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Books on the topic 'Host disease resistance'

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Author: Grafiati
Published: 10 December 2022
Last updated: 29 January 2023

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1

S, Sadasivam. Molecular host plant resistance to pests. New York: Marcel Dekker, 2003.

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2

Host management in crop pathosystems. New York: Macmillan, 1987.

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3

Vidhyasekaran, P. Fungal pathogenesis in plants and crops: Molecular biology and host defense mechanisms. New York: M. Dekker, 1997.

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4

Flanders, Kathy L. Host plant resistance in Solanum germplasm: An appraisal of resistance to Colorado potato beetle, Potato leafhopper and Potato flea beetle. St.Paul,Minn: Minnesota Agricultural Experiment Station, University of Minnesota, 1992.

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5

Flanders, Kathy Linn. Host plant resistance in Solanum germplasm: An appraisal of resistance to Colorado potato beetle, potato leafhopper and potato flea beetle. St. Paul, Minn: Minnesota Agricultural Experiment Station, University of Minnesota, 1992.

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6

Deverall, Brian J. Defence mechanisms of plants. Cambridge: Cambridge University Press, 2009.

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7

1928-, Day Peter R., Jellis G. J, and British Society for Plant Pathology., eds. Genetics and plant pathogenesis. Oxford [Oxfordshire]: Blackwell Scientific Publications, 1987.

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8

Fungal pathogenesis in plants and crops: Molecular biology and host defense mechanisms. 2nd ed. Boca Raton: CRC Press, 2008.

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9

Or.) International Workshop on the Genetics of Host-Parasite Interactions in Forestry (4th 2011 Eugene. Proceedings of the Fourth International Workshop on the Genetics of Host-Parasite Interactions in Forestry: Disease and insect resistance in forest trees : July 31 to August 5, 2011, Eugene, Oregon, USA. Albany, CA: U.S. Dept. of Agriculture, Forest Service, Pacific Southwest Research Station, 2012.

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10

NATO Advanced Research Workshop on Biology and Molecular Biology of Plant-Pathogen Interactions (1985 Ilminster). Biology and molecular biology of plant-pathogen interactions: Proceedings of the NATO Advanced Research Workshop on Biology and Molecular Biology of Plant-Pathogen Interactions held at Dillington College, Ilminster, U.K., September 1-6, 1985. New York: Springer, 1986.

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11

A, Heinrichs E., ed. Plant stress-insect interactions. New York: Wiley, 1988.

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12

A, Bailey John, ed. Biology and molecular biology of plant-pathogen interactions. Berlin: Springer-Verlag, 1986.

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13

Pande, S. High throughout phenotyping of pigeonpea diseases: Stepwise identification of host plant resistance. Patancheru: International Crops Research Institute for the Semi-Arid Tropics, 2012.

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14

Pande, S. High throughput phenotyping of chickpea diseases: Stepwise identification of host plant resistance. Patancheru, Andhra Pradesh, India: International Crops Research Institute for the Semi-Arid Tropics, 2012.

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15

Derek, Wakelin, and Blackwell J. M, eds. Genetics of resistance to bacterial and parasitic infection. London: Taylor & Francis, 1988.

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16

Plant pathogenesis and resistance: Biochemistry and physiology of plant-microbe interactions. Dordrecht: Kluwer Academic, 2001.

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17

1951-, Wäckers F. L., Rijn, P. C. J. van 1958-, and Bruin J, eds. Plant-provided food for carnivorous insects: A protective mutualism and its applications. Cambridge, UK: Cambridge University Press, 2005.

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18

Sadasivam, S., and B. Thayumanayan. Molecular Host Plant Resistance to Pests. Taylor & Francis Group, 2003.

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19

Sadasivam, S., and B. Thayumanayan. Molecular Host Plant Resistance to Pests. Taylor & Francis Group, 2003.

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20

Sadasivam, S., and B. Thayumanayan. Molecular Host Plant Resistance to Pests. Taylor & Francis Group, 2003.

