Relevant bibliographies by topics / Haemodynamic monitoring

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  1. Journal articles
  2. Dissertations / Theses
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  5. Conference papers

Academic literature on the topic 'Haemodynamic monitoring'

Author: Grafiati
Published: 4 June 2021
Last updated: 27 April 2022

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Journal articles on the topic "Haemodynamic monitoring"

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Bossaert, Leo L., Hendrik E. Demey, Raf De Jongh, and Luc Heytens. "Haemodynamic Monitoring." Drugs 41, no. 6 (June 1991): 857–74. http://dx.doi.org/10.2165/00003495-199141060-00004.

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Peruzzi, William T., and Jeffery S. Vender. "Haemodynamic monitoring." Current Opinion in Anaesthesiology 4, no. 1 (February 1991): 47–52. http://dx.doi.org/10.1097/00001503-199102000-00008.

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Payen, Didier. "Haemodynamic monitoring." Current Opinion in Anaesthesiology 4, no. 6 (December 1991): 821–26. http://dx.doi.org/10.1097/00001503-199112000-00012.

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Poelaert, Jan I. T. "Haemodynamic monitoring." Current Opinion in Anaesthesiology 14, no. 1 (February 2001): 27–32. http://dx.doi.org/10.1097/00001503-200102000-00005.

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Pinsky, Michael R. "Functional haemodynamic monitoring." Current Opinion in Critical Care 20, no. 3 (June 2014): 288–93. http://dx.doi.org/10.1097/mcc.0000000000000090.

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Clinton, Heidi. "Haemodynamic Monitoring in Theatre." British Journal of Anaesthetic and Recovery Nursing 4, no. 1 (February 2003): 10–16. http://dx.doi.org/10.1017/s1742645600002114.

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AbstractThe number of devices available to monitor the haemodynamic status of patients is increasing. Practitioners need to be aware of the non-invasive and invasive methods available in order to care for their patients safely and effectively. This article reviews a number of noninvasive measurements of haemodynamic function, in addition to invasive methods such as arterial blood pressure, central venous pressure and pulmonary artery pressure monitoring. It is argued that using these methods in combination provides a comprehensive haemodynamic assessment.
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Garretson, Sharon. "Haemodynamic monitoring: arterial catheters." Nursing Standard 19, no. 31 (April 13, 2005): 55–64. http://dx.doi.org/10.7748/ns2005.04.19.31.55.c3843.

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Pang, Qianyun, Jan Hendrickx, Hong-Liang Liu, and Jan Poelaert. "Contemporary perioperative haemodynamic monitoring." Anaesthesiology Intensive Therapy 51, no. 2 (2019): 147–58. http://dx.doi.org/10.5114/ait.2019.86279.

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Hofer, Christoph K., Maurizio Cecconi, Gernot Marx, and Giorgio della Rocca. "Minimally invasive haemodynamic monitoring." European Journal of Anaesthesiology 26, no. 12 (December 2009): 996–1002. http://dx.doi.org/10.1097/eja.0b013e3283300d55.

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Garretson, Sharon. "Haemodynamic monitoring: arterial catheters." Nursing Standard 19, no. 31 (April 13, 2005): 55–67. http://dx.doi.org/10.7748/ns.19.31.55.s51.

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Dissertations / Theses on the topic "Haemodynamic monitoring"

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Currey, Judy A., and mikewood@deakin edu au. "Critical care nurses' haemodynamic decision making." Deakin University. School of Nursing, 2003. http://tux.lib.deakin.edu.au./adt-VDU/public/adt-VDU20050728.094123.

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For cardiac surgical patients, the immediate 2-hour recovery period is distinguished by potentially life-threatening haemodynamic instability. To ensure optimum patient outcomes, nurses of varying levels of experience must make rapid and accurate decisions in response to episodes of haemodynamic instability. Decision complexity, nurses’ characteristics, and environmental characteristics, have each been found to influence nurses' decision making in some form. However, the effect of the interplay between these influences on decision outcomes has not been investigated. The aim of the research reported in this thesis was to explore variability in critical care nurses' haemodynamic decision making as a function of interplay between haemodynamic decision complexity, nurses' experience, and specific environmental characteristics by applying a naturalistic decision making design. Thirty-eight nurses were observed recovering patients in the immediate 2-hour period after cardiac surgery. A follow-up semi-structured interview was conducted. A naturalistic decision making approach was used. An organising framework for the goals of therapy related to maintaining haemodynamic stability after cardiac surgery was developed to assist the observation and analysis of practice. The three goals of therapy were the optimisation of cardiovascular performance, the promotion of haemostasia, and the reestablishment of normothermia. The research was conducted in two phases. Phase One explored issues related to observation as method, and identified emergent themes. Phase Two incorporated findings of Phase 1, investigating the variability in nurses' haemodynamic decision making in relation to the three goals of therapy. The findings showed that patients had a high acuity after cardiac surgery and suffered numerous episodes of haemodynamic instability during the immediate 2-hour recovery period. The quality of nurses' decision making in relation to the three goals of therapy was influenced by the experience of the nurse and social interactions with colleagues. Experienced nurses demonstrated decision making that reflected the ability to recognise subtle changes in haemodynamic cues, integrate complex combinations of cues, and respond rapidly to instability. The quality of inexperienced nurses' decision making varied according to the level and form of decision support as well as the complexity of the task. When assistance was provided by nursing colleagues during the reception and recovery of patients, the characteristics of team decision making were observed. Team decision making in this context was categorised as either integrated or non integrated. Team decision making influenced nurses' emotions and actions and decision making practices. Findings revealed nurses' experience affected interactions with other team members and their perceptions of assuming responsibility for complex patients. Interplay between decision complexity, nurses' experience, and the environment in which decisions were made influenced the quality of nurses' decision making and created an environment of team decision making, which, in turn, influenced nurses' emotional responses and practice outcomes. The observed variability in haemodynamic decision making has implications for nurse education, nursing practice, and system processes regarding patient allocation and clinical supervision.
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Saxena, Rohit. "An assessment of two novel tools for advanced haemodynamic monitoring in critically ill children." Thesis, King's College London (University of London), 2016. http://kclpure.kcl.ac.uk/portal/en/theses/an-assessment-of-two-novel-tools-for-advanced-haemodynamic-monitoring-in-critically-ill-children(f246dfbb-72ed-4b2e-a394-b9d28444ccec).html.

