Journal articles: 'Homeostatis'

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Goumenjuk, E. "Hormonal treatment and lipid homeostatis in premenopausal women." Pathophysiology 5 (June 1998): 46. http://dx.doi.org/10.1016/s0928-4680(98)80454-9.

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Lazovic, Biljana, Mirjana Zlatkovic-Svenda, Tijana Durmic, Zoran Stajic, Vesna Djuric, and Vladimir Zugic. "The regulation role of carotid body peripheral chemoreceptors in physiological and pathophysiological conditions." Medical review 69, no. 11-12 (2016): 385–90. http://dx.doi.org/10.2298/mpns1612385l.

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Introduction. The major oxygen sensors in the human body are peripheral chemoreceptors, also known as interoreceptors-as connected with internal organs, located in the aortic arch and in the body of the common carotid artery. Chemoreceptor function under physiological conditions. Stimulation of peripheral chemoreceptors during enviromental hypoxia causes a reflex-mediated increased ventilation, followed by the increase of the muscle sympatic activity, aiming to maintain tissue oxygen homeostatis, as well as glucosae homeostatis. Besides that, peripheral chemoreceptors interact with central chemoreceptors, responsible for carbon dioxide changes, and they are able to modulate each other. Chemoreceptor function in pathophysiological conditions. Investigations of respiratory function in many pathological processes, such as hypertension, obstructive sleep apnea, congestive heart failure and many other diseases that are presented with enhanced peripheral chemosensitivity and impaired functional sympatholysis ultimately determine the peripheral chemoreceptor role and significance of peripheral chemoreceptors in the process of those pathological conditions development. Considering this, the presumed influence of peripheral chemoreceptors is important in patients having the above mentioned pathology. Conclusion. The importance and the role of peripheral chemoreceptors in the course of the breathing control is still controversial, despite many scientific attempts to solve this problem. The main objective of this review is to give the latest data on the peripheral chemoreceptor role and to highlight the importance of peripheral chemoreceptors for maintaining of oxygen homeostasis in pateints with hypoxia caused by either physiological or pathological conditions.

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Meilana, Anna, Nurrani Mustika Dewi, and Andi Wijaya. "Heterogeneous Stem Cells in Skin Homeostatis and Wound Repair." Indonesian Biomedical Journal 7, no. 2 (August 1, 2015): 87. http://dx.doi.org/10.18585/inabj.v7i2.74.

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BACKGROUND: The skin protects mammals from insults, infection and dehydration and enables thermoregulation and sensory perception. Various skin-resident cells carry out these diverse functions. Constant turnover of cells and healing upon injury necessitate multiple reservoirs of stem cells. The skin is a complex organ harboring several distinct populations of stem cells and a rich array of cell types. Advances in genetic and imaging tools have brought new findings about the lineage relationships between skin stem cells and their progeny. Such knowledge may offer novel avenues for therapeutics and regenerative medicine.CONTENT: In the past years, our view of the mechanisms that govern skin homeostasis and regeneration have markedly changed. New populations of stem cells have been identified that behave spatio-temporally differently in healthy tissues and in situations of damage, indicating that a great level of stem cell heterogeneity is present in the skin. There are believed to be distinct populations of stem cells in different locations. The lineages that they feed are normally constrained by signals from their local environment, but they can give rise to all epidermal lineages in response to appropriate stimuli. Given the richness of structures such as blood vessels, subcutaneous fat, innervation and the accumulation of fibroblasts under the upper parts of the rete ridges (in the case of human skin), it is reasonable to speculate that the microenvironment might be essential for interfollicular epidermal homeostasis. The bloodstream is probably the main source of long-range signals reaching the skin, and cues provided by the vascular niche might be essential for skin homeostasis.SUMMARY: A key function of the interfollicular epidermis is to act as a protective interface between the body and the external environment, and it contains several architectural elements that enable it to fulfill this function. All elements of the epidermis play active roles in regulating skin function, which might not have been anticipated from their role in maintaining skin integrity. Skin cell research benefits from the integration of complementary technologies and disciplines. How skin function is regulated and how it may be possible to intervene to treat a variety of skin conditions. Ultimately also impairing the maintenance of self-renewing satellite cells. Therefore, only anti-aging strategies taking both factors, the stem cell niche and the stem cells per se, into consideration may ultimately be successful.KEYWORDS: epidermis, hair follicle, fibroblast, skin stem cells, homeostasis, regeneration

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Nef, Holger, Petra Kindle, Antje Technau, Christoph Bode, Jutta Schaper, and Albrecht Elsässer. "Alterations of calcium homeostatis induce contractile dysfunction in hibernating myocardium." Journal of Molecular and Cellular Cardiology 33, no. 6 (June 2001): A83. http://dx.doi.org/10.1016/s0022-2828(01)90331-1.

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Wu, Jian Kang, Zhipei Huang, Zhiqiang Zhang, Wendong Xiao, and Hong Jiang. "Quantitative Assessment of Autonomic Regulation of the Cardiac System." Journal of Healthcare Engineering 2019 (April 21, 2019): 1–8. http://dx.doi.org/10.1155/2019/4501502.

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Autonomic neural system (ANS) regulates the circulation to provide optimal perfusion of every organ in accordance with its metabolic needs, and the quantitative assessment of autonomic regulation is crucial for personalized medicine in cardiovascular diseases. In this paper, we propose the Dystatis to quantitatively evaluate autonomic regulation of the human cardiac system, based on homeostatis and probabilistic graphic model, where homeostatis explains ANS regulation while the probability graphic model systematically defines the regulation process for quantitative assessment. The indices and measurement methods for three well-designed scenarios are also illustrated to evaluate the proposed Dystatis: (1) heart rate variability (HRV), blood pressure variability (BPV), and respiration synchronization (Synch) in resting situation; (2) chronotropic competence indices (CCI) in graded exercise testing; and (3) baroreflex sensitivity (BRS), sympathetic nerve activity (SNA), and parasympathetic nerve activity (PNA) in orthostatic testing. The previous clinical results have shown that the proposed method and indices for autonomic cardiac system regulation have great potential in prediction, diagnosis, and rehabilitation of cardiovascular diseases, hypertension, and diabetes.

