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Journal articles on the topic 'Homer'

Author: Grafiati

Published: 4 June 2021

Last updated: 30 July 2024

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1

Reibring, Claes-Göran, Kristina Hallberg, Anders Linde, and Amel Gritli-Linde. "Distinct and Overlapping Expression Patterns of the Homer Family of Scaffolding Proteins and Their Encoding Genes in Developing Murine Cephalic Tissues." International Journal of Molecular Sciences 21, no. 4 (February 13, 2020): 1264. http://dx.doi.org/10.3390/ijms21041264.

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In mammals Homer1, Homer2 and Homer3 constitute a family of scaffolding proteins with key roles in Ca2+ signaling and Ca2+ transport. In rodents, Homer proteins and mRNAs have been shown to be expressed in various postnatal tissues and to be enriched in brain. However, whether the Homers are expressed in developing tissues is hitherto largely unknown. In this work, we used immunohistochemistry and in situ hybridization to analyze the expression patterns of Homer1, Homer2 and Homer3 in developing cephalic structures. Our study revealed that the three Homer proteins and their encoding genes are expressed in a wide range of developing tissues and organs, including the brain, tooth, eye, cochlea, salivary glands, olfactory and respiratory mucosae, bone and taste buds. We show that although overall the three Homers exhibit overlapping distribution patterns, the proteins localize at distinct subcellular domains in several cell types, that in both undifferentiated and differentiated cells Homer proteins are concentrated in puncta and that the vascular endothelium is enriched with Homer3 mRNA and protein. Our findings suggest that Homer proteins may have differential and overlapping functions and are expected to be of value for future research aiming at deciphering the roles of Homer proteins during embryonic development.

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2

Son, Aran, Namju Kang, Sue Young Oh, Ki Woo Kim, Shmuel Muallem, Yu-Mi Yang, and Dong Min Shin. "Homer2 and Homer3 modulate RANKL-induced NFATc1 signaling in osteoclastogenesis and bone metabolism." Journal of Endocrinology 242, no. 3 (September 2019): 241–49. http://dx.doi.org/10.1530/joe-19-0123.

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The receptor activator of nuclear factor-kappa B ligand (RANKL) induces osteoclastogenesis by induction of Ca2+ oscillation, calcineurin activation and translocation into the nucleus of nuclear factor of activated T cells type c1 (NFATc1). Homer proteins are scaffold proteins. They regulate Ca2+ signaling by modulating the activity of multiple Ca2+ signaling proteins. Homers 2 and 3, but not Homer1, also independently affect the interaction between NFATc1 and calcineurin. However, to date, whether and how the Homers are involved in osteoclastogenesis remains unknown. In the present study, we investigated Homer2 and Homer3 roles in Ca2+ signaling and NFATc1 function during osteoclast differentiation. Deletion of Homer2/Homer3 (Homer2/3) markedly decreased the bone density of the tibia, resulting in bone erosion. RANKL-induced osteoclast differentiation is greatly facilitated in Homer2/3 DKO bone marrow-derived monocytes/macrophages (BMMs) due to increased NFATc1 expression and nuclear translocation. However, these findings did not alter RANKL-induced Ca2+ oscillations. Of note, RANKL treatment inhibited Homer proteins interaction with NFATc1, but it was restored by cyclosporine A treatment to inhibit calcineurin. Finally, RANKL treatment of Homer2/3 DKO BMMs significantly increased the formation of multinucleated cells. These findings suggest a novel potent mode of bone homeostasis regulation through osteoclasts differentiation. Specifically, we found that Homer2 and Homer3 regulate NFATc1 function through its interaction with calcineurin to regulate RANKL-induced osteoclastogenesis and bone metabolism.

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3

Zhu, Man, Jiangcheng Zuo, Ji Shen, Wei Jing, Ping Luo, Nandi Li, Xue Wen, et al. "Diagnostic Potential of Differentially Expressed Homer1 and Homer2 in Ischemic Stroke." Cellular Physiology and Biochemistry 39, no. 6 (2016): 2353–63. http://dx.doi.org/10.1159/000447927.