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Background: Critically ill children require accurate haemodynamic assessment to evaluate the severity of illness or response to therapy. Clinical estimation of cardiac output is inaccurate. Hence, advanced haemodynamic monitoring devices may help guide physicians towards the most appropriate treatment strategy. However, none of the currently available monitors for children fulfil all the criteria of an ideal device. Methods: We evaluated two novel minimally invasive haemodynamic monitoring devices in 100 critically ill children. The transpulmonary ultrasound dilution (TPUD) method is a validated indicator dilution based technique for measuring cardiac output in children. Pressure recording analytical method (PRAM) is an arterial pulse contour based method and is not yet validated in children. We compared PRAM with TPUD both in terms of agreement with absolute values of CO and also quantified the ability of PRAM to track changes in CO in response to therapy. We also evaluated the ability of TPUD to identify, and quantify, small anatomic shunts. Finally, a range of variables measured by TPUD and PRAM were assessed for their ability to predict response to fluid bolus administration. The contribution of baseline myocardial contractility towards that response was also evaluated. Results: PRAM showed unacceptable level of error for estimation of absolute values of CO and was unable to accurately track changes in CO. TPUD could identify small anatomic shunts. All of the volumetric variables were unable to predict accurately for fluid responsiveness. Myocardial contractility was found to be an important determinant of the response of stroke volume to fluid bolus administration. Conclusion: A revision of the current algorithm of PRAM is recommended for measurement of CO in children. The predictive ability of the studied variables was poor to moderate for determining response of stroke volume to fluid bolus administration.
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Mathavakkannan, Suresh. "Techniques to assess volume status and haemodynamic stability in patients on haemodialysis." Thesis, University of Hertfordshire, 2010. http://hdl.handle.net/2299/4811.

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Volume overload is a common feature in patients on haemodialysis (HD). This contributes significantly to the cardiovascular disease burden seen in these patients. Clinical assessments of the volume state are often inaccurate. Techniques such as interdialytic blood pressure, relative blood volume monitoring, bioimpedance are available to improve clinical effectives. However all these techniques exhibit significant shortcomings in their accuracy, reliability and applicability at the bed side. We evaluated the usefulness of a dual compartment monitoring technique using Continuous Segmental Bioimpedance Spectroscopy (CSBIS) and Relative Blood Volume (RBV) as a tool to assess hydration status and determine dry weight. We also sought to evaluate the role of Atrial Natriuretic Peptide (ANP) and B-type Natriuretic Peptide (BNP) as a volume marker in dialysis patients. The Retrospective analysis of a historical cohort (n = 376, 55 Diabetic) showed a significant reduction in post-dialysis weights in the first three months of dialysis (72.5 to 70kg, p<0.027) with a non-significant increase in weight between months 6-12. The use of anti-hypertensive agents reduced insignificantly in the first 3 months, increased marginally between months 3-6 and significantly increased over the subsequent 6 months. The residual urea clearance (KRU) fell and dialysis times increased. The cohort was very different to that dialysing at Tassin and showed a dissociation between weight reduction and BP control. This may relate to occult volume overload. CSBIS-RBV monitoring in 9 patients with pulse ultrafiltration (pulse UF) showed distinct reproducible patterns relating to extra cellular fluid (ECF) and RBV rebound. An empirical Refill Ratio was then used to define the patterns of change and this was related to the state of their hydration. A value closer to unity was consistent with the attainment of best achievable target weight. The refill ratio fell significantly between the first (earlier) and third (last) rebound phase (1.97 ± 0.92 vs 1.32 ± 0.2). CSBIS monitoring was then carried out in 31 subjects, whilst varying dialysate composition, temperature and patient posture to analyse the effects of these changes on the ECF trace and to ascertain whether any of these interventions can trigger a change in the slope of the ECF trace distinct to that caused by UF. Only, isovolemic HD caused a change in both RBV and ECF in some patients that was explained by volume re-distribution due to gravitational shifts, poor vascular reactivity, sodium gradient between plasma and dialysate and the use of vasodilating antihypertensive agents. This has not been described previously. These will need to be explored further. The study did demonstrate a significant lack of comparability of absolute values of RECF between dialysis sessions even in the same patient. This too has not been described previously. This is likely to be due to subtle changes in fluid distribution between compartments. Therefore a relative changes must be studied. This sensitivity to subtle changes may increase the usefulness of the technique for ECF tracking through dialysis. The potential of dual compartment monitoring to track volume changes in real time was further explored in 29 patients of whom 21 achieved weight reductions and were able to be restudied. The Refill Ratio decreased significantly in the 21 patients who had their dry weights reduced by 0.95 ± 1.13 kg (1.41 ± 0.25 vs 1.25 ± 0.31). Blood pressure changes did not reach statistical significance. The technique was then used to examine differences in vascular refill between a 36oC and isothermic dialysis session in 20 stable prevalent patients. Pulse UF was carried out in both these sessions. There were no significant differences in Refill Ratios, energy removed and blood pressure response between the two sessions. The core temperature (CT) of these patients was close to 36oC and administering isothermic HD did not confer any additional benefit. Mean BNP levels in 12 patients during isovolemic HD and HD with UF did not relate to volume changes. ANP concentrations fell during a dialysis session in 11 patients from a mean 249 ± 143 pg/ml (mean ± SD) at the start of dialysis to 77 ± 65 pg/ml at the end of the session (p<0.001). During isolated UF levels did not change but fell in the ensuing sham phase indicating a time lag between volume loss and decreased generation. (136±99 pg/ml to 101±77.2 pg/ml; p<0.02) In a subsequent study ANP concentrations were measured throughout dialysis and in the post-HD period for 2 hours. A rebound in ANP concentration was observed occurring at around 90 min post-HD. The degree of this rebound may reflect the prevailing fluid state and merit further study. We have shown the utility of dual compartment monitoring with CSBIS-RBV technique and its potential in assessing volume changes in real time in haemodialysis patients. We have also shown the potential of ANP as an independent marker of volume status in the same setting. Both these techniques merit further study.
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Vaquer, Araujo Sergi. "Evaluation of femoral dP/dt max as a marker of cardiac function in critically ill patients." Doctoral thesis, Universitat Autònoma de Barcelona, 2021. http://hdl.handle.net/10803/673247.

