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Books on the topic 'Human papillomavirus infection'

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Published: 4 June 2021
Last updated: 30 May 2022

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1

Jong-Tieben, Linda M. de. Human papillomavirus infection and skin cancer in renal transplant recipients. Leiden: University of Leiden, 1998.

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2

Perinot, Glenis Michele. A study of human papillomavirus infection: Recombinant DNA synthesis and analysis. Sudbury, Ont: Laurentian University Press, 1995.

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3

Michael, Paul Simon. PCR screening for human papillomavirus infections, and evaluation of the estimated infection prevalence for a population of females. Sudbury, Ont: Laurentian University, Department of Biology, 1999.

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4

IARC, Working Group on the Evaluation of Carcinogenic Risks to Humans (1995 Lyon France). Human papillomaviruses. [Lyon, France?]: International Agency for Research on Cancer, 1995.

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5

Dizon, Don S. Questions & answers about human papilloma virus (HPV). Sudbury, Mass: Jones and Bartlett Publishers, 2011.

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6

Syrjänen, Kari J. Human papillomavirus (HPV) involvement in esophageal carcinogenesis. New York: Nova Biomedical Books, 2010.

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7

Dizon, Don S. Dx/Rx: Human papilloma virus. Sudbury, MA: Jones & Bartlett Learning, 2011.

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8

Dizon, Don S. Questions & answers about human papilloma virus (HPV). Sudbury, Mass: Jones and Bartlett Publishers, 2011.

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9

R, Stuckey Ashley, and Krychman Michael L, eds. Dx/Rx: Human papilloma virus. Sudbury, MA: Jones & Bartlett Learning, 2011.

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10

Rosenblatt, Alberto. Human Papillomavirus: A Practical Guide for Urologists. Berlin, Heidelberg: Springer-Verlag Berlin Heidelberg, 2009.

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11

Bidal, Michel. Recombinant DNA technology and its use in trying to conquer cervical cancer association to infections with Human papillomavirus type 16. Sudbury, Ont: Laurentian University, 1991.

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12

Parker, Philip M., and James N. Parker. Human papilloma virus: A medical dictionary, bibliography, and annotated research guide to Internet references. San Diego, CA: ICON Health Publications, 2003.

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13

Sexually transmitted diseases sourcebook: Basic consumer health information about sexual health and the screening, diagnosis, treatment, and prevention of common sexually transmitted diseases (STDs), including chancroid, chlamydia, gonorrhea, herpes, hepatitis, human immunodeficiency virus/acquired immunodeficiency syndrome (HIV/AIDS), human papillomavirus (HPV), syphilis, and trichomoniasis ; along with facts about risk factors and complications, trends and disparities in infection rates, tips for discussing STDs with sexual partners, a glossary of related terms, and resources for additional help and information. 5th ed. Detroit, MI: Omnigraphics, Inc., 2013.

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14

Sexual health information for teens: Health tips about sexual development, reproduction, contraception, and sexually transmitted infections including facts about puberty, sexuality, birth control, HIV/AIDS, human papillomavirus, chlamydia, gonorrhea, herpes, and more. Detroit, MI: Omnigraphics, Inc., 2016.

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15

Magill, Elizabeth. Sexual health information for teens: Health tips about sexual development, reproduction, contraception, and sexually transmitted infections : including facts about puberty, sexuality, birth control, hiv/aids, human papillomavirus, chlamydia, gonorrhea, herpes, and more. 3rd ed. Detroit, Mich: Omnigraphics, 2011.

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16

Genital warts: Human papillomavirus infection. London: E. Arnold, 1995.

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17

Negari, Shelly Ben-Harush, and Jessica A. Kahn. Human Papillomavirus. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190604813.003.0009.

