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Relevant bibliographies by topics / HIV pathogenicity; Gene therapy / Books

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Books on the topic 'HIV pathogenicity; Gene therapy'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Bauer, Gerhard, and Joseph S. Anderson. Gene Therapy for HIV. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-0434-1.

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2

Singwi, Sanjeev. HIV gene therapy using nucleases. Ottawa: National Library of Canada, 1996.

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3

Smith, Clay. Gene Therapy for HIV Infection. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-662-11821-4.

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4

Hengge, Ulrich R. Immunotreatment and gene therapy of HIV infection. Bremen: Uni-Med, 2004.

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5

Berkhout, Ben, Hildegund C. J. Ertl, and Marc S. Weinberg, eds. Gene Therapy for HIV and Chronic Infections. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2432-5.

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6

Lombardi, Rocco Anthony. GAG and ENV trans-dominant mutants for use in ANTI-HIV-1 gene therapy. Ottawa: National Library of Canada, 1996.

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7

1937-, Matsushita Masaaki, and Fukunishi Isao, eds. Cutting edge medicine and liaison psychiatry: Psychiatric problems of organ transplantation, cancer, HIV/AIDS, and genetic therapy : proceedings of the 13th Tokyo Institute of Psychiatry International Symposium held in Tokyo on 29-30 September 1998. Amsterdam: Elsevier, 1999.

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8

1957-, Smith Clay, ed. Gene therapy for HIV infection. Georgetown, Tex: R.G. Landes, 1998.

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9

Berkhout, Ben, Hildegund C. J. Ertl, and Marc S. Weinberg. Gene Therapy for HIV and Chronic Infections. Springer, 2015.

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10

Bauer, Gerhard, and Joseph S. Anderson. Gene Therapy for HIV: From Inception to a Possible Cure. Springer, 2014.

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11

Factor, Phillip H. Gene Therapy for Acute and Acquired Diseases. Springer, 2012.

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12

Gene Therapy for Acute and Acquired Diseases. Springer, 2001.

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13

Medina, Maria Fe C. Strategies for isolation and expression of ribozymes for use in HIV gene therapy. 2000.

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14

Ramezani, Ali. Development and testing of mono- and multimeric hammerhead ribozymes for HIV-1 gene therapy. 2002.

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15

G, Giraldo, ed. Development and applications of vaccines and gene therapy in AIDS. Basel: Karger, 1996.

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16

Goba Ma Khosi: The Final Solution. Lulu Publishing, 2009.

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17

Culshaw, Rebecca. Science Sold Out: Does HIV Really Cause AIDS? North Atlantic Books, 2007.

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18

Cured: How the Berlin patients defeated HIV and forever changed medical science. Dutton, 2014.

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19

(Editor), I. Scharrer, and W. Schramm (Editor), eds. 30th Hemophilia Symposium Hamburg 1999: HIV Infection and Epidemiology in Hemophilia; Gene Therapy in Hemophilia A and B; Therapy of Hepatitis C; Inhibitors ... Pediatric Hemostasiology; Case Reports. Springer, 2000.

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20

Juelg, Boris, and Rajesh Gandhi. HIV Cure Strategies. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190493097.003.0006.

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Although current antiretroviral therapy (ART) is highly effective at controlling HIV-1 replication, it does not eradicate or cure the infection. HIV-1 persists quiescently in cellular reservoirs, not detected by the immune system due to the lack of active viral replication; these reservoirs represent the major obstacle for cure approaches. Reversal of HIV-1 latency and induction of virus expression by a variety of interventions may render infected cells susceptible to immune recognition and active clearance. Strategies to boost immune responses via vaccination, immunomodulation, or gene therapy are being evaluated with the aim of achieving HIV-1 control without antiretroviral therapy, if not viral eradication.

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21

Naicker, Saraladevi, and Graham Paget. HIV and renal disease. Edited by Vivekanand Jha. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0187_update_001.

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The human immunodeficiency virus (HIV) infection epidemic has particularly affected the poorest regions of the world. HIV can directly or indirectly affect different aspects of renal function, and results in a variable expression of kidney disease.Acute kidney injury (AKI) occurs in approximately 20% of hospitalized patients. The prevalence of chronic kidney disease (CKD) amongst HIV-infected patients is reported at 3.5–38% in different regions of the world. The complex interplay between the pheno- and/or genotypic variants of the virus, the genetic make-up of the host, and environmental factors determine the clinical manifestations of renal disease. The association of APOL1 gene variants G1 and G2 with the risk of focal segmental glomerulosclerosis explains the high frequency of HIV-associated nephropathy (HIVAN) in populations of black ethnicity.Anti-retroviral therapy (ART) is effective in preventing progression of HIVAN. Some of the drugs used in ART regimens are potentially nephrotoxic and require dose adjustment or even avoidance in CKD. Progression to end-stage renal disease (ESRD) in HIVAN has been reported to correlate with the extent of chronic damage quantified by renal biopsy.HIV-infected patients requiring dialysis, who are stable on ART, are achieving survival rates comparable to those of non-HIV dialysis populations. Similarly, HIV infection does not seem to adversely affect patient and graft survival rates after kidney transplantation, and there has been no increase in the prevalence of opportunistic infections in transplant recipients on effective ART.

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22

Retroviruses of Human A.I.D.S. and Related Animal Diseases (Colloque des Cent Gardes 26-27-28 Octobre 1989 Marnes-La Coquette/Paris - France). Pasteur Vaccins, 1989.

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