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Dissertations / Theses on the topic 'Heart disease; Cardiac metabolism'

Author: Grafiati

Published: 4 June 2021

Last updated: 8 February 2022

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1

Hopkins, James Charles Alex. "Myocardial glycogen, glucose uptake and insulin sensitivity : interrelations and changes with disease." Thesis, University of Oxford, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.363766.

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2

Luongo, Timothy Scott. "The Role of Mitochondrial Calcium Exchange in Cardiac Physiology and Disease." Diss., Temple University Libraries, 2017. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/437718.

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Biomedical Sciences<br>Ph.D.<br>The high metabolic demand of the heart makes it essential that an efficient and tightly controlled system be in place to regulate energy production. Contractility is mediated by a variable flux in intracellular calcium (iCa2+), which is proposed to be integrated into mitochondria to regulate cardiac energetics. Moreover, mitochondrial Ca2+ (mCa2+)-overload is known to activate the mitochondrial permeability transition pore (MPTP) and induce cell death. However, the true function of cardiac mCa2+ in physiology remains unknown. Recent studies have reported that th

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3

Steeples, Violetta Rae. "Metabolic modulation through deletion of hypoxia-inducible factor-1α and fumarate hydratase in the heart". Thesis, University of Oxford, 2015. http://ora.ox.ac.uk/objects/uuid:f546ca24-6226-4846-b492-30de26836e94.

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Hypoxia inducible factor-1α (HIF-1α) plays a critical role in the oxygen homeostasis of all metazoans. HIF-1α is a master transcriptional regulator which coordinates the adaptive response to low oxygen tension. Through activation of a plethora of downstream target genes, HIF-1α facilitates oxygenation by promoting angiogenesis and blood vessel dilation, in addition to modulating metabolic pathways to inhibit oxidative phosphorylation and promote glycolytic energy production. Given the critical roles of hypoxia, insufficient blood supply and perturbed energetics in the p

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4

Mahmod, Masliza. "Multiparametric cardiovascular magnetic resonance for the assessment of cardiac function and metabolism in hypertrophy and heart failure." Thesis, University of Oxford, 2013. http://ora.ox.ac.uk/objects/uuid:ff24c167-e00d-4c6d-9809-82203979ba7a.

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Both hypertrophied and failing hearts are characterised by pathological left ventricular (LV) remodelling, impaired myocardial energy status and alteration in substrate metabolism. Cardiac magnetic resonance imaging (CMR) and magnetic resonance spectroscopy (MRS) are powerful tools in the characterisation of these disease conditions. More recent techniques have allowed assessment of myocardial steatosis using <sup>1</sup>H-MRS and tissue oxygenation using blood oxygen level dependent (BOLD) CMR. In hypertrophy and heart failure, studies on steatosis and the relationship with other parameters s

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5

Dewan, Aaraf. "A Unique Role for Sarcolemmal Membrane Associated Protein Isoform 1 (SLMAP1) as a Regulator of Cardiac Metabolism and Endosomal Recycling." Thesis, Université d'Ottawa / University of Ottawa, 2016. http://hdl.handle.net/10393/35088.

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Altered glucose metabolism is the underlying factor in many metabolic disorders, including diabetes. A novel protein recently linked to diabetes through animal and clinical studies is Sarcolemmal Membrane Associated Protein (SLMAP) but its role in metabolism remains undefined. The data here reveals a novel role for SLMAP isoform1 in glucose metabolism within the myocardium. Neonatal cardiomyocytes (NCMs) harvested from hearts of transgenic mice expressing SLMAP1, presented with increased glucose uptake, glycolytic rate, as well as glucose transporter 4 (GLUT4) expressions with minimal impact

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6

Wachowiak, Paul Stephen. "Relationships among Cynical Hostility, Metabolic Syndrome, and Cardiac Structure and Function in Multi-Ethnic Post-Myocardial Infarction Patients: A Structural Modeling Approach." Scholarly Repository, 2009. http://scholarlyrepository.miami.edu/oa_dissertations/291.

