Relevant bibliographies by topics / Haemodynamics

Academic literature on the topic 'Haemodynamics'

Author: Grafiati
Published: 4 June 2021
Last updated: 20 February 2023

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Journal articles on the topic "Haemodynamics"

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Kuboki, Takuo, Kenji Maekawa, and Glenn Clark. "Intramuscular haemodynamics using near infra-red spectroscopy as a research strategy to understand chronic muscle pain pathophysiology." Spectroscopy 19, no. 1 (2005): 27–36. http://dx.doi.org/10.1155/2005/148586.

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Recently, localized intramuscular haemodynamic disturbance has been recognized as one of the cardinal features of a chronically painful muscle. To test this hypothesis, we have carried out several studies that have assessed intramuscular haemodynamics to understand more about haemodynamic alteration under conditions of an experimentally induced vasoreactive challenges in healthy controls and in patients with chronic muscle pain using near infra?red (NIR) spectroscopy. In addition, we have validated the NIR based haemodynamic signals by comparing against a water signal intensity change taken from a transverse T2?weighted magnetic resonance image (MRI) of the muscles. As a result, we discovered that muscle pain patients have a diminished ability to vasodilate in painful muscle. We also provided powerful evidences that NIR data (total haemoglobin level), transcutaneously taken from the muscles in humans could be an analogue of intramuscular haemodynamics.
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Hartkamp, Nolan S., Esben T. Petersen, Michael A. Chappell, Thomas W. Okell, Maarten Uyttenboogaart, Clark J. Zeebregts, and Reinoud PH Bokkers. "Relationship between haemodynamic impairment and collateral blood flow in carotid artery disease." Journal of Cerebral Blood Flow & Metabolism 38, no. 11 (August 4, 2017): 2021–32. http://dx.doi.org/10.1177/0271678x17724027.

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Collateral blood flow plays a pivotal role in steno-occlusive internal carotid artery (ICA) disease to prevent irreversible ischaemic damage. Our aim was to investigate the effect of carotid artery disease upon cerebral perfusion and cerebrovascular reactivity and whether haemodynamic impairment is influenced at brain tissue level by the existence of primary and/or secondary collateral. Eighty-eight patients with steno-occlusive ICA disease and 29 healthy controls underwent MR examination. The presence of collaterals was determined with time-of-flight, two-dimensional phase contrast MRA and territorial arterial spin labeling (ASL) imaging. Cerebral blood flow and cerebrovascular reactivity were assessed with ASL before and after acetazolamide. Cerebral haemodynamics were normal in asymptomatic ICA stenosis patients, as opposed to patients with ICA occlusion, in whom the haemodynamics in both hemispheres were compromised. Haemodynamic impairment in the affected brain region was always present in symptomatic patients. The degree of collateral blood flow was inversely correlated with haemodynamic impairment. Recruitment of secondary collaterals only occurred in symptomatic ICA occlusion patients. In conclusion, both CBF and cerebrovascular reactivity were found to be reduced in symptomatic patients with steno-occlusive ICA disease. The presence of collateral flow is associated with further haemodynamic impairment. Recruitment of secondary collaterals is associated with severe haemodynamic impairment.
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Raj, Leah M., and Leslie A. Saxon. "Haemodynamic Monitoring Devices in Heart Failure: Maximising Benefit with Digitally Enabled Patient Centric Care." Arrhythmia & Electrophysiology Review 7, no. 4 (2018): 1. http://dx.doi.org/10.15420/aer.2018.32.3.

