Log in

Relevant bibliographies by topics / Human papillomavirus infection

Contents

Journal articles
Dissertations / Theses
Books
Book chapters
Conference papers
Reports

Academic literature on the topic 'Human papillomavirus infection'

Author: Grafiati

Published: 4 June 2021

Last updated: 30 May 2022

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Human papillomavirus infection.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "Human papillomavirus infection"

1

Sharipova, I. P., and E. I. Musabaev. "HUMAN PAPILLOMAVIRUS INFECTION AND CERVICAL CANCER (OWERWIEW)." UZBEK MEDICAL JOURNAL 2, no. 4 (April 30, 2021): 23–29. http://dx.doi.org/10.26739/2181-0664-2021-4-4.

Full text

Abstract:

Viral infections are responsible for 15–20% of all human cancers. Infection with oncogenic viruses can contribute to various stages of carcinogenesis. Despite effective screening methods, cervical cancer continues to be a major public health problem. There are large differences in morbidity and mortality from cervical cancer by geographic region. The age-specific prevalence of HPV varies widely in different populations and has shown two peaks of HPV positiveness in young and older women. Around the world, there have been many studies on the epidemiology of HPV infection and oncogenic properties due to different HPV genotypes. However, there are still many countries where population prevalence has not yet been determined. Moreover, screening strategies for cervical cancer differ from country to country. Organized cervical screening programs are potentially more effectivethan opportunistic screening programs.Key words:Human papillomavirus, cervical cancer, screening, dysplasia

APA, Harvard, Vancouver, ISO, and other styles

2

HIROMURA, Katsuhiko, Masaharu GUNJI, Masahiko FUJINO, and Masafumi ITO. "Human papillomavirus infection in healthy women in Japan." Journal of the Japanese Society of Clinical Cytology 53, no. 5 (2014): 366–70. http://dx.doi.org/10.5795/jjscc.53.366.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Peh, Woei Ling, Kate Middleton, Neil Christensen, Philip Nicholls, Kiyofumi Egawa, Karl Sotlar, Janet Brandsma, et al. "Life Cycle Heterogeneity in Animal Models of Human Papillomavirus-Associated Disease." Journal of Virology 76, no. 20 (October 15, 2002): 10401–16. http://dx.doi.org/10.1128/jvi.76.20.10401-10416.2002.

Full text

Abstract:

ABSTRACT Animal papillomaviruses are widely used as models to study papillomavirus infection in humans despite differences in genome organization and tissue tropism. Here, we have investigated the extent to which animal models of papillomavirus infection resemble human disease by comparing the life cycles of 10 different papillomavirus types. Three phases in the life cycles of all viruses were apparent using antibodies that distinguish between early events, the onset of viral genome amplification, and the expression of capsid proteins. The initiation of these phases follows a highly ordered pattern that appears important for the production of virus particles. The viruses examined included canine oral papillomavirus, rabbit oral papillomavirus (ROPV), cottontail rabbit papillomavirus (CRPV), bovine papillomavirus type 1, and human papillomavirus types 1, 2, 11, and 16. Each papillomavirus type showed a distinctive gene expression pattern that could be explained in part by differences in tissue tropism, transmission route, and persistence. As the timing of life cycle events affects the accessibility of viral antigens to the immune system, the ideal model system should resemble human mucosal infection if vaccine design is to be effective. Of the model systems examined here, only ROPV had a tissue tropism and a life cycle organization that resembled those of the human mucosal types. ROPV appears most appropriate for studies of the life cycles of mucosal papillomavirus types and for the development of prophylactic vaccines. The persistence of abortive infections caused by CRPV offers advantages for the development of therapeutic vaccines.

