Log in

Relevant bibliographies by topics / Herpes virus 8 / Journal articles

To see the other types of publications on this topic, follow the link: Herpes virus 8.

Journal articles on the topic 'Herpes virus 8'

Author: Grafiati

Published: 4 June 2021

Last updated: 20 February 2023

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 50 journal articles for your research on the topic 'Herpes virus 8.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse journal articles on a wide variety of disciplines and organise your bibliography correctly.

1

Landy, Helain J., and John H. Grossman. "Herpes Simplex Virus." Obstetrics and Gynecology Clinics of North America 16, no. 3 (September 1989): 495–515. http://dx.doi.org/10.1016/s0889-8545(21)00405-8.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Meignin, Véronique, and Lionel Galicier. "Hémopathies lymphoïdes et HHV-8 (human herpes virus 8)." Annales de Pathologie 29, no. 5 (October 2009): 376–82. http://dx.doi.org/10.1016/j.annpat.2009.09.006.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Porter, S. R., L. Di Alberti, and N. Kumar. "Human herpes virus 8 (Kaposi’s sarcoma herpesvirus)." Oral Oncology 34, no. 1 (January 1998): 5–14. http://dx.doi.org/10.1016/s1368-8375(97)00038-9.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Rodríguez Arrieta, Luis Antonio, Gener Mahaht Rada, Luis Antonio Salazar, and Rita Magola Sierra. "Sarcoma de Kaposi y enfermedad de Castleman multicéntrica VHH-8 positivo - VIH Negativo." Acta Médica Colombiana 43, no. 1S (May 20, 2019): 30–32. http://dx.doi.org/10.36104/amc.2018.1357.

Full text

Abstract:

El sarcoma de Kaposi y la enfermedad de Castleman multicéntrica/virus herpes humano-8 positivo/virus de la inmunodeficiencia humana negativo representan un subgrupo etiológico dentro del espectro clínico de las enfermedades asociadas al virus herpes humano-8 positivo, inicialmente fueron consideradas dos entidades aisladas. El virus tiene un papel protagónico por su tropismo por el tejido linfoide y su potencial oncogénico. La presencia de sarcoma de Kaposi y enfermedad de Castleman multicéntrica/virus herpes humano-8 (VHH-8) positivo/virus de la inmunodeficiencia humana (VIH) negativo sugiere la posibilidad de una deficiencia inmunológica subyacente. Se presenta un caso de una mujer de 67 años de edad con dermatosis en extremidades de curso indolente y aparición posterior de múltiples adenomegalias, esplenomegalia además síntomas sistémicos. Se destaca el abordaje diagnóstico, la coexistencia de dos enfermedades etiológicamente ligadas al virus herpes humano-8, sin asociación de inmunosupresión

APA, Harvard, Vancouver, ISO, and other styles

5

Cathomas, G. "Human herpes virus 8: a new virus discloses its face." Virchows Archiv 436, no. 3 (March 10, 2000): 195–206. http://dx.doi.org/10.1007/s004280050031.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Díaz-García, Juan Daniel, Juan Pablo Venzor-Castellanos, Karen Hopf-Estandia, and Eunice Rojas-Zaldívar. "PLasmablastic lymphoma with coinfection by Epstein Barr Virus and Herpes Human Virus 8: a case report." ACTUALIDAD MEDICA 104, no. 808 (December 31, 2019): 188–90. http://dx.doi.org/10.15568/am.2019.808.cc03.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Marlina, Erni, Ali Yusran, and Zohra Nazaruddin. "Diagnosis dan tatalaksana nyeri pada rongga mulut yang disebabkan oleh infeksi virus herpes Diagnosis and management of pain in oral cavity caused by herpes virus infection." Journal of Dentomaxillofacial Science 11, no. 1 (February 28, 2012): 33. http://dx.doi.org/10.15562/jdmfs.v11i1.291.

