Desai, Mehul J. "Intervertebral Disc Temperature Mapping During Disc Biacuplasty in the Human Cadaver." Pain Physician 2;18, no. 2;3 (March 14, 2015): E217—E223. http://dx.doi.org/10.36076/ppj/2015.18.e217.

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Background: Intradiscal biacuplasty (IDB) is a novel heating therapy using cooled radiofrequency (RF), which may offer relief for discogenic pain. Effective neuroablation may be achieved intradiscally at higher lesion temperatures. The safety of intradiscal heating at elevated temperatures using cooled RF has never been reported. Objective: The purpose of this study is to map the intradiscal and peridiscal temperatures when IDB is performed at increased temperature using a modified lesion approach. The resulting temperature profiles are used to assess the safety and theoretical efficacy of this approach to ablate nociceptors in the posterior annulus. Study Design: Research article. Methods: Eleven lumbar discs in a non-perfused human cadaver were treated by IDB. Temperature profiles in the disc during bipolar lesion at 50°C followed by 2 monopolar lesions at 60°C were mapped using custom thermocouples. Temperatures inside the disc, at the nerve roots, and in the midline ventral epidural space were monitored in real-time using a data-collection system with custom RF filters. Setting: Human research laboratory. Results: Higher maximum temperature was reached intradiscally, and a larger volume of tissue was exposed to neuroablative temperature (> 45°C). Temperature at the nerve roots and in the epidural space increased by 2.4°C ± 2.6°C and 4.9°C ± 1.9°C (mean ± SD), respectively, during bipolar lesion. Similarly, temperature increased by 2.2°C ± 1.9°C and 0.8°C ± 1.3°C at the nerve roots and in the epidural space, respectively, during monopolar lesion. Limitations: Limitations include the ex vivo setting which lacks perfusion and may not reproduce in vivo conditions such as cerebrospinal fluid dynamics. Conclusions: The modified treatment paradigm showed intradiscal heating is achieved and is concentrated in the posterior annulus, suggesting minimal risk of thermal damage to the neighboring neural structures. Clinical benefits should be evaluated. Key words: Spine, biacuplasty, thermal, disc, intervertebral
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Kelemen, Christina, Shu Chien, and G. M. Artmann. "Temperature Transition of Human Hemoglobin at Body Temperature: Effects of Calcium." Biophysical Journal 80, no. 6 (June 2001): 2622–30. http://dx.doi.org/10.1016/s0006-3495(01)76232-7.

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&NA;. "▪ Effect of Ambient Temperature on Human Pain and Temperature Perception." Obstetric Anesthesia Digest 20, no. 3 (September 2000): 118–19. http://dx.doi.org/10.1097/00132582-200020030-00017.

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28

Absolonová, Karolína, Miluše  Dobisíková, Michal  Beran, Jarmila  Zocová, and Petr  Velemínský. "The temperature of cremation and its effect on the microstructure of the human rib compact bone." Anthropologischer Anzeiger 69, no. 4 (September 1, 2012): 439–60. http://dx.doi.org/10.1127/0003-5548/2012/0213.

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29

Kohshi, K., and N. Konda. "Human auditory brain stem response during induced hyperthermia." Journal of Applied Physiology 69, no. 4 (October 1, 1990): 1419–22. http://dx.doi.org/10.1152/jappl.1990.69.4.1419.

