Relevant bibliographies by topics / HLA histocompatibility antigens

Academic literature on the topic 'HLA histocompatibility antigens'

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Published: 4 June 2021
Last updated: 30 July 2024

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Journal articles on the topic "HLA histocompatibility antigens"

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Barbosa, J., H. Noreen, L. Emme, F. Goetz, R. Simmons, A. DeLeiva, J. Najarian, and E. J. Yunis. "Histocompatibility (HLA) Antigens and Diabetic Microangiopathy*." Tissue Antigens 7, no. 4 (December 11, 2008): 233–37. http://dx.doi.org/10.1111/j.1399-0039.1976.tb01060.x.

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Veerapathran, Anandharaman, Joseph Pidala, Francisca Beato, William E. Janssen, Xue-Zhong Yu, and Claudio Anasetti. "Tregs Specific for Minor Histocompatibility Antigens." Blood 120, no. 21 (November 16, 2012): 1891. http://dx.doi.org/10.1182/blood.v120.21.1891.1891.

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Abstract Abstract 1891 Background: The risk of acute GVHD after HSCT is increased in male recipients of female grafts. Disparities for the male-associated H-Y and other minor histocompatibility antigens (mHAs) have the capacity to sensitize alloreactive donor T cells and cause GVHD in HLA-matched recipients. These mHAs are polymorphic proteins that differ between donor and recipient and are presented as peptides by HLA molecules on recipient or donor antigen-presenting cells to donor immune cells. Currently, there is no evidence that minor histocompatibility antigen specific Tregs exist. Earlier in our laboratory, we have measured the frequency, growth requirements, and function of human blood Tregs specific for allo-MHC. In the present study, we sought to detect the frequency, expansion kinetics and characteristics of the minor antigens specific Tregs in the blood of HLA-matched sibling pair. Methods: CD4+CD25+CD127− Tregs were isolated by immunoabsorption from sibling donors, and cultured with HLA-matched sibling recipient antigen-presenting cells in the presence of IL-2, IL-15 and rapamycin. We detected 30–50 fold increase in H-TdR uptake at 6 days in Treg cultures stimulated by HLA-identical sibling compared to self DC. The precursor frequency of mHA-specific Tregs are between 7 and 43 (median - 13) cells per one million blood Tregs. The frequency of mHA-specific conventional CD4 T cells among total blood CD4 T cells is similar in HLA-matched sibling donors. Ex vivo expanded mHA-specific Tregs maintained higher levels of Foxp3 expression, retained the lymphoid homing receptor CD62L and a chemokine receptor, CCR7, suggesting that they are functional and are able to migrate to lymphoid tissue in vivo. Split well assay on day 12 demonstrated the mHA specificity, since Treg responded to restimulation with DC from the original HLA-identical sibling, but not self DC. The mHA-specific Tregs expanded to more than 100 fold in vitro, and exhibited antigen specific suppression. When Tregs were cultured at limiting dilution, we obtained 6 mHA-specific Treg clones that retained TGF-beta secretion in response to the sibling's mHA-disparate DC but not self DC. Conclusion: We demonstrated for the first time that it is possible to detect and expand mHA specific Tregs from HLA-matched sibling pairs, immunotherapy with mHA-specific Tregs may prevent GVHD. Disclosures: No relevant conflicts of interest to declare.
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Mu�oz, G., M. P. L�pez-Corell, J. F. Taboada, E. Ferrer, and M. D�az-Llopis. "Fuch's heterochromic cyclitis and HLA histocompatibility antigens." International Ophthalmology 18, no. 3 (1994): 127–30. http://dx.doi.org/10.1007/bf00915960.

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Razumova, I. Yu, A. A. Godzenko, I. A. Guseva, and O. K. Vorob’eva. "Uveitis-associated HLA class 1 histocompatibility antigens." Vestnik oftal'mologii 133, no. 5 (2017): 11. http://dx.doi.org/10.17116/oftalma2017133511-15.