Full text

Abstract:

There are 80 types of known herpes virus, 8 of them can cause infection on humans. They are herpes simplex virus(HSV) 1 and 2, varicella zoster virus (VZV), cytomegalovirus, Epstein-Barr virus, human herpes virus (HHV6) Aand B, and paramyxovirus. HSV1, HSV2, and VZV are the virus known to cause oral mucosal diseases. This paperaims to review and discuss orofacial pain caused by herpes virus infection. Detail anamnesis about prodromal signand symptom with clinical features that vesicles, labial and intraoral lesions, and unilateral distribution of lesionsare characterized oral herpes virus infections. It can be concluded that detailed anamnesis and an understandingabout oral clinical sign and symptom may confirm diagnosis of herpes virus infections.

APA, Harvard, Vancouver, ISO, and other styles

8

Ariza-Heredia, Ella J., and Raymund R. Razonable. "Human Herpes Virus 8 in Solid Organ Transplantation." Transplantation 92, no. 8 (October 2011): 837–44. http://dx.doi.org/10.1097/tp.0b013e31823104ec.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Bernit, Emmanuelle, Véronique Veit, Christine Zandotti, Julie Gachon, Nicolas Schleinitz, and Jean Robert Harlé. "Chronic Lymphadenopathies and Human Herpes Virus Type 8." Scandinavian Journal of Infectious Diseases 34, no. 8 (January 2002): 625–26. http://dx.doi.org/10.1080/00365540210147552.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Leao, Jair Carneiro, Andreza Barkokebas Santos de Faria, Déborah Daniela Diniz Fonseca, Luiz Alcino Monteiro Gueiros, Igor Henrique Morais Silva, and Stephen R. Porter. "Intrahost genetic variability of human herpes virus-8." Journal of Medical Virology 85, no. 4 (February 15, 2013): 636–45. http://dx.doi.org/10.1002/jmv.23491.

Full text

APA, Harvard, Vancouver, ISO, and other styles

11

Volpi, A., L. Sarmati, B. Suligoi, M. Montano, G. Rezza, and M. Andreoni. "Correlates of human herpes virus-8 and herpes simplex virus type 2 infections in Northern Cameroon." Journal of Medical Virology 74, no. 3 (2004): 467–72. http://dx.doi.org/10.1002/jmv.20200.

Full text

APA, Harvard, Vancouver, ISO, and other styles

12

Mertz, Gregory J. "Genital Herpes Simplex Virus Infections." Medical Clinics of North America 74, no. 6 (November 1990): 1433–54. http://dx.doi.org/10.1016/s0025-7125(16)30489-8.

Full text

APA, Harvard, Vancouver, ISO, and other styles

13

Annunziato, Paula W. "Herpes Simplex Virus Infections." Pediatrics In Review 17, no. 12 (December 1, 1996): 415–23. http://dx.doi.org/10.1542/pir.17.12.415.

Full text

Abstract:

Case Report A term female developed fever and tachypnea and had a transient episode of poor color and tone on the third day of life. She was delivered by cesarean section 5 hours after rupture of membranes to a 19-year-old, gravida 4, para 0 mother whose serology was negative for syphilis, human immunodeficiency virus (HIV), and hepatitis B surface antigen. The mother had no history of sexually transmitted diseases. Apgar scores were 8 at 1 minute and 9 at 10 minutes. When the infant was admitted to the nursery, scalp abrasions were noticed by the nurse. On the third day of life, a sepsis evaluation was initiated, and the infant received ampicillin and gentamicin intravenously: no bacterial infection was found. On the fifth day of life, vesicular lesions were noticed on her scalp. Bilateral interstitial infiltrates were present On chest radiograph and laboratory studies revealed mild elevations in liver function tests. There were no cerebrospinal fluid (CSF) abnormalities. She was started on intravenous acyclovir, and herpes simplex virus (HSV)-2 subsequently was isolated from both her pharynx and scalp lesions. After receiving intravenous acyclovir for 3 weeks, she was discharged with no evidence of residual sequelae. Four days after acyclovir was discontinued, new scalp vesicles appeared and the infant developed a temperature of 38.8°C (102°F).