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Belkacemi, Louiza, Ghalem Selselet-Attou, Emeline Hupkens, Evrard Nguidjoe, Karim Louchami, Abdullah Sener, and Willy J. Malaisse. "Intermittent Fasting Modulation of the Diabetic Syndrome in Streptozotocin-Injected Rats." International Journal of Endocrinology 2012 (2012): 1–12. http://dx.doi.org/10.1155/2012/962012.

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This study investigates the effects of intermittent overnight fasting in streptozotocin-induced diabetic rats (STZ rats). Over 30 days, groups of 5-6 control or STZ rats were allowed free food access, starved overnight, or exposed to a restricted food supply comparable to that ingested by the intermittently fasting animals. Intermittent fasting improved glucose tolerance, increased plasma insulin, and lowered Homeostatis Model Assessment index. Caloric restriction failed to cause such beneficial effects. Theβ-cell mass, as well as individualβ-cell and islet area, was higher in intermittently fasting than in nonfasting STZ rats, whilst the percentage of apoptoticβ-cells appeared lower in the former than latter STZ rats. In the calorie-restricted STZ rats, comparable findings were restricted to individual islet area and percentage of apoptotic cells. Hence, it is proposed that intermittent fasting could represent a possible approach to prevent or minimize disturbances of glucose homeostasis in human subjects.

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Nie, Guangjun, Alex D. Sheftel, Sangwon F. Kim, and Prem Ponka. "Overexpression of Mitochondrial Ferritin Causes Cytosolic Iron Starvation and Changes Cellular Iron Homeostatis." Blood 104, no. 11 (November 16, 2004): 3195. http://dx.doi.org/10.1182/blood.v104.11.3195.3195.

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Abstract Cytosolic ferritin is well known to sequester and store iron and, consequently, protect cells against iron-mediated free radical damage. However, the function of the newly discovered mitochondrial ferritin (MtFt) is unknown. To examine the role of MtFt in cellular iron metabolism, we established a cell line that stably overexpresses the mouse MtFt under the control of tetracycline. The overexpression of MtFt caused a dose-dependent iron deficiency in the cytosol that was revealed by increased RNA-biding activity of iron regulatory proteins (IRPs) with a concomitant increase in transferrin receptor levels and a decrease in cytosolic ferritin. Consequently, the induction of MtFt resulted in a dramatic increase (2-fold) in cellular iron uptake from transferrin, most of which ended up in MtFt within mitochondria. Similarly, the induction of MtFt caused a shift of iron from cytosolic ferritin to MtFt. Moreover, the cell permeable iron chelator salicylaldehyde isonicotinoyl hydrazone (SIH) mobilized approximately twice as much iron from wild-type cells as compared to those overexpressing MtFt suggesting that iron in this protein is less accessible to SIH. Furthermore, the expression of MtFt was associated with a decrease in the activity of both mitochondrial and cytosolic aconitase activity, the latter being in agreement with the increase of IRP binding activity. In conclusion, our results indicate that the overexpression of MtFt causes a dramatic change in intracellular iron homeostasis and that the shunt of iron to mitochondrial storage protein likely limits its availability for functional iron proteins.

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Mounaji, Khadija, Nour-Eddine Erraiss, and Maurice Wegnez. "Identification of Metallothionein in Pleurodeles waltl." Zeitschrift für Naturforschung C 57, no. 7-8 (August 1, 2002): 727–31. http://dx.doi.org/10.1515/znc-2002-7-830.

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The characterization of metallothionein in the Urodele amphibian species Pleurodeles waltl was achieved. A simple and rapid method for identification of metallothionein, based on its strong affinity for cadmium (109Cd), was used. We were able to show that metallothionein is constitutively synthesized in liver, ovary and brain. The property of metallothionein to strongly bind essential (Zn, Cu) as well as toxic (Cd, Hg) metals is consistent with a dual role in cellular metabolism, i.e. homeostatis and detoxification of heavy metal ions.

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Ozyazici, Sefa, Faruk Karateke, Umit Turan, Adnan Kuvvetli, Huseyin Kilavuz, Burak Karakaya, Pınar Ozaltun, Murat Alısık, and Ozcan Erel. "A Novel Oxidative Stress Mediator in Acute Appendicitis: Thiol/Disulphide Homeostasis." Mediators of Inflammation 2016 (2016): 1–6. http://dx.doi.org/10.1155/2016/6761050.

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Aim. To investigate the role of a novel oxidative stress marker, thiol/disulphide homeostasis, in patients diagnosed with acute appendicitis (AA).Methods. In this study, seventy-one (43 male and 28 female) patients diagnosed with AA and 71 (30 male and 41 female) healthy volunteers were included. Age, gender, body mass index (BMI), haemoglobin (Hb), white blood cell (WBC), c-reactive protein (CRP), and thiol/disulphide homeostasis parameters (native thiol, total thiol, disulphide, disulphide/native thiol, native thiol/total thiol, and disulphide/total thiol ratios) were compared between the groups. Thiol/disulphide homeostasis was determined by a newly developed method by Erel and Neselioglu.Results. The native thiol, total thiol, and the native thiol/total thiol ratio levels were statistically significantly decreased in the AA compared with the control group (p<0.001). Disulphide level and the ratios of disulphide/native thiol and disulphide/total thiol were higher in the AA group than in the control group (p<0.001). There was a negative correlation of CRP with native thiol, total thiol, and native thiol/total thiol ratio while there was a positive correlation of CRP with disulphide/native thiol and disulphide/total thiol in the AA group. In the stepwise regression model, risk factors as disulphide/native thiol (OR = 1.368;p=0.018) and CRP (OR = 1.635;p=0.003) were determined as predictors of perforated appendicitis compared to the nonperforated group.Conclusion. This is the first study examining the thiol/disulphide homeostasis as a diagnostic aid in AA and establishing thiol/disulphide homeostatis balance shifted towards the disulphide formation due to thiol oxidation. Further studies are needed to optimize the use of this novel oxidative stress marker in AA.

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Oldner, Anders, Michel Goiny, Urban Ungerstedt, and Alf Sollevi. "SPLANCHNIC HOMEOSTATIS DURING ENDOTOXIN CHALLENGE IN THE PIG AS ASSESSED BY MICRODIALYSIS AND TONOMETRY." Shock 6, no. 3 (September 1996): 188–93. http://dx.doi.org/10.1097/00024382-199609000-00006.