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BACKGROUND: Risk factors associated with Metabolic Syndrome (MetS) have been implicated in cardiovascular disease (CVD) development and outcomes. Few studies have investigated relationships between psychological variables, MetS factors, and indices of cardiac structure and function (CSF) among healthy individuals in a single conceptual model. No studies to date have analyzed such relationships in patients with CVD. METHODS: The present study examined associations between cynical hostility (CynHo), MetS factors, and CSF in 186 multi-ethnic post-myocardial infarction (MI) patients. Structura

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7

Cutter, Zachary S. "EFFECTS OF THE NA-CL CO-TRANSPORTER (NCC) IN WESTERN DIET INDUCED METABOLIC AND CARDIAC DYSFUNCTION." VCU Scholars Compass, 2018. https://scholarscompass.vcu.edu/etd/5431.

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Interleukin-18 (IL-18) is a pro-inflammatory cytokine known to be involved in maintaining metabolic homeostasis; however, also capable of inducing cardiac dysfunction. Additionally, IL-18, has been shown to bind to a novel receptor, the Na-Cl Co-transporter (NCC). We hypothesized that NCC mediates IL-18 metabolic and cardiac signaling in mice. Using male C57BL/6J mice, we compared the metabolic and cardiac function changes after at least 8 weeks of high-saturated fat high sugar diet (Western Diet) in NCC knockout (NCCKO), IL-18 knockout (IL-18KO), and wild-type mice. We show that NCCKO mice ha

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8

Dumaresq, Danielle Maia Holanda. "AvaliaÃÃo dos efeitos metabÃlico e oxidativo em cirurgia cardÃaca pediÃtrica: influÃncia da tÃcnica anestÃsica." Universidade Federal do CearÃ, 2006. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=409.

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Instituto Dr. JosÃ Frota<br>A cirurgia cardÃaca pediÃtrica freqÃentemente necessita de circulaÃÃo extracorpÃrea (CEC) durante a intervenÃÃo cirÃrgica. A combinaÃÃo de estresse cirÃrgico e CEC evoca uma resposta inflamatÃria sistÃmica multifatorial, com ativaÃÃo das cascatas humoral e celular. Somado Ã isto, a CEC proporciona perÃodos de isquemia-reperfusÃo, levando Ã condiÃÃes favorÃveis para formaÃÃo de radicais livres e criando uma situaÃÃo de desequilÃbrio que Ã denominada de estresse oxidativo. As espÃcies reativas do oxigÃnio (ERO) formadas durante o perÃodo de isquemia-reperfusÃo, estÃo

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9

Murray, Andrew James. "Control of cardiac metabolism and efficiency." Thesis, University of Oxford, 2003. http://ora.ox.ac.uk/objects/uuid:858cc1f9-7ba0-4999-a1c8-614a950888c2.

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10

Quigley, Gillian Margaret. "Inflammation of the heart in heart disease." Thesis, University of Manchester, 2013. https://www.research.manchester.ac.uk/portal/en/theses/inflammation-of-the-heart-in-heart-disease(eae19e58-aeb4-4673-924e-1dbd1c831fec).html.

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Heart failure patients have dysfunction of the cardiac conduction system that contributes to a high burden of arrhythmias including atrial fibrillation and sudden cardiac death. Heart failure has been associated with the inflammatory response, but it is unknown if inflammation is playing a role in the remodelling of the cardiac conduction system in heart failure. Inflammation has been shown to be present in the myocardium from failing hearts and it is known to have detrimental effects on cardiac function, inducing fibrosis, remodelling of ion channels and even arrhythmias. However, the effect

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11

Berryman, Barbara Michelle Swindell. "Design of a cardiac fitness and lifestyle management tool for phase III cardiac rehabilitation patients." Thesis, Georgia Institute of Technology, 2002. http://hdl.handle.net/1853/23732.

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12

Turner, J. E. "Collagen metabolism in normal heart and during cardiac hypertrophy." Thesis, Imperial College London, 1988. http://hdl.handle.net/10044/1/47290.

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13

Steele, Ian Conrad. "Pathophysiology of chronic cardiac failure." Thesis, Queen's University Belfast, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.337046.

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14

Kreshel, Leigh Anne. "Increasing energy expenditure of cardiac rehabilitation patients." Electronic thesis, 2002. http://dspace.zsr.wfu.edu/jspui/handle/10339/175.

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15

Hsiao, Lien-Cheng. "Cardiac stem cell therapy for heart failure." Thesis, University of Oxford, 2012. https://ora.ox.ac.uk/objects/uuid:c4fcb449-2d05-4dc6-9a8d-f7450c0b200c.