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DAMGAARD-JENSEN, L., and F. KISSMEYER-NIELSEN. "HLA HISTOCOMPATIBILITY ANTIGENS IN OPEN-ANGLE GLAUCOMA." Acta Ophthalmologica 56, no. 3 (May 27, 2009): 384–88. http://dx.doi.org/10.1111/j.1755-3768.1978.tb05691.x.

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Falkenburg, J. H., H. M. Goselink, D. van der Harst, S. A. van Luxemburg-Heijs, Y. M. Kooy-Winkelaar, L. M. Faber, J. de Kroon, A. Brand, W. E. Fibbe, and R. Willemze. "Growth inhibition of clonogenic leukemic precursor cells by minor histocompatibility antigen-specific cytotoxic T lymphocytes." Journal of Experimental Medicine 174, no. 1 (July 1, 1991): 27–33. http://dx.doi.org/10.1084/jem.174.1.27.

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Minor histocompatibility (mH) antigens appear to play a major role in bone marrow transplantation (BMT) using HLA-identical donors. Previously, we reported the isolation of major histocompatibility complex (MHC)-restricted mH antigen-specific cytotoxic T lymphocytes (CTL) from patients with graft-vs.-host disease or rejection after HLA-identical BMT. We have demonstrated that mH antigens can be recognized on hematopoietic progenitor cells, and residual recipient CTL specific for mH antigens expressed on donor hematopoietic progenitor cells may be responsible for graft rejection in spite of intensive conditioning regimens in HLA-identical BMT. Here, we investigated whether mH antigen-specific CTL directed against the mH antigens HA-1 to HA-5 and the male-specific antigen H-Y were capable of antigen-specific inhibition of in vitro growth of clonogenic leukemic precursor cells. We demonstrate that mH antigen-specific CTL against all mH antigens tested can lyse freshly obtained myeloid leukemic cells, that these mH antigen-specific CTL can inhibit their clonogenic leukemic growth in vitro, and that this recognition is MHC restricted. We illustrate that leukemic (precursor) cells can escape elimination by mH antigen-specific CTL by impaired expression of the relevant MHC restriction molecule. We suggest that mH antigen-specific MHC-restricted CTL may be involved in vivo in the graft-vs.-leukemia reactivity after BMT.
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IBRAGIMOV, SH I., G. T. MANSUROVA, U. A. MAMATKULOV, and M. R. MAKHSUDOV. "Studying of relationship of Histocompatibility antigens and N-acetylation phenotype in patients of Uzbek populations with psoriasis." Vestnik dermatologii i venerologii 86, no. 1 (February 15, 2010): 67–69. http://dx.doi.org/10.25208/vdv834.

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There studied the distribution of Histocompatibility antigens and N-acetylation phenotype in patients of Uzbek populations with psoriasis and it has been revealed, that HLA antigenes A11, A28, В5, В13 and Cw3 are antigenes of predisposition to presented disease and the individuals with slow N-acetylation phenotype prevail among them. The relationship of more frequent associated Histocompatibility antigens and N-acetylation phenotype marked by the prevalence of patients with a slow phenotype in the dermatosis has been revealed in patients of Uzbek populations with psoriasis.
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Ramilyeva, I. R., Zh K. Burkitbaev, S. A. Abdrakhmanova, A. A. Turganbekova, D. K. Baimukasheva, and E. B. Zhiburt. "DISTRIBUTION PATTERN FOR HLA SPECIFICITIES IN THE PATIENTS WITH ACUTE MYELOID LEUKEMIA." Medical Immunology (Russia) 21, no. 5 (December 13, 2019): 965–72. http://dx.doi.org/10.15789/1563-0625-2019-5-965-972.