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Oldner, Anders, Michel Goiny, Urban Ungerstedt, and Alf Sollevi. "SPLANCHNIC HOMEOSTATIS DURING ENDOTOXIN CHALLENGE IN THE PIG AS ASSESSED BY MICRODIALYSIS AND TONOMETRY." Shock 6 (September 1996): 188–93. http://dx.doi.org/10.1097/00024382-199609010-00006.

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Elly, Nur, Indaryani Indaryani, and Novi Lasmadasari. "Studi Prevalensi dan Faktor-Faktor yang Berpengaruh terhadap Status Defisiensi Vitamin D pada Ibu Hamil." Jurnal Kebidanan dan Keperawatan Aisyiyah 16, no. 2 (February 8, 2021): 206–16. http://dx.doi.org/10.31101/jkk.1902.

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Vitamin D berfungsi mengatur pemeliharaan homeostatis kalsium dan fosfor, mineralisasi tulang, regulasi imun, proliferasi sel dan mencegah gangguan penyakit. Kekurangan vitamin D pada ibu hamil menyebabkan bayi lahir pendek, lingkar kepala dan dada kecil serta berat badan lahir rendah, sedangkan bagi ibu dapat menyebabkan hipertensi, diabetes gestasional, osteomalasia dan osteoporosis. Penelitian ini bertujuan untuk melihat prevalensi dan faktoryang mempengaruhi status vitamin D pada ibu hamil.Metode penelitian menggunakan survey analitik dengan pendekatan crossectional. Hasil analisis statistik didapatkan bahwa p value paparan sinar matahari adalah 0,041 dan penambahan berat badan adalah 0,032.

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Kawano, Miyuki, Kazuei Igarashi, and Yoshimi Kakinuma. "The Na+-Responsive ntpOperon Is Indispensable for Homeostatis of K+ and Na+ in Enterococcus hirae at Limited Proton Potential." Journal of Bacteriology 180, no. 18 (September 15, 1998): 4942–45. http://dx.doi.org/10.1128/jb.180.18.4942-4945.1998.

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ABSTRACT Enterococcus hirae ATCC 9790 grew well in Na+-deficient, low-K+ medium, but growth was inhibited by carbonylcyanide m-chlorophenylhydrazone (CCCP). Growth inhibition and decrease of cellular K+levels in the presence of CCCP were relieved by the addition of Na+ and a high concentration of K+. In contrast, in the mutant defective in Na+-ATPase or the NtpJ component of the KtrII K+ uptake system, CCCP-induced growth inhibition was rescued by a high concentration of K+but not of Na+. These transporters are thus indispensable for homeostatis of K+ and Na+ at low proton potential.

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Zhang, Fenghua, Suping Xing, and Zongpeng Li. "Antagonistic effects of lycopene on cadmium-induced hippocampal dysfunctions in autophagy, calcium homeostatis and redox." Oncotarget 8, no. 27 (May 29, 2017): 44720–31. http://dx.doi.org/10.18632/oncotarget.18249.

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Suzić, Slavica, Ljubinka Radunović, Violeta Janković, and Radmila S̆egović. "Effects of protein-free diet in amino acid homeostatis of rat blood plasma and gut contents." FEBS Letters 216, no. 2 (June 1, 1987): 287–90. http://dx.doi.org/10.1016/0014-5793(87)80707-x.

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Darwis, Darlan. "Kegawatan Demam Berdarah Dengue pada Anak." Sari Pediatri 4, no. 4 (December 6, 2016): 156. http://dx.doi.org/10.14238/sp4.4.2003.156-62.

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Kegawatan demam berdarah dengue (DBD) yang mengancam nyawa adalah disfungsisirkulasi atau syok hipovolemik yang disebabkan oleh peningkatan permeabilitas kapilardan perdarahan, sehingga terjadi plasma leakage, penurunan perfusi organ, penurunansuplai oksigen dan nutrien untuk sel yang dapat berlanjut dengan gagal organ multipledan kematian. Tata laksana kegawatan DBD berorientasi pada pendekatan patofisiologikmulti system terpadu yang diarahkan pada pemenuhan kebutuhan oksigen dan nutrien,termasuk didalamnya tunjangan ventilasi, pemberian oksigen, resusitasi cairan dan obatresusitasi. Resusitasi cairan paling baik diberikan saat syok kompensasi, dengan pemberiancairan kristaloid atau koloid secara agresif 10-30 ml/kgbb dalam 6-10 menit untukmeningkatkan preload, curah jantung, volume sirkulasi efektif, memperbaiki perfusiorgan, sehingga mekanisme homeostatis atau mekanisme kompensasi tidak digunakanlagi dan kesembuhan segera pasien sindrom syok dengue dapat diharapkan.

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Wang, Lin-Shu, and Peizheng Ma. "The homeostasis solution – Mechanical homeostasis in architecturally homeostatic buildings." Applied Energy 162 (January 2016): 183–96. http://dx.doi.org/10.1016/j.apenergy.2015.10.058.

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van Blitterswijk, Wim J., Jan Damen, Henk Hilkmann, and John de Widt. "Alterations in biosynthesis and homeostatis of cholesterol and in lipoprotein patterns in mice bearing a transplanted lymphoid tumor." Biochimica et Biophysica Acta (BBA) - Biomembranes 816, no. 1 (June 1985): 46–56. http://dx.doi.org/10.1016/0005-2736(85)90391-8.

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Liu, Yuanbin. "Understanding the biological activity of amyloid proteins in vitro: From inhibited cellular mtt reduction to altered cellular cholesterol homeostatis." Progress in Neuro-Psychopharmacology and Biological Psychiatry 23, no. 3 (April 1999): 377–95. http://dx.doi.org/10.1016/s0278-5846(99)00003-2.

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Ferenčík, Miroslav, Viera tvrtinová, and Ivan Hulín. "Defects in Regulation of Local Immune Responses Resulting in Atherosclerosis." Clinical and Developmental Immunology 12, no. 3 (2005): 225–34. http://dx.doi.org/10.1080/17402520500182295.