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Background: Ischemic stroke (IS) is an extremely heterogeneous disease with variable pathogenesis. Due to the lack of early diagnostic marker, the mortality rate of IS remains high worldwide. The family of Homer plays an important role in the pathology of atherosclerotic plaque. In this study, we have investigated its expression pattern and clinical significance in IS. Methods: RT-qPCR was performed to detect the expression of Homer1, Homer2, and Homer3. Results: We found that the mRNA levels of Homer1 (p<0.001) and Homer2 (p<0.001), but not Homer3, in large-artery atherosclerosis (LAA) strokes were significantly upregulated than those in non-LAA strokes and controls. Multinomial logistic regression analyses showed that, although none of the Homer was associated with non-LAA strokes, higher Homer1 (adjusted OR=1.337, 95% CI: 1.227-1.458) and Homer2 (adjusted OR=1.099, 95% CI: 1.062-1.138) levels showed significant associations with increased odds of having LAA stroke, compared with the controls. The receiver operating characteristic (ROC) curves showed that the combination of Homer1 and Homer2 had a better diagnostic accuracy to differentiate LAA strokes from non-LAA strokes and controls, and the sensitivity and specificity ratios were 80.5%/90.4% and 98.0%/70.3%, respectively. Conclusion: Our data suggested that Homer1 and Homer2 might be considered as novel diagnostic biomarkers for LAA stroke.

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4

Shin, Dong Min, Marlin Dehoff, Xiang Luo, Shin Hyeok Kang, Jiangchen Tu, Surendra K. Nayak, Elliott M. Ross, Paul F. Worley, and Shmuel Muallem. "Homer 2 tunes G protein–coupled receptors stimulus intensity by regulating RGS proteins and PLCβ GAP activities." Journal of Cell Biology 162, no. 2 (July 8, 2003): 293–303. http://dx.doi.org/10.1083/jcb.200210109.

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Homers are scaffolding proteins that bind G protein–coupled receptors (GPCRs), inositol 1,4,5-triphosphate (IP3) receptors (IP3Rs), ryanodine receptors, and TRP channels. However, their role in Ca2+ signaling in vivo is not known. Characterization of Ca2+ signaling in pancreatic acinar cells from Homer2−/− and Homer3−/− mice showed that Homer 3 has no discernible role in Ca2+ signaling in these cells. In contrast, we found that Homer 2 tunes intensity of Ca2+ signaling by GPCRs to regulate the frequency of [Ca2+]i oscillations. Thus, deletion of Homer 2 increased stimulus intensity by increasing the potency for agonists acting on various GPCRs to activate PLCβ and evoke Ca2+ release and oscillations. This was not due to aberrant localization of IP3Rs in cellular microdomains or IP3R channel activity. Rather, deletion of Homer 2 reduced the effectiveness of exogenous regulators of G proteins signaling proteins (RGS) to inhibit Ca2+ signaling in vivo. Moreover, Homer 2 preferentially bound to PLCβ in pancreatic acini and brain extracts and stimulated GAP activity of RGS4 and of PLCβ in an in vitro reconstitution system, with minimal effect on PLCβ-mediated PIP2 hydrolysis. These findings describe a novel, unexpected function of Homer proteins, demonstrate that RGS proteins and PLCβ GAP activities are regulated functions, and provide a molecular mechanism for tuning signal intensity generated by GPCRs and, thus, the characteristics of [Ca2+]i oscillations.

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5

Johnson, Samuel, Gustavo Althoff, and Mauri Furlan. "Translating Homer / Traduzindo Homero." Scientia Traductionis, no. 16 (June 23, 2016): 20. http://dx.doi.org/10.5007/1980-4237.2014n16p20.

