Relevant bibliographies by topics / Hormonal

Academic literature on the topic 'Hormonal'

Author: Grafiati
Published: 4 June 2021
Last updated: 19 February 2023

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Journal articles on the topic "Hormonal"

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Abreu Lomba, Alin, Karen Dayana González, Maria Antonia Escobar, Antonio Jose Paredes, and Andres Emilson Delgado Truque. "Hipofunción hipofisaria y trastornos relacionados." Revista Colombiana Salud Libre 15, no. 1 (July 7, 2021): e407279. http://dx.doi.org/10.18041/1900-7841/rcslibre.2020v15n1.7279.

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La hipofunción hipofisiaria o también conocida como hipopituitarismo se refiere a la disminución en la secreción de una o mas hormonas hipofisiarias, esto dependerá de la porción glandular comprometida. La glándula se divide en la hipófisis anterior o adenohipófisis en donde se producen 6 hormonas bien conocidas como Hormona de Crecimiento (GH), la Corticotropina (ACTH), la Tirotropina (TSH), la Hormona Folículo Estimulante (FSH), la Hormona Luteinizante (LH) y la Prolactina (PRL). En la hipófisis posterior o neurohipofisis se almacena continuamente y después se secretan la Hormona Antidiurética (ADH) y la oxitocina. Por lo tanto, el grado de compromiso glandular determina si el déficit de estas hormonas es parcial o total con consecuencias clínicas leves o severas. El diagnóstico en muchas oportunidades se realiza con pruebas de estimulación o evaluaciones periódicas múltiples de función hipofisiaria con medición de niveles hormonales. Las terapias de reemplazo hormonal varían dependiendo del déficit encontrado, así como la etapa de vida del paciente.
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Paris, Claudia, Ana Zepeda, Mónica Muñoz, Adela Camus, Paula Catalán, Cristian Sotomayor, Rosario Luengo, Carolina Schulin-Zeuthem, Mariela Brieba, and Patricia Romero Fuentes. "Reserva ovárica y falla ovárica prematura en niñas y adolescentes postrasplante de progenitores hematopoyéticos." Andes Pediatrica 93, no. 1 (February 14, 2022): 19. http://dx.doi.org/10.32641/andespediatr.v93i1.3693.

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El aumento de la sobrevida de niñas y adolescentes después de un trasplante de progenitores hemato- poyéticos (TPH) ha permitido conocer los efectos tardíos de esta terapia. Objetivo: medir la reserva y la falla ováricas prematura después de un TPH.Pacientes y Método: estudio descriptivo, transversal de niñas y adolescentes trasplantadas entre 1999-2011. Se realizó examen ginecológico, exámenes hormonales y ecotomografía ginecológica transabdominal, y se consignó desarrollo puberal pre TPH. Se registraron los siguientes datos de la ficha clínica: patología de base, tipo de acondicionamiento, uso de radioterapia en el acondicionamiento, edad al momento del TPH y haber tenido o tener enfermedad del injerto contra el huésped (EICH) aguda o crónica. Los exámenes hormonales incluyeron hormona folículo estimulante (FSH), hormona luteinizante (LH), estradiol, prolactina (PRL), hormona tiroestimulante (TSH), tiroxina libre, testosterona total, proteína transportadora de hormonas sexuales (SHBG) y hormona antimulleriana (AMH). Análisis estadístico: chi cuadrado y exacto de Fisher con P < 0,05. Resultados fueron expresados como mediana (rango). Resultados: Se evaluaron 41 pacientes, edad al momento del TPH de 6,8 años (1,5-14,1) y 14,8 años (rango: 4-25,4) al momen- to de la evaluación. El 93% de las pacientes trasplantadas tenían patologías oncológicas y recibieron regímenes de acondicionamiento mieloablativos. Todas mostraron una reserva ovárica disminuida. El 72% de las pacientes presentaron una falla ovárica prematura (FOP). Conclusiones: Todas las pacientes estudiadas tenían una reserva ovárica disminuida y la mayoría de ellas una alta prevalencia de FOP. Es indispensable una evaluación ginecológica antes del TPH y un seguimiento posterior para monitorización hormonal e inicio del reemplazo hormonal.
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Becerra Urrego, Rodrigo, and Juan Carlos Galvis Rincón. "Perfil hormonal del envejecimiento muscular." Revista Repertorio de Medicina y Cirugía 20, no. 4 (December 1, 2011): 217–24. http://dx.doi.org/10.31260/repertmedcir.v20.n4.2011.772.

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El concepto de sarcopenia implica la pérdida de la masa y la fuerza musculares asociadas con el envejecimiento. Se produce por múltiples factores, como el daño oxidativo, pérdida de unidades motoras alfa de la médula espinal, disminución de la calidad y masa musculares, descenso de hormonas anabólicas como dehidroepiandrosterona, testosterona, estrógenos, hormona del crecimiento, factor de crecimiento ligado a la insulina tipo IGF-1, hormona paratiroidea e insulina, así como aumento de diferentes interleuquinas y del cortisol, inactividad física voluntaria y dieta hipocalórica. En esta revisión analizamos el papel de algunas hormonas que afectan el balance dinámico entre estímulos anabólicos y catabólicos en el músculo y la relación entre estos cambios hormonales y el desarrollo de sarcopenia.
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Rueda-Galvis, Myriam Vanessa, and Carlos Alfonso Builes-Barrera. "Fisiología de la tiroides e hipotiroidismo en el embarazo. Revisión de tema." Medicina y Laboratorio 26, no. 1 (January 11, 2022): 15–33. http://dx.doi.org/10.36384/01232576.557.

