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Relevant bibliographies by topics / Histocompatibilty complex

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Academic literature on the topic 'Histocompatibilty complex'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "Histocompatibilty complex"

1

Qin, J., C. Mamotte, N. E. Cockett, J. D. Wetherall, and D. M. Groth. "A map of the class III region of the sheep major histocompatibilty complex." BMC Genomics 9, no. 1 (2008): 409. http://dx.doi.org/10.1186/1471-2164-9-409.

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Olufowobi, Olanrewaju Teslim, Babatunde Moses Ilori, Olajide Olowofeso, Olajide Mark Sogunle, and Adewunmi Omolade Omotoso. "Genetic variation of the major histocompatibilty complex-B haplotypes in Nigerian local chicken populations." Agricultura Tropica et Subtropica 53, no. 4 (December 1, 2020): 175–81. http://dx.doi.org/10.2478/ats-2020-0017.

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AbstractTo understand the genetic basis and mechanism underlying the differences in the level of immunity among and within chicken populations in Nigeria, it is important to start from the Major Histocompability Complex (MHC) region particularly as it serves as a reservoir for genes of the immune system. The B-complex of chicken major histocompatibility complex, located on microchromosome 16, consists of gene classes responsible for immunity through antigen presentation to T cells. A highly polymorphic tandem repeat marker (LEI0258) located within the B-complex has been a marker of choice for genotyping to identify major histocompatibility complex-B haplotypes and to study the genetic diversity of chicken populations. This study was carried out to determine the genetic variations, at the LEI0258 locus, in three Nigerian local chicken populations; Normal feather, Frizzle feather and Naked neck. The allelic and genotypic profiles of each representative from each population were determined through polymerase chain reaction amplification of the repeat region. The genetic diversity parameters, analysis of molecular variance and evolutionary relationship were determined using GenAlex, FSTAT, Arlequin and POPTREEW, respectively. 76 % of the entire population was heterozygous at the LEI0258 locus. Analysis of molecular variance revealed that large proportion of the total variations across populations was due to variation between individuals (79 %), whereas variations among the populations and among individuals within populations only accounted for less than 1 % and 21 %, respectively. Using Anak Titan as an exotic outgroup, the evolutionary relationship among the Nigerian local chicken populations was studied and a Nei-based dendrogram showed two major clades separating the exotic population from the Nigerian local chicken populations. The identified diversity at the locus could be exploited for usage in further breeding programmes especially for disease resistance and fitness in locally adapted chicken populations in Nigerian.

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Hirata, Jun, Kazuyoshi Hosomichi, Saori Sakaue, Masahiro Kanai, Hirofumi Nakaoka, Kazuyoshi Ishigaki, Ken Suzuki, et al. "Genetic and phenotypic landscape of the major histocompatibilty complex region in the Japanese population." Nature Genetics 51, no. 3 (January 28, 2019): 470–80. http://dx.doi.org/10.1038/s41588-018-0336-0.

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Nikbakht-sangari, M., A. K. Qayumi, P. Keown, V. Duronio, and K. Horley. "Platelet-Activating Factor Plays a Role in the Mechanism of Major Histocompatibilty Complex in T Lymphocytes." Immunological Investigations 28, no. 4 (January 1999): 223–33. http://dx.doi.org/10.3109/08820139909060857.

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Lee, K. W., B. Choi, Y. M. Kim, C. W. Cho, H. Park, J. I. Moon, G. S. Choi, J. B. Park, and S. J. Kim. "Major Histocompatibilty Complex–Restricted Adaptive Immune Responses to CT26 Colon Cancer Cell Line in Mixed Allogeneic Chimera." Transplantation Proceedings 49, no. 5 (June 2017): 1153–59. http://dx.doi.org/10.1016/j.transproceed.2017.03.011.

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Shiels, Carol, Suhail A. Islam, Radost Vatcheva, Peter Sasieni, Michael J. E. Sternberg, Paul S. Freemont, and Denise Sheer. "PML bodies associate specifically with the MHC gene cluster in interphase nuclei." Journal of Cell Science 114, no. 20 (October 15, 2001): 3705–16. http://dx.doi.org/10.1242/jcs.114.20.3705.

