Relevant bibliographies by topics / Herpetiformi

Academic literature on the topic 'Herpetiformi'

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Published: 1 April 2023

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Journal articles on the topic "Herpetiformi"

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Fernandes, Iolanda Conde, Madalena Sanches, Rosário Alves, and Manuela Selores. "Case for diagnosis." Anais Brasileiros de Dermatologia 87, no. 6 (December 2012): 933–35. http://dx.doi.org/10.1590/s0365-05962012000600023.

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We report a clinical case of a rare variant of pemphigus - pemphigus herpetiformis - which combines the clinical features of dermatitis herpetiformis with the immunological findings of pemphigus. Due to its atypical presentation, it is frequently misdiagnosed as dermatitis herpetiformis. It is basically characterized by the herpetiform pattern of skin lesions, severe pruritus and by the presence of eosinophilic spongiosis confirmed on histopathology. We call attention to the excellent response to dapsone.
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Poór, Adrienn Katalin, Judit Hársing, Bernadett Hidvégi, Péter Holló, and Sarolta Kárpáti. "Impetigo herpetiformis." Bőrgyógyászati és Venerológiai Szemle 88, no. 4 (October 31, 2012): 117–20. http://dx.doi.org/10.7188/bvsz.2012.88.4.3.

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Miziara, Ivan Dieb, Bernardo Cunha Araujo Filho, and Raimar Weber. "Aids e estomatite aftóide recidivante." Revista Brasileira de Otorrinolaringologia 71, no. 4 (August 2005): 517–20. http://dx.doi.org/10.1590/s0034-72992005000400020.

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O estado de imunodeficiência em pacientes HIV positivos tem sido causa de episódios severos de Estomatite Aftóide Recidivante (EAR). OBJETIVO: Este estudo objetiva estabelecer evidências da relação entre o surgimento (ou agravamento) de EAR com o estado de imunossupressão causado pelo vírus HIV, por meio da contagem de células CD4+, CD8+ e da carga viral infectante. FORMA DE ESTUDO: estudo de série. MATERIAL E MÉTODO: Noventa e quatro pacientes HIV (1)-positivos (25 mulheres e 69 homens) com EAR foram acompanhados no ambulatório de Aids da Divisão de Clínica ORL do Hospital das Clínicas da FMUSP no período de janeiro de 1998 a dezembro de 2003. A idade dos pacientes variou de 19 a 63 anos (média = 35,3 anos). RESULTADO: Os pacientes com Aids e soropositivos apresentaram, respectivamente, oito aftas e duas aftas por surto. Da mesma maneira, os pacientes portadores de úlceras do tipo major apresentaram menor contagem de células CD8+, CD4+ e relação CD4+/CD8+ e maior valor médio da carga viral do que os pacientes portadores de aftas herpetiformes e minor. Entre os portadores de aftas minor e herpetiforme não houve diferença estatística. CONCLUSÕES O aparecimento das lesões, principalmente as do tipo major, está diretamente relacionado ao estado imunitário do paciente soropositivo, acarretando déficits nutricionais e piora na qualidade de vida. Desta forma, o diagnóstico e tratamento da EAR é um desafio que não deve ser desprezado.
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Nakajima, Kimiko. "Recent Advances in Dermatitis Herpetiformis." Clinical and Developmental Immunology 2012 (2012): 1–4. http://dx.doi.org/10.1155/2012/914162.

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Dermatitis herpetiformis is an autoimmune bullous disease that is associated with gluten sensitivity which typically presents as celiac disease. As both conditions are multifactorial disorders, it is not clear how specific pathogenetic mechanisms may lead to the dysregulation of immune responses in the skin and small bowel, respectively. Recent studies have demonstrated that IgA and antibodies against epidermal transglutaminase 3 play an important role in the pathogenesis of dermatitis herpetiformis. Here, we review recent immunopathological progress in understanding the pathogenesis of dermatitis herpetiformis.
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Ruvinskaya, Guzel R., E. N. Silantieva, and A. V. Anokhina. "The need for a multidisciplinary approach to the diagnosis and treatment procedures of isolated lesions of the oral mucosa in herpetiform Dühring dermatitis." Russian Journal of Dentistry 24, no. 2 (October 3, 2020): 99–103. http://dx.doi.org/10.17816/1728-2802-2020-24-2-99-103.