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Durante el embarazo se generan múltiples cambios fisiológicos a nivel hormonal para llevar a cabo de manera satisfactoria la gestación. Uno de los ejes hormonales con cambios más importantes que repercuten de manera directa en el desarrollo fetal y bienestar materno es el tiroideo, el cual presenta modificaciones para lograr suplir las necesidades de hormona tiroidea tanto materna como fetal, principalmente en las primeras etapas del embarazo. Entre estas, se describen cambios en la cantidad de proteínas transportadoras de hormonas, aumento en el estímulo y producción de hormonas tiroideas, incremento del aclaramiento renal de yodo y alteración en la actividad de las desyodinasas. Estos mecanismos ofrecen suficiente hormona tiroidea al feto, el cual es dependiente del aporte materno. Un desajuste en cualquiera de estos mecanismos, puede conducir al desarrollo de hipotiroidismo con múltiples complicaciones, como la pérdida del embarazo e hipertensión gestacional, entre otras. Una tamización oportuna y un tratamiento temprano pueden evitar estos desenlaces adversos. De ahí la necesidad fundamental de conocer y comprender el comportamiento del eje tiroideo en la gestación.
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Dong, Bing, and Feng-Qi Zhao. "Involvement of the ubiquitous Oct-1 transcription factor in hormonal induction of β-casein gene expression." Biochemical Journal 401, no. 1 (December 11, 2006): 57–64. http://dx.doi.org/10.1042/bj20060570.

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Transcription of the milk protein β-casein gene is induced by the lactogenic hormones Prl (prolactin) and glucocorticoids. Multiple transcription factors involved in this induction have been identified, including the STAT5 (signal transducer and activator of transcription 5) and the GR (glucocorticoid receptor). Our previous studies have identified a binding site for the ubiquitous Oct-1 (octamer-binding transcription factor 1) protein in the lactogenic hormonal regulatory region of the mouse β-casein promoter. In the present study, we report that Oct-1 is indeed expressed and binds to the β-casein promoter in mammary epithelial cells. Oct-1 activates hormonally induced β-casein promoter activity in a dose-dependent manner. Hormonal induction of promoter activity was decreased not only by mutating the Oct-1-binding site from ATTAGCAT to GCTAGCAT, which abolishes Oct-1 binding (50% decrease, P<0.01), but also by changing the site to the consensus Oct-1-binding motif ATTTGCAT (40% decrease, P<0.01). Reversing the Oct-1-binding site reduced hormonal induction by 70% (P<0.01), showing that orientation of Oct-1 binding is also critical in hormonal action. In transient transfection experiments, Oct-1 collaboratively transactivated the β-casein gene promoter with STAT5 and/or GR in the presence of Prl receptor in cells treated with the lactogenic hormones. The C-terminus of Oct-1 was not essential to its function. The results of the present study provide biochemical evidence that the ubiquitous Oct-1 transcription factor may be involved in hormonally regulated, tissue-specific β-casein gene expression.
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Hunt, Joan S., Lance Miller, and Jeralyn Sue Platt. "Hormonal Regulation of Uterine Macrophages." Developmental Immunology 6, no. 1-2 (1998): 105–10. http://dx.doi.org/10.1155/1998/87527.

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Macrophages are major cellular inhabitants of cycling and pregnant mammalian uteri. Their densities and patterns of tissue distribution in this organ fluctuate in concert with levels of circulating female sex steroid hormones, estrogens and progesterone, and their production of various effector molecules also may be hormonally regulated. Hormonal control may be achieved by direct binding to receptors or by indirect pathways where hormones modulate production of various autocrine and paracrine cytokines and growth factors that then target to resident macrophages and influence their secretory profiles. In this paper, we marshall evidence supporting the concept that progesterone acts as a powerful negative regulator of these versatile cells, reducing their migration into the uterus and impairing their ability to produce potent effector molecules such as nitric oxide that could interfere with the success of pregnancy.
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Gonzáles Campos, Oscar, Rosario Gutiérrez, Juan Matzumura, and Alfredo Larrañaga. "Terapia Hormonal de Reemplazo." Revista Peruana de Ginecología y Obstetricia 42, no. 4 (August 2, 2015): 184–94. http://dx.doi.org/10.31403/rpgo.v42i1743.

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Shrestha, Smriti. "Correlation of Hormonal Profile and Lipid Levels with Female Adult Acne in a Tertiary Care Center of Nepal." Journal of Nepal Health Research Council 16, no. 2 (July 3, 2018): 222–27. http://dx.doi.org/10.33314/jnhrc.v16i2.1178.

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Background: Acne beyond 25 years of age is frequently associated with hormonal derangement in women. Hormonal association provides the impetus for hormonal therapy as well as underpins the need for blood investigations in this population. Hence, we aim to estimate the presence of hormonal derangement and lipid alteration in female adult acne.Methods: A prospective, observational study was conducted in Dhulikhel Hospital from July 2015 to February 2016. Females older than 25 years with acne were taken in the study after informed consent. Total 100 patients were enrolled after sample size estimation. Hormonal paneland lipid profile were measured. Hormones tested were androgens, C-peptide and thyroid stimulating hormone.Data analysis was done with SPSS-23. Bivariate analysis was done by chi-square test for categorical data.Results: In this study, majority of patients were younger than 30 years (70.5%) and perioral area most commonly involved. Hormonal alteration was seen in 37.2% patients, among which17.9% had hyperandrogenism, 15.4% had abnormal thyroid level and 10.3% had high C-peptides respectively. Lipid profile was altered in 15.4% patients. Hormonal alteration had significant association with irregular menstruation (P<0.05) but not acne severity. Conclusions: We observed hormonal alteration frequently in females with adult acne, which comprised of various hormonal parameters including hyperandrogenism.Hormonal alteration reflects deranged metabolic milieu and we suggest that wide hormonal panel should be done in female adult acne. Relationship of hormones with menstrual irregularity but not with acne severity, suggest that clinical symptoms should lead hormonal investigations in all grades of acne.Keywords: Acne; adult; hormones; lipid; thyroid.
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Khudaiberdiev, Sadik Tursunovich. "HORMONAL FUNCTION OF ADENOHYPOPHISIS." American Journal of Medical Sciences and Pharmaceutical Research 04, no. 02 (February 1, 2022): 33–37. http://dx.doi.org/10.37547/tajmspr/volume04issue02-08.

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This article discusses hormonal function of adenohypophysis, which produces a number of hormones that regulate the growth of the child. The production of adenohypophysis hormones, in turn, depends on liberins and statins, hormones of the hypothalamus that enter the pituitary portal system.
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Usselman, Charlotte W., Chantelle A. Nielson, Torri A. Luchyshyn, Tamara I. Gimon, Nicole S. Coverdale, Stan H. M. Van Uum, and J. Kevin Shoemaker. "Hormone phase influences sympathetic responses to high levels of lower body negative pressure in young healthy women." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 311, no. 5 (November 1, 2016): R957—R963. http://dx.doi.org/10.1152/ajpregu.00190.2016.

