Relevant bibliographies by topics / Histamine, brain, memory, feeding behavior

Academic literature on the topic 'Histamine, brain, memory, feeding behavior'

Author: Grafiati
Published: 10 March 2023

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Journal articles on the topic "Histamine, brain, memory, feeding behavior"

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Morimoto, T. "Brain histamine and feeding behavior." Behavioural Brain Research 124, no. 2 (October 15, 2001): 145–50. http://dx.doi.org/10.1016/s0166-4328(01)00225-x.

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Haas, Helmut L., Olga A. Sergeeva, and Oliver Selbach. "Histamine in the Nervous System." Physiological Reviews 88, no. 3 (July 2008): 1183–241. http://dx.doi.org/10.1152/physrev.00043.2007.

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Histamine is a transmitter in the nervous system and a signaling molecule in the gut, the skin, and the immune system. Histaminergic neurons in mammalian brain are located exclusively in the tuberomamillary nucleus of the posterior hypothalamus and send their axons all over the central nervous system. Active solely during waking, they maintain wakefulness and attention. Three of the four known histamine receptors and binding to glutamate NMDA receptors serve multiple functions in the brain, particularly control of excitability and plasticity. H1 and H2 receptor-mediated actions are mostly excitatory; H3 receptors act as inhibitory auto- and heteroreceptors. Mutual interactions with other transmitter systems form a network that links basic homeostatic and higher brain functions, including sleep-wake regulation, circadian and feeding rhythms, immunity, learning, and memory in health and disease.
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Köhler, Cristiano André, Weber Cláudio da Silva, Fernando Benetti, and Juliana Sartori Bonini. "Histaminergic Mechanisms for Modulation of Memory Systems." Neural Plasticity 2011 (2011): 1–16. http://dx.doi.org/10.1155/2011/328602.

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Encoding for several memory types requires neural changes and the activity of distinct regions across the brain. These areas receive broad projections originating in nuclei located in the brainstem which are capable of modulating the activity of a particular area. The histaminergic system is one of the major modulatory systems, and it regulates basic homeostatic and higher functions including arousal, circadian, and feeding rhythms, and cognition. There is now evidence that histamine can modulate learning in different types of behavioral tasks, but the exact course of modulation and its mechanisms are controversial. In the present paper we review the involvement of the histaminergic system and the effects histaminergic receptor agonists/antagonists have on the performance of tasks associated with the main memory types as well as evidence provided by studies with knockout models. Thus, we aim to summarize the possible effects histamine has on modulation of circuits involved in memory formation.
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Fukagawa, Koji, Hiroshi Etou, Kazuma Fujimoto, Kazuo Kurata, Kenji Terada, Yasufumi Okabe, and Toshiie Sakata. "Feeding behavior in response to histamine H1-receptor antagonist in the rat brain." Japanese Journal of Pharmacology 40 (1986): 175. http://dx.doi.org/10.1016/s0021-5198(19)59272-1.

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Gotoh, Koro, Takayuki Masaki, Seiichi Chiba, Hisae Ando, Kansuke Fujiwara, Takanobu Shimasaki, Kimihiko Mitsutomi, et al. "Brain-derived neurotrophic factor, corticotropin-releasing factor, and hypothalamic neuronal histamine interact to regulate feeding behavior." Journal of Neurochemistry 125, no. 4 (March 19, 2013): 588–98. http://dx.doi.org/10.1111/jnc.12213.

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Fülöp, András K., Anna Földes, Edit Buzás, Krisztina Hegyi, Ildikó H. Miklós, László Romics, Monika Kleiber, András Nagy, András Falus, and Krisztina J. Kovács. "Hyperleptinemia, Visceral Adiposity, and Decreased Glucose Tolerance in Mice with a Targeted Disruption of the Histidine Decarboxylase Gene." Endocrinology 144, no. 10 (October 1, 2003): 4306–14. http://dx.doi.org/10.1210/en.2003-0222.

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Histamine has been referred to as an anorexic factor that decreases appetite and fat accumulation and affects feeding behavior. Tuberomammillary histaminergic neurons have been implicated in central mediation of peripheral metabolic signals such as leptin, and centrally released histamine inhibits ob gene expression. Here we have characterized the metabolic phenotype of mice that completely lack the ability to produce histamine because of targeted disruption of the key enzyme in histamine biosynthesis (histidine decarboxylase, HDC). Histochemical analyses confirmed the lack of HDC mRNA, histamine immunoreactivity, and histaminergic innervation throughout the brain of gene knockout mouse. Aged histamine-deficient (HDC−/−) mice are characterized by visceral adiposity, increased amount of brown adipose tissue, impaired glucose tolerance, hyperinsulinemia, and hyperleptinemia. Histamine-deficient animals are not hyperphagic but gain more weight and are calorically more efficient than wild-type controls. These metabolic changes presumably are due to the impaired regulatory loop between leptin and hypothalamic histamine that results in orexigenic dominance through decreased energy expenditure, attenuated ability to induce uncoupling protein-1 mRNA in the brown adipose tissue and defect in mobilizing energy stores. Our results further support the role of histamine in regulation of energy homeostasis.
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Schmidt, Stefan, Miriam Richter, Dirk Montag, Tina Sartorius, Verena Gawlik, Anita M. Hennige, Stephan Scherneck, et al. "Neuronal functions, feeding behavior, and energy balance in Slc2a3+/− mice." American Journal of Physiology-Endocrinology and Metabolism 295, no. 5 (November 2008): E1084—E1094. http://dx.doi.org/10.1152/ajpendo.90491.2008.