APA, Harvard, Vancouver, ISO, and other styles

4

Park, Jong Sup. "Human Papillomavirus Infection." Journal of the Korean Medical Association 45, no. 4 (2002): 430. http://dx.doi.org/10.5124/jkma.2002.45.4.430.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Beutner, Karl R. "Human papillomavirus infection." Journal of the American Academy of Dermatology 20, no. 1 (January 1989): 114–23. http://dx.doi.org/10.1016/s0190-9622(89)80004-0.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Adimora, Adaora A., and E. Byrd Quinlivan. "Human papillomavirus infection." Postgraduate Medicine 98, no. 3 (September 1995): 109–20. http://dx.doi.org/10.1080/00325481.1995.11946045.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Oriel, J. D. "Human papillomavirus infection." Current Opinion in Infectious Diseases 2, no. 1 (February 1989): 2–6. http://dx.doi.org/10.1097/00001432-198902010-00002.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Oriel, J. D. "Human papillomavirus infection." Current Opinion in Infectious Diseases 3, no. 1 (February 1990): 24–29. http://dx.doi.org/10.1097/00001432-199002000-00005.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Reed, B. D. "Human papillomavirus infection." Archives of Family Medicine 3, no. 10 (October 1, 1994): 855–56. http://dx.doi.org/10.1001/archfami.3.10.855.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Torpy, Janet M., Alison E. Burke, and Richard M. Glass. "Human Papillomavirus Infection." JAMA 297, no. 8 (February 28, 2007): 912. http://dx.doi.org/10.1001/jama.297.8.912.

Full text

APA, Harvard, Vancouver, ISO, and other styles

More sources

Dissertations / Theses on the topic "Human papillomavirus infection"

1

Aaltonen, Leena-Maija. "Laryngeal human Papillomavirus infection." Helsinki : University of Helsinki, 1999. http://ethesis.helsinki.fi/julkaisut/laa/haart/vk/aaltonen/.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Geijersstam, Veronika af. "Dynamics of oncogenic human papillomavirus infection /." Stockholm, 2000. http://diss.kib.ki.se/2000/91-628-4459-8/.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Healey, Sylvia M. "Human papillomavirus infection and cervical dysplasia in Nunavut." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape4/PQDD_0011/MQ52906.pdf.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Bratton, Kristin. "The Role of Furin Cleavage in Human Papillomavirus Infection." Thesis, The University of Arizona, 2012. http://hdl.handle.net/10150/243893.

Full text

Abstract:

The early stages of Human Papillomavirus infection proceeds through a series of steps that involve interactions between cell surface molecules and viral capsid proteins. While the role of viral protein L1 in internalization of the virus has been well characterized, the role of minor capsid protein L2 in internalization and infection is less clear. However, cleavage of L2 by furin, a proprotein convertase, and the resulting exposure of an N-terminal region, the RG-1 epitope, has been shown to be a critical step in infection. In this study, we aimed to explicitly show furin cleavage during infection and identify the cellular conditions required for this cleavage event and establishment of infection. An assay to detect furin cleavage of L2 was developed, and for the first time, furin cleavage was directly shown during infection. In vivo data suggests that cyclophilin B, a peptidyl-prolyl isomerase believed to playa role in the conformational changes that occur prior to internalization, may playa less prominent role than previously thought. Understanding the role of L2 in entry and infection provides a more comprehensive picture of the mechanism of papillomavirus infection and exposure of the RG-1 epitope, the major target for next-generation broadly protective pan-HPV vaccines.

APA, Harvard, Vancouver, ISO, and other styles

5

Li, Wenqin. "Novel intranasal GNRs-DNA vaccines against human papillomavirus infection." Thesis, University of Strathclyde, 2015. http://digitool.lib.strath.ac.uk:80/R/?func=dbin-jump-full&object_id=28807.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Meys, Rhonda. "Aspects of human papillomavirus (HPV) disease in human immunodeficiency virus (HIV) infection." Thesis, Imperial College London, 2013. http://hdl.handle.net/10044/1/10730.

Full text

Abstract:

Cutaneous and genital human papillomavirus (HPV) infection in HIV patients, on suppressive anti-retroviral therapy (ART), poses under-investigated clinical challenges. HPV in HIV may represent a form of immune reconstitution associated disease (IRAD). HPV disease and IRADs have been separately correlated with human leucocyte antigen (HLA) genotype. HLA might also influence HPV in HIV. Comprehensive HPV typing of persistent warts obtained from HIV infected and healthy subjects was performed. Cutaneous HPV types were detected using nested PCR/sequencing and newly developed (Luminex based) HSLPCR/ MPG; genital and beta HPV types were identified using a reverse hybridisation line probe assay. Real time PCR was employed to determine HPV DNA viral loads. HLA alleles were defined in HIV infected and healthy patients by Luminex-based molecular typing using DNA derived from blood. The HPV profile of cutaneous and genital HIV warts differs significantly from warts from healthy individuals. In HIV, HPV 7 has been confirmed to be an important HPV type in cutaneous warts (p=0.001). In genital warts in HIV, HPV 11 is the predominant HPV type (p=0.15) and HPV 6 is less common (p=0.002), contrasting with the usual finding that HPV 6 is the principal type in the general population. Cross-over of HPV types between cutaneous and genital sites suggests that HPV tropism is less important than previously thought. An excess of beta HPV types, predominantly as mixed infections, is seen in cutaneous warts in HIV (p<0.0005). The HLA class I allele group HLA-B*44 (as the allele HLA-B*44:02 and the haplotype HLA-B*44, -C*05) has been identified more frequently in HIV than in controls (p=0.004, allele group; p=0.0006, allele; p=0.001, haplotype). The class II allele HLA-DQB1*06 may also be of interest (p=0.03). However, the differences are reduced after correction for multiple testing. Further work is required to ascertain if these HPV types and alleles are of importance.