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El màxim increment de pressió per unitat de temps (dP/dtmax) mesurat a nivell de l’arteria femoral és considerat per molts com un marcador fiable i mínimament invasiu de la contractilitat cardíaca. A diferència de la mesura del dP/dtmax al ventricle esquerre, el dP/dtmax femoral te lloc durant la fase d’ejecció ventricular del cicle cardíac i, per tant, hauria d’estar subjecte als efectes de la post-càrrega i dependre del to arterial. A demès, i de forma similar al dP/dtmax del ventricle esquerre, la mesura del dP/dtmax femoral podria estar subjecte a variacions en la pre-càrrega cardíaca a través de múltiples mecanismes. L’objectiu de la present tesi doctoral és el d’analitzar els efectes de la pre-càrrega i la post-càrrega en la mesura del dP/dtmax femoral, i avaluar la seva validesa com a marcador de contractilitat cardíaca. Es van realitzar dos estudis per respondre a les hipòtesis formulades. En el primer es van analitzar els canvis en el dP/dtmax femoral de manera prospectiva durant canvis en la contractilitat cardíaca (variacions en la dosi d’infusió de dobutamina), post-càrrega (variacions en la dosi d’infusió de noradrenalina) i en la pre-càrrega (mitjançant l’administració d’una càrrega de volum estàndard o una maniobra d’elevació de les cames) en pacients crítics. En el segon, es va realitzar un estudi retrospectiu en pacients crítics que havien rebut una càrrega de volum estàndard on es va analitzar la rellevància de la pre-càrrega dependència sobre la resposta del dP/dtmax femoral a canvis en la pre-càrrega cardíaca. Els resultats suggereixen que, tot i que pot existir una relació entre les mesures de dP/dtmax femoral i ventricular, el dP/dtmax femoral és altament susceptible a canvis en la post-carrega ventricular i el to arterial, i canvia amb els canvis de pre-càrrega en pacients en situació de pre-càrrega dependència. Addicionalment, el dP/dtmax femoral va mostrar una relació quasi directe amb la pressió de pols en tots els casos, el qué suggereix que qualsevol canvi en aquesta pot influenciar la mesura del dP/dtmax femoral. Basant-se en els resultats obtinguts, es pot concloure amb una raonable certesa que el dP/dtmax femoral no és un marcador adequat de la contractilitat cardíaca, donat que es veu influenciat pels canvis en la pre i post-càrrega ventricular, i per tant, no s’hauria d’emprar com a tal en la practica clínica habitual.
El máximo incremento de presión por unidad de tiempo (dP/dtmax) medido a nivel de la arteria femoral es considerado por muchos como un marcador fiable y mínimamente invasivo de la contractilidad cardíaca. A diferencia de la medida del dP/dtmax en el ventrículo izquierdo, el dP/dtmax femoral tiene lugar durante la fase de eyección ventricular del ciclo cardíaco y, por tanto, debería estar sujeta a los efectos de la post-carga y depender del tono arterial. Además, y de forma similar al dP/dtmax del ventrículo izquierdo, la medida del dP/dtmax femoral podría estar sujeta a variaciones en la pre-carga cardíaca a través de múltiples mecanismos. El objetivo de la presente tesis doctoral es el analizar los efectos de la pre-carga y la post-carga en la medida del dP/dtmax femoral, y evaluar su validez como marcador de contractilidad cardíaca. Se realizaron dos estudios para responder a las hipótesis formuladas. En el primero se analizaron los cambios en el dP/dtmax femoral de manera prospectiva durante cambios en la contractilidad cardíaca (variaciones en la dosis de infusión de dobutamina), post-carga (variaciones en la dosis de infusión de noradrenalina) y en la pre-carga (mediante la administración de una carga de volumen estándar o una maniobra de elevación de las piernas) de pacientes críticos. En el segundo, se realizó un estudio retrospectivo en pacientes críticos que habían recibido una carga de volumen estándar donde se analizó la relevancia de la pre-carga dependencia sobre la respuesta del dP/dtmax femoral a cambios en la pre-carga cardíaca. Los resultados sugieren que, a pesar de que puede existir una relación entre las medidas de dP/dtmax femoral y ventricular, el dP/dtmax femoral es altamente susceptible a cambios en la post-carga ventricular y el tono arterial, y cambia con las variaciones de pre-carga en pacientes en situación de pre-carga dependencia. Adicionalmente, el dP/dtmax femoral mostró una relación cuasi directa con la presión de pulso en todos los casos, lo que sugiere que cualquier cambio en ésta puede influenciar la medida del dP/dtmax femoral. De acuerdo con los resultados obtenidos, se puede concluir con una certeza razonable que el dP/dtmax femoral no es un marcador adecuado de la contractilidad cardíaca, dado que se ve influenciado por los cambios en la pre y post-carga ventricular, y por tanto, no se debería utilizar como tal en la práctica clínica habitual.
Femoral dP/dtmax (the maximum rise of pressure over time) has been considered by many as a minimally invasive, peripheral marker of left ventricle contractility. However, in contrast to left ventricular dP/dtmax, femoral dP/dtmax occurs during the ejection phase of the cardiac cycle and should therefore be subject to afterload and arterial load variations. Furthermore, similar to the left ventricle dP/dtmax, femoral dP/dtmax might be subject to preload variations through several potential mechanisms. The objective of the present work was to elucidate the effects of left ventricle loading conditions (preload and afterload) on the measure of femoral dP/dtmax and to assess the reliability of this marker as an estimator of cardiac contractility. Two studies were performed to address the hypothesis formulated for the present doctoral work. In the first, changes in femoral dP/dtmax were prospectively monitored during contractility (dobutamine infusion change), afterload (norepinephrine dose change) and preload (passive leg raising or volume expansion) variations in critically ill patients. In the second, a retrospective analysis of a database of critically ill patients receiving fluid infusion was analysed to evaluate the relevance of preload-dependence on the responsiveness of femoral dP/dtmax to preload variations. Results suggested that although a link between femoral dP/dtmax and left ventricle contractility variations may exists, femoral dP/dtmax is highly susceptible to changes in afterload and arterial load, and varies with preload increases in preload-dependent patients. Furthermore, femoral dP/dtmax retained an almost direct correlation with pulse pressure in all cases, suggesting that any change in the later should also influence the former. According to present results, it can therefore be concluded with reasonable certainly that femoral dP/dtmax is not an adequate marker of cardiac contractility as it is affected by left ventricular loading conditions and should not be used as such in clinical practice.
Universitat Autònoma de Barcelona. Programa de Doctorat en Medicina
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Daly, Jonathan. "Video camera monitoring to detect changes in haemodynamics." Thesis, University of Oxford, 2016. https://ora.ox.ac.uk/objects/uuid:e84f2acf-f35c-4257-a4c3-209c5da9cbee.

