Relevant bibliographies by topics / Human herpes virus

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Human herpes virus'

Author: Grafiati
Published: 4 June 2021
Last updated: 20 February 2023

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Journal articles on the topic "Human herpes virus"

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Baz, Kıymet Baz, Gönül Aslan, Ayşegül Usta Güney, Seda Tezcan, Ayça Cordan Yazıcı, Bahar Taşdelen, Güliz İkizoğlu, and Gürol Emekdaş. "Human Herpes Virus-6 and Human Herpes Virus -7 in Pityriasis Rosea." Turkish Journal of Dermatology / Türk Dermatoloji Dergisi 7, no. 3 (September 5, 2013): 142–44. http://dx.doi.org/10.4274/tdd.1611.

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Kelly, Teresa, and Saurabh Guleria. "Human herpes virus-6 encephalitis." Journal of Pediatric Neuroradiology 01, no. 02 (July 28, 2015): 143–44. http://dx.doi.org/10.3233/pnr-2012-022.

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Bragina, E. E. "Viral infection of sperm. Part 2. Human herpes viruses, human immunodeficiency virus, hepatitis C virus, Zika virus (review)." Andrology and Genital Surgery 21, no. 4 (February 12, 2021): 20–30. http://dx.doi.org/10.17650/2070-9781-2020-21-4-20-30.

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Intragametal viral infection of spermatozoa can cause true vertical transmission of viruses through germ cells. Currently, human immunodeficiency virus, hepatitis C viruses, herpes simplex virus, cytomegalovirus, Zika virus have been detected in spermatozoa. The possibility of vertical transmission of human immunodeficiency virus, cytomegalovirus, herpes simplex virus and Zika virus has been proven.Intragametal infection of spermatozoa with viruses of the herpes group leads to abnormalities in the development of the embryo and can cause spontaneous abortions both during natural conception and when using assisted reproductive technologies.The development of adequate methods for diagnosing an intragametal spermatozoa virus infection will make it possible to find out, at least in some patients, the cause of infertility and pregnancy abnormalities and apply appropriate antiviral therapy in preparation for natural conception or the use of assisted reproductive technologies.
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Tanaka, Toshio, Kazuhiro Kogawa, Hidenori Sasa, Shigeaki Nonoyama, Kenichi Furuya, and Kenji Sato. "Rapid and simultaneous detection of 6 types of human herpes virus (herpes simplex virus, varicella-zoster virus, Epstein-Barr virus, cytomegalovirus, human herpes virus 6A/B, and human herpes virus 7) by multiplex PCR assay." Biomedical Research 30, no. 5 (2009): 279–85. http://dx.doi.org/10.2220/biomedres.30.279.

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Ali, Jwan A. "Detection of Human Herpes Virus-6 in Children Suffering from Febrile Convulsion." International Journal of Psychosocial Rehabilitation 24, no. 5 (April 20, 2020): 3043–49. http://dx.doi.org/10.37200/ijpr/v24i5/pr202010.

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Sunur, Stella, Izazi Hari Purwoko, Yulia Farida Yahya, and Raden Pamudji. "Genital Herpes in Human Immunodeficiency Virus Infected Patients." Bioscientia Medicina : Journal of Biomedicine and Translational Research 5, no. 3 (May 4, 2021): 764–78. http://dx.doi.org/10.32539/bsm.v5i3.342.

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Genital herpes is a recurrent, lifelong sexual transmitted infection caused by HSV, especially type 2. Genital herpes is the most common infection in HIV patient. HSV-2 can increase the risk of HIV acquisition 2 to 3 times. Clinical manifestations of genital herpes can be different between HIV- infected and non-HIV patients. HIV-infected patients have a high risk of developing chronic and severe genital ulcers with atypical manifestation, prolonged healing, and resistant to treatment, depends on CD4 count. Genital herpes can be diagnosed based on history, clinical manifestation, laboratory and histopathologic examination. Management of genital herpes includes education and counseling patients and sexual partners, systemic antiviral, and symptomatic treatment.
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Sunur, Stella, Izazi Hari Purwoko, Yulia Farida Yahya, and Raden Pamudji. "Genital Herpes in Human Immunodeficiency Virus Infected Patients." Bioscientia Medicina : Journal of Biomedicine and Translational Research 5, no. 8 (May 4, 2021): 758–72. http://dx.doi.org/10.32539/bsm.v5i8.342.