APA, Harvard, Vancouver, ISO, and other styles

7

Pintos, Vega Luis Javier. "Human papillomavirus infection and oral cancer : a case-control study." Thesis, McGill University, 2002. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=84413.

Full text

Abstract:

Introduction. Human papillomavirus (HPV) has been detected with varying frequency in oral cancers and in normal oral tissues. The main objective of the present study was to examine the association between HPV infection and risk of developing oral cancer.
Methodology. This investigation, as a component of an international multi-centre study coordinated by the IARC, followed a hospital-based case-control design. Cases consisted of newly diagnosed patients with primary squamous cell carcinoma of the oral cavity, including mouth and oropharynx. Controls were frequency matched to cases by sex, age, and hospital. All subjects were interviewed to elicit detail information on known and putative risk factors.
Oral exfoliated cells were collected from all subjects for detection of HPV DNA using the PGMY09/11 PCR protocol. Antibodies against HPV 16, 18, and 31 capsids were detected in patients' plasma using an immunoassay technique. Logistic regression was used for estimation of odds ratios (ORs) and 95% confidence intervals (CI) of oral cancer for HPV and other candidate risk factors.
Results. A total of 72 cases and 129 controls were recruited. HPV DNA was detected in 19% of cases (14 out of 72), and in 5% of controls (6 out of 129). Analysis for cancers related to Waldeyer's ring (palatine tonsil and base of tongue) showed that the OR of disease for detection of high risk HPV types was 19.32 (95%CI:2.3--159.5), after adjustment for socio-demographic characteristics, tobacco and alcohol consumption. The adjusted OR of disease for HPV 16 seropositivity was 31.51 (95%CI:4.5--219.7). Analysis for non tonsillar oral cancers showed that the OR for detection of high risk HPV DNA in oral cells and for seropositivity were 2.14 (95%CI:0.4--13.0) and 3.16 (95%CI:0.8--13.0), respectively.
Discussion. The results from this study provide evidence supporting a strong association between HPV infection and cancers of the oropharynx, especially those arising from Waldeyer's ring. On the other hand, the association with non tonsillar oral cancers was of much lower magnitude. The biological evidence establishing a firm etiologic link remains to be established for the latter subsites, whereas the association between HPV and Waldeyer's ring carcinomas is consistent with a causal link.

APA, Harvard, Vancouver, ISO, and other styles

8

Burchell, Ann. "Human papillomavirus infection and transmission among couples through heterosexual activity." Thesis, McGill University, 2009. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=40745.

Full text

Abstract:

Human papillomavirus (HPV) is the most common sexually transmitted infection (STI). The vast majority of these infections clear spontaneously. The small proportion that persists may result in substantial morbidity and treatment costs. High oncogenic risk HPV (HR-HPV) types, including HPV-16 and 18, are recognised unequivocally as the main causal factor for cervical cancer, and may also cause other anogenital neoplasms and head and neck cancers. Infections with types that have low oncogenic risk (LR-HPV), such as HPV-6 and -11, are associated with benign lesions including genital warts. Many projections of the impact of the new HPV vaccines and screening technologies use dynamic transmission models which require sound estimates of the probability of transmission upon exposure. Furthermore, biological and practical limitations of the current vaccines require that we explore as many prevention options as possible. A critical research question is whether condoms provide protection. The main aims of the thesis were to characterize patterns of HPV infection among heterosexual couples in a new relationship, to identify risk factors for HPV infection, and to estimate HPV transmission probabilities per partnership and per coital act. I carried out a preliminary Monte Carlo simulation to estimate the probability of HPV transmission, then designed and conducted a study of heterosexual couples. The thesis objectives were addressed using baseline data from the ongoing HITCH Cohort Study (HPV Infection and Transmission among Couples through Heterosexual activity). The study population consists of young (aged 18-24) women attending a university or junior college (CEGEP) in Montreal and their male partners. Results from the simulation analysis suggested that HPV is highly transmissible, which was confirmed by the cross-sectional analysis of the HITCH study. Among the 263 couples enrolled between 05/2005 and 08/2008, HPV prevalence was 56% among women and men. In nearly two th
Le virus du papillome humain (VPH) est l'infection transmissible sexuellement (ITS) la plus répandue. Une grande majorité des infections à VPH se résorbent spontanément. Cependant, la petite proportion d’infections à VPH persistantes peut avoir des coûts substantiels de traitement et de morbidité comme conséquence. Des génotypes de VPH à haut risque oncologique (HR-HPV), y compris HPV-16 et 18, sont identifiés sans équivoque comme facteur causal principal pour le cancer cervical, et peuvent également causer d'autres cancers anogénitaux, de la tête et du cou. Les infections avec des génotypes de VPH à bas risque oncologique (LR-HPV), comme HPV-6 et -11, sont associées aux lésions bénignes comprenant les verrues génitales. Plusieurs projections de l'impact des nouveaux vaccins de VPH et des techniques de dépistage utilisent des modèles de transmission dynamiques qui exigent des évaluations précises de la probabilité de transmission lors de l'exposition. En outre, les limitations biologiques et pratiques des vaccins courants exigent que nous explorions autant d'options de prévention que possibles. Une question critique de recherches est de savoir si les condoms assurent une protection. Les objectifs principaux de la thèse consistaient à caractériser des modèles typiques d’infection au VPH parmi les couples hétérosexuels dans une nouvelle relation, identifier des facteurs de risque pour l'infection au VPH, et estimer les probabilités de transmission du VPH par relation de couple et par acte coïtal. J'ai effectué une simulation préliminaire de Monte Carlo pour estimer la probabilité de transmission de VPH, puis j’ai conçu et entrepris une étude des couples hétérosexuels. Les objectifs de thèse ont été élaborés en utilisant les données de base de l'étude de cohorte HITCH (HPV Infection and Transmission among Couples through Heterosexual activity) qui se poursuit toujours. La population de l’étude est composée$

APA, Harvard, Vancouver, ISO, and other styles

9

Caparros, Wanderley Wilson. "Development and study of potential immunotherapies against human papillomavirus infection." Thesis, King's College London (University of London), 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.300044.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Bronnimann, Matthew Phillip, and Matthew Phillip Bronnimann. "Penetration of Host Membrane Barriers by Human Papillomavirus During Infection." Diss., The University of Arizona, 2016. http://hdl.handle.net/10150/623159.

Full text

Abstract:

Human Papillomaviruses (HPVs) are circular double-stranded DNA (dsDNA) viruses that infect human cutaneous and mucosal tissue. Most HPV infections are benign or cause only minor pathologies. However, infection with one of the ~15 high risk types of HPV is associated with a variety of head/neck and anogenital cancers. All told, HPV infection is thought to cause ~5% of all human cancers and cause ~275,000 deaths per year. Despite causing immense morbidity and mortality, many aspects of how HPV virions successfully establish infection in host cells remain poorly characterized. Infection begins with HPV virions binding the cell surface, where they are modified by the host protease furin. The HPV virions are then endocytosed by association with an unknown entry receptor(s). After endocytosis the HPV minor capsid protein L2 acts as a chaperone to ensure that the viral genome (vDNA) traffics from endosomes to the trans-Golgi network (TGN) and eventually the nucleus, where HPV replication occurs. En route to the nucleus, the L2/vDNA complex must translocate across limiting intracellular membranes. The details of these critical processes remain poorly characterized. In this work we investigate the viral and host factors involved in the penetration of host membranes by the HPV L2/vDNA complex. First, we elucidated many of the viral and host factors necessary for furin cleavage of L2. We also demonstrate that furin cleavage mediates the homo-oligomerization and membrane insertion of L2. Finally we demonstrate that complete translocation across the limiting membrane is dependent on host cell entry into mitosis. Overall this work provides novel insight into the molecular mechanisms used by HPV virions to breach host membranes and establish infection.