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Cardiovascular disease is a leading cause of death worldwide and becomes increasingly prevalent in the elderly population. Independent of etiopathogenesis, heart failure (HF) is the final common stage of numerous heart diseases. Cardiac stem cell (CSC) therapy has emerged as a promising cell-based strategy for treatment of HF. However, cell replacement is not able to fully restore a structurally damaged myocardium in advanced and end-stage HF. The objective of this project was to test the following hypotheses: that a bioengineered heart extracellular matrix (ECM) with preserved intact geometri

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16

Suh, Doug Young. "Knowledge-based boundary detection system : on MRI cardiac image sequences." Diss., Georgia Institute of Technology, 1990. http://hdl.handle.net/1853/13291.

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17

Kay, Edmund. "Cardiac acoustics : understanding and detecting heart murmurs." Thesis, University of Cambridge, 2018. https://www.repository.cam.ac.uk/handle/1810/275992.

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18

Chong, Yuen-yu. "Psychosocial smoking cessation interventions for hospitalized patients with cardiac disease." Click to view the E-thesis via HKUTO, 2009. http://sunzi.lib.hku.hk/hkuto/record/B43251146.

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19

Roberts, Timothy Lloyd. "Linoleic acid and sudden cardiac death." Thesis, University College London (University of London), 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.281772.

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20

Rigatto, Claudio. "Cardiac disease in renal transplant recipients /." St. John's, NF : [s.n.], 2001.

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21

莊婉瑜 and Yuen-yu Chong. "Psychosocial smoking cessation interventions for hospitalized patientswith cardiac disease." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hub.hku.hk/bib/B43251146.

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22

Morgan, Eric E. "The Cardiac Fatty Acid Metabolic Pathway in Heart Failure." Case Western Reserve University School of Graduate Studies / OhioLINK, 2006. http://rave.ohiolink.edu/etdc/view?acc_num=case1138394643.

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23

Mahy, Ian Richard John. "Observations on human peripheral microvascular function in cardiac disease." Thesis, Imperial College London, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.308969.

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24

McCance, Alastai J. "Systemic and cardiac noradrenaline kinetics in ischaemic heart disease in man." Thesis, University of Oxford, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.235891.

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25

Jamshidi, Yalda. "Role of PPAR#alpha# in coronary heart disease and cardiac hypertrophy." Thesis, University College London (University of London), 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.252393.

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26

Zhang, Huajun. "Functional characterisation of cardiac progenitors from patients with ischaemic heart disease." Thesis, University of Oxford, 2013. http://ora.ox.ac.uk/objects/uuid:3b8a7199-c077-436c-bb89-cd354efe4414.

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Ischaemic heart disease (IHD) is the leading cause of death worldwide. Currently, even optimal medical therapies do not attenuate deterioration of the left ventricular (LV) function completely. Stem cell therapies, and recently cardiac stem cell therapies, have emerged as potential novel treatments for IHD. However, clinical evidence from randomised controlled studies has shown mixed results. Thus understanding what patient-related factors may affect the therapeutic performance of the cells may help improving treatment outcomes. The studies described in this thesis aim to understand how cardia

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27

De’Ath, Henry D. I. "Trauma associated cardiac injury & dysfunction." Thesis, Queen Mary, University of London, 2013. http://qmro.qmul.ac.uk/xmlui/handle/123456789/8466.

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The existence of a trauma induced secondary cardiac injury (TISCI) remains in doubt. The risk factors and pathological processes that lead to its development are not known, whilst the effects of TISCI on injured patient outcome are uncertain. Concurrently, the incidence of coronary heart disease (CHD) in a trauma population and its influence on mortality are inconclusive. The aim of this research project was to address these specific areas of uncertainty. Critically injured patients (n=135) were retrospectively investigated for the incidence and nature of adverse cardiac events (ACEs), and lev

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28

Shaw, I., BS Shaw, and GA Brown. "Influence of strength training on cardiac risk prevention in individuals without cardiovascular disease." African Journal for Physical, Health Education, Recreation and Dance, 2009. http://encore.tut.ac.za/iii/cpro/DigitalItemViewPage.external?sp=1001650.