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Promyelocytic leukemia (PML) bodies are nuclear multi-protein domains. The observations that viruses transcribe their genomes adjacent to PML bodies and that nascent RNA accumulates at their periphery suggest that PML bodies function in transcription. We have used immuno-FISH in primary human fibroblasts to determine the 3D spatial organisation of gene-rich and gene-poor chromosomal regions relative to PML bodies. We find a highly non-random association of the gene-rich major histocompatibilty complex (MHC) on chromosome 6 with PML bodies. This association is specific for the centromeric end of the MHC and extends over a genomic region of at least 1.6 megabases. We also show that PML association is maintained when a subsection of this region is integrated into another chromosomal location. This is the first demonstration that PML bodies have specific chromosomal associations and supports a model for PML bodies as part of a functional nuclear compartment.

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Mahy, Nicola L., Paul E. Perry, and Wendy A. Bickmore. "Gene density and transcription influence the localization of chromatin outside of chromosome territories detectable by FISH." Journal of Cell Biology 159, no. 5 (December 9, 2002): 753–63. http://dx.doi.org/10.1083/jcb.200207115.

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Genes can be transcribed from within chromosome territories; however, the major histocompatibilty complex locus has been reported extending away from chromosome territories, and the incidence of this correlates with transcription from the region. A similar result has been seen for the epidermal differentiation complex region of chromosome 1. These data suggested that chromatin decondensation away from the surface of chromosome territories may result from, and/or may facilitate, transcription of densely packed genes subject to coordinate regulation. To investigate whether localization outside of the visible confines of chromosome territories can also occur for regions that are not coordinately regulated, we have examined the spatial organization of human 11p15.5 and the syntenic region on mouse chromosome 7. This region is gene rich but its genes are not coordinately expressed, rather overall high levels of transcription occur in several cell types. We found that chromatin from 11p15.5 frequently extends away from the chromosome 11 territory. Localization outside of territories was also detected for other regions of high gene density and high levels of transcription. This is shown to be partly dependent on ongoing transcription. We suggest that local gene density and transcription, rather than the activity of individual genes, influences the organization of chromosomes in the nucleus.

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Porto, Graça, Eugénia Cruz, Maria José Teles, and Maria de Sousa. "HFE Related Hemochromatosis: Uncovering the Inextricable Link between Iron Homeostasis and the Immunological System." Pharmaceuticals 12, no. 3 (August 22, 2019): 122. http://dx.doi.org/10.3390/ph12030122.

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The HFE gene (OMIM 235200), most commonly associated with the genetic iron overload disorder Hemochromatosis, was identified by Feder et al. in 1996, as a major histocompatibilty complex (MHC) class I like gene, first designated human leukocyte antigen-H (HLA-H). This discovery was thus accomplished 20 years after the realization of the first link between the then “idiopathic” hemochromatosis and the human leukocyte antigens (HLA). The availability of a good genetic marker in subjects homozygous for the C282Y variant in HFE (hereditary Fe), the reliability in serum markers such as transferrin saturation and serum ferritin, plus the establishment of noninvasive methods for the estimation of hepatic iron overload, all transformed hemochromatosis into a unique age related disease where prevention became the major goal. We were challenged by the finding of iron overload in a 9-year-old boy homozygous for the C282Y HFE variant, with two brothers aged 11 and 5 also homozygous for the mutation. We report a 20 year follow-up during which the three boys were seen yearly with serial determinations of iron parameters and lymphocyte counts. This paper is divided in three sections: Learning, applying, and questioning. The result is the illustration of hemochromatosis as an age related disease in the transition from childhood to adult life and the confirmation of the inextricable link between iron overload and the cells of the immune system.

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MAROSI, BÉLA, KAREN M. KIEMNEC-TYBURCZY, IOAN V. GHIRA, TIBOR SOS TIBOR SOS, and OCTAVIAN POPESCU. "Identification and characterization of major histocompatibility complex class IIB alleles in three species of European ranid frogs." Indian Journal of Applied Research 3, no. 9 (October 1, 2011): 4–6. http://dx.doi.org/10.15373/2249555x/sept2013/2.

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Zorc, Minja, Jernej Ogorevc, and Peter Dovc. "The new bovine reference genome assembly provides new insight into genomic organization of the bovine major histocompatibility complex." Journal of Central European Agriculture 20, no. 4 (2019): 1111–15. http://dx.doi.org/10.5513/jcea01/20.4.2679.

