Log in

Relevant bibliographies by topics / Human herpes virus / Journal articles

To see the other types of publications on this topic, follow the link: Human herpes virus.

Journal articles on the topic 'Human herpes virus'

Author: Grafiati

Published: 4 June 2021

Last updated: 20 February 2023

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 50 journal articles for your research on the topic 'Human herpes virus.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse journal articles on a wide variety of disciplines and organise your bibliography correctly.

1

Baz, Kıymet Baz, Gönül Aslan, Ayşegül Usta Güney, Seda Tezcan, Ayça Cordan Yazıcı, Bahar Taşdelen, Güliz İkizoğlu, and Gürol Emekdaş. "Human Herpes Virus-6 and Human Herpes Virus -7 in Pityriasis Rosea." Turkish Journal of Dermatology / Türk Dermatoloji Dergisi 7, no. 3 (September 5, 2013): 142–44. http://dx.doi.org/10.4274/tdd.1611.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Kelly, Teresa, and Saurabh Guleria. "Human herpes virus-6 encephalitis." Journal of Pediatric Neuroradiology 01, no. 02 (July 28, 2015): 143–44. http://dx.doi.org/10.3233/pnr-2012-022.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Bragina, E. E. "Viral infection of sperm. Part 2. Human herpes viruses, human immunodeficiency virus, hepatitis C virus, Zika virus (review)." Andrology and Genital Surgery 21, no. 4 (February 12, 2021): 20–30. http://dx.doi.org/10.17650/2070-9781-2020-21-4-20-30.

Full text

Abstract:

Intragametal viral infection of spermatozoa can cause true vertical transmission of viruses through germ cells. Currently, human immunodeficiency virus, hepatitis C viruses, herpes simplex virus, cytomegalovirus, Zika virus have been detected in spermatozoa. The possibility of vertical transmission of human immunodeficiency virus, cytomegalovirus, herpes simplex virus and Zika virus has been proven.Intragametal infection of spermatozoa with viruses of the herpes group leads to abnormalities in the development of the embryo and can cause spontaneous abortions both during natural conception and when using assisted reproductive technologies.The development of adequate methods for diagnosing an intragametal spermatozoa virus infection will make it possible to find out, at least in some patients, the cause of infertility and pregnancy abnormalities and apply appropriate antiviral therapy in preparation for natural conception or the use of assisted reproductive technologies.

APA, Harvard, Vancouver, ISO, and other styles

4

Tanaka, Toshio, Kazuhiro Kogawa, Hidenori Sasa, Shigeaki Nonoyama, Kenichi Furuya, and Kenji Sato. "Rapid and simultaneous detection of 6 types of human herpes virus (herpes simplex virus, varicella-zoster virus, Epstein-Barr virus, cytomegalovirus, human herpes virus 6A/B, and human herpes virus 7) by multiplex PCR assay." Biomedical Research 30, no. 5 (2009): 279–85. http://dx.doi.org/10.2220/biomedres.30.279.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Ali, Jwan A. "Detection of Human Herpes Virus-6 in Children Suffering from Febrile Convulsion." International Journal of Psychosocial Rehabilitation 24, no. 5 (April 20, 2020): 3043–49. http://dx.doi.org/10.37200/ijpr/v24i5/pr202010.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Sunur, Stella, Izazi Hari Purwoko, Yulia Farida Yahya, and Raden Pamudji. "Genital Herpes in Human Immunodeficiency Virus Infected Patients." Bioscientia Medicina : Journal of Biomedicine and Translational Research 5, no. 3 (May 4, 2021): 764–78. http://dx.doi.org/10.32539/bsm.v5i3.342.

Full text

Abstract:

Genital herpes is a recurrent, lifelong sexual transmitted infection caused by HSV, especially type 2. Genital herpes is the most common infection in HIV patient. HSV-2 can increase the risk of HIV acquisition 2 to 3 times. Clinical manifestations of genital herpes can be different between HIV- infected and non-HIV patients. HIV-infected patients have a high risk of developing chronic and severe genital ulcers with atypical manifestation, prolonged healing, and resistant to treatment, depends on CD4 count. Genital herpes can be diagnosed based on history, clinical manifestation, laboratory and histopathologic examination. Management of genital herpes includes education and counseling patients and sexual partners, systemic antiviral, and symptomatic treatment.

