Relevant bibliographies by topics / HERG

Academic literature on the topic 'HERG'

Author: Grafiati
Published: 4 June 2021
Last updated: 6 July 2024

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Journal articles on the topic "HERG"

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Trudeau, Matthew C., Jeffrey W. Warmke, Barry Ganetzky, and Gail A. Robertson. "HERG Sequence Correction." Science 272, no. 5265 (May 24, 1996): 1087. http://dx.doi.org/10.1126/science.272.5265.1087.c.

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LIAN, JIANGFANG, JIAN GUO, XIAOYAN HUANG, XI YANG, GUOCHANG HUANG, HAIYAN MAO, HUAN HUAN SUN, YANNA BA, and JIANQING ZHOU. "miRNAs Regulate hERG." Journal of Cardiovascular Electrophysiology 27, no. 12 (September 26, 2016): 1472–82. http://dx.doi.org/10.1111/jce.13084.

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Trudeau, M. C., J. W. Warmke, B. Ganetzky, and G. A. Robertson. "HERG Sequence Correction." Science 272, no. 5265 (May 24, 1996): 1083j—1087. http://dx.doi.org/10.1126/science.272.5265.1083j.

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Trudeau, M. C., J. W. Warmke, B. Ganetzky, and G. A. Robertson. "HERG Sequence Correction." Science 272, no. 5265 (May 24, 1996): 1087c. http://dx.doi.org/10.1126/science.272.5265.1087c.

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Czodrowski, Paul. "hERG Me Out." Journal of Chemical Information and Modeling 53, no. 9 (August 21, 2013): 2240–51. http://dx.doi.org/10.1021/ci400308z.

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Zhou, Qinlian, Agnieszka Lis, and G. Bett. "Modeling HERG Isoforms." Biophysical Journal 104, no. 2 (January 2013): 264a. http://dx.doi.org/10.1016/j.bpj.2012.11.1485.

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Inanobe, Atsushi, Kazuharu Furutani, and Yoshihisa Kurachi⁎. "Facilitation of hERG Current Occurs in Various hERG Channel Blockers." Journal of Molecular and Cellular Cardiology 45, no. 4 (October 2008): S13. http://dx.doi.org/10.1016/j.yjmcc.2008.09.632.

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Kim, Ki-Suk, Sang-Joon Park, Hankwang Na, Hae-Sung Park, and Eun-Joo Kim. "EFFECT OF HERG CURRENT BY DRUGS IN hERG TRAFFICKING MUTANTS." Journal of Pharmacological and Toxicological Methods 56, no. 2 (September 2007): e5. http://dx.doi.org/10.1016/j.vascn.2007.02.010.

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Friederich, P., A. Solth, S. Schillemeit, and D. Isbrandt. "Local anaesthetic sensitivities of cloned HERG channels from human heart: comparison with HERG/MiRP1 and HERG/MiRP1 T8A." British Journal of Anaesthesia 92, no. 1 (January 2004): 93–101. http://dx.doi.org/10.1093/bja/aeh026.

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El Harchi, Aziza, Dario Melgari, and Jules C. Hancox. "Investigation of the Influence of hERG 1b on hERG Channel Pharmacology." Biophysical Journal 104, no. 2 (January 2013): 297a. http://dx.doi.org/10.1016/j.bpj.2012.11.1655.

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Dissertations / Theses on the topic "HERG"

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Ridley, John Malcolm. "Pharmacology of the HERG K⁺ channel." Thesis, University of Bristol, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.411068.

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Thomson, Steven James. "Deactivation gating and pharmacology of hERG potassium channel." Thesis, University of Leicester, 2012. http://hdl.handle.net/2381/11071.

