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1

Stauropoulos, Dimitrios James. An analysis of the interplay between telomeric factors and DNA repair proteins, in the human ALT pathway and cellular response to genomic double strand breaks. 2005.

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2

Moral, Amelia Marti del, and Guillermo Zalba Goñi. Telomeres Diet and Human Disease. Taylor & Francis Group, 2021.

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Marti, Amelia, and Guillermo Zalba. Telomeres, Diet and Human Disease: Advances and Therapeutic Opportunities. Taylor & Francis Group, 2017.

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Telomeres, Diet and Human Disease: Advances and Therapeutic Opportunities. Taylor & Francis Group, 2017.

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Marti, Amelia, and Guillermo Zalba. Telomeres, Diet and Human Disease: Advances and Therapeutic Opportunities. Taylor & Francis Group, 2017.

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Marti, Amelia, and Guillermo Zalba. Telomeres, Diet and Human Disease: Advances and Therapeutic Opportunities. Taylor & Francis Group, 2017.

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Marti, Amelia, and Guillermo Zalba. Telomeres, Diet and Human Disease: Advances and Therapeutic Opportunities. Taylor & Francis Group, 2017.

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8

Blackburn, Elizabeth, and Elissa Epel. Telomere Effect: A Revolutionary Approach to Living Younger, Healthier, Longer. Grand Central Publishing, 2017.

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Telomere Effect: A Revolutionary Approach to Living Younger, Healthier, Longer. Orion Publishing Group, Limited, 2018.

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10

Gray, Doug, Carole Proctor, and Tom Kirkwood. Biological aspects of human ageing. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199644957.003.0001.

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At the molecular and cellular levels human ageing is characterized by the accumulation of unrepaired random damage, and an accompanying loss of function. A major source of damage is oxidative stress caused by the generation of reactive oxygen species as a by-product of respiration. DNA and proteins are both susceptible to damage but whereas DNA damage repair systems exist, faulty proteins are generally removed by protein degradation systems. During ageing these systems become less efficient and the subsequent accumulation of damaged protein promotes protein aggregation, a process which is especially problematic in the ageing brain. Other aspects of ageing include genetic and epigenetic changes, mitochondrial dysfunction, telomere shortening, and cellular senescence, all subject to stochasticity. The complexity of the biology of ageing has led to an increase in the use of systems biology approaches whereby the use of mathematical modelling and bioinformatic tools complement the more traditional experimental approaches.
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Blackburn, Dr Elizabeth, and Dr Elissa Epel. The Telomere Effect: A Revolutionary Approach to Living Younger, Healthier, Longer. Grand Central Publishing, 2017.

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The Telomere Effect: A Revolutionary Approach to Living Younger, Healthier, Longer. Grand Central Publishing, 2018.

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Blackburn, Elizabeth H. The telomere effect : (large print): A revolutionary approach to living younger, healthier, longer. 2017.

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author, Epel Elissa, ed. The telomere effect: A revolutionary approach to living younger, healthier, longer. 2017.

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15

Massey, Douglas S., and Brandon Wagner. Segregation, Stigma, and Stratification: A Biosocial Model. Edited by Brenda Major, John F. Dovidio, and Bruce G. Link. Oxford University Press, 2017. http://dx.doi.org/10.1093/oxfordhb/9780190243470.013.14.

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This chapter reviews research on segregation’s effect in generating concentrated poverty and stigma, and it explores the biological consequences of exposure to these conditions for health and socioeconomic status. High levels of segregation interact with high levels of poverty to produce concentrated poverty for African Americans and Hispanics in many metropolitan areas. In addition to objective circumstances of deprivation, the concentration of poverty also brings about the stigmatization of the segregated group. The differential exposure of Blacks and Hispanics to concentrated neighborhood disadvantage and its correlates, in turn, functions to shorten telomeres, increase allostatic load, and alter gene expression in deleterious ways. In so doing, it compromises health and cognitive ability, the two critical components of human capital formation, thus systematically undermining the socioeconomic prospects of African Americans and Hispanics in today’s post-industrial, information economy.
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Zapico, Sara C. Mechanisms Linking Aging, Diseases and Biological Age Estimation. Taylor & Francis Group, 2017.

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Zapico, Sara C. Mechanisms Linking Aging Diseases and Biological Age Estimation. Taylor & Francis Group, 2021.

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Mechanisms Linking Aging, Diseases and Biological Age Estimation. Taylor & Francis Group, 2016.

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Zapico, Sara C. Mechanisms Linking Aging, Diseases and Biological Age Estimation. Taylor & Francis Group, 2017.

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