APA, Harvard, Vancouver, ISO, and other styles

14

Fitzpatrick, Lisa K., Lovemore Gwanzura, Xing-Quan Zhang, Peter Mason, Ordias Chikuni, Robert Schooley, and David Katzenstein. "EPIDEMIOLOGIC STUDIES OF HUMAN HERPES VIRUS-8 IN ZIMBABWE." Journal of Acquired Immune Deficiency Syndromes and Human Retrovirology 17, no. 4 (April 1998): A14. http://dx.doi.org/10.1097/00042560-199804010-00025.

Full text

APA, Harvard, Vancouver, ISO, and other styles

15

Woolley, Ian, Vicente Boix, and Mark N. Polizzotto. "Human herpes virus 8-related illness: still with us." AIDS 36, no. 14 (November 15, 2022): 2067–69. http://dx.doi.org/10.1097/qad.0000000000003383.

Full text

APA, Harvard, Vancouver, ISO, and other styles

16

Borchers, Kerstin, and Hanns Ludwig. "Simian Agent 8—A herpes simplex-like monkey virus." Comparative Immunology, Microbiology and Infectious Diseases 14, no. 2 (January 1991): 125–32. http://dx.doi.org/10.1016/0147-9571(91)90126-x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

17

Weston, William L., Sylvia L. Brice, Joy D. Jester, Scott Stockert, J. Clark Huff, and Alfred T. Lane. "Herpes Simplex Virus in Childhood Erythema Multiforme." Pediatrics 89, no. 1 (January 1, 1992): 32–34. http://dx.doi.org/10.1542/peds.89.1.32.

Full text

Abstract:

Although an association between herpes simplex virus (HSV) infection and erythema multiforme (EM) minor has been documented in adults, this has not been reported in the pediatric population. This study assessed the potential role of HSV infection in the pathogenesis of EM minor in children. Erythema multiforme skin lesions from 20 children, aged 1 to 16 years, were examined for the presence of HSV by using the polymerase chain reaction. The children included all fit strict clinical criteria for EM minor. Ten had a clinical history of an antecedent herpes infection ("herpes-associated EM"), and 10 did not ("idiopathic EM"). Herpes simplex virus DNA was detected in skin lesions of 8 of 10 children with herpes-associated EM and in 8 of 10 with idiopathic EM. Control skin biopsies from children with other bullous inflammatory diseases were negative. In addition, no HSV could be detected in a biopsy of normal uninvolved skin of a child in whom HSV was present in lesional skin. In situ hybridization on selected biopsies by means of an HSV-specific riboprobe confirmed the presence of HSV and localized it to the epidermis. It is concluded that HSV is a significant precipitating factor for EM minor in children, as it is in adults, and that clinicians should maintain a high index of suspicion of HSV even in the absence of a known history of herpes infection.

APA, Harvard, Vancouver, ISO, and other styles

18

Millichap, J. Gordon. "Herpes Simplex Virus and Surgical Epilepsy." Pediatric Neurology Briefs 11, no. 8 (August 1, 1997): 64. http://dx.doi.org/10.15844/pedneurbriefs-11-8-15.

Full text

APA, Harvard, Vancouver, ISO, and other styles

19

de los Santos Moreno, A., D. Jiménez Gallo, C. Palomar Muñoz, and B. E. Montenegro Puche. "Infecciones por virus del grupo herpes." Medicine - Programa de Formación Médica Continuada Acreditado 11, no. 50 (March 2014): 2946–53. http://dx.doi.org/10.1016/s0304-5412(14)70721-8.

Full text

APA, Harvard, Vancouver, ISO, and other styles

20

Kalman, Concetta M., and Oscar L. Laskin. "Herpes zoster and zosteriform herpes simplex virus infections in immunocompetent adults." American Journal of Medicine 81, no. 5 (November 1986): 775–78. http://dx.doi.org/10.1016/0002-9343(86)90343-8.

Full text

APA, Harvard, Vancouver, ISO, and other styles

21

López-Martínez, Jael, María del Pilar Gabriel de la Torre, Cristian Cruz Ochoa, Lucía Martínez-Martínez, and Miguel Ángel Mayoral Chávez. "Los virus como detonante oncogénico." Tequio 1, no. 2 (January 27, 2018): 57–67. http://dx.doi.org/10.53331/teq.v1i2.5493.