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The article presents a study on the distribution of gene polymorphisms in the histocompatibility antigens among the patients diagnosed with AML, and healthy donors in the Republic of Kazakhstan, as well as features of the HLA-A*, *B, Cw*, DRB1*, DQB1* distribution among the patients with acute myeloid leukemia (AML). HLA typing and data processing were performed at the Research and Production Center of Transfusiology, Nur-Sultan. A total of 3808 people were examined, including 3621 healthy blood donors and 187 patients diagnosed with AML. Genomic DNA for HLA typing was isolated from peripheral blood leukocytes by proteinase method using columns with silica membrane and using a set of reagents PROTRANS DNA BOX (Protrans, Germany). Typing of HLA-A, B, C, DRB1, DQB1 in the patients and blood donors was performed by polymerase chain reaction using commercial reagent kits from Protrans (PROTRANS HLA- A*/B*/DRB1* Cyclerplate System, PROTRANS HLA-C* Cyclerplate System, PROTRANS HLA-DQB1* Cyclerplate System).HLA-A*31 (OR = 1.8; CI 1.16-2.79; p < 0.01) proved to be more common in the group of patients compared to the control group, which suggesting an association between AML and presence of this antigen. The control group showed an increased frequency of HLA-A*02 antigen (OR = 0.55; CI 0.41-0.75; p < 0.01). This antigen may be, therefore, exert a protective effect in AML development.The studies of major histocompatibility complex which include HLA genes, did significantly expanded the understanding of HLA antigens which may have strong associative links with distinct diseases, and moderately or poorly expressed links in other disorders. Analysis of the literature data showed that myeloid leukemia is characterized by decreased frequency of HLA-B13, B14, B40 antigens, most often determined by antigens B16, Bw 22, B27. In this study, HLA-A*31, B*37 were associated with AML. Phenotypes with antigens HLA-A*02, B*27, C*02, DRB1*01, *04, DQB1*06 have a probable protective effect on the development of this pathology.The study has determined some features of histocompatibility gene distribution in AML patients, detection of HLA-markers that determine the risk or resistance to the occurrence of this disease. We have established characteristic specific markers of HLA system among AML patients in Kazakhstan, which may be associated with higher risk of the disease.
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Romanyuk, D. S., A. M. Pilunov, G. A. Efimov, A. V. Bogolyubova, and E. N. Parovichnikova. "Minor histocompatibility antigens represented in HLA-A*02:01 and their search strategies." Oncohematology 18, no. 3 (September 13, 2023): 115–24. http://dx.doi.org/10.17650/1818-8346-2023-18-3-115-124.

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Minor histocompatibility antigens (MiHAs) are polymorphic peptides on the cell surface derived from self-proteins that are capable to induce an immune response during allogeneic hematopoietic stem cells transplantation. Their presentation occurs in the context of the certain major histocompatibility complex (HLA – human leucocyte antigen) alleles. One of the most common HLA alleles is HLA-A*02:01. Accordingly, for a significant number of donors and recipients pairs, it is possible to use the MiHAs presented in the HLA-A*02:01 as a target for relapsed leukemia therapy. This review discusses the main known MiHAs presented in the context of HLA-A*02:01, their characteristics and approaches used for identification. The described approaches may be used to search for new MiHAs for immunotherapy.
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Stern, Martin, Loredana Ruggeri, Marusca Capanni, Antonella Mancusi, and Andrea Velardi. "Human leukocyte antigens A23, A24, and A32 but not A25 are ligands for KIR3DL1." Blood 112, no. 3 (August 1, 2008): 708–10. http://dx.doi.org/10.1182/blood-2008-02-137521.