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Atherosclerosis is nowadays generally accepted as an inflammatory disease but the mechanism of its origin and development have not yet been fully clarified. The present review focuses on the role of the local immune system as one of the key players in the pathogenesis of the complex process. Its part represented by vascular-associated lymphoid tissue (VALT) within the arterial wall participates directly in the vascular wall's homeostatis. Its inordinate activation during ontogenic development of an individual, this formerly defensive and physiologic mechanism transform into a pathological process resulting in an impairing inflammation. Hsp60, CRP and oxidized or otherwise modified LDL are serious candidates for triggering these pathological changes. The principal role is played by anti-Hsp60 antibodies and by shear stress originating on the surface of endothelium due to blood flow. The experimental and clinical data supporting this immunological hypothesis of atherosclerosis are discussed.

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Taqwiem, Ahsani. "KELISANAN DALAM NOVEL PEREMPUAN HUJAN KARYA HAMAMI ADABY (ORALITY IN PEREMPUAN HUJAN NOVEL BY HAMAMI ADABY)." JURNAL BAHASA, SASTRA DAN PEMBELAJARANNYA 6, no. 2 (July 19, 2017): 275. http://dx.doi.org/10.20527/jbsp.v6i2.3758.

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Kelisanan dalam Novel Perempuan Hujan karya Hamami Adaby. Perkembangan kebudayaantidak bisa melepaskan diri sepenuhnya dari kebudayaan sebelumnya, maka dapat dipahami bahwatradisi tulis tidak serta-merta menghapuskan unsur-unsur tradisi lisan yang mendahuluinya. Sastraadalah salah satu unsur di dalam kebudayaan manusia, oleh karena itu perkembangan sastra selalusejalan dengan perkembangan kebudayaan manusia.Secara objektif, penelitian ini bertujuan untukmendeskripsikan tentang wujud kelisanan di dalam novel Perempuan Hujan karya Hamami Adaby.Penelitian ini merupakan penelitian kualitatif dengan menggunakan pendekatan strukturalismedinamik.Di dalam penelitian ini sumber data yang digunakan adalah sumber data primer.Data-datayang digunakan diambil langsung dari novel Perempuan Hujan karya Hamami Adaby.Sedangkaninstrumen utama adalah peneliti sendiri.Hasil penelitian menunjukkan bahwa berdasarkan data-datayang berhasil dikumpulkan secara umum maka dapat disimpulkan bahwa di dalam novel PerempuanHujan masih terdapat pengaruh kelisanan yang dapat ditelurusi.Dengan analisis yang cermat,ditemukan enam unsur-unsur kelisanan pada novel Perempuan Hujan. Unsur-unsur itu antara lainadalah bersifat homeostatis serta adanya pemikiran yang kontekstual; menggunakan gaya penambahdan kopius; adanya penyajian tokoh datar dan ajaran moral.Kata-kata kunci: novel, karakteristik kelisanan, wujud kelisanan

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Meiliana, Anna, and Andi Wijaya. "Gut Hormones and Energy Balance, The Future for Obesity Therapy?" Indonesian Biomedical Journal 1, no. 3 (December 1, 2009): 33. http://dx.doi.org/10.18585/inabj.v1i3.99.

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BACKGROUND: The prevalence of obesity is increasing in both developed and developing countries along with associated diseases such as type 2 diabetes and coronary heart disease. The recent discovery of a number of gut hormones that play a role in appetite regulation and are released or suppressed in response to a meal may offer new targets for the treatments of obesity.CONTENT: In addition to the obvious role of the gut in the digestion and absorption of nutrient, the intestine and associated visceral organs, including the pancreas, liver, and visceral adipose depots, have important sensing and signaling roles in the regulation of energy homeostatis. Signals reflecting energy stores, recent nutritional state, and other parameters are integrated in the central nervous system, particularly in the hypotalamus, to coordinate energy intake and expenditure.SUMMARY: Our understanding of the role of the gut in energy balance and insights into gut-derived signals will stimulate previously unexplored therapeutics for obesity and other disorders of energy balance.KEYWORDS: obesity, energy, balance, gut hormones, satiation, satiety

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Soep, Soep. "LATIHAN FISIK PENDERITA DM TERHADAP PENURUNAN KONSENTRASI GLUKOSA DARAH, LDL DAN PENINGKATAN HDL." Jurnal Ilmiah PANNMED (Pharmacist, Analyst, Nurse, Nutrition, Midwivery, Environment, Dentist) 16, no. 1 (May 7, 2021): 51–57. http://dx.doi.org/10.36911/pannmed.v16i1.997.

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Latar belakang; Diabetes melitus merupakan sindrom metabolisme yang dikarakteristikkan dengan kehilangan homeostatis glukosa sehingga menyebabkan kerusakan pada metabolisme glukosa, kolesterol, LDL dan HDL. Latihan fisik merupakan salah satu pilar penatalaksanaan DM. Tujuan dari penelitian ini yaitu untuk mengetahui pengaruh latihan fisik terhadap kadar glukosa, kolesterol total, LDL dan HDL. Metode Penelitian; Penelitian ini merupakan Kuasi-Eksperimental dengan rancangan pre-post-test group design. Jumlah sampel dalam penelitian ini yaitu 47 orang. Latihan fisik dilaksanakan selama 4 minggu,5 kali/minggu dengan durasi 30 menit. Hasil Penelitian; Hasil penelitian menunjukkan bahwa ada pengaruh latihan fisik pada penderita DM terhadap penurunan glukosa darah, LDL dan peningkatan HDL di Wilayah Kerja Puskesmas Medan Helvetia dengan nilai p<0,05. Penelitian ini dapat dijadikan masukan bagi dalam pengambilan kebijakan dan peningkatan mutu pelayanan petugas kesehatan dalam menyelenggarakan kegiatan olah raga bagi penderita diabetes mellitus sebagai bagian dari penatalaksanaan diabetes mellitus dalam mengontrol gula darah dan profil lipid.

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Wilde, Gerald J. S. "Beyond the concept of risk homeostatis: Suggestions for research and application towards the prevention of accidents and lifestyle-related disease." Accident Analysis & Prevention 18, no. 5 (October 1986): 377–401. http://dx.doi.org/10.1016/0001-4575(86)90012-6.

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Zakharov, V. M., and I. E. Trofimov. "Homeostatic mechanisms of biological systems: Development homeostasis." Russian Journal of Developmental Biology 45, no. 3 (May 2014): 105–16. http://dx.doi.org/10.1134/s1062360414030096.