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Dissertations / Theses on the topic "Histocompatibilty complex"

1

Willer, Cristen. "Genetic and environmental susceptibility to multiple sclerosis." Thesis, University of Oxford, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.275379.

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Lincoln, Matthew R. "Candidate gene studies and fine-mapping of the mjor histocompatibilty complex association in multiple sclerosis." Thesis, University of Oxford, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.427634.

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Berggren, Bremdal Karin. "Evolution of MHC Genes and MHC Gene Expression." Doctoral thesis, Uppsala universitet, Evolutionsbiologi, 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-122011.

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Polymorphism in coding regions and regions controlling gene expression is the major determinant of adaptive differences in natural populations. Genes of the major histocompatibility complex (MHC) possess a high level of genetic variation, which is maintained by selection over long coalescence times. MHC genes encode antigen-presenting molecules in the adaptive immune system, which protects the host from infectious diseases. However, MHC molecules may also present self-peptides and for most autoimmune diseases there is a genetic factor associated with the MHC. MHC genes have been used to learn about the interplay of selection and historical population events. In domestic dogs and their progenitor, the wolf, I explored factors associated with domestication and breed formation and their influence not only on MHC coding regions but also on the haplotypic structure of the class II region. Polymorphism and strong selection was demonstrated in the proximal promoters of MHC genes in dogs and wolves. Hence, genetic variation associated with MHC gene expression may have at least equal importance for a well functioning immune system. Associations between promoter sequences and particular coding alleles suggested allele-specific expression patterns. SNP haplotypes of the MHC class II region revealed ancestral as well as convergent haplotypes, in which combinations of alleles are kept by selection. Interestingly, weaker allelic associations were found between different genes and between coding regions and promoters in dogs compared to wolves. Potentially, this could cause insufficient defense against infections and predispose dogs to autoimmune diseases. For example, I identified a site in the promoter region that showed a consistent difference between haplotypes conferring susceptibility and protection to diabetes in dogs, which should be investigated further. Furthermore, I investigated how selection and demographic changes associated with glacial and inter-glacial periods have affected MHC variation in European hedgehogs and extended the prevailing knowledge concerning their population history.

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Hirni, Helen. "The Major histocompatibility complex in horses /." [S.l : s.n.], 1988. http://www.ub.unibe.ch/content/bibliotheken_sammlungen/sondersammlungen/dissen_bestellformular/index_ger.html.

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Laguna, Goya Rocío. "Major histocompatibility complex and stem cells." Thesis, University of Cambridge, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.608773.

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Brown, Jason John. "Polymorphisms of the equine Major Histocompatibility Complex." Thesis, University of Liverpool, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.406666.

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Paterson, Stephen. "Major histocompatibility complex variation in Soay sheep." Thesis, University of Cambridge, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.627271.

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Kemp, Stephen John. "The major histocompatibility complex of African cattle." Thesis, University of Edinburgh, 1985. http://hdl.handle.net/1842/15146.

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Kendall, Elaine. "Molecular characterisation of the human major histocompatibility complex." Thesis, University of Oxford, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.333402.

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10

Takousis, Petrus. "DNA replication in the human major histocompatibility complex." Thesis, University College London (University of London), 2007. http://discovery.ucl.ac.uk/1445119/.

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DNA replication is a vital component of the eukaryotic cell cycle. During the course of S-phase, numerous origins of replication become activated along each chromosome. Several adjacent origins fire synchronously to replicate large sections of a chromosome at specific times. Early studies identified a relationship between cytogenetic bands and replication timing: GC-rich R-bands replicate early while AT-rich and gene poor G-bands replicate late, apparently regardless of differentiation and developmental status. Subsequent studies revealed that other factors such as transcriptional status also influence the replication programme. The aim of this thesis is to examine the organisation of DNA replication in the human Major Histocompatibility Complex (MHC) on chromosome 6, and understand how it relates to gene expression and inherent genomic properties. A previous investigation from the Human Cytogenetics Laboratory using fluorescence in situ hybridisation (FISH) suggested that replication timing of the MHC is organised into distinct zones, with the MHC class II region, an AT-rich isochore, replicating later than neighbouring regions. Using a biochemical approach, the entire MHC was found to replicate within the first half of S-phase in cell lines derived from different tissues. Subsequent analysis of a B-lymphoblastoid cell line using a high resolution tiling path array for the MHC confirmed that a large proportion of the MHC class II replicates later than its neighbours. The data suggested the existence within the MHC of replication origins that fire at distinct times in S-phase. An investigation of replication initiation in the MHC revealed the presence of several potential initiation sites, which were further analysed by quantitative PCR. The gene-rich MHC class III was found to have a relatively large number of replication initiation sites. Overall, these results suggest that either specific origins of replication or zones of initiation can fulfill the replication requirements of a region.