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Abstract Inhibitory killer cell immunoglobulin receptors (KIR) bind to major histocompatibility complex antigens. Concise knowledge of KIR ligands allows prediction of natural killer (NK)–cell alloreactivity after hematopoietic stem cell transplantation. KIR3DL1 binds to the Bw4 epitope on HLA-B antigens. Although the same epitope is also found on 4 HLA-A antigens (HLA-A23/24/25/32), these are not currently regarded as KIR3DL1 ligands. We show that expression of HLA A*2301, A*2402, or A*3201 but not HLA A*2501 protects target cells from lysis by KIR3DL1+ NK cells. KIR3DL1+ NK cells from donors expressing the Bw4 epitope on an HLA-A antigen only are fully functional and capable of lysing Bw4− target cells. HLA A25 differs at amino acid 90, close to the serologic Bw4 epitope, from A23/24/32 and from Bw4+ HLA-B antigens. These data suggest that HLA-A antigens should be taken into consideration when assessing the potential for NK alloreactivity after hematopoietic stem cell transplantation.
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Dissertations / Theses on the topic "HLA histocompatibility antigens"

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Talken, Beth L. "Assembly of the Lw¹⁶ and Ld class I MHC molecules." free to MU campus, to others for purchase, 1996. http://wwwlib.umi.com/cr/mo/fullcit?p9720532.

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Pang, Ha Sang. "Identification of CD8+ T cell epitopes from HCA661 presented by HLA-A2 molecules /." View abstract or full-text, 2006. http://library.ust.hk/cgi/db/thesis.pl?BIOL%202006%20PANG.

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葉德俊 and Tak-chun Timothy Yip. "Characterization of a monoclonal antibody reactive against major histocompatibility complex class II antigens." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1992. http://hub.hku.hk/bib/B3123334X.

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Yip, Tak-chun Timothy. "Characterization of a monoclonal antibody reactive against major histocompatibility complex class II antigens /." [Hong Kong] : University of Hong Kong, 1992. http://sunzi.lib.hku.hk/hkuto/record.jsp?B13478771.

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Hume, Clifford Robert. "Regulation of HLA class II expression in class II negative mutant B-cell lines /." Access full-text from WCMC, 1989. http://proquest.umi.com/pqdweb?did=745028251&sid=1&Fmt=2&clientId=8424&RQT=309&VName=PQD.

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Halley, Lorna Louise. "The investigation of the HLA system and wheat gluten in determining risk of schizophrenia." Thesis, University of the Highlands and Islands, 2015. https://pure.uhi.ac.uk/portal/en/studentthesis/the-investigation-of-the-hla-system-and-wheat-gluten-in-determining-risk-of-schizophrenia(60acd449-c659-4743-9dbd-4392c0fc015a).html.

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Genome-wide association (GWA) studies confirmed that the HLA genes were strongly associated with schizophrenia but the HLA variants identified by GWA studies had a lower frequency in patients with schizophrenia than control subjects. The HLA molecules have function of presenting peptide antigens to T lymphocytes in order to initiate an immune response. Environmental factors such as infection and dietary proteins have been found to be associated with schizophrenia. This PhD program has thus focused on the following objectives: (1) identification of genetic variants for schizophrenia in the HLA region and (2) investigation of circulating antibodies to linear peptide antigens derived from wheat gluten in schizophrenia. The major findings from this work are as follows: 1. The HLA-DQ2.5 variants were strongly associated with schizophrenia; this finding is consistent with that from GWA study. 2. The NOTCH4 association was replicated in our study samples, in which a CNV in exon 19 of the gene may be associated with risk of schizophrenia. 3. There was no HLA-II variant identified to be associated with a high risk of schizophrenia but a CNV present in the HLA-DQ/DR region might confer risk of the disease. 4. The levels of circulating antibodies to linear peptide antigens derived from wheat gluten were significantly lower in schizophrenia patients than controls. This finding is inconsistent with previous studies that showed elevated levels of circulating antibodies in schizophrenia across subpopulations. In conclusion, it is likely that not one but many HLA variants lead to risk of schizophrenia development. From this research it is likely that anti-gluten antibodies are not an environmental trigger for this disease. Further investigation is needed to clarify the role of the HLA region in bridging the gap between genetic make-up and environmental factors in developing schizophrenia.
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Kosmoliaptsis, Vasilis. "Investigation into the immunogenicity of human leukocyte antigen mismatches in kidney transplantation." Thesis, University of Cambridge, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.609630.