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hERG (Kv11.1) encodes the α-subunit of the potassium (K+) channel that carries IKr, an important current for repolarisation of the cardiac action potential. Alterations of hERG current, either through inherited mutations that alter gating or through drugs that block the pore, are associated with Long QT syndrome, cardiac arrhythmias and sudden death. The N-terminus has an important role in regulating deactivation, a gating process that is important for timing of the hERG current during cardiac action potentials. Removing the entire N-terminus accelerates deactivation. A crystal structure of part of the N-terminus (residues 26-135) was solved in 1998 and showed it contained a PAS domain, but it did not resolve the structure of the functionally important first 26 residues (NT 1-26). Here we present an NMR structure of residues 1-135. The structure reveals that residues 1-10 are unordered and residues 11-24 form an amphipathic helix one face of which is positively charged. Neutralising the positive charge accelerates deactivation to similar rates as if the whole of the N-terminus has been removed. Neutralising negative charge in the C-terminus also accelerates deactivation. We propose a model where the N and C-termini interact to stabilise the open state of the channel and slow deactivation. Exactly how changes in membrane voltage are transduced into movement of the activation gate is not fully understood. In hERG, the mutation V659A dramatically slows deactivation. Val659 is located in a region where hERG’s activation gate is believed to lie. From the structure of Kv2.1 it can be seen the S4-S5 linker forms a cuff around S6 where the activation gate is thought to be. Using cysteine cross-linking experiments we show that V659C interacts with E544C and Y545C in the S4-S5 linker to lock the channel in the open state. Trapping of drugs in the inner cavity of hERG has been an important model used to help explain why hERG is blocked by so many drugs and with high potency. A series of derivatives of E-4031, a well characterised high-affinity hERG blocker, were made that progressively increased the length of the molecule. Results in this thesis showed these compounds had binding kinetics completely different from E-4031 and none were trapped in the inner cavity. An alternative model of strongly state-dependent drug binding rather than drug-trapping is proposed. Together, the results in this thesis present new insights on the structural basis for deactivation gating and drug binding in hERG channels.
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Pettini, Francesco. "Molecular Dynamics simulation of the hERG channel assisting Precision Medicine in Channelopathies." Doctoral thesis, Università di Siena, 2023. https://hdl.handle.net/11365/1227475.

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In the last two decades, a revolution in biology has shifted the traditional reductive approach to a bottom-up study of virtual models. This discipline, known as System Biology, integrates information coming from individual components, in order to predict the functioning of biological systems, with the idea that complex systems are made up of many independent components that can interact within well-structured networks changing over time, and that the functional properties of biological systems emerge as a consequence of interactions among their components. This paradigm shift is enabled by rapid advancements in technologies providing high-throughput instruments able to analyse in detail biological processes at the single molecule and single cell scale. The vast amount of data produced by these experimental techniques asks for adequate methods of analyses. The present dissertation focues on structural based methods for simulating the functioning of biological molecules, and in particular on the role of Molecular Dynamics simulations. The advantage of Molecular Dynamics simulations is that it is based on physical description of the systems, and consequently it might offer an atomistic description of the process under investigation. The first chapter of this thesis will provide an introduction on the role of Molecular Genetics and Biology in Medicine, also considering new challenges for the prediction of protein interactions and for development of Precision Medicine. In the Second Chapters, Molecular Dynamics simulations will be discussed, with an emphasis on the methods for data analysis adopted in the research projects presented in the second part of the thesis. The third Chapter will be present the main research project produced during my PhD: the study of inactivation and drug binding in the hERG potassium channel. Side project and parallel collaborations are briefly discussed in the fourth Chapter, followed by concluding remarks.
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Blütner, Carmen. "Der Einfluss des Antiemetikums Droperidol auf den HERG-Ionenkanal /." Hamburg, 2008. http://opac.nebis.ch/cgi-bin/showAbstract.pl?sys=000254071.

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Shimizu, Atsuya, Ryoko Niwa, Zhibo Lu, Haruo Honjo, and Kaichiro Kamiya. "Effects of Dronedarone on HERG and KCNQ1/KCNE1 Channels." Research Institute of Environmental Medicine, Nagoya University, 2003. http://hdl.handle.net/2237/7585.