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Samuel Johnson (1709-1784), crítico, ensaísta, jornalista, poeta, educador e lexicógrafo, é considerado uma das personalidades mais proeminentes no mundo intelectual da Bretanha do século XVIII. Em 1777, ele recebeu a proposta de um grupo de livreiros para escreveruma série de vidas de poetas ingleses, e entre 1779-81 foi publicada a obra TheLives of the English Poets, a qual contém a vida de Alexander Pope (1688-1744),de onde extraímos o excerto abaixo. (Robinson, 2002). Samuel Johnson elogia o trabalho de Pope na tradução de Homero e a sua contribuição para a versificação em inglês. E observa que a tradução de Pope não é fiel e não tem a simplicidade do original. Johnson, contudo, justifica as variações apresentadas por Pope em sua tradução em razão da distância existente entre aslínguas, as épocas, os lugares.

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6

Clay, Jenny Strauss. "Homer Really was Homer." Classical Review 55, no. 1 (March 2005): 8–9. http://dx.doi.org/10.1093/clrevj/bni005.

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7

Bortoloso, Elena, Nadia Pilati, Aram Megighian, Elisa Tibaldo, Dorianna Sandonà, and Pompeo Volpe. "Transition of Homer isoforms during skeletal muscle regeneration." American Journal of Physiology-Cell Physiology 290, no. 3 (March 2006): C711—C718. http://dx.doi.org/10.1152/ajpcell.00217.2005.

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Homer represents a new and diversified family of proteins that includes several isoforms, Homer 1, 2, and 3; some of these isoforms have been reported to be present in striated muscles. In this study, the presence of Homer isoforms 1a, 1b/c/d, 2b, and 3 was thoroughly investigated in rat skeletal muscles under resting conditions. Transition in Homer isoforms compositon was studied under experimental conditions of short-term and long-term adaptation, e.g., fatigue and regeneration, respectively. First, we show that Homer 1a was constitutively expressed and was transiently upregulated during regeneration. In C2C12 cell cultures, Homer 1a was also upregulated during formation of myotubes. No change of Homer 1a was observed in fatigue. Second, Homer 1b/c/d and Homer 2b were positively and linearly related to muscle mass change during regeneration, and third, Homer 3 was not detectable under resting conditions but was transiently expressed during regeneration although with a temporal pattern distinct from that of Homer 1a. Thus a switch in Homer isoforms is associated to muscle differentiation and regeneration. Homers may play a role not only in signal transduction of skeletal muscle, in particular regulation of Ca2+ release from sarcoplasmic reticulum (Ward CW, Feng W, Tu J, Pessah IN, Worley PF, and Schneider MF. Homer protein increases activation of Ca2+ sparks in permeabilized skeletal muscle. J Biol Chem 279: 5781–5787, 2004), but also in adaptation.

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8

Bortoloso, Elena, Aram Megighian, Sandra Furlan, Luisa Gorza, and Pompeo Volpe. "Homer 2 antagonizes protein degradation in slow-twitch skeletal muscles." American Journal of Physiology-Cell Physiology 304, no. 1 (January 1, 2013): C68—C77. http://dx.doi.org/10.1152/ajpcell.00108.2012.

