Relevant bibliographies by topics / Human malaria

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Human malaria'

Author: Grafiati
Published: 4 June 2021
Last updated: 3 February 2022

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Journal articles on the topic "Human malaria"

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BUTCHER, G. A., and G. H. MITCHELL. "The role ofPlasmodium knowlesiin the history of malaria research." Parasitology 145, no. 1 (November 10, 2016): 6–17. http://dx.doi.org/10.1017/s0031182016001888.

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SUMMARYIn recent years, a malaria infection of humans in South East Asia, originally diagnosed as a known human-infecting species,Plasmodium malariae, has been identified as a simian parasite,Plasmodium knowlesi.This species had been subject to considerable investigation in monkeys since the 1930s. With the development of continuous culture of the erythrocytic stages of the human malarial parasite,Plasmodium falciparumin 1976, the emphasis in research shifted away from knowlesi. However, its importance as a human pathogen has provoked a renewed interest inP. knowlesi, not least because it too can be maintained in continuous culture and thus provides an experimental model. In fact, this parasite species has a long history in malaria research, and the purpose of this chapter is to outline approximately the first 50 years of this history.
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Haldar, Kasturi, and Narla Mohandas. "Malaria, erythrocytic infection, and anemia." Hematology 2009, no. 1 (January 1, 2009): 87–93. http://dx.doi.org/10.1182/asheducation-2009.1.87.

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Abstract Malaria is a major world health problem. It results from infection of parasites belonging to the genus Plasmodium. Plasmodium falciparum and Plasmodium vivax cause the major human malarias, with P falciparum being the more virulent. During their blood stages of infection, both P falciparum and P vivax induce anemia. Severe malarial anemia caused by P falciparum is responsible for approximately a third of the deaths associated with disease. Malarial anemia appears to be multi-factorial. It involves increased removal of circulating erythrocytes as well as decreased production of erythrocytes in the bone marrow. The molecular mechanisms underlying malarial anemia are largely unknown. Over the last five years, malaria parasite ligands have been investigated for their remodeling of erythrocytes and possible roles in destruction of mature erythrocytes. Polymorphisms in cytokines have been associated with susceptibility to severe malarial anemia: these cytokines and malaria “toxins” likely function by perturbing erythropoiesis. Finally a number of co-infections increase susceptibility to malarial anemia, likely because they exacerbate inflammation caused by malaria. Because of the complexities involved, the study of severe malarial anemia may need a “systems approach” to yield comprehensive understanding of defects in both erythropoiesis and immunity associated with disease. New and emerging tools such as (i) mathematical modeling of the dynamics of host control of malarial infection, (ii) ex vivo perfusion of human spleen to measure both infected and uninfected erythrocyte retention, and (iii) in vitro development of erythroid progenitors to dissect responsiveness to cytokine imbalance or malaria toxins, may be especially useful to develop integrated mechanistic insights and therapies to control this major and fatal disease pathology.
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Sabbatani, Sergio, Roberto Manfredi, and Sirio Fiorino. "Malaria infection and the anthropological evolution." Saúde e Sociedade 19, no. 1 (March 2010): 64–83. http://dx.doi.org/10.1590/s0104-12902010000100006.

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During the evolution of the genus Homo, with regard to species habilis, erectus and sapiens, malaria infection played a key biological role, influencing the anthropological development too. Plasmodia causing malaria developed two kinds of evolution, according to a biological and philogenetical point of view. In particular, Plasmodium vivax, Plasmodium malariae, and Plasmodium ovale, would have either coevolved with human mankind (coevolution), or reached human species during the most ancient phases of genus Homo evolution. On the other hand, Plasmodium falciparum has been transmitted to humans by monkeys in a more recent period, probably between the end of Mesolithic and the beginning of Neolithic age. The authors show both direct and indirect biomolecular evidences of malaria infection, detected in buried subjects, dating to the Ancient World, and brought to light in the course of archeological excavations in some relevant Mediterranean sites. In this literature review the Authors organize present scientific evidences: these confirm the malarial role in affecting the evolution of populations in Mediterranean countries. The people living in several different regions on the Mediterranean Sea sides, the cradle of western civilization, have been progressively influenced by malaria, in the course of the spread of this endemic disease during the last millennia. In addition, populations affected by endemic malaria developed cultural, dietary and behaviour adaptations, contributing to decrease the risk of disease. These habits were not probably fully conscious. Nevertheless it may be thought that both these customs and biological modifications, caused by malarial plasmodia, favoured the emergence of groups of people with a greater resistance against malaria. All these considered factors decreased demographical impact, influencing in a favourable way the general development and growth of civilization.
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Lamikanra, Abigail A., Douglas Brown, Alexandre Potocnik, Climent Casals-Pascual, Jean Langhorne, and David J. Roberts. "Malarial anemia: of mice and men." Blood 110, no. 1 (July 1, 2007): 18–28. http://dx.doi.org/10.1182/blood-2006-09-018069.