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Human papillomavirus (HPV) is a ubiquitous, single-stranded DNA virus that is commonly sexually transmitted and an important cause of cervical cancer. Manifestations of infection in the perinatal and childhood periods are recurrent respiratory papillomatosis (RRP) and anogenital warts (AGWs). Children with respiratory papillomatosis most commonly develop papillomas in the larynx, but papillomas may develop in any location along the respiratory tract. Although RRP is rare, it is the most common benign neoplasm of the larynx among children and the second-most-frequent cause of childhood hoarseness. AGWs are uncommon in the perinatal period and typically benign. They may develop on the vulva, hymen, vagina, urethra, or perianal area in girls and on the perianal area in boys. The clinical manifestations, epidemiology, diagnostic studies, and management strategies pertinent to these infections are reviewed.
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18

Renzo, Barrasso, and Gross G, eds. Human papilloma virus infection: A clinical atlas. Berlin: Ullstein Mosby, 1997.

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19

Tindle, Robert W. Vaccines for Human Papillomavirus Infection and Disease. Landes Bioscience, 1999.

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20

W, Tindle Robert, ed. Vaccines for human papillomavirus infection and anogenital disease. Austin: R.G. Landes, 1999.

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21

Genital Warts: Human Papillomavirus Infection (Hodder Arnold Publication). A Hodder Arnold Publication, 1995.

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22

Cothran, Mary Mccaa. HUMAN PAPILLOMAVIRUS INFECTION, CIGARETTE SMOKING AND CERVICAL INTRAEPITHELIAL NEOPLASIA. 1996.

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23

Black, Riva. Oral papillary squamous cell carcinoma: Its relation to human papillomavirus infection and associated cell cycle deregulation. 2005.

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24

Bulterys, Marc, Julia Brotherton, and Ding-Shinn Chen. Prevention of Infection-Related Cancers. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0066.

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This chapter discusses primary prevention measures that disrupt transmission of oncogenic infections. It begins by discussing vaccination against hepatitis B virus (HBV) and human papillomavirus (HPV), two major causes of cancer for which safe and effective vaccines are currently available. It briefly discusses the importance of treatment and prophylaxis against human immunodeficiency virus type 1 (HIV-1), which potentiates the virulence of other viral infections as well as directly increasing the incidence of non-Hodgkin lymphoma. It does not discuss the treatment of HBV or hepatitis C virus (HCV) infection, since these are considered in Chapters 25 and 33. Also beyond the scope of this chapter are the randomized clinical trials currently underway to assess the efficacy and feasibility of eradication of Helicobacter pylori (Chapters 24, 31), vaccination against Epstein-Barr virus (EBV) (Chapters 24, 26, 39), or the prevention of schistosomiasis and liver flukes (Chapters 24, 33, and 52).
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25

Burchell, Ann, and Eduardo Franco1. The impact of immunization on cancer control: the example of HPV vaccination. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199550173.003.0006.

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Chapter 6 reviews briefly the role of infections as causal agents in cancer, describes anti-hepatitis B virus (HBV) immunization as the first cancer vaccine paradigm, and finally focuses on the latest paradigm of prophylactic vaccination against human papillomavirus (HPV) infection as the new front in cancer prevention.
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26

Cobo, Fernando. Human papillomavirus infections. Woodhead Publishing Limited, 2012. http://dx.doi.org/10.1533/9781908818171.

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27

Franceschi, Silvia, Hashem B. El-Serag, David Forman, Robert Newton, and Martyn Plummer. Infectious Agents. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0024.

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Eleven infectious agents (seven viruses, three parasites, and one bacterium) have been classified by the International Agency for Research on Cancer as carcinogenic to humans for one or more cancer sites: hepatitis B virus; hepatitis C virus; thirteen types of human papillomavirus (HPV); human immunodeficiency virus type 1 (HIV-1); human T-cell leukemia virus type 1; Epstein-Barr virus; Kaposi sarcoma herpesvirus; Helicobacter pylori; Opisthorchis viverrini; Clonorchis sinensis; and Schistosoma haematobium. Other infectious agents, such as Merkel cell polyomavirus, Plasmodium falciparum, and cutaneous HPVs, have been classified as “probably carcinogenic” or “possibly carcinogenic.” Accurate biomarkers of chronic infection have been essential for estimating risk and ascribing a causal role to infectious agents in cancer. Of the 14 million cases of cancer estimated to have occurred worldwide in 2012, 2.2 million were caused by infectious agents. Vaccination and screen-and-treat programs have the potential for greatly reducing the burden of cancer caused by infections.
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28

J, Syrjänen Kari, Gissmann L. 1949-, and Koss Leopold G, eds. Papillomaviruses and human disease. Berlin: Springer-Verlag, 1987.

