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Author: Grafiati

Published: 8 July 2023

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1

Ragin, Margaret M. Recovery of sulfur from phosphogypsum: Conversion of calcium sulfate to calcium sulfide. Washington, D.C. (2401 E St., N.W., MS #9800, Washington 20241): U.S. Dept. of the Interior, Bureau of Mines, 1990.

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2

Ragin, Margaret M. Recovery of sulfur from phosphogypsum: Conversion of calcium sulfate to calcium sulfide. Pgh. [i.e. Pittsburgh] PA: United States Dept. of the Interior, Bureau of Mines, 1990.

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3

Götz-Neunhoeffer, Friedlinde. Modelle zur Kinetik der Hydratation von Calciumaluminatzement mit Calciumsulfat aus kristallchemischer und mineralogischer sicht. Erlangen: Universitätsbund Erlangen-Nürnberg, 2006.

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4

Zhang, Zhongjie. Stability of calcium sulfate base course in a wet environment. Baton Rouge, LA: Louisiana Transportation Research Center, 2006.

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5

A, Rice David, ed. Recovery of sulfur from phosphogypsum: Conversion of calcium sulfide to sulfur. [Washington, D.C.]: Bureau of Mines, U.S. Dept. of the Interior, 1990.

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6

A, Tyburczy James, Ahrens T. J. 1936-, and United States. National Aeronautics and Space Administration., eds. Shock-induced devolatization of calcium sulfate and implications for K-T extinctions. [Washington, DC: National Aeronautics and Space Administration, 1993.

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7

A, Tyburczy James, Ahrens T. J. 1936-, and United States. National Aeronautics and Space Administration., eds. Shock-induced devolatization of calcium sulfate and implications for K-T extinctions. [Washington, DC: National Aeronautics and Space Administration, 1993.

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8

A, Tyburczy James, Ahrens T. J. 1936-, and United States. National Aeronautics and Space Administration., eds. Shock-induced devolatization of calcium sulfate and implications for K-T extinctions. [Washington, DC: National Aeronautics and Space Administration, 1993.

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9

Hao, Yue-Li. Inhibition of acid production in coal refuse amended with calcium sulfate and calcium sulfite-containing flue gas desulfurization by-products. Columbus, OH: Graduate School, Ohio State University, 1998.

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10

Newton, Peter J. Sodium sulfate corrosion of silicon carbide fiber-reinforced calcium aluminosilicate glass-ceramic matrix composites. Monterey, Calif: Naval Postgraduate School, 1994.

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11

Services, Dearborn Environmental Consulting. Treatability of wastewaters from high CaO and CaSO4 content FBC wastes. Ottawa, Ont., Canada: Environment Canada, 1986.

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12

Engineers, National Association of Corrosion. Laboratory screening tests to determine the ability of scale inhibitors to prevent the precipitation of calcium sulfate and calcium carbonate from solution (for oil and gas production systems). Houston: NACE, 1995.

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13

National Association of Corrosion Engineers., ed. Standard test method: Laboratory screening tests to determine the ability of scale inhibitors to prevent the precipitation of calcium sulfate and calcium carbonate from solution : (for oil and gas production systems). Houston: NACE, 2001.

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14

Wells, Keith Buckley. Optically stimulated fluorescence in samarium doped calcium sulfate. 1989.

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15

Ullman's Encyclopedia of Industrial Chemistry. Vol. A4 Benzyl alcohol to calcium sulfate. 5th ed. Deerfield Beach: VCH, 1985.

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16

National Aeronautics and Space Administration (NASA) Staff. Shock-Induced Devolatization of Calcium Sulfate and Implications for K-T Extinctions. Independently Published, 2018.

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17

Davis, H. T., and Edited by: H. J. Arpe. Benzyl Alcohol to Calcium Sulfate, Volume A4, Ullmann's Encyclopedia of Industrial Chemistry, 5th Edition. 5th ed. Wiley-VCH, 1985.

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18

Abhishek, Abhishek, and Michael Doherty. Pathophysiology of calcium pyrophosphate deposition. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0049.