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NIWA, Ryoko, Zhibo LU, Haruo HONJO, and Kaichiro KAMIYA. "Voltage-Dependent Effects of Bepridil on D540K HERG Channels." Research Institute of Environmental Medicine, Nagoya University, 2002. http://hdl.handle.net/2237/2798.

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Niwa, Ryoko, Atsuya Shimizu, Zhibo Lu, Haruo Honjo, and Kaichiro Kamiya. "Voltage-Dependent Effects of Amiodarone on D540K HERG Channels." Research Institute of Environmental Medicine, Nagoya University, 2003. http://hdl.handle.net/2237/7587.

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Du, Chunyun. "The effects of acidosis on the hERG potassium channel." Thesis, University of Bristol, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555619.

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The human ether-a-go-go-related gene (hERG) encodes channels mediating the rapid delayed rectifier K+ current (IKr). IKr participates in cardiac action potential (AP) repolarisation and may also protect the ventricles against premature stimulation. The heart is exposed to acidosis (low pH) in a number of pathological conditions including myocardial ischemia. Extracellular acidosis is known to modulate hERG current (IhERG) function, although a number of aspects of the modulation remain incompletely understood. The aims of this investigation were to establish the effects of acidosis on: (i) IhERG amplitude, kinetics and the response to premature stimulation at mammalian physiological temperature; (ii) the hERG blocking potency of selected anti arrhythmic drugs. Whole-cell patch-clamp recordings of IhERG were made from mammalian cells (CHO or HEK 293) at 37 QC. Lowering external pH from 7.4 to 6.3 reduced the magnitude of IhERG by reducing macroscopic hERG conductance and modulating IhERG kinetics, with positively shifted activation and accelerated deactivation. Results from experiments using an acidic pipette solution showed that the actions of protons occurred from the external surface and not from secondary intracellular acidosis. Experimental and computer simulation work demonstrated that acidosis impairs the protective role of IhERG against premature stimulation. The effects of extracellular acidosis on IhERG kinetics were preserved when the shortened hERG I b isoform was studied, indicating that a full-length N-terminus is not necessary for acidic modulation of hERG channel function. Interestingly, the inhibitory effect of acidosis on IhERG was greater for hERG 1 band hERG lall b than for hERG la. Extracellular acidosis decreased the hERG blocking potency of flecainide, dofetilide and ranolazine, whilst the potency of amiodarone was unaffected. IhERG inactivation was found to be important for ranolazine's inhibitory action and a series of S6 and inner helix residues (Y652, F656, T623, S624 and V625) were identified as contributing to ranolazine binding. I.
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DJUIDJE, ERNESTINE NICAISE. "Design, Synthesis and Biological activities of Benzothiazole, Benzimidazole and Imidazopyrimidine polyphenols as multifunctional molecules against oxidative stress sustained processes." Doctoral thesis, Università degli studi di Ferrara, 2018. http://hdl.handle.net/11392/2488156.

