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Books on the topic 'Hepatitis C – Patients'

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Published: 4 June 2021
Last updated: 4 February 2022

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1

Napolitano, Ralph. Hepatitis C survival secrets: With critical insights your doctor won't share. Pine Bush, NY: HTX, 2010.

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2

Smith, Suzy. Hepatitis C through a patient's eyes: Hope for healing. Livermore, CA: WingSpan Press, 2006.

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3

Scheu, Gerhard. Deliktische Produktverantwortung für Hepatitis C-Infektionen hämophiler Patienten. Baden-Baden: Nomos, 1999.

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4

New South Wales. Anti-Discrimination Board. C change: Report of the enquiry into hepatitis C related discrimination. [Sydney]: The Board, 2001.

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5

Pozitivno. Sofii︠a︡: Bŭlgarski pisatel, 2012.

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6

Mullah, Nadiah Mohammad Ali. A study of the incidence of viral hepatitis B and C in patients undergoing haemodialysis in Jeddah. Salford: University of Salford, 1992.

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7

Mohamed Hatem Fathi El-Saied Wali. Natural history, factors affecting severity and progression rate of hepatitis c virus (HCV) infection in liver transplanted and non-transplanted patients. Birmingham: University of Birmingham, 2002.

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8

Parker, James N., and Philip M. Parker. The 2002 official patient's sourcebook on hepatitis C. Edited by Icon Group International Inc and NetLibrary Inc. San Diego, Calif: Icon Health Publications, 2002.

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9

Office, General Accounting. VA health care: Further efforts needed to improve hepatitis C testing for at-risk veterans : report to the Chairman, Subcommittee on National Security, Emerging Threats, and International Relations, Committee on Government Reform, House of Representatives. Washington, D.C: United States General Accounting Office, 2003.

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10

Office, General Accounting. VA health care: Improvements needed in hepatitus C disease management practices : report to the Chairman, Subcommittee on National Security, Veterans Affairs, and International Relations, Committee on Government Reform, House of Representatives. Washington, D.C: United States General Accounting Office, 2003.

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11

Office, General Accounting. VA health care: Laundry service operations and costs : report to the Chairman, Subcommittee on Oversight and Investigations, Committee on Veterans' Affairs, House of Representatives. Washington, D.C. (P.O. Box 37050, Washington, D.C. 20013): The Office, 1999.

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12

Office, General Accounting. VA health care: VA's management of drugs on its national formulary : report to the Ranking Minority Member, Committee on Veterans' Affairs, U.S. Senate. Washington, D.C. (P.O. Box 37050, Washington, D.C. 20013): The Office, 1999.

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13

Office, General Accounting. VA health care: Expanded eligibility has increased outpatient pharmacy use and expenditures : report to the chairman, Committee on Veterans' Affairs, House of Representatives. Washington, D.C. (P.O. Box 37050, Washington 20013): U.S. General Accounting Office, 2002.

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14

Office, General Accounting. VA health care: Access for Chattanooga-area veterans needs improvement : report to congressional requesters. Washington, D.C. (P.O. Box 37050, Washington 20013): U.S. General Accounting Office, 2004.

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15

Office, General Accounting. VA health care: Status of efforts to improve efficiency and access : report to the chairman, Subcommitte on VA, HUD, and Independent Agencies, Committee on Appropriations, U.S. Senate. Washington, D.C. (P.O. Box 37050, Washington, D.C. 20013): The Office, 1998.

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16

Office, General Accounting. VA health care: Effects of facility realignment on construction needs are unknown : report to the Chairman, Subcommittee on Hospitals and Health Care, Committee on Veterans' Affairs, House of Representatives. Washington, D.C: The Office, 1995.

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17

Office, General Accounting. VA health care: Issues affecting eligibility reform efforts : report to the Chairman, Committee on Veterans' Affairs, U.S. Senate. Washington, D.C: The Office, 1996.

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18

Office, General Accounting. VA health care: Medical centers are not correcting identified quality assurance problems : report to the chairman, Committee on Veterans' Affairs, U.S. Senate. Washington, D.C: The Office, 1992.