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Homer represents a new and diversified family of proteins made up of several isoforms. The presence of Homer isoforms, referable to 1b/c and 2a/b, was investigated in fast- and slow-twitch skeletal muscles from both rat and mouse. Homer 1b/c was identical irrespective of the muscle, and Homer 2a/b was instead characteristic of the slow-twitch phenotype. Transition in Homer isoform composition was studied in two established experimental models of atrophy, i.e., denervation and disuse of slow-twitch skeletal muscles of the rat. No change of Homer 1b/c was observed up to 14 days after denervation, whereas Homer 2a/b was found to be significantly decreased at 7 and 14 days after denervation by 70 and 90%, respectively, and in parallel to reduction of muscle mass; 3 days after denervation, relative mRNA was reduced by 90% and remained low thereafter. Seven-day hindlimb suspension decreased Homer 2a/b protein by 70%. Reconstitution of Homer 2 complement by in vivo transfection of denervated soleus allowed partial rescue of the atrophic phenotype, as far as muscle mass, muscle fiber size, and ubiquitinazion are concerned. The counteracting effects of exogenous Homer 2 were mediated by downregulation of MuRF1, Atrogin, and Myogenin, i.e., all genes known to be upregulated at the onset of atrophy. On the other hand, slow-to-fast transition of denervated soleus, another landmark of denervation atrophy, was not rescued by Homer 2 replacement. The present data show that 1) downregulation of Homer 2 is an early event of atrophy, and 2) Homer 2 participates in the control of ubiquitinization and ensuing proteolysis via transcriptional downregulation of MuRF1, Atrogin, and Myogenin. Homers are key players of skeletal muscle plasticity, and Homer 2 is required for trophic homeostasis of slow-twitch skeletal muscles.

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9

Scully, Stephen, and Paolo Vivante. "Homer." Classical World 79, no. 6 (1986): 417. http://dx.doi.org/10.2307/4349952.

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10

Merialdo, Paolo, Paolo Atzeni, Marco Magnante, Giansalvatore Mecca, and Marco Pecorone. "HOMER." ACM SIGMOD Record 29, no. 2 (June 2000): 586. http://dx.doi.org/10.1145/335191.335497.

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11

Ganzarolli de Oliveira, Joao Vicente. "Homer." Annals of Language and Literature 3, no. 2 (2019): 1–3. http://dx.doi.org/10.22259/2637-5869.0302001.

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12

Blottner, Dieter, Gabor Trautmann, Sandra Furlan, Guido Gambara, Katharina Block, Martina Gutsmann, Lian-Wen Sun, et al. "Reciprocal Homer1a and Homer2 Isoform Expression Is a Key Mechanism for Muscle Soleus Atrophy in Spaceflown Mice." International Journal of Molecular Sciences 23, no. 1 (December 22, 2021): 75. http://dx.doi.org/10.3390/ijms23010075.

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The molecular mechanisms of skeletal muscle atrophy under extended periods of either disuse or microgravity are not yet fully understood. The transition of Homer isoforms may play a key role during neuromuscular junction (NMJ) imbalance/plasticity in space. Here, we investigated the expression pattern of Homer short and long isoforms by gene array, qPCR, biochemistry, and laser confocal microscopy in skeletal muscles from male C57Bl/N6 mice (n = 5) housed for 30 days in space (Bion-flight = BF) compared to muscles from Bion biosatellite on the ground-housed animals (Bion ground = BG) and from standard cage housed animals (Flight control = FC). A comparison study was carried out with muscles of rats subjected to hindlimb unloading (HU). Gene array and qPCR results showed an increase in Homer1a transcripts, the short dominant negative isoform, in soleus (SOL) muscle after 30 days in microgravity, whereas it was only transiently increased after four days of HU. Conversely, Homer2 long-form was downregulated in SOL muscle in both models. Homer immunofluorescence intensity analysis at the NMJ of BF and HU animals showed comparable outcomes in SOL but not in the extensor digitorum longus (EDL) muscle. Reduced Homer crosslinking at the NMJ consequent to increased Homer1a and/or reduced Homer2 may contribute to muscle-type specific atrophy resulting from microgravity and HU disuse suggesting mutual mechanisms.

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13

Pozdnev, Mikhail M. "Potemkin’s Homer." Philologia Classica 13, no. 2 (2018): 288–302. http://dx.doi.org/10.21638/11701/spbu20.2018.209.

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14

Dillon, John B., and Gordon Teskey. "Milton's Homer." PMLA 101, no. 5 (October 1986): 857. http://dx.doi.org/10.2307/462361.

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15

Madhu, R. Chandran. "Plato’s Homer." Ancient Philosophy 19 (1999): 87–95. http://dx.doi.org/10.5840/ancientphil199919special52.

