Relevant bibliographies by topics / Haemostasis / Books
To see the other types of publications on this topic, follow the link: Haemostasis.

Books on the topic 'Haemostasis'

Author: Grafiati
Published: 4 June 2021
Last updated: 20 February 2023

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 50 books for your research on the topic 'Haemostasis.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse books on a wide variety of disciplines and organise your bibliography correctly.

1

Monagle, Paul, ed. Haemostasis. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-339-8.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Faughnan, Marian. Copper and Haemostasis. [S.l: The Author], 1998.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
3

Boldt, David H. Update on haemostasis. Edinburgh: Churchill Livinstone, 1990.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
4

Geddis, Alison Elaine. Haemostasis in hypercholesterolemia. [S.l: The Author], 1992.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
5

Roberts, Harold Ross, ed. Haemophilia and Haemostasis. Oxford, UK: Blackwell Publishing Ltd, 2007. http://dx.doi.org/10.1002/9780470692554.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Monagle, Paul T. Haemostasis: Methods and protocols. New York: Humana Press, 2013.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
7

Zirlik, Andreas, Christoph Bode, and Meinrad Gawaz, eds. Platelets, Haemostasis and Inflammation. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-66224-4.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Harenberg, Job, Dieter L. Heene, Gerd Stehle, and Gotthard Schettler, eds. New Trends in Haemostasis. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-84318-1.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

Szpalski, Marek, Richard B. Weiskopf, Robert Gunzburg, and Max Aebi, eds. Haemostasis in Spine Surgery. Berlin/Heidelberg: Springer-Verlag, 2005. http://dx.doi.org/10.1007/b139006.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Giuseppe, Remuzzi, and Rossi Ennio Claudio 1931-, eds. Haemostasis and the kidney. London: Butterworths, 1989.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
11

Maki, Masahiro. Haemostasis and thrombosis in obstetrics and gynecology. Marburg: Medizinische Verlagsgesellschaft, 1991.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
12

Scott, Melanie. The effect of phospholipid composition on haemostasis. Leicester: De Montfort University, 2004.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
13

International Congress on Thrombosis and Haemostasis. (11th 1987 Brussels, Belgium). Thrombosis and haemostasis: XI congress, Brussels, 1987. Leuven (Belgium): International Society on Thrombosis and Haemostasis, Leuven University Press, 1987.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
14

Heslin, Jacinta. A study of paediatric haematology and haemostasis. [S.l: The Author], 1992.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
15

Holmes, Valerie Anne. Early markers of haemostasis in normal pregnancy. [s.l: The Author], 2002.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
16

1958-, Greer I. A., Turpie, A. G. G. 1939-, and Forbes C. D. 1938-, eds. Haemostasis and thrombosis in obstetrics and gynaecology. London: Chapman & Hall Medical, 1992.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
17

1949-, Williams C. E., ed. Critical factors in haemostasis: Evaluation and development. Chichester, England: Ellis Horwood, 1988.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
18

Paul A. Owren & the golden era of haemostasis. Norway?]: Gazettebok, 2005.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
19

Ma, Alice, H. R. Roberts, and Daniel Carrizosa. Haemophilia and haemostasis: A case-based approach to management. Edited by Haemostasis Forum and Wiley online library. Malden, MA: Blackwell Pub., 2007.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
20

Dr, Cooper David N., ed. The molecular genetics of haemostasis and its inherited disorders. Oxford: Oxford University Press, 1994.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
21

Job, Harenberg, and Heidelberger Akademie der Wissenschaften, eds. New trends in haemostasis: Coagulation proteins, endothelium, and tissue factors. Berlin: Springer-Verlag, 1990.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
22

(Editor), E. W. Salzman, and S. Sherry (Editor), eds. Fragmin (Haemostasis,). S. Karger AG (Switzerland), 1986.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
23

Anticoagulanti, Federazione Centri Sorveglianza. Haemostasis: A Guide to Oral Anticoagulant Therapy (Haemostasis). S Karger Pub, 1998.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
24

Haemostasis and Cancer. Kluwer Academic Publishers Group, 1998.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
25

L, Bloom Arthur, and Thomas Duncan P, eds. Haemostasis and thrombosis. 2nd ed. Edinburgh: Churchill Livingstone, 1987.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
26