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Calcium pyrophosphate (CPP) dihydrate crystals form extracellularly. Their formation requires sufficient extracellular inorganic pyrophosphate (ePPi), calcium, and pro-nucleating factors. As inorganic pyrophosphate (PPi) cannot cross cell membranes passively due to its large size, ePPi results either from hydrolysis of extracellular ATP by the enzyme ectonucleotide pyrophosphatase/phosphodiesterase 1 (also known as plasma cell membrane glycoprotein 1) or from the transcellular transport of PPi by ANKH. ePPi is hydrolyzed to phosphate (Pi) by tissue non-specific alkaline phosphatase. The level of extracellular PPi and Pi is tightly regulated by several interlinked feedback mechanisms and growth factors. The relative concentration of Pi and PPi determines whether CPP or hydroxyapatite crystal is formed, with low Pi/PPi ratio resulting in CPP crystal formation, while a high Pi/PPi ratio promotes basic calcium phosphate crystal formation. CPP crystals are deposited in the cartilage matrix (preferentially in the middle layer) or in areas of chondroid metaplasia. Hypertrophic chondrocytes and specific cartilage matrix changes (e.g. high levels of dermatan sulfate and S-100 protein) are related to CPP crystal deposition and growth. CPP crystals cause inflammation by engaging with the NALP3 inflammasome, and with other components of the innate immune system, and is marked with a prolonged neutrophilic inflitrate. The pathogenesis of resolution of CPP crystal-induced inflammation is not well understood.

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19

Ullmann's Encyclopedia of Industrial Chemistry: Benzyl Alcohol to Calcium Sulfate, Vol A4 (Ullmann's Encyclopedia of Industrial Chemistry 5th ed Vol a). 5th ed. Vch Pub, 1985.

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20

Parker, Philip M. The World Market for Modelling Pastes, Dental Wax or Dental Impression Compounds in Sets for Retail Sale, and Calcined Gypsum or Calcium Sulfate Based Dental Products: A 2007 Global Trade Perspective. ICON Group International, Inc., 2006.

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21

The World Market for Modelling Pastes, Dental Wax or Dental Impression Compounds in Sets for Retail Sale, and Calcined Gypsum or Calcium Sulfate Based Dental Products: A 2004 Global Trade Perspective. Icon Group International, Inc., 2005.

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22

Fisher, David. Self-Healing Concrete. Materials Research Forum LLC, 2021. http://dx.doi.org/10.21741/9781644901373.

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Self-healing techniques are most successful in preventing concrete from cracking or breaking. The book reviews the most promising methods, including the use of polymers, epoxy resins, fungi or cementitious composites; biomineralization, continuing hydration or carbonation or wet/dry cycling. Various micro-organisms are able to produce favorable effects, such as denitrification, calcium carbonate formation, sulfate reduction or the production of methane. The book references 289 original resources and includes their direct web link for in-depth reading.

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23

Nadkarni, Vinay, Robert M. Sutton, and Robert A. Berg. Resuscitation and Stabilization. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199918027.003.0001.

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The authors provide essential information on providing care during the four phases of cardiac arrest and cardiopulmonary resuscitation (CPR): (1) prearrest, (2) no flow (untreated cardiac arrest), (3) low flow (CPR), and (4) postresuscitation. They discuss the most common precipitating events for out-of-hospital and in-hospital cardiac arrests in children and the how to deliver high-quality CPR. Medications used to treat cardiac arrest are presented (specifically vasopressors, including epinephrine; antiarrhythmics, including lidocaine, amiodarone, and magnesium sulfate; calcium; and sodium bicarbonate), along with the clinical indications and rationale for their use. Appropriate goals for care after return of spontaneous circulation are delineated, with guidelines for targeted temperature management and blood pressure.

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24

Chau, Anthony T. Magnesium Toxicity. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0058.

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Magnesium sulfate is currently the drug of choice for prophylaxis against eclampsia in women with preeclampsia and first-line treatment for eclampsia. In this population, medication administration error is a frequent cause of magnesium toxicity. Symptoms of toxicity are linked to increasing serum concentrations, but routine monitoring is not recommended. Instead, deep tendon reflexes and respiratory rate are the most commonly monitored parameters. However, magnesium serum concentration should be monitored when magnesium toxicity is suspected or in patients at high risk of toxicity. The gastrointestinal and central nervous systems are usually affected first. As the serum concentration becomes extremely high, neuromuscular and cardiovascular effects may occur, leading to respiratory failure and cardiac arrest. In addition to standard advanced life support measures, calcium is the mainstay of treatment and in some cases forced diuresis and dialysis may also be considered.

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