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19

Office, General Accounting. VA health care: Variabilities in outpatient care eligibility and rationing decisions : report to the Chairman, Subcommittee on Oversight and Investigations, Committee on Veterans' Affairs, House of Representatives. Washington, D.C: The Office, 1993.

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20

Office, General Accounting. VA health care: More veterans are being served, but better oversight is needed : report to Congressional requesters. Washington, D.C. (P.O. Box 37050, Washington, D.C. 20013): The Office, 1998.

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21

Office, General Accounting. VA health care: Retargeting needed to better meet veterans' changing needs : report to Congressional requesters. Washington, D.C: The Office, 1995.

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22

Hepatitis C: A complete guide for patients and families. Johns Hopkins University Press, 2015.

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23

Primedia. PULSE Plus: Approaching Psychiatric Patients (Pulse Plus). Delmar Learning, 2001.

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24

Fabrizi, Fabrizio. Hepatitis C. Edited by Vivekanand Jha. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0186.

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Hepatitis C virus (HCV) is associated with a large spectrum of histopathological lesions in both native and transplanted kidneys. The exact frequency of kidney damage in HCV-infected patients remains unknown, but the most frequent associated renal lesion is membranoproliferative glomerulonephritis (MPGN) with deposition of immunoglobulin and complement (MPGN type 1), usually in the context of type II mixed cryoglobulinaemia associated with a monoclonal IgM which binds IgG.Various approaches have been tried for the treatment of HCV-related glomerulonephritis, including immunosuppressive therapy (corticosteroids and cytotoxic agents), plasma exchange, and antiviral agents. Use of antiviral drugs for treatment of HCV-associated glomerulonephritis has shown encouraging results. Immunosuppressive therapy is recommended for cryoglobulinaemic kidney disease. Two distinct approaches should be considered for the treatment of HCV-associated cryoglobulinaemic glomerulonephritis according to the level of proteinuria and kidney failure. Preliminary evidence on rituximab therapy for HCV-related cryoglobulinaemic glomerulonephritis exists but several questions related to its use remain unclear.
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25

Prater, Mark, and C. L. Carden. Silent Storm: Finding Spiritual Shelter During Hepatitis C. New Hope Publishers (AL), 2005.

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26

Michaels, Gabriel. The Legendary Bulldog. BookShelf Press, 2006.

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27

Witkos, Maciej. Predictors of antiviral therapy in a cohort of hepatitis C patients. 2004.

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28

Wilson, Deanna. Hepatitis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0035.

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Hepatitis A (HAV) and E (HEV) viruses are spread via the fecal-oral route. Hepatitis B virus (HBV) exposure is via occupational or recreational activities. Hepatitis D virus (HDV; also spread parentally) can only coinfect or superinfect those with chronic HBV. Hepatitis C (HCV) transmission is predominantly parenteral; the highest risk group is injection drug users. Prodromal-period patients with acute hepatitis present with vague constitutional symptoms when serum transaminases peak, with elevated serum bilirubin and varying levels of hepatic protein synthesis impairment; during the icteric phase, patients develop abdominal pain, hepatomegaly, and jaundice. Acute hepatitis has limited therapy; treatment is predominantly supportive. However, most adults with acute phase HAV, HBV, HDV, and HEV spontaneously clear the virus. Most individuals with HCV develop chronic hepatitis. Patients with known HAV, HBV, or HEV exposures may be eligible for post-exposure prophylaxis to reduce their risk of infection.
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29

Porter, Lucinda K. Hepatitis C treatment one step at a time: Inspirational readings and practical tips for successful hepatitis c treatment. 2013.

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30

Canada, Canada Health, Canada. Hepatitis C Prevention, Support and Research Program., and Barrington Research Group Inc, eds. Collection of project case studies, Hepatitis C Prevention, Support and Research Program. [Ottawa]: Hepatitis C Prevention, Support and Research Program, 2003.

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31

Locke, Kenneth Andrew. Descriptive evaluation of the section 8 process for interferon treatment of patients with chronic hepatitis C in Ontario. 2006.