APA, Harvard, Vancouver, ISO, and other styles

7

Sunur, Stella, Izazi Hari Purwoko, Yulia Farida Yahya, and Raden Pamudji. "Genital Herpes in Human Immunodeficiency Virus Infected Patients." Bioscientia Medicina : Journal of Biomedicine and Translational Research 5, no. 8 (May 4, 2021): 758–72. http://dx.doi.org/10.32539/bsm.v5i8.342.

Full text

Abstract:

Genital herpes is a recurrent, lifelong sexual transmitted infection caused by HSV, especially type 2. Genital herpes is the most common infection in HIV patient. HSV-2 can increase the risk of HIV acquisition 2 to 3 times. Clinical manifestations of genital herpes can be different between HIV- infected and non-HIV patients. HIV-infected patients have a high risk of developing chronic and severe genital ulcers with atypical manifestation, prolonged healing, and resistant to treatment, depends on CD4 count. Genital herpes can be diagnosed based on history, clinical manifestation, laboratory and histopathologic examination. Management of genital herpes includes education and counseling patients and sexual partners, systemic antiviral, and symptomatic treatment.

APA, Harvard, Vancouver, ISO, and other styles

8

Nikolskiy, M. A. "THE HUMAN HERPES VIRUS TYPE 7." Russian Journal of Infection and Immunity 3, no. 1 (July 7, 2014): 15. http://dx.doi.org/10.15789/2220-7619-2013-1-15-20.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Okuno, T., H. Sao, and K. Yamanishi. "Human herpes virus 6(HHV-6)." Uirusu 41, no. 2 (1991): 65–76. http://dx.doi.org/10.2222/jsv.41.65.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Sanghavi, Akta, Deepak Dave, Prasad Nadig, Tulsi Sanghavi, and Nirali Khanpara. "Human herpes virus: Bacteria and periodontium." Journal of Oral Disease Marker 1, no. 1 (2017): 5–9. http://dx.doi.org/10.15713/ins.jodm.3.

Full text

APA, Harvard, Vancouver, ISO, and other styles

11

Galich, E. N., I. L. Solovyeva, A. I. Kuselman, А. А. Solovyeva, K. A. Zakuraeva, and V. A. Lankov. "Phagocytosis and Antibody Mediated Immunity in Children Infected with Human Herpes Virus 6." Doctor.Ru 160, no. 5 (2019): 23–26. http://dx.doi.org/10.31550/1727-2378-2019-160-5-23-26.

Full text

APA, Harvard, Vancouver, ISO, and other styles

12

Bagg, Jeremy. "Human herpesvirus-6: the latest human herpes virus." Journal of Oral Pathology and Medicine 20, no. 10 (November 1991): 465–68. http://dx.doi.org/10.1111/j.1600-0714.1991.tb00404.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

13

Prou, O., and A. M. Couroucé. "Human B lymphotropic virus (HBLV) ou Human Herpes Virus (HHV.6)." Revue Française de Transfusion et d'Hémobiologie 32, no. 3 (June 1989): 203–13. http://dx.doi.org/10.1016/s1140-4639(89)80041-3.

Full text

APA, Harvard, Vancouver, ISO, and other styles

14

Díaz-García, Juan Daniel, Juan Pablo Venzor-Castellanos, Karen Hopf-Estandia, and Eunice Rojas-Zaldívar. "PLasmablastic lymphoma with coinfection by Epstein Barr Virus and Herpes Human Virus 8: a case report." ACTUALIDAD MEDICA 104, no. 808 (December 31, 2019): 188–90. http://dx.doi.org/10.15568/am.2019.808.cc03.