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Patients in hospital can be prone to sudden, life-threatening changes in their cardiovascular state. Haemodynamic parameters such as blood pressure, pulse transit time (PTT) and perfusion can be monitored in clinical situations to identify these changes as early as possible. Continuous blood pressure is usually monitored using a catheter placed into a major artery, but this is invasive and involves risk to the patient. In the last decade, the field of non-contact vital sign monitoring has emerged, with growing evidence that the remote photoplethysmogram (rPPG) signal can be used to estimate vital signs using video cameras. If the analysis of the rPPG signal can be expanded to include the estimation of haemodynamic parameters, it could result in methods for the continuous, non-contact monitoring of a subject's haemodynamic state. In a physiology study, a series of video recordings were made of 43 healthy volunteers. The subjects sat in a purpose-built chamber, and the composition of the air was carefully adjusted to cause the subjects to experience large, controlled changes in blood oxygen levels. To validate the video camera algorithms, reference data were also collected. Along with the volunteer study, a clinical study was performed to acquire data in a challenging clinical environment. Data were collected from patients on haemodialysis in the Renal Unit, a population likely to experience sudden changes in haemodynamics. The reference data from the Renal Unit study were analysed to determine the extent to which PTT and mean arterial pressure (MAP) are related. The correlation coefficients and linear fits were found on a global and a per-subject basis. In addition, the video recordings from the Physiology study were processed to derive rPPG signals, and these signals were analysed to obtain estimates for PTT. Local rPPG signals were also derived for different regions of interest, and the waveforms were analysed using a novel application of the technique of signal averaging to produce spatial maps of perfusion and blood flow. The correlation between conventionally measured PTT and MAP was found to be weaker in the haemodialysis population than has been shown elsewhere in the literature, except for a sub-set of patients. The results of the video analysis showed that PTT could be estimated robustly and consistently, although direct validation of these estimates was not possible because of the different method used to calculate the reference PTT. For most subjects, the spatial mapping methods produced robust maps that were consistent over time. These results suggest that it is possible to detect changes in haemodynamics using a video camera, and that this could have applications in healthcare, providing that challenges such as subject movement and clinical validation can be overcome.
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Thomas, Nicholas. "On the application of the Doppler effect in pulsed Doppler flowmeters and the effect of certain propagation and scattering artifacts." Thesis, King's College London (University of London), 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.297092.