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We tested the hypothesis that sympathetic responses to baroreceptor unloading may be affected by circulating sex hormones. During lower body negative pressure at −30, −60, and −80 mmHg, muscle sympathetic nerve activity (MSNA), heart rate, and blood pressure were recorded in women who were taking ( n = 8) or not taking ( n = 9) hormonal contraceptives. All women were tested twice, once during the low-hormone phase (i.e., the early follicular phase of the menstrual cycle and the placebo phase of hormonal contraceptive use), and again during the high-hormone phase (i.e., the midluteal phase of the menstrual cycle and active phase of contraceptive use). During baroreceptor unloading, the reductions in stroke volume and resultant increases in MSNA and total peripheral resistance were greater in high-hormone than low-hormone phases in both groups. When normalized to the fall in stroke volume, increases in MSNA were no longer different between hormone phases. While stroke volume and sympathetic responses were similar between women taking and not taking hormonal contraceptives, mean arterial pressure was maintained during baroreceptor unloading in women not taking hormonal contraceptives but not in women using hormonal contraceptives. These data suggest that differences in sympathetic activation between hormone phases, as elicited by lower body negative pressure, are the result of hormonally mediated changes in the hemodynamic consequences of negative pressure, rather than centrally driven alterations to sympathetic regulation.
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Dissertations / Theses on the topic "Hormonal"

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Hussein, Mohamed Osman. "Hormonal modulation of Leydig cell membrane luteinizing hormone receptors /." The Ohio State University, 1986. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487267024995937.

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Kirkpatrick, Bridgette Lee 1966. "Hormonal regulation of gonadotropin releasing hormone receptor expression in the ewe." Diss., The University of Arizona, 1998. http://hdl.handle.net/10150/282660.

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Endocrine regulation of expression of GnRH receptors is an important step in the control of reproduction. During the early follicular phase of the estrous cycle in the ewe, GnRH receptor expression increases in preparation for the preovulatory surge of LH. The studies described herein were designed to further elucidate the hormonal interactions controlling GnRH receptor expression. In long-term ovariectomized ewes, neither removal of progesterone, nor the presence of estradiol affected the expression of GnRH receptors. However, in ewes ovariectomized during the luteal phase of the estrous cycle and immediately implanted with progesterone and estradiol for 48 hours, low levels of estradiol for 24 hours were required to increase GnRH receptor mRNA following the removal of progesterone. In ovariectomized ewes following hypothalamic-pituitary disconnection, low levels of estradiol and pulsatile GnRH were required to increase GnRH receptor expression within 24 hours of treatment initiation. These results suggest an interaction between estradiol and GnRH is involved in increasing GnRH receptor expression during the periovulatory period. How progesterone, estradiol and, GnRH interact to increase GnRH receptors is unknown, but a possible candidate involved in mediating these interactions may be the cell specific transcription factor, steroidogenic factor-1 (SF-1). SF-1 mRNA increased within 24 hours of treatment of ewes with prostaglandin F₂(α) compared to ewes in the luteal phase of the estrous cycle. This suggests that progesterone may have an inhibitory effect on SF-1 mRNA. SF-1 mRNA was similar between ovariectomized ewes and ovariectomized ewes following hypothalamic-pituitary disconnection treated with estradiol and GnRH. Treatment with estradiol or GnRH alone did not increase SF-1 mRNA. The results of these experiments suggest that progesterone removal as well as the presence of estradiol and GnRH are required to increase GnRH receptor expression during the early follicular phase in the ewe. Further, the transcription factor, SF-1 may be involved in mediating the effects of these hormones on GnRH receptor expression.
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Rodrigues, Mark. "Hormonal and non-hormonal factors associated with cognition in post-menopausal women." University of Western Australia. School of Psychiatry and Clinical Neurosciences, 2004. http://theses.library.uwa.edu.au/adt-WU2004.0075.

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[Truncated abstract.] Alzheimer’s disease (AD) is the most common form of dementia world-wide accounting for more than two thirds of all dementia cases. AD is characterised by the presence of extracellular amyloid plaques, neurofibrillary tangles and congophillic amyloid angiopathy in the brain tissue of affected individuals. Of these neuropathological features the extracellular amyloid plaques are the most characteristic containing a peptide termed amyloid- beta (Aβ); the major protein component of these structures. In addition a number of genetic risk factors for AD have been identified. Of these the ε4 allele of the apolipoprotein E (APOE) gene found on chromosome 19 is considered to be the main genetic risk factor attributing to about 40-60% of all AD cases in most populations. Although there is strong evidence that genetic risk factors play an important role in AD they do not actually trigger the disease process. Deficits in memory and learning are the most common clinical signs of AD in the initial stages of the disease. Neuropsychological tests such as the CAMCOG and California Verbal Learning Test (CVLT) are important diagnostic tools used for the assessment of cognition. The CAMCOG is an accurate and efficient measure of global cognitive ability, while the CVLT is more specific to areas of cognition influenced in the early stages of the disease such as verbal memory. Substantial evidence indicates that changes in sex hormones following menopause in women are important factors in AD. Specifically, the reduced levels of oestrogen in post-menopausal women have been linked to cognitive decline and an increased risk of dementia. In addition the elevated level of the gonadotropins, a characteristic of the post-menopausal period, have been implicated with the disease. Numerous nonhormonal factors such as age and education may also be associated with the development and progression of cognitive decline.
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Nyeko, Moini Brian Anyau. "Hormonal Contraceptives for men." Thesis, Uppsala universitet, Institutionen för farmaceutisk biovetenskap, 2018. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-422360.