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Datu, Olvie, and Fransisco P. Sumalong. "EFEK PEMBERIAN ALPHA LIPOIC ACID PADA ENDOTEL TIKUS PUTIH YANG DIINDUKSI LIPOPOLISAKARIDA." PHARMACON 9, no. 1 (February 28, 2020): 125. http://dx.doi.org/10.35799/pha.9.2020.27418.

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ABSTRACT Endothelial dysfunction play a major role in the pathogenesis of vascular diseases, especially atherosclerosis. Endothelial dysfunction causes endothelial cells are activated and resulting homeostasis changes in endothelial cell. Rats were induced by LPS 0.5 mg/kg and treated with ALA at doses of 30 mg/kg, 60 mg/kg and 120 mg/kg BW for 2 weeks which given 1 hour before the LPS administration. This study show that LPS causes morphological changes in rat aorta based on histochemical staining show that endothelial cell are irregular, not homogeneous and increase permeability but in group that received show that ALA can improve the morphological of endothelial cell by reduces ROS, regenerates of exogenous and endogenous antioxidants. Administration of ALA and LPS reduce the rats body weight but not significant compared to the control group. Therefore can be concluded that administration of ALA can prevent endothelial dysfunction. Keywords: Endothel dysfunction, alpha lipoic acid, lipopolysaccharide ABSTRAK Disfungsi endotel berperan penting pada pathogenesis penyakit vascular, khususnya aterosklerosis. Disfungsi endotel menyebabkan sel endotel teraktivasi dan terjadi perubahan homeostatis pada sel endotel. Tikus diinduksi dengan LPS 0.5 mg/Kg BB dan selanjutnya di terapi dengan ALA pada dosis 30mg/Kg BB, 60mg/Kg BB, dan 120mg/Kg BB selama 2 minggu 1 jam sebelum pemerian LPS. Penelitian ini menujukan bahwa LPS dapat menyebabkan perubahan morfologi pada sel nedotel berdasakan pada hasil pewarnaan jaringan yang menunjukan sel endotel yang tersusun tidak beraturan, tidak homogeny dan terjadi peningkatan permeabilitas tapi pada kelompok perlakuan yang mendapatkan terapi ALA menunjukan ALA dapat memperbaiki morfologi sel endotel dengan mengurangi ROS, meningkatkan antioksidan eksogen dan endogen. Pemberian ALA dan LPS tidak menyebabkan perubahan berat badan tikus yang signifikan di bandingkan dengan kelompok control. Sehingga dapat disimpulkan pemberian ALA dapat mencegah disfungsi endotel. Kata Kunci: disfungsi endotel, histokimia, alpha lipoic acid, lipopolisakarida

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Krizanac-Bengez, Ljiljana, Marc R. Mayberg, and Damir Janigro. "The cerebral vasculature as a therapeutic target for neurological disorders and the role of shear stress in vascular homeostatis and pathophysiology." Neurological Research 26, no. 8 (December 2004): 846–53. http://dx.doi.org/10.1179/016164104x3789.

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Thwaites, C. J., N. B. Baillie, and W. Kasa. "Effects of dehydration on the heat tolerance of male and female New Zealand White rabbits." Journal of Agricultural Science 115, no. 3 (December 1990): 437–40. http://dx.doi.org/10.1017/s0021859600075894.

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SUMMARYMale and female New Zealand White rabbits were exposed for 3h to 34 °C and 36 °C (both at 40% r.h.) when hydrated and dehydrated. Females had lower rectal and skin temperatures and respiratory rates than males (P < 0·001). Differences between the sexes in rectal temperature were greater at 36 °C than at 34 °C. Withholding water for 24h significantly increased the responses in rectal temperature; the differentials between hydrated and dehydrated males and females being 0·3 °C and 0·2 °C, respectively. In contrast, respiratory rates were lower in dehydrated than in hydrated rabbits, suggesting that the former were attempting water homeostatis at the expense of thermoregulation.The results suggest that the performance of rabbits in the tropics is likely to be maximized when drinking water is available at all times, and that of males, particularly breeding bucks, might be improved simply by housing them in the coolest available location. Significant individual differences in observed responses point to the need for genetic studies of heat tolerance and the possibility of developing better adapted genotypes.

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Kang, Bit Na, Deepak A. Deshpande, K. G. Tirumurugaan, Reynold A. Panettieri, Timothy F. Walseth, and Mathur S. Kannan. "Adenoviral mediated anti-sense CD38 attenuates TNF-α-induced changes in calcium homeostasis of human airway smooth muscle cells." Canadian Journal of Physiology and Pharmacology 83, no. 8-9 (August 1, 2005): 799–804. http://dx.doi.org/10.1139/y05-081.

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CD38 is a membrane-bound protein involved in the synthesis and degradation of cyclic-ADP-ribose (cADPR). cADPR mobilizes calcium from intracellular stores in airway smooth muscle cells. To determine the role of CD38/cADPR signaling in calcium regulation in human airway smooth muscle (HASM) cells, we downregulated CD38 expression using a recombinant replication-defective adenovirus with anti-sense human CD38 (Ad-asCD38). CD38 expression was determined by RT-PCR and real-time quantitative PCR, and ADP-ribosyl cyclase (cyclase) activity was determined by competitive binding assay. In HASM cells infected with Ad-asCD38, TNF-α-induced, augmented-CD38 expression and cyclase activity were significantly lower than in TNF-α-treated cells. The net intracellular calcium responses to 10 nmol/L bradykinin were measured in HASM cells by fluorescence imaging. In cells infected with Ad-asCD38 in the presence of TNF-α, the net intracellular Ca2+ responses were significantly lower than in cells treated with TNF-α in the presence of the control vector (p < 0.001). These results provide evidence for the feasibility of using adenoviral vectors for gene transfer to down regulate gene expression, and confirm the role of CD38 in calcium homeostatis in ASM cells.Key words: cyclic ADP-Ribose, cytokine, smooth muscle.

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Westerterp-Plantenga, Margriet S., Manuela P. G. M. Lejeune, Astrid J. P. G. Smeets, and Natalie D. Luscombe-Marsh. "Sex differences in energy homeostatis following a diet relatively high in protein exchanged with carbohydrate, assessed in a respiration chamber in humans." Physiology & Behavior 97, no. 3-4 (June 2009): 414–19. http://dx.doi.org/10.1016/j.physbeh.2009.03.010.