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32

Verslype, Chris. Detection of Hepatitis C Virus-Core & Envelope (E2) Proteins in the Liver: A Tool to Study Patients With Non-A to E Hepatitis (Acta Biomedica Lovansiensa, 261). Leuven Univ Pr, 2002.

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33

Protocol to estimate mortality from cirrhosis and hepatocellular carcinoma attributable to viral hepatitis B and C. Pan American Health Organization, 2021. http://dx.doi.org/10.37774/9789275123768.

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One of the goals to be achieved by 2030 of the Global Health Sector Strategy on Viral Hepatitis 2016-2021 is to reduce mortality from hepatitis viruses B (HBV) and C (HCV). To measure and monitor it, countries need to implement a systematic process to generate national estimates of mortality from viral hepatitis, which many lack. This document is aimed at the institutions and/or ministries in charge of monitoring progress in each country. The main objective of this protocol is to present simple methods to estimate the proportion of patients with cirrhosis and hepatocellular carcinoma who have HBV and HCV infection, and then calculate the national mortality due to these sequelae attributable to viral hepatitis, preferably within a surveillance system. In addition, a general framework is provided on how the surveillance system should function, how to collect the data, and ethical considerations. The surveillance system will be based on sentinel centers where information will be collected from patients with cirrhosis and hepatocellular carcinoma. These data will be used to estimate the fraction of cirrhosis and hepatocellular carcinoma attributable to HBV and HCV. On the other hand, data will be collected on the number of deaths nationwide from cirrhosis and hepatocellular carcinoma. With this information, mortality from cirrhosis and hepatocellular carcinoma attributable to HBV and HCV will be estimated.
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34

editor, Flight Nancy, ed. Demon in my blood: My fight with Hep C-- and a miracle cure. Greystone Books, 2017.

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35

Huyghebaert, Trudy. Is the Child-Pugh classification or other factors useful to predict hepatotoxicity following HAART in patients co-infected with HIV and hepatitis C? 2000.

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36

Berden, Jo H. M., and Jack F. M. Wetzels. Immunological investigation of the patient with renal disease. Edited by Christopher G. Winearls. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0017.

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Laboratory techniques (electrophoresis, indirect immunofluorescence, ELISA, and immunoblotting) required for immunological investigation of the patient with renal disease are described. Renal disease-related aspects of immunoglobulins (immunoglobulin A, paraproteins, cryoglobulins), complement, antinuclear antibodies, anti-C1q antibodies, antineutrophil cytoplasmic antibodies, anti-glomerular basement membrane antibodies, antipodocyte antibodies, antiphospholipid antibodies, and antimicrobial responses (streptococci, hepatitis C, hepatitis B) are reviewed. Laboratory assays which evaluate the immune response, in particular the identification of (auto)-antibodies are valuable tools in establishing a diagnosis and/or monitoring of the activity of the disease. Guidelines are given for immunological studies in patients with specific renal syndromes including nephrotic syndrome, rapidly progressive glomerulonephritis, systemic lupus erythematosus, and thrombotic microangiopathy.
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37

Primedia. PULSE Plus: Patient Assisted Medications (Pulse Plus). Delmar Learning, 2000.

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38

MD, Barbara McGovern. Care of the Patient with Hepatitis C Virus Infection, An Issue of Infectious Disease Clinics. Saunders, 2012.

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39

Smith, Suzy. Hepatitis C Through a Patient's Eyes; Hope for Healing. WingSpan Press, 2006.

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40

Maier. Hepatitis C - Hepatitisfolgen. Ratgeber für Patienten und ihre Angehörigen. Thieme, Stuttgart, 2002.

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41

Winston, Jonathan, Etti Zeldis, John A. Grimaldi, and Esteban Martínez. HIV-Associated Nephropathy, End-Stage Renal Disease, Dialysis, and Kidney Transplant. Edited by Mary Ann Cohen, Jack M. Gorman, Jeffrey M. Jacobson, Paul Volberding, and Scott Letendre. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199392742.003.0044.