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Background: Contraceptives for men has existed for centuries. However, the only contraceptives available for men are condoms, withdrawal during sexual intercourse or undergoing Vas occlusion. The issues currently with these methods are that many couples are not comfortable with using condoms and vasectomy is a non-reversible contraceptive as it requires surgery. Currently, only research has been made with regards to hormonal contraceptives for men. The basis for hormonal contraceptives for men is that it disturbs hypothalamic-pituitary–gonadal axis, by suppressing GnRH, LH and FSH in order to suppress spermatogenesis. The substances that have been clinically tested as a potential hormonal contraceptive for men are androgens, androgens together progestins and synthetic androgens. Aim: The aim of this thesis was to examine the effectiveness of the substances that have been used in past and recent clinical trials and the adverse drug reactions/effects. Method: Results from the clinical trials were collected through PubMed with regards to preordained criteria, which resulted in 18 scientific articles being used in the results. Results: Overall, the results showed that the majority of substances used in previous and recent clinical trials are effective in suppressing spermatogenesis. The results also showed that a majority of substances used in previous and recent clinical trials had severe adverse drug effects severe amongst the participants. Discussion and Conclusion:  In summary, the research shows that amongst the substances used in recent and current clinical trials, that synthetic androgens have the best potential as a hormonal contraceptive for men with regards to effectiveness and adverse drug effects.
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GUENEAU, SYLVIE. "Le cancer du sein chez l'homme : approche hormonale a propos de 20 cas." Aix-Marseille 2, 1990. http://www.theses.fr/1990AIX20151.

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Ligerot, Yasmine. "Rôle des strigolactones dans le développement de l’architecture aérienne de la plante en interaction avec les autres hormones végétales." Thesis, Université Paris-Saclay (ComUE), 2015. http://www.theses.fr/2015SACLS118/document.

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La croissance et le développement des plantes sont sous l’influence de nombreux facteurs génétiques et environnementaux. Parmi eux les hormones végétales participent aux multiples processus qui conduisent à la formation d’une plante via la mise en place d’un réseau complexe d’interactions et de rétro-contrôles. Le contrôle de la ramification implique trois d’entre-elles : l’auxine et les strigolactones (SLs) qui inhibent le démarrage des bourgeons et les cytokinines (CKs) qui le stimulent. Différents mécanismes d’interactions entre ces signaux dans le contrôle de la ramification sont connus. L’auxine contrôle les niveaux de SLs et de CKs, et les voies SLs et CKs convergent vers la même cible, le facteur de transcription BRC1.Chez le pois, les mutants hyper-ramifiés ramosus (rms) sont affectés dans la voie SLs. Les gènes RMS1 (PsMAX4) et RMS5 (PsMAX3) contrôlent la biosynthèse des SLs alors que RMS3 (PsD14) (récepteur) et RMS4 (PsMAX2) sont impliqués dans la perception des SLs. En plus de partager un phénotype hyper-ramifié, les mutants rms présentent un phénotype de nanisme. Cependant le rôle des SLs dans le contrôle de la taille des plantes n’était pas encore connu. Nous avons montré que les SLs contrôlent la taille des plantes en jouant sur le processus de division cellulaire indépendamment de la voie gibbérelline.Les mutants rms montrent des caractéristiques physiologiques similaires : un haut niveau d’expression des gènes de biosynthèse des SL et une faible teneur en CKs dans la sève xylémienne (X-CKs). En revanche, le mutant hyper-ramifié rms2 présente une faible expression des gènes de biosynthèse des SLs et une forte teneur en X-CKs. De précédentes études suggèrent que rms2 est affecté dans un signal tige-racine de rétro-contrôle régulant la biosynthèse des SLs et le niveau de X-CKs. La nature biochimique de ce signal est inconnue et il a été proposé que ce signal soit auxine-indépendant.Nous avons montré que le gène RMS2 est l’homologue chez le pois des gènes AFB4/5 d’Arabidopsis codant pour une protéine à boîte-F de la famille des récepteurs de l’auxine TIR1/AFBs (Auxin signaling F-Box). Ce qui suggère que le signal de rétro-contrôle RMS2-dépendant est l’auxine. De plus nos résultats montrent que les SLs jouent un rôle de répresseur du niveau d’auxine dans la tige via la régulation de l’expression des gènes du métabolisme de l’auxine.L’ensemble de ces résultats montrent que dans le processus de contrôle de la ramification les interactions entre auxine et SLs sont complexes. Les multiples mécanismes en jeux aboutissent à la formation d’une boucle de régulation dans laquelle chaque hormone est capable de contrôler la biosynthèse de l’autre
The different processes of plant growth and development are under the influence of growth regulators which interact in complex hormonal networks and feedback mechanisms. The control of shoot branching involves 3 key plant hormones, auxin, cytokinins (CKs) and strigolactones (SLs). Auxin and SLs repress axillary bud outgrowth whereas CKs stimulate it. Different mechanisms of interactions between these signals have already been suggested in controlling shoot branching. Auxin controls SLs and CKs levels, and SLs and CKs pathways converge on the same target, the TCP transcription factor BRC1 in the axillary bud. In pea, the high shoot branching of ramosus (rms) mutants are known for being impaired in the SLs pathway. RMS1 (PsMAX4) and RMS5 (PsMAX3) genes are involved in SL biosynthesis while RMS3 (PsD14) (receptor) and RMS4 (PsMAX2) are involved in SL perception. In addition to sharing their high branching phenotype, the rms mutants display a dwarf phenotype. However the role of SLs in controlling plant height was unknown. Here we show that SLs control internode length by acting on cell division in a Gibberellin-independent way.The rms mutants show similar physiological characteristics: high expression of the SL-biosynthesis genes and very low xylem-sap CKs (X-CKs) content. In contrast, the rms2 mutant with similar shoot phenotype shows very low expression of SL-biosynthesis genes and high X-CKs content. Previous studies suggested that rms2 was affected in a shoot-to-root feedback signal controlling both SL biosynthesis and X-CKs level. Whether this feedback signal was auxin or not was highly discussed. Here we demonstrated that the RMS2 gene is the pea homologue of the Arabidopsis AFB4/5 gene, encoding an F-box protein which belongs to the TIR1/AFB (Auxin signaling F-Box) auxin receptor family. This suggests that the RMS2-dependent feedback signal is very likely auxin. Moreover our results suggest a role for SLs in the repression of auxin content in pea stem via the regulation of auxin metabolism gene expression. These results highlighted that for the control of shoot branching, interactions between auxin and strigolactones involve multiple mechanisms leading to regulation loop where both hormones are able to regulate the biosynthesis of each other
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Isaksson, Friman Erika. "Hormonal treatments and the breast : effects on sex steroid receptor expression and proliferation /." Stockholm : [Karolinska institutets bibl.], 2002. http://diss.kib.ki.se/2002/91-7349-182-9/.