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29

Vigil, Karen J. Sexually Transmitted Diseases. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190493097.003.0044.

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Sexually transmitted diseases (STDs) are common in HIV-infected patients. Education and counseling on changes in sexual behaviors of patients with STDs and their sexual partners, identification of asymptomatic infection, and effective diagnosis and treatment are the cornerstone for prevention. HIV-infected patients with syphilis should have a detailed neurologic examination. Penicillin is the treatment of choice for syphilis. Gonococcal infection is an important cause of urethritis, cervicitis, pharyngitis, and proctitis in HIV-infected sexually active patients. Dual therapy for gonorrhea and chlamydia is recommended. Most Chlamydia trachomatis infections are asymptomatic and thus detected only by routine, periodic screening. Human papillomavirus is the most common STD in the United States.
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30

Syrjänen, Kari J., and Stina M. Syrjänen. Papillomavirus Infections in Human Pathology. Wiley, 2000.

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31

G, Gross, and Krogh Geo von, eds. Human papillomavirus infections in dermatovenereology. Boca Raton: CRC Press, 1997.

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32

(Editor), Jane C. Sterling, and Stephen K. Tyring (Editor), eds. Human Papillomaviruses: Clinical and Scientific Advances. A Hodder Arnold Publication, 2001.

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33

Jane, Sterling, and Tyring Stephen K, eds. Human papillomaviruses: Clinical and scientific advances. London: Arnold, 2001.

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34

(Contributor), WHO, ed. Human Papillomaviruses (IARC Monographs on the Evaluation of Carcinogenic Risks to H). World Health Organisation, 1995.

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35

Human Papillomavirus: Bench to Bedside. S. Karger, 2014.

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36

Human Papilloma Viruses (Perspectives in Medical Virology). Elsevier Science, 2002.

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37

Franklin, Yvette. Human Papillomaviruses: Detection, Infection and Protection. Nova Science Publishers, Incorporated, 2020.

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38

Franklin, Yvette. Human Papillomaviruses: Detection, Infection and Protection. Nova Science Publishers, Incorporated, 2020.

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39

A, Stanley Margaret, and International Workshop on HPV Immunology (2nd : 1993 : Cambridge, England), eds. Immunology of human papillomaviruses. New York: Plenum Press, 1994.

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40

Herrero, Rolando, and Raul Murillo. Cervical Cancer. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0048.

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Cervical cancer is the fourth most common cancer in women worldwide, with more than 500,000 cases and 250,000 deaths per year. The disease is characterized by marked regional differences, with more than 80% of the cases and deaths occurring in developing countries. The etiology and natural history of the disease are very well studied, with persistent infection with one of thirteen human papillomavirus (HPV) types now considered to be a necessary cause. The molecular mechanisms have also been elucidated and are mediated mainly by the expression of viral oncogenes that interfere with cellular pathways. The two most common HPV types, namely HPV-16 and HPV-18, are associated with about 70% of all cases around the world. Immunologic (e.g., HIV infection), hormonal (e.g., high parity), environmental (e.g., smoking), and genetic (e.g., HLA type) cofactors determine the risk of persistence and cancer among women harboring HPV infection.
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41

van Assen, Sander, and Marc Bijl. Vaccination in immunocompromised adults. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0094.

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This chapter addresses all important questions regarding vaccination of patients with autoimmune inflammatory rheumatic diseases (AIIRD). First, the incidence of vaccine-preventable infections in these patients is discussed. Pulmonary infections, including influenza and pneumococcal infection, occur more often in AIIRD patients; herpes zoster and human papillomavirus are also more frequent. The efficacy of vaccination for all European registered vaccines is discussed. Treatment with disease-modifying anti-rheumatic drugs (DMARDs) and biologicals (in particular TNFα‎-blocking agents) do not hamper, or only slightly hamper, the immune responses to most vaccines. Rituximab is an exception, severely reducing humoral responses following influenza and pneumococcal vaccination, at least during the first 6 months after administration. Safety of vaccination is an important issue in patients with autoimmune diseases, since increased disease activity of the underlying disease as a result of vaccination is theoretically possible. The available evidence is summarized, suggesting that vaccination is safe in AIIRD patients. Live vaccines, however, are contraindicated in immunosuppressed patients with AIIRD. Finally, the European League Against Rheumatism (EULAR) recommendations are highlighted, summarizing the 'do's' and 'don'ts' of vaccination in adults with AIIRD.
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42