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16

Kelsall, Malcolm. "Byronic Homer." Byron Journal 35, no. 1 (June 2007): 1–10. http://dx.doi.org/10.3828/bj.35.1.2.

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17

Perkins, Sid. "Odyssey's Homer." Science News 161, no. 23 (June 8, 2002): 355. http://dx.doi.org/10.2307/4013534.

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18

AUSTIN, NORMAN. "HOMER WHO?" Arion: A Journal of the Humanities and the Classics 19, no. 2 (2011): 121–53. http://dx.doi.org/10.1353/arn.2011.0039.

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19

Aftergood, Steven. "Homer II." Bulletin of the Atomic Scientists 57, no. 2 (March 1, 2001): 71–73. http://dx.doi.org/10.2968/057002021.

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20

Ranasinghe, Nalin. "Ransoming Homer." Philotheos 11 (2011): 14–21. http://dx.doi.org/10.5840/philotheos2011112.

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21

Scodel, R. "Review: Homer." Notes and Queries 51, no. 4 (December 1, 2004): 427–28. http://dx.doi.org/10.1093/nq/51.4.427.

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22

Scodel, Ruth. "Review: Homer." Notes and Queries 51, no. 4 (December 1, 2004): 427–28. http://dx.doi.org/10.1093/nq/510427.

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23

Finkelberg, Margalit. "Timēandaretēin Homer." Classical Quarterly 48, no. 1 (May 1998): 14–28. http://dx.doi.org/10.1017/s0009838800038751.

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Much effort has been invested by scholars in defining the specific character of the Homeric values as against those that obtained at later periods of Greek history. The distinction between the ‘shame-culture’ and the ‘guilt-culture’ introduced by E. R. Dodds, and that between the ‘competitive’ and the ‘cooperative’ values advocated by A. W. H. Adkins, are among the more influential ones. Although Adkins's taxonomy encountered some acute criticism, notably from A. A. Long, it has become generally adopted both in the scholarly literature and in general philosophical discussions of Greek ethics. Objections to Adkins's approach have mainly concentrated on demonstrating that his denial of the cooperative values to Homer is untenable on general grounds and is not supported by Homeric evidence. Characteristically, Adkins's thesis concerning the centrality to Homer's ethics of the so-called ‘competitive values‘ has never received similar attention, probably owing to the fact that this is the point at which his picture of the Homeric society concurs with the influential reconstructions by W Jaeger and M. I. Finley. The present study oftimēandaretē, generally held to be the two competitive values central to the Homeric poems, purports to address this issue.

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24

Hubbell, Larry. "Fred Homer." PS: Political Science & Politics 39, no. 02 (April 2006): 367–68. http://dx.doi.org/10.1017/s1049096506220584.

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25

Landais, C., M. Aletti, J. B. Soureau, J. M. Cournac, E. Poisnel, S. Sagnier, J. F. Paris, P. Carli, and S. Mouly. "Chauffe Homer !" La Revue de Médecine Interne 31, no. 2 (February 2010): 153–56. http://dx.doi.org/10.1016/j.revmed.2009.10.001.

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26

Jastrun, Mieczysław. "Reading Homer." New England Review 40, no. 2 (2019): 62. http://dx.doi.org/10.1353/ner.2019.0047.

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27

Tzetzes, John. "Before Homer." Aristei. Aristeas: vestnik klassicheskoi filologii i antichnoi istorii 24 (2021): 188–202. http://dx.doi.org/10.53084/22209050_2021_24_189.

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28

Jardin, Isaac, Letizia Albarrán, Nuria Bermejo, Ginés M. Salido, and Juan A. Rosado. "Homers regulate calcium entry and aggregation in human platelets: a role for Homers in the association between STIM1 and Orai1." Biochemical Journal 445, no. 1 (June 15, 2012): 29–38. http://dx.doi.org/10.1042/bj20120471.