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ICDs and resynchronisation devices are routinely implanted in patients with heart failure for primary prevention of sudden cardiac death or to treat the condition. The addition of device features and algorithms that directly or indirectly monitor cardiac haemodynamics to assess heart failure status can provide additional benefit by treating heart failure more continuously. Established and emerging devices and sensors aimed at treating or measuring cardiac haemodynamics represent the next era of heart failure disease management. Digitally enabled models of heart failure care, based on frequent haemodynamic measurements, will increasingly involve patients in their own disease management. Software tools and services tailored to provide patients with personalised information to guide diet, activity, medications and haemodynamic management offer an unprecedented opportunity to improve patient outcomes. This will enable physicians to care for larger populations because management will be exception based, automated and no longer depend on one-to-one patient and physician interactions.
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Bull, C. "Haemodynamics." Current Opinion in Cardiology 1, no. 1 (January 1986): 88–92. http://dx.doi.org/10.1097/00001573-198601000-00016.

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Oslizlok, P. "Haemodynamics." Current Opinion in Cardiology 2, no. 1 (January 1987): 118–22. http://dx.doi.org/10.1097/00001573-198701010-00023.

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Bull, C. "Haemodynamics." Current Opinion in Cardiology 3, no. 1 (January 1988): 71–77. http://dx.doi.org/10.1097/00001573-198801000-00012.

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Obokata, Masaru, Thomas P. Olson, Yogesh N. V. Reddy, Vojtech Melenovsky, Garvan C. Kane, and Barry A. Borlaug. "Haemodynamics, dyspnoea, and pulmonary reserve in heart failure with preserved ejection fraction." European Heart Journal 39, no. 30 (May 19, 2018): 2810–21. http://dx.doi.org/10.1093/eurheartj/ehy268.

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Abstract Aims Increases in left ventricular filling pressure are a fundamental haemodynamic abnormality in heart failure with preserved ejection fraction (HFpEF). However, very little is known regarding how elevated filling pressures cause pulmonary abnormalities or symptoms of dyspnoea. We sought to determine the relationships between simultaneously measured central haemodynamics, symptoms, and lung ventilatory and gas exchange abnormalities during exercise in HFpEF. Methods and results Subjects with invasively-proven HFpEF (n = 50) and non-cardiac causes of dyspnoea (controls, n = 24) underwent cardiac catheterization at rest and during exercise with simultaneous expired gas analysis. During submaximal (20 W) exercise, subjects with HFpEF displayed higher pulmonary capillary wedge pressures (PCWP) and pulmonary artery pressures, higher Borg perceived dyspnoea scores, and increased ventilatory drive and respiratory rate. At peak exercise, ventilation reserve was reduced in HFpEF compared with controls, with greater dead space ventilation (higher VD/VT). Increasing exercise PCWP was directly correlated with higher perceived dyspnoea scores, lower peak exercise capacity, greater ventilatory drive, worse New York Heart Association (NYHA) functional class, and impaired pulmonary ventilation reserve. Conclusion This study provides the first evidence linking altered exercise haemodynamics to pulmonary abnormalities and symptoms of dyspnoea in patients with HFpEF. Further study is required to identify the mechanisms by which haemodynamic derangements affect lung function and symptoms and to test novel therapies targeting exercise haemodynamics in HFpEF.
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Warmerdam, Evangeline, Gregor J. Krings, Tim Leiner, and Heynric B. Grotenhuis. "Three-dimensional and four-dimensional flow assessment in congenital heart disease." Heart 106, no. 6 (December 19, 2019): 421–26. http://dx.doi.org/10.1136/heartjnl-2019-315797.