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Genital herpes is a recurrent, lifelong sexual transmitted infection caused by HSV, especially type 2. Genital herpes is the most common infection in HIV patient. HSV-2 can increase the risk of HIV acquisition 2 to 3 times. Clinical manifestations of genital herpes can be different between HIV- infected and non-HIV patients. HIV-infected patients have a high risk of developing chronic and severe genital ulcers with atypical manifestation, prolonged healing, and resistant to treatment, depends on CD4 count. Genital herpes can be diagnosed based on history, clinical manifestation, laboratory and histopathologic examination. Management of genital herpes includes education and counseling patients and sexual partners, systemic antiviral, and symptomatic treatment.
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Nikolskiy, M. A. "THE HUMAN HERPES VIRUS TYPE 7." Russian Journal of Infection and Immunity 3, no. 1 (July 7, 2014): 15. http://dx.doi.org/10.15789/2220-7619-2013-1-15-20.

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Okuno, T., H. Sao, and K. Yamanishi. "Human herpes virus 6(HHV-6)." Uirusu 41, no. 2 (1991): 65–76. http://dx.doi.org/10.2222/jsv.41.65.

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Sanghavi, Akta, Deepak Dave, Prasad Nadig, Tulsi Sanghavi, and Nirali Khanpara. "Human herpes virus: Bacteria and periodontium." Journal of Oral Disease Marker 1, no. 1 (2017): 5–9. http://dx.doi.org/10.15713/ins.jodm.3.

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Dissertations / Theses on the topic "Human herpes virus"

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Carbone, Ilaria <1984&gt. "Human herpes virus and Alzheimer’s disease." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2013. http://amsdottorato.unibo.it/5255/.

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Alzheimer’s disease (AD) is a chronic and progressive neurodegenerative disorder and according to the WHO it is estimated that 36 millions of people worldwide currently suffer from AD. Genetic and environmental factors interact in a complex interplay that might affect pathogenic mechanisms leading to age-related neurodegeneration. The hypothesis is that the presence of allelic polymorphisms in selected genes affecting individual brain susceptibility to infection by the herpes virus family during aging, may contribute to neuronal loss, inflammation and amyloid deposition. Herpes virus family show features relevant to AD, since they infect a large proportion of human population, develop a latent form persisting for several years, are difficult to eliminate by immune responses especially when latency has been established and are able to infect neurons. The association between AD and herpes viruses infection has been investigated. In particular the investigation focused on CMV, EBV and HHV-6 in DNA samples from peripheral blood of a large cohort of patients with clinical diagnosis of AD and age matched CTR, from a longitudinal population study, and DNA samples from brain tissue of patients with neuropathological diagnosis of definitive AD. An association between the presence of EBV and HHV-6 DNA from PBL positivity with the cognitive deterioration and progression to AD has been focused. Moreover, IgG plasma levels in CTR and AD to these viruses were tested. CMV and EBV IgG plasma levels were higher in elderly subjects that developed clinical AD at the end of the five year follow up. Our findings support the notion that persistent cycles of latency and reactivation of herpes viruses may contribute to impair systemic immune response and induce altered inflammatory process that in turn affect cognitive decline during aging.
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Halawi, Mustafa. "Immune responses against human herpes virus 6." Thesis, University of Liverpool, 2015. http://livrepository.liverpool.ac.uk/2012959/.