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Schulte, Kathleen Q. "Mutagenized HLA DNA Constructs: Tools for Validating Molecular HLA Typing Methodologies." Thesis, University of North Texas, 1999. https://digital.library.unt.edu/ark:/67531/metadc500888/.

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This study describes the development and validation of mutagenized cloned DNA constructs, which correspond to the polymorphic regions of the class II region of the HLA complex. The constructs were used to verify the allelic specificity of primers and probes in polymerase chain reaction (PCR)-based HLA typing assays such as Sequence Specific Primers (SSP) and Sequence Specific Oligonucleotide Probes (SSOP). The constructs consisted of the entire polymorphic region of exon 2 of class II HLA allele sequences that included primer annealing sites or probe hybridization sites. An HLA allele sequence was inserted into a plasmid, cloned, then mutagenized to match a specific HLA allele, and finally, the correct clone was verified by bidirectional sequencing of the insert. Thus, the construct created a cloned reference DNA sample for any specific allele, and can be used to validate the accuracy of various molecular methodologies.
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Yamamoto, Masaru. "Synthesis and oxidation studies of sulfur containing inhibitors for human leukocyte elastase : (2) synthesis of cyclic peptide analogs for tissue factor pathway inhibitor (TFPI) : Part 2 synthesis and evaluation of aziridinecarboxylic acid analogs as a new family of cysteine proteinase inhibitors." Diss., Georgia Institute of Technology, 1993. http://hdl.handle.net/1853/25953.

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Chang, Yea-wen. "Application of molecular genetic techniques to the study of major histocompatibility complex class II allelic associations with insulin-dependent diabetes mellitus in Chinese /." Hong Kong : University of Hong Kong, 1997. http://sunzi.lib.hku.hk/hkuto/record.jsp?B18539919.

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Books on the topic "HLA histocompatibility antigens"

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Bo, Dupont, Ceppellini Ruggero, and International Histocompatibility Workshop and Conference (10th : 1987 : Princeton, N.J. and New York, N.Y.), eds. Immunobiology of HLA. New York: Springer-Verlag, 1989.

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J, Crumpton M., ed. HLA in medicine. Edinburgh: Churchill Livingstone for the British Council, 1987.

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1929-, Honda Y., Juji T. 1935-, and International Symposium on Narcolepsy (2nd : 1985 : Stanford University), eds. HLA in narcolepsy. Berlin: Springer-Verlag, 1988.

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Gorer, Symposium (1983 London England). Major histocompatibility system: The Gorer Symposium. Oxford: Blackwell Scientific Publications, 1985.

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Lee, John, 1935 July 30- and Red Cross International Histocompatibility Workshop (1st : 1990 : Beijing, China), eds. The HLA system: A new approach. New York: Springer-Verlag, 1990.

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Abrahámová, Jitka. HLA system and some neoplastic diseases. Praha: Univerzita Karlova, 1988.

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Robert, Lechler, ed. HLA and disease. London: Academic Press, 1994.

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Scott, Murphy, ed. The HLA system: Basic biology and clinical applications. Bethesda, Md: American Association of Blood Banks, 1999.

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G, Solheim Bjarte, Ferrone Soldano 1940-, and Möller Erna, eds. The HLA system in clinical transplantation: Basic concepts and importance. Berlin: Springer-Verlag, 1993.

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1946-, Silver Jack, ed. Molecular biology of HLA class II antigens. Boca Raton, Fla: CRC Press, 1990.

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Book chapters on the topic "HLA histocompatibility antigens"

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Spierings, Eric, Alejandro Madrigal, and Katharina Fleischhauer. "Histocompatibility." In The EBMT Handbook, 73–84. Cham: Springer International Publishing, 2024. http://dx.doi.org/10.1007/978-3-031-44080-9_9.