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Hair, W. Morton. "Studies in male hormonal contraception." Thesis, University of Edinburgh, 2004. http://hdl.handle.net/1842/24662.

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To date androgens remain a central part of any hormonal contraceptive for men although understanding the heterogeneity of the suppression response within populations may require consideration of less orthodox hormonal regulatory systems. Development of minimally invasive, long acting androgen formulations is also necessary to provide an acceptable, convenient and reliable form of androgen delivery which men themselves can administer. Experimental studies in animals have established prolactin as a progonadal hormone in the testis and accessory glands. To explore the role of prolactin in men we investigated the localization and functional activation of the prolactin receptor in the human testis and accessory tissues by immunohistochemistry, RT-PCR and activation of the Janus Kinase/Signal Transducer and Activator of Transcription and Mitogen Activated Protein kinase and Extracellular signal-Regulated Kinase signalling pathways. Expression of prolactin receptor was localized to the Leydig cells and differentiating cells of the testis, the epithelium of vas deferens, epididymis, prostate and seminal vesicles. Functional activation of prolactin receptor was demonstrated in fresh samples of vas deferens. The second study investigated whether concomitant suppression of PRL with the dopamine receptor agonist quinagolide (Q), would enhance the efficacy of testosterone (T) as a contraceptive in men. Volunteers were treated orally with Q, to chronically suppress PRL secretion. A high and an intermediate dose of T was selected to establish whether PRL inhibition would allow use of a lower dose of androgen to induce azoospermia in men. Q did not seem to confer additional efficacy but difficulties in chronic suppression of PRL precludes definitive assessment. The final study describes a clinical trial employing a subject administered, non-invasive hormonal contraceptive regime. Men received transdermal T patches and the synthetic progestagen desogestrel orally in a down ward dose finding study. Transdermal T was less effective than injectable androgen formulations and the minimally effective dose of DSG is 150 mg/day.
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Naguib, Raouf Edouard. "Hormonal modulation of renal autoregulation." Thesis, McGill University, 1995. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=22864.

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Autoregulation of renal blood flow is mediated by two mechanisms. The myogenic response operates at $ approx$0.1-0.2 Hz and tubuloglomerular feedback (TGF) operates at $ approx$0.03-0.05 Hz. Atrial natriuretic factor (ANF) dilates pre-glomerular resistance vessels, in which autoregulation occurs, and has been reported to inhibit TGF. We tested the potential actions of ANF on TGF-mediated autoregulation using Sprague-Dawley rats anesthetized with isoflurane. Renal perfusion pressure (RPP) was manipulated by a servo-controlled clamp placed on the aorta between the renal arteries. Time constants for constriction and dilatation were consistent with operation of TGF. ANF did not affect either the magnitude or the time constants of the response.
To test the contribution of TGF resetting to the wide dynamic range of steady state autoregulation experiments, the autoregulatory range was first defined by conventional stepwise reduction of RPP. As ANG II is necessary for TGF resetting, the experiment was repeated in the presence of Losartan, a competitive ANG Il-AT$ sb1$ receptor antagonist. The results do not support the hypothesis that TGF resetting contributes to the wide dynamic range of steady state autoregulation. In fact, the lower limit of autoregulation was extended to a lower RPP. In the Losartan experiment, this shift was not apparent, suggesting that it was ANG II-dependent. (Abstract shortened by UMI.)
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Kunovac, Kallak Theodora. "Hormonal Regulation of Vaginal Mucosa." Doctoral thesis, Uppsala universitet, Institutionen för kvinnors och barns hälsa, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-246215.

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Vaginal atrophy symptoms such as dryness, irritation, and itching, are common after menopause. Vaginal estrogen therapy is the most effective treatment but not appropriate for all women. Women with estrogen-responsive breast cancer treated with aromatase inhibitor (AI) treatment, suppressing estrogen levels, often suffer from more pronounced vaginal atrophy symptoms. However, vaginal estrogen treatment is not recommended, leaving them without effective treatment options. The aim of this thesis was to study the effect of long-term anti-estrogen therapy on circulating estrogen levels and biochemical factors in vaginal mucosa in relation to morphological changes and clinical signs of vaginal atrophy. Circulating estrogen levels were analyzed by use of mass spectrometry and radioimmunoassay. Immunohistochemistry was used to study vaginal proliferation and steroid hormone receptors in vaginal mucosa. Vaginal gene expression was studied by use of microarray technology and bioinformatic tools, and validated by use of quantitative real-time PCR and immunohistochemistry. An estrogenic regulation of aquaporins and a possible role in vaginal dryness was investigated in vaginal mucosa and in Vk2E6E7 cells. Aromatase inhibitor-treated women had higher than expected estradiol and estrone levels but still significantly lower than other postmenopausal women. Aromatase was detected in vaginal tissue, the slightly stronger staining in vaginal mucosa from AI-treated women, suggest a local inhibition of vaginal aromatase in addition to the systemic suppression. Vaginal mucosa from AI-treated women had weak progesterone receptor, and strong androgen receptor staining intensity. Low estrogen levels lead to low expression of genes involved in cell adhesion, proliferation, and differentiation as well as weak aquaporin 3 protein immunostaining. The higher than expected estrogen levels in AI-treated women suggest that estrogen levels might previously have been underestimated. Systemic estrogen suppression by treatment with AIs, and possibly also by local inhibition of vaginal aromatase, results in reduced cell adhesion, proliferation, differentiation, and weak aquaporin 3 protein staining. Low proliferation and poor differentiation leads to fewer and less differentiated superficial cells affecting epithelial function and possibly also causing vaginal symptoms. Aquaporin 3 with a possible role in vaginal dryness, cell proliferation, and differentiation should be further explored for the development of non-hormonal treatment options for vaginal symptoms.
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Books on the topic "Hormonal"

1

Govil, Prabodh Kumar. Hormonal Fencing: Hormonal Fencing. Jaipur,India: Monika Prakashan Jaipur, 2014.