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Human herpes virus 6 (HHV6) infects the majority of individuals in childhood, followed by a lifelong asymptomatic latent infection. However, in immunosuppressed individuals reactivation of HHV6 can cause significant clinical pathology. Recent successes with adoptive T cell therapy against other viral infections, notably the human herpes viruses Epstein-Barr virus (EBV) and Human cytomegalovirus (HCMV), suggest that this may be a useful therapeutic approach for HHV6-driven disease in immunosuppressed individuals. However, very few studies have been carried out analysing the immune response to HHV6 in any detail. This thesis was aimed at characterising the CD8+ T cell response to HHV6 in a group of healthy individuals, with the aim of mapping and characterising novel CD8+ T cell epitopes. Initial studies included four HHV6B antigens (U11, U39, U54 and U90), predicted to be immunogenic based on their HCMV homologues. Whole antigen peptide mixes (pepmixes) were used to stimulate peripheral blood mononuclear cells (PBMC) from healthy subjects. T cell responses were analysed by intracellular cytokine staining (ICS) after overnight stimulation and/or by interferon-γ (IFN-γ) ELISpot assay after 10 days of stimulation. For responses to U11 and U90, peptides libraries were used to map minimum CD8+ restricted epitopes. Further characterisation of HHV6B-specific T cells was carried out by identifying the HLA restriction elements and determining whether these T cells were capable of killing HHV6B-infected cells. PBMC from 30 healthy donors were stimulated with pepmixes corresponding to HHV-6B antigens U11, U39, U54 and U90. A weak CD8+ response (0.02-0.2%) to U90 and U54 was observed in a number of donors. Short-term in-vitro reactivations of PBMC (in 25 healthy donors) with HHV6B pepmixes followed by analysis of antigen and peptide specific response were performed by IFN-γ ELISpot assay. T cell responses to U54, U90, U11 and U39 were observed in 88%, 84%, 76% and 72% of the donors, respectively. Subsequently, the breadth of epitope specificity within U90 and U11 was screened for 9 healthy donors; with successful identification of 10 CD8+ T cells specific (9-mer) epitopes within these antigens. Seven of them were within U90 antigens and three of them were within U11 antigens. Allelic association of the U90 epitopes were; VEESIKEIL - B40 (60), FESLLFPEL - B40 (60), NLITAAKNI - A2, ITAAKNIGI - A2, LNIDPSESI - A1, PSKSKKIKL - A29, NHCFINHFV - B39. Allelic association of the 2 U11 epitopes were LKTQRRHKF - B37 and GILDFGVKL - A2; the HLA association for FNAVYSQRV was not identified. CD8+ T cell populations specific to some of these epitopes were also able to kill HHV6B infected cells. HHV6B T cells responses are detectable in healthy donors. Peptide specific responses against U11 and U90 have been mapped and characterised. These findings are relevant to the development of T cell mediated immunotherapy of HHV6-associated diseases.
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Sayers, Charlotte. "Herpes simplex virus type 1 infection of human keratinocyte cells." Thesis, Imperial College London, 2013. http://hdl.handle.net/10044/1/11112.

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Infection of herpes simplex virus type 1 (HSV-1) begins at the epidermis, a stratified layer composed primarily of keratinocytes. The physiologically relevant cell type for the study of HSV-1 assembly is therefore the human keratinocyte. Nonetheless, relatively little is known about the replication of HSV-1 in this natural host cell. Comparison of virus growth in monolayers of keratinocyte cells and Vero cells, a routinely used cell line for HSV-1 studies, revealed that these keratinocytes support a more productive virus replication than Vero cells. Furthermore, newly assembled virus is produced more rapidly in keratinocytes and this enhancement occurs prior to, or upon the initiation of immediate early gene transcription. This augmented replication in keratinocytes can be at least partially attributed to the method of entry of the virus. We have found by penetration assays and electron microscopy that the virus is able to penetrate keratinocyte cells much more rapidly than Vero cells. We have also shown that the virus entry mechanism is more efficient at lower temperatures in nTERT cells, with virus entering cells at temperatures as low as 7°C. Additionally preliminary work implies that depletion of one of the herpes virus entry receptors, Nectin-1, does not affect entry into nTERT cells, whereas entry is reduced up to 65% in HeLa cells. Taken together, these results imply a role for other entry receptors, possibly as yet unidentified, in the entry of human keratinocyte cells. This work also identifies a role for cellular Rab proteins, GTPases essential for the regulation of vesicle trafficking, in HSV-1 infection of keratinocytes. In particular Rab6, which was also found to play a role in infected HeLa cells (Elliott Group), appears to have a similar function in both these cells and together support a model for HSV-1 morphogenesis involving Rab-regulated vesicle trafficking of viral glycoproteins to the cell surface. Several other Rabs identified by this screen now provide interesting opportunities to elucidate further roles of Rab proteins in HSV-1 infection of keratinocyte cells. This project has broadly characterised the replication of HSV-1 in keratinocyte cells and explored the role of Rab GTPases in virus trafficking within keratinocytes - a cell type that is physiologically relevant to infection.
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Simmonds, Peter. "Detection of antibody responses to infection with herpes simplex virus and human immunodeficiency virus." Thesis, University of Edinburgh, 1988. http://hdl.handle.net/1842/26933.