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Reactive oxygen species regulate several essential physiological processes such as cell proliferation, differentiation, vascular tone and inflammation. However, their higher concentrations can have deleterious effects on many molecules including protein, lipid, RNA and DNA which leads to cell destruction causing several diseases including Parkinson’s diseases, inflammatory disease, cardiovascular, cancer, diabetes, Alzheimer’s disease, cataracts, autism and aging. Most of these diseases involve more physiopathological indications. Nevertheless, different pharmacological strategies, such as the use of multifunctional drugs, have been designed to prevent or restore redox imbalances and to treat complex diseases. The present research project comes from the desire to synthesize multifunctional compounds with the capacity to present or treat multifactorial diseases such as cancer. For this purple, isosteric modification was performed on 2-Phenyl-1H-benzimidazol-5-sulfonic acid (PBSA) and three set of compounds were obtained: benzimidazole, benzothiazole and imidazopyrimidine derivatives. Synthesized compounds were evaluated for their UV-filter, antioxidant, antifungal and antiproliferative activites. Photoprotective capacity was determined using spectrophotometric transmittance technique. DPPH and FRAP were performed to determine antioxidant activity. Diffusion method in Sabouraud Dextrose Agar (SDA) was used to evaluate anti-dermatophyte activity, while for broth microdilution method in RPMI was used to investigate anti-candida activity. Finally, MTS essay were performed to determine antioroliferative activity. For benzimidazole derivatives, compound DE 35 was found to be a potential candidate in the development of multifunctional drugs, while for benzothiazole and imidazopyrimidine we respectively have 4g/4k and 14g. In addition, these set of compounds might have possible application as a drug for the treatment of neoplastic diseases such as childhood leukemia, pancreatic cancer and melanoma.
Le specie reattive dell'ossigeno (ROS) regolano diversi processi fisiologici essenziali come la proliferazione cellulare, la differenziazione e il tono vascolare. Tuttavia, un'alta concentrazione di ROS può avere effetti indesiderati su molte molecole tra cui proteine, lipidi, RNA e DNA che portano alla distruzione cellulare causando varie pathologie ad esempio: malattie infiammatorie, malattie cardiovascolari, cancro, diabete, cataratta, autismo invecchiamento malattia di Parkinson e malattia Alzheimer. La maggior parte di queste malattie coinvolge più indicazioni fisiopatologiche. Diverse strategie farmacologiche, come l'uso di farmaci multifunzionali, sono state progettate per prevenire o ripristinare gli squilibri di riduzione dell'ossidazione e per trattare malattie complesse. Il presente progetto di ricerca nasce dal desiderio di sintetizzare composti multifunzionali con la capacità di prevenire o curare malattie multifattoriali come il cancro. Per questo motivo, le modifiche isosteriche sono state effettuate sull’ acido 2-fenil-1H-benzimidazol-5-solfonico (PBSA) e sono state ottenute tre serie di composti: derivati benzimidazolici, benzotiazolici e imidazopirimidici. I composti sintetizzati sono poi stati valutati per le loro attività UV filtrante, antiossidante, antifungina e antiproliferativa. La capacità fotoprotettiva è stata determinata utilizzando la tecnica di trasmittanza spettrofotometrica. DPPH e FRAP sono stati eseguiti per determinare l'attività antiossidante. Il metodo di diffusione in Sabouraud Dextrose Agar (SDA) è stato utilizzato per valutare l'attività anti-dermatofiti, mentre il metodo di microdiluizione del brodo RPMI è stato utilizzato per studiare l'attività anti-candida. Infine, è stato eseguito il saggio MTS per determinare l'attività anti-proliferativa. Per i derivati benzimidazolici, il composto DE 35 è risultato essere il potenziale candidato nello sviluppo di farmaci multifunzionali, mentre per i benzotiazolici e l'imidazopirimidici abbiamo rispettivamente 4g / 4k e 14g. Inoltre, questi set di composti potrebbero avere una possibile applicazione come farmaco per il trattamento di malattie neoplastiche come: la leucemia infantile, il cancro del pancreas e il melanoma.
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Ouille, Aude. "Evaluation des risques torsadogènes en pharmacologie de sécurité : du test hERG à la télémétrie sur animal éveillé, vers une évolution des recommandations ?" Thesis, Tours, 2009. http://www.theses.fr/2009TOUR4016.