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AbstractHuman leukocyte antigen (HLA) molecules are the most important histocompatibility antigens, due to their genetic polymorphism and their key role in peptide antigen presentation and T-cell alloreactivity. While full matching for the most relevant HLA loci had been regarded as a prerequisite for successful transplantation until recently, the introduction of posttransplant cyclophosphamide (PTCy) as immune prophylaxis has also allowed successful transplantation across multiple HLA mismatches, thus also enabling access to transplantation for patients without a fully compatible donor. The rules governing high-risk/nonpermissive HLA mismatches, identified in the past as immunopeptidome overlaps, expression levels, and predicted indirectly recognized HLA epitopes (PIRCHEs), will have to be redefined in the PTCy area to further improve patient outcomes.
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Lozano Chinga, Michell M., David Buchbinder, and Jolan E. Walter. "HistocompatIbility Antigens (HLA) and Transplantation." In Absolute Allergy and Immunology Board Review, 21–42. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-12867-7_3.

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Collins, T., J. S. Pober, and J. L. Strominger. "Physiologic Regulation of Class II Major Histocompatibility Complex Gene Expression." In HLA Class II Antigens, 14–31. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-70367-6_2.

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Pla, M., and P. Ivanyi. "Cross-reactions of Class II Histocompatibility Antigens of Various Species." In HLA Class II Antigens, 128–53. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-70367-6_8.

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Schenkel-Brunner, Helmut. "Antigens of the Major Histocompatibility Complex (HLA Antigens)." In Human Blood Groups, 595–605. Vienna: Springer Vienna, 2000. http://dx.doi.org/10.1007/978-3-7091-6294-1_31.

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Schenkel-Brunner, Helmut. "The Antigens of the Major Histocompatibility Complex (HLA Antigens)." In Human Blood Groups, 415–25. Vienna: Springer Vienna, 1995. http://dx.doi.org/10.1007/978-3-7091-3686-7_25.

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Milford, Edgar L., and Edmond J. Yunis. "New HLA Antigens Identified in the Serologic Studies of the Tenth International Histocompatibility Workshop, 1987." In Immunobiology of HLA, 52–53. New York, NY: Springer New York, 1989. http://dx.doi.org/10.1007/978-1-4612-3552-1_9.

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MacSween, Joseph M., James A. Collicut, Stuart L. Eastwood, and Allan D. Cohen. "An In Vitro Model to Investigate the Importance for Renal Transplant Survival of Mismatches for Specific Histocompatibility Antigens." In Immunobiology of HLA, 507–8. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-662-39946-0_217.

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Dessi, V., H. Lazarus, E. Raniezi, E. Celada, R. J. Winchester, and A. Nuñez-Roldan. "Five Human IgG Monoclonal Antibodies Obtained from a Single Polytransfused Patient Specific for Different Class I and Class II Major Histocompatibility Antigens." In Immunobiology of HLA, 339–40. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-662-39946-0_128.

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Lawrance, S. K., L. Karlsson, J. Price, V. Quaranta, Y. Ron, J. Sprent, and P. A. Peterson. "T Cell Recognition of Major Histocompatibility Complex Antigens in HLA Class II Transgenic Mice." In Transgenic Mice and Mutants in MHC Research, 247–56. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-75442-5_33.

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Conference papers on the topic "HLA histocompatibility antigens"

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Godzenko, A., I. Guseva, and I. Razumova. "THU0682 Human histocompatibility antigens (HLA) class 1 in anterior uveitis patients with and without spondyloarthritis." In Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.2773.

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Keefe, Robert, Tim Mayall, Egidio Cofano, Marlyn Anguelov, Constantinos Panousis, and Paola Sette. "275 Manufacture of allogeneic, HLA-matched, TCR-edited T-cell therapy reactive against minor histocompatibility antigen 1 to treat acute myeloid leukemia in combination with CD34 HSCT with the potential for high potency and durability." In SITC 37th Annual Meeting (SITC 2022) Abstracts. BMJ Publishing Group Ltd, 2022. http://dx.doi.org/10.1136/jitc-2022-sitc2022.0275.

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