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Kleinman, R. L. Directory of hormonal contraceptives =: Répertoire des contraceptifs hormonaux = Guía de anticonceptivos hormonales. London: IPPF International Office, 1988.

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Li, Jonathan J., Satyabrata Nandi, and Sara Antonia Li, eds. Hormonal Carcinogenesis. New York, NY: Springer New York, 1992. http://dx.doi.org/10.1007/978-1-4613-9208-8.

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Shulman, Neil. Hormonal Balance. Chicago: Bull Publishing, 2008.

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Équilibre hormonal. Montréal]: Modus Vivendi, 2006.

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Humans, IARC Working Group on the Evaluation of Carcinogenic Risks to. Hormonal contraception and post-menopausal hormonal therapy. Lyon, France: IARC, 1999.

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J, Li Jonathan, and International Symposium on Hormonal Carcinogenesis (5th : 2006 : Montpellier, France), eds. Hormonal carcinogenesis V. New York, NY: Springer, 2008.

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Li, Jonathan J., Sara A. Li, Suresh Mohla, Henri Rochefort, and Thierry Maudelonde, eds. Hormonal Carcinogenesis V. New York, NY: Springer New York, 2008. http://dx.doi.org/10.1007/978-0-387-69080-3.

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Li, J. Jonathan, Sara Antonia Li, Jan-Åke Gustafsson, Satyabrata Nandi, and Lea I. Sekely, eds. Hormonal Carcinogenesis II. New York, NY: Springer New York, 1996. http://dx.doi.org/10.1007/978-1-4612-2332-0.

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Li, Jonathan J., Sara A. Li, and Antonio Llombart-Bosch, eds. Hormonal Carcinogenesis IV. Boston, MA: Springer US, 2005. http://dx.doi.org/10.1007/b102371.

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Book chapters on the topic "Hormonal"

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Seifert, Roland. "Sex Hormones: Hormonal Contraception and Hormone Replacement Therapy." In Basic Knowledge of Pharmacology, 287–301. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-18899-3_24.

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Hutson, John M., Jørgen M. Thorup, and Spencer W. Beasley. "Hormonal Treatment." In Descent of the Testis, 149–58. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-25910-9_9.

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Kaaks, R. "Hormonal Carcinogenesis." In Mechanisms in Carcinogenesis and Cancer Prevention, 141–67. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-662-08602-5_9.

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Cowper, Ann, and Cyril Young. "Hormonal contraception." In Family Planning, 52–74. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4899-3266-2_7.

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Hamilton, Colin. "Hormonal Influences." In Cognition and Sex Differences, 132–49. London: Macmillan Education UK, 2008. http://dx.doi.org/10.1007/978-1-137-01967-7_7.

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Bruni, Vincenzina, Metella Dei, and Francesca Pampaloni. "Hormonal Contraception." In Endocrinology, 1–30. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-03594-5_7-1.

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Tubiana-Hulin, M., and P. Soulié. "Hormonal Therapy." In Bone Metastases, 55–71. London: Springer London, 2002. http://dx.doi.org/10.1007/978-1-4471-3251-6_6.

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Rübe, Claudia E., Bernadine R. Donahue, Jay S. Cooper, Caspian Oliai, Yan Yu, Laura Doyle, Rene Rubin, et al. "Hormonal Agents." In Encyclopedia of Radiation Oncology, 332–33. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-540-85516-3_749.

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Arcos, Joseph C., Mary F. Argus, and Yin-tak Woo. "Hormonal Carcinogenesis." In Chemical Induction of Cancer, 419–30. Boston, MA: Birkhäuser Boston, 1995. http://dx.doi.org/10.1007/978-1-4612-4076-1_23.

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Banerjee, Sushanta K. "Hormonal Carcinogenesis." In Encyclopedia of Cancer, 1–4. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-27841-9_2809-2.

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Conference papers on the topic "Hormonal"

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Kusumawati, Widya, and Lely Khulafa’ur Rosidah. "The Effect of Hormonal Contraception on Body Mass Index among Women in Reproductive Age." In The 7th International Conference on Public Health 2020. Masters Program in Public Health, Universitas Sebelas Maret, 2020. http://dx.doi.org/10.26911/the7thicph.03.20.

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ABSTRACT Background: Hormonal contraception is still a popular contraceptive method for most women. More than six million women worldwide use the injectable hormonal contraceptive method. The hormones estrogen and progesterone contained in hormonal birth control can affect the increase of sodium and fluids. This will affect the fat layer and appetite which will cause weight gain, thus impacting body mass index (BMI). This study aimed to determine the effect of hormonal contraception on BMI. Subjects and Method: This was a cross-sectional study conducted in Ngampel village, Mojoroto District, Kediri, East Java, in June – July 2019. The total of 30 women in reproductive age were selected using purposive sampling technique. The independent variable was the use of hormonal contraception. the dependent variable was BMI. The data were collected by questionnaire then analyzed using Chi Square test. Results: The use of hormonal contraception was increased the BMI among women in reproductive age. Conclusion: The use of hormonal contraception can give an effect on BMI. Sometimes, woman body should be given the opportunity to rest using non-hormonal birth control. Keywords: family planning, hormonal contraception, body mass index Correspondence: Widya Kusumawati. Dharma Husada Kediri Academy of Midwifery. Jl. Coverage No 41 A Kediri City. Email: widya.koesoemawati@gmail.com. Mobile: 085722223910. DOI: https://doi.org/10.26911/the7thicph.03.20
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Bunting, Haley A., Ryan T. Cassilly, Brian Jin, Christopher S. Ahmad, Louis U. Bigliani, William N. Levine, and Thomas R. Gardner. "Effect of Hormone Therapy on Tensile Strain of the Macaque Inferior Glenohumeral Ligament." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53531.