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Döker, Serhat, Sandra Mounicou, Mehmet Doğan, and Ryszard Lobinski. "Probing the metal-homeostatis effects of the administration of chromium(vi) to mice by ICP MS and size-exclusion chromatography-ICP MS." Metallomics 2, no. 8 (2010): 549. http://dx.doi.org/10.1039/c004508j.

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Mäkinen, Susanna, Thomas van Groen, Jared Clarke, Anders Thornell, Dale Corbett, Mikko Hiltunen, Hilkka Soininen, and Jukka Jolkkonen. "Coaccumulation of Calcium and β-Amyloid in the Thalamus after Transient Middle Cerebral Artery Occlusion in Rats." Journal of Cerebral Blood Flow & Metabolism 28, no. 2 (July 25, 2007): 263–68. http://dx.doi.org/10.1038/sj.jcbfm.9600529.

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Transient occlusion of the middle cerebral artery (MCAO) in rats leads to abnormal accumulation of β-amyloid (Aβ) peptides in the thalamus. This study investigated the chemical composition of these deposits. Adult male human β-amyloid precursor protein (APP) overexpressing ( hAPP695) rats and their wild-type littermates were subjected to transient MCAO for 2 h or sham operation. After 26-week survival time, histological examination revealed an overlapping distribution pattern for rodent and human Aβ in the thalamus of hAPP695 rats subjected to MCAO. X-ray microanalysis showed that the deposits did not contain significant amount of iron, zinc, or copper typical to senile plaques. In contrast, the deposit both in hAPP695 and non-transgenic rats contained calcium and phosphorus in a ratio (1.28 ± 0.15) characteristic to hydroxyapatites. Alizarin red staining confirmed that calcium coaccumulated in these Aβ deposits. It is suggested that APP expression is induced by ischemic insult in cortical neurons adjacent to infarct, which in turn is reflected as increased release of Aβ peptides by their corticothalamic axon endings. This together with insufficient clearance or atypical degradation of Aβ peptides lead to dysregulation of calcium homeostatis and coaccumulation in the thalamus.

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Alam, MZ, L. Regioneiri, and MAS Santos. "Determination of amino acids misincorporation in recombinant protein by mass spectrometry." Bangladesh Journal of Animal Science 42, no. 1 (July 22, 2013): 11–19. http://dx.doi.org/10.3329/bjas.v42i1.15760.

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The synthesis of protein according to genetic code of a gene determines the basis of life and a stable proteome is necessary for cell homeostatis. However, errors occur naturally during translation of protein from its mRNA, which varies from 10-3 to 10-4 per codon. These errors are more frequent in recombinant protein overexpressed in heterologous hosts and affect protein functionality. The increasing amount of nonfunctional protein is often related to mistranslation of a gene under stress. In the present study, Saccharomyces cerevisiae as a host organism to overexpress E. coli lacZ gene fusion with GST to quantify misincorporation of amino acid in GST-? galactosidase recombinant protein. The yeast was treated with various stressors such as ethanol, chromium (CrO3), and aminoglycoside antibiotic - geneticin (G418) to induce protein aggregation. The misincorporation of amino acids was studied in soluble protein fractions by mass-spectrometry to determine how much misincorporation occur. We found that under experimental stress conditions the misincorporation of amino acids ranges from 5.6 ×10-3 to 8 × 10-3, which represents 60-80 fold higher than reported level. DOI: http://dx.doi.org/10.3329/bjas.v42i1.15760 Bang. J. Anim. Sci. 2013. 42 (1): 11-19

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Benhaiem, Sarah, Heribert Hofer, Martin Dehnhard, Janine Helms, and Marion L. East. "Sibling competition and hunger increase allostatic load in spotted hyaenas." Biology Letters 9, no. 3 (June 23, 2013): 20130040. http://dx.doi.org/10.1098/rsbl.2013.0040.

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Allostatis is the process of maintaining homeostatis through behavioural or physiological responses to challenges, and its cumulative energetic cost is termed allostatic load. The allostatic load hypothesis predicts that hunger and the mechanisms that establish and maintain social dominance should have a strong impact on allostatic load. In spotted hyaenas, dominance between twin siblings emerges during intense early competition for maternal milk and involves trained winner/loser effects . Conflict over access to teats declines with age as behavioural dominance conventions are established. In young litters, the allostatic load of subordinates measured in terms of faecal glucocorticoid metabolite concentrations (fGMCs) should be higher than that of dominants. When low milk provisioning threatens survival, hungry subordinates are more assertive, particularly when competing against a dominant sister. Dominants challenged by assertive subordinates should have allostatic loads and fGMCs above those of dominants with subordinates that adhere to dominance conventions. We show that in young litters, subordinates had significantly higher fGMCs than dominants, and dominant sisters had significantly higher fGMCs than dominant brothers. When hungry, both dominants and subordinates had significantly higher fGMCs than when fed. Our results provide evidence that hunger and sibling competition affect allostatic load in spotted hyaenas.

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Faizan, Shahla, Saima Kausar, and Neelima Akhtar. "Influence of Wastewater Application and Fertilizer use on Growth, Photosynthesis, Nutrient Homeostatis, Yield and Heavy Metal Accumulation in Okra (Abelmoschus esculentus L. Moench)." Pakistan Journal of Biological Sciences 17, no. 5 (April 15, 2014): 630–40. http://dx.doi.org/10.3923/pjbs.2014.630.640.

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Rivas-Crespo, M. Francisco. "Comment on Boyko and Jensen. Do We Know What Homeostatis Model Assessment Measures? If Not, Does It Matter? Diabetes Care 2007;30:2725–2728." Diabetes Care 38, no. 12 (November 24, 2015): e213-e213. http://dx.doi.org/10.2337/dc15-1172.

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Gainey, Melanie A., and Daniel E. Feldman. "Multiple shared mechanisms for homeostatic plasticity in rodent somatosensory and visual cortex." Philosophical Transactions of the Royal Society B: Biological Sciences 372, no. 1715 (March 5, 2017): 20160157. http://dx.doi.org/10.1098/rstb.2016.0157.