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Homozygous deletion of the gene of the neuronal glucose transporter GLUT3 ( Slc2a3) in mice results in embryonic lethality, whereas heterozygotes ( Slc2a3+/−) are viable. Here, we describe the characterization of heterozygous mice with regard to neuronal function, glucose homeostasis, and, since GLUT3 might be a component of the neuronal glucose-sensing mechanism, food intake and energy balance. Levels of GLUT3 mRNA and protein in brain were reduced by 50% in Slc2a3+/− mice. Electrographic features examined by electroencephalographic recordings give evidence for slightly but significantly enhanced cerebrocortical activity in Slc2a3+/− mice. In addition, Slc2a3+/− mice were slightly more sensitive to an acoustic startle stimulus (elevated startle amplitude and reduced prepulse inhibition). However, systemic behavioral testing revealed no other functional abnormalities, e.g., in coordination, reflexes, motor abilities, anxiety, learning, and memory. Furthermore, no differences in body weight, blood glucose, and insulin levels were detected between wild-type and Slc2a3+/− littermates. Food intake as monitored randomly or after intracerebroventricular administration of 2-deoxyglucose or d-glucose, or food choice for carbohydrates/fat was not affected in Slc2a3+/− mice. Taken together, our data indicate that, in contrast to Slc2a1, a single allele of Slc2a3 is sufficient for maintenance of neuronal energy supply, motor abilities, learning and memory, and feeding behavior.
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Zhang, Ting, Min Jung Kim, Min Ju Kim, Xuangao Wu, Hye Jeong Yang, Heng Yuan, Shaokai Huang, Sun Myung Yoon, Keun-Nam Kim, and Sunmin Park. "Long-Term Effect of Porcine Brain Enzyme Hydrolysate Intake on Scopolamine-Induced Memory Impairment in Rats." International Journal of Molecular Sciences 23, no. 6 (March 20, 2022): 3361. http://dx.doi.org/10.3390/ijms23063361.

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No study has revealed the effect of porcine brain enzyme hydrolysate (PBEH) on memory impairment. We aimed to examine the hypothesis that PBEH intake modulates memory deficits and cognitive behavior in scopolamine (SC)-induced amnesia rats, and its mechanism, including gut microbiota changes, was determined. Sprague–Dawley male rats had intraperitoneal injections of SC (2 mg/kg body weight/day) at 30 min after daily feeding of casein (MD-control), PBEH (7 mg total nitrogen/mL) at 0.053 mL (Low-PBEH), 0.159 mL (Medium-PBEH), 0.478 mL (High-PBEH), or 10 mg donepezil (Positive-control) per kilogram body weight per day through a feeding needle for six weeks. The Normal-control rats had casein feeding without SC injection. PBEH dose-dependently protected against memory deficits determined by passive avoidance test, Y-maze, water-maze, and novel object recognition test in SC-induced rats compared to the MD-control. The High-PBEH group had a similar memory function to the Positive-control group. Systemic insulin resistance determined by HOMA-IR was lower in the PBEH groups than in the Normal-control but not the Positive-control. In parallel with systemic insulin resistance, decreased cholesterol and increased glycogen contents in the hippocampus in the Medium-PBEH and High-PBEH represented reduced brain insulin resistance. PBEH intake prevented the increment of serum TNF-α and IL-1β concentrations in the SC-injected rats. Hippocampal lipid peroxide and TNF-α contents and mRNA TNF-α and IL-1β expression were dose-dependently reduced in PBEH and Positive-control. PBEH decreased the hippocampal acetylcholinesterase activity compared to the MD-control, but not as much as the Positive-control. PBEH intake increased the α-diversity of the gut microbiota compared to the MD-control, and the gut microbiota community was separated from MD-control. In metagenome function analysis, PBEH increased the energy metabolism-related pathways of the gut microbiota, including citric acid cycle, oxidative phosphorylation, glycolysis, and amino acid metabolism, which were lower in the MD-control than the Normal-control. In conclusion, alleviated memory deficit by PBEH was associated potentially with not only reducing acetylcholinesterase activity but also improving brain insulin resistance and neuroinflammation potentially through modulating gut microbiota. PBEH intake (1.5–4.5 mL of 7 mg total nitrogen/mL for human equivalent) can be a potential therapeutic agent for improving memory impairment.
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MONGE-SANCHEZ, Diana, Marcelıno MONTİEL-HERRERA, Denısse GARCİA VİLLA, Guillermo LOPEZ, J. Abraham DOMÍNGUEZ-AVİLA, and Gustavo GONZÁLEZ-AGUİLAR. "Fasting alters p75NTR and AgRP mRNA expression in rat olfactory bulb and hippocampus." Journal of Cellular Neuroscience and Oxidative Stress 14, no. 2 (January 31, 2023): 1074–84. http://dx.doi.org/10.37212/jcnos.1168800.

