Relevant bibliographies by topics / Host Immune Respose

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  1. Journal articles

Academic literature on the topic 'Host Immune Respose'

Author: Grafiati
Published: 6 September 2023

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Journal articles on the topic "Host Immune Respose"

1

Hamadou, Takieddine, Imene Hamadou, Ahmed Menad, Somia Bouameur, and Souad Ameddah. "COVID-19 : histoire, pathogenèse et réponse immunitaire de l'hôte." Batna Journal of Medical Sciences (BJMS) 8, no. 1 (2021): 52–58. http://dx.doi.org/10.48087/bjmsra.2021.8110.

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By the end of 2019, pneumonia of unknown etiology occurred in Wuhan, China. Local hospitals started receiving patients presenting symptoms like dry cough, fatigue, and breathing difficulties, most of these patients were linked to the Huanan seafood market, Wuhan, China. The pandemic was afterward confirmed to be associated with a novel coronavirus. The virus spread quickly from Wuhan to other provinces of China, then from china to the rest of the world causing thereby one of the most brutal pandemics in the world’s history. SARS-CoV2 has a long incubation period ranging from 3 to 7 days and can go up to 14 days in some cases which makes the infection difficult to be detected early and subsequently the disease spread harder to be controlled. SARS-CoV-2 is a single-stranded RNA virus with 4 main structural proteins, the spike (S) glycoprotein, the small envelope (E) the glycoprotein, the membrane (M) glycoprotein as well as the nucleocapsid (N) protein. Current knowledge about the virus shows that it uses its spike protein to invade host cells, mainly the alveolar epithelial cells. The the lung is the most targeted organ among many other organs like the heart, small intestine, and kidneys that are vulnerable to SARS-CoV-2 infection. The COVID-19 is known to be mild in most cases, but in some cases, it can be severe or even fatal. In the severe cases, acute respiratory distress syndrome was reported, and the the capability of SARS-CoV-2 to infect many organs can lead to multiorgan failure and death. SARS-CoV-2 invasion induces several immune responses that could be efficient for infection clearance in mild cases, while in severe cases, the immune response dysfunctions can even contribute to the disease aggravation. Neither the the pathogenic mechanism by which SARS-CoV-2 infects host cells, nor the host immune response to its infection have been fully understood, hence further studies are needed to give further evidence about these two phenomena. Keywords: COVID-19, SARS-CoV-2, Coronavirus, Structural proteins, Immune response.
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2

Haig, David M., Jackie Thomson, Colin McInnes, et al. "Orf virus immuno-modulation and the host immune response." Veterinary Immunology and Immunopathology 87, no. 3-4 (2002): 395–99. http://dx.doi.org/10.1016/s0165-2427(02)00087-9.

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3

Roilides, Emmanuel, Emmanuel Roilides, Maria Simitsopoulou, Aspasia Katragkou, and Thomas J. Walsh. "Host immune response againstScedosporiumspecies." Medical Mycology 47, no. 4 (2009): 433–40. http://dx.doi.org/10.1080/13693780902738006.

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4

Trasia, Reqgi First. "Host Immune Response to Malaria." International Islamic Medical Journal 2, no. 2 (2021): 67–71. http://dx.doi.org/10.33086/iimj.v2i2.1681.

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Malaria is still a health threat, especially for children and pregnant women in endemic areas. The World Health Organization (WHO) reports 228 million cases of malaria occur worldwide and an estimated 405,000 deaths from malaria globally in 2018. A series of malaria control efforts according to WHO recommendations have been carried out widely. However, these programs face obstacles. Therefore, the existence of an effective malaria vaccine is absolutely necessary in a series of malaria control strategies. Development of a malaria vaccine requires a basic concept regarding the host's immune response to malaria. Unfortunately, only a few in Indonesia have reviewed how the immune response is. This article will present an understanding of how the human immune system responds to Plasmodium falciparum.
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5

Cheng, E. "Imaging the Host Immune Response." Science Translational Medicine 2, no. 40 (2010): 40ec113. http://dx.doi.org/10.1126/scitranslmed.3001469.

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6

Li, Chaozheng, Shaoping Weng, and Jianguo He. "WSSV–host interaction: Host response and immune evasion." Fish & Shellfish Immunology 84 (January 2019): 558–71. http://dx.doi.org/10.1016/j.fsi.2018.10.043.