APA, Harvard, Vancouver, ISO, and other styles

More sources

Books on the topic "Human papillomavirus infection"

1

Jong-Tieben, Linda M. de. Human papillomavirus infection and skin cancer in renal transplant recipients. Leiden: University of Leiden, 1998.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

2

Perinot, Glenis Michele. A study of human papillomavirus infection: Recombinant DNA synthesis and analysis. Sudbury, Ont: Laurentian University Press, 1995.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

3

Michael, Paul Simon. PCR screening for human papillomavirus infections, and evaluation of the estimated infection prevalence for a population of females. Sudbury, Ont: Laurentian University, Department of Biology, 1999.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

4

IARC, Working Group on the Evaluation of Carcinogenic Risks to Humans (1995 Lyon France). Human papillomaviruses. [Lyon, France?]: International Agency for Research on Cancer, 1995.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

5

Dizon, Don S. Questions & answers about human papilloma virus (HPV). Sudbury, Mass: Jones and Bartlett Publishers, 2011.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

6

Syrjänen, Kari J. Human papillomavirus (HPV) involvement in esophageal carcinogenesis. New York: Nova Biomedical Books, 2010.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

7

Dizon, Don S. Dx/Rx: Human papilloma virus. Sudbury, MA: Jones & Bartlett Learning, 2011.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

8

Dizon, Don S. Questions & answers about human papilloma virus (HPV). Sudbury, Mass: Jones and Bartlett Publishers, 2011.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

9

R, Stuckey Ashley, and Krychman Michael L, eds. Dx/Rx: Human papilloma virus. Sudbury, MA: Jones & Bartlett Learning, 2011.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

10

Rosenblatt, Alberto. Human Papillomavirus: A Practical Guide for Urologists. Berlin, Heidelberg: Springer-Verlag Berlin Heidelberg, 2009.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

More sources

Book chapters on the topic "Human papillomavirus infection"

1

Suryadevara, Manika. "Human Papillomavirus Infection." In Introduction to Clinical Infectious Diseases, 181–90. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-91080-2_17.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Szalai, Zsuzsanna Z. "Human Papillomavirus Infection." In Harper's Textbook of Pediatric Dermatology, 47.1–47.12. Oxford, UK: Wiley-Blackwell, 2011. http://dx.doi.org/10.1002/9781444345384.ch47.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Brendle, Sarah A., Stephanie M. Bywaters, and Neil D. Christensen. "Pathogenesis of Infection by Human Papillomavirus." In Human Papillomavirus, 47–57. Basel: S. KARGER AG, 2014. http://dx.doi.org/10.1159/000355963.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Ikenberg, Hans. "Laboratory Diagnosis of Human Papillomavirus Infection." In Human Papillomavirus, 166–74. Basel: S. KARGER AG, 2014. http://dx.doi.org/10.1159/000356515.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Stanley, Margaret A., and Jane C. Sterling. "Host Responses to Infection with Human Papillomavirus." In Human Papillomavirus, 58–74. Basel: S. KARGER AG, 2014. http://dx.doi.org/10.1159/000355964.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Tschandl, Philipp, Cliff Rosendahl, and Harald Kittler. "Cutaneous Human Papillomavirus Infection: Manifestations and Diagnosis." In Human Papillomavirus, 92–97. Basel: S. KARGER AG, 2014. http://dx.doi.org/10.1159/000355966.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Rosenblatt, Alberto, and Homero Gustavo de Campos Guidi. "Human Papillomavirus Infection in HIV-Infected Individuals." In Human Papillomavirus, 163–77. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-70974-9_9.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Gross, Gerd. "Genitoanal Human Papillomavirus Infection and Associated Neoplasias." In Human Papillomavirus, 98–122. Basel: S. KARGER AG, 2014. http://dx.doi.org/10.1159/000358423.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Ramírez-Fort, Marigdalia K., Shiu-Chung Au, Syed Ali Javed, and Daniel S. Loo. "Management of Cutaneous Human Papillomavirus Infection: Pharmacotherapies." In Human Papillomavirus, 175–85. Basel: S. KARGER AG, 2014. http://dx.doi.org/10.1159/000356069.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Ramírez-Fort, Marigdalia K., Hakeem Sam, and Ernest K. Manders. "Management of Cutaneous Human Papillomavirus Infection: Surgery." In Human Papillomavirus, 186–96. Basel: S. KARGER AG, 2014. http://dx.doi.org/10.1159/000356070.

