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Books on the topic 'Homeostasis'

Author: Grafiati

Published: 4 June 2021

Last updated: 21 September 2024

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1

Gelissen, Ingrid C., and Andrew J. Brown, eds. Cholesterol Homeostasis. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6875-6.

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Snow, Andrew L., and Michael J. Lenardo, eds. Immune Homeostasis. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-290-2.

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Carafoli, Ernesto, and Joachim Krebs, eds. Calcium Homeostasis. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-58306-3.

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4

Pool, Ontario Assessment Instrument, ed. Homeostasis: Draft. Toronto: Minister of Education, Ontario, 1989.

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5

Kumar, Vijay, ed. Immune Homeostasis. New York, NY: Springer US, 2024. http://dx.doi.org/10.1007/978-1-0716-3754-8.

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6

Smith, Robert Elijah. Mammalian homeostasis. Burlington, N.C: Carolina Biological Supply Co., 1987.

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7

Banfalvi, Gaspar. Homeostasis - Tumor - Metastasis. Dordrecht: Springer Netherlands, 2014. http://dx.doi.org/10.1007/978-94-007-7335-6.

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8

Jeong, Jeeyon, ed. Plant Iron Homeostasis. New York, NY: Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-3183-6.

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9

1937-, Brenner Barry M., and Stein Jay H. 1937-, eds. Body fluid homeostasis. New York: Churchill Livingstone, 1987.

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10

Panda, Sanjib Kumar, and Yoshiharu Y. Yamamoto, eds. Redox Homeostasis in Plants. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-95315-1.

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11

Hong, Hee-Jeon, ed. Bacterial Cell Wall Homeostasis. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3676-2.

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12

Massry, Shaul G., Michel Olmer, and Eberhard Ritz, eds. Phosphate and Mineral Homeostasis. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5206-8.

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13

Hyndman, Kelly Anne, and Thomas L. Pannabecker, eds. Sodium and Water Homeostasis. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-3213-9.

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14

Bradshaw, S. Donald. Homeostasis in Desert Reptiles. Berlin, Heidelberg: Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60355-6.

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15

N, Fawcett Josephine, ed. Pathophysiology, homeostasis and nursing. New York: Routledge, 2003.

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16

Bradshaw, S. D. Homeostasis in desert reptiles. Berlin: Springer Verlag, 1997.

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17

International Workshop on Phosphate and Other Minerals (7th 1985 Marseille, France). Phosphate and mineral homeostasis. New York: Plenum Press, 1986.

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18

P, Ferrari, ed. Electrolyte and fluid homeostasis. London: Baillière Tindall, 2003.

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19

V, Gisolfi Carl, and Lamb David R, eds. Fluid homeostasis during exercise. Carmel, IN: Benchmark Press, 1990.

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20

Ishimura, Yuzuru, Hideo Shimada, and Makoto Suematsu, eds. Oxygen Homeostasis and Its Dynamics. Tokyo: Springer Japan, 1998. http://dx.doi.org/10.1007/978-4-431-68476-3.

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21

Wang, Donna H., ed. Molecular Sensors for Cardiovascular Homeostasis. Boston, MA: Springer US, 2007. http://dx.doi.org/10.1007/978-0-387-47530-1.

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22

Maret, W., ed. Zinc Biochemistry, Physiology, and Homeostasis. Dordrecht: Springer Netherlands, 2001. http://dx.doi.org/10.1007/978-94-017-3728-9.

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23

1951-, Häussinger D., ed. PH homeostasis: Mechanisms and control. London: Academic Press, 1988.

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24

1935-, Ishimura Yuzuru, Shimada H. 1948-, Suematsu M. 1957-, and Keio International Symposium for Life Sciences and Medicine (1st : 1996 : Tokyo, Japan), eds. Oxygen homeostasis and its dynamics. Tokyo: Springer, 1998.

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25

J, McVicar Andrew, and Baird Nicola 1962-, eds. Perioperative practice: Fundamentals of homeostasis. New York: Routledge, 2002.

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26

John, Clancy. Perioperative practice: Fundamentals of homeostasis. London: Routledge, 2002.

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27

Snow, Andrew L., and Michael Lenardo. Immune homeostasis: Methods and protocols. New York: Humana Press, 2013.