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Antiretroviral therapy has changed the phenotype of HIV-related kidney disease to a more chronic disease model. In addition to HIV-associated nephropathy (HIVAN), patients with HIV may experience kidney dysfunction related to other chronic illnesses, such as diabetes, hypertension, and hepatitis C. Patients with HIV should be monitored for the development of chronic kidney disease and the potential nephrotoxicity of antiretroviral therapy. For patients with HIV who progress to end-stage renal disease, the outcomes on dialysis and management of the dialysis procedure are similar to the outcomes of patients without HIV. Renal transplantation is a promising treatment option for HIV patients with end-stage renal disease, despite certain barriers inherent in the transplant evaluation process. Concomitant HIV and end-stage renal disease, with the stress of dialysis, can exacerbate psychiatric illness.
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42

Lopes, Eurides, and Jennifer Husson. Solid Organ Transplantation in HIV-Infected Individuals. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190493097.003.0025.

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End-organ disease has become a major cause of morbidity and mortality in HIV-infected patients due to increased life expectancy, increasing the demand for organ transplantation in these patients. The care of HIV-infected transplant recipients warrants a multidisciplinary team approach, including the transplant team, pharmacists, infectious disease specialists, nurses, and patients and their families. The immunosuppression of HIV-infected recipients post-transplant does not appear to further advance HIV disease. The post-transplant risk for HIV-infected recipients of opportunistic infections does not appear to be increased by immunosuppression. However, the overall rate of infections is high, and it is even higher in hepatitis C virus (HCV) co-infected transplant recipients. HIV/HCV co-infected recipients have worse outcomes compared to both liver and kidney HIV-infected recipients.
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43

Bacon, Bruce R. The Non-responding Hepatitis C Patient: Options And Variables (Round Table Series). Royal Society of Medicine Press, 2000.

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44

Fabrizi, Fabrizio, and Patrice Cacoub. The patient with cryoglobulinaemia. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0151.

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AbstractCryoglobulinaemia is characterized by the presence in the blood of proteins showing the that precipitate when serum is cooled. Clinically recognised cryoprecipitates are predominantly immunoglobulin-containing. In Type 1 cryoglobulinaemia, the precipitate is formed from a monoclonal paraprotein, usually IgG. In Type 2, a monoclonal IgM binds IgG to form a mixed precipitate. Type 3 cryoglobulins do not contain a monoclonal element.Type 1 cryoglobulins are a rare cause of renal disease, but cause a membranoproliferative glomerulonephritis (MPGN) with nephrotic syndrome and haematuria and usually with severe cutaneous involvement.Type 2 is most typically associated with renal disease, again characterized by MPGN and haematuria, with variable cutaneous signs and vasculitis in other organs. Many cases are associated with Hepatitis C virus (HCV) infection – but not all.Therapeutic approaches include optimal antiviral regimen, immunosuppressive therapy (corticosteroids, rituximab, and cytotoxic agents), and plasma exchange. Treatment of HCV-related mixed cryoglobulinaemia vasculitis should be adjusted according to the clinico-biological presentation.
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45

The Official Patient's Sourcebook on Hepatitis C Infection: A Revised and Updated Directory for the Internet Age. Icon Health Publications, 2005.

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46

Publications, ICON Health. The 2002 Official Patient's Sourcebook on Hepatitis C: A Revised and Updated Directory for the Internet Age. Icon Health Publications, 2002.

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47

VA health care: Telephone service should be more accessible to patients : report to the Honorable Frank H. Murkowski, United States Senate. Washington, D.C: The Office, 1991.

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48

Office, General Accounting. VA health care: Allegations concerning VA's patient mortality study : report to the chairman, Committee on Veterans' Affairs, U.S. Senate. Washington, D.C: U.S. General Accounting Office, 1989.

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49

VA health care: Inadequate enforcement of federal ethics requirements at VA medical centers : supplement to report to the Chairman, Subcommittee on Oversight and Investigations, Committee on Veterans' Affairs, House of Representatives. Washington, D.C: The Office, 1993.

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50

VA health care: Resource allocation methodology should improve VA's financial management : briefing report to the Committee on Veterans' Affairs, United States Senate. Washington, D.C: The Office, 1987.

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