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Severe malaria is manifest by a variety of clinical syndromes dependent on properties of both the host and the parasite. In young infants, severe malarial anemia (SMA) is the most common syndrome of severe disease and contributes substantially to the considerable mortality and morbidity from malaria. There is now growing evidence, from both human and mouse studies of malaria, to show that anemia is due not only to increased hemolysis of infected and clearance of uninfected red blood cells (RBCs) but also to an inability of the infected host to produce an adequate erythroid response. In this review, we will summarize the recent clinical and experimental studies of malaria to highlight similarities and differences in human and mouse pathology that result in anemia and so inform the use of mouse models in the study of severe malarial anemia in humans.
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Merrick, Catherine J. "Plasmodium falciparum." Emerging Topics in Life Sciences 1, no. 6 (December 22, 2017): 517–23. http://dx.doi.org/10.1042/etls20170099.

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Plasmodium falciparum is a protozoan parasite that causes the most severe form of human malaria. Five other Plasmodium species can also infect humans — P. vivax, P. malariae, P. ovale curtisi, P. ovale wallikeri and P. knowlesi — but P. falciparum is the most prevalent Plasmodium species in the African region, where 90% of all malaria occurs, and it is this species that causes the great majority of malaria deaths. These were reported by the WHO at 438 000 in 2015 from an estimated 214 million cases; importantly, however, figures for the global burden of malaria tend to have wide margins of error due to poor and inaccurate reporting. In this Perspective, features of P. falciparum that are unique among human malaria parasites are highlighted, and current issues surrounding the control and treatment of this major human pathogen are discussed.
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VON MÜHLEN, André. "COMPUTER IMAGE ANALYSIS OF MALARIAL PLASMODIUM VIVAX IN HUMAN RED BLOOD CELLS." Periódico Tchê Química 02, no. 1 (August 20, 2004): 42–51. http://dx.doi.org/10.52571/ptq.v1.n02.2004.agosto/9_pgs_42_51.pdf.

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This project aims to study computer image analysis of malarial parasites using morphological operators as its main method of approach. Malaria is a life-threatening parasitic disease transmitted through female Anopheles mosquitoes (1). It is found throughout the tropical and subtropical regions of the world (figure 1), and affects over 300 million people annually. As globalization, ease and frequency of travel increase, malaria cases may occur in any country (2). Over one million people die due to malaria every year (3).
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Aikawa, Masamichi. "Human Cerebral Malaria *." American Journal of Tropical Medicine and Hygiene 39, no. 1 (July 1, 1988): 3–10. http://dx.doi.org/10.4269/ajtmh.1988.39.3.

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OO, MAUNG MAUNG, MASAMICHI AIKAWA, THAN THAN, TIN MAUNG AYE, PE THAN MYINT, IKUO IGARASHI, and WILLIAM C. SCHOENE. "Human Cerebral Malaria." Journal of Neuropathology and Experimental Neurology 46, no. 2 (March 1987): 223–31. http://dx.doi.org/10.1097/00005072-198703000-00009.

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Slater, A. "Human malaria parasites." Biomedicine & Pharmacotherapy 46, no. 10 (January 1992): 502. http://dx.doi.org/10.1016/0753-3322(92)90014-x.

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Singh, Balbir. "Plasmodium knowlesi: an update." Microbiology Australia 37, no. 1 (2016): 39. http://dx.doi.org/10.1071/ma16014.