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28

Bowker, Lesley K., James D. Price, Ku Shah, and Sarah C. Smith. Homeostasis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198738381.003.0014.

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This chapter provides information on volume depletion and dehydration, management of dehydration, assessment of hyponatraemia, treatment of hyponatraemia, syndrome of inappropriate antidiuretic hormone secretion (SIADH), hypernatraemia, diagnosis of hypothermia, management of hypothermia, and heat-related illness.

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29

Aréchiga Urtuzuástegui, Hugo. Homeostasis. Universidad Nacional Autónoma de México, Centro de Investigaciones Interdisciplinarias en Ciencias y Humanidades, 2012. http://dx.doi.org/10.22201/ceiich.9786070235672e.2012.

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30

Mason, Peggy. Introduction to Homeostasis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190237493.003.0026.

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Three common misconceptions regarding homeostasis are dispelled. First, the brain has the unique ability to mount an anticipatory defense against changes that could potentially push the body’s physiology out of homeostatic range. Such anticipation of needed adjustments is contrasted to the model of homeostasis as a servomechanism. Second, homeostasis depends on many neurons, not just those in the hypothalamus. Yet hypothalamic neurons play a special role in the integration of challenges and coordination of diverse effector reactions. Third, the idea that homeostasis is the purview of the autonomic nervous system is corrected. As exemplified by respiration and micturition, the brain employs skeletal muscle as well as autonomic targets in supporting visceral life. Finally, the allostatic perspective on the brain’s contribution to staying alive is contrasted with the standard homeostatic perspective and illustrated by examples.

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31

Paradise, Christopher J., and A. Malcolm Campbell. Population Homeostasis. Momentum Press, 2016.

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32

Marples, David, and Søren Nielsen. Water homeostasis. Edited by Robert Unwin. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0022_update_001.

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Under normal circumstances, the maintenance of water balance is a question of balancing urine output against oral water intake, after allowance for the largely unregulated loss of water through other routes (respiratory, transcutaneous, and via the gastrointestinal tract). Normally, this is managed by the feedback mechanisms controlling thirst and diuresis, but in a medical context it is important to allow for other forms of administration that may not be under the control of the patient, and other routes of fluid loss, such as haemorrhage and drains. Electrolyte and water homeostasis are closely interrelated: the major trigger for both antidiuretic hormone (vasopressin) release (and hence renal water retention) and thirst is plasma osmolality. Sodium and chloride are the major solutes in extracellular fluid so are major determinants of body water content and circulating volume.

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33

Doucet, Alain, and Gilles Crambert. Potassium homeostasis. Edited by Robert Unwin. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0023.

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The equilibrium between the concentration of K+ in the extracellular space (low) and the intracellular compartment (high) is crucial for maintaining the electrical properties of excitable and non-excitable cells, because it determines the membrane resting potential. The high intracellular concentration of K+ (120–140 mmol/L) also contributes to the intracellular osmolarity, a determinant of cell volume. It is therefore crucial to finely tune both extracellular and intracellular K+ concentrations. There is a coordinated regulation between processes/mechanisms that store/release K+ from internal stores (internal balance) and those that retain/excrete K+ (external balance).

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34

Murer, Heini, Jürg Biber, and Carsten A. Wagner. Phosphate homeostasis. Edited by Robert Unwin. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0025.

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Inorganic phosphate ions (H2PO4−/ HPO42−) (abbreviated as Pi) are involved in formation of bone and generation of high-energy bonds (e.g. ATP), metabolic pathways, and regulation of cellular functions. In addition, Pi is a component of biological membranes and nucleic acids. Only about 1% of total body Pi content is present in extracellular fluids, at a plasma concentration in adults within the range 0.8–1.4 mMol/L (at pH 7.4 mostly as HPO42−), with diurnal variations of approximately 0.2 mM. A small amount of plasma Pi is bound to proteins or forms complexes with calcium. Under normal, balanced conditions, absorption of dietary Pi along the small intestine equals the output of Pi via kidney and faeces. Renal excretion of Pi represents the key determinant for the adjustment of normal Pi plasma concentrations. Renal reabsorption of Pi occurs along the proximal tubules by sodium-dependent Pi cotransporters that are strictly localized at the apical brush border membrane. Parathyroid hormone (PTH) and FGF23 are key regulators amongst a myriad of factors controlling excretion of Pi in urine, mostly by changes of the apical abundance of Na/Pi cotransporters. Hypophosphataemia may result in osteomalacia, rickets, muscle weakness, and haemolysis. Hyperphosphataemia can lead to hyperparathyroidism and severe calcifications in different tissues.