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The defeat of the oral mucosa in herpetiform Dhring dermatitis is a rare pathology. According to the literature, it is detected in 9.6% 10% of cases. Granular deposits of immunoglobulin A in the papillae of the dermis in herpetiform Dhring dermatitis are associated with gluten enteropathy, lead to the development of inflammatory processes on the skin and oral mucosa and the appearance of polymorphic itchy rashes and chronic recurrent course of the disease. The article describes a clinical case of a patient diagnosed with herpetiform dermatitis, manifestations in the oral cavity (ICD-10 L13.0X code). The need for a multidisciplinary approach to the diagnosis, differential diagnosis, treatment and dynamic observation of isolated lesions of the oral mucosa in herpetiform Dhring dermatitis is indicated. A scheme of measures required by a dentist is developed.
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Novikov, Yuri A., Denis V. Zaslavsky, Olga V. Pravdina, Elena A. Zykova, Anastasia S. Lipatnikova, Elena S. Bolshakova, Elena S. Manylova, and Lyudmila N. Drozdova. "During’s herpetiform dermatitis in pediatric dermatology: issues of diagnostics and treatment." Pediatrician (St. Petersburg) 11, no. 6 (December 31, 2020): 79–86. http://dx.doi.org/10.17816/ped11679-86.

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This article presents a case of clinical observation of a 5-year-old child with herpetiformis dermatitis (Duhrings). This rare dermatosis is characterized by a chronic relapsing course, the presence of itching polymorphic rashes, typical histological and immunomorphological signs. The diagnosis was made on the basis of the clinical picture, histological and immunohistochemical studies of skin biopsy, as well as the results of HLA typing by PCR. Clinical observation of this case is of interest to practicing physicians-dermatologists due to the rare occurrence of Duhrings herpetiformis in children, the complexity of differential diagnostic search, which requires further generalization of experience using histological, immunohistochemical and molecular genetic research methods. The disease is clearly differentiated from other rashes with the formation of subepidermal blisters according to histological, immunological and gastrointestinal criteria. The prevalence of dermatosis in various populations of the Europian race ranges from 10 to 39 cases per 100,000 population. Duhrings dermatitis herpetiformis can develop at any age (cases of the childhood form of Dhrings dermatitis herpetiformis have been reported), but most often the disease occurs at the age of 4050 years. Dermatitis herpetiformis persists indefinitely with variable severity. In patients with Duhrings dermatitis, associated gluten-sensitive enteropathy is often noted, which in most cases is asymptomatic.
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Villegas de la Lama, Juan Carlos, and Marina Lacalle-Calderón. "Mujer de 45 años con lesiones digitales pruriginosas." Revista Española de Casos Clínicos en Medicina Interna 7, no. 2 (August 9, 2022): 9–11. http://dx.doi.org/10.32818/reccmi.a7n2a4.

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La dermatitis herpetiforme es una erupción cutánea autoinmune infrecuente que pone de manifiesto una sensibilidad al gluten. Los pacientes desarrollan típicamente unas pápulas y vesículas inflamatorias intensamente pruriginosas en los antebrazos, las rodillas, el cuero cabelludo o los glúteos. La inmensa mayoría de los pacientes con dermatitis herpetiforme también asocia una enteropatía sensible al gluten (enfermedad celíaca), que normalmente es oligosintomática o asintomática. Este caso clínico es una presentación atípica de dermatitis herpetiforme, dado que la primera manifestación cutánea de nuestra paciente fue una combinación de máculas purpúricas, pápulas y petequias dolorosas localizadas en regiones inusuales: los pulpejos de los dedos.
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Klimov, L. Ya, V. A. Kuryaninova, Yu A. Dmitrieva, Ya D. Mironova, A. V. Yagupova, S. V. Dolbnya, M. V. Stoyan, S. N. Kashnikova, T. A. Ivenskaya, and E. A. Cherkasova. "Dermatitis herpetiformis Duhring as one of the forms of gluten-associated pathology: a review of the literature and a description of a clinical case." Meditsinskiy sovet = Medical Council, no. 1 (March 5, 2022): 301–11. http://dx.doi.org/10.21518/2079-701x-2022-16-1-301-311.