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A continuous monitoring of auditory brain stem response (ABR) and esophageal (Tes) and rectal temperatures (Tre) were recorded in male undergraduate subjects to investigate a relationship between the interpeak latencies (IPLs) and core temperature. The average change of Tes (36.8-39.5 degrees C) was achieved by immersing the subjects in a temperature-controlled water bath (30-42 degrees C). The IPLs became shorter with the rise in body temperature and were correlated with both Tes and Tre. The average slopes for IPL(I-III) and IPL(I-V) were significantly higher than those for IPL(III-V). The present study of humans indicated that changes of IPL(I-III) and IPL(I-V) were 0.11 and 0.16 ms, respectively, per 1 degree C change in core temperature during induced hyperthermia.
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30

Zak, Roksana B., Robert J. Shute, Matthew W. S. Heesch, D. Taylor La Salle, Matthew P. Bubak, Nicholas E. Dinan, Terence L. Laursen, and Dustin R. Slivka. "Impact of hot and cold exposure on human skeletal muscle gene expression." Applied Physiology, Nutrition, and Metabolism 42, no. 3 (March 2017): 319–25. http://dx.doi.org/10.1139/apnm-2016-0415.

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Many human diseases lead to a loss of skeletal muscle metabolic function and mass. Local and environmental temperature can modulate the exercise-stimulated response of several genes involved in mitochondrial biogenesis and skeletal muscle function in a human model. However, the impact of environmental temperature, independent of exercise, has not been addressed in a human model. Thus, the purpose of this study was to compare the effects of exposure to hot, cold, and room temperature conditions on skeletal muscle gene expression related to mitochondrial biogenesis and muscle mass. Recreationally trained male subjects (n = 12) had muscle biopsies taken from the vastus lateralis before and after 3 h of exposure to hot (33 °C), cold (7 °C), or room temperature (20 °C) conditions. Temperature had no effect on most of the genes related to mitochondrial biogenesis, myogenesis, or proteolysis (p > 0.05). Core temperature was significantly higher in hot and cold environments compared with room temperature (37.2 ± 0.1 °C, p = 0.001; 37.1 ± 0.1 °C, p = 0.013; 36.9 ± 0.1 °C, respectively). Whole-body oxygen consumption was also significantly higher in hot and cold compared with room temperature (0.38 ± 0.01 L·min−1, p < 0.001; 0.52 ± 0.03 L·min−1, p < 0.001; 0.35 ± 0.01 L·min−1, respectively). In conclusion, these data show that acute temperature exposure alone does not elicit significant changes in skeletal muscle gene expression. When considered in conjunction with previous research, exercise appears to be a necessary component to observe gene expression alterations between different environmental temperatures in humans.
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31

Young, Kenneth L., Claudia Kassouf, Monika B. Dolinska, David Eric Anderson, and Yuri V. Sergeev. "Human Tyrosinase: Temperature-Dependent Kinetics of Oxidase Activity." International Journal of Molecular Sciences 21, no. 3 (January 30, 2020): 895. http://dx.doi.org/10.3390/ijms21030895.

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Human tyrosinase (Tyr) is involved in pigment biosynthesis, where mutations in its corresponding gene TYR have been linked to oculocutaneous albinism 1, an autosomal recessive disorder. Although the enzymatic capabilities of Tyr have been well-characterized, the thermodynamic driving forces underlying melanogenesis remain unknown. Here, we analyze protein binding using the diphenol oxidase behavior of Tyr and van ’t Hoff temperature-dependent analysis. Recombinant Tyr was expressed and purified using a combination of affinity and size-exclusion chromatography. Michaelis-Menten constants were measured spectrophotometrically from diphenol oxidase reactions of Tyr, using L-3,4-dihydroxyphenylalanine (L-DOPA) as a substrate, at temperatures: 25, 31, 37, and 43 °C. Under the same conditions, the Tyr structure and the L-DOPA binding activity were simulated using 3 ns molecular dynamics and docking. The thermal Michaelis-Menten kinetics data were subjected to the van ‘t Hoff analysis and fitted with the computational model. The temperature-dependent analysis suggests that the association of L-DOPA with Tyr is a spontaneous enthalpy-driven reaction, which becomes unfavorable at the final step of dopachrome formation.
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32

Rising, R., A. Keys, E. Ravussin, and C. Bogardus. "Concomitant interindividual variation in body temperature and metabolic rate." American Journal of Physiology-Endocrinology and Metabolism 263, no. 4 (October 1, 1992): E730—E734. http://dx.doi.org/10.1152/ajpendo.1992.263.4.e730.