Full text

Abstract:

Viruses are intracellular pathogens, which are replicated within a cell, using their own machinery to generate more viral particles. Viral replication can affect normal host cell genes, thereby modulating signaling pathways that control cell proliferation, differentiation, and death; genomic integrity, and immune-mediated recognition. Viruses, known as oncogenic viruses, cause about 20% of human cancers. The phenomenon of a basic viral infection to tumorigenesis is long due to the involvement of several factors, such as immune complications, cellular mutations, and exposure to other cancer agents. Epstein-Barr virus (EBV), Kaposi’s sarcoma-associated with herpes, also known as type-8 Herpes virus (HHV-8), human papillomavirus (HPV), hepatitis B virus (HBV), hepatitis C virus (HCV) and type-1 human T-cell lymphotropic virus (HTLV-1) are involved in the development of humans cancers.

APA, Harvard, Vancouver, ISO, and other styles

22

Page, Robert N., Michael Harvey, Roy King, and Paul B. Googe. "Human Herpes Virus 8 in Skin Disease: An Immunoperoxidase Analysis." Journal of Histotechnology 28, no. 2 (June 2005): 67–70. http://dx.doi.org/10.1179/his.2005.28.2.67.

Full text

APA, Harvard, Vancouver, ISO, and other styles

23

Yun, James J., Veronica Preda, and Brad Frankum. "Chronic Human Herpes Virus 8 Infection Successfully Treated With Valganciclovir." World Allergy Organization Journal 2, no. 7 (2009): 128–29. http://dx.doi.org/10.1097/wox.0b013e3181abe7b6.

Full text

APA, Harvard, Vancouver, ISO, and other styles

24

BAHADORAN, P. "Human herpes virus type 8 in thymoma-associated Kaposi's sarcoma." Journal of the European Academy of Dermatology and Venereology 11 (September 1998): S174. http://dx.doi.org/10.1016/s0926-9959(98)95203-5.

Full text

APA, Harvard, Vancouver, ISO, and other styles

25

Devriese, P. P. "Bell Palsy and Herpes Simplex Virus." Annals of Internal Medicine 125, no. 8 (October 15, 1996): 698. http://dx.doi.org/10.7326/0003-4819-125-8-199610150-00023.

Full text

APA, Harvard, Vancouver, ISO, and other styles

26

Murakami, Shingo. "Bell Palsy and Herpes Simplex Virus." Annals of Internal Medicine 125, no. 8 (October 15, 1996): 698. http://dx.doi.org/10.7326/0003-4819-125-8-199610150-00024.

Full text

APA, Harvard, Vancouver, ISO, and other styles

27

Guo, Hongyan, Heather S. Koehler, Edward S. Mocarski, and Richard D. Dix. "RIPK3 and caspase 8 collaborate to limit herpes simplex encephalitis." PLOS Pathogens 18, no. 9 (September 19, 2022): e1010857. http://dx.doi.org/10.1371/journal.ppat.1010857.

Full text

Abstract:

Invasion of the brain by herpes simplex virus 1 (HSV1) can lead to the development of herpes simplex encephalitis (HSE) that is often associated with significant morbidity and mortality regardless of therapeutic intervention. Both virus and host immune factors dictate HSE onset and progression. Because programmed cell death pathways including necroptosis are important antiviral defense mechanisms in HSV-1-associated peripheral diseases, they might also play critical roles in HSV1 neuropathogenesis. HSV1-encoded ICP6 prevents receptor-interacting protein kinase 3 (RIPK3)-mediated necroptosis during infection of human cells, but it also acts as a species-dependent inducer of necroptosis in murine cells and thereby restricts virus replication. We therefore used an established mouse model of HSE to investigate RIPK3-mediated necroptosis impact on HSV1 neuropathogenesis. Following corneal HSV1 inoculation, RIPK3 knockout mice showed increased susceptibility to HSE when compared with wildtype mice indicating RIPK3 helps to limit HSE progression. RIPK3-mediated defense against HSE was found to be independent of the kinase domain necessary to drive necroptosis implicating that a death independent function of RIPK3 protects against HSE. Conversely the pro-necroptotic kinase function RIPK3 served to limit viral replication in corneal tissue implicating a tissue-specific RIPK3 function in limiting HSV-1. Further evaluation of the kinase-independent mechanism to restrict HSE revealed that the RIPK3 signaling partner, caspase 8, contributes to limiting HSE neuropathogenesis. Increased HSE susceptibility from loss of caspase 8 and RIPK3 correlated with decreased levels of chemokines, cytokines, and antiviral lymphocytes recruitment to the brain. We conclude that RIPK3 contributes toward host control of HSV1 replication in a tissue-specific fashion. Whereas RIPK3-mediated necroptosis restricts virus replication within the cornea, kinase-independent induction of inflammation by RIPK3 in collaboration with caspase 8 restricts virus replication within the brain during HSE neuropathogenesis.

APA, Harvard, Vancouver, ISO, and other styles

28

Millichap, J. Gordon. "Herpes Simplex Virus Type 2 Neurologic Complications." Pediatric Neurology Briefs 22, no. 6 (June 1, 2008): 46. http://dx.doi.org/10.15844/pedneurbriefs-22-6-8.

Full text

APA, Harvard, Vancouver, ISO, and other styles

29

Lavery, Eric A., and Walter J. Coyle. "Herpes simplex virus and the alimentary tract." Current Gastroenterology Reports 10, no. 4 (August 2008): 417–23. http://dx.doi.org/10.1007/s11894-008-0078-8.

Full text

APA, Harvard, Vancouver, ISO, and other styles

30

Littler, E., I. W. Halliburton, K. L. Powell, B. W. Snowden, and J. R. Arrand. "Immunological Conservation between Epstein-Barr Virus and Herpes Simplex Virus." Journal of General Virology 69, no. 8 (August 1, 1988): 2021–31. http://dx.doi.org/10.1099/0022-1317-69-8-2021.

Full text

APA, Harvard, Vancouver, ISO, and other styles

31

Cohen, Philip R. "Tests for Detecting Herpes Simplex Virus and Varicellazoster Virus Infections." Dermatologic Clinics 12, no. 1 (January 1994): 51–68. http://dx.doi.org/10.1016/s0733-8635(18)30201-8.

Full text

APA, Harvard, Vancouver, ISO, and other styles

32

Buzzetti, María Pía, Vanina Silva, Alvaro Dendi, Gabriela Vidal, and Helena Sobrero. "Oftalmia neonatal causada por Virus Herpes Simple tipo I." Andes Pediatrica 93, no. 5 (October 26, 2022): 749. http://dx.doi.org/10.32641/andespediatr.v93i5.4115.

Full text

Abstract:

Las conjuntivitis y oftalmias neonatales poseen diversas etiologías, entre ellas las virales. Dentro de éstas, debido a la potencial gravedad, destaca la provocada por el Virus Herpes Simple, tanto serotipo 1 como 2. Se trata de una entidad rara, poco frecuente, pero con una alta tasa de morbimortalidad de no mediar diagnóstico y tratamiento oportuno. Objetivo: Describir un caso de oftalmia neonatal a Herpes Virus tipo I, sus características clínicas y diagnóstico. Caso Clínico: Neonato de 8 días de vida, sin antecedentes a destacar, que presenta edema y eritema de párpados, acompañado de secreción ocular bilateral. Por medio de técnica PCR se diagnosticó infección por Herpes Virus tipo I, sin elementos de enfermedad diseminada ni afección del sistema nervioso central. Recibió tratamiento completo con Aciclovir intravenoso con mejoría clínica completa. Conclusiones: El Virus Herpes Simple debe considerarse como diagnóstico diferencial en la conjuntivitis neonatal. El diagnóstico y tratamiento temprano y oportuno es de vital importancia.