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Thellman, Nichole Nikki M. "A human neuronal model for herpes simplex virus latency and reactivation." Thesis, Van Andel Research Institute, 2017. http://pqdtopen.proquest.com/#viewpdf?dispub=10284545.

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A defining characteristic of alphaherpesviruses is the establishment of lifelong latency in host sensory ganglia with occasional reactivation causing recurrent lytic infections. Much remains unknown regarding the cellular and viral mechanisms involved in HSV exit from latency. We hypothesize that VP16 recruits chromatin-remodeling enzymes to immediate early gene promoters on compact-latent chromatin as a necessary step for reactivation. In order to test this hypothesis, a robust in vitro assay in which HSV latency can be established in neurons was required. In this dissertation, I explored the use of a human sensory neuron cell line as a novel in vitro model of HSV-1 latency and reactivation. HD10.6 cells were derived from embryonic human dorsal root ganglia and immortalized by a tetracycline-regulated v-myc oncogene. HD10.6 cells mature to express a sensory neuron-associated phenotype when treated with doxycycline which suppresses proliferation mediated by the v-myc oncogene. Infection at a low MOI in the presence of acyclovir results in a quiescent infection resembling latency in matured cells. HD10.6 cells provide a novel context in which to study the host and viral mechanisms of HSV-1 latency establishment, maintenance, and reactivation.

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Brown, Elizabeth L. "Consequences of genital herpes simplex virus infection among vulnerable populations /." Thesis, Connect to this title online; UW restricted, 2006. http://hdl.handle.net/1773/10885.

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Held, Kathrin. "Control of herpes simplex virus type 1 latency in human trigeminal ganglia." Diss., lmu, 2012. http://nbn-resolving.de/urn:nbn:de:bvb:19-142865.

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Baker, Kevin. "Investigations into the ocular involvement of human herpes and papilloma virus infections." Thesis, University of Liverpool, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.266252.

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Peat, D. S. "The effect of herpes simplex virus type 1 on chromosomes of human cells." Thesis, University of Cambridge, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.383841.

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O'Leary, John James. "Molecular analysis of Kaposi's sarcoma associated herpes virus (KSHV) in immunocompromised patients." Thesis, University of Oxford, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.360468.

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Books on the topic "Human herpes virus"

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Media, Springer Science+Business, ed. Herpes simplex virus: Methods and protocols. New York: Humana Press, 2014.

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Marie, Studahl, Cinque Paola, and Bergström T, eds. Herpes simplex viruses. Boca Raton: Taylor & Francis, 2006.

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Johnson-Delaney, Cathy A. Herpes B virus in non-human primates: A bibliography, 1969-1989. Seattle, Wash: Primate Information Center, Regional Primate Research Center, University of Washington, 1989.

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Tabery, Helena M. Herpes simplex virus epithelial keratitis: In vivo morphology in the human cornea. Heidelberg: Springer, 2010.

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Skinner, Gordon R. B. The immunological relatedness, immunogenicity and oncogenicity of herpes simplex virus: Development of a vaccinetowards prevention of herpes genitalis in human subjects. Birmingham: University of Birmingham, 1989.