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Aldrich, Clive Jeffrey. "Intrapartum fetal cerebral oxygenation and haemodynamics assessed by near infrared spectroscopy (NIRS)." Thesis, University of Southampton, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.320699.

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Wesołowski, Roman. "Development of arterial spin labelling methods for monitoring cerebral haemodynamics." Thesis, University of Nottingham, 2010. http://eprints.nottingham.ac.uk/13854/.

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The work described in this thesis was carried out at the Sir Peter Mansfield Magnetic Resonance Centre at the University of Nottingham between March 2006 and December 2009. All work described in this thesis was performed by the author, except where indicated. This thesis aims to develop and implement ASL techniques to measure haemodynamic responses to neural activity. The development of a new technique Double Acquisition Background Suppression (DABS) is presented as a remedy for a newly discovered artefact affecting Philips Achieva 7 T scanners and other sources of variation in baseline signals such as physiological noise. The new technique (DABS) was developed for simultaneous acquisition of ASL (with suppressed static tissue signal) and BOLD data using the FAIR scheme. This method not only provided a solution to obtaining ASL data at 7 T, despite the Roman Artefact, but also proved to reduce the contribution of physiological noise to ASL images, which is problematic, especially at ultra-high magnetic field strengths. The statistical verification was carried out based on the neural activation induced by a finger-tapping stimulus. A simplified model for quantifying CBVa.with the Look-Locker sampling method is proposed in this thesis to overcome the need for the Step-wise Compartmental Model (SCM). The Look-Locker sampling scheme acquires multiple readout pulses following the labelling and provides an estimation of transit time as well as CBVa. Here the simplified model is used to assess changes due to visual stimulation and validated against the SCM model. The application of LL-FAIR to form CBF and CBVa weighted data with improved SNR compared to traditional single TI FAIR technique is then shown. This method uses a summation over LL-EPI readout pulses and is used to asses the temporal characteristics and absolute changes in CBF and CBVa haemodynamic responses to a short (4.8 s) and long (9.6 s) visual stimulus. LL-FAIR methods are then used to appraise the neural coupling of haemodynamic parameters and assess Grubb's relationship. CBF and CBVa. Data were collected together with CBVtot data from a bolus injection of contrast agent. Assessing Grubb's power-law (CBVtot = CBFCI:)for neuronal activation, which was originally derived in primates during a steady state response of hypercapnia, a was found in this human study to be between 0.22 ± 0.08 and 0.29, dependent on the analysis method. In addition, the power-law relationship between CBVtot and CBVa.was assessed, and resulted in a similar relation, yielding aTA = 0.42 ± 0.14 and 0.40. Since CBF is thought to be driven by CBVa.the power-law between these parameters was also tested with a value of aFA = 1.35 ± 0.64 and 1.21, found in close agreement with earlier animal work.
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Donnelly, Joseph. "Intracranial monitoring after severe traumatic brain injury." Thesis, University of Cambridge, 2018. https://www.repository.cam.ac.uk/handle/1810/271422.

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Intracranial monitoring after severe traumatic brain injury offers the possibility for early detection and amelioration of physiological insults. In this thesis, I explore cerebral insults due raised intracranial pressure, decreased cerebral perfusion pressure and impaired cerebral pressure reactivity after traumatic brain injury. In chapter 2, the importance of intracranial pressure, cerebral perfusion pressure and pressure reactivity in regulating the cerebral circulation is elucidated along with a summary of the existing evidence supporting intracranial monitoring in traumatic brain injury. In chapter 4, intracranial pressure, cerebral perfusion pressure, and pressure reactivity insults are demonstrated to be common, prognostically important, and responsive to long-term changes in management policies. Further, while these insults often occur independently, coexisting insults portend worse prognosis. In chapter 5, I examine possible imaging antecedents of raised intracranial pressure and demonstrate that initial subarachnoid haemorrhage is associated with the subsequent development of elevated intracranial pressure. In addition, elevated glucose during the intensive care stay is associated with worse pressure reactivity. Cortical blood flow and brain tissue oxygenation are demonstrated to be sensitive to increases in intracranial pressure in chapter 6. In chapter 7, a method is proposed to estimate the cerebral perfusion pressure limits of reactivity in real-time, which may allow for more nuanced intensive care treatment. Finally, I explore a recently developed visualisation technique for intracranial physiological insults and apply it to the cerebral perfusion pressure limits of reactivity. Taken together, this thesis outlines the scope, risk factors and consequences of intracranial insults after severe traumatic brain injury. Novel signal processing applications are presented that may serve to facilitate a physiological, personalised and precision approach to patient therapy.
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Thomson, Stephen D. "Exercise testing and non-invasive haemodynamics in the assessment and monitoring of pulmonary hypertension : novel submaximal and peak exercise variables." Thesis, University of Glasgow, 2017. http://theses.gla.ac.uk/8918/.