Full text

APA, Harvard, Vancouver, ISO, and other styles

15

Golden, M. P., S. Kim, S. M. Hammer, E. A. Ladd, P. A. Schaffer, N. DeLuca, and M. A. Albrecht. "Activation of Human Immunodeficiency Virus by Herpes Simplex Virus." Journal of Infectious Diseases 166, no. 3 (September 1, 1992): 494–99. http://dx.doi.org/10.1093/infdis/166.3.494.

Full text

APA, Harvard, Vancouver, ISO, and other styles

16

Lachmann, Robin. "Herpes simplex virus latency." Expert Reviews in Molecular Medicine 5, no. 29 (December 5, 2003): 1–14. http://dx.doi.org/10.1017/s1462399403006975.

Full text

Abstract:

Herpes simplex virus type 1 (HSV-1) and type 2 (HSV-2) are ubiquitous human pathogens. They share with other herpesviruses the ability to establish lifelong latent infection of the host. Periodic reactivation from latency is responsible for most of the clinical disease burden of HSV infection. This review focuses on what we have learned from molecular studies in model systems of HSV latency, and the implications these findings have for treating recurrent HSV disease.

APA, Harvard, Vancouver, ISO, and other styles

17

Marlina, Erni, Ali Yusran, and Zohra Nazaruddin. "Diagnosis dan tatalaksana nyeri pada rongga mulut yang disebabkan oleh infeksi virus herpes Diagnosis and management of pain in oral cavity caused by herpes virus infection." Journal of Dentomaxillofacial Science 11, no. 1 (February 28, 2012): 33. http://dx.doi.org/10.15562/jdmfs.v11i1.291.

Full text

Abstract:

There are 80 types of known herpes virus, 8 of them can cause infection on humans. They are herpes simplex virus(HSV) 1 and 2, varicella zoster virus (VZV), cytomegalovirus, Epstein-Barr virus, human herpes virus (HHV6) Aand B, and paramyxovirus. HSV1, HSV2, and VZV are the virus known to cause oral mucosal diseases. This paperaims to review and discuss orofacial pain caused by herpes virus infection. Detail anamnesis about prodromal signand symptom with clinical features that vesicles, labial and intraoral lesions, and unilateral distribution of lesionsare characterized oral herpes virus infections. It can be concluded that detailed anamnesis and an understandingabout oral clinical sign and symptom may confirm diagnosis of herpes virus infections.

APA, Harvard, Vancouver, ISO, and other styles

18

DEATLY, A. M., A. T. HAASE, P. H. FEWSTER, E. LEWIS, and M. J. BALL. "Human herpes virus infections and Alzheimer's disease." Neuropathology and Applied Neurobiology 16, no. 3 (June 1990): 213–23. http://dx.doi.org/10.1111/j.1365-2990.1990.tb01158.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

19

Buchbinder, S. P., M. H. Katz, N. A. Hessol, J. Y. Liu, P. M. O'Malley, R. Underwood, and S. D. Holmberg. "Herpes Zoster and Human Immunodeficiency Virus Infection." Journal of Infectious Diseases 166, no. 5 (November 1, 1992): 1153–56. http://dx.doi.org/10.1093/infdis/166.5.1153.

Full text

APA, Harvard, Vancouver, ISO, and other styles

20

DUA, H. S. "Herpes simplex virus in the human cornea." British Journal of Ophthalmology 84, no. 6 (June 1, 2000): 560–61. http://dx.doi.org/10.1136/bjo.84.6.560.

Full text

APA, Harvard, Vancouver, ISO, and other styles

21

SANCHETEE, PC, and YD SINGH. "HERPES ZOSTER IN HUMAN IMMUNODEFICIENCY VIRUS INFECTION." Medical Journal Armed Forces India 52, no. 1 (January 1996): 1–2. http://dx.doi.org/10.1016/s0377-1237(17)30823-7.

Full text

APA, Harvard, Vancouver, ISO, and other styles

22

Porter, S. R., L. Di Alberti, and N. Kumar. "Human herpes virus 8 (Kaposi’s sarcoma herpesvirus)." Oral Oncology 34, no. 1 (January 1998): 5–14. http://dx.doi.org/10.1016/s1368-8375(97)00038-9.