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Toutes les molécules en développement préclinique (ICH S7B) doivent être testées pour évaluer leur potentiel torsadogène. Le but de ce travail est d’établir le profil électrophysiologique de molécules torsadogènes connues afin de mieux comprendre le mécanisme de déclenchement des Torsades de Pointes et de déterminer des points clés nous permettant de prédire les molécules à risque. Il existe une base de données, TdPScreen®, combinant données cliniques et tests réalisés sur fibres de Purkinje de chien, qui permet d’attribuer un score pro-arythmique aux molécules testées. Treize molécules connues ont été choisies dans cette base de données, et testées en patch-clamp sur des cellules HEK293 exprimant le canal hERG (IKR), le canal KvLQT1+MinK (IKS), le canal Kir2.1 (IK1), le canal NaV1.5 (INa), ou le canal CaV1.2+? (ICaL). Des investigations in vivo ont également été réalisées, afin de mettre en évidence l’impact du système nerveux autonome sur l’allongement de l’intervalle QT lors d’études de pharmacologie de sécurité
According to the ICH S7B guidelines, the torsadogenic risk of new drug candidates must be evaluated before clinical trials. The aim of this work was to establish the electrophysiological profile of known torsadogenic drugs to better understand the mechanism triggering the Torsades de Pointe and defined key points for prediction of proarrhythmic risk. TdPScreen®, a predictive tool, based on clinical data and the model of isolated canine Purkinje fibres allows determination of a proarrhythmic score. Thirteen drugs were chosen in this data base, and tested in patch-clamp on HEK293 cells expressing different channels: hERG (IKR), KvLQT1+MinK (IKS), Kir2.1 (IK1), NaV1.5 (INa), or CaV1.2+? (ICaL). In vivo investigations were also performed, to bring to light the impact of the autonomic nervous system on QT interval prolongation in safety pharmacology
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Books on the topic "HERG"

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Chʻoe, Han-sŏk. hERG chʻaenŏl ŭl iyong han simhyŏl kwanʼgye anjŏnsŏng yangni yŏnʼgu =: Study on cardiovascular safety of drugs using hERG channel. [Seoul]: Sikpʻum Ŭiyakpʻum Anjŏnchʻŏng, 2007.

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Chadwick, Derek J., and Jamie Goode, eds. The hERG Cardiac Potassium Channel: Structure, Function and Long QT Syndrome. Chichester, UK: John Wiley & Sons, Ltd, 2005. http://dx.doi.org/10.1002/047002142x.

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Derek, Chadwick, Goode Jamie, and Novartis Foundation, eds. The hERG cardial potassium channel: Structure, function, and long QT syndrome. New York: J. Wiley, 2005.

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Suprihono, Agus. Hera Heru. [Yogyakarta]: Taman Budaya Propinsi Daerah Istimewa Yogyakarta, 2001.

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Kudlinski, Kathleen V. Hero over here. New York, N.Y., U.S.A: Puffin Books, 1992.

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Kudlinski, Kathleen V. Hero over here. New York, N.Y., U.S.A: Viking, 1990.

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Block, Lawrence. Here comes a hero. Thorndike, ME: Center Point Publishing, 2002.

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Varela, Gabrielle C. I made Herc a hero, by Phil. New York: Disney Press, 1997.

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Varela, Gabrielle C. Disney's Hercules: I made Herc a hero, by Phil. New York: Disney Press, 1997.

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Malcolm Hillier's herb garden. London: Dorling Kindersley, 1996.

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Book chapters on the topic "HERG"

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Ficker, Eckhard, Adrienne Dennis, Yuri Kuryshev, Barbara A. Wible, and Arthur M. Brown. "hERG Channel Trafficking." In The hERG Cardiac Potassium Channel: Structure, Function and Long QT Syndrome, 57–74. Chichester, UK: John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/047002142x.ch6.

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Leishman, Derek J., and Zoran Rankovic. "Drug Discovery vs hERG." In Topics in Medicinal Chemistry, 225–59. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/7355_2014_38.

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Leishman, Derek J., and Zoran Rankovic. "Erratum to: Drug Discovery vs hERG." In Topics in Medicinal Chemistry, 385. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/7355_2014_72.

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Finlayson, Keith, and John Sharkey. "A High-Throughput Binding Assay for HERG." In Optimization in Drug Discovery, 353–68. Totowa, NJ: Humana Press, 2004. http://dx.doi.org/10.1385/1-59259-800-5:353.