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21

Sadasivam, S., and B. Thayumanayan. Molecular Host Plant Resistance to Pests. Taylor & Francis Group, 2003.

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22

Sadasivam, S., and B. Thayumanavan. Molecular Host Plant Resistance to Pests. Taylor & Francis Group, 2003.

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23

Sadasivam, S., and B. Thayumanayan. Molecular Host Plant Resistance to Pests. Taylor & Francis Group, 2003.

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24

V, Groth James, Bushnell William R, and American Phytopathological Society Meeting, eds. Genetic basis of biochemical mechanisms of plant disease. St. Paul, Minn: APS press, 1985.

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25

Doeschl-Wilson, Andrea B., and Ilias Kyriazakis, eds. Should we aim for genetic improvement in host resistance or tolerance to infectious disease? Frontiers Media SA, 2013. http://dx.doi.org/10.3389/978-2-88919-105-5.

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26

Vidhyasekaran, P. Fungal Pathogenesis in Plants and Crops: Molecular Biology and Host Defense Mechanisms. Taylor & Francis Group, 2008.

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27

Vidhyasekaran, P. Fungal Pathogenesis in Plants and Crops: Molecular Biology and Host Defense Mechanisms, Second Edition. Taylor & Francis Group, 2007.

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28

Smith, C. Michael. Plant Resistance to Arthropods: Molecular and Conventional Approaches. Springer, 2006.

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29

Deverall, Brian J. Defence Mechanisms of Plants. Cambridge University Press, 2010.

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30

Deverall, Brian J. Defence Mechanisms of Plants. Cambridge University Press, 2011.

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31

N, Andreev L., Gorlenko Mikhail Vladimirovich, and Glavnyĭ botanicheskiĭ sad (Akademii͡a︡ nauk SSSR), eds. Obligatnyĭ parazitizm: T͡s︡itofiziologicheskie aspekty : sbornik nauchnykh stateĭ. Moskva: "Nauka", 1991.

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32

J, Boethel David, and Eikenbary Raymond D, eds. Interactions of plant resistance and parasitoids and predators of insects. Chichester: Ellis Horwood, 1986.

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33

Legrand, M., and B. Fritig. Mechanisms of Plant Defense Responses. Springer, 2012.

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34

Legrand, M., and B. Fritig. Mechanisms of Plant Defense Responses. Springer, 2012.

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35

Vidhyasekaran, P. Fungal Pathogenesis in Plants and Crops: Molecular Biology and Host Defense Mechanisms, Second Edition. Taylor & Francis Group, 2007.

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36

Vidhyasekaran, P. Fungal Pathogenesis in Plants and Crops: Molecular Biology and Host Defense Mechanisms, Second Edition (Books in Soils, Plants, and the Environment). 2nd ed. CRC, 2007.

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37

A, Blanchette R., and Biggs A. R, eds. Defense mechanisms of woody plants against fungi. Berlin: Springer-Verlag, 1992.

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38

Schmid-Hempel, Paul. Evolutionary Parasitology. 2nd ed. Oxford University Press, 2021. http://dx.doi.org/10.1093/oso/9780198832140.001.0001.

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Parasites are ubiquitous and shape almost every aspect of their hosts, including physiology, behaviour, life histories, the structure of the microbiota, and entire communities. Hence, parasitism is one of the most potent forces in nature and, without parasites, the world would look very different. The book gives an overview over the parasite groups and the diversity of defences that hosts have evolved, such as immune systems. Principles of evolutionary biology and ecology analyse major elements of host–parasite interactions, including virulence, infection processes, tolerance, resistance, specificity, memory, polymorphisms, within-host dynamics, diseases spaces, and many other aspects. Genetics is always one of the key elements in these topics. Modelling, furthermore, can predict best strategies for host and parasites. Similarly, the spread of an infectious disease in epidemiology combines with molecular data and genomics. Furthermore, parasites have evolved ways to overcome defences and to manipulate their hosts. Hosts and parasites, therefore, continuously co-evolve, with changes sometimes occurring very rapidly, and sometimes requiring geological times. Many infectious diseases of humans have emerged from a zoonotic origin, in processes governed by the basic principles discussed in the different sections. Hence, this book integrates different fields to study the diversity of host–parasite processes and phenomena. It summarizes the essential topics for the study of evolutionary parasitology and will be useful for a broad audience.
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39

Traugott, Matthew Steven. Effects of tomato allelochemicals on growth, consumption and behavior of an insect predator. 1994.