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Homer is a family of cytoplasmic adaptor proteins that play different roles in cell function, including the regulation of G-protein-coupled receptors. These proteins contain an Ena (Enabled)/VASP (vasodilator-stimulated phosphoprotein) homology 1 domain that binds to the PPXXF sequence motif, which is present in different Ca2+-handling proteins such as IP3 (inositol 1,4,5-trisphosphate) receptors and TRPC (transient receptor potential canonical) channels. In the present study we show evidence for a role of Homer proteins in the STIM1 (stromal interaction molecule 1)–Orai1 association, as well as in the TRPC1–IP3RII (type II IP3 receptor) interaction, which might be of relevance in platelet function. Treatment of human platelets with thapsigargin or thrombin results in a Ca2+-independent association of Homer1 with TRPC1 and IP3RII. In addition, thapsigargin and thrombin enhanced the association of Homer1 with STIM1 and Orai1 in a Ca2+-dependent manner. Interference with Homer function by introduction of the synthetic PPKKFR peptide into cells, which emulates the proline-rich sequences of the PPXXF motif, reduced STIM1–Orai1 and TRPC1– IP3RII associations, as compared with the introduction of the inactive PPKKRR peptide. The PPKKFR peptide attenuates thrombin-evoked Ca2+ entry and the maintenance of thapsigargin-induced store-operated Ca2+ entry. Finally, the PPKKFR peptide attenuated thrombin-induced platelet aggregation. The findings of the present study support an important role for Homer proteins in thrombin-stimulated platelet function, which is likely to be mediated by the support of agonist-induced Ca2+ entry.

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29

Lumby, Catharine, and Kath Albury. "Homer versus Homer: Digital Media, Literacy and Child Protection." Media International Australia 128, no. 1 (August 2008): 80–87. http://dx.doi.org/10.1177/1329878x0812800110.

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Despite growing work on the educational potential of digital media, literacy debates in Australia have remained locked in a banal opposition between serious educational aims and trivial entertainment media. To reinvigorate these debates, this article overviews progressive approaches to media literacy and case studies debates around the sexualisation of girls and young women in popular media. Ultimately, the authors — drawing on their submission to the recent Senate Inquiry on the subject — identify two ways to reset the media education and literacy agenda by incorporating a more productive engagement with digital media literacy.

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30

Böhme, Robert. "Homer Alpha 1." Emerita 54, no. 2 (December 30, 1986): 203–35. http://dx.doi.org/10.3989/emerita.1986.v54.i2.647.

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31

Ulf, Christoph. "Homer’s World oder die Welt Homers? Kontextualisierungen zur „Homer Encyclopedia“." Historische Zeitschrift 295, no. 1 (September 2012): 105–17. http://dx.doi.org/10.1524/hzhz.2012.0354.

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32

Verzemnieks, Inara. "Homer Dill's Undead." Iowa Review 45, no. 2 (September 2015): 76–86. http://dx.doi.org/10.17077/0021-065x.7592.

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33

Çağrı Mutlu, Esra. "Homer, the Teacher." Beytulhikme An International Journal of Philosophy 8, no. 1 (July 21, 2018): 319–38. http://dx.doi.org/10.18491/beytulhikme.446509.

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34

Semêdo, Rafael de Almeida. "Rhetoric in Homer?" Nuntius Antiquus 16, no. 1 (July 31, 2020): 13–34. http://dx.doi.org/10.35699/1983-3636..21481.

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This article discusses the possibility of exploring the field of rhetoric within the Homeric poems. Is it adequate to employ the term “rhetoric” in discussions of Homeric poetry? We contend, following Knudsen (2014), that yes, the Iliad and the Odyssey provide us with the earliest instances of rhetorical activity in Antiquity. Firstly, we address why some scholars disregard that possibility, then argue why we disagree with them. Finally, we apply the elements of our theoretical discussion to an analysis of Odysseus’ supplication to Nausicaa in Odyssey 6, focusing on: a) the introduction by the Homeric narrator with the terms kerdíon, kerdaléos, and meilíkhios; and b) Odysseus’ strategic speaking when trying to convince Nausicaa to provide him with clothes and information about the way to town.