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Singh, Jitendra N., and Shyam S. Sharma. "hERG Potassium Channels in Drug Discovery and Development." In Ion Channels and Their Inhibitors, 149–90. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-19922-6_6.

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Brown, Arthur M. "hERG Block, QT Liability and Sudden Cardiac Death." In The hERG Cardiac Potassium Channel: Structure, Function and Long QT Syndrome, 118–35. Chichester, UK: John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/047002142x.ch10.

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Recanatini, Maurizio, Andrea Cavalli, and Matteo Masetti. "In Silico Modelling-Pharmacophores and hERG Channel Models." In The hERG Cardiac Potassium Channel: Structure, Function and Long QT Syndrome, 171–85. Chichester, UK: John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/047002142x.ch14.

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Piper, David R., Michael C. Sanguinetti, and Martin Tristani-Firouzi. "Voltage Sensor Movement in the hERG K+ Channel." In The hERG Cardiac Potassium Channel: Structure, Function and Long QT Syndrome, 46–56. Chichester, UK: John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/047002142x.ch5.

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Schiesaro, Andrea, and Gerhard F. Ecker. "Prediction of hERG Channel Inhibition Using In Silico Techniques." In Ion Channels and Their Inhibitors, 191–239. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-19922-6_7.

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Arcangeli, Annarosa. "Expression and Role of hERG Channels in Cancer Cells." In The hERG Cardiac Potassium Channel: Structure, Function and Long QT Syndrome, 225–34. Chichester, UK: John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/047002142x.ch17.

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Conference papers on the topic "HERG"

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De Bie, Johan, Brian Chiu, David Mortara, Cristiana Corsi, and Stefano Severi. "Quantification of hERG Block from the ECG." In 2017 Computing in Cardiology Conference. Computing in Cardiology, 2017. http://dx.doi.org/10.22489/cinc.2017.239-123.

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Grienke, U., CE Mair, JM Kratz, and JM Rollinger. "hERG channel blockers in medicinal plants: Hysteria or serious problem?" In GA 2017 – Book of Abstracts. Georg Thieme Verlag KG, 2017. http://dx.doi.org/10.1055/s-0037-1608031.

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Hu, Jieying, Ming Huang, Naoaki Ono, Ye Chen-Izu, Leighton T. Izu, and Shigehiko Kanaya. "Cardiotoxicity Prediction Based on Integreted hERG Database with Molecular Convolution Model." In 2019 IEEE International Conference on Bioinformatics and Biomedicine (BIBM). IEEE, 2019. http://dx.doi.org/10.1109/bibm47256.2019.8983163.

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Gonzalez, Rodolfo, Edgar Cardenas, and Alain Manzo. "Pore hERG mutation A561V increases dofetilide proarrhythmic risk. A simulation study." In 2017 IEEE International Autumn Meeting on Power, Electronics and Computing (ROPEC). IEEE, 2017. http://dx.doi.org/10.1109/ropec.2017.8261689.

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Hirano-Iwata, A., A. Oshima, Y. Ishinari, Y. Kimura, and M. Niwano. "A Chip-Based Stable Lipid Bilayers for Recording hERG Channel Activities." In 2013 International Conference on Solid State Devices and Materials. The Japan Society of Applied Physics, 2013. http://dx.doi.org/10.7567/ssdm.2013.g-1-3.

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Sarkar, Aditya, and Arnav Bhavsar. "Virtual Screening of Pharmaceutical Compounds with hERG Inhibitory Activity (Cardiotoxicity) using Ensemble Learning." In 8th International Conference on Bioimaging. SCITEPRESS - Science and Technology Publications, 2021. http://dx.doi.org/10.5220/0010267700002865.

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Sarkar, Aditya, and Arnav Bhavsar. "Virtual Screening of Pharmaceutical Compounds with hERG Inhibitory Activity (Cardiotoxicity) using Ensemble Learning." In 8th International Conference on Bioimaging. SCITEPRESS - Science and Technology Publications, 2021. http://dx.doi.org/10.5220/0010267701520159.