Full text

APA, Harvard, Vancouver, ISO, and other styles

Conference papers on the topic "Human papillomavirus infection"

1

Rocha, Willker Menezes da, Camila Freze Baez, Larissa Alves Afonso, Fernanda Nahoum Carestiato, Marianna Tavares Venceslau Gonçalves, Rafael Brandão Varella, and Silvia Maria Baeta Cavalcanti. "The use of DNA microarray assay as a diagnostic tool to study penile cancer associated with human papillomavirus." In XIII Congresso da Sociedade Brasileira de DST - IX Congresso Brasileiro de AIDS - IV Congresso Latino Americano de IST/HIV/AIDS. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/dst-2177-8264-202133p159.

Full text

Abstract:

Introduction: The genital infection by the human papillomavirus (HPV) can result in a sexually transmitted disease associated with precursor lesions for carcinogenesis in the genital tract. In recent years, evidence was accumulated defining HPV as the etiologic agent of cervical cancer; however, the etiology of penile cancer is still open and lacks studies. This study aims to contribute to the epidemiologic knowledge regarding the prevalence of this virus in malignant lesions of the male genital tract, using the DNA microarray assay, a technique that allows the simultaneous detection of up to 32 different HPV genotypes. Objective: The aim of this study was to investigate the presence of HPV in penile malignant lesions, to genotype HPV, when present, to correlate the HPV infection and its genotypes with the histopathological data. Methods: A total of 112 penile cancer samples was collected in a cross-sectional study. The detection methodology consisted of (1) detecting the presence of HPV DNA by the polymerase chain reaction (PCR) technique with generic primers, (2) genotyping the HPV using the DNA microarray assay, and (3) correlation of the histopathology, tumor invasiveness, and the dispersion of malignant cells by the lymph nodes with the presence of HPV. Results: The HPV prevalence was 57.1% (64). The most prevalent genotype was the HPV16 (32.8%), followed by HPV6 (23.4%); HPV18, HPV35, and HPV45 (12.5%); HPV31 (10.9%); and HPV70 (7.8%). Of the HPV-positive samples, 25% were mixed infections. Conclusion: The role of the HPV infection was significant within the multifactorial etiology of penile cancer. There was statistical significance between the lesion invasiveness and the presence of high-risk HPV infection. Thus, genotype surveillance can promote a better understanding of the role of HPV genotypes in male cancer development, and the DNA microarray assay proved to be an efficient tool for both the epidemiological study and the diagnostics of the HPV.

APA, Harvard, Vancouver, ISO, and other styles

2

Safarova, R. I. "Clinical and epidemiological features of pathological processes of the neck uterine infection associated with human papillomavirus infection." In General question of world science. "Science of Russia", 2019. http://dx.doi.org/10.18411/gq-31-07-2019-33.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Liu, Stephanie S., Kelvin KY Chan, Rebecca CY Leung, May HM Luk, Sue ST Lo, Daniel YT Fong, Annie NY Cheung, Zhong Qiu Lin, and Hextan YS Ngan. "Abstract 5732: Human papillomavirus infection in Southern Chinese women - a population-based study." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-5732.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Gupta, Ishita, Ayesha Jabeen, Maria K. Smatti, Hamda A. Al-Thawadi, Gheyath K. Nasrallah, Ali A. Sultan, Moussa Al-Khalaf, Semir Vranic, and Ala-Eddin Al-Moustafa. "Co-Prevalence of Human Papillomavirus and Epstein Barr Virus in Healthy Blood Donors in Qatar." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0120.