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There is significant variation in metabolic rate in humans, independent of differences in body size, body composition, age, and gender. Although it has been generally held that the normal human "set-point" body temperature is 37 degrees C, these interindividual variations in metabolic rate also suggest possible variations in body temperature. To examine the possibility of correlations between metabolic rate and body temperature, triplicate measurements of oral temperatures were made before and after measurement of 24-h energy expenditure in a respiratory chamber in 23 Pima Indian men. Fasting oral temperatures varied more between individuals than can be attributed to methodological errors or intraindividual variation. Oral temperatures correlated with sleeping (r = 0.80, P < 0.0001), and 24-h (r = 0.48, P < 0.02) metabolic rates adjusted for differences in body size, body composition, and age. Similarly, in the 32 Caucasian men of the Minnesota Semi-Starvation Study, oral temperature correlated with adjusted metabolic rate, and the interindividual differences in body temperature were maintained throughout semistarvation and refeeding. These results suggest that a low body temperature and a low metabolic rate might be two signs of an obesity-prone syndrome in humans.
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Corbett, Ron, Abbot Laptook, and Paul Weatherall. "Noninvasive Measurements of Human Brain Temperature Using Volume-Localized Proton Magnetic Resonance Spectroscopy." Journal of Cerebral Blood Flow & Metabolism 17, no. 4 (April 1997): 363–69. http://dx.doi.org/10.1097/00004647-199704000-00001.

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Elucidation of the role of cerebral hyperthermia as a secondary factor that worsens outcome after brain injury, and the therapeutic application of modest brain hypothermia would benefit from noninvasive measurements of absolute brain temperature. The present study was performed to evaluate the feasibility of using 1H magnetic resonance (MR) spectroscopy to measure absolute brain temperature in human subjects on a clinical imaging spectroscopy system operating at a field strength of 1.5 T. In vivo calibration results were obtained from swine brain during whole-body heating and cooling, with concurrent measurements of brain temperature via implanted probes. Plots of the frequency differences between the in vivo MR peaks of water and N-acetyl-aspartate and related compounds (NAX), or water and choline and other trimethylamines versus brain temperature were linear over the temperature range studied (28–40°C). These relationships were used to estimate brain temperature from 1H MR spectra obtained from 10 adult human volunteers from 4 cm3-volumes selected from the frontal lobe and thalamus. Oral and forehead temperatures were monitored concurrently with MR data collection to verify normothermia in all the subjects studied. Temperatures determined using N-acetyl-aspartate or choline as the chemical shift reference did not differ significantly, and therefore results from these estimates were averaged. The brain temperature (mean ± SD) measured from the frontal lobe (37.2 = 0.6°C) and thalamus (37.7 ± 0.6°C) were significantly different from each other (paired t-test, p = 0.035). We conclude that 1H MR spectroscopy provides a viable noninvasive means of measuring regional brain temperatures in normal subjects and is a promising approach for measuring temperatures in brain-injured subjects.
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34

Johnson, J. M. "Nonthermoregulatory control of human skin blood flow." Journal of Applied Physiology 61, no. 5 (November 1, 1986): 1613–22. http://dx.doi.org/10.1152/jappl.1986.61.5.1613.