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40

Downy Mildews Biology Mechanisms Of Resistance And Population Ecology. Springer, 2011.

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41

Michaels, Frank H. The contribution of various host factors to resistance to bovine endotoxemia. 1986.

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42

Institute of Medicine (U.S.). Forum on Microbial Threats., ed. Ending the war metaphor: The changing agenda for unraveling the host-microbe relationship : workshop summary. Washington, DC: National Academies Press, 2006.

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43

Ending the War Metaphor: The Changing Agenda for Unraveling the Host-Microbe Relationship - Workshop Summary. National Academies Press, 2006.

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44

Dawson, Susan. Other bacterial diseasesStaphylococcal zoonoses. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0026.

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Staphylococcal species are common commensals of the skin and mucous membranes of humans and animals but only in very recent years has zoonotic infections been recognised. They can also be associated with infection and disease, especially coagulase positive organisms. Staphylococcus aureus is relatively frequently carried by humans in the nasal passages and is a cause of infections in people including bacteraemias in hospitalised patients. More recently some strains of Staphylococcus aureus have acquired a resistance gene (mecA) which renders them resistant to meticillin (meticillin-resistant Staphylococcus aureus, MRSA). MRSA isolates are of major importance in healthcare situations as well as increasingly in the community. Animals can also be carriers of Staphylococcus aureus although less frequently than humans and MRSA can be carried or infect several different host species. For companion animals such as dogs and cats, the most frequently isolated MRSA strains are similar to the common local human healthcare strains; thus for the UK, EMRSA-15 and -16. This suggests a reverse zoonosis with spill over from the human population into their companion animals. In horses the situation is different, with some horses carrying or infected with human epidemic strains but others infected with strains less frequently seen in people. For food-producing animals the picture is different again with a particular strain, ST398, which appears to circulate endemically in animal populations, such as pigs, and can spill over into the human population where it can cause carriage as well as infection and disease. The transmission appears to be by direct contact with animals rather than through the food-chain.Where risk factors for infection with MRSA have been studied in animals they appear similar to some of the risks for human infection. Therefore, for control of MRSA in animals measures such as improved hygiene and good antibacterial stewardship are important.
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45

Grabe, Magnus, and Björn Wullt. Urinary tract infection. Edited by Rob Pickard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0004.

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Infections of the urinary tract are among the most frequent infections encountered in the community and hospital environments. They range from harmless self-curing cystitis to severe pyelonephritis with life-threatening sepsis. Urinary tract infections are often recurrent. Host defence is crucial to control the infection but can also be deleterious in terms of scar formation. Early diagnosis, determination of severity, evaluation of possible risk factors, and assumption of possible pathogen are essential aspects to initiate efficient treatment. Urine culture with antibiotic sensitivity testing is the most important tool to confirm a suspected clinical diagnosis and direct treatment. Patients with urological disease are particularly susceptible to urinary tract infections, and healthcare-associated urinary infections are observed in approximately 10% of hospitalized urological patients. In view of the worsening resistance pattern of common urinary pathogens against available antimicrobial agents, it is important to comply with recommended treatment regimens.
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46

Dubey, J. P. Toxoplasmosis, sarcocystosis, isosporosis, and cyclosporosis. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0054.