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Congenital heart disease (CHD) is the most common form of congenital defects, with an incidence of 8 per 1000 births. Due to major advances in diagnostics, perioperative care and surgical techniques, the survival rate of patients with CHD has improved dramatically. Conversely, although 70%–95% of infants with CHD survive into adulthood, the rate of long-term morbidity, which often requires (repeat) intervention, has increased. Recently, the role of altered haemodynamics in cardiac development and CHD has become a subject of interest. Patients with CHD often have abnormal blood flow patterns, either due to the primary cardiac defect or as a consequence of the surgical intervention(s). Research suggests that these abnormal blood flow patterns may contribute to diminished cardiac and vascular function. Serial assessment of haemodynamic parameters in patients with CHD may allow for improved understanding of the often complex haemodynamics in these patients and thereby potentially guide the timing and nature of interventions with the aim of preventing progression of cardiovascular deterioration. In this article we will discuss two novel non-invasive four-dimensional (4D) techniques to evaluate cardiovascular haemodynamics: 4D-flow cardiac magnetic resonance and computational fluid dynamics. This review focuses on the additional value of these two modalities in the evaluation of patients with CHD with abnormal flow patterns, who could benefit from advanced haemodynamic evaluation: patients with coarctation of the aorta, bicuspid aortic valve, tetralogy of Fallot and patients after Fontan palliation.
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Kabir, MS, AAS Majumder, AFKU Ahmed, KA Rubaiyat, A. Jhan, ASMG Rabbani, and SMS Haque. "Echocardiographic Evaluation of Pulmonary Haemodynamics - A Review." Cardiovascular Journal 13, no. 2 (April 15, 2021): 200–216. http://dx.doi.org/10.3329/cardio.v13i2.52975.

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Pulmonary haemodynamic status has got significant clinical and prognostic significance both in structural and nonstructural heart diseases. But diagnostic accuracy depends on much care in evaluation and logical correlation of different echocardiographic parameters. Though the Doppler studies play crucial role in estimating Pulmonary haemodynamics, careful observation of 2-D measurements of different structures as well as RV systolic and diastolic function are vital issues to be addressed and correlated for diagnostic accuracy. Tricuspid Valve, RVOT and Pulmonary valve pathologies also demand vivid correlation while estimating PA pressures and pulmonary vascular resistance. Mere presence of elevated pulmonary artery systolic pressure is not enough to define pulmonary hypertension (PH). Before declaring a patient having PH a comprehensive evaluation of right heart haemodynamics is to be performed carefully to avoid unnecessary cardiac catheterization or to avoid inadvertent initiation of PH therapy. Cardiovasc. j. 2021; 13(2): 200-216
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Miranda, William R., Barry A. Borlaug, Donald J. Hagler, Heidi M. Connolly, and Alexander C. Egbe. "Haemodynamic profiles in adult Fontan patients: associated haemodynamics and prognosis." European Journal of Heart Failure 21, no. 6 (January 23, 2019): 803–9. http://dx.doi.org/10.1002/ejhf.1365.

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Dissertations / Theses on the topic "Haemodynamics"

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O'Brien, Aoife Bernadette. "Pulmonary Haemodynamics." Thesis, Imperial College London, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.398251.

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Leunissen, Karel Maria Lucas. "Haemodynamics during haemodialysis." Maastricht : Maastricht : Rijksuniversiteit Limburg ; University Library, Maastricht University [Host], 1988. http://arno.unimaas.nl/show.cgi?fid=5410.

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Peacock, J. A. "Heart valve haemodynamics." Thesis, University of Oxford, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.371560.

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Begbuna, Veronica. "Haemodynamics in chronic venous disease." Thesis, Imperial College London, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.401816.

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Zabielski, Leszek. "Helical pipe flow in haemodynamics." Thesis, Imperial College London, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.338931.

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Moorhead, Katherine Tracey. "Autoregulation modelling of cerebral haemodynamics." Thesis, University of Canterbury. Mechanical Engineering, 2005. http://hdl.handle.net/10092/6844.