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This review presents information on the prevalence, pathogenesis, clinical manifestations, diagnosis and treatment of Duhring’s dermatitis herpetiformis. Although the disease was first clinically described in 1884 by the American dermatologist L.A. Duhring, the study of its pathogenesis and the search for prognostic markers of its occurrence continue. Against the background of a significant expansion of ideas about gluten-dependent diseases and conditions, views on the mechanisms of autoimmune skin damage in dermatitis herpetiformis are detailed. A strong association with hereditary predisposition through the major human leukocyte histocompatibility complex (HLA) DQ2 and DQ8, and a role of epidermal transglutaminase as a major autoantigen in dermatitis herpetiformis are shown. The hypotheses explaining the decline in the incidence of dermatitis herpetiformis in recent decades against the background of increased and more effective serological screening and the resulting earlier diagnosis of celiac disease are commented on. A typical clinical picture of dermatitis herpetiformis, in which erythematous papules, plaques, vesicles are seen, usually clustered on the flexural surfaces of the extremities. Secondary elements are erosions, excoriations and crusts due to rupture of blisters and due to scratching caused by intense itching. A generally favourable prognosis for life and disease is shown with a gluten-free diet and the use of dapsone, glucocorticoids and, if these are ineffective, immunosuppressants. The authors describe a clinical case of the disease in an adolescent girl with a typical clinical history and characteristic rashes on the extensor surfaces of the limbs. The authors show that drug therapy without a gluten-free diet cannot be considered effective, and that the diet for dermatitis herpetiformis, like that for celiac disease, is lifelong. The growing understanding of gluten-associated pathology, which includes dermatitis herpetiformis, in recent decades has led to an intensive search for diagnostic and prognostic markers, as well as the development of ways to correct this group of diseases, including those not related to the lifelong elimination of cereal prolamines.
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Sárdy, Miklós, Sarolta Kárpáti, Barbara Merkl, Mats Paulsson, and Neil Smyth. "Epidermal Transglutaminase (TGase 3) Is the Autoantigen of Dermatitis Herpetiformis." Journal of Experimental Medicine 195, no. 6 (March 18, 2002): 747–57. http://dx.doi.org/10.1084/jem.20011299.

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Gluten sensitivity typically presents as celiac disease, a common chronic small intestinal disorder. However, in certain individuals it is associated with dermatitis herpetiformis, a blistering skin disease characterized by granular IgA deposits in the papillary dermis. While tissue transglutaminase has been implicated as the major autoantigen of gluten sensitive disease, there has been no explanation as to why this condition appears in two distinct forms. Here we show that while sera from patients with either form of gluten sensitive disease react both with tissue transglutaminase and the related enzyme epidermal (type 3) transglutaminase, antibodies in patients having dermatitis herpetiformis show a markedly higher avidity for epidermal transglutaminase. Further, these patients have an antibody population specific for this enzyme. We also show that the IgA precipitates in the papillary dermis of patients with dermatitis herpetiformis, the defining signs of the disease, contain epidermal transglutaminase, but not tissue transglutaminase or keratinocyte transglutaminase. These findings demonstrate that epidermal transglutaminase, rather than tissue transglutaminase, is the dominant autoantigen in dermatitis herpetiformis and explain why skin symptoms appear in a proportion of patients having gluten sensitive disease.
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Cinats, Allison K., Laurie M. Parsons, and Richard M. Haber. "Facial Involvement in Dermatitis Herpetiformis: A Case Report and Review of the Literature." Journal of Cutaneous Medicine and Surgery 23, no. 1 (August 13, 2018): 35–37. http://dx.doi.org/10.1177/1203475418795818.

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Dermatitis herpetiformis is a cutaneous manifestation of celiac disease that classically presents as a symmetric pruritic vesicular eruption on extensor surfaces. Typical locations include elbows, knees, and buttocks. Facial involvement has been reported rarely. Here, we report a case of a 44-year-old woman with dermatitis herpetiformis presenting as pruritic vesicles on the face that had previously been misdiagnosed as allergic contact dermatitis. Diagnosis was confirmed with direct immunofluorescence demonstrating granular IgA in the papillary dermis. This eruption cleared with topical dapsone 5% gel and a gluten-free diet. We report this case to raise awareness of facial involvement in dermatitis herpetiformis as well as the possibility of topical dapsone as a therapeutic option.
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Dissertations / Theses on the topic "Herpetiformi"

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ROUE, ISABELLE. "Dermatite herpetiforme : a propos d'un cas ; analyse critique de la litterature." Lille 2, 1992. http://www.theses.fr/1992LIL2M277.