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The effect of hormone treatment on the material properties of ligaments has been extensively studied for the anterior cruciate ligament (ACL). However, there have been few studies on the effects of hormones on the material properties of the shoulder. Shoulder ligaments contribute to overall shoulder stability, and a change in ligament properties could contribute to a change in overall shoulder laxity. Cynomolgus monkeys have served as nonhuman primate models in studies examining the effects of hormone replacement therapy on the cardiovascular system, as well as serving as a model for menopause, reproductive support structures, the knee joint and shoulder joint. The cynomolgus macaques are commonly used for studies involving hormone replacement therapy because they have 28-day menstrual cycles with very similar hormonal patterns to that of women. This study uses female cynomologus macaque (Macaca fascicularis) monkeys as an animal model of the human shoulder to determine if estrogen has an effect on the strain distribution of the inferior glenohumeral ligament (IGHL).
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Gardner, Thomas R., Ryan T. Cassilly, Brian Jin, Anuli N. Mkparu, Christoper S. Ahmad, Louis U. Bigliani, and William N. Levine. "Effect of Estrogen on Viscoelastic Properties of the Anterior Pouch of the Macaque Animal Model of the Inferior Glenohumeral Ligament." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206835.

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The effect of hormone treatment on the material properties of ligaments has been extensively studied for the anterior cruciate ligament (ACL). However, there have been very few studies on the effects of hormones on the material properties of the shoulder. These shoulder ligaments contribute to overall shoulder stability, and a change in ligament properties could contribute to a change in overall shoulder laxity. This study uses female cynomologus macaque (Macaca fascicularis) monkeys as an animal model of the human shoulder to determine if estrogen has an effect on the viscoelastic properties of the anterior pouch of the inferior glenohumeral ligament (IGHL). Cynomolgus monkeys have served as nonhuman primate models in several studies, including monkey models of menopause, the effects of hormone replacement therapy on the cardiovascular system, reproductive support organs, and the knee joint. The cynomolgus macaques are commonly used for studies involving hormone replacement therapy because they have 28-day menstrual cycles with very similar hormonal patterns to that of women.
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Caldwell, Jon-Michael E., Ryan T. Cassilly, Haley A. Bunting, Christopher S. Ahmad, Louis U. Bigliani, William N. Levine, and Thomas R. Gardner. "Effects of Hormone Therapy on Regional Surface Strain as a Function of Applied Strain in the Macaque Inferior Glenohumeral Ligament." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80840.

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Hormones such as estrogen are known to have an effect on the biomechanical properties of certain ligaments such as the anterior cruciate ligament in the knee; however, relatively little is known about its effect on the ligaments of the shoulder. The inferior glenohumeral ligament (IGHL) is a static stabilizer of the shoulder that prevents anterior translation of the humeral head. Alterations to the properties of this ligament can result in capsular stretching, increased laxity, and ultimately instability. The cynomolgus macaque (Macaca fascicularis) shares many hormonal similarities with humans including a 28-day menstrual cycle and is a commonly used model for hormone replacement therapy studies. This study uses the female cynomolgus monkey as a model of the human shoulder to determine if estrogen has an effect on the regional surface strain behavior of the inferior glenohumeral ligament.
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Magalhães, Paolla Giovanna Rossito de, Marina Buldrini Filogonio Seraidarian, Bernardo Tardin Caetano, Barbara Oliveira Paixão, Tassila Oliveira Nery de Freitas, Daniel Vasconcelos de Pinho Tavares, Gabriella Braga da Cunha Silva, et al. "Case Report: Reversible Cerebral Vasoconstriction Syndrome in a cancer patient using ZoladexTM." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.142.

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Context: The Reversible Cerebral Vasoconstriction Syndrome (SVCR) is characterized by rapid and reversible vasoconstriction and segmental dilation of cerebral arteries, usually preceded by thunderclap headache. The involvement of second and third-order branches of the cerebral arteries is the most commom finding in a cerebral angiography. This report is about a SVCR case with atypical involvement, significantly compromising extracranial vessels and raising the hypothesis of association between the use of hormonal blocker gosserelin acetate (ZoladexTM) with SVCR. Case report: Female, 39 years old, with breast cancer and bone metastasis using ZoladexTM that presented with a sudden headache and vomiting, progressing to global afasia and paresis in the right upper limb. Magnetic resonance identified hyperacute intraparenchymal hematoma in left frontoparietal convexity and subarachnoid haemorrhage. Cerebral angiography showed irregularities in the distal branches (M3 and M4) of the middle cerebral arteries, as well as in the superficial temporal artery, characterized by focal strictures. Conclusion: Studies show that hormonal fluctuations in the postpartum period can trigger SVCR due to the drop in estrogen and progesterone (gonadotropins). During postpartum, the stimulus of breastfeeding increases prolactin levels leading to GnRH suppression, which decreases the level of gonadotropins. ZoladexTM is a GnRH analogue and its chronic administration results in suppression of these hormones - similar to the postpartum period. Therefore, there may be an association of hormonal blockers with SVCR.
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Afriyanti, Neta, Eti Poncorini Pamungkasari, and Hanung Prasetya. "The Effect of Hormonal Contraceptive Use on the Risk of Depression in Women of Reproductive Age: Evidence from Gunungkidul, Yogyakarta." In The 7th International Conference on Public Health 2020. Masters Program in Public Health, Universitas Sebelas Maret, 2020. http://dx.doi.org/10.26911/the7thicph.03.121.