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The Circle of Willis (CoW) is a ring-like structure of blood vessels found at the base of the brain. Its main function is to distribute a constant flow oxygen-rich arterial blood to the cerebral mass, despite changes in afferent pressures or flows. This objective is achieved by a local mechanism known as autoregulation, whereby the resistance in small vessels branching from the CoW changes by vasodilation or vasoconstriction of the smooth muscle cells surrounding the vessel. A one-dimensional (1D) model of the CoW is developed to simulate a series of possible clinical scenarios such as occlusions in afferent arteries, absent or string-like circulus vessels, or arterial infarctions. A series of studies investigates various features of autoregulatory behaviour. Firstly, a simple model is created to verify solution methods; secondly, the model is validated against a three-dimensional (3D) Computational Fluid Dynamics (CFD) model; and lastly, the decentralised nature of cerebral autoregulation is investigated. Finally, an advanced, metabolic model of autoregulation is created, incorporating the successful aspects of the early model, as well as more physiologically accurate dynamics. The advanced model captures cerebral haemodynamic autoregulation by using a Proportional-Integral-Derivative (PID) controller to modify efferent artery resistances and partial pressures of oxygen to maintain optimal efferent flow rates and oxygen supply to the cerebral mass for a given circle geometry and afferent blood pressure. This advanced model is physiologically relevant, matching the accepted physiological responses of blood flow as a function of arterial pressure, tissue oxygen partial pressure as a function of blood flow, as well as limited transient clinical data. Results match accepted physiological response and exhibit excellent correlation with the limited clinical data available. In addition, a set of boundary conditions and geometry is presented for which the autoregulated system cannot provide the necessary efferent flow rates and perfusion, representing a condition with increased risk of stroke and highlighting the importance of modelling the haemodynamics of the Circle of Willis. The system model created is computationally simple so it can be used to identify at-risk cerebral arterial geometries and conditions prior to surgery or other clinical procedures. In addition, the solution for the CoW arterial system is obtained in a far shorter time period using this time-varying resistance model than with higher dimensional CFD methods, and requires significantly less computational effort while retaining a high level of accuracy.
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Dalton, Matthew W. "Experimental modelling of vascular haemodynamics." Thesis, Nottingham Trent University, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.273762.

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Dury, R. J. "Understanding haemodynamics in neurodegenerative disease." Thesis, University of Nottingham, 2018. http://eprints.nottingham.ac.uk/50380/.

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In this thesis, the haemodynamic, functional and structural changes in Alzheimer's Disease, Huntington's Disease and Multiple Sclerosis are assessed at 7T. Across all chapters, there is a focus on the use of Arterial Spin Labelling (ASL) to provide haemodynamic measures of perfusion (or cerebral blood flow) and transit time (TT) to provide a useful marker of disease. Arterial Spin Labelling (ASL) has the advantage that it is a non-invasive method to measure perfusion using magnetic resonance imaging (MRI). Clinically, perfusion is assessed using contrast-enhanced techniques which requires the intravenous administration of an exogenous gadolinium-based contrast agent, such as Prohance-TM and Gadovist-TM. Contrast-enhanced techniques typically provide higher SNR than ASL methods, however the non-invasive nature of ASL makes it a safe method suited for repeated measures in any subjects, including those with poor renal clearance. Additionally, gadolinium contrast agents have been shown to accumulate in neuronal tissue, and until the clinical significance of this is determined, contrast-enhanced scans should be performed with caution. In Chapter 5, arterial spin labelling is used to assess cerebral perfusion in a patient group with Alzheimer's Disease (AD) and compared with an age-matched healthy control group (HC). Functional MRI (fMRI) is used to assess functional connectivity within the default mode network (DMN) and measures compared between the AD and HC group. In addition, high resolution structural data is acquired to assess the effects of atrophy in AD. Results demonstrate a significant decrease in grey matter perfusion and a significant increase in grey matter transit time in the AD group compared the HC group. A trend showing a decrease in functional connectivity in the DMN was found in the AD group as compared to the HC group. As expected, significant grey matter loss and cortical thinning were observed in the AD group compared to the HC group. Secondly, haemodynamic and vascular changes in a Huntington's Disease (HD) patient group are assessed and compared with healthy age matched controls (HC). Phase contrast angiography is used to assess vessel density and vessel radius distributions between the two groups. Structural data was also acquired to assess grey matter volume and cortical thickness differences between the two groups. A significant reduction in perfusion was found in grey matter, putamen and the caudate in the HD group compared to the HC group. The ASL transit time was found to be significantly increased in the caudate and putamen in the HD group compared to the HC group. Phase contrast angiography data showed an increase in the frequency of smaller vessels (0.15-0.35mm) in the HD group compared to the HC group, whereas larger vessels appeared more frequently in the HC group. A significant reduction in grey matter volume was also observed in the HD group compared to the HC group, which manifested as thinning of the cortical ribbon. In the final study of this thesis, high spatial resolution arterial spin labelling is used to assess perfusion inside cortical lesions and compare with perfusion in surrounding normal appearing grey matter in a Multiple Sclerosis (MS) patient group. Grey matter perfusion as a function of distance from the cortical lesions was also assessed. It was found that cortical lesions have reduced perfusion compared to surrounding normal appearing grey matter. Perfusion increased and stabilised immediately outside of the cortical lesion itself, suggesting that the perfusion deficit observed is highly spatially localised.
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Williams, Stephen A. "Microvascular haemodynamics in essential hypertension." Thesis, Imperial College London, 1989. http://hdl.handle.net/10044/1/47712.