Full text

APA, Harvard, Vancouver, ISO, and other styles

23

Moore, F. G. A., and C. Wolfson. "Human herpes virus 6 and multiple sclerosis." Acta Neurologica Scandinavica 106, no. 2 (June 27, 2002): 63–83. http://dx.doi.org/10.1034/j.1600-0404.2002.01251.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

24

Tolstano, Axel A., José Luis Villarreal C, Franklin E. Torres J, Edward Lozano H., Nelys P. Movilla P, and Lina M. Barroso M. "DIAGNÓSTICO MOLECULAR DE INFECCIÓN POR VIRUS HERPES SIMPLEX TIPO 1 EN TEJIDO ATEROSCLERÓTICO HUMANO." Biociencias 13, no. 2 (December 5, 2018): 97–110. http://dx.doi.org/10.18041/2390-0512/biociencias.2.5001.

Full text

Abstract:

Introducción: Las enfermedades del sistema circulatorio representan uno de los mayores problemas de salud pública a nivel mundial, nacional y regional. El mecanismo patogénico que subyace esta patología es la aterosclerosis. Existen varios factores que favorecen la etiopatogenia de la lesión aterosclerótica. Las infecciones, juegan un papel importante. La infección por el Virus del Herpes Simplex se ha considerado como un factor de riesgo emergente. Objetivo: Realizar diagnóstico molecular de infección por Virus Herpes Simplex tipo 1 y tipo 2 en tejido aterosclerótico humano. Método: Se realizó extracción de ADN viral a partir de ateromas usando el kit comercial PureGenomeTM Tissue DNA Extraction. La amplificación del material genético viral se realizó por PCR en tiempo real (qPCR) con el kit comercial “Human Herpes Virus 2 (Herpes simplex type 2) UL36 region genesig Standard Kit y Human Herpes Virus 1 (Herpes simplex type 1) Capsid assembly and DNA maturation gene. Genesig Standard Kit”. Resultados: En total se obtuvieron 102 muestras de ateromas, extraídas de diferentes fuentes anatómicas. Tres muestras resultaron positivas para VHS tipo 1 (3/102). Ninguna muestra evidenció material genético para VHS tipo 2 (0/102). Conclusión: La etiopatogenia de la aterosclerosis es un proceso altamente complejo. Los virus juegan un papel importante, en especial la infección por Virus del herpes simplex tipo 1. La infección por este virus genera cambios a nivel de las células vasculares y no vasculares, favoreciendo el acumulo de lipoproteínas de baja densidad químicamente oxidadas, importantes para la aterogénesis.

APA, Harvard, Vancouver, ISO, and other styles

25

Jovanović, Marina. "Genital Herpes / Genitalni herpes." Serbian Journal of Dermatology and Venerology 3, no. 1 (January 1, 2011): 7–22. http://dx.doi.org/10.2478/v10249-011-0033-9.

Full text

Abstract:

Abstract Genital herpes is a chronic, nearly always active herpes simplex virus (HSV) infection of sacral ganglia, that may appear bilaterally and in more ganglia than previously thought. It represents one of the most prevalent sexually transmitted infections, and the most frequent cause of genital ulcer disease in the general populations of developed countries. It is caused by HSV type-2 (HSV-2) in 60-80% of cases, with HSV-1 infections causing the remainder. Genital herpes caused by HSV-1 is on the rise. Since genital HSV-1 infections have higher risk for transmission from mother to infant during delivery than HSV-2, they account for 30% of all cases of neonatal herpes. Serological studies have found prevalence of HSV-2 in the general population of developed contries to be up to 25%. Thirty years ago, herpes was defined as “Today’s Scarlet Letter”in the absence of reliable serological tests and highly effective medications, for diagnosis and treatment of genital herpes. In 2000, apart from virus isolation in cell culture (70% sensitivity), that has long been regarded as the diagnostic gold standard, type specific serological tests and higly effective antiviral agents have evolved. However, the following questions were raised: should serological testing be routinely recommended in asymptomatic patients; can antiviral therapy reduce asymptomatic shedding of the virus; can antiviral therapy reduce sexual transmission of infection; can antiviral therapy reduce acquisitation of viral copathogens, such as human immunodeficiency virus (HIV)? Now, ten years later, we know the answers. Type specific HSV DNA detection by real-time PCR assays (100% sensitivity) are tests of choice for every person with recurrent genital ulcers lasting more than 4 days, and must be available in those laboratories currently performing a significant number of PCR tests for different purposes. Type specific IgG serology assays are indicated in all asymptomatic persons who are at increased risk for HSV infection. In sexually active patients experiencing ≥ 6 recurrences per year, daily supressive dose of acyclovir, valacyclovir or famciclovir should be discontinued after a maximum of a year of continuous antiviral therapy in order to reassess recurrence frequency. If necessary, the therapy should be restarted after at least two recurrences. With such expansive diagnostic possibilities and more aggressive therapeutic approaches, we can define genital herpes not as a “Scarlet Letter”, but as a “widespread untoward consequence of human sexuality”.