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Books on the topic "Histocompatibilty complex"

1

Kasahara, Masanori, ed. Major Histocompatibility Complex. Tokyo: Springer Japan, 2000. http://dx.doi.org/10.1007/978-4-431-65868-9.

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2

Natural history of the major histocompatibility complex. New York: Wiley, 1986.

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3

Rammensee, Hans-Georg. MHC ligands and peptide motifs. Austin, Tex: Landes Bioscience, 1997.

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4

Klein, Jan. Natural history of the major histocompatibility complex. New York: Wiley, 1986.

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5

NATO Advanced Research Workshop on MHC Evolution (1991 Key Biscayne, Fla.). Molecular evolution of the major histocompatibility complex. Berlin: Springer-Verlag, 1991.

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Klein, Jan, and Dagmar Klein, eds. Molecular Evolution of the Major Histocompatibility Complex. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-84622-9.

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7

Aizawa, Miki. Major histocompatibility complex of the rat, rattus norvegicus. Sapporo: Hokkaido University School of Medicine, 1988.

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8

Konenkov, V. I. Medit͡s︡inskai͡a︡ i ėkologicheskai͡a︡ immunogenetika. Novosibirsk: [SO RAMN], 1999.

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9

Harding, Clifford V. MHC molecules and antigen processing. Austin, Tex: R.G. Landes Co., 1997.

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10

Aizawa, Miki. Major histocompatibility complex of the rat, Rattus norvegicus: Its structure and function. Sapporo: Hokkaido University School of Medicine, 1988.

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Book chapters on the topic "Histocompatibilty complex"

1

Auffray, Charles. "Molecular Anatomy of the Chicken Major Histocompatibilty B Complex." In Improving Genetic Disease Resistance in Farm Animals, 99–103. Dordrecht: Springer Netherlands, 1989. http://dx.doi.org/10.1007/978-94-009-1057-7_11.

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Chaudhary, Reema. "Major Histocompatibility Complex." In Encyclopedia of Animal Cognition and Behavior, 1–4. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-47829-6_551-1.

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Cruse, Julius M., and Robert E. Lewis. "Major Histocompatibility Complex." In Atlas of Immunology, 77–89. Berlin, Heidelberg: Springer Berlin Heidelberg, 1999. http://dx.doi.org/10.1007/978-3-662-11196-3_4.

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Rajalingam, Raja, Qiuheng Zhang, J. Michael Cecka, and Elaine F. Reed. "Major histocompatibility complex." In Transplant Immunology, 85–102. Chichester, UK: John Wiley & Sons, Ltd, 2015. http://dx.doi.org/10.1002/9781119072997.ch5.

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Klohe, Ellen, and Janardan P. Pandey. "Major histocompatibility complex." In Medical Immunology, 23–36. 7th edition. | Boca Raton : Taylor & Francis, 2020.: CRC Press, 2019. http://dx.doi.org/10.1201/9780429278990-3.

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Renz, H., and B. Gierten. "Major Histocompatibility Complex." In Springer Reference Medizin, 1560–61. Berlin, Heidelberg: Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-48986-4_2012.

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Renz, H., and B. Gierten. "Major Histocompatibility Complex." In Lexikon der Medizinischen Laboratoriumsdiagnostik, 1. Berlin, Heidelberg: Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-49054-9_2012-1.

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Matsuura, Akihiro, and Miyuki Kinebuchi. "Rat TL and CD1." In Major Histocompatibility Complex, 222–35. Tokyo: Springer Japan, 2000. http://dx.doi.org/10.1007/978-4-431-65868-9_16.

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Afanassieff, Marielle, Ronald M. Goto, Jennifer Ha, Rima Zoorob, Charles Auffray, Françoise Coudert, W. Elwood Briles, and Marcia M. Miller. "Are chicken Rfp-Y class I genes classical or non-classical?" In Major Histocompatibility Complex, 236–47. Tokyo: Springer Japan, 2000. http://dx.doi.org/10.1007/978-4-431-65868-9_17.

