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Journal articles on the topic 'Hepatitis B virus'

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Published: 4 June 2021
Last updated: 31 July 2024

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1

Schaefer, Stephan. "Hepatitis B virus taxonomy and hepatitis B virus genotypes." World Journal of Gastroenterology 13, no. 1 (2007): 14. http://dx.doi.org/10.3748/wjg.v13.i1.14.

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2

AYGEN, Bilgehan, Mustafa Kemal ÇELEN, İftihar KÖKSAL, Selma TOSUN, Oğuz KARABAY, Tansu YAMAZHAN, Orhan YILDIZ, Celal AYAZ, and Fehmi TABAK. "The Prevalence and Epidemiological Characteristics of Hepatitis B Virus and Hepatitis C Virus Coinfection in Turkey." Turkiye Klinikleri Journal of Medical Sciences 33, no. 5 (2013): 1245–49. http://dx.doi.org/10.5336/medsci.2012-32319.

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3

Adil, B., O. Fatih, I. Volkan, B. Bora, E. Veysel, K. Koray, K. Cemalettin, I. Burak, and Y. Sezai. "Hepatitis B Virus and Hepatitis D Virus Recurrence in Patients Undergoing Liver Transplantation for Hepatitis B Virus and Hepatitis B Virus Plus Hepatitis D Virus." Transplantation Proceedings 48, no. 6 (July 2016): 2119–23. http://dx.doi.org/10.1016/j.transproceed.2016.02.076.

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4

Godoy, C., S. Sopena, J. Gregori, D. Tabernero, M. Homs, R. Casillas, A. Ruiz, et al. "Hepatitis B virus quasispecies complexity is lower in hepatitis B virus–hepatitis D virus infection than in hepatitis B virus monoinfection." Journal of Hepatology 66, no. 1 (2017): S483—S484. http://dx.doi.org/10.1016/s0168-8278(17)31360-0.

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5

Nayak, Akshatha P., Insha Firoz, Ravindra Prabhu, Jayanth Nayak, Veena NK, and Megha Nagaraj Nayak. "Development of Immunity to Hepatitis B Virus Following Hepatitis B Vaccination in Hemodialysis Patient." Annals of International Medical and Dental Research 9, no. 2 (April 2023): 20–23. http://dx.doi.org/10.53339/aimdr.2023.9.2.4.

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Background: Hepatitis B infection is common in Dialysis population. Hemodialysis patients have high risk of hepatitis B virus transmission not only due to frequent blood or blood product transmission, decreased response to Hepatitis B vaccine and length on hemodialysis but also due to their immunosuppressed state. Hepatitis B vaccination has the potential to reduce the risk of HBsAg infection in dialysis units. Effective vaccination, blood donor screening, the use of erythropoietin and the isolation of HBV carriers have successfully regulated HBV infection in hemodialysis units (1). This study aims to assess the immunity to HBV the seroconversion of HBsAg infection in hemodialysis unit. This retrospective observational study evaluated serological markers, hepatitis B vaccination status and co morbidities which can affect the immunity levels of patients undergoing hemodialysis. The patient’s data were collected from laboratory investigations and patient record for analysis. Out of 153 CKD-5D patients on maintenance hemodialysis, 39 patients had anti HBs titer <10U/ml, 30 patients had anti HBs titer between 10-100U/ml, 38 patients had anti HBs titer between100-1000 U/ml, 21 patients had anti HBs titer >1000U/ml and 24 patients didn’t check their titer value. Hypertension was the common co morbidity followed by anaemia and diabetes mellitus.
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6

Genesca, J., R. Jardi, M. Buti, L. Vives, S. Prat, J. I. Esteban, R. Esteban, and J. Guardia. "Hepatitis b virus replication in acute hepatitis B, acute hepatitis B virus-hepatitis delta virus coinfection and acute hepatitis delta superinfection." Hepatology 7, no. 3 (May 1987): 569–72. http://dx.doi.org/10.1002/hep.1840070325.

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7

Ganem, Don. "Hepatitis B Virus." Expert Review of Anti-infective Therapy 1, no. 3 (September 2003): 357–58. http://dx.doi.org/10.1586/14787210.1.3.357.

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8

XIAO, JIANBO, Lei Zhang, XIAOQING CHEN, Ming Xu *, and XINYU JIANG. "HEPATITIS B VIRUS." Professional Medical Journal 14, no. 02 (September 6, 2007): 241–47. http://dx.doi.org/10.29309/tpmj/2007.14.02.4883.

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Dried leaves of Marchantia convoluta are largely used to protect livers,and to treat tumefaction of skins in China. Flavonoids from Marchantia Convoluta (MCF) was one of the mostpotentially effective anti-inflammatory. MCF was studied here for its ability to inhibit the proliferation of 2,2,15 cells(clone cells derived from HepG2 cells that were transected with a plasmid containing HBV, DNA). All concentrations(5,10,20 and 40 :g/ml) of MCF inhibit hepatitis B surface antigen (HbsAg) and hepatitis B E antigen (HbeAg) in thecultured medium released from 2.2.15 cells. Analysis of morphological changes of MCF-treated phase- contrastmicroscope revealed a possible model of action for MCF to inhibit Proliferation of 2.2.15 cells by inducing apoptosis.
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9

Bredfeldt, James E. "Hepatitis B virus." Postgraduate Medicine 78, no. 6 (November 1985): 71–83. http://dx.doi.org/10.1080/00325481.1985.11699185.

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10

Seitz, R. "Hepatitis B Virus." Transfusion Medicine and Hemotherapy 27, no. 5 (2000): 226–34. http://dx.doi.org/10.1159/000025279.

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11

Tiollais, Pierre, and Marie-Annick Buendia. "Hepatitis B Virus." Scientific American 264, no. 4 (April 1991): 116–23. http://dx.doi.org/10.1038/scientificamerican0491-116.

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12

Hassanein, Tarek I. "Hepatitis B Virus." Clinics in Liver Disease 20, no. 4 (November 2016): i. http://dx.doi.org/10.1016/s1089-3261(16)30077-0.

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13

Shih, Chiaho, Ching-Chun Yang, Gansukh Choijilsuren, Chih-Hsu Chang, and An-Ting Liou. "Hepatitis B Virus." Trends in Microbiology 26, no. 4 (April 2018): 386–87. http://dx.doi.org/10.1016/j.tim.2018.01.009.

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14

&NA;. "Hepatitis B Virus." Pediatric Infectious Disease Journal 32 (November 2013): K—1—K—17. http://dx.doi.org/10.1097/01.inf.0000437867.26573.9a.

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15

Higgins, Patricia A. "Hepatitis B virus." Journal of the American Academy of Physician Assistants 29, no. 8 (August 2016): 48–49. http://dx.doi.org/10.1097/01.jaa.0000484310.19556.82.

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16

&NA;. "Hepatitis B virus." Advances in Anatomic Pathology 1, no. 2 (September 1994): 108. http://dx.doi.org/10.1097/00125480-199409000-00015.

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17

Yoffe, Boris, and Christine A. Noonan. "Hepatitis B virus." Digestive Diseases and Sciences 37, no. 1 (January 1992): 1–9. http://dx.doi.org/10.1007/bf01308334.

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18

Sir, Donna, David K. Ann, and Jing-hsiung James Ou. "Autophagy by hepatitis B virus and for hepatitis B virus." Autophagy 6, no. 4 (May 16, 2010): 548–49. http://dx.doi.org/10.4161/auto.6.4.11669.

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19

Lamontagne, Jason. "Hepatitis B virus and microRNAs: Complex interactions affecting hepatitis B virus replication and hepatitis B virus-associated diseases." World Journal of Gastroenterology 21, no. 24 (2015): 7375. http://dx.doi.org/10.3748/wjg.v21.i24.7375.

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20

Anjum, Muhammad Usman, Muhammad Safdar Khan, Nazar Muhamamd Afridi, and Syed Humayun Shah. "HEPATITIS B AND C VIRUS INFECTIONS." Professional Medical Journal 22, no. 10 (October 10, 2015): 1284–88. http://dx.doi.org/10.29309/tpmj/2015.22.10.981.

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Patients with end stage renal disease require haemodialysis as a part of theirtreatment. The incidence of hepatitis B and C virus infection is quite high in patients undergoingmaintenance haemodialysis than in general population. This risk is specifically associatedwith use of blood and its products as well as repeated intravascular access in these patients.Objectives: To determine the seropositivity of hepatitis B and C virus infection in patientsreceiving haemodialysis. Design: Descriptive cross sectional study. Setting: NephrologyDepartment, Ayub Teaching Hospital, Abbottabad, Pakistan. Period: From October 2014 toApril 2015. Methods: Five hundred patients were included in the study based on inclusionand exclusion criteria. Demographic data was recorded and detailed history was taken fromeach patient specifically about the no of blood transfusions received, the frequency of dialysisand the dialysis done in other centers. All patients were checked for the presence of hepatitisB surface antigen (HbsAg) and antibodies to HCV using third generation enzyme linkedimmunoassay (ELISA). Results: Mean age of study sample was 46±5 years with 60.8 % males.Incidence of hepatitis positive cases was 164 (32.8 %), out of which 66 (13.2 %) patients wereHBV positive and 98 (19.08 %) patients were HCV positive. The hepatitis B and C infectionswere more common in males than females. Seropositivity of HBV and HCV was higher (HBV18.1 % and HCV 22.2 %) among haemodialysis patients who have received more than threeblood transfusions. The frequency of HBV and HCV infections increases significantly with theincrease in frequency of dialysis, with 49 (17.11 %) patients were HBV positive and 70 (24.5%) patients were HCV positive cases, who have received haemodialysis for more than fivetimes. There were 48 (15.7 %) HBV positive cases as well as 68 (22.3 %) HCV positive cases inpatients who have received their treatment from a single center. Conclusion: Hepatitis B andC infection is quite common in patients undergoing haemodialysis. The risk of these infectionscan be reduced by following infection control guidelines, proper training of the staff and strictscreening of blood and blood products specifically for hepatitis C virus.
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21

Sekiba, Kazuma, Motoyuki Otsuka, Motoko Ohno, Mari Yamagami, Takahiro Kishikawa, Tatsunori Suzuki, Rei Ishibashi, Takahiro Seimiya, Eri Tanaka, and Kazuhiko Koike. "Hepatitis B virus pathogenesis: Fresh insights into hepatitis B virus RNA." World Journal of Gastroenterology 24, no. 21 (June 7, 2018): 2261–68. http://dx.doi.org/10.3748/wjg.v24.i21.2261.

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22

Mowat, A. P., and G. Mieli-Vergani. "Perinatal hepatitis B virus detection by hepatitis B virus-DNA analysis." Archives of Disease in Childhood 60, no. 5 (May 1, 1985): 495–96. http://dx.doi.org/10.1136/adc.60.5.495-b.

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23

De Virgiliis, S., F. Frau, G. Sanna, M. P. Turco, A. L. Figus, G. Cornacchia, and A. Cao. "Perinatal hepatitis B virus detection by hepatitis B virus-DNA analysis." Archives of Disease in Childhood 60, no. 1 (January 1, 1985): 56–58. http://dx.doi.org/10.1136/adc.60.1.56.

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24

DE COCK, KEVIN M. "Hepatitis B Virus DNA in Fulminant Hepatitis B." Annals of Internal Medicine 105, no. 4 (October 1, 1986): 546. http://dx.doi.org/10.7326/0003-4819-105-4-546.

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25

Triani, Eva. "Infeksi Hepatitis B Tersamar (Occult Hepatitis B Infection) dan Kanker Hati Primer." Unram Medical Journal 5, no. 3 (September 28, 2016): 45. http://dx.doi.org/10.29303/jku.v5i3.303.

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Infeksi virus hepatitis B tersamar merupakan infeksi yang masih menjadi masalah karena prevalensinya yang cukup tinggi dan berpengaruh pada keselamatan terutama dalam praktik donor darah. Virus Hepatitis B merupakan virus yang paling sering ditransmisikan melalui transfusi dan terdapat kemungkinan transmisi berasal dari virus hepatitis B tersamar. Padahal, telah diketahui bahwa terdapat hubungan yang erat antara infeksi hepatitis B tersamar dan kejadian sirosis dan kanker hati primer.
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26

Jung, Janghan, and Mindie H. Nguyen. "Liver-Related Mortality in Hepatitis B Virus Core Antibody+/Hepatitis B Virus Surface Antigen− Patients: Occult Hepatitis B Virus, Hepatitis B Virus Reactivation, and Hepatocellular Carcinoma Development." American Journal of Gastroenterology 118, no. 1 (September 16, 2022): 24–25. http://dx.doi.org/10.14309/ajg.0000000000002030.

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27

Soni, Dr Payal, Dr Dipa Kinariwala, Dr Neeta Khandelwal, Dr Mehul Patel, Dr P. K. Shah Dr. P. K. Shah, and Dr M. M. Vegad Dr. M. M. Vegad. "Co-Infection of Hepatitis C Virus and Hepatitis B Virus in Human Immuno Deficiency Virus Positive Population in Ahmedabad, Gujarat." International Journal of Scientific Research 2, no. 9 (June 1, 2012): 17–18. http://dx.doi.org/10.15373/22778179/sep2013/158.