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Turner, John Barrie. "An investigation of the structure of gliadins and its implications in gluten enteropathy." Thesis, Manchester Metropolitan University, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.337275.

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Rocha, Souto Luciana. "Frequência de Auto-anticorpos tireoideanos em pacientes com dermatite herpetiforme." Universidade Federal de Pernambuco, 2003. https://repositorio.ufpe.br/handle/123456789/7384.

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Made available in DSpace on 2014-06-12T18:31:52Z (GMT). No. of bitstreams: 2 arquivo8080_1.pdf: 168508 bytes, checksum: 987b692e6b7f2fdbcdd2fa53acd024a7 (MD5) license.txt: 1748 bytes, checksum: 8a4605be74aa9ea9d79846c1fba20a33 (MD5) Previous issue date: 2003
O objetivo deste estudo foi determinar a freqüência de anticorpos antitireoglobulina e antimicrossomais e realizar a dosagem sérica dos hormônios tiroideanos em 41 pacientes com Dermatite Herpetiforme e 120 pacientes do grupo controle. O desenho do estudo foi o comparativo entre um grupo de casos (portadores de D.H.) e um grupo controle. Foram estudados retrospectivamente 41 pacientes do ambulatório de Dermatologia do Hospital das Clínicas da Universidade Federal de Pernambuco entre Janeiro de 1990 e Dezembro de 2000 que preencheram os critérios para o diagnóstico (características clínicas e hispopatológicas). Os níveis de TSH, T3, T4 e a presença de anticorpos antimicrossomais e antitireoglobulina de acordo com o sexo e a idade forma avaliados em ambos os grupos. No grupo de pacientes com D.H. a idade média foi de 34 anos com mínima de 21 e máxima de 51 anos, sendo 60% dos pacientes do sexo feminino. A freqüência de anticorpos antitireoglobulina utilizando o método de hemaglutinação foi de 7,3% em pacientes com D.H e em 1,7% no grupo controle. A presença de anticorpos antimicrossomais foi detectada em 36% dos pacientes com D.H. e em 11,7% no grupo controle, esta diferença foi estatisticamente significativa (p<0,000). Dois pacientes com hipotiroidismo e um com hipertiroidismo foram diagnosticados no grupo com D.H., e no controle foi detectado um caso de hipertiroidismo e outro de hipotiroidismo. Estes resultados demonstram a occorrência freqüente de anticorpos antitiroideanos em pacientes com Dermatite Herpetiforme, embora a falência tiroideana seja menos comumente associada a esta condição
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Varpuluoma, O. (Outi). "Drugs, dermatitis herpetiformis and celiac disease as risk factors for bullous pemphigoid in Finland." Doctoral thesis, Oulun yliopisto, 2019. http://urn.fi/urn:isbn:9789526221922.