APA, Harvard, Vancouver, ISO, and other styles

33

SULIGOI, B., E. POZIO, M. ANDREONI, R. T. DANAYA, L. SARMATI, G. REZZA, I. L. OWEN, and S. BOROS. "INFECTION WITH HUMAN IMMUNODEFICIENCY VIRUS, HERPES SIMPLEX VIRUS TYPE 2, AND HUMAN HERPES VIRUS 8 IN REMOTE VILLAGES OF SOUTHWESTERN PAPUA NEW GUINEA." American Journal of Tropical Medicine and Hygiene 72, no. 1 (January 1, 2005): 33–36. http://dx.doi.org/10.4269/ajtmh.2005.72.33.

Full text

APA, Harvard, Vancouver, ISO, and other styles

34

Tugizov, S. M., J. Y. Webster-Cyriaque, S. Syrianen, A. Chattopadyay, H. Sroussi, L. Zhang, and A. Kaushal. "Mechanisms of Viral Infections Associated with HIV." Advances in Dental Research 23, no. 1 (March 25, 2011): 130–36. http://dx.doi.org/10.1177/0022034511400076.

Full text

Abstract:

HIV infection is commonly associated with activation and dissemination of several other viral pathogens, including herpes simplex virus 1/2, human cytomegalovirus, human herpesvirus 8, Epstein-Barr virus, Varicella Zoster virus, and human papillomavirus, which behave as opportunistic agents and cause various diseases in immunocompromised hosts. The increased frequency and severity of diseases caused by these viruses in HIV-infected individuals is due mainly to dysfunction of both the adaptive and innate immune responses to viral pathogens. In addition, molecular interactions between HIV and these opportunistic viruses are likely to play critical roles in the progression of disease, including neoplasia. This report reviews the critical aspects of HIV interaction with opportunistic viruses, including Epstein-Barr virus, human cytomegalovirus, herpes simplex virus, Varicella Zoster virus, human herpesvirus 8, and human papillomavirus.

APA, Harvard, Vancouver, ISO, and other styles

35

Dwyer, Dominic E., and Anthony L. Cunningham. "4 Herpes simplex virus infection in pregnancy." Baillière's Clinical Obstetrics and Gynaecology 7, no. 1 (March 1993): 75–105. http://dx.doi.org/10.1016/s0950-3552(05)80148-8.

Full text

APA, Harvard, Vancouver, ISO, and other styles

36

Billiau, A. "Immunobiology of infection with herpes simplex virus." Antiviral Research 5, no. 3 (June 1985): 191. http://dx.doi.org/10.1016/0166-3542(85)90051-8.

Full text

APA, Harvard, Vancouver, ISO, and other styles

37

Jha, R., G. Narayen, S. Sinha, K. Kadeer, and K. N. Prasad. "Symptomatic herpes virus infections in postrenal transplant." Transplantation Proceedings 35, no. 1 (February 2003): 284–85. http://dx.doi.org/10.1016/s0041-1345(02)03836-8.

Full text

APA, Harvard, Vancouver, ISO, and other styles

38

Cambouropoulos, Peter J., and Anthony A. Nash. "Herpes simplex virus — Pathogenesis, immunobiology and control." Immunology Today 14, no. 3 (January 1993): 144–45. http://dx.doi.org/10.1016/0167-5699(93)90223-8.

Full text

APA, Harvard, Vancouver, ISO, and other styles

39

Qədir qızı Əbilova, Rübayə, Gülnarə Alışa qızı Cəfərova, and Hafiz Maarif oğlu Osmanov. "The role of viruses and bacteria in the development of cancer." NATURE AND SCIENCE 11, no. 06 (August 23, 2021): 5–10. http://dx.doi.org/10.36719/2707-1146/11/5-10.