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Tabery, Helena M. Varicella-Zoster Virus Epithelial Keratitis in Herpes Zoster Ophthalmicus: In Vivo Morphology in the Human Cornea. Berlin, Heidelberg: Springer-Verlag Berlin Heidelberg, 2011.

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West, A. Investigations by mass spectrometry of the interactions of novel serine protease inhibitors with Herpes Simplex Virus type 2 and Human Cytomegalovirus proteases. [s.l.]: typescript, 1999.

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Current issues in clinical neurovirology: Pathogenesis, diagnosis and treatment. Philadelphia, Pa: Saunders, 2008.

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Sexually transmitted diseases sourcebook: Basic consumer health information about sexual health and the screening, diagnosis, treatment, and prevention of common sexually transmitted diseases (STDs), including chancroid, chlamydia, gonorrhea, herpes, hepatitis, human immunodeficiency virus/acquired immunodeficiency syndrome (HIV/AIDS), human papillomavirus (HPV), syphilis, and trichomoniasis ; along with facts about risk factors and complications, trends and disparities in infection rates, tips for discussing STDs with sexual partners, a glossary of related terms, and resources for additional help and information. 5th ed. Detroit, MI: Omnigraphics, Inc., 2013.

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Brown, David W. G. Herpes B virus (Cercopithecine Herpes 1). Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0036.

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Herpes B virus or Cercopithecine herpes 1 as it is formally classified causes a persistent infection of monkeys of the Macaca genus. In monkey colonies and social groups, it is transmitted by close contact and sexually. Human infection is rare with less than 50 human cases described it has been seen in monkey handlers exposed to infected monkeys following bites, scratches and abraded skin. Infection has also been recognized in two cases following exposure through laboratory work. Following an incubation period of 9-59 days typically an ascending encephalomyelitis develops which is fatal in 80% of cases. Prevention and control of the risk of B virus is based on avoiding direct contact with infected animals by screening, following handling guidelines for monkeys used in biomedical research and rigorous laboratory safety precautions. Treatment with acyclovir has been successful and halved mortality in recent cases. It is also recommended for prophylaxis in potential exposures.
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Book chapters on the topic "Human herpes virus"

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Applewhite, Brooks P., and Jonathan Lester. "Human Herpes Virus." In Encyclopedia of Ophthalmology, 1–5. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-35951-4_799-1.

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Applewhite, Brooks P., and Jonathan Lester. "Human Herpes Virus." In Encyclopedia of Ophthalmology, 886–90. Berlin, Heidelberg: Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-540-69000-9_799.

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Steiner, Israel. "Herpes Simplex Virus Meningoencephalitis." In Viral Infections of the Human Nervous System, 47–63. Basel: Springer Basel, 2012. http://dx.doi.org/10.1007/978-3-0348-0425-7_3.

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Coia, John, and Heather Cubie. "Human Herpes virus 6 (HHV6)." In The Immunoassay Kit Directory, 795–97. Dordrecht: Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-009-0359-3_20.

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Dabrowski, C. E., X. Qiu, and S. S. Abdel-Meguid. "The Human Herpes-Virus Proteases." In Handbook of Experimental Pharmacology, 95–115. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-57092-6_6.

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Glorioso, Joseph, Mary Ann Bender, David Fink, and Neal Deluca. "Herpes simplex virus vectors." In Molecular and Cell Biology of Human Gene Therapeutics, 33–63. Dordrecht: Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-011-0547-7_3.

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Balachandran, N., D. E. Oba, and L. M. Hutt-Fletcher. "Cross Reactivity between EBV and Herpes Simplex Virus." In Epstein-Barr Virus and Human Disease, 263–64. Totowa, NJ: Humana Press, 1987. http://dx.doi.org/10.1007/978-1-4612-4590-2_59.

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Stevens, Jack G. "Transcripts Associated with Herpes Simplex Virus Latency." In Immunobiology and Prophylaxis of Human Herpesvirus Infections, 199–204. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4684-5853-4_20.

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Kurata, T., T. Sata, T. Iwasaki, T. Onodera, and A. Toniolo. "Herpes Virus Infections in Human Pancreas Islets and Animal Models." In Virus Infections and Diabetes Mellitus, 143–60. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-2085-2_8.