Cobo, Fernando. Human Papillomavirus Infections: From the Laboratory to Clinical Practice. Woodhead Publishing, 2012.

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43

Human Papillomavirus Infections in Dermatovenereology (Crc Series in Dermatology). Informa Healthcare, 1996.

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44

Hashibe, Mia, Erich M. Sturgis, Jacques Ferlay, and Deborah M. Winn. Oral Cavity, Oropharynx, Lip, and Salivary Glands. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0029.

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Cancers of the oral cavity, oropharynx, lip, and salivary glands are malignancies of the head and neck. Some of these cancer sites share risk factors, although each has distinctive anatomic, epidemiologic, and clinical features. Oral cavity cancers arise on the inner lip and buccal mucosa, anterior two-thirds of the tongue, gum, hard palate, and floor of mouth. These cancers are strongly associated with the use of smoked and smokeless tobacco products, heavy alcohol consumption, and chewing of betel quid or pan, but only minimally associated with prior infection with human papillomavirus (HPV). In contrast, oropharyngeal cancers affect the posterior one-third (base) of the tongue, tonsils, soft palate, and other oropharyngeal tissues and are strongly associated with HPV-16 infection as well as with the use of tobacco, alcohol, and betel quid. In principle, tumors of the oral cavity, oropharynx, and lip are among the most preventable forms of cancer.
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45

Everything You Need to Know About Human Papillomavirus. Rosen Publishing Group, 2001.

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46

Winkler, Barbara. Clinical Practice of Gynecology: Human Papillomavirus Infections (Clinical Practice of Gynecology). McGraw-Hill/Appleton & Lange, 1990.

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47

Djajadiningrat, Rosa, and Simon Horenblas. Penile cancer. Edited by James W. F. Catto. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0093.

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Penile cancer is a rare malignancy in the Western world, but in Asia, Africa, and South Africa the incidence is much higher. Risk factors, including phimosis, human papillomavirus (HPV), smoking, chronic inflammatory conditions, psoralen ultraviolet photochemotherapy, genital warts, and HIV infection play a role in the pathogenesis of penile cancer. Approximately 95% of all penile tumours are squamous cell carcinomas (PSCC) and the large majority arise from the prepuce or glans. PSCC has a strong tendency for lymphatic dissemination, but cure can still be attained in patients with inguinal involvement. The most commonly used staging system is the 2009 TNM classification for penile cancer. Surgical resection has been the mainstay of treatment in penile carcinoma, including penile-preserving techniques, partial and total penectomy. The aim of surgery is minimizing loss of anatomy and function, without jeopardizing oncological results.
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48

Hausen, H. Zur. Human Pathogenic Papillomaviruses (Current Topics in Microbiology and Immunology). Springer-Verlag Telos, 1999.

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49

Török, M. Estée, Fiona J. Cooke, and Ed Moran. Sexually transmitted infections. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199671328.003.0018.

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This chapter covers the diagnosis and management of sexually transmitted infections, including bacterial vaginosis, with causes including vaginal discharge, vulvovaginal candidiasis, and trichomoniasis. The chapter also covers vulvovaginal candidiasis, genital warts or anogenital warts caused by human papillomavirus, tropical genital ulceration (which is commoner in patients presenting with sexually transmitted infections in the developing world and is an important factor in the spread of HIV), genital herpes, pelvic inflammatory disease, toxic shock syndrome, gonorrhoea, chlamydia, trichomoniasis, and syphilis.
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50

Tommasino, Massimo. Papillomaviruses in Human Cancer: The Role Of E6 And E7 Oncoproteins. Springer, 2013.

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