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Pulmonary hypertension is a disease characterised by progressive pulmonary vascular remodelling and obliteration with consequent development of right heart failure and ultimately death. First described many decades ago with a median survival of less than 3 years and no available treatments, the development of disease specific pulmonary vasodilator therapy has led to only modest improvements in survival and it remains an almost universally fatal disease. One of the key symptoms of pulmonary hypertension is exercise intolerance, primarily a consequence of the underlying right ventricular failure and an inability to augment stroke volume on exercise. The gold standard diagnostic test is right heart catheterisation but this is unattractive as a tool for ongoing monitoring as it is invasive and not without risk, albeit that risk is small. As a result most monitoring of disease progression and of treatment response is carried out using surrogate markers, often exercise based such as the 6 minute walk test. Increasing attention is focused on the role of exercise both in that monitoring of patients and also in helping to understand better the pathophysiology. The work presented in this thesis therefore aimed to explore novel exercise derived variables and noninvasive haemodynamic measurement as tools to improve our understanding of the disease limitation, to enhance our monitoring of treatment response and to give additional prognostic information. In Chapter 3 the role of peripheral muscle oxygen extraction and exercise limitation was explored by performing right heart catheterisation on exercise with measurement of mixed venous oxygen saturation. This demonstrated that patients with pulmonary hypertension demonstrate no evidence of impaired oxygen extraction and that they appear to extract at least as much oxygen on exercise as healthy individuals have been shown to in other studies. This indicates that impairment of oxygen extraction is not a cause of exercise limitation in pulmonary hypertension. 3 Chapter 4 describes a series of studies evaluating the potential role of the oxygen uptake efficiency slope in pulmonary hypertension. This variable derived from the oxygen consumption and ventilation across an incremental cardiopulmonary exercise test has demonstrated promise as a potential submaximal measure of exercise performance and predictor of survival in left heart failure. The studies conducted demonstrated that this variable is a measure of peak exercise performance in pulmonary hypertension, that it can be measure on submaximal levels of exercise and that it predicts survival in patients with Group 1 and Group 4 disease. The studies described in Chapter 5 investigated the rates of recovery of heart rate and oxygen consumption after exercise and found that both could predict survival. In particular the rate of recovery of heart rate after exercise was demonstrated to be a strong predictor of survival on multivariate analysis, thus providing a further method of assessing prognosis with exercise. Finally the ability of noninvasive measures of stroke volume to predict outcome was explored in the studies detailed in Chapter 6. The underlying haemodynamic abnormalities are not assessed when surrogate measures such as exercise testing are employed in patient follow up. Standard practice is to review patients 3 to 4 months after any change in treatment and to assess them using these surrogate measures. Acute haemodynamic changes are able to be detected invasively immediately after administration of pulmonary vasodilator therapy. This study therefore investigated the ability of two noninvasive methods of measuring stroke volume, inert gas rebreathing and cardiac MRI, to detect treatment response after only 2 weeks and assess how this related to functional improvement at the standard 4 months. The study found that haemodynamic changes were able to be detected at 2 weeks and these appeared to relate to changes in 6 minute walk distance at the same time point but did not appear to relate to 6 minute walk distance at 4 months. This study however did not reach its recruitment target and therefore further work is needed in this area.
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Books on the topic "Haemodynamic monitoring"

1

Mäkivirta, Aki. Use of the median filter in haemodynamic monitoring. Espoo: Technical Research Centre of Finland, 1992.

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2

Dillon, A. Haemodynamic profiles and the critically ill patient: A practical guide. Edited by Coombs M. A and Lyon J. Oxford (England): BIOS Scientific Publishers, 1997.

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3

Cold, Georg E., and Niels Juul, eds. Monitoring of Cerebral and Spinal Haemodynamics During Neurosurgery. Berlin, Heidelberg: Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-77873-8.

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Haemodynamic Monitoring And Manipulation. M&K; Update Ltd, 2009.

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(Editor), Jean-Francois Dhainaut, and Didier Payen (Editor), eds. Strategy in Bedside Haemodynamic Monitoring (Update in Intensive Care & Emergency Medicine). Springer-Verlag Berlin and Heidelberg GmbH & Co. K, 1991.

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6

Leach, Dr Richard, and Professor Kevin Moore. Practical procedures and monitoring. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199565979.003.00019.

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Chapter 19 covers practical procedures and monitoring of patients presenting with vascular and haemodynamic, respiratory, gastrointestinal, and other conditions, as well as additional various practical procedures.
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HAEMODYNAMIC PROFILES AND THE CRITICALLY ILL PATIENT (Understanding Cardiac Output Studies). Taylor & Francis, 1996.

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8

Bloos, Frank, and Konrad Reinhart. Mixed and central venous oxygen saturation monitoring in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0134.