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Flajnik, Martin F., and Yuko Ohta. "Xenopus class I proteins." In Major Histocompatibility Complex, 248–59. Tokyo: Springer Japan, 2000. http://dx.doi.org/10.1007/978-4-431-65868-9_18.

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Conference papers on the topic "Histocompatibilty complex"

1

Qiu, Xuemei, Feng Zhang, Xiangying Meng, Yibing Zhou, and Xiuli Wang. "Evolution of the Major Histocompatibility Complex Genes in Fish." In 2009 3rd International Conference on Bioinformatics and Biomedical Engineering (iCBBE). IEEE, 2009. http://dx.doi.org/10.1109/icbbe.2009.5162809.

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Rowland, A., G. Levine, L. Schnabel, A. Berglund, D. Antczak, D. Miller, and Watts AE. "Allorecognition of Mesenchymal Stem Cells is Dependent on Major Histocompatibility Complex Haplotype." In Abstracts of the 47th Annual Conference of the Veterinary Orthopedic Society. Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1712896.

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Sroka, Ewa Maria, Rodrigo Prado Martins, Chrysoula Daskalogianni, Sebastien Apcher, and Robin Fahraeus. "Abstract B187: Origins of neoantigens for the major histocompatibility complex class I pathway." In Abstracts: Fourth CRI-CIMT-EATI-AACR International Cancer Immunotherapy Conference: Translating Science into Survival; September 30 - October 3, 2018; New York, NY. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/2326-6074.cricimteatiaacr18-b187.

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Wang, Jinbu, and Brian Y. Chen. "On Conformations of Peptides Bound to Class I Major Histocompatibility Complexes." In BCB '19: 10th ACM International Conference on Bioinformatics, Computational Biology and Health Informatics. New York, NY, USA: ACM, 2019. http://dx.doi.org/10.1145/3307339.3343868.

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Ford, Michael, Richard Jones, David Allen, Ravi Amunugama, Paul Del Rizzo, Michael Pisano, James Mobley, et al. "Abstract B80: Mass spectrometric characterization of peptides associated with molecules of the major histocompatibility complex." In Abstracts: AACR Special Conference on Tumor Immunology and Immunotherapy; October 1-4, 2017; Boston, MA. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/2326-6074.tumimm17-b80.

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Ford, Michael, Richard Jones, David Allen, Ravi Amunugama, Paul Del Rizzo, Michael Pisano, James Mobley, Paul Domanski, Bill Ho, and Daniel Bochar. "Abstract 1673: Mass spectrometric characterization of peptides associated with molecules of the major histocompatibility complex." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-1673.

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Knoche, Shelby M., and Joyce C. Solheim. "Abstract 1817: Epidermal growth factor receptor alters major histocompatibility complex class I expression on pancreatic cancer cells." In Proceedings: AACR Annual Meeting 2021; April 10-15, 2021 and May 17-21, 2021; Philadelphia, PA. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.am2021-1817.

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Bustos, MA, JIJ Orozco, MP Salomon, DSB Hoon, and DM Marzese. "Abstract P1-05-02: CRISPR/Cas9-guided editing of spliceosome factors enhances major histocompatibility complex proteins in triple-negative breast cancer." In Abstracts: 2017 San Antonio Breast Cancer Symposium; December 5-9, 2017; San Antonio, Texas. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.sabcs17-p1-05-02.

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Klimanov, Igor A., Svetlana Soodaeva, Timur Li, Nailya Kubysheva, Larisa Postnikova, Viktor Novikov, and Lidiya Nikitina. "Level of soluble molecules of major histocompatibility complex CLASS II in serum, sputum and exhaled breath condensate in COPD of patients with an exacerbation of COPD." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa885.

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Reports on the topic "Histocompatibilty complex"

1

Hammamieh, Rasha. Apoptosis Use Case: In Silico Evaluation of a Library of Small Molecule Pharmacophore Models for Blocking the Formation of SEB-Major Histocompatibility Class II Complexes. Fort Belvoir, VA: Defense Technical Information Center, April 2007. http://dx.doi.org/10.21236/ada482295.

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