Full text

Abstract:

Recently, there have been some scientific studies proving the role of viruses and bacteria in the development of cancer. Among them are eighteen types of pathogens (Helicobacter pylori, hepatitis B virus (HBV), hepatitis C virus (HCV), Opisthorchis viverrini, Clonorchis sinensis, Schistosoma haematobium, human papillomavirus (HPV), Barr (EBV) virus, Ephthia virus-human cell 1 (HTLV-1), human herpes virus type 8 (HHV-8) and human immunodeficiency virus type 1 (HIV-1), belong to group 1 carcinogens. Further study of the role of viruses and bacteria in the development of cancer is of great importance for the early prevention of cancer. Key words: cancer, viruses, bacteria

APA, Harvard, Vancouver, ISO, and other styles

40

Jenkins, C., Y. Sorour, E. Blake, R. Elliot, A. I. Al-Sabah, and J. Green. "Human-immunodeficiency-virus-negative, Human-herpes-virus-8-negative Abdominal Cavity Primary Effusion Lymphoma." Clinical Oncology 17, no. 8 (December 2005): 636–38. http://dx.doi.org/10.1016/j.clon.2005.05.012.

Full text

APA, Harvard, Vancouver, ISO, and other styles

41

Sari, Nendika Dyah Ayu Murika, and Eki Dyan Larasakti. "TRANSMISI DAN REAKTIVASI VIRUS HERPES SIMPLEKS TIPE 1 (LAPORAN KASUS)." Jurnal Kesehatan Gigi dan Mulut (JKGM) 3, no. 1 (July 27, 2021): 1–6. http://dx.doi.org/10.36086/jkgm.v3i1.661.

Full text

Abstract:

Background : Herpes simplex virus (HSV) type 1 can cause recurrent infection (reactivation) in the form of herpes labialis. This viral infection can be transmitted through contact with oral secretions, with a 8-10% prevalence of patients) and 90% of close contact with patients. The purpose of this case study is to describe herpes labialis caused by contact with the patient and its treatment. Case report : A 23-year-old woman came to RSIGM Sultan Agung Semarang with the chief complaint of sores in the corner of her mouth since 4 days ago with fever, which ruptured two days later. Extraoral examination revealed 4 mm erosions with clear borders and multiple 1 mm vesicles with clear borders surrounded by erythematous based on the labial commissure dextra. Herpes labialis was diagnosed. Conclusion : Herpes labialis can be transmitted through direct contact or through oral secretions such as saliva. Giving acyclovir cream three times a day is effective for relieving symptoms and hastening healing. These lesions can be healed without scar.

APA, Harvard, Vancouver, ISO, and other styles

42

Gumber, S., U. Arora, and P. Devi. "Occurrence of Cytomegalo Virus and Herpes Simplex Virus Infections in Pregnancy." Indian Journal of Medical Microbiology 26, no. 2 (April 2008): 204–5. http://dx.doi.org/10.1016/s0255-0857(21)01953-8.

Full text

APA, Harvard, Vancouver, ISO, and other styles

43

S. Mohamed, Amira, Hanaa H.A. Gomaa, and Fadia M. Attia. "Assessment of Human Herpes Virus 8 Infection among Breast Cancer Patients." International Journal of Current Microbiology and Applied Sciences 6, no. 10 (October 10, 2017): 661–68. http://dx.doi.org/10.20546/ijcmas.2017.610.081.

Full text

APA, Harvard, Vancouver, ISO, and other styles

44

O'Donnell, Patrick J., Wayne H. Duke, and Liron Pantanowitz. "Absence of human herpes virus-8 (HHV8) in nephrogenic systemic fibrosis." BMC Research Notes 1, no. 1 (2008): 82. http://dx.doi.org/10.1186/1756-0500-1-82.

Full text

APA, Harvard, Vancouver, ISO, and other styles

45

Sheldon, Julie, Stéphanie Henry, Michel Mourad, Monique Bodéus, Jean‐Paul Squifflet, Thomas F. Schulz, and Patrick Goubau. "Human herpes virus 8 infection in kidney transplant patients in Belgium." Nephrology Dialysis Transplantation 15, no. 9 (September 1, 2000): 1443–45. http://dx.doi.org/10.1093/ndt/15.9.1443.

Full text

APA, Harvard, Vancouver, ISO, and other styles

46

Damery, B., and A. Cremieux. "Virucidal activity against Herpes and Vaccinia virus of 8 antiseptic formulations." International Journal of Pharmaceutics 49, no. 3 (February 1989): 205–8. http://dx.doi.org/10.1016/0378-5173(89)90343-8.