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Abstract It has widely been shown that exercise, particularly aerobic exercise, has extensive cardioprotective benefits and is an important tool in the prevention of coronary heart disease (CHD). The present investigation aimed to determine the multivariate impact of strength training, designed to prevent the development of CHD, on the Framingham Risk Assessment (FRA) score. Twenty-eight healthy untrained men with low CHD risk (mean age 28 years and 7 months) participated in an eight-week (3- d/wk) strength training programme. Self-administered smoking records, resting blood pressures, total c

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29

Taylor, Carolyn W. "Breast cancer radiotherapy and heart disease." Thesis, University of Oxford, 2008. http://ora.ox.ac.uk/objects/uuid:c9dda3ca-8cb3-4a38-938d-0b75b4f6471d.

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Introduction: Some past breast cancer radiotherapy regimens led to an increased risk of death from heart disease. Although heart dose from breast cancer radiotherapy has generally reduced over the past few decades, there may still be some cardiac risk. Estimation of future risk for women irradiated today requires both measurement of their cardiac dose and dose-response relationships, which depend on cardiac dosimetry of past regimens, in conjunction with long-term follow-up data. Methods: Virtual simulation and computed tomography 3-dimensional treatment planning on a representative patient we

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30

Zhang, Wen. "Cardiac function, energetics and substrate metabolism in mouse models of disease." Thesis, University of Oxford, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.444950.

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31

Stewart, Simon. "Optimising therapeutic efficacy in acute and chronic cardiac disease states /." Title page, contents and abstract only, 1999. http://web4.library.adelaide.edu.au/theses/09PH/09phs851.pdf.

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32

GREGORY, KIMBERLY NICOLE. "SARCOPLASMIC RETICULUM CALCIUM CYCLING AND CARDIAC DISEASE." University of Cincinnati / OhioLINK, 2005. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1116008332.

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33

d'Arcy, Joanna Louise. "Valvular heart disease : novel epidemiological and imaging studies." Thesis, University of Edinburgh, 2016. http://hdl.handle.net/1842/25476.

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Since living conditions have improved and antibiotics have entered routine use, valvular heart disease (VHD) in the developed world is mostly degenerative in origin, rather than rheumatic. Our population is increasing with age, and therefore the burden of VHD is likely to increase. Despite this, the epidemiology & prognostication in VHD remain poorly understood. A better understanding of the prevalence of VHD in our population, and improved methods of predicting outcomes, are essential if we are to be better equipped to meet the challenges of this new “epidemic”. This thesis aims to improve ou

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34

Soo, Lin Hoe. "Out-of-hospital cardiac arrest in Nottinghamshire." Thesis, University of Nottingham, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.341974.

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35

Varian, Kenneth Dean. "Cardiac Myofilament Calcium Sensitivity in Health and Disease." The Ohio State University, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=osu1211898886.

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36

Thow, Morag Kennedy. "A study of the recruitment patterns of women in cardiac rehabilitation and the evaluation of an exercise based cardiac rehabilitation programme in women post myocardial infarction." Thesis, Glasgow Caledonian University, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.369996.

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37

Luk, Ting-hin, and 陸庭軒. "Effect of cardiac rehabilitation on vascular function in patients withcoronary artery disease." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B45153000.

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38

di, Bernardo Diego. "Computer modelling of cardiac repolarisation for the analysis of the electrocardiogram." Thesis, University of Newcastle Upon Tyne, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.364809.

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39

Simnett, Sarah Jacqueline. "Relaxation processes in cardiac muscle." Thesis, University of Oxford, 1993. http://ora.ox.ac.uk/objects/uuid:9b670123-f816-42be-8d74-c37917af200b.

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40

Leong, Yuk-yan Pauline, and 梁玉恩. "The effectiveness of exercise-based cardiac rehabilitation program for secondary prevention of coronary heart disease : a systematic review." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hdl.handle.net/10722/193828.

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Objective: To examine the effect of exercise-based cardiac rehabilitation program for secondary prevention of coronary heart disease on cardiac-related mortality, recurrent cardiovascular event and quality of life. Methods: All studies published between 1990 and 2013 in PubMed, and from 1980 to 2013 in EMBASE, which evaluated the effectiveness of exercise-based cardiac rehabilitation program for coronary heart disease. Using the specific keywords “Cardiac rehabilitation”, “Coronary heart disease” OR “Ischemic heart disease” [MeSH], “Exercise” OR “Physical activities” AND “Quality of life”

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41

Sammut, Eva Clare. "Advanced cardiac magnetic resonance imaging in heart failure and coronary artery disease." Thesis, King's College London (University of London), 2018. https://kclpure.kcl.ac.uk/portal/en/theses/advanced-cardiac-magnetic-resonance-imaging-in-heart-failure-and-coronary-artery-disease(edf62114-1762-4830-a456-2aa8886ef130).html.