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Abstract Bullous pemphigoid (BP) is the most common autoimmune blistering disease. It mostly affects elderly patients and is characterized by intense pruritus and blistering or bullae. Treatment options include topical and systemic corticosteroids, other immunosuppressive drugs and doxycycline. Disease course may be chronic and relapses are common. The incidence of BP has been reported to have increased in the last few decades, but the reason for this trend is not known. The aim of this thesis was to study the risk factors of BP. Firstly, the influence of the use of dipeptidyl peptidase (DPP-4) inhibitors was analyzed as a risk factor, and then those of other oral diabetes medications. This study also aimed to determine whether drugs used for psychiatric and neurologic conditions are risk factors for BP. Finally, previously diagnosed dermatitis herpetiformis (DH) and celiac disease (CD) were examined as potential risk factors for subsequent BP. For this retrospective, matched case-control study, patient data were obtained from the Finnish Care Register for Health Care database, and data on reimbursed drugs from the Social Insurance Institution of Finland. In the present study, prior use of DPP-4 inhibitors was found to increase the risk of BP twofold and in particular, vildagliptin increased the risk tenfold. The mean time between the initiation of vildagliptin and diagnosis of BP was 449 days. Metformin and other conventional diabetes drugs were not risk factors for BP. Several drugs used for neurological and psychiatric diseases were associated with an elevated risk for BP, but no pharmacological or chemical properties of these drugs emerged as candidates to explain the increased risk. A prior diagnosis of DH increased the risk of BP 22-fold and a diagnosis of CD doubled it. Dapsone had been used in the two years before BP diagnosis by 44% of patients whose BP was preceded by DH. The mean time between the diagnoses of DH and BP was 3.3 years. This study confirms the view that DPP-4 inhibitors increase the risk for BP. No such association was found with other classes of diabetes drugs and therefore their use can be continued following a diagnosis of BP. Doctors treating patients with DH should be aware of the association between DH and BP, and be particularly vigilant if a DH patient’s skin symptoms change or become unresponsive to a gluten-free diet and/or dapsone
Tiivistelmä Rakkulainen pemfigoidi (pemfigoidi) on yleisin ihon autoimmuunirakkulatauti. Pemfigoidi on pääasiassa ikääntyneiden sairaus, ja sen tyypillisiä oireita ovat kova kutina ja rakkulat iholla. Pemfigoidin hoitoon käytetään paikallisia ja systeemisiä kortikosteroideja, muita immunosuppressiivisia lääkkeitä sekä doksisykliiniä. Taudinkulku on usein krooninen ja uusiutumiset ovat yleisiä. Rakkulaisen pemfigoidin ilmaantuvuuden on raportoitu lisääntyneen, mutta syitä tähän muutokseen ei täysin ymmärretä. Tämän tutkimuksen tavoite oli tutkia pemfigoidin riskitekijöitä Suomessa. Retrospektiivisessä tapaus-verrokkitutkimuksessa käytettiin aineistona Terveyden ja hyvinvoinnin laitoksen hoitoilmoitusrekisteristä poimittuja pemfigoidipotilaita (N=3397) ja verrokkeina ihon tyvisolusyöpäpotilaita (N=12941). Tiedot korvatuista lääkeostoista saatiin Kelan lääkekorvausrekisteristä. Tutkimuksessa todettiin DPP-4:n salpaajien kaksinkertaistavan pemfigoidin riskin ja DPP-4:n salpaaja vildagliptiini lisäsi riskiä jopa kymmenkertaiseksi. Vildagliptiinin aloituksen ja pemfigoidin toteamisen välillä kului keskimäärin 449 vuorokautta. Metformiini ja muut tutkitut suun kautta otettavat diabeteslääkkeet eivät lisänneet pemfigoidin riskiä. Useiden psykiatrisiin ja neurologisiin sairauksiin käytettävien lääkkeiden todettiin lisäävän pemfigoidin riskiä. Pemfigoidin on kuvattu voivan puhjeta ihokeliakian jälkeen, mutta laajempia tutkimuksia näiden sairauksien yhteydestä ei oltu aiemmin tehty. Tämän vuoksi samassa potilasaineistossa tutkittiin ihokeliakiaa ja keliakiaa pemfigoidin riskitekijöinä. Edeltävä ihokeliakia lisäsi pemfigoidin toteamisen riskiä selvästi, jopa 22-kertaiseksi ja keliakia kaksikertaiseksi. Huomattava osa potilaista oli ostanut ihokeliakian hoitoon käytettävää dapsonia edeltävän 2 vuoden aikana ennen pemfigoidin toteamista, mikä voi kertoa ihokeliakian oireiden aktiivisuudesta. Tämä tutkimus vahvistaa näkemystä siitä, että DPP-4:n salpaajat ovat pemfigoidin riskitekijä. Muut tutkitut diabeteslääkkeet eivät lisänneet riskiä ja voidaan ajatella, että ne eivät edelleen hankaloita aiemmin todetun pemfigoidin oireita. Koska ihokeliakian todettiin olevan pemfigoidin riskitekijä, tulee näitä potilaita hoitavan lääkärin muistaa pemfigoidin mahdollisuus, jos ihokeliakian oireet muuttuvat tai hoitovaste menetetään
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Millar, Baxter W. "An investigation of the significance of polymorphonuclear leukocyte in vitro chemotaxis in dermatitis herpetiformis and psoriasis." Thesis, University of Strathclyde, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.291994.

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Karell, Kati. "Dissecting genetic susceptibility to gluten sensitivity : HLA-linked risk factors in coeliac disease and dermatitis herpetiformis." Helsinki : University of Helsinki, 2003. http://ethesis.helsinki.fi/julkaisut/mat/bioti/vk/karell/.