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Haemodynamic resuscitation should target goals that reflect the tissue oxygen needs of an individual patient. Venous oximetry may be such a tool. Oxygen saturation of blood in the pulmonary artery contains venous blood from the whole body and is referred to as mixed oxygen saturation (SvO2). Measurement of oxygen saturation in blood obtained from a central venous catheter is referred to as central venous oxygen saturation (ScvO2). Both values are not identical since a catheter placed into the superior vena cava only represents venous blood draining the upper body. While it is not possible, in the clinical setting, to predict SvO2 from ScvO2, changes in SvO2 are adequately mirrored by changes in ScvO2. Post-operative patients and patients admitted to intensive care with a low ScvO2 show a higher morbidity and mortality. Early goal-directed therapy (EGDT) combines several haemodynamic goals into a treatment algorithm, including a ScvO2 target. However, recent studies do not support the systematic use of this protocolized approach. A normal value of SvO2 or ScvO2 saturation does not always exclude tissue hypoxia, since it is not possible to identify an inadequate oxygen supply in single organs. A further limitation of this technique is that organ dysfunction can progress, or serum lactate increases, despite normal or even increased venous oximetry values.
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Arulkumaran, Nishkantha, and Maurizio Cecconi. Cardiac output assessment in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0136.

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Haemodynamic monitoring facilitates effective resuscitation and the rapid assessment of the response to time-dependent vasoactive and fluid therapyin different shock states. Since the introduction of the pulmonary artery catheter, several minimally and non-invasive CO monitoring devices have been introduced to provide continuous monitoring and a dynamic profile of fluid responsiveness. Several of these monitors provide additional haemodynamic parameters including dynamic indices of preload and volumetric indices. Patient outcome is dependent accurate acquisition and interpretation of data and subsequent management. Whilst data from CO monitors offer valuable information on global hamodynamics, they do not preclude tissue hypoperfusion. Furthermore, there is no ‘ideal’ CO value for an individual patient, and the trend in haemodynamic parameters in response to therapy may be more informative than the absolute values. CO monitoring should be based upon the patient’s needs, the clinical scenario, and the experience of the treating physician.
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10

Sidhu, Kulraj S., Mfonobong Essiet, and Maxime Cannesson. Cardiac and vascular physiology in anaesthetic practice. Edited by Jonathan G. Hardman. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0001.

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This chapter discusses key components of cardiovascular physiology applicable to clinical practice in the field of anaesthesiology. From theory development to ground-breaking innovations, the history of cardiac and vascular anatomy, as well as physiology, is presented. Utilizing knowledge of structure and function, parameters created have allowed adequate patient clinical assessment and guided interventions. A review of concepts reveals the impact of multiple physiological variables on a patient’s haemodynamic state and the need for more accurate and efficient measurements. In particular, it is noted that a more reliable index of ventricular contractility is the end-systolic elastance rather than the ejection fraction. Constant direct preload assessment has not yet been achieved but continues to be determined through surrogate variables, and continuous cardiac output monitoring for oxygen delivery, although advancing, has limitations. Considering the effect of compound factors perioperatively, especially heart failure, modifies the goals and interventions of anaesthetists to achieve improved outcomes. Therefore, medical management prior to surgery and complete assessment through history, physical examination, and diagnostic tests are a priority. This chapter also details the expectations following volume expansion to augment haemodynamics during surgery, the concept of functional haemodynamic monitoring, and limitations to the parameters applied in assessing fluid responsiveness. Challenging the accuracy of conventional indices to predict volume status led to the use of goal-directed therapy, reducing morbidity and minimizing length of hospital stay. The mainstay of this chapter is to reinforce the relevance of advances in haemodynamic monitoring and homeostasis optimization by anaesthetists during surgery, using fundamental concepts of cardiovascular physiology.
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Book chapters on the topic "Haemodynamic monitoring"

1

Boyle, Maureen, and Judy Bothamley. "Haemodynamic monitoring." In Critical Care Assessment by Midwives, 19–29. New York, NY : Routledge, 2018.: Routledge, 2018. http://dx.doi.org/10.4324/9781315183657-3.

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Woodrow, Philip. "Haemodynamic monitoring." In Intensive Care Nursing, 190–203. Fourth edition. | Abingdon, Oxon ; New York, NY : Routledge, 2018.: Routledge, 2018. http://dx.doi.org/10.4324/9781315231174-20.

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Guarracino, Fabio, and Rubia Baldassarri. "Haemodynamic Monitoring." In ECMO-Extracorporeal Life Support in Adults, 375–82. Milano: Springer Milan, 2014. http://dx.doi.org/10.1007/978-88-470-5427-1_32.

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Edwards, Mark R., and Rupert M. Pearse. "Perioperative Haemodynamic Optimisation." In Hemodynamic Monitoring, 457–67. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-69269-2_36.

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Frasacco, Giulia, and Luigi Tritapepe. "Haemodynamic Monitoring During Anaesthesia." In Practical Trends in Anesthesia and Intensive Care 2017, 87–101. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-61325-3_6.

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Magder, S. "Principles of haemodynamic monitoring." In Anaesthesia, Pain, Intensive Care and Emergency Medicine — A.P.I.C.E., 619–34. Milano: Springer Milan, 2003. http://dx.doi.org/10.1007/978-88-470-2215-7_1.