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ABSTRACT Background: Estrogen and progesterone hormones have been hypothesized to play a role in the cause of depressive symptoms in female. Clinical studies have indicated that changes in estrogen levels may trigger depressive episodes among women at risk for depression. A previous study found that use of combined oral contraceptives among women who previously had experienced emotional adverse effects resulted in mood deterioration and changes in emotional brain reactivity. This study aimed to examine the effect of hormonal contraceptive use on the risk of depression in women of reproductive age. Subjects and Method: A cross-sectional study was carried out in Gunungkidul, Yogyakarta. A sample of 200 women of reproductive age was selected by purposive sampling. The dependent variable was depression symptom. The independent variables were age, education, duration of contraceptive use, type of contraceptive use, employment, and family income. Depression symptom was measured by Beck Depression Inventory II (BDI II). The other variables were collected by questionnaire. The data were analyzed by a multiple logistic regression run on Stata 13. Results: Depression symptoms increased with hormonal contraception (OR= 3.74; 95% CI= 1.61 to 8.65; p= 0.001) and duration of contraceptive use ≥36 months (OR= 6.33; 95% CI= 2.36 to 16.97; p <0.001). Depression symptoms decreased with age ≥29 years (OR= 0.12; 95% CI= 0.04 to 0.34; p <0.001), family income ≥Rp 1,571,000 (OR= 0.10; 95% CI= 0.04 to 0.24; p <0.001), education ≥Senior high school (OR= 0.18; 95% CI= 0.07 to 0.45; p<0.001), and working at home (OR= 0.37; 95% CI= 0.16 to 0.83; p= 0.016). Conclusion: Depression symptoms increase with hormonal contraception and duration of contraceptive use ≥36 months. Depression symptoms decrease with age ≥29 years, family income ≥Rp 1,571,000, education ≥Senior high school, and working at home. Keywords: hormonal contraceptive, depression Correspondence: Neta Afriyanti. Masters Program in Public Health, Universitas Sebelas Maret. Jl. Ir. Sutami 36 A, Surakarta 57126, Central Java. Email: neta.friyanti16@gmail.com DOI: https://doi.org/10.26911/the7thicph.03.121
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Lomin, S. N., O. O. Kolachevskaya, and G. A. Romanov. "Hormonal regulation of tuberization in potatoes." In IX Congress of society physiologists of plants of Russia "Plant physiology is the basis for creating plants of the future". Kazan University Press, 2019. http://dx.doi.org/10.26907/978-5-00130-204-9-2019-263.

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Ashlock, Daniel, Christopher Kuusela, and Nicholas Rogers. "Hormonal systems for prisoners dilemma agents." In 2011 IEEE Conference on Computational Intelligence and Games (CIG). IEEE, 2011. http://dx.doi.org/10.1109/cig.2011.6031990.

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Jain, Vikas. "Massive peripheral giant cell granuloma associated with pregnancy." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685368.

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Peripheral giant cell granuloma (PGCG) is a relatively Common reactive exophytic lesion of the oral cavity. The influence of hormones has been suggested as contributory factor in PGCG development and predominance of these lesions in young females as well as some previously reported pregnancy related cases support this belief. It has been observed that majority of lesions present in the 4th decade of life, when hormonal changes are more pronounced. Cailluette and Mattar in their study found that peripheral giant cell granuloma are under the influence of the ovarian hormones. However Chambers and Spector suggested peripheral giant cell granuloma to be enhanced by pregnancy rather than being pregnancy dependent. The responsiveness of gingiva to these hormones along with the immunosuppressive actions of the hormones contributes to the growth of the lesion. Clinically, PGCGs may present as polypoid or nodular lesions, predominantly bluish red with a smooth shiny or mamillated surface.This poster will review the literature available on the association of Massive Peripheral Giant Cell Granuloma With Pregnancy with focus on possible causes of PGCG during pregnancy.
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Dascaliuc, Alexandru. "Hormesis, screening and practical use of biostimulators in agriculture." In International Scientific Symposium "Plant Protection – Achievements and Prospects". Institute of Genetics, Physiology and Plant Protection, Republic of Moldova, 2020. http://dx.doi.org/10.53040/9789975347204.44.

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The term hormesis describes the biphasic response of any biological system to increasing the dose of the stress factor of a different nature, characterized in that low doses have stimulating, beneficial effects. In contrast, high doses cause harmful, inhibitory effects. The hormonal response is practically universal, being stimulated by the action of toxic substances, heavy metal ions, hormones, including physical factors. The standard type of response to different factors suggests installing these evolving mechanisms, so they are of particular interest in elucidating plant adaptation mechanisms to various stressors, including developing screening methods and practical use of biostimulators. The practical use of hormesis principles was the theoretical basis for elaboration and rational use of the biostimulator Reglalg in agriculture.
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Reports on the topic "Hormonal"

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Simpson, Jennifer K., Francesmary Modugno, Joel L. Weissfeld, Lewis Kuller, Victor Vogel, and Joseph P. Costantino. Hormonal Determinants of Mammographic Density. Fort Belvoir, VA: Defense Technical Information Center, August 2004. http://dx.doi.org/10.21236/ada432434.

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Simpson, Jennifer K., and Francemary Modugno. Hormonal Determinants of Mammographic Density. Fort Belvoir, VA: Defense Technical Information Center, August 2003. http://dx.doi.org/10.21236/ada420073.

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Kolev, Ognyan I., and Budin B. Mihov. Hormonal Changes Following Prenauseogenic Vestibular Stimulation. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, June 2018. http://dx.doi.org/10.7546/crabs.2018.06.16.

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Gerbi, Susan A., Alexander Brodsky, and Ben Raphael. Hormonal Involvement in Breast Cancer Gene Amplification. Fort Belvoir, VA: Defense Technical Information Center, October 2008. http://dx.doi.org/10.21236/ada501716.

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Gerbi, Susan A., Alexander Brodsky, and Ben Raphael. Hormonal Involvement in Breast Cancer Gene Amplification. Fort Belvoir, VA: Defense Technical Information Center, October 2010. http://dx.doi.org/10.21236/ada541789.

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Stachenfeld, Nina. Hormonal Contraception, Body Water Balance and Thermoregulation. Fort Belvoir, VA: Defense Technical Information Center, July 2001. http://dx.doi.org/10.21236/ada410450.

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Stachenfeld, Nina. Hormonal Contraception, Body Water Balance and Thermoregulation. Fort Belvoir, VA: Defense Technical Information Center, October 2000. http://dx.doi.org/10.21236/ada391575.

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Tom, Moshe, Milton Fingerman, and Esther Lubzens. Hormonal Control of Reproduction in Penaeid Shrimp. United States Department of Agriculture, October 1992. http://dx.doi.org/10.32747/1992.7603803.bard.

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Cohen, Jerry, and Ephraim Epstein. Hormonal and Development Control of Root Initiation. United States Department of Agriculture, January 1993. http://dx.doi.org/10.32747/1993.7603517.bard.

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Xian, Wa, and Jeffrey M. Rosen. Hormonal Regulation of Mammary Gland Development and Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, December 2005. http://dx.doi.org/10.21236/ada444687.

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