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35

Trepiccione, Francesco, and Giovambattista Capasso. Calcium homeostasis. Edited by Robert Unwin. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0026.

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Ca2+ homeostasis is achieved through a fine balance among three main organs: the intestine, the kidney, and bone. Blood levels of Ca2+ are accurately tuned through the Ca2+ sensing receptors and regulated by several hormones, including parathyroid hormone (PTH), active vitamin D, and calcitonin. The most recent findings in Ca2+ handling are described. The role of the Ca2+ sensing receptor, as well as Klotho, a new player participating in Ca2+ homeostasis, are described. Finally, the effects of diuretics, calcineurin inhibitors, and the link between hypertension and Ca2+ metabolism are reviewed.

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36

Houillier, Pascal. Magnesium homeostasis. Edited by Robert Unwin. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0027.

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Magnesium is critically important in the process of energy release. Although most magnesium is stored outside the extracellular fluid compartment, the regulated concentration appears in blood. Urinary magnesium excretion can decrease rapidly to low values when magnesium entry rate into the extracellular fluid volume is low, which has several important implications: cell and bone magnesium do not play a major role in the defence of blood magnesium concentration; while a major role is played by the kidney and especially the renal tubule, which adapts to match the urinary magnesium excretion and net entry of magnesium into extracellular fluid. In the kidney, magnesium is reabsorbed in the proximal tubule, the thick ascending limb of the loop of Henle (TALH), and the distal convoluted tubule (DCT). Magnesium absorption is mainly paracellular in the proximal tubule and TALH, whereas it is transcellular in the DCT. The hormone(s) regulating renal magnesium transport and blood magnesium concentration are not fully understood. Renal tubular magnesium transport is altered by a number of hormones, mainly in the TALH and DCT. Parathyroid hormone, calcitonin, arginine vasopressin, ß-adrenergic agonists, and epidermal growth factor, all increase renal tubular magnesium reabsorption; in contrast, prostaglandin E2 decreases magnesium reabsorption. Non-hormonal factors also influence magnesium reabsorption: it is decreased by high blood concentrations of calcium and magnesium, probably via the action of divalent cations on the calcium-sensing receptor; metabolic acidosis decreases, and metabolic alkalosis increases, renal magnesium reabsorption.

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37

Szablewski, Leszek, ed. Glucose Homeostasis. InTech, 2014. http://dx.doi.org/10.5772/57190.

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38

Campbell, A. Malcolm, and Christopher J. Paradise. Organismal Homeostasis. Momentum Press, 2016.

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39

Campbell, A. Malcolm, and Christopher J. Paradise. Ecological Homeostasis. Momentum Press, 2016.

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40

Kompa, Craig Michael. Idealized Homeostasis. Vantage Press, 1995.

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41

Paradise, Christopher J., and A. Malcolm Campbell. Organismal Homeostasis. Momentum Press, 2016.

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42

Paradise, Christopher J., and A. Malcolm Campbell. Ecological Homeostasis. Momentum Press, 2016.

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43

Mirzaei, Jehan. Mission: Homeostasis. iUniverse, Inc., 2006.

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44

Krebs, J., and E. Carafoli. Calcium Homeostasis. Springer Berlin / Heidelberg, 2012.

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45

Kelly, Jeffery W., Franz-Ulrich Hartl, and Richard Morimoto. Protein Homeostasis. Cold Spring Harbor Laboratory Press, 2019.

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46

Witherspoon. Homeostasis IBM. Not Avail, 1998.

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47

Krebs, J., and E. Carafoli. Calcium Homeostasis. Springer London, Limited, 2012.

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48

Paradise, Christopher J., and A. Malcolm Campbell. Ecological Homeostasis. Momentum Press, 2016.

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49

Campbell, A. Malcolm, and Christopher J. Paradise. Ecological Homeostasis. Momentum Press, 2016.

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50

Calcium Homeostasis. Springer, 2011.

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