Full text

Abstract:

Introduction: Infections with human oncoviruses such as high-risk human papillomaviruses (HPVs) and Epstein-Barr virus (EBV) are globally prevalent in the adult population. Both viruses are strongly associated with several types of human carcinomas such as cervical, head and neck, nasopharyngeal and gastric. In the present study, we explored the prevalence of these two oncoviruses in the healthy population of Qatar. Methods: The study included 385 healthy blood donors that reflect diverse nationalities in the Qatari community (Qatar, Egypt, Syria, Jordan, Pakistan, and India). DNA was extracted from the peripheral blood and genotyping was done using PCR and nested-PCR targeting E6 and E7 as well as LMP1 genes of HPVs and EBV, respectively. Results: The age of participants (378 males and 7 females) ranged between 19 and 68 years (mean 37.12 ± 9.3 years). Our data indicate that 55% and 61% of the tested samples were HPVs and EBV positive, respectively. Moreover, we found that there was (40%) co-presence of both HPVs and EBV in our samples. The most common high-risk HPV types in Qatar included HPV 59 (55%), 31 (54%), 52 (49%), 51 (49%), 58 (47%) and 35 (46%). While, HPV 16 and 18 were detected in 38% and 36% of the samples, respectively. Notably, all samples showed multiple HPVs infections. Conclusion: Our study reveals for the first time a high prevalence of both EBV and HPVs among healthy individuals in Qatar. More significantly, most cases had multiple HPV types infection in addition to the co-presence of both viruses in a substantial proportion of the samples. Given the important possible cooperative role of these viruses in human carcinogenesis, preventive measures using available and upcoming vaccines are of paramount importance.

APA, Harvard, Vancouver, ISO, and other styles

5

Cavalcanti, Silvia Maria Baeta, Thaissa Isaias Cordeiro, Fernanda Nahoum Carestiato, Willker Menezes da Rocha, Daniele Ceperuelo Lisboa, Raquel da Hora Barbosa, Mauro Romero Leal Passos, and Tegnus Depes De Gouvea. "P3.207 Human papillomavirus infection in oral and anogenital sites: prevalence and rates of concordance." In STI and HIV World Congress Abstracts, July 9–12 2017, Rio de Janeiro, Brazil. BMJ Publishing Group Ltd, 2017. http://dx.doi.org/10.1136/sextrans-2017-053264.442.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Cordeiro, Thaissa. "P3.220 Human papillomavirus infection in oral and anogenital sites: prevalence and rates of concordance." In STI and HIV World Congress Abstracts, July 9–12 2017, Rio de Janeiro, Brazil. BMJ Publishing Group Ltd, 2017. http://dx.doi.org/10.1136/sextrans-2017-053264.455.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

RSQ, Simões, Silva EP, and Barth OM. "Lb3.246 Prevalence and risk factors of human papillomavirus infection in unimmunized women in brazil." In STI and HIV World Congress Abstracts, July 9–12 2017, Rio de Janeiro, Brazil. BMJ Publishing Group Ltd, 2017. http://dx.doi.org/10.1136/sextrans-2017-053264.481.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Ondondo, Raphael, Elizabeth Bukusi, Zipporah Nganga, Michael Kiptoo, and Solomon Mpoke. "O16.3 Cytokine immune response against natural human papillomavirus infection among men in kisumu, kenya." In Abstracts for the STI & HIV World Congress (Joint Meeting of the 23rd ISSTDR and 20th IUSTI), July 14–17, 2019, Vancouver, Canada. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/sextrans-2019-sti.195.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Kops, Natalia, Marina Bessel, Gerson Fernando Pereira, Flavia Souza, Adele Benzaken, Luisa Villa, Barbara Mello, and Eliana Wendland. "P828 Use of hormonal contraceptives and genital human papillomavirus infection: a national multicenter study." In Abstracts for the STI & HIV World Congress (Joint Meeting of the 23rd ISSTDR and 20th IUSTI), July 14–17, 2019, Vancouver, Canada. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/sextrans-2019-sti.873.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Rollison, Dana E., Michelle R. Iannacone, Jane L. Messina, Neil A. Fenske, Basil S. Cherpelis, Vernon K. Sondak, Richard G. Roetzheim, et al. "Abstract 4835: Genetic variation inEVER1/EVER2and beta human papillomavirus infection in cutaneous squamous cell carcinoma." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-4835.

Full text

APA, Harvard, Vancouver, ISO, and other styles

Reports on the topic "Human papillomavirus infection"

1

Wang, Huanhuan, Yuyu Zhang, Wei Bai, Bin Wang, Jinlong Wei, Ji Rui, Ying Xin, Lihua Dong, and Xin Jiang. Feasibility of immunohistochemical p16 staining in the diagnosis of human papillomavirus infection in patients with squamous cell carcinoma of the head and neck: A systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, July 2020. http://dx.doi.org/10.37766/inplasy2020.7.0068.

Full text

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!