Full text

APA, Harvard, Vancouver, ISO, and other styles

47

Enbom, M., T. Tolfvenstam, H. Ghebrekidan, U. Rudén, M. Grandien, B. Wahren, and A. Linde. "Seroprevalence of human herpes virus 8 in different Eritrean population groups." Journal of Clinical Virology 14, no. 3 (December 1999): 167–72. http://dx.doi.org/10.1016/s1386-6532(99)00061-x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

48

Slacmeulder, Michel, Frederic Geissmann, Yves Lepelletier, Jean-Christophe Fournet, Nicole Brousse, Caroline Thomas, Jean Donadieu, and Antoine Gessain. "No association between Langerhans cell histiocytosis and human herpes virus 8." Medical and Pediatric Oncology 39, no. 3 (July 23, 2002): 187–89. http://dx.doi.org/10.1002/mpo.10114.

Full text

APA, Harvard, Vancouver, ISO, and other styles

49

Cheng, Frankie Wai Tsoi, Paul Kay Sheung Chan, Wing Kwan Leung, Vincent Lee, Matthew MK Shing, Chi Kong Li, and Ting Fan Leung. "Lymphoproliferative Response to Herpes Simplex Virus, Cytomegalovirus, Epstein-Barr Virus, Varicella Zoster Virus, Human Herpes Virus 6, 7 and 8 Antigen Stimulation in Pediatric Allogeneic Stem Cell Transplant Recipients." Blood 114, no. 22 (November 20, 2009): 4663. http://dx.doi.org/10.1182/blood.v114.22.4663.4663.

Full text

Abstract:

Abstract Abstract 4663 Introduction Delayed immune reconstitution is a major cause of transplant-related mortality in allogeneic stem cell transplant setting. Reactivation of herpes viruses is a common post-transplant complication. In this study, we evaluate the recovery of cell-mediated immunity (CMI) to various herpes viruses by measuring lymphoproliferative response (LPR) to specific recall antigens. Materials and Methods Cell-mediated immunity was evaluated by the in-vitro lymphoproliferative response of peripheral blood mononuclear cells (PBMC) to specific purified HSV, VZV, CMV, EBV, HHV-6, 7, 8 antigens. Results were expressed as stimulation index (SI). SI≥a3 was regarded as positive lymphoproliferative response (LPR). Serial measurements were made at before transplant, and monthly during the initial 6 months post-transplant then quarterly till 12 months post-transplant. Results From 2001-2004, 36 patients (M=19; F=17) with median age 10.5 years old were recruited. Hematological malignancies accounted for 58.3% of cases. Most transplants were from matched siblings (MSD) with peripheral blood stem cells (PBSC) as the source of stem cells. Altogether 50% of subjects showed positive LPR to HSV, CMV and VZV antigens at 2-4 months post-transplant; A significant upsurge of LPR were observed at 4-6 months post-transplant. However, no positive LPR to HSV, HHV-6,7 and 8 antigens were observed. The antibody status of donor and recipient for HSV, CMV and VZV were associated with the timing of recovery of CMI. Donor choice and stem cell source were important determinants of eventual LPR at day 100 post-transplant. Conclusion More than half of transplant recipients developed satisfactory LPR to HSV, CMV and VZV at 2-4 months after transplant. Pre-transplant serostatus of donor and recipient, donor choice and stem cell are important determinants of LPR to herpes viruses. Disclosures: No relevant conflicts of interest to declare.

APA, Harvard, Vancouver, ISO, and other styles

50

Nocera, A., M. Corbellino, U. Valente, S. Barocci, F. Torre, R. De Palma, A. Sementa, et al. "Posttransplant human herpes virus 8 infection and seroconversion in a kaposi’s sarcoma affected kidney recipient transplanted from a human herpes virus 8 positive living related donor." Transplantation Proceedings 30, no. 5 (August 1998): 2095–96. http://dx.doi.org/10.1016/s0041-1345(98)00550-8.

Full text

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!