APA, Harvard, Vancouver, ISO, and other styles

26

Flores, Sonia C., and Sharilyn Almodovar. "Human Immunodeficiency Virus, Herpes Virus Infections, and Pulmonary Vascular Disease." Pulmonary Circulation 3, no. 1 (January 2013): 165–70. http://dx.doi.org/10.4103/2045-8932.109955.

Full text

APA, Harvard, Vancouver, ISO, and other styles

27

Cathomas, G. "Human herpes virus 8: a new virus discloses its face." Virchows Archiv 436, no. 3 (March 10, 2000): 195–206. http://dx.doi.org/10.1007/s004280050031.

Full text

APA, Harvard, Vancouver, ISO, and other styles

28

CHESKY, MARISA, ROSANA SCALCO, LUCIANE FAILACE, STEVEN READ, and LUIZ FERNANDO JOBIM. "Polymerase chain reaction for the laboratory diagnosis of aseptic meningitis and encephalitis." Arquivos de Neuro-Psiquiatria 58, no. 3B (September 2000): 836–42. http://dx.doi.org/10.1590/s0004-282x2000000500008.

Full text

Abstract:

A protocol for testing cerebrospinal fluid specimens using a range of PCR assays for the diagnosis of central nervous system infection was developed and used to test prospectively 383 specimens. PCR assays were used for the detection of adenovirus, Borrelia burgdorferi, enteroviruses, Epstein Barr virus, cytomegalovirus, herpes simplex virus, human herpes virus type 6, JC virus, Leptospira interrogans, Listeria monocytogenes, lymphocytic choriomeningitis virus, measles virus, mumps virus, Mycobacterium sp., Mycoplasma pneumoniae, Toxoplasma gondii and varicella zoster virus. Of the 383 specimens tested in this study, 46 (12.0%) were found to be positive. The microorganisms detected were CMV, enterovirus, Epstein Barr virus, herpes simplex virus, human herpes virus type 6, JC virus, L. monocytogenes, Mycobacterium genus, Toxoplasma gondii and varicella zoster virus. The introduction of the PCR protocol described has improved the diagnosis of a range of central nervous system infections in our laboratory. We believe however that further evaluation of these assays in immunocompromised patients is necessary to better determine the predictive value of positive PCR results in these patient groups.

APA, Harvard, Vancouver, ISO, and other styles

29

Annunziato, Paula W. "Herpes Simplex Virus Infections." Pediatrics In Review 17, no. 12 (December 1, 1996): 415–23. http://dx.doi.org/10.1542/pir.17.12.415.