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There were only four species of Plasmodium that were thought to cause malaria in humans until a large number of human infections by Plasmodium knowlesi, a malaria parasite typically found in long-tailed and pig-tailed macaques, were reported in 2004 in Malaysian Borneo. Since then, cases of knowlesi malaria have been reported throughout South-east Asia and also in travellers returning from the region. This article describes the molecular, entomological and epidemiological data which indicate that P. knowlesi is an ancient parasite that is primarily zoonotic, and there are three highly divergent sub-populations. It also describes the detection methods for P. knowlesi, which is morphologicaly similar to P. malariae, and the clinical features and treatment of this malaria parasite that is potentially fatal.
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Dissertations / Theses on the topic "Human malaria"

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Hodgson, Susanne H. "Using the controlled human malaria infection model to investigate immunity to malaria." Thesis, University of Oxford, 2015. https://ora.ox.ac.uk/objects/uuid:b3c9a2eb-beab-4ef6-bd8d-483390f316b8.

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Controlled human malaria infection (CHMI) studies, where healthy volunteers are infected with Plasmodium falciparum have become a vital tool to accelerate vaccine and drug development. As CHMI trials are carried out in a controlled environment, they allow unprecedented, detailed evaluation of parasite growth dynamics and immunological responses to infection. Though commonly performed in malaria-naïve populations, CHMI trials have rarely been conducted in malaria-endemic regions and to date, have not been used to investigate naturally acquired immunity (NAI) to P. falciparum infection. This thesis describes the first CHMI study in Kenya and the first attempt to use the modern CHMI model to explore the dynamics and mechanisms of NAI. Using samples collected post-CHMI from both UK volunteers and Kenyan subjects with varying prior exposure to P. falciparum, this work reports and compares the findings of key in vitro assays including GIA, ADRB activity and changes in gene expression in order to understand the effect of NAI on these measures.
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Bruce, Marian Cooke. "Intra-host dynamics of human malaria parasites." Thesis, University of Oxford, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.298193.

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Goncalves, B. "The human infectious reservoir of Falciparum malaria." Thesis, London School of Hygiene and Tropical Medicine (University of London), 2017. http://researchonline.lshtm.ac.uk/4646827/.

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Malaria control strategies are often targeted at individuals who suffer most morbidity, not at those driving transmission. This is in part due to our limited understanding, described in Chapter 1, of the human infectious reservoir of falciparum malaria – i.e. individuals responsible for human-to-mosquito transmission of Plasmodium falciparum parasites in endemic areas. This work, whose objectives are listed in Chapter 2, assessed the prevalence of infectiousness in naturally exposed human populations during dry and wet seasons. Exposure to Anopheles mosquitoes, a measure of transmission opportunities, was also quantified; and to determine the value of infectiousness-reducing interventions, the use of primaquine to block transmission from infectious individuals was investigated. Experimental infections of mosquitoes were performed to determine malaria infectivity of randomly selected individuals in two villages in Burkina Faso. Molecular assays were used to quantify parasite, including gametocyte, densities. Less than 10% of the population was infectious to mosquitoes. These results are presented in a manuscript that included data from other study sites (Chapter 3). To assess exposure to malaria vectors, bloodfed mosquitoes were collected indoors in one of the study villages in Burkina Faso. A multiplex PCR assay targeting nine human microsatellites and a gender-specific marker was used to identify the human sources of mosquito blood meals. Although there was substantial variation in the number of mosquito bites each individual received (Chapter 4), on average adults received more mosquito bites than children. This suggests that, despite their lower infectiousness, adults are major contributors to malaria transmission in endemic areas. An efficacy trial of single low dose primaquine was performed in Burkina Faso and pre- and post-treatment infectiousness were quantified by mosquito feeding experiments to assess primaquine’s infectiousness-reducing activity (Chapter 5). Individuals receiving primaquine cleared gametocytes faster than individuals who received artemether-lumefantrine alone. Feeding assays, however, suggest that artemether-lumefantrine blocks most parasite transmission after treatment administration. In Chapter 6, these findings, and how they can inform future control strategies, are discussed.
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Weber, Grace E. "Memory B Cell Dysfunction in Human Malaria." Case Western Reserve University School of Graduate Studies / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=case1512731469728517.