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We compare the circuit and cellular mechanisms for homeostatic plasticity that have been discovered in rodent somatosensory (S1) and visual (V1) cortex. Both areas use similar mechanisms to restore mean firing rate after sensory deprivation. Two time scales of homeostasis are evident, with distinct mechanisms. Slow homeostasis occurs over several days, and is mediated by homeostatic synaptic scaling in excitatory networks and, in some cases, homeostatic adjustment of pyramidal cell intrinsic excitability. Fast homeostasis occurs within less than 1 day, and is mediated by rapid disinhibition, implemented by activity-dependent plasticity in parvalbumin interneuron circuits. These processes interact with Hebbian synaptic plasticity to maintain cortical firing rates during learned adjustments in sensory representations. This article is part of the themed issue ‘Integrating Hebbian and homeostatic plasticity’.

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Clancy, John, and Andrew McVicar. "Homeostasis 2: Nurses as external agents of homeostatic control." British Journal of Nursing 20, no. 4 (February 23, 2011): 232–38. http://dx.doi.org/10.12968/bjon.2011.20.4.232.

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Meizlish, Matthew L., Ruth A. Franklin, Xu Zhou, and Ruslan Medzhitov. "Tissue Homeostasis and Inflammation." Annual Review of Immunology 39, no. 1 (April 26, 2021): 557–81. http://dx.doi.org/10.1146/annurev-immunol-061020-053734.

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There is a growing interest in understanding tissue organization, homeostasis, and inflammation. However, despite an abundance of data, the organizing principles of tissue biology remain poorly defined. Here, we present a perspective on tissue organization based on the relationships between cell types and the functions that they perform. We provide a formal definition of tissue homeostasis as a collection of circuits that regulate specific variables within the tissue environment, and we describe how the functional organization of tissues allows for the maintenance of both tissue and systemic homeostasis. This leads to a natural definition of inflammation as a response to deviations from homeostasis that cannot be reversed by homeostatic mechanisms alone. We describe how inflammatory signals act on the same cellular functions involved in normal tissue organization and homeostasis in order to coordinate emergency responses to perturbations and ultimately return the system to a homeostatic state. Finally, we consider the hierarchy of homeostatic and inflammatory circuits and the implications for the development of inflammatory diseases.

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Sumner, T., J. Hetherington, R. M. Seymour, L. Li, M. Varela Rey, S. Yamaji, P. Saffrey, et al. "A composite computational model of liver glucose homeostasis. II. Exploring system behaviour." Journal of The Royal Society Interface 9, no. 69 (February 8, 2012): 701–6. http://dx.doi.org/10.1098/rsif.2011.0783.

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Using a composite model of the glucose homeostasis system, consisting of seven interconnected submodels, we enumerate the possible behaviours of the model in response to variation of liver insulin sensitivity and dietary glucose variability. The model can reproduce published experimental manipulations of the glucose homeostasis system and clearly illustrates several important properties of glucose homeostasis—boundedness in model parameters of the region of efficient homeostasis, existence of an insulin sensitivity that allows effective homeostatic control and the importance of transient and oscillatory behaviour in characterizing homeostatic failure. Bifurcation analysis shows that the appearance of a stable limit cycle can be identified.

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Pérez-Torres, Israel, Blanca Ibarra, Elizabeth Soria-Castro, Rocío Torrico-Lavayen, Natalia Pavón, Eulises Diaz-Diaz, Pedro L. Flores, Oscar Infante, and Guadalupe Baños. "Effect of glycine on the cyclooxygenase pathway of the kidney arachidonic acid metabolism in a rat model of metabolic syndrome." Canadian Journal of Physiology and Pharmacology 89, no. 12 (December 2011): 899–910. http://dx.doi.org/10.1139/y11-086.

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The kidneys are organs that can be severely impaired by metabolic syndrome (MS). This is characterized by the association of various pathologies such as hypertension, dyslipidemia, and type-2 diabetes. Glycine, a nonessential amino acid, is known to possess various protective effects in the kidney, such as a decrease in the deterioration of renal function and a reduction of the damage caused by hypoxia. In a rat model of MS, the effect of glycine on the cyclooxygenase (COX) pathway of arachidonic acid (AA) metabolism was studied in isolated perfused kidney. MS was induced in Wistar rats by feeding them a 30% sucrose solution for 16 weeks. The addition of 1% glycine to their drinking water containing 30% sucrose, for 8 weeks, reduced high blood pressure, triglyceride levels, insulin concentration, homeostatis model assessment (HOMA) index, albuminuria, AA concentration in kidney homogenate, renal perfusion pressure, prostaglandin levels, PLA2expression, and COX isoform expression, compared with MS rats that did not receive the glycine supplement. Glycine receptor expression decreased significantly with MS, but glycine treatment increased it. The results suggest that in the MS model, 1% glycine treatment protects the kidney from damage provoked by the high sucrose consumption, by acting as an anti-inflammatory on the COX pathway of AA metabolism in kidney.

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Sakamuri, Anil, Sujatha Pitla, Uday Kumar Putcha, Sugeedha Jayapal, Sailaja Pothana, Sai Santosh Vadakattu, Nagabhushan Reddy Konapalli, Siva Sankara Vara Prasad Sakamuri, and Ahamed Ibrahim. "Transient Decrease in Circulatory Testosterone and Homocysteine Precedes the Development of Metabolic Syndrome Features in Fructose-Fed Sprague Dawley Rats." Journal of Nutrition and Metabolism 2016 (2016): 1–11. http://dx.doi.org/10.1155/2016/7510840.

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Background. Increased fructose consumption is linked to the development of metabolic syndrome (MS). Here we investigated the time course of development of MS features in high-fructose-fed Sprague Dawley rats along with circulatory testosterone and homocysteine levels.Methods. Rats were divided into control and experimental groups and fed with diets containing 54.5% starch and fructose, respectively, for 4, 12, and 24 weeks. Plasma testosterone and homocysteine levels were measured along with insulin, glucose, and lipids. Body composition, insulin resistance, and hepatic lipids were measured.Results. Increase in hepatic triglyceride content was first observed in metabolic disturbance followed by hypertriglyceridemia and systemic insulin resistance in fructose-fed rats. Hepatic lipids were increased in time-dependent manner by fructose-feeding starting from 4 weeks, but circulatory triglyceride levels were increased after 12 weeks. Fasting insulin and Homeostatis Model Assessment of Insulin Resistance (HOMA-IR) were increased after 12 weeks of fructose-feeding. Decreased visceral adiposity, circulatory testosterone, and homocysteine levels were observed after 4 weeks of fructose-feeding, which were normalized at 12 and 24 weeks.Conclusions. We conclude that transient decrease in circulatory testosterone and homocysteine levels and increased hepatic triglyceride content are the earliest metabolic disturbances that preceded hypertriglyceridemia and insulin resistance in fructose-fed SD rats.