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Recent technical developments have increased the scope for cardiac MRI as a research tool. This work presents novel approaches to the assessment of patients with coronary disease and heart failure. The thesis explores coronary physiology and microarchitecture, ischaemia and perfusion CMR, and in particular the quantification of ischaemia. In addition, in-vivo cardiac diffusion tensor imaging is employed to characterize microarchitecture in heart failure. This work comes to a number of conclusions. We have been able to show, for the first time in patients with heart failure, that even in patien

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42

Clarkson, Peter Bruce Mark. "Studies of left ventricular diastolic function inhealth and disease." Thesis, University of Dundee, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.337397.

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43

Colegrave, Melanie. "Expression of #beta#-cardiac myosin in a myogenic cell line." Thesis, King's College London (University of London), 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.342254.

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44

Klipp, Robert Carl. "Catecholamine Interactions with the Cardiac Ryanodine Receptor." PDXScholar, 2013. https://pdxscholar.library.pdx.edu/open_access_etds/1439.

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The cardiac ryanodine receptor (RyR2) is a Ca2+ ion channel found in the sarcoplasmic reticulum (SR), an intracellular membranous Ca2+ storage system. It is well known that a destabilization of RyR2 can lead to a Ca2+ flux out of the SR, which results in an overload of intracellular Ca2+; this can also lead to arrhythmias and heart failure. The catecholamines play a large role in the regulation of RyR2; stimulation of the Beta-adrenergic receptor on the cell membrane can lead to a hyperphosphorylation of RyR2, making it more leaky to Ca2+. We have previously shown that strong electron donors w

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45

Tien, Pamela. "Reductive metabolism of aliphatic tertiary amine n-oxides." Thesis, De Montfort University, 1999. http://hdl.handle.net/2086/10714.

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This study is based on a proposal concerning the feasibility of using aliphatic tertiary amine N-oxides as antiarrhythmic agent prodrugs. Lignocaine was selected as a candidate for prodrug development, because the N-oxide is a non-active, polar derivative of lignocaine and the drug of choice for ventricular arrhythmia, a symptom associated with ischaemic episodes leading to regions of transiently hypoxic heart tissue. An HPLC analytical method was developed to study the metabolism of lignocaine N-oxide. The rapid and sensitive analysis of lignocaine and its metabolites was demonstrated with go

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46

Rahimi-Larijani, B. "Dynamic Fourier phase and amplitude analysis and computational techniques in gated cardiac scintigraphy." Thesis, University of Brighton, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.382224.

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47

Mansor, Latt Shahril. "Effect of hypoxia on cardiac metabolism and function in the type 2 diabetic heart." Thesis, University of Oxford, 2015. https://ora.ox.ac.uk/objects/uuid:e84a3068-0c7d-46d7-a37f-3433cc06b3d4.

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Type 2 diabetic patients have impaired cardiac ischaemia-reperfusion recovery and higher rates of mortality following a myocardial infarction. Hypoxia is a key component of ischaemia and therefore, this thesis is aimed to investigate the effect of hypoxia on metabolism and contractile function of the type 2 diabetic heart. In combination with high fat feeding, different doses of streptozotocin (STZ) (15, 20, 25 and 30 mg/kg) were used to determine the optimal dose needed for induction of diabetes in male Wistar rats. A novel type 2 diabetic model was developed and characterised by hyperinsulin

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48

Watkins, Hugh Christian. "Demonstrations that cardiac troponin T mutations cause hypertrophic cardiomyopathy." Thesis, St George's, University of London, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.269224.

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49

de, Gannes Matthew K. "Dioxin Impact on Cardiac Development, Structure, Function, and Health, and Implications for Disease." University of Cincinnati / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1613748020897261.

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50

Chung, Jae-Hoon. "Regulation of Human Cardiac Muscle Contraction and Relaxation in Health and Disease." The Ohio State University, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=osu1522851185767187.

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