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7

Bhopale, Mahendra. "Experimental Hookworm Infection in Laboratory animals: Parasite behavior, Immune response and Chemotherapeutic Studies." Biotechnology and Bioprocessing 2, no. 5 (2021): 01–03. http://dx.doi.org/10.31579/2766-2314/040.

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Hookworm disease is known to be caused allergic manifestation and severe anemic pathogenicity in man and canine hosts. Attempts have been made to establish laboratory models of Necator americaus, Ancylostoma duodenale, and Ancylostoma ceylanicum, together with canine parasite, Ancylostoma caninum. The studies include pathophysiological aspects of the host-parasite relationship, and develop to establish patent infection. Immunological approach to selecting antigen for diagnosis and protective immunity purpose using larval and adult worm antigens and their secretions became the focus with the subsequent discovery of cloning in vaccine development as main research interest. Chemotherapy of newer drug screening in laboratory models ultimately selected to use for preventive chemotherapy in hookworm endemic areas using recommended drugs.
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8

Jamieson, Amanda Mercedes, Meredith Crane, Yun Xu, and Kayla Lee. "Immune triage: prioritization of host immune responses." Journal of Immunology 196, no. 1_Supplement (2016): 197.20. http://dx.doi.org/10.4049/jimmunol.196.supp.197.20.

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Abstract The immune response is important in many functions, including host defense against pathogens, wound healing, development, response to cancer, and maintenance of homeostatic physiological responses. We are interested in the concept of immune triage, in that a given host must be able to deal effectively with multiple insults, and at times prioritize immune responses. It is important for the overall health status of the host that the immune system responds effectively to protect essential organs. We have developed several mouse models, focusing on the lung immune response, that allow us to examine different aspects of immune triage. The lung is an essential and delicate organ and thus pulmonary immune responses must be tightly regulated. We have determined that lung infection with influenza A virus (IAV) alters the response to bacterial lung infections. Depending on the bacterial infection, previous infection with IAV can suppress or augment the immune response to bacteria. We have also determined that pulmonary infection with IAV alters many aspects of the systemic immune response. There is a global suppression to systemic bacterial infection, and a decrease in the wound healing response. Our data indicate that the immune system prioritizes lung infections over many other responses. This is most likely due to the importance of the lung in host survival. We have established several regulatory mechanisms by which this immune triage occurs. By understanding how the immune system responds to multiple insults we can improve our understanding of the immune network on a global level.
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9

Elaskandrany, Miar, Rohin Patel, Mintoo Patel, George Miller, Deepak Saxena, and Anjana Saxena. "Fungi, host immune response, and tumorigenesis." American Journal of Physiology-Gastrointestinal and Liver Physiology 321, no. 2 (2021): G213—G222. http://dx.doi.org/10.1152/ajpgi.00025.2021.

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Advances in -omics analyses have tremendously enhanced our understanding of the role of the microbiome in human health and disease. Most research is focused on the bacteriome, but scientists have now realized the significance of the virome and microbial dysbiosis as well, particularly in noninfectious diseases such as cancer. In this review, we summarize the role of mycobiome in tumorigenesis, with a dismal prognosis, and attention to pancreatic ductal adenocarcinoma (PDAC). We also discuss bacterial and mycobial interactions to the host’s immune response that is prevalently responsible for resistance to cancer therapy, including immunotherapy. We reported that the Malassezia species associated with scalp and skin infections, colonize in human PDAC tumors and accelerate tumorigenesis via activating the C3 complement-mannose-binding lectin (MBL) pathway. PDAC tumors thrive in an immunosuppressive microenvironment with desmoplastic stroma and a dysbiotic microbiome. Host-microbiome interactions in the tumor milieu pose a significant threat in driving the indolent immune behavior of the tumor. Microbial intervention in multimodal cancer therapy is a promising novel approach to modify an immunotolerant (“cold”) tumor microenvironment to an immunocompetent (“hot”) milieu that is effective in eliminating tumorigenesis.
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10

Visser, Douwe H., Regan S. Solomons, Katharina Ronacher, et al. "Host Immune Response to Tuberculous Meningitis." Clinical Infectious Diseases 60, no. 2 (2014): 177–87. http://dx.doi.org/10.1093/cid/ciu781.

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