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Although it is well accepted that skin blood flow (SkBF) in humans is controlled by thermoregulatory reflexes, the conclusion that the cutaneous circulation is also controlled by reflexes of nonthermoregulatory origin is not universally held. This review considers the extent to which the cutaneous circulation participates in baroreceptor-mediated reflexes and in the reflexes associated with exercise. Exercise is explored in some detail, because it elicits both thermoregulatory and nonthermoregulatory reflexes. The overall conclusion reached is that thermoregulatory control of SkBF is subject to modification by or competition from several other sources. The fundamental pattern for control of SkBF is described by the threshold and slope of the SkBF-internal temperature relationship. Reflex effects of skin temperature act to shift the threshold of this relationship such that lower levels of skin temperature are associated with higher threshold internal temperatures at which cutaneous vasodilation begins. Similarly, baroreceptor reflexes, reflexes associated with exercise, and effects of some cardiovascular disease also operate against this background. Although modification of the SkBF-internal temperature slope is occasionally seen, the most consistent effect of these nonthermoregulatory factors is to elevate the threshold internal temperature for cutaneous vasodilation. The consequence of this modification of thermoregulatory control of SkBF is that temperature regulation will often suffer when increases in SkBF are delayed or limited. Blood flow to other regions, possibly including active skeletal muscle, may also be compromised when thermoregulatory demands for SkBF are high.
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35

Wara, S. T., I. Oghogho, A. Abayomi-Alli, C. D. Odikayor, and M. S. Essien. "Human Body Resistance and Temperature Measurement Device." Advanced Materials Research 62-64 (February 2009): 153–58. http://dx.doi.org/10.4028/www.scientific.net/amr.62-64.153.

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This paper discusses the design and construction of a human body resistance and temperature measurement device. The device measures the human body temperature and resistance when the sensing probes are placed in contact with the human skin. The design analysis was based on simple electronic circuit theories leading to specification and choice of components used for the construction of the system. After the construction and testing with various individuals the human body resistance and temperature was found to be within the ranges of 1KΩ to 210KΩ and 36.10C to 37.50C respectively. The paper discuses the various effect of current on the human body and their implication. The system can be adapted to various fields such as bio-technology, security (lie detector), safety equipment in industries and companies to determine insulation.
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36

Zainol, Zaina Norhallis, Masine Md Tap, and Haslinda Mohamed Kamar. "Prediction human skin temperature in comfort level." Science Proceedings Series 1, no. 3 (May 18, 2019): 1–4. http://dx.doi.org/10.31580/sps.v1i3.857.

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Thermal comfort is the human subject perceive satisfaction to the work environment. The thermal comfort need to be achieve towards productive working environment. The comfort level of the subject is affected by the human skin temperature. To assess the skin temperature with the sorrounding by conducting human experiment in the climatic chamber. It is rigorous and complex experiment.This study was developed to predict human skin temperature in comfort level with the finite element method and the bioheat equation. The bioheat equation is a consideration of metabolic heat generation and the blood perfusion to solve heat transfer of the living tissue. It is to determine the skin temperature focussing at the human arm. From the study, it is found that the predicted skin temperature value were in well agreement with the experimental results. The percentage error insignificant with acceptable error of 1.05%.
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37

Lam, David A., and Jeffrey A. Miron. "The Effects of Temperature on Human Fertility." Demography 33, no. 3 (August 1996): 291. http://dx.doi.org/10.2307/2061762.

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38

Warner, David S. "Direct intraoperative measurement of human brain temperature." Journal of Neurosurgical Anesthesiology 10, no. 1 (January 1998): 57. http://dx.doi.org/10.1097/00008506-199801000-00015.

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39

Hu, Ting, Ying Sun, Xuebin Zhang, Seung-Ki Min, and Yeon-Hee Kim. "Human influence on frequency of temperature extremes." Environmental Research Letters 15, no. 6 (June 8, 2020): 064014. http://dx.doi.org/10.1088/1748-9326/ab8497.

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40

Pušnik, Igor, and Andraž Miklavec. "Dilemmas in Measurement of Human Body Temperature." Instrumentation Science & Technology 37, no. 5 (August 10, 2009): 516–30. http://dx.doi.org/10.1080/10739140903149061.

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41

Lin, Shao, Wayne Lawrence, Ziqiang Lin, Edward Fitzgerald, Guang-Hui Dong, and Scott Sheridan. "Temperature Variation, Transitional Season, and Human Health." ISEE Conference Abstracts 2017, no. 1 (February 2018): 310. http://dx.doi.org/10.1289/isee.2017.2017-310.