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Hall, Emma Louise. "Quantitative methods to assess cerebral haemodynamics." Thesis, University of Nottingham, 2012. http://eprints.nottingham.ac.uk/12673/.

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In this thesis methods for the assessment of cerebral haemodynamics using 7 T Magnetic Resonance Imaging (MRI) are described. The measurement of haemodynamic parameters, such as cerebral blood flow (CBF), is an important clinical tool. Arterial Spin Labelling (ASL) is a non-invasive technique for CBF measurement using MRI. ASL methodology for ultra high field (7 T) MRI was developed, including investigation of the optimal readout strategy. Look-Locker 3D-EPI is demonstrated to give large volume coverage improving on previous studies. Applications of methods developed to monitor functional activity, through flow or arterial blood volume, in healthy volunteers and in patients with low grade gliomas using Look-Locker ASL are described. The effect of an increased level of carbon dioxide in the blood (hypercapnia) was studied using ASL and functional MRI; hypercapnia is a potent vasodilator and has a large impact on haemodynamics. These measures were used to estimate the increase in oxygen metabolism associated with a simple motor task. To study the physiology behind the hypercapnic response, magnetoencephalography was used to measure the impact of hypercapnia on neuronal activity. It was shown that hypercapnia induces widespread desynchronisation in a wide frequency range, up to ~ 50 Hz, with peaks in the sensory-motor areas. This suggests that hypercapnia is not iso-metabolic, which is an assumption of calibrated BOLD. A Look-Locker gradient echo sequence is described for the quantitative monitoring of a gadolinium contrast agent uptake through the change in longitudinal relaxation rate. This sequence was used to measure cerebral blood volume in Multiple Sclerosis patients. Further development of the sequence yielded a high resolution anatomical scan with reduced artefacts due to field inhomogeneities associated with ultra high field imaging. This allows whole head images acquired at sub-millimetre resolution in a short scan time, for application in patient studies.
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Books on the topic "Haemodynamics"

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Cold, Georg E., and Niels Juul, eds. Monitoring of Cerebral and Spinal Haemodynamics During Neurosurgery. Berlin, Heidelberg: Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-77873-8.

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Gwynn, Brian Rodney. The influence of arteriovenous fistulae on in-situ vein graft haemodynamics. Birmingham: University of Birmingham, 1987.

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Huang, Chunlong. A study of the action of hormones and drugs on regional haemodynamics of the rat kidney. Birmingham: University of Birmingham, 1994.

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Mäkivirta, Aki. Use of the median filter in haemodynamic monitoring. Espoo: Technical Research Centre of Finland, 1992.

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Dillon, A. Haemodynamic profiles and the critically ill patient: A practical guide. Edited by Coombs M. A and Lyon J. Oxford (England): BIOS Scientific Publishers, 1997.

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Franceschi, Claude. Conservative haemodynamic cure of incompetent and varicose veins in ambulatory patients. Précy-sous-Thil: Éditions de l'Armançon, 1993.