Full text

Abstract:

Case Report A term female developed fever and tachypnea and had a transient episode of poor color and tone on the third day of life. She was delivered by cesarean section 5 hours after rupture of membranes to a 19-year-old, gravida 4, para 0 mother whose serology was negative for syphilis, human immunodeficiency virus (HIV), and hepatitis B surface antigen. The mother had no history of sexually transmitted diseases. Apgar scores were 8 at 1 minute and 9 at 10 minutes. When the infant was admitted to the nursery, scalp abrasions were noticed by the nurse. On the third day of life, a sepsis evaluation was initiated, and the infant received ampicillin and gentamicin intravenously: no bacterial infection was found. On the fifth day of life, vesicular lesions were noticed on her scalp. Bilateral interstitial infiltrates were present On chest radiograph and laboratory studies revealed mild elevations in liver function tests. There were no cerebrospinal fluid (CSF) abnormalities. She was started on intravenous acyclovir, and herpes simplex virus (HSV)-2 subsequently was isolated from both her pharynx and scalp lesions. After receiving intravenous acyclovir for 3 weeks, she was discharged with no evidence of residual sequelae. Four days after acyclovir was discontinued, new scalp vesicles appeared and the infant developed a temperature of 38.8°C (102°F).

APA, Harvard, Vancouver, ISO, and other styles

30

Hayashi, Kyoko, Toshimitsu Hayashi, and Akio Tomoda. "Phenoxazine Derivatives Inactivate Human Cytomegalovirus, Herpes Simplex Virus-1, and Herpes Simplex Virus-2 In Vitro." Journal of Pharmacological Sciences 106, no. 3 (2008): 369–75. http://dx.doi.org/10.1254/jphs.fp0071679.

Full text

APA, Harvard, Vancouver, ISO, and other styles

31

Volpi, A., L. Sarmati, B. Suligoi, M. Montano, G. Rezza, and M. Andreoni. "Correlates of human herpes virus-8 and herpes simplex virus type 2 infections in Northern Cameroon." Journal of Medical Virology 74, no. 3 (2004): 467–72. http://dx.doi.org/10.1002/jmv.20200.

Full text

APA, Harvard, Vancouver, ISO, and other styles

32

Pechnikov, Y. "Herpes febrilis и herpes genitalis." Kazan medical journal 22, no. 2 (December 24, 2020): 248–49. http://dx.doi.org/10.17816/kazmj52948.

Full text

Abstract:

It is still unclear whether the causative agents of both of these diseases are identical or different. Lipschutz (Arch. F. Derm. U. Sypb., 1925, Bd. 149) indicates the difference between them on the basis of: 1) his own observations during experimental infection of human skin with a virus of both forms: 2) different duration of evolution of keratitis in a rabbit after vaccination of both viruses and 3) is different in histological skin lesions. A comparison of a number of clinical observations also indicates a difference in the etiological moment for both herpes species.

APA, Harvard, Vancouver, ISO, and other styles

33

Weissenböck, H., J. A. Hainfellner, J. Berger, I. Kasper, and H. Budka. "Naturally Occurring Herpes Simplex Encephalitis in a Domestic Rabbit (Oryctolagus cuniculus)." Veterinary Pathology 34, no. 1 (January 1997): 44–47. http://dx.doi.org/10.1177/030098589703400107.

Full text

Abstract:

An approximately 1-year-old domestic rabbit showed severe neurologic signs with circling and turning somersaults. Histologically, a nonsuppurative meningoencephalitis with neuronal cell necrosis and numerous intranuclear inclusion bodies in neurons and glial cells was found. Electron microscopic examination revealed herpesvirus particles in affected cells. A human herpes simplex virus was identified by means of immunocytochemistry and in situ hybridization as the causal agent and was further classified as herpes simplex virus 1 by polymerase chain reaction analysis. Because encephalitis is easily induced in rabbits by experimental infection with herpes simplex virus, the source of infection is suspected to be a human with herpes labialis who had close contact with the rabbit.

APA, Harvard, Vancouver, ISO, and other styles

34

SULIGOI, B., E. POZIO, M. ANDREONI, R. T. DANAYA, L. SARMATI, G. REZZA, I. L. OWEN, and S. BOROS. "INFECTION WITH HUMAN IMMUNODEFICIENCY VIRUS, HERPES SIMPLEX VIRUS TYPE 2, AND HUMAN HERPES VIRUS 8 IN REMOTE VILLAGES OF SOUTHWESTERN PAPUA NEW GUINEA." American Journal of Tropical Medicine and Hygiene 72, no. 1 (January 1, 2005): 33–36. http://dx.doi.org/10.4269/ajtmh.2005.72.33.