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Jevon, Philip, Beverley Ewens, and Jagtar Singh Pooni. "Monitoring Cardiovascular Function 2: Haemodynamic Monitoring." In Monitoring the Critically III Patient, 115–53. West Sussex, UK: John Wiley & Sons, Ltd,., 2013. http://dx.doi.org/10.1002/9781118702932.ch5.

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Poli, M. Dei, B. Allaria, A. Trivellato, and P. Ferrario. "Technical Aspects of Haemodynamic Monitoring." In Anaesthesia, Pain, Intensive Care and Emergency Medicine — A.P.I.C.E., 417–22. Milano: Springer Milan, 1996. http://dx.doi.org/10.1007/978-88-470-2203-4_37.

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Blasi, Annabel, Gianni Biancofiore, and David Green. "Haemodynamic Monitoring During Liver Transplant Surgery." In Anesthesia for Hepatico-Pancreatic-Biliary Surgery and Transplantation, 195–208. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-51331-3_10.

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Frasacco, Giulia, Mario Mezzapesa, Giovanni Carriero, Fernando Piscioneri, and Luigi Tritapepe. "ICU Echocardiography and Noninvasive Haemodynamic Monitoring." In Textbook of Echocardiography for Intensivists and Emergency Physicians, 431–38. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-99891-6_43.

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Conference papers on the topic "Haemodynamic monitoring"

1

Donadello, K., E. Pedrazzoli, G. Frison, V. Schweiger, M. Waldner, and E. Polati. "ESRA19-0658 Continuous non-invasive haemodynamic monitoring during caesarean section in hypertensive patients." In Abstracts of the European Society of Regional Anesthesia, September 11–14, 2019. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/rapm-2019-esraabs2019.477.

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Emarose, S., R. Asokan, and D. Valayaputtur. "Continuous monitoring of heart rate variability and haemodynamic stability of an automobile driver to prevent road accidents." In 2012 Third International Conference on Computing, Communication and Networking Technologies (ICCCNT 2012). IEEE, 2012. http://dx.doi.org/10.1109/icccnt.2012.6395936.

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3

Toschi, N., A. Canichella, F. Coniglione, E. Sabato, F. della Badia Giussi, M. Dauri, A. F. Sabato, et al. "Intraoperative haemodynamic monitoring: A pilot study on integrated data collection, processing and modelling for extracting vital signs and beyond." In 2010 3rd International Symposium on Applied Sciences in Biomedical and Communication Technologies (ISABEL 2010). IEEE, 2010. http://dx.doi.org/10.1109/isabel.2010.5702766.

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4

Bellucci, S., E. Cambau, B. Candalot, and J. P. Caen. "PHARMACOLOGICAL STUDIES OF PLATELET ANTIAGGREGANTS USING AN IN VITRO MODEL OF PRIMARY HEMOSTASIS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643434.

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We used a new device simulating in vitro primary haemostasis : more precisely the reactivity of blood to collagen and ADP. Thus an artificial vessel was created consisting of two main parts : a glass capillary (ID 140 um, length 16 mm, siliconized) simulating the haemodynamic resistance of an arteriole and an aperture (ID 150 um) reflecting the injured part of a cut arteriole. This aperture was performed in a cellulose acetate filter covered with collagen type I (3 mg/ml) to provide a defined surface for the adhesion of platelets and soaked with ADP in a concentration similar to that of injured endothelial cells (2 x 10-2 M). The mean - sd control values were 110 ± 24 s, 156 -± 40 ul (n = 25) and correlated well with in vivo bleeding time values (p< 0.01). We studied the effect on this test of classical antiaggregant drugs which act on primary hemostasis by different mechanisms of action. Acetylsalycilic acid (Egic laboratories) prolonged this test for concentrations above 10−5 M, ticlopidine (Millot-Solac laboratories) above 3 × 10−4 M, prostacyclin (Wellcome laboratories) above 5 Õ 10−9 M, the synthetic octapeptide LYS-PRO-GLY-GLU-PRO-GLY-PR0-LYS derived from type III collagen (gift from Y. Legrand) above 5 × 10−4 M. We evidenced a synergistic action between collagen octapeptide and ticlopidine. Thus this device permits the screening of new drugs for their effects on primary hemostasis and the study of ex vivo repeated measurements for the monitoring of antiaggregant therapy.
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Pathak, Soumi. "An acute cardiac complication of HIPEC." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685385.

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Recently, cytoreductive surgery (CRS) followed by hyperthermic intraperitoneal chemotherapy (HIPEC) has been described for both treatment and prevention of locoregional cancer of various origin. As this procedure involves large amount of blood and fluid loss during the CRS phase, and haemodynamic, metabolic, and coagulation changes during the HIPEC phase, thus thorough study and evaluation is needed to reduce the morbidity and mortality associated with this newer modality in treatment of cancer patients. We hereby describe a case report where a patient developed acute cardiac dysfunction in the immediate postoperative period following CRS with HIPEC. A 65 years old patient weighing 62 kg had undergone CRS with HIPEC for ovarian carcinoma. She had a blood loss of 1.5 L and ascetic fluid drainage of 1.5 L. Intraoperatively fluid was given according to stroke volume variation and two pack cell was transfused to maintain haemoglