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Journal articles on the topic 'Hemochromatosis'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 July 2024

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1

Torbenson, Michael S., and Lori A. Erickson. "Hemochromatosis." Mayo Clinic Proceedings 97, no. 2 (February 2022): 423–24. http://dx.doi.org/10.1016/j.mayocp.2021.12.008.

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2

McElroy, Vanessa. "Hemochromatosis." Journal of Diagnostic Medical Sonography 25, no. 6 (October 22, 2009): 325–28. http://dx.doi.org/10.1177/8756479309344625.

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3

Ashinsky, Douglas. "Hemochromatosis." Postgraduate Medicine 91, no. 4 (March 1992): 137–45. http://dx.doi.org/10.1080/00325481.1992.11701249.

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4

Vogt, J. H. "Hemochromatosis." Acta Pathologica Microbiologica Scandinavica 21, no. 3 (August 14, 2009): 461–71. http://dx.doi.org/10.1111/j.1699-0463.1944.tb04959.x.

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5

Ramrakhiani, Sanjay, and Bruce R. Bacon. "Hemochromatosis." Journal of Clinical Gastroenterology 27, no. 1 (July 1998): 41–46. http://dx.doi.org/10.1097/00004836-199807000-00008.

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6

Wortsman, Jacobo. "Hemochromatosis." New England Journal of Medicine 335, no. 24 (December 12, 1996): 1815. http://dx.doi.org/10.1056/nejm199612123352406.

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7

Seligman, Paul A. "Hemochromatosis." Primary Care Case Reviews 4, no. 1 (March 2001): 40–46. http://dx.doi.org/10.1097/00129300-200103000-00007.

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8

Kent Holland, H., and Jerry L. Spivak. "Hemochromatosis." Medical Clinics of North America 73, no. 4 (July 1989): 831–45. http://dx.doi.org/10.1016/s0025-7125(16)30641-1.

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9

Powell, Lawrie W., and Thomas R. Yapp. "HEMOCHROMATOSIS." Clinics in Liver Disease 4, no. 1 (February 2000): 211–28. http://dx.doi.org/10.1016/s1089-3261(05)70104-5.

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10

Adams, Paul C. "Hemochromatosis." Clinics in Liver Disease 8, no. 4 (November 2004): 735–53. http://dx.doi.org/10.1016/j.cld.2004.06.002.

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11

Uba, Angela, and James Litynsky. "HEMOCHROMATOSIS." Southern Medical Journal 92, Supplement (November 1999): S32. http://dx.doi.org/10.1097/00007611-199911001-00064.

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12

Regan, Elizabeth Neville. "Hemochromatosis." Nurse Practitioner 34, no. 6 (June 2009): 25–29. http://dx.doi.org/10.1097/01.npr.0000352285.81981.d5.

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13

DeSOUZA, PATRICIA. "Hemochromatosis." Gastroenterology Nursing 21, no. 5 (September 1998): 210–12. http://dx.doi.org/10.1097/00001610-199809000-00004.

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14

Olynyk, John K., and Grant A. Ramm. "Hemochromatosis." New England Journal of Medicine 387, no. 23 (December 8, 2022): 2159–70. http://dx.doi.org/10.1056/nejmra2119758.

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15

Bacon, Bruce R. "Hemochromatosis." Current Treatment Options in Gastroenterology 2, no. 1 (January 1999): 58–60. http://dx.doi.org/10.1007/s11938-999-0019-0.

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16

Crosby, William H. "Hemochromatosis." Archives of Internal Medicine 146, no. 6 (June 1, 1986): 1209. http://dx.doi.org/10.1001/archinte.1986.00360180229034.

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17

Crosby, William H. "Hemochromatosis." Archives of Internal Medicine 146, no. 10 (October 1, 1986): 1910. http://dx.doi.org/10.1001/archinte.1986.00360220054009.

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18

Peters, Sonya. "Hemochromatosis." JAAPA 36, no. 10 (October 2023): 41–42. http://dx.doi.org/10.1097/01.jaa.0000977708.62318.43.

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19

Parker, Christina N., Kathleen J. Finlayson, Emma J. Hall, Natasha Pitman, Wing Kei Chu, and Daniel F. Wallace. "Exploring the Association between Hemochromatosis and Lower-Limb Venous Disease." Advances in Skin & Wound Care 37, no. 1 (January 2024): 32–39. http://dx.doi.org/10.1097/asw.0000000000000080.

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ABSTRACT OBJECTIVE Chronic venous disease is a circulatory system dysfunction that has the potential to lead to venous leg ulceration. Although research on the influence of specific gene variants on chronic venous disease has been limited, a few studies have reported an association between hemochromatosis and chronic venous disease. However, no studies have looked at the prevalence of lower-limb venous disease and leg ulcers in people with hemochromatosis. This study aimed to review the existing literature for any association between venous disease and hemochromatosis and investigate the prevalence of venous disease and leg ulcers in people with hemochromatosis. METHODS Scoping systematic literature review and cross-sectional study surveying people with hemochromatosis. RESULTS This scoping systematic literature review included nine articles and indicated a link between hemochromatosis and venous disease/leg ulcers, although further studies are needed to support this link. Analysis of survey results from people with hemochromatosis found a 9.2% prevalence of leg ulcers in those with self-reported hemochromatosis, considerably higher than the 1% to 3% expected, suggesting that hemochromatosis gene variants may be associated with the pathogenesis of chronic venous disease and leg ulcers. CONCLUSIONS This is the first known study to complete a review of the literature regarding hemochromatosis and venous leg ulcers and document the association between hemochromatosis and venous disease/leg ulcers. There is a lack of research in this area and hence limited evidence to guide practice.
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20

Oh, Chang-Kyu, and Yuseok Moon. "Dietary and Sentinel Factors Leading to Hemochromatosis." Nutrients 11, no. 5 (May 10, 2019): 1047. http://dx.doi.org/10.3390/nu11051047.

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Although hereditary hemochromatosis is associated with the mutation of genes involved in iron transport and metabolism, secondary hemochromatosis is due to external factors, such as intended or unintended iron overload, hemolysis-linked iron exposure or other stress-impaired iron metabolism. The present review addresses diet-linked etiologies of hemochromatosis and their pathogenesis in the network of genes and nutrients. Although the mechanistic association to diet-linked etiologies can be complicated, the stress sentinels are pivotally involved in the pathological processes of secondary hemochromatosis in response to iron excess and other external stresses. Moreover, the mutations in these sentineling pathway-linked genes increase susceptibility to secondary hemochromatosis. Thus, the crosstalk between nutrients and genes would verify the complex procedures in the clinical outcomes of secondary hemochromatosis and chronic complications, such as malignancy. All of this evidence provides crucial insights into comprehensive clinical or nutritional interventions for hemochromatosis.
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21

Zoller, Heinz, and Benjamin Henninger. "Pathogenesis, Diagnosis and Treatment of Hemochromatosis." Digestive Diseases 34, no. 4 (2016): 364–73. http://dx.doi.org/10.1159/000444549.

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Hemochromatosis is a common cause of chronic liver disease and HFE genotyping allows decisive and non-invasive diagnosis. Molecular and clinical genetic studies have led to the identification of genes other than HFE in patients with inherited diseases associated with increased hepatic iron storage that can cause hemochromatosis, which adds complexity to a diagnostic approach to patients with suspected hemochromatosis. Despite major advances in genetics, hepatic iron quantification by non-invasive methods therefore remains the key to the diagnosis of hemochromatosis. Although associated with homozygosity for the C282Y polymorphism in the HFE gene in >80% of patients, hemochromatosis is a complex genetic disease with strong environmental disease modifiers. Testing for mutations in the non-HFE hemochromatosis genes transferrin receptor 2, hemojuvelin, HAMP and SLC40A1 is complex, costly and time-consuming. Demonstration of hepatic iron overload by liver biopsy or MRI is therefore required before such complex tests are carried out. The pathogenesis of chronic liver disease in hemochromatosis is mainly attributed to the redox potential of tissue iron, and only the more recent studies have focused on the toxic properties of circulating iron. Considering the fact that an increased saturation of transferrin and high iron in plasma are the hallmark of all hemochromatosis forms, an alternative view would be that toxic iron in the circulation is involved in the pathogenesis of hemochromatosis. Recent studies have shown an increased concentration of redox-active iron in plasma in patients with increased transferrin saturation. This finding supports the hypothesis that tissue iron may be the ‘smoking gun' of iron-induced organ damage. Taken together, caring for patients with suspected or established hemochromatosis still remains a challenge, where understanding the genetics, biochemistry and cell biology of hemochromatosis will aid better diagnosis and treatment of affected individuals.
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22

Press, Richard D. "Hereditary Hemochromatosis." Archives of Pathology & Laboratory Medicine 123, no. 11 (November 1, 1999): 1053–59. http://dx.doi.org/10.5858/1999-123-1053-hh.

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Abstract Objective.—To review the current state-of-the-art regarding the role of iron- and DNA-based testing on the detection, treatment, and prevention of hereditary hemochromatosis (HH), the most common single-gene disorder in white people. Sources.—Review of the medical literature, with particular emphasis on recent reports of the impact of DNA-based testing on the detection of symptomatic and presymptomatic patients with HH. Conclusions.—Hereditary hemochromatosis, a common autosomal recessive iron overload disorder (with a population prevalence of 0.3%–0.8%), is a common cause of preventable liver, heart, joint, and endocrine disease. Since the associated clinical signs and symptoms are nonspecific, an accurate HH diagnosis demands both a high index of suspicion and the direct laboratory demonstration of elevated iron parameters. The substantial public health burden of HH as a common, deadly, detectable, and treatable chronic disease has led the College of American Pathologists to recommend that “systematic screening for hemochromatosis is warranted for all persons over the age of 20 years.” The recent discovery that most HH cases are the result of a single well-conserved homozygous missense mutation (C282Y) within a novel transferrin-receptor binding protein (HFE) has given rise to diagnostic clinical tests for the DNA-based detection of this pathologic mutation. This direct HFE mutation test can now be used not only to confirm the diagnosis of HH in those with symptomatic disease, but also, perhaps more importantly, to detect those with presymptomatic iron overload in whom future disease manifestations may be prevented (with phlebotomy therapy).
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23

McDonnell, Sharon M., and David Witte. "Hereditary hemochromatosis." Postgraduate Medicine 102, no. 6 (December 1997): 83–94. http://dx.doi.org/10.3810/pgm.1997.12.378.

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24

Geller, Stephen A., and Fernando P. F. de Campos. "Hereditary hemochromatosis." Autopsy and Case Reports 5, no. 1 (2015): 7–10. http://dx.doi.org/10.4322/acr.2014.043.

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25

Eustace, Stephen J., Nancy D. Baker, Howard H. C. Lan, and David Dorfman. "Hemochromatosis Arthropathy." Radiologic Clinics of North America 34, no. 2 (March 1996): 441–45. http://dx.doi.org/10.1016/s0033-8389(22)00477-8.

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26

Hershko, Chaim. "Hemochromatosis redefined." Blood 139, no. 20 (May 19, 2022): 3001–2. http://dx.doi.org/10.1182/blood.2021014036.

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27

Prows, Cynthia A. "HEREDITARY HEMOCHROMATOSIS." Nursing Clinics of North America 35, no. 3 (September 2000): 707–17. http://dx.doi.org/10.1016/s0029-6465(22)02512-9.

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28

Murray, K. F., and K. V. Kowdley. "Neonatal Hemochromatosis." PEDIATRICS 108, no. 4 (October 1, 2001): 960–64. http://dx.doi.org/10.1542/peds.108.4.960.

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29

Olynyk, John K., and Bruce R. Bacon. "Hereditary hemochromatosis." Postgraduate Medicine 96, no. 5 (November 1994): 151–65. http://dx.doi.org/10.1080/00325481.1994.11945914.

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30

Pietrangelo, Antonello. "Hereditary Hemochromatosis." Annual Review of Nutrition 26, no. 1 (August 2006): 251–70. http://dx.doi.org/10.1146/annurev.nutr.26.061505.111226.

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31

Jonas, Maureen M., Yosef Aaron Kavveblum, and Rita Fojaco. "Neonatal Hemochromatosis." Journal of Pediatric Gastroenterology and Nutrition 6, no. 6 (November 1987): 984–88. http://dx.doi.org/10.1097/00005176-198711000-00029.

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32

Rand, Elizabeth B., Daniel T. McClenathan, and Peter F. Whitington. "Neonatal Hemochromatosis." Journal of Pediatric Gastroenterology and Nutrition 15, no. 3 (October 1992): 325–29. http://dx.doi.org/10.1097/00005176-199210000-00017.

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33

Starreveld, J. S., J. P. van Dijk, and H. G. van Eijk. "Neonatal Hemochromatosis." Journal of Pediatric Gastroenterology and Nutrition 17, no. 3 (October 1993): 347. http://dx.doi.org/10.1097/00005176-199310000-00029.

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34

Brissot, Pierre. "Hereditary hemochromatosis." Hematology 18, no. 6 (November 2013): 370–71. http://dx.doi.org/10.1179/1024533213z.000000000222.

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35

Kaltwasser, J. P., K. P. Schalk, and E. Werner. "Juvenile Hemochromatosis." Annals of the New York Academy of Sciences 526, no. 1 Hemochromatos (June 1988): 339–41. http://dx.doi.org/10.1111/j.1749-6632.1988.tb55521.x.

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36

Passen, Edward L., E. Rene Rodriguez, Alexander Neumann, Carmela D. Tan, and Joseph E. Parrillo. "Cardiac Hemochromatosis." Circulation 94, no. 9 (November 1996): 2302–3. http://dx.doi.org/10.1161/01.cir.94.9.2302.

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37

Ajioka, Richard S., and James P. Kushner. "Hereditary hemochromatosis." Seminars in Hematology 39, no. 4 (October 2002): 235–41. http://dx.doi.org/10.1053/shem.2002.35634.

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38

Camaschella, Clara, Antonella Roetto, and Marco De Gobbi. "Juvenile hemochromatosis." Seminars in Hematology 39, no. 4 (October 2002): 242–48. http://dx.doi.org/10.1053/shem.2002.35635.

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39

Pearson, Lauree, Robin Bissinger, and Kelly R. Romero. "Neonatal Hemochromatosis." Advances in Neonatal Care 9, no. 2 (April 2009): 72–76. http://dx.doi.org/10.1097/anc.0b013e31819ac020.

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40

Franchini, Massimo, and Dino Veneri. "Hereditary hemochromatosis." Hematology 10, no. 2 (April 1, 2005): 145–49. http://dx.doi.org/10.1080/10245330500065771.

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41

Tennyson, Gary S., and Sami R. Achem. "Genetic Hemochromatosis." Digestive Diseases 14, no. 5 (1996): 330. http://dx.doi.org/10.1159/000171564.

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42

Quigley, Patricia. "Hereditary hemochromatosis." Nursing 46, no. 5 (May 2016): 36–43. http://dx.doi.org/10.1097/01.nurse.0000482258.55896.d8.

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43

Knisely, A. S., Giorgina Mieli-Vergani, and Peter F. Whitington. "Neonatal hemochromatosis." Gastroenterology Clinics of North America 32, no. 3 (September 2003): 877–89. http://dx.doi.org/10.1016/s0889-8553(03)00050-5.

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44

Blisard, Karen S., and Sue A. Bartow. "Neonatal hemochromatosis." Human Pathology 17, no. 4 (April 1986): 376–83. http://dx.doi.org/10.1016/s0046-8177(86)80461-0.

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45

Templin, Christian, Stefanie Pertschy, and Arnd Schaefer. "Cardiac hemochromatosis." International Journal of Cardiology 116, no. 3 (April 2007): e109-e110. http://dx.doi.org/10.1016/j.ijcard.2006.08.102.

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46

Witte, David L., William H. Crosby, Corwin Q. Edwards, Virgil F. Fairbanks, and Frank A. Mitros. "Hereditary hemochromatosis." Clinica Chimica Acta 245, no. 2 (February 1996): 139–200. http://dx.doi.org/10.1016/0009-8981(95)06212-2.

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47

Pietrangelo, Antonello. "Hereditary hemochromatosis." Biochimica et Biophysica Acta (BBA) - Molecular Cell Research 1763, no. 7 (July 2006): 700–710. http://dx.doi.org/10.1016/j.bbamcr.2006.05.013.

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48

Herbert, Victor. "Hereditary Hemochromatosis." Annals of Internal Medicine 135, no. 12 (December 18, 2001): 1091. http://dx.doi.org/10.7326/0003-4819-135-12-200112180-00019.

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49

Amidon, Phillip B., and Rhonda D. Jankovich. "Hereditary Hemochromatosis." Annals of Internal Medicine 135, no. 12 (December 18, 2001): 1091. http://dx.doi.org/10.7326/0003-4819-135-12-200112180-00020.

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50

Barbosa, K??tia Val??ria Bastos Dias, A??cio Fl??vio Meirelles de Souza, J??lio Maria Fonseca Chebli, Fernando Augusto Proietti, Ricardo Souza Portes Meirelles, and Juliano Leite de Souza. "Hereditary Hemochromatosis." Journal of Clinical Gastroenterology 39, no. 5 (May 2005): 430–34. http://dx.doi.org/10.1097/01.mcg.0000159218.85537.e5.

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