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Partanen, Juhani. Cardiovascular responses induced by haemodynamic interventions and inotropics: A series of noninvasive studies. Helsinki: University Central Hospital, 1989.

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Fry, J. E. Effect of smoking on arterial stiffness and haemodynamic parameters in type 1 diabetes mellitus. Roehampton: University of Surrey Roehampton, 2004.

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Alfred Benzon Symposium (37th 1993 Royal Danish Academy of Sciences and Letters). Brain lesions in the newborn: Hypoxic and haemodynamic pathogenesis : Alfred Benzon Symposium 37 : proceedings of a symposium held at the Royal Society of Sciences and Letters, 15-19 August 1993. Edited by Greisen Gorm, Larsen J[0]rgen Falck, Lou Hans C, and Alfred Benzon Foundation. Copenhagen: Munksgaard, 1994.

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Cardiovascular Haemodynamics and Doppler Waveforms Explained. Greenwich Medical Media, 2001.

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Book chapters on the topic "Haemodynamics"

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Poelaert, Jan, Ruggero Amà, and Karl Skarvan. "Haemodynamics." In Transoesophageal Echocardiography in Anaesthesia and Intensive Care Medicine, 176–95. London, UK: BMJ Publishing Group, 2008. http://dx.doi.org/10.1002/9780470760239.ch10.

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McAndrew, Katherine, Maurizio Cecconi, and Andrew Rhodes. "Perioperative Haemodynamics." In Hemodynamic Monitoring, 107–15. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-69269-2_11.

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Jansen, Shirley, Michael Lawrence-Brown, Siamak Mishani, Christopher Lagat, Brian Evans, Kurt Liffman, and Ilija D. Šutalo. "Vascular Haemodynamics." In Mechanisms of Vascular Disease, 141–69. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-43683-4_7.

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Tu, Jiyuan, Kiao Inthavong, and Kelvin Kian Loong Wong. "Fundamentals of Haemodynamics." In Computational Hemodynamics – Theory, Modelling and Applications, 67–94. Dordrecht: Springer Netherlands, 2015. http://dx.doi.org/10.1007/978-94-017-9594-4_4.

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Sloth, Erik, Christian Alcaraz Frederiksen, and Peter Juhl-Olsen. "Non-Invasive Haemodynamics." In Emergency Point-of-Care Ultrasound, 375–78. Chichester, UK: John Wiley & Sons, Ltd, 2017. http://dx.doi.org/10.1002/9781119072874.ch35.

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Tu, Jiyuan, Kiao Inthavong, and Kelvin Kian Loong Wong. "Computational Haemodynamics—An Introduction." In Computational Hemodynamics – Theory, Modelling and Applications, 1–19. Dordrecht: Springer Netherlands, 2015. http://dx.doi.org/10.1007/978-94-017-9594-4_1.

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de Silva, D., and D. Kühne. "Haemodynamics in Interventional Neuroradiology." In Imaging of Brain Metabolism Spine and Cord Interventional Neuroradiology Free Communications, 397. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-74337-5_113.

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Smith, Brendan E., and Veronica M. Madigan. "Doppler Assessment of Haemodynamics." In Emergency Point-of-Care Ultrasound, 379–85. Chichester, UK: John Wiley & Sons, Ltd, 2017. http://dx.doi.org/10.1002/9781119072874.ch36.

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Bradley, Stanley E., Franz J. Ingelfinger, and Geraldine P. Bradley. "Determinants of Hepatic Haemodynamics." In Ciba Foundation Symposium - Visceral Circulation, 219–32. Chichester, UK: John Wiley & Sons, Ltd., 2008. http://dx.doi.org/10.1002/9780470718810.ch22.

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Brookes, Murray, and William J. Revell. "Introduction to bone haemodynamics." In Blood Supply of Bone, 203–9. London: Springer London, 1998. http://dx.doi.org/10.1007/978-1-4471-1543-4_15.