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Kaneko, Akira. "Malaria on islands : human and parasite diversities and implications for malaria control in Vanuatu /." Stockholm, 1999. http://diss.kib.ki.se/1999/19990927kane.

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Nisar, Samia. "Role of ATP2B4 and human malaria : looking for functional genetic variants associated with malaria." Thesis, Aix-Marseille, 2020. http://theses.univ-amu.fr.lama.univ-amu.fr/200911_NISAR_992dobfs271wcdsgy656twqjfn399ockic_TH.pdf.

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GWAS pour le paludisme grave identifié 30 variantes génétiques situées dans régions non codantes, avec seulement quelques associations répliquées dans des populations indépendantes. Dans cette étude, nous avons cherché à identifier les variantes génétiques potentielles situées dans ces loci et à démontrer leur activité fonctionnelle. Nous avons systématiquement étudié l'effet régulateur des SNP en déséquilibre liaison avec les tagSNPs associés au paludisme sévère dans plusieurs populations. L'annotation et priorisation ont conduit à l'identification d'une région régulatrice contenant 5 SNP ATP2B4 en déséquilibre liaison avec le tagSNP. Nous confirmé l'association de rs10900585 et trouvé des associations significatives de paludisme sévère avec nos candidats dans population sénégalaise. Nous montré que cette région avait à la fois une activité promoteur et un activateur et que l'individu et combinaison de SNP avaient un effet en utilisant des dosages de luciférase. En outre, la délétion médiée par CRISPR / Cas9 de cette région a diminué le transcrit ATP2B4 et les niveaux de protéines et a augmenté la concentration intracellulaire de Ca2+ dans les cellules K562. Ensemble, nos données montrent les variantes génétiques associées au paludisme grave modifient l'activité d'un promoteur avec une fonction d'activateur. Nous montré que cet amplificateur contrôle l'expression de l'ATP2B4 qui code l'ATPase 4 (PMCA4) transportant le calcium dans la membrane plasmique, qui est la principale pompe à calcium des globules rouges. La modification de l'activité de cet Epromoter affecte le risque de paludisme sévère probablement par l'effet de la concentration de calcium sur la parasitémie
Genome-wide association studies (GWAS) for severe malaria have identified 30 genetic variants mostly located in non-coding regions, with only few associations replicated in independent populations. In this study, we aimed at identifying potential causal genetic variants located in these loci and demonstrate their functional activity. We systematically investigated the regulatory effect of the SNPs in linkage disequilibrium with the tagSNPs associated with severe malaria in several populations. Annotating and prioritizing genetic variants led to the identification of a regulatory region containing 5 ATP2B4 SNPs in linkage disequilibrium with the tagSNP rs10900585. We confirmed the association of rs10900585 and also found significant associations of severe malaria with our candidate SNPs (rs11240734, rs1541252, rs1541253, rs1541254, and rs1541255) in a Senegalese population. Then, we showed that this region had both a promoter and an enhancer activity and that both individual SNPs and the combination of SNPs had an effect using luciferase reporter assays. In addition, CRISPR/Cas9-mediated deletion of this region decreased ATP2B4 transcript and protein levels and increased Ca2+ intracellular concentration in K562 cell line. Taken together, our data show that severe malaria associated genetic variants alters the activity of a promoter with enhancer function. We showed that this enhancer controls the expression of ATP2B4 that encodes plasma membrane calcium-transporting ATPase 4 (PMCA4), which is the major calcium pump on red blood cells. Altering the activity of this Epromoter affects the risk of severe malaria probably through calcium concentration effect on parasitaemia
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Walker, Alison Dalgity. "Protein variation in the malaria parasite Plasmodium falciparum." Thesis, University of Edinburgh, 1986. http://hdl.handle.net/1842/13171.

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Berthoud, Tamara Katherine. "Human cellular immune responses to candidate malaria vaccines." Thesis, University of Oxford, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.445763.