L, Bloom Arthur, and Thomas Duncan P. 1929-, eds. Haemostasis and thrombosis. 2nd ed. Edinburgh: Churchill Livingstone, 1987.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
27

L, Bloom Arthur, ed. Haemostasis and thrombosis. 3rd ed. Edinburgh: Churchill Livingstone, 1994.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
28

(Editor), Nadey S. Hakim, and Ruben Canelo (Editor), eds. Haemostasis in Surgery. Imperial College Press, 2007.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
29

Loo, Jurgen Van De. Diabetes and Haemostasis. S Karger AG, 1987.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
30

Provan, Drew, Trevor Baglin, Inderjeet Dokal, and Johannes de Vos. Haemostasis and thrombosis. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199683307.003.0010.

Full text
Abstract:
Assessing haemostasis - The coagulation system - Laboratory tests - Platelets - Bleeding - Bleeding: laboratory investigations - Bleeding: therapeutic products - von Willebrand disease - Haemophilia A and B - Rare congenital coagulation disorders - Congenital thrombocytopenias - Congenital platelet function defects - Congenital vascular disorders - Haemorrhagic disease of the newborn - Thrombocytopenia (acquired) - Specific thrombocytopenic syndromes - Disseminated intravascular coagulation - Liver disease - Renal disease - Acquired anticoagulants and inhibitors - Treatment of spontaneous FVIII inhibitor - Acquired disorders of platelet function - Henoch–Schönlein purpura - Perioperative bleeding and massive blood loss - Massive blood loss - Heparin - Heparin induced thrombocytopenia/with thrombosis (HIT/T) - Oral anticoagulant therapy (warfarin, VKAs) - New oral anticoagulant drugs - Thrombosis - Risk assessment and thromboprophylaxis - Example of VTE risk assessment - Heritable thrombophilia - Acquired thrombophilia - Thrombotic thrombocytopenic purpura (TTP) - Haemolytic uraemic syndrome (HUS) - Heparin-induced thrombocytopenia (HIT)
APA, Harvard, Vancouver, ISO, and other styles
31

Provan, Drew, Trevor Baglin, Inderjeet Dokal, Johannes de Vos, and Angela Theodoulou. Haemostasis and thrombosis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199683307.003.0010_update_001.

Full text
Abstract:
Assessing haemostasis - The coagulation system - Laboratory tests - Platelets - Bleeding - Bleeding: laboratory investigations - Bleeding: therapeutic products - von Willebrand disease - Haemophilia A and B - Rare congenital coagulation disorders - Congenital thrombocytopenias - Congenital platelet function defects - Congenital vascular disorders - Haemorrhagic disease of the newborn - Thrombocytopenia (acquired) - Specific thrombocytopenic syndromes - Disseminated intravascular coagulation - Liver disease - Renal disease - Acquired anticoagulants and inhibitors - Treatment of spontaneous FVIII inhibitor - Acquired disorders of platelet function - Henoch–Schönlein purpura - Perioperative bleeding and massive blood loss - Massive blood loss - Heparin - Heparin induced thrombocytopenia/with thrombosis (HIT/T) - Oral anticoagulant therapy (warfarin, VKAs) - New oral anticoagulant drugs - Thrombosis - Risk assessment and thromboprophylaxis - Example of VTE risk assessment - Heritable thrombophilia - Acquired thrombophilia - Thrombotic thrombocytopenic purpura (TTP) - Haemolytic uraemic syndrome (HUS) - Heparin-induced thrombocytopenia (HIT)
APA, Harvard, Vancouver, ISO, and other styles
32

Hakim, Nadey S., and Ruben Canelo. Haemostasis in Surgery. PUBLISHED BY IMPERIAL COLLEGE PRESS AND DISTRIBUTED BY WORLD SCIENTIFIC PUBLISHING CO., 2007. http://dx.doi.org/10.1142/p466.