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Miyata, Ryusuke, Daisuke Tadaki, Daichi Yamaura, Shun Araki, Madoka Sato, Maki Komiya, Teng Ma, Hideaki Yamamoto, Michio Niwano, and Ayumi Hirano-Iwata. "Solvent-free Lipid Bilayer Microarray for Parallel Recordings of Transmembrane hERG Channel Activities." In 2021 International Conference on Solid State Devices and Materials. The Japan Society of Applied Physics, 2021. http://dx.doi.org/10.7567/ssdm.2021.g-6-07.

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Couderc, J. P., W. Zareba, and A. J. Moss. "Discrimination of herg carrier from non-carrier adult patients with borderline prolonged QTc interval." In Computers in Cardiology, 2005. IEEE, 2005. http://dx.doi.org/10.1109/cic.2005.1588050.

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WU, LONG-MEI, KAZUO UEDA, YUJI HIRANO, TETSUSHI FURUKAWA, and MASAYASU HIRAOKA. "HERG POTASSIUM CHANNEL IS REGULATED BY PROTEIN TYROSINE KINASE (PTK) IN HUMAN EMBRYONIC KIDNEY CELLS." In Proceedings of the 31st International Congress on Electrocardiology. WORLD SCIENTIFIC, 2005. http://dx.doi.org/10.1142/9789812702234_0009.

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Reports on the topic "HERG"

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Disis, Mary L. Phase II Study of a HER-2/neu (HER2) Intracellular Domain (ICD) Peptide-Based Vaccine Administered to Stage IIIB and IV HER2 Positive Breast Cancer Patients Receiving Trastuzumab Monotherapy. Fort Belvoir, VA: Defense Technical Information Center, May 2008. http://dx.doi.org/10.21236/ada486630.

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Martínez-García, A., and A. Gómez-Aguilar. Magallanes: building the hero. Revista Latina de Comunicación Social, February 2019. http://dx.doi.org/10.4185/rlcs-2019-1348en.

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Marshak, David. Portlet Standards Are Here. Boston, MA: Patricia Seybold Group, November 2003. http://dx.doi.org/10.1571/ta11-26-03.

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Freeman, A. E. Gene, Joe Detrick, and David H. Kelley. Dairy Breeding Research Herd. Ames (Iowa): Iowa State University, January 2004. http://dx.doi.org/10.31274/ans_air-180814-25.

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Tinsley, David. The Hero at Rest. Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.6826.

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Nugroho, Heru Santoso Wahito. HERU SANTOSO WAHITO NUGROHO. FORIKES, 2021. http://dx.doi.org/10.33846/hswn.

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Disis, Mary L. Phase II Study of HER-2/neu Intracellular Domain Peptide-Based Vaccine Administered to Stage IV HER2 Positive Breast Cancer Patients Receiving Trastuzumab. Fort Belvoir, VA: Defense Technical Information Center, May 2006. http://dx.doi.org/10.21236/ada456015.

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Disis, Mary L. Study of HER-2/neu Intracellular Domain Peptide-Based Vaccine Administered to Stage IV HER2 Positive Breast Cancer Patients Receiving Trastuzumab. Phase 2. Fort Belvoir, VA: Defense Technical Information Center, May 2009. http://dx.doi.org/10.21236/ada505213.

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Disis, Mary L. Phase II Study of HER-2/neu Intracellular Domain Peptide-Based Vaccine Administered to Stage IV HER2 Positive Breast Cancer Patients Receiving Trastuzumab. Fort Belvoir, VA: Defense Technical Information Center, May 2007. http://dx.doi.org/10.21236/ada471552.

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Disis, Mary L. Phase II Study of a HER-2/neu Intracellular Domain Peptide-Based Vaccine Administered to Stage IV HER2 Positive Breast Cancer Patients Receiving Trastuzumab. Fort Belvoir, VA: Defense Technical Information Center, May 2010. http://dx.doi.org/10.21236/ada533841.

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