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Buitrago, Amanda Elena Maestre. "Immunity to malaria using the rodent malaria parasite Plasmodium chabaudi AS as a model of the human malaria Plasmodium falciparum." Thesis, University of Glasgow, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.298916.

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Maestre, Buitrago Amanda Elena. "Immunity to malaria using the rodent malaria parasite Plasmodium chabaudi chabaudi AS as a model of the human malaria Plasmodium falciparum." Thesis, University of Glasgow, 1997. http://theses.gla.ac.uk/2036/.

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The role of IFN in acquisition of immunity against erythrocyte forms of P.c. chabaudi AS was studied. Inbred NIH mice given the construct 7 days before malaria infection, showed a significant delay in the onset and in the level of the recrudescent parasitaemia in comparison with controls. No differences, however, were observed in the recrudescent parasitaemia between the groups. NIH mice infected with malaria 3 days after or on the same day as the administration of the IFN construct, showed a primary peak of infection similar to controls, but the resolution of this patent parasitaemia occurred 1 or 2 days earlier in the experimental mice when compared with controls. In the same experiment, mice given the construct 10 days before malaria infection had a similar course of infection as controls. Simultaneous inoculation with two S. typhimurium constructs: IFN and TNF, 8 days before malaria infection resulted in a course of parasitaemia similar to that observed in mice given the IFN construct alone. On the other hand, inoculation of 'susceptible' inbred A/J mice with S. typhimurium/IFN 3 or 8 days before malaria infection had no effect on the course of the parasitaemia when compared with controls. The immune mechanisms involved in the better control of the malaria infection of NIH mice given S. typhimurium/ IFN, seem to be independent of nitric oxide (NO) production, since increased levels of the molecule were demonstrable around the peak of the primary parasitaemia in control groups but not in experimental mice. In the latter basal levels of serum NO were observed from the period after the S. typhimurium/ IFN inoculation until up to three days after the peak of the parasitaemia.
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Books on the topic "Human malaria"

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Sharma, V. P. Seroepidemiology of human malaria: A multicentric study. Delhi: Malaria Research Centre, ICMR, 1989.

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Oldstone, Michael B. A., ed. Cytotoxic T-Lymphocytes in Human Viral and Malaria Infections. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-78530-6.

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Payne, David. Biosafety in vivo and in vitro studies of human malaria. [Geneva]: World Health Organization, 1990.

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Iqbal, M. P. Investigation of the TFIIB and fibrillarin genes in the human malaria parasite plasmodium falciparum. Manchester: UMIST, 1996.

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Fighting malaria: Progress and challenges : hearing before the Subcommittee on Africa, Global Health, and Human Rights of the Committee on Foreign Affairs, House of Representatives, One Hundred Twelfth Congress, first session, December 5, 2011. Washington: U.S. G.P.O., 2011.

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The ten-thousand year fever: Rethinking human and wild primate malarias. Walnut Creek, CA: Left Coast Press, 2011.

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Qing dai Yunnan zhang qi yu sheng tai bian qian yan jiu. Beijing: Zhongguo she hui ke xue chu ban she, 2007.

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United States. Congress. House. Committee on Foreign Affairs. Subcommittee on Africa, Global Health, Global Human Rights, and International Organizations. The U.S. contribution to the fight against malaria: Hearing and meeting before the Subcommittee on Africa, Global Health, Global Human Rights, and International Organizations of the Committee on Foreign Affairs, House of Representatives, One Hundred Thirteenth Congress, first session, May 17, 2013. Washington: U.S. Government Printing Office., 2013.

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United States. Congress. House. Committee on International Relations. Subcommittee on Africa, Global Human Rights, and International Operations. Malaria and TB: Implementing proven treatment and eradication methods : hearing before the Subcommittee on Africa, Global Human Rights, and International Operations of the Committee on International Relations, House of Representatives, One Hundred Ninth Congress, first session, April 26, 2005. Washington: U.S. G.P.O., 2005.

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Mosquito empires: Ecology and war in the Greater Caribbean, 1620 - 1914. New York: Cambridge University Press, 2010.