Full text
APA, Harvard, Vancouver, ISO, and other styles
33

Monasterio, J., J. A. Iriarte, and Y. Saez. Antiphospholipid Antibodies: Journal : Haemostasis, Vol 24, No 3, 1994 (Haemostasis). S Karger Pub, 1994.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
34

Critical Factors in Haemostasis. Wiley-VCH, 1989.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
35

Monagle, Paul. Haemostasis: Methods and Protocols. Humana Press, 2016.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
36

Gawaz, Meinrad, Andreas Zirlik, and Christoph Bode. Platelets, Haemostasis and Inflammation. Springer, 2019.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
37

Haemostasis in Spine Surgery. Springer, 2005.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
38

Gawaz, Meinrad, Andreas Zirlik, and Christoph Bode. Platelets, Haemostasis and Inflammation. Springer, 2018.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
39

(Editor), Marek Szpalski, Robert Gunzburg (Editor), Richard B. Weiskopf (Editor), and Max Aebi (Editor), eds. Haemostasis in Spine Surgery. Springer, 2005.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
40

Trends in haemostasis 1995. Budapest: Akadémiai Kiadó, 1995.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
41

Mannucci, Pier Mannuccio. Bleeding and haemostasis disorders. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0070.

Full text
Abstract:
The main cause of haemostasis defects and related bleeding complications in patients with acute coronary syndromes admitted to the intensive cardiac care unit is the use of multiple antithrombotic drugs, alone or concomitantly with invasive procedures such as percutaneous coronary intervention with stent deployment and coronary artery bypass surgery. These drugs, that act upon several components of haemostasis (platelet function, coagulation, fibrinolysis), are associated with bleeding complications, particularly in elderly patients (more so in women than in men), those who are underweight, and those with comorbid conditions such as renal and liver insufficiency and diabetes. The identification of patients at higher risk of bleeding is the most important preventive strategy. Red cell and platelet transfusions, which may become necessary in patients with severe bleeding, should be used with caution, because transfused patients with acute coronary syndrome have a high rate of adverse outcomes (death, myocardial infarction, and stroke). To reduce the need of transfusion, haemostatic agents that decrease blood loss and transfusion requirements (antifibrinolytic amino acids, plasmatic prothrombin complex concentrates, recombinant factor VIIa) may be considered. However, the efficacy of these agents in the control of bleeding complications in acute coronary syndrome is not unequivocally established, and there is concern for an increased risk of re-thrombosis. A low platelet count is another cause of bleeding in the intensive cardiac care unit. The main aetiologies are drugs (unfractionated heparin and glycoprotein IIb/IIIa inhibitors), thrombotic microangiopathies, such as thrombotic thrombocytopenic purpura, and disseminated intravascular coagulation, that are often paradoxically associated with thrombotic manifestations. In conclusion, evidence-based recommendations for the management of bleeding in patients admitted to the intensive cardiac care unit are lacking. Accurate assessments of the risk of bleeding in the individual and prevention measures are the most valid strategies.
APA, Harvard, Vancouver, ISO, and other styles
42

Mannucci, Pier Mannuccio. Bleeding and haemostasis disorders. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199687039.003.0070_update_001.

Full text
Abstract:
The main cause of haemostasis defects and related bleeding complications in patients with acute coronary syndromes admitted to the intensive cardiac care unit is the use of multiple antithrombotic drugs, alone or concomitantly with invasive procedures such as percutaneous coronary intervention with stent deployment and coronary artery bypass surgery. These drugs, that act upon several components of haemostasis (platelet function, coagulation, fibrinolysis), are associated with bleeding complications, particularly in elderly patients (more so in women than in men), those who are underweight, and those with comorbid conditions such as renal and liver insufficiency and diabetes. The identification of patients at higher risk of bleeding is the most important preventive strategy. Red cell and platelet transfusions, which may become necessary in patients with severe bleeding, should be used with caution, because transfused patients with acute coronary syndrome have a high rate of adverse outcomes (death, myocardial infarction, and stroke). To reduce the need of transfusion, haemostatic agents that decrease blood loss and transfusion requirements (antifibrinolytic amino acids, plasmatic prothrombin complex concentrates, recombinant factor VIIa) may be considered. However, the efficacy of these agents in the control of bleeding complications in acute coronary syndrome is not unequivocally established, and there is concern for an increased risk of re-thrombosis. A low platelet count is another cause of bleeding in the intensive cardiac care unit. The main aetiologies are drugs (unfractionated heparin and glycoprotein IIb/IIIa inhibitors), thrombotic microangiopathies, such as thrombotic thrombocytopenic purpura, and disseminated intravascular coagulation, that are often paradoxically associated with thrombotic manifestations. In conclusion, evidence-based recommendations for the management of bleeding in patients admitted to the intensive cardiac care unit are lacking. Accurate assessments of the risk of bleeding in the individual and prevention measures are the most valid strategies.
APA, Harvard, Vancouver, ISO, and other styles
43

Mannucci, Pier Mannuccio. Bleeding and haemostasis disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199687039.003.0070_update_002.