Full text

APA, Harvard, Vancouver, ISO, and other styles

35

Strumia, R., C. Roveggio, A. Rotola, P. Monini, and E. Cassai. "Keratoacanthomas: human papillomavirus and herpes simplex virus associated?" Journal of the European Academy of Dermatology and Venereology 8, no. 2 (April 1997): 130–36. http://dx.doi.org/10.1111/j.1468-3083.1997.tb00201.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

36

Bastow, K. "New Acridone Inhibitors of Human Herpes Virus Replication." Current Drug Target -Infectious Disorders 4, no. 4 (December 1, 2004): 323–30. http://dx.doi.org/10.2174/1568005043340533.

Full text

APA, Harvard, Vancouver, ISO, and other styles

37

Ariza-Heredia, Ella J., and Raymund R. Razonable. "Human Herpes Virus 8 in Solid Organ Transplantation." Transplantation 92, no. 8 (October 2011): 837–44. http://dx.doi.org/10.1097/tp.0b013e31823104ec.

Full text

APA, Harvard, Vancouver, ISO, and other styles

38

Contreras, A., and J. Slots. "Typing of herpes simplex virus from human periodontium." Oral Microbiology and Immunology 16, no. 1 (February 2001): 63–64. http://dx.doi.org/10.1034/j.1399-302x.2001.160111.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

39

Bernit, Emmanuelle, Véronique Veit, Christine Zandotti, Julie Gachon, Nicolas Schleinitz, and Jean Robert Harlé. "Chronic Lymphadenopathies and Human Herpes Virus Type 8." Scandinavian Journal of Infectious Diseases 34, no. 8 (January 2002): 625–26. http://dx.doi.org/10.1080/00365540210147552.

Full text

APA, Harvard, Vancouver, ISO, and other styles

40

Balakrishna, Jayalakshmi P., Tapan Bhavsar, Alina Nicolae, Mark Raffeld, Elaine S. Jaffe, and Stefania Pittaluga. "Human Herpes Virus 6 (HHV-6)–associated Lymphadenitis." American Journal of Surgical Pathology 42, no. 10 (October 2018): 1402–8. http://dx.doi.org/10.1097/pas.0000000000001121.

Full text

APA, Harvard, Vancouver, ISO, and other styles

41

Strumìa, R. "Keratoacanthomas: human papillomavirus and herpes simplex virus associated?" Journal of the European Academy of Dermatology and Venereology 8, no. 2 (April 1997): 130–36. http://dx.doi.org/10.1016/s0926-9959(96)00575-2.

Full text

APA, Harvard, Vancouver, ISO, and other styles

42

Nussenblatt, Robert B., and Alan G. Palestine. "Human Immunodeficiency Virus, Herpes Zoster, and the Retina." American Journal of Ophthalmology 112, no. 2 (August 1991): 206–7. http://dx.doi.org/10.1016/s0002-9394(14)76703-0.

Full text

APA, Harvard, Vancouver, ISO, and other styles

43

Remeijer, Lies, Albert Osterhaus, and Georges Verjans. "Human herpes simplex virus keratitis: the pathogenesis revisited." Ocular Immunology and Inflammation 12, no. 4 (January 2004): 255–85. http://dx.doi.org/10.1080/092739490500363.

Full text

APA, Harvard, Vancouver, ISO, and other styles

44

Croen, Kenneth D., Jeffrey M. Ostrove, Lubo J. Dragovic, John E. Smialek, and Stephen E. Straus. "Latent Herpes Simplex Virus in Human Trigeminal Ganglia." New England Journal of Medicine 317, no. 23 (December 3, 1987): 1427–32. http://dx.doi.org/10.1056/nejm198712033172302.

Full text

APA, Harvard, Vancouver, ISO, and other styles

45

Kim, S. U., and L. F. Kastrukoff. "HERPES SIMPLEX VIRUS INFECTION IN CULTURED HUMAN OLIGODEWDROCYTES." Journal of Neuropathology and Experimental Neurology 46, no. 3 (May 1987): 347. http://dx.doi.org/10.1097/00005072-198705000-00053.