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Book chapters on the topic "Human malaria"

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Vekemans, Johan. "Immune Responses to the RTS,S/AS01 Malaria Vaccine Candidate: Lessons from Human Immunology, Parasitologic and Clinical Evaluations." In Malaria, 139–56. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-45210-4_7.

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Salman, Ahmed M., Catherin Marin Mogollon, Jing-wen Lin, Fiona J. A. van Pul, Chris J. Janse, and Shahid M. Khan. "Generation of Transgenic Rodent Malaria Parasites Expressing Human Malaria Parasite Proteins." In Malaria Vaccines, 257–86. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2815-6_21.

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Foquet, Lander, Philip Meuleman, Cornelus C. Hermsen, Robert Sauerwein, and Geert Leroux-Roels. "Assessment of Parasite Liver-Stage Burden in Human-Liver Chimeric Mice." In Malaria Vaccines, 59–68. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2815-6_5.

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Timmann, Christian, and Christian G. Meyer. "Human Genetic Factors and Resistance to Malaria." In Encyclopedia of Malaria, 1–16. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4614-8757-9_138-1.

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Ndour, Papa Alioune, Innocent Safeukui, Seidina Diakité, Julien Duez, Stéphane Jauréguiberry, and Pierre Buffet. "Role of the Spleen in Human Malaria." In Encyclopedia of Malaria, 1–24. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4614-8757-9_89-1.

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Wang, Ying. "Malaria in China." In Treatment of Human Parasitosis in Traditional Chinese Medicine, 53–65. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-39824-7_5.

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Williams, Thomas N. "Human Genetic Resistance to Malaria." In Advances in Experimental Medicine and Biology, 243–53. New York, NY: Springer US, 2008. http://dx.doi.org/10.1007/978-0-387-79838-7_20.

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Giribaldi, Giuliana, Sarah D’Alessandro, Mauro Prato, and Nicoletta Basilico. "Etiopathogenesis and Pathophysiology of Malaria." In Human and Mosquito Lysozymes, 1–18. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-09432-8_1.

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Marinotti, Osvaldo, and Anthony A. James. "The Transcriptome of Human Malaria Vectors." In Molecular Approaches to Malaria, 516–30. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555817558.ch27.

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Mellanby, Kenneth. "Chapter 15 Malaria." In Human Guinea Pigs, by Kenneth Mellanby: A Reprint with Commentaries, 97–100. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-37697-0_17.

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Conference papers on the topic "Human malaria"

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Sa-ngamuang, Chaitawat. "Human mobility and malaria transmission." In 2020 IEEE International Conference on Healthcare Informatics (ICHI). IEEE, 2020. http://dx.doi.org/10.1109/ichi48887.2020.9374375.

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Soyemi, Jumoke, Itunuoluwa Isewon, Jelili Oyelade, and Ezekiel Adebiyi. "Functional enrichment of human protein complexes in malaria parasites." In 2017 International Conference on Computing Networking and Informatics (ICCNI). IEEE, 2017. http://dx.doi.org/10.1109/iccni.2017.8123791.

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Addawe, Joel, and Aprimelle Kris Pajimola. "Dynamics of climate-based malaria transmission model with age-structured human population." In THE 4TH INTERNATIONAL CONFERENCE ON QUANTITATIVE SCIENCES AND ITS APPLICATIONS (ICOQSIA 2016). Author(s), 2016. http://dx.doi.org/10.1063/1.4966069.

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Joseph, G. Arul, and S. Balamuralitharan. "Global stability analysis of human SEIV-mosquitoes SEI model for malaria transmission." In 1ST INTERNATIONAL CONFERENCE ON MATHEMATICAL TECHNIQUES AND APPLICATIONS: ICMTA2020. AIP Publishing, 2020. http://dx.doi.org/10.1063/5.0025240.

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Ohm, Johanna R. "What is 'fitness'? Defining fitness and appropriate proxies for mosquitoes that transmit human malaria." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.114615.

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Kim, Kyoohyun, HyeOk Yoon, and YongKeun Park. "3-D Imaging of Malaria-infected Human Red Blood Cells Using Optical Diffraction Tomography." In CLEO: Science and Innovations. Washington, D.C.: OSA, 2014. http://dx.doi.org/10.1364/cleo_si.2014.sm4p.2.