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Leivo, T. (Tomi). "Basement membrane zone proteins, epithelial integrins and TGF-β system in reepithelialization, dermatitis herpetiformis and psoriasis:modulation by isotretinoin, betamethasone and calcipotriol." Doctoral thesis, University of Oulu, 2000. http://urn.fi/urn:isbn:951425712X.

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Abstract TGF-βs are cytokines that signal through the receptor complex of type I and type II receptors. Hemidesmosome (BP180, BP230, plectin/HD1, α6β4 integrin), anchoring filaments (laminin 5), and anchoring fibrils (collagen VII) form a hemidesmosomal adhesion complex that provides stable adherence of keratinocytes to the epidermal basement membrane. Nidogen, collagen IV, and laminins are components of the basement membrane, integrins are cell adhesion molecules, and tenascin-C is a matrix protein. The expression of TGF-β receptors I and II was studied in normal epidermis and lesional and non-lesional psoriatic epidermis by immunohistochemistry. TGF-β1 and TGF-β2 in suction blister fluid and serum were determined by enzyme-linked immunoassay. Suction blister fluid and serum samples were obtained from acne patients before and after oral isotretinoin treatment. Suction blister fluid samples were also obtained from healthy volunteers in two age groups from a control site and a betamethasone-pretreated site. The expression of BP180, BP230, plectin/HD1, α6 integrin, β4 integrin, laminin 5, collagen VII, collagen IV, nidogen, laminin α3 chain, and laminin β1g1 chains was studied in uninvolved dermatitis herpetiformis skin by the immunofluorescence technique. The ultrastructure of the hemidesmosomal inner plaque was studied in uninvolved dermatitis herpetiformis skin by electron microscopy. The suction blister method was used to study intact blisters, open wounds (=blister roofs removed right after blister induction) and calcipotriol-pretreated open wounds in healthy volunteers. The reepithelialization rate and the expression of BP180, BP230, plectin/HD1, β4 integrin, laminin 5, collagen VII, laminin α5 chain, laminin β1 chain, tenascin-C, αvβ5 integrin, β5 integrin, α5 integrin, and α9 integrin during reepithelialization were studied by haematoxylin and eosin stainings and the immunofluorescence technique. BP180, BP230, and plectin/HD1 expression were analyzed by body site to exclude regional variation. In normal epidermis, TGF-β receptors I and II were detected in the basal epidermis. Diffusion calculations suggest that circulation is likely to be a major source of TGF-β for TGF-β receptors in the basal epidermis. Downregulation of TGF-β receptors I and II was seen in lesional psoriatic epidermis, suggesting that hyperproliferating lesional epidermis may have lost TGF-β-mediated growth inhibition. Isotretinoin did not affect the serum TGF-β1 or TGF-β2 levels, but caused a 19% local increase in suction blister fluid TGF-β1. Betamethasone caused a 17% decrease in suction blister fluid TGF-β1, presumably due to glucocorticoid-induced vasoconstriction. Modulation of the interstitial fluid TGF-β1 concentration may be one mechanism by which isotretinoin and betamethasone mediate their effects in skin. Immunoreactivity for BP230 and plectin/HD1 was decreased in the basement membrane zone in uninvolved dermatitis herpetiformis skin in a significant proportion of the patients, suggesting distinct molecular changes in BP230 and plectin/HD1. This may be a factor contributing to blister formation. Reepithelialization rate was considerably slower in intact blisters than in open wounds and was not affected by calcipotriol. BP230 and plectin/HD1 appeared earlier in intact blisters than in open wounds. Reepithelialization took place on a continuous laminin sheath in intact blisters, but the laminin sheath in open wounds was partially discontinuous. It was a novel finding that integrin αvβ5 and integrin β5 antibodies showed divergent distributions in regenerating epidermis. The present results suggest that, in some bullous diseases, removal of the blister roof could accelerate blister healing, calcipotriol treatment does not delay wound epithelialization, a continuous laminin sheath may inhibit reepithelialization, and the formation of the hemidesmosomal inner plaque at the leading edge takes place earlier in the more slowly reepithelializing intact blisters than in open wounds.
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Meneses, Mariana Marques Moura Cardoso. "Dermatoses bolhosas auto-imunes na idade pediátrica- Estudo retrospetivo." Master's thesis, 2018. http://hdl.handle.net/10316/82798.