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Conference papers on the topic "Haemodynamics"

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"Investigation Haemodynamics in Parabolic Flight." In 55th International Astronautical Congress of the International Astronautical Federation, the International Academy of Astronautics, and the International Institute of Space Law. Reston, Virigina: American Institute of Aeronautics and Astronautics, 2004. http://dx.doi.org/10.2514/6.iac-04-g.3.06.

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Soulis, J. V. "Computational haemodynamics of left coronary artery." In BIOMEDICINE 2003, edited by T. M. Farmakis, G. D. Giannoglou, O. Faltsi, D. Sofialidis, J. Josipovic, and G. E. Louridas. Southampton, UK: WIT Press, 2003. http://dx.doi.org/10.2495/bio030181.

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Barbe, Kurt, Wendy Van Moer, and Guy Nagels. "Exploring the fractional haemodynamics in fMRI data." In 2011 IEEE International Symposium on Medical Measurements and Applications (MeMeA). IEEE, 2011. http://dx.doi.org/10.1109/memea.2011.5966686.

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Klöoking, H.-P., and K. Sedlarik. "EFFECT OF PENTOSAN POLYSULPHATE IN MINIPIGS WITH EXPERIMENTAL PULMONARY EMBOLI." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643250.

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In anaesthetized minipigs emboli were induced by reinjection of electrically activated autologous blood. The animals were then infused with either saline or pentosan polysulphate (SP 54) for three hours; 2 mg SP 54/kg body weight injected as bolus followed by intravenous infusion of 1 mg SP 54/kg body weight per hour. Before and during infusion of SP 54 or saline, we determined haemodynamics, degree of pulmonary arterial occlusion, blood gases and plasminogen activator levels. As a result of treatment with SP 54 a decrease in mean pulmonary arterial pressure, an increase in blood flow of pulmonary artery, and an increase in plasminogen activator activity was seen, compared with control animals who received saline infusion only. The efficacy of SP 54 treatment was demonstrated by a mean haemodynamlc and blood flow improvement of about 50 %, three hours after starting the experiment.
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Boutsianis, Evangelos, Thomas Frauenfelder, Hitendu Dave, Jurg Grunenfelder, Simon Wildermuth, Gregor Zund, Marko Turina, Dimos Poulikakos, and Yiannis Ventikos. "Cardiovascular Haemodynamic Simulations of Anatomically Accurate Coronaries." In ASME 2003 International Mechanical Engineering Congress and Exposition. ASMEDC, 2003. http://dx.doi.org/10.1115/imece2003-42728.

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The present study is devoted to the investigation of the pulsatile blood flow within the first few vessels of the Left Coronary Artery (LCA) vasculature of an anatomically accurate porcine coronary tree. Transient computational fluid dynamics simulations were performed under realistic pulsatile volume inflow boundary conditions. The numerical results have provided a comprehensive collection of information regarding the haemodynamics within the LCA and its major branches, namely the Left Anterior Descending (LAD) and the Left Circumflex (LCX) arteries. The underlying principle of developing computational techniques, which would eventually allow for the realistic simulation of the vascular haemodynamics of patients, lies on the capacity of such tools for predictive diagnostics and non-invasive, hence simulation-based, surgical planning.
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Williamson, P., P. D. Docherty, S. G. Yazdi, M. Jermy, A. Khanafer, N. Kabaliuk, and P. H. Geoghegan. "PIV Analysis of Stented Haemodynamics in the Descending Aorta." In 2019 41st Annual International Conference of the IEEE Engineering in Medicine & Biology Society (EMBC). IEEE, 2019. http://dx.doi.org/10.1109/embc.2019.8856823.

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Zhang, Y., F. Scopesi, G. Serra, J. W. Sun, and P. Rolfe. "Tissue oxygenation and haemodynamics measurement with spatially resolved NIRS." In Sixth International Symposium on Precision Engineering Measurements and Instrumentation. SPIE, 2010. http://dx.doi.org/10.1117/12.885441.