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Shi, Xinyan, Duo Peng, Yanfei Liu, Xiaofen Miao, Hui Ye, and Jun Zhang. "Advantages of Serum Anti-Müllerian Hormone as a Marker for Polycystic Ovarian Syndrome." Laboratory Medicine 50, no. 3 (December 10, 2018): 236–42. http://dx.doi.org/10.1093/labmed/lmy068.

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Abstract Objective To evaluate the advantages of using anti-Müllerian hormone (AMH) in the diagnosis of polycystic ovary syndrome (PCOS). Methods We compared data from 56 Han Chinese patients with PCOS and 52 female control Han Chinese individuals in terms of biochemical markers and the antral follicle count (AFC). Then, the correlation between serum AMH levels and various endocrine and metabolic factors was analyzed. We generated receiver operating characteristic (ROC) curves to assess the usefulness of AMH, AFC, the luteinizing hormone/follicle-stimulating hormone (LH/FSH) ratio, and testosterone (T), to discriminate between the PCOS and control groups. Results The LH/FSH ratio, homeostatis model assessment of insulin resistance (HOMA-IR), testosterone (T), blood glucose (Glu), triglyceride (TG), body mass index (BMI), AFC and, in particular, serum AMH level were significantly higher in the PCOS group than in the control group (P <.05; P <.001). The serum AMH level had a significant positive effect on the likelihood of AFC changes in patients with PCOS and control-group women. The areas under the ROC curve (AUC) of AMH, AFC, LH/FSH ratio, and T were 0.952, 0.778, 0.760, and 0.665, respectively. The best cutoff point for AMH was 6.09 ng per mL, with sensitivity of 88% and specificity of 88%.

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Curtsinger, James W. "Reproductive Homeostasis and Senescence in Drosophila melanogaster." Journals of Gerontology: Series A 74, no. 10 (December 6, 2018): 1533–38. http://dx.doi.org/10.1093/gerona/gly274.

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Abstract The homeostatic properties of reproduction in aging female Drosophila melanogaster are investigated. Classic studies based on cohort analysis suggest that homeostatic capacity declines gradually as daily oviposition rates decline in aging flies. Analysis at the level of individuals gives a very different picture: reproductive homeostasis remains relatively constant for most of adult life until a critical point when oviposition either ceases entirely or continues in dysregulated fashion. The collapse of homeostatic capacity is abrupt. Enhanced homeostasis is associated with increased lifetime fecundity and improved prospects for survival. The fractal concept of lacunarity can be used to parameterize the “roughness” of individual fecundity trajectories and is inversely related to homeostatic capacity.

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Boyko, Edward J. "Response to Comment on Boyko and Jensen. Do We Know What Homeostatis Model Assessment Measures? If Not, Does It Matter? Diabetes Care 2007;30:2725–2728." Diabetes Care 38, no. 12 (November 24, 2015): e214-e214. http://dx.doi.org/10.2337/dci15-0021.

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Pourhassan, M., C.-C. Glüer, P. Pick, W. Tigges, and M. J. Müller. "Impact of weight loss-associated changes in detailed body composition as assessed by whole-body MRI on plasma insulin levels and homeostatis model assessment index." European Journal of Clinical Nutrition 71, no. 2 (October 19, 2016): 212–18. http://dx.doi.org/10.1038/ejcn.2016.189.

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Pelletier, Luc G., Camille Guertin, J. Paige Pope, and Meredith Rocchi. "Homeostasis balance, homeostasis imbalance or distinct motivational processes? Comments on Marks (2015) ‘Homeostatic Theory of Obesity’." Health Psychology Open 3, no. 1 (January 13, 2016): 205510291562451. http://dx.doi.org/10.1177/2055102915624512.

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Zhang, Huimin, Yang Li, Xiani Yao, Gang Liang, and Diqiu Yu. "POSITIVE REGULATOR OF IRON HOMEOSTASIS1, OsPRI1, Facilitates Iron Homeostasis." Plant Physiology 175, no. 1 (July 27, 2017): 543–54. http://dx.doi.org/10.1104/pp.17.00794.

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Kim, Ji-Yong, Yong Ju Yun, Joshua Jeong, C. Yoon Kim, Klaus-Robert Müller, and Seong-Whan Lee. "Leaf-inspired homeostatic cellulose biosensors." Science Advances 7, no. 16 (April 2021): eabe7432. http://dx.doi.org/10.1126/sciadv.abe7432.

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An incompatibility between skin homeostasis and existing biosensor interfaces inhibits long-term electrophysiological signal measurement. Inspired by the leaf homeostasis system, we developed the first homeostatic cellulose biosensor with functions of protection, sensation, self-regulation, and biosafety. Moreover, we find that a mesoporous cellulose membrane transforms into homeostatic material with properties that include high ion conductivity, excellent flexibility and stability, appropriate adhesion force, and self-healing effects when swollen in a saline solution. The proposed biosensor is found to maintain a stable skin-sensor interface through homeostasis even when challenged by various stresses, such as a dynamic environment, severe detachment, dense hair, sweat, and long-term measurement. Last, we demonstrate the high usability of our homeostatic biosensor for continuous and stable measurement of electrophysiological signals and give a showcase application in the field of brain-computer interfacing where the biosensors and machine learning together help to control real-time applications beyond the laboratory at unprecedented versatility.

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Kurniawan, Shahdevi Nandar. "INTRACELLULAR Ca2+ HOMEOSTASIS." MNJ (Malang Neurology Journal) 1, no. 1 (January 1, 2015): 36–45. http://dx.doi.org/10.21776/ub.mnj.2015.001.01.7.

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Journal articles on the topic 'Homeostatis'

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