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Abstract:
Trabalho Final do Mestrado Integrado em Medicina apresentado à Faculdade de Medicina
Introdução: As dermatoses bolhosas na infância, embora sejam pouco prevalentes, apresentam elevada morbilidade, com impacto significativo na qualidade de vida. Dividem-se em dois grandes grupos: adquiridas e hereditárias. Serão alvo deste estudo as formas auto-imunes, nomeadamente a dermatite herpetiforme, o penfigóide bolhoso e a dermatose IgA linear. Estas relacionam-se com a ação de auto-anticorpos contra componentes das proteínas de adesão intercelular e célula-matriz, resultando num conjunto de alterações cuja manifestação cutânea primária e fundamental consiste no aparecimento de vesículas e bolhas.Objetivo: Analisar a casuística do Centro Hospitalar e Universitário de Coimbra e compará-la com os dados publicados na literatura médica, em termos de idade, formas de apresentação, exames complementares de diagnóstico, tratamento e evolução/prognóstico. Materiais e métodos: Estudo retrospetivo de doentes com diagnóstico de dermatoses bolhosas auto-imunes (DBAIs), que foram seguidos no Serviço de Dermatologia e Venereologia do Centro Hospitalar e Universitário de Coimbra no período de 2007-2017. Foram consultadas bases de dados (PubMed, Índex, b-on e web of science), assim como o livro “Dermatology-volume 2” de Bolognia JL et al.. A informação obtida foi posteriormente analisada e comparada.Resultados: Dos 6 doentes com DBAIs, 4 (66,6%) foram diagnosticados com penfigóide bolhoso (PB), 1 (16,6%) com dermatite herpetiforme (DH) e 1 (16,66%) com dermatose IgA linear (LABD). Verificou-se um predomínio pelo sexo masculino, tendo sido diagnosticadas, na maioria, na idade pré-escolar. À exceção do doente com DH, todos realizaram terapêutica farmacológica com agentes sistémicos, com remissão das lesões após um período de 12±10 meses. Conclusão: De acordo com estudos anteriores, a LABD é a dermatose bolhosa auto-imune mais frequente na idade pediátrica. O estudo da população pediátrica do Centro Hospitalar e Universitário de Coimbra demonstrou predominância do PB. Todos os doentes envolvidos apresentaram boa evolução clínica, após a implementação de terapêutica, reforçando o bom prognóstico destas patologias nesta faixa etária. Estudos adicionais serão necessários para uma melhor compreensão da prevalência e características clínicas destas patologias na população pediátrica do nosso País.
Introduction: Bullous dermatosis in childhood, although not very prevalent, present high morbidity, with a significant impact on the quality of life. They are divided into two main groups: acquired and hereditary. This study will focus on autoimmune forms, namely dermatitis herpetiformis, bullous pemphigoid and linear IgA dermatosis. These are related to the action of autoantibodies against intercellular and cell-matrix adhesion proteins, resulting in a set of alterations whose primary and fundamental cutaneous manifestation consists of vesicles and blisters.Objective: To compare the casuistry of the Dermatology Department of Centro Hospitalar e Universitário de Coimbra with the data published in the medical literature, in terms of age, forms of presentation, complementary diagnostic tests, treatment and evolution/prognosis.Materials and methods: 6 patients were diagnosed with DBAIs and followed at the Dermatology and Venereology Department of the Centro Hospitalar e Universitário de Coimbra. Databases such as PubMed, Index, b-on and web of science were consulted, as well as the book “Dermatology-volume 2” by Bolognia JL et al. The information obtained was subsequently analysed by comparing variables such as age of presentation, clinical manifestations, complementary diagnostic tests, treatment and evolution/prognosis.Results: Of the 6 patients with DBAIs, 4 (66.6%) were diagnosed with bullous pemphigoid (PB), 1 (16.6%) with dermatitis herpetiformis (DH) and 1 (16,66) with linear IgA dermatosis. In all dermatosis, a predominance was found in males, and most of them were diagnosed at preschool age. Almost all patients were treated with systemic agents, which led to clinical remission after a period of 12 ± 10 months. Conclusion: According to previous studies, LABD is the most common autoimmune bullous dermatosis in pediatric age. The study of the pediatric population of the Centro Hospitalar e Universitário de Coimbra showed a predominance of PB. All the patients involved in this study showed a good evolution after the treatment, reinforcing the good prognosis of these diseases. However, more studies are needed to better understand the prevalence and clinical characteristics of these pathologies in the pediatric population of our country.
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Books on the topic "Herpetiformi"

1

Society, Coeliac. Dermatitis herpetiformis. High Wycombe: Coeliac Society, 1992.