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28

GUPTA, NEHA, SIMA K. BHATT, and AMI M. SHAH. "Seroprevalence of Hepatitis-B Virus, Human Immunodeficiency Virus & Hepatitis-C Virus in Corneal Donors in Tertiary Care Centre, Ahmedabad." International Journal of Scientific Research 3, no. 2 (June 1, 2012): 455–56. http://dx.doi.org/10.15373/22778179/feb2014/150.

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29

Aliu, Tawakalitu Bidemi, Abafi J. Majiyebo, Amos Ndarubu Tsado, Habiba Abdulsalam Ibrahim, and Eustace B. Berinyuy. "Biology and molecular pathogenesis of hepatitis B virus infection." BIOMED natural and applied science 2, no. 2 (June 26, 2022): 28–36. http://dx.doi.org/10.53858/bnas02022836.

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Hepatitis B virus (HBV) remains a global public health infection. Over the past several decades, the basic principles of HBV gene expression and replication as well as the viral and host determinants governing infection outcomes have been largely uncovered. The clinical manifestations of HBV infection vary from an acute and chronic form of the disease. During the acute phase of the infection, the disease manifestations vary from subclinical hepatitis to anicteric hepatitis, icteric hepatitis, and fulminant hepatitis while during the chronic infection, manifest in different from ranging from asymptomatic carrier state to chronic hepatitis, cirrhosis, and hepatocellular carcinoma. The clinical outcome of the infection depends upon the level of HBV replication, age at infection, and the immune status of the host. HBV integration into the host genome often serves as a relevant source of hepatitis B surface antigen (HBsAg) expression during chronic infection, possibly triggering an immune response. If HBV is not eliminated, a delicate balance between viral replication and immune defence prevails which may lead to chronic hepatitis and liver cirrhosis. It is generally acknowledged that the humoral antibody response contributes to the clearance of circulating virus particles and the prevention of viral spread within the host while the cellular immune response eliminates infected cells.
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30

Estévez Escobar, M. "New therapeutic approaches in Hepatitis B and Hepatitis D." Revista Andaluza de Patología Digestiva 46, no. 5 (October 30, 2023): 258–68. http://dx.doi.org/10.37352/2023465.3.

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Resumen El virus de la hepatitis B y el virus de la hepatitis D ocasionan un espectro variable de enfermedades hepáticas, desde cuadros asintomáticos hasta hepatitis fulminante o hepatitis crónica. Debido a que, en su forma crónica, la enfermedad puede tener consecuencias fatales sobre el huésped por desarrollo de cirrosis, disfunción hepática y desarrollo de hepatocarcinoma, es importante el diagnóstico precoz para iniciar un tratamiento que cambie la historia natural de la enfermedad, mejorando así el pronóstico del sujeto. En la actual revisión, se recorren las opciones disponibles de tratamiento en hepatitis B así como las novedades terapéuticas que persiguen obtener, no solo la supresión viral, sino la curación funcional. Además, se describe el actual escenario terapéutico en hepatitis D y el impacto que podrán tener sobre su manejo las nuevas moléculas en investigación, especialmente, Bulevirtide.
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31

Beck, Juergen. "Hepatitis B virus replication." World Journal of Gastroenterology 13, no. 1 (2007): 48. http://dx.doi.org/10.3748/wjg.v13.i1.48.

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32

Bruss, Volker. "Hepatitis B virus morphogenesis." World Journal of Gastroenterology 13, no. 1 (2007): 65. http://dx.doi.org/10.3748/wjg.v13.i1.65.

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33

Zulqarnain, Arif, Imran Iqbal, and Naveed Anjum. "HEPATITIS–B VIRUS INFECTION;." Professional Medical Journal 21, no. 05 (December 14, 2018): 950–55. http://dx.doi.org/10.29309/tpmj/2014.21.05.2526.

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Objective: To describe the clinical presentations of hepatitis B virus infectionin children. Methodology: Children presenting with symptoms of liver diseases and otherdiseases who were found to be HBsAg positive by screening or ELISA method were enrolled.Children suffering from thalassemia, hemophilia and hemolytic anemia, which need multipletransfusions, were excluded. On the basis of history, examination and investigations the clinicalpresentation of the patient was categorized. Basic demographic data, relevant clinical history,physical examination, lab investigations and clinical presentations category were entered in thepredesigned proforma. As this is the descriptive study, no hypothesis were required. Design:Descriptive case series. Setting: Paediatric unit-2 NishtarHospital Multan. Period: 16th May2012 to 15th November 2012. Results: Study results consist of relative frequencies of differentclinical presentations of HBsAg positive patients. Fifty children who were HBsAg positive wereenrolled in a six month period. Out of 50 patients, 21 (42%) were of hepatic encephalopathy,14 (28%) were with acute hepatitis, 12 (24%) were cirrhosis, 2 (4%) were asymptomatic carrierand 1 (2%) was presented with chronic hepatitis B. There were 40 (80%) males and 10 (20%)females. The overall male to female ratio was 4:1. Conclusions: Most common presentationwas hepatic encephalopathy which has a very bad prognosis, next comes the acute hepatitisand then cirrhosis. There is another inference that males are more and severely affected byhepatitis-B virus infection.
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34