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Alshorman, Abdullah A. "Haemodynamics of Disturbed Flow Under Variable Shear Rate Effect." In ASME 7th Biennial Conference on Engineering Systems Design and Analysis. ASMEDC, 2004. http://dx.doi.org/10.1115/esda2004-58407.

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The local blood haemodynamics at curvatures and branches of the arteries system has a crucial role in controlling the localization of blood cells (i.e., leukocytes and platelets) at these sites and at sites of inflammation or vascular injuries respectively. Commonly, the local flow has different shapes of flow separation, recirculation, and non uniform shear stress distribution. To investigate these mechanisms, simulation of cell rolling and adhesion has been performed under variable position shear rate to determine the ranges of shear rates that allow for either adhesion or rolling. The results demonstrate that the cell had the three stages of cell-surface interactions (capture, adhesive rolling and secondary adhesion) when it starts at low shear rate G = 9.06 s−1. Nevertheless, at higher shear rate (i.e., 41.12 and 85.32 s−1) the cell rolls slowly for short time before its rolling velocity gradually increases to reach a maximum value as the shear rate gets higher. On the other hand, cell adhesion is dominant within the distance between the step and the stagnation point; such that the cells in this range rolsl shortly in the direction of shear rate then adheres to the surface.
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9

Peiffer, Véronique, Ethan M. Rowland, Stephanie G. Cremers, Peter D. Weinberg, and Spencer J. Sherwin. "Age-Related Differences in Haemodynamics of the Rabbit Aorta and Comparison With Average Maps of Atherosclerotic Lesion Prevalence." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80310.

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The focal occurrence of atherosclerotic lesions around branching points and in curved vessels has led to the hypothesis that haemodynamics plays an important role in the development of the disease. The spatial distribution of atherosclerosis is also age-dependent in humans and rabbits [1]; detailed maps of disease prevalence in aortas of immature and mature rabbits were presented recently [2]. Vincent et al. [3] applied computational fluid dynamics to study blood flow in the aorta of a mature rabbit, but they did not investigate age-dependent effects. The purpose of the present study was to quantify differences in aortic haemodynamics between immature and mature rabbits, and to compare averaged distributions of the flow-related wall shear stress with maps of disease prevalence. As blood flow is heavily dependent on vessel geometry, the shapes of the aorta in the two age groups were also compared.
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10

Boutsianis, Evangelos, Thomas Frauenfelder, Simon Wildermuth, Dimos Poulikakos, and Yiannis Ventikos. "Anatomically Accurate Haemodynamic Simulations of Abdominal Aortic Aneurysms." In ASME 2003 International Mechanical Engineering Congress and Exposition. ASMEDC, 2003. http://dx.doi.org/10.1115/imece2003-42766.

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The pulsatile blood flow field in a patient-specific pathology of a large Abdominal Aortic Aneurysm (AAA) is being simulated, both pre and post interventionally. The anatomies of the aortic wall and blood lumen have been derived by digitized Computerized Tomography (CT) scans. Three dimensional unsteady computational fluid dynamics simulations have provided a comprehensive collection of quantitative information on the haemodynamics and the flow features that present themselves in both the temporal and spatial spaces. The focus lies on alterations in the haemodynamics triggered by the interventional procedure itself, which consists of the endoluminal introduction of a stent-graft. Significant information may also be deduced concerning the hydrodynamic loading of such implants. Computational tools of this nature, along with the non-invasive CT or Magnetic Resonance (MR) aortic imaging techniques, could enable an objective assessment of the possible effects of any interventional scenario in a virtual noninvasive environment both proximally and distally to the diseased region.
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Reports on the topic "Haemodynamics"

1

Zhou, Suzhen, Denghua Li, Lieliang Zhang, and Juxiang Li. The anti-inflammatory and haemodynamic effects of levosimendan on advanced heart failure patients : a meta-analysis of randomized controlled trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2022. http://dx.doi.org/10.37766/inplasy2022.5.0097.

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