Full text
Abstract:
The main cause of haemostasis defects and related bleeding complications in patients with acute coronary syndromes admitted to the intensive cardiac care unit is the use of multiple antithrombotic drugs, alone or concomitantly with invasive procedures such as percutaneous coronary intervention with stent deployment and coronary artery bypass surgery. These drugs, that act upon several components of haemostasis (platelet function, coagulation, fibrinolysis), are associated with bleeding complications, particularly in elderly patients (more so in women than in men), those who are underweight, and those with comorbid conditions such as renal and liver insufficiency and diabetes. The identification of patients at higher risk of bleeding is the most important preventive strategy. Red cell and platelet transfusions, which may become necessary in patients with severe bleeding, should be used with caution, because transfused patients with acute coronary syndrome have a high rate of adverse outcomes (death, myocardial infarction, and stroke). To reduce the need of transfusion, haemostatic agents that decrease blood loss and transfusion requirements (antifibrinolytic amino acids, plasmatic prothrombin complex concentrates, recombinant factor VIIa) may be considered. However, the efficacy of these agents in the control of bleeding complications in acute coronary syndrome is not unequivocally established, and there is concern for an increased risk of re-thrombosis. A low platelet count is another cause of bleeding in the intensive cardiac care unit. The main aetiologies are drug usage (unfractionated heparin and glycoprotein IIb/IIIa inhibitors), such thrombotic microangiopathies as thrombotic thrombocytopenic purpura and disseminated intravascular coagulation, that are often paradoxically associated with thrombotic manifestations. In conclusion, evidence-based recommendations for the management of bleeding in patients admitted to the intensive cardiac care unit are lacking. Accurate assessments of the risk of bleeding in the individual and prevention measures are the most valid strategies.
APA, Harvard, Vancouver, ISO, and other styles
44

Mannucci, Pier Mannuccio, and Maddalena Lettino. Bleeding and haemostasis disorders. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0070_update_003.

Full text
Abstract:
The main cause of haemostasis defects and related bleeding complications in patients with acute coronary syndromes admitted to the intensive cardiac care unit is the use of multiple antithrombotic drugs, alone or concomitantly with invasive procedures such as percutaneous coronary intervention with stent deployment and coronary artery bypass surgery. These drugs, that act upon several components of haemostasis (platelet function, coagulation, fibrinolysis), are associated with bleeding complications, particularly in elderly patients (more so in women than in men), those who are underweight, and those with comorbid conditions such as renal and liver insufficiency and diabetes. The identification of patients at higher risk of bleeding is the most important preventive strategy. Red cell and platelet transfusions, which may become necessary in patients with severe bleeding, should be used with caution, because transfused patients with acute coronary syndrome have a high rate of adverse outcomes (death, myocardial infarction, and stroke).
APA, Harvard, Vancouver, ISO, and other styles
45

Kay, L. A. Essentials of Haemostasis and Thrombosis. Churchill Livingstone, 1988.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
46

(Editor), Haverkate F., and Frits Haverkate (Editor), eds. Vascular Factors in Dementia (Haemostasis). Not Avail, 1999.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
47

Hedner, Ulla. New Aspects on Thromboembolism (Haemostasis). S Karger Ag, 1993.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
48

International Congress on Thrombosis (Haemostasis). S Karger AG, 1998.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
49

Pasechnik, I. N., and S. A. Burns, eds. Haemostasis Disorders In Surgical Patients. OOO «GEOTAR-Media» Publishing Group, 2021. http://dx.doi.org/10.33029/9704-6395-6-hae-2021-1-296.

Full text
APA, Harvard, Vancouver, ISO, and other styles
50

Alexander J. C. 1837-1900 Skene. Electro-Haemostasis in Operative Surgery. Creative Media Partners, LLC, 2018.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
You might also be interested in the bibliographies on the topic 'Haemostasis' for other source types:
Journal articles Dissertations / Theses
We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!

To the bibliography