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Ashe, Siobhan Nanette Patricia. Ig A-class endomysial antibodies and their demonstration in dermatitis herpetiformis. [S.l: The Author], 1991.

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Ruth, Rolph, ed. The gluten-free diet book: A guide to celiac sprue, dermatitis herpetiformis, and gluten-free cookery. New York: Arco Pub., 1985.

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Rawcliffe, Peter. The Gluten-free diet book: A guide to coeliac disease, dermatitis herpetiformis and gluten-free cookery. London: Optima, 1990.

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Rawcliffe, Peter. The gluten-free diet book: A guide to coeliac disease, dermatitis, herpetiformis and gluten-free cookery. Cape Town: Timmins, 1985.

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Rawcliffe, Peter. The gluten-free diet book: A guide to coeliac disease, dermatitis herpetiformis and gluten-free cookery. London: Martin Dunitz, 1985.

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Coeliac Society of the United Kingdom., ed. The food list: A list of gluten-free manufactured products produced to help those who have been medically diagnosed as having Coeliac Disease or Dermatitis Herpetiformis. High Wycombe: The Coeliac Society, 1998.

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Dermantitis Herpetiformis: Detailed Guide on Dermatitis Herpetiformis, Its Signs and Symptoms and Remedies. Independently Published, 2022.

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White, Camille. Dermatitis Herpetiformis Cure Manual: Guide to Understand How to Treat, Prevent, Strive and Reverse Dermatitis Herpetiformis Completely. Independently Published, 2022.

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Gerry, Rolland. Coloring Book - You Will Get Better - Dermatitis Herpetiformis. Independently Published, 2021.

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Book chapters on the topic "Herpetiformi"

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Gloster, Hugh Morris, Lauren E. Gebauer, and Rachel L. Mistur. "Dermatitis Herpetiformis." In Absolute Dermatology Review, 77–79. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-03218-4_25.

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Patton, Timothy, and Neil J. Korman. "Dermatitis Herpetiformis." In Autoimmune Bullous Diseases, 163–73. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-26728-9_10.

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Laws, Phillip, and Neil H. Shear. "Pemphigus Herpetiformis." In Autoimmune Bullous Diseases, 41–55. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-26728-9_3.

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Jukic, Ines Lakos, and Branka Marinović. "Dermatitis Herpetiformis." In European Handbook of Dermatological Treatments, 189–97. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-662-45139-7_19.

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Fry, Lionel. "Dermatitis herpetiformis." In Management of Blistering Diseases, 139–60. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4899-7190-6_11.

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Norman, Robert A., and Edward M. Young. "Dermatitis Herpetiformis." In Atlas of Geriatric Dermatology, 43–46. London: Springer London, 2013. http://dx.doi.org/10.1007/978-1-4471-4579-0_5.

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Fry, Lionel, Fenella T. Wojnarowska, and Parvin Shahrad. "Dermatitis Herpetiformis." In Illustrated Encyclopedia of Dermatology, 29–39. Dordrecht: Springer Netherlands, 1985. http://dx.doi.org/10.1007/978-94-010-9390-3_6.

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Leonard, Jonathan N. "Dermatitis Herpetiformis." In Harper's Textbook of Pediatric Dermatology, 90.1–90.8. Oxford, UK: Wiley-Blackwell, 2011. http://dx.doi.org/10.1002/9781444345384.ch90.

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Kárpáti, Sarolta. "Dermatitis Herpetiformis." In Clinical Cases in Autoimmune Blistering Diseases, 113–17. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-10148-4_15.

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Salmi, Teea, and Kaisa Hervonen. "Dermatitis Herpetiformis." In Diagnosis and Management of Gluten-Associated Disorders, 17–25. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-56722-4_2.

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Conference papers on the topic "Herpetiformi"

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Hannusch, D. C., J. L. Mendonça, G. S. Rizzo, F. S. Rossi, and L. M. Lopes. "Dermatite Herpetiforme: Relato De Caso E Revisão Da Literatura." In II Congresso Brasileiro de Medicina Hospitalar. São Paulo: Editora Edgard Blücher, 2014. http://dx.doi.org/10.5151/medpro-ii-cbmh-043.

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