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Classic non-homeostatic structures involved in food intake regulation are reciprocally influenced by metabolic signals. Orexigenic peptides expressed in the olfactory bulb (OB) and hippocampus (HP) modulate olfactory processing and memory, respectively. Hypothalamic circuits also modulate feeding behavior by activating and releasing Agouti-related peptide (AgRP) in response to orexigenic signals. An adequate response to fasting requires the expression of p75 neurotrophin receptor (p75NTR) in AgRP neurons. The present study aimed to determine whether there is a role for p75NTR and AgRP in the OB and HP on the feeding behavior of fasted rats. A group of fasted rats (FG) was confronted with a decision-making paradigm in a T-maze containing a standard chow pellet (CP), and the same pellet coated with a phenolic-rich avocado paste extract (AVO) on either end; their OB and HP were then analyzed with histological and molecular tools. FG rats had briefer feeding latencies, as compared to control rats fed ad libitum (median latencies: 55.4 vs 191.7 min, p = 0.032). They also had reduced cell counts in both brain structures, as compared to satiated rats. AgRP mRNA was not expressed in the HP of either group, however, it was found in the OB. p75NTR mRNA was expressed in both brain structures of FG rats. These results suggest that contrasting metabolic states (fasted or satiated) motivate different feeding responses, which are influenced by p75NTR and AgRP mRNA expression in non-homeostatic food intake brain structures.
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Nässel, Dick R. "Leucokinin and Associated Neuropeptides Regulate Multiple Aspects of Physiology and Behavior in Drosophila." International Journal of Molecular Sciences 22, no. 4 (February 16, 2021): 1940. http://dx.doi.org/10.3390/ijms22041940.

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Leucokinins (LKs) constitute a family of neuropeptides identified in numerous insects and many other invertebrates. LKs act on G-protein-coupled receptors that display only distant relations to other known receptors. In adult Drosophila, 26 neurons/neurosecretory cells of three main types express LK. The four brain interneurons are of two types, and these are implicated in several important functions in the fly’s behavior and physiology, including feeding, sleep–metabolism interactions, state-dependent memory formation, as well as modulation of gustatory sensitivity and nociception. The 22 neurosecretory cells (abdominal LK neurons, ABLKs) of the abdominal neuromeres co-express LK and a diuretic hormone (DH44), and together, these regulate water and ion homeostasis and associated stress as well as food intake. In Drosophila larvae, LK neurons modulate locomotion, escape responses and aspects of ecdysis behavior. A set of lateral neurosecretory cells, ALKs (anterior LK neurons), in the brain express LK in larvae, but inconsistently so in adults. These ALKs co-express three other neuropeptides and regulate water and ion homeostasis, feeding, and drinking, but the specific role of LK is not yet known. This review summarizes Drosophila data on embryonic lineages of LK neurons, functional roles of individual LK neuron types, interactions with other peptidergic systems, and orchestrating functions of LK.
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Dissertations / Theses on the topic "Histamine, brain, memory, feeding behavior"

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FABBRI, ROBERTA. "THE BRAIN HISTAMINERGIC SYSTEM AS A MODULATOR OF AVERSIVE MEMORY AND FEEDING BEHAVIOUR." Doctoral thesis, 2015. http://hdl.handle.net/2158/1026120.

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PROVENSI, GUSTAVO. "Differential modulation of the brain histaminergic system: effects on cognition and feeding behaviour." Doctoral thesis, 2014. http://hdl.handle.net/2158/854511.

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Book chapters on the topic "Histamine, brain, memory, feeding behavior"

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Komatsuzaki, Yoshimasa, Ayaka Itoh, and Minoru Saito. "Green Tea-Derived Catechins have Beneficial Effects on Cognition in the Pond Snail." In Basic and Applied Malacology [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.99789.

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Green tea has been used as a medicine in East Asia for thousands of years. Plant-derived compounds called flavanols, which are included in green tea, may have potentials to help maintain healthy brain function. In this chapter, we review the effects of flavanols, e.g. epicatechin (EpiC), on cognitive ability in the pond snail, Lymnaea stagnalis. In this decade, the Lukowiak’s group has tested the effects of EpiC on cognition ability in Lymnaea. In a Lymnaea model system, they showed that EpiC and EpiC-containing foods have a rapid and activity-dependent effect enhancing the formation of long-term memory (LTM) following operant conditioning of aerial respiratory behavior. In the last part of this chapter, we also introduce our study for the effects of EpiC on LTM formation in another model system in Lymnaea. This study showed that EpiC increases the persistence of LTM formed by classical conditioning of feeding behavior, and suggested that EpiC alters some electrophysiological properties of a neuron in the feeding system.
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