Cottone, James A., and Raghunath Puttaiah. "HEPATITIS B VIRUS INFECTION." Dental Clinics of North America 40, no. 2 (April 1996): 293–307. http://dx.doi.org/10.1016/s0011-8532(22)00143-4.

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35

Dienstag, Jules L. "Hepatitis B Virus Infection." New England Journal of Medicine 359, no. 14 (October 2, 2008): 1486–500. http://dx.doi.org/10.1056/nejmra0801644.

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36

MacLachlan, J. H., and B. C. Cowie. "Hepatitis B Virus Epidemiology." Cold Spring Harbor Perspectives in Medicine 5, no. 5 (May 1, 2015): a021410. http://dx.doi.org/10.1101/cshperspect.a021410.

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37

Jang, Yeonsoo, Sunjoo Boo, and Hyera Yoo. "Hepatitis B Virus Infection." Gastroenterology Nursing 41, no. 5 (2018): 388–95. http://dx.doi.org/10.1097/sga.0000000000000335.

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38

Hicks, Ralph A., John W. Cullen, Mary Anne Jackson, and V. Fred Burry. "Hepatitis B Virus Vaccine." Clinical Pediatrics 28, no. 8 (August 1989): 359–65. http://dx.doi.org/10.1177/000992288902800805.

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39

Tiollais, Pierre, Christine Pourcel, and Anne Dejean. "The hepatitis B virus." Nature 317, no. 6037 (October 1985): 489–95. http://dx.doi.org/10.1038/317489a0.

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40

Lee, William M. "Hepatitis B Virus Infection." New England Journal of Medicine 337, no. 24 (December 11, 1997): 1733–45. http://dx.doi.org/10.1056/nejm199712113372406.

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41

Liaw, Yun-Fan, and Chia-Ming Chu. "Hepatitis B virus infection." Lancet 373, no. 9663 (February 2009): 582–92. http://dx.doi.org/10.1016/s0140-6736(09)60207-5.

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42

Kramvis, Anna, Michael Kew, and Guido François. "Hepatitis B virus genotypes." Vaccine 23, no. 19 (March 2005): 2409–23. http://dx.doi.org/10.1016/j.vaccine.2004.10.045.

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43

Brunetto, Maurizia Rossana, Ulisses Aragon Rodriguez, and Ferruccio Bonino. "Hepatitis B Virus Mutants." Intervirology 42, no. 2-3 (1999): 69–80. http://dx.doi.org/10.1159/000024968.

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44

Trépo, Christian, Henry L. Y. Chan, and Anna Lok. "Hepatitis B virus infection." Lancet 384, no. 9959 (December 2014): 2053–63. http://dx.doi.org/10.1016/s0140-6736(14)60220-8.

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45

Hassanein, Tarek I. "Hepatitis B Virus Update." Clinics in Liver Disease 23, no. 3 (August 2019): i. http://dx.doi.org/10.1016/s1089-3261(19)30030-3.

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46

Chang, Mei-Hwei. "Hepatitis B virus infection." Seminars in Fetal and Neonatal Medicine 12, no. 3 (June 2007): 160–67. http://dx.doi.org/10.1016/j.siny.2007.01.013.

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47

Not Available, Not Available. "Hepatitis-B-Virus (HBV)." Bundesgesundheitsblatt - Gesundheitsforschung - Gesundheitsschutz 43, no. 3 (March 15, 2000): 240–48. http://dx.doi.org/10.1007/s001030050047.

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48

MacDermott, Richard. "Fingerprinting hepatitis B virus." Gastroenterology 90, no. 4 (April 1986): 1085–86. http://dx.doi.org/10.1016/0016-5085(86)90891-7.

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49

Poss, Jane. "Hepatitis B virus infection." Journal of Pediatric Health Care 4, no. 3 (May 1990): 158. http://dx.doi.org/10.1016/0891-5245(90)90059-f.

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50

Hassanein, Tarek I. "Hepatitis B Virus Update." Clinics in Liver Disease 23, no. 3 (August 2019): xiii—xiv. http://dx.doi.org/10.1016/j.cld.2019.05.002.

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