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Toxoplasmosis is a protozoan disease caused by Toxoplasma gondii. It is widely prevalent in humans and animals throughout the world, especially in the western hemisphere. Virtually all warm-blooded animals can act as intermediate hosts but the life cycle is completed only in cats, the definitive host. Cats excrete the resistant stage of T. gondii (oocysts) in faeces, and oocysts can survive in the environment for months. Humans become infected congenitally, by ingesting undercooked infected meat, or by ingesting food and water contaminated with oocysts from cat faeces. It can cause mental retardation and loss of vision in congenitally infected children and deaths in immunosuppressed patients, especially those with AIDS. There is no vaccine to control toxoplasmosis in humans at the present time but one is available for reduction of fetal losses in sheep.
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47

Huang, Jeng-Sheng. Plant Pathogenesis and Resistance: Biochemistry And Physiology Of Plant-Microbe Interactions. Jeng Sheng Huang, 2010.

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48

Huang, Jeng-Sheng. Plant Pathogenesis and Resistance: Biochemistry and Physiology of Plant-Microbe Interactions. Springer, 2001.

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49

Jacobs, Samantha E., Catherine B. Small, and Thomas J. Walsh. Fungal diseases of the respiratory tract. Edited by Christopher C. Kibbler, Richard Barton, Neil A. R. Gow, Susan Howell, Donna M. MacCallum, and Rohini J. Manuel. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755388.003.0030.

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Fungal respiratory infections are important causes of morbidity and mortality in immunocompromised patients. Invasive aspergillosis remains the most common invasive fungal infection whereas other filamentous fungi, such as Fusarium spp., Mucorales, and Scedosporium spp., are increasing in frequency, particularly in neutropenic hosts. Endemic mycoses, including those due to Histoplasma capsulatum, Coccidioides spp., and Talaromyces marneffei, are increasingly prevalent in patients with cell-mediated immunodeficiencies in respective geographic regions. Culture remains the gold standard of diagnosis but has limited sensitivity and often requires invasive procedures. Non-invasive diagnostic tests, including the serum sandwich enzyme immunoassay for the detection of galactomannan, the (1→3)-β‎-D-glucan assay, and molecular amplification methods have been developed to facilitate early and accurate diagnosis. Successful therapy depends upon early initiation of antifungal agents and reversal of immunosuppression. Lipid formulations of amphotericin B and newer generation triazoles including voriconazole, posaconazole, and isavuconazole have expanded the ability to treat multi-drug resistant pathogens more effectively and with less toxicity.
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50

Jex, Aaron R., Rachel M. Chalmers, Huw V. Smith, Giovanni Widmer, Vincent McDonald, and Robin B. Gasser. Cryptosporidiosis. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0053.

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Cryptosporidium species represent a genus of parasitic protozoa (Apicomplexa) that are transmitted via the faecal-oral route and commonly infect the epithelial tissues of the gastric or intestinal (or sometimes the respiratory) tract of many vertebrates, including humans. Infection occurs following the ingestion of viable and resistant oocysts, through direct host-to-host contact or in contaminated food, drinking or recreational water. Infection can be transmitted via anthroponotic (human-to-human, human-to-animal) or zoonotic (animal-to-human or animal-to-animal) pathways, depending upon the species of Cryptosporidium. Although infection can be asymptomatic, common symptoms of disease (cryptosporidiosis) include diarrhoea, colic (abdominal pain), nausea or vomiting, dehydration and/or fever. In humans, cryptosporidial infection in immunocompetent patients is usually short-lived (days to weeks) and eliminated following the stimulation of an effective immune response. However, infection in immunodeficient individuals (e.g., those with HIV/AIDS) can be chronic and fatal (in the absence of immunotherapy), as there are few effective anti-cryptosporidial drugs and no vaccines available. The present chapter provides an account of the history, taxonomy and biology, genomics and genetics of Cryptosporidium, the epidemiology, pathogenesis, treatment and control of cryptosporidiosis and the advances in tools for the identification and characterisation of Cryptosporidium species and the diagnosis of cryptosporidiosis.
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