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Borysiewicz, L. K., and J. G. P. Sissons. "Cytotoxic T Cells and Human Herpes Virus Infections." In Current Topics in Microbiology and Immunology, 123–50. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-78530-6_8.

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Conference papers on the topic "Human herpes virus"

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Le Hingrat, Quentin, Mada Ghanem, Aurélie Cazes, Benoit Visseaux, Gilles Collin, Diane Descamps, Charlotte Charpentier, and Bruno Crestani. "No association between Human herpes virus, Herpes virus Saimiri and idiopathic pulmonary fibrosis." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa1319.

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Gandhiraj, D., A. C. Miller, C. Zhang, J. K. Mwangi, D. Picker, E. Charbek, and D. Stoeckel. "Human Herpes Virus-8 Negative Primary Effusion Lymphoma." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6971.

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AL, Cunningham, M. Kim, Truong NR, Sandgren KJ, Harman AN, Bertram KM, L. Bosnjak, et al. "O06.2 Initial interactions of herpes simplex virus with human skin dendritic cells." In STI and HIV World Congress Abstracts, July 9–12 2017, Rio de Janeiro, Brazil. BMJ Publishing Group Ltd, 2017. http://dx.doi.org/10.1136/sextrans-2017-053264.31.

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Liu, R., J. Wang, R. Liu, and S. Rabkin. "Abstract P2-18-01: Treatment of Human Breast Tumors by Oncolytic Herpes Simplex Virus." In Abstracts: Thirty-Third Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 8‐12, 2010; San Antonio, TX. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/0008-5472.sabcs10-p2-18-01.

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"A COMPUTATIONAL STRATEGY TO INVESTIGATE RELEVANT SIMILARITIES BETWEEN VIRUS AND HUMAN PROTEINS - Local High Similarities between Herpes and Human Proteins." In International Conference on Bioinformatics Models, Methods and Algorithms. SciTePress - Science and and Technology Publications, 2011. http://dx.doi.org/10.5220/0003156801830188.

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Henning, Jill D., Kathrine Kercher, Clareann H. Bunker, Robert E. Ferrel, Alan L. Patrick, and Frank J. Jenkins. "Abstract B006: Analysis of the interaction of human herpes virus 8 and gp130 in prostate cancer risk." In Abstracts: AACR Special Conference: Prostate Cancer: Advances in Basic, Translational, and Clinical Research; December 2-5, 2017; Orlando, Florida. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.prca2017-b006.

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Tan, Gewen, Hideki Kasuya, Tevfik Tolga Sahin, Toshio Shikano, Suguru Yamada, Akiyuki Kanzaki, Kazuo Yamamura, et al. "Abstract 2981: Combination therapy of oncolytic herpes virus HF10 and bevacizumab against experimental model of human breast carcinoma xenograft." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-2981.

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Cheng, L., J. Wang, and R. Liu. "Abstract P1-04-01: A novel oncolytic herpes simplex virus, GD116, has enhanced antitumour efficacy in human breast cancer." In Abstracts: 2017 San Antonio Breast Cancer Symposium; December 5-9, 2017; San Antonio, Texas. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.sabcs17-p1-04-01.

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Ghatol, Abhijeet, and Nikhil Barot. "A Case Of Human Herpes Virus 8 (HHV 8)-Associated Extracavitary Lymphoma In A Patient With Disseminated Kaposi Sarcoma And HIV." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a2970.

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Tan, Gewen, Hideki Kasuya, Tevfik T. Sahin, Toshio Shikano, Suguru Yamada, Akiyuki Kanzaki, Kazuo Yamamura, et al. "Abstract 476: The effect of combination of oncolytic herpes virus HF10 with bevacizumab in the treatment of human breast carcinoma xenograft." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-476.

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Reports on the topic "Human herpes virus"

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Ye, Shanli. DNA Sequences Involved in the Regulation of Human c-myc Gene Expression by Herpes Simplex Virus Type 1 (HSV-1). Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.7097.

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