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35

Reyes, Alfonso. "Homer in Cuernavaca." Translation and Literature 9, no. 1 (March 2000): 91–106. http://dx.doi.org/10.3366/tal.2000.9.1.91.

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36

Reyes, Alfonso. "Homer in Cuernavaca." Translation and Literature 9, Part_1 (January 2000): 91–106. http://dx.doi.org/10.3366/tal.2000.9.part_1.91.

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37

Ferguson, GC. "Homer and Alzheimer." Journal of the Royal College of Physicians of Edinburgh 30, no. 4 (December 2000): 361. http://dx.doi.org/10.1177/147827150003000419.

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38

VAN COILLIE, GEERT. "HOMER ON COMPETITION." Bijdragen 71, no. 2 (January 2010): 115–31. http://dx.doi.org/10.2143/bij.71.2.2051600.

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39

Millinger, J. Ferguson, and Helen A. Cooper. "Winslow Homer Watercolors." New England Quarterly 60, no. 2 (June 1987): 305. http://dx.doi.org/10.2307/365619.

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40

Izdebski, Krzysztof. "Herbert Homer Dedo." Journal of Voice 36, no. 1 (January 2022): 1. http://dx.doi.org/10.1016/j.jvoice.2021.08.019.

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41

LOGAN, WILLIAM. "KEATS'S CHAPMAN'S HOMER." Yale Review 102, no. 2 (2014): 1–25. http://dx.doi.org/10.1353/tyr.2014.0041.

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42

Doherty, Lillian E., and Jasper Griffin. "Homer: The Odyssey." Classical World 83, no. 2 (1989): 124. http://dx.doi.org/10.2307/4350572.

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43

Farenga, Vincent, and Frank J. Nisetich. "Pindar and Homer." Classical World 84, no. 5 (1991): 400. http://dx.doi.org/10.2307/4350870.

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44

Pearcy, Lee T., Homer, and Robert Fagles. "Homer: The Iliad." Classical World 85, no. 1 (1991): 52. http://dx.doi.org/10.2307/4350988.

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45

Parameswaran, Uma, and Rabindranath Maharaj. "Homer in Flight." World Literature Today 73, no. 4 (1999): 752. http://dx.doi.org/10.2307/40155175.

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46

Lee, Robert E., Robert H. Young, and Benjamin Castleman. "James Homer Wright." American Journal of Surgical Pathology 26, no. 1 (January 2002): 88–96. http://dx.doi.org/10.1097/00000478-200201000-00011.

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47

Kozłowski, Jan M. "Homer a Eucharystia." Vox Patrum 57 (June 15, 2012): 351–57. http://dx.doi.org/10.31743/vp.4136.

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An excerpt from the fifth book of the Iliad, in which Homer explains why gods are immortal, sheds light upon the famous passage in Ignatius of Antioch’s Letter to the Ephesians that defines the Eucharistic bread as ‘the medicine of immortality’. By implying that consumption of bread and wine is the cause of human mortality, Homer enables us to notice the revolutionary character of Eucharistic meal as presented by Ignatius: in the Eucharist the Christian dynamic of approaching eternal life not through ecstatic denial of human nature, but rather through its affirmation, finds its fullest expression.

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48

Smock, Frederick. "On Reading Homer." Iowa Review 28, no. 2 (July 1998): 126–27. http://dx.doi.org/10.17077/0021-065x.5027.

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49

Segvic, †. Heda. "Homer in Plato'sProtagoras." Classical Philology 101, no. 3 (July 2006): 247–62. http://dx.doi.org/10.1086/511016.

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Zipser, Edward J., Greg Holland, and William Gray. "Robert Homer Simpson." Physics Today 68, no. 11 (November 2015): 62–63. http://dx.doi.org/10.1063/pt.3.2990.

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