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42

Childs, Charmaine, and Graham Machin. "Reliability issues in human brain temperature measurement." Critical Care 13, no. 4 (2009): R106. http://dx.doi.org/10.1186/cc7943.

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43

Lee, Chan Mi, Seon-Pil Jin, Eun Jin Doh, Dong Hun Lee, and Jin Ho Chung. "Regional Variation of Human Skin Surface Temperature." Annals of Dermatology 31, no. 3 (2019): 349. http://dx.doi.org/10.5021/ad.2019.31.3.349.

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44

Zhang, W., X. Y. Guo, G. Y. Hu, W. B. Liu, J. W. Shay, and A. B. Deisseroth. "A temperature-sensitive mutant of human p53." EMBO Journal 13, no. 11 (June 1994): 2535–44. http://dx.doi.org/10.1002/j.1460-2075.1994.tb06543.x.

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45

Kozyreva, T. V., and E. Ya Tkachenko. "Effect of menthol on human temperature sensitivity." Human Physiology 34, no. 2 (March 2008): 221–25. http://dx.doi.org/10.1134/s0362119708020138.

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46

Slivka, D., C. Dumke, T. Tucker, J. Cuddy, and B. Ruby. "Human mRNA Response to Exercise and Temperature." International Journal of Sports Medicine 33, no. 02 (November 23, 2011): 94–100. http://dx.doi.org/10.1055/s-0031-1287799.

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47

Stone, J. Gilbert, Robert R. Goodman, Kristy Z. Baker, Christopher J. Baker, and Robert A. Solomon. "Direct Intraoperative Measurement of Human Brain Temperature." Neurosurgery 41, no. 1 (July 1, 1997): 20–24. http://dx.doi.org/10.1097/00006123-199707000-00007.

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48

Cabanac, M., and H. Brinnel. "The pathology of human temperature regulation: Thermiatrics." Experientia 43, no. 1 (January 1987): 19–27. http://dx.doi.org/10.1007/bf01940348.

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49

LABOW, R. "Temperature affects human sarcoplasmic reticulum calcium atpase." Journal of Molecular and Cellular Cardiology 22 (July 1990): 12. http://dx.doi.org/10.1016/0022-2828(90)90160-4.

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50

Kenney, W. Larry, and Thayne A. Munce. "Invited Review: Aging and human temperature regulation." Journal of Applied Physiology 95, no. 6 (December 2003): 2598–603. http://dx.doi.org/10.1152/japplphysiol.00202.2003.

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This mini-review focuses on the effects of aging on human temperature regulation. Although comprehensive reviews have been published on this topic (Kenney WL. Exercise and Sport Sciences Reviews, Baltimore: Williams & Wilkins, 1997, p. 41-76; Pandolf KB. Exp Aging Res 17: 189-204, 1991; Van Someren EJ, Raymann RJ, Scherder EJ, Daanen HA, and Swaab DF. Ageing Res Rev 1: 721-778, 2002; and Young AJ. Exp Aging Res 17: 205-213, 1991), this mini-review concisely summarizes the present state of knowledge about human temperature regulation and aging in thermoneutral conditions, as well as during hypo- and hyperthermic challenges. First, we discuss age-related effects on baseline body core temperature and phasing rhythms of the circadian temperature cycle. We then examine the altered physiological responses to cold stress that result from aging, including attenuated peripheral vasoconstriction and reduced cold-induced metabolic heat production. Finally, we present the age-related changes in sweating and cardiovascular function associated with heat stress. Although epidemiological evidence of increased mortality among older adults from hypo- and hyperthermia exists, this outcome does not reflect an inability to thermoregulate with advanced age. In fact, studies that have attempted to separate the effects of chronological age from concurrent factors, such as fitness level, body composition, and the effects of chronic disease, have shown that thermal tolerance appears to be minimally compromised by age.
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