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Beay, Lazarus Kalvein, Kasbawati, and Syamsuddin Toaha. "Effects of human and mosquito migrations on the dynamical behavior of the spread of malaria." In SYMPOSIUM ON BIOMATHEMATICS (SYMOMATH 2016). Author(s), 2017. http://dx.doi.org/10.1063/1.4978975.

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Custer, Jonathan R., Michael Kariuki, Brenda T. Beerntsen, and John A. Viator. "Photoacoustic detection of hemozoin in human mononuclear cells as an early indicator of malaria infection." In BiOS, edited by Alexander A. Oraevsky and Lihong V. Wang. SPIE, 2010. http://dx.doi.org/10.1117/12.841399.

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Diyasa, I. Gede Susrama Mas, Akhmad Fauzi, Ariyono Setiawan, Moch Idhom, Radical Rakhman Wahid, and Alfath Daryl Alhajir. "Pre-trained Deep Convolutional Neural Network for Detecting Malaria on the Human Blood Smear Images." In 2021 International Conference on Artificial Intelligence in Information and Communication (ICAIIC). IEEE, 2021. http://dx.doi.org/10.1109/icaiic51459.2021.9415183.

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Rakshit, Pranati, and Kriti Bhowmik. "Detection of presence of parasites in human RBC in case of diagnosing malaria using image processing." In 2013 IEEE Second International Conference on Image Information Processing (ICIIP). IEEE, 2013. http://dx.doi.org/10.1109/iciip.2013.6707610.

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Reports on the topic "Human malaria"

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Magowan, C., J. T. Brown, N. Mohandas, and W. Meyer-Ilse. X-ray microscopy of human malaria. Office of Scientific and Technical Information (OSTI), April 1997. http://dx.doi.org/10.2172/603461.

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Obaldia, Nicanor. Evaluation of Drug and Vaccine Candidates in the Human Malaria/Aotus Monkey Model. Fort Belvoir, VA: Defense Technical Information Center, July 2001. http://dx.doi.org/10.21236/ada393971.

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Obaldia III, Nicanor. Evaluation of Drug and Vaccine Candidates in the Human Malaria/Aotus Monkey Model. Fort Belvoir, VA: Defense Technical Information Center, March 1999. http://dx.doi.org/10.21236/ada373724.

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Prachumsri, Jetsumon. Proteomic Study of Human Malaria Parasite Plasmodium Vivax Liver Stages for Development of Vaccines and Drugs. Fort Belvoir, VA: Defense Technical Information Center, October 2008. http://dx.doi.org/10.21236/ada494445.

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Bautista, José Manuel. Adaptación evolutiva a la malaria en el ser humano (Especial Premio Lasker). Sociedad Española de Bioquímica y Biología Molecular (SEBBM), November 2011. http://dx.doi.org/10.18567/sebbmdiv_anc.2011.11.1.

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S. Abdellatif, Omar, Ali Behbehani, and Mauricio Landin. Malaysia COVID-19 Governmental Response. UN Compliance Research Group, September 2021. http://dx.doi.org/10.52008/mly0501.

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Abstract:
The International Health Regulations (2005) are legally binding on 196 States Parties, Including all WHO Member States. The IHR aims to keep the world informed about public health risks, through committing all signatories to cooperate together in combating any future “illness or medical condition, irrespective of origin or source, that presents or could present significant harm to humans.” Under IHR, countries agreed to strengthen their public health capacities and notify the WHO of any such illness in their populations. The WHO would be the centralized body for all countries facing a health threat, with the power to declare a “public health emergency of international concern,” issue recommendations, and work with countries to tackle a crisis. Although, with the sudden and rapid spread of COVID-19 in the world, many countries varied in implementing the WHO guidelines and health recommendations. While some countries followed the WHO guidelines, others imposed travel restrictions against the WHO’s recommendations. Some refused to share their data with the organization. Others banned the export of medical equipment, even in the face of global shortages. The UN Compliance Research group will focus during the current cycle on analyzing the compliance of the WHO member states to the organizations guidelines during the COVID-19 pandemic.
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