Full text

APA, Harvard, Vancouver, ISO, and other styles

46

Salahuddin, Syed Zaki, Ann S. Kelley, Gerhard R. F. Krueger, Steven F. Josephs, Sudhir Gupta, and Dharam V. Ablashi. "Human herpes virus-6 (HHV-6) in diseases." Clinical and Diagnostic Virology 1, no. 2 (July 1993): 81–100. http://dx.doi.org/10.1016/0928-0197(93)90016-x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

47

Guo, Hongyan, Shinya Omoto, Philip A. Harris, Joshua N. Finger, John Bertin, Peter J. Gough, William J. Kaiser, and Edward S. Mocarski. "Herpes Simplex Virus Suppresses Necroptosis in Human Cells." Cell Host & Microbe 17, no. 2 (February 2015): 243–51. http://dx.doi.org/10.1016/j.chom.2015.01.003.

Full text

APA, Harvard, Vancouver, ISO, and other styles

48

Leao, Jair Carneiro, Andreza Barkokebas Santos de Faria, Déborah Daniela Diniz Fonseca, Luiz Alcino Monteiro Gueiros, Igor Henrique Morais Silva, and Stephen R. Porter. "Intrahost genetic variability of human herpes virus-8." Journal of Medical Virology 85, no. 4 (February 15, 2013): 636–45. http://dx.doi.org/10.1002/jmv.23491.

Full text

APA, Harvard, Vancouver, ISO, and other styles

49

Arvin, A. M. "Varicella-zoster virus." Clinical Microbiology Reviews 9, no. 3 (July 1996): 361–81. http://dx.doi.org/10.1128/cmr.9.3.361.

Full text

Abstract:

Varicella-zoster virus (VZV) is a ubiquitous human alphaherpesvirus that causes varicella (chicken pox) and herpes zoster (shingles). Varicella is a common childhood illness, characterized by fever, viremia, and scattered vesicular lesions of the skin. As is characteristic of the alphaherpesviruses, VZV establishes latency in cells of the dorsal root ganglia. Herpes zoster, caused by VZV reactivation, is a localized, painful, vesicular rash involving one or adjacent dermatomes. The incidence of herpes zoster increases with age or immunosuppression. The VZV virion consists of a nucleocapsid surrounding a core that contains the linear, double-stranded DNA genome; a protein tegument separates the capsid from the lipid envelope, which incorporates the major viral glycoproteins. VZV is found in a worldwide geographic distribution but is more prevalent in temperate climates. Primary VZV infection elicits immunoglobulin G (IgG), IgM, and IgA antibodies, which bind to many classes of viral proteins. Virus-specific cellular immunity is critical for controlling viral replication in healthy and immunocompromised patients with primary or recurrent VZV infections. Rapid laboratory confirmation of the diagnosis of varicella or herpes zoster, which can be accomplished by detecting viral proteins or DNA, is important to determine the need for antiviral therapy. Acyclovir is licensed for treatment of varicella and herpes zoster, and acyclovir, valacyclovir, and famciclovir are approved for herpes zoster. Passive antibody prophylaxis with varicella-zoster immune globulin is indicated for susceptible high-risk patients exposed to varicella. A live attenuated varicella vaccine (Oka/Merck strain) is now recommended for routine childhood immunization.

APA, Harvard, Vancouver, ISO, and other styles

50

McIntosh, E. David G. "Development of vaccines against gonorrhoea, syphilis, chlamydia, herpes simplex virus, human immunodeficiency virus and Zika virus." Clinical Microbiology and Antimicrobial Chemotherapy 21, no. 4 (2019): 253–60. http://dx.doi.org/10.36488/cmac.2019.4.253-260.

Full text

Abstract:

The success in preventing hepatitis B virus and human papillomavirus infections by means of vaccination paves the way for the development of other vaccines to prevent sexually transmitted infections (STIs) such as gonorrhoea, syphilis, chlamydia, herpes simplex virus, human immunodeficiency virus and Zika virus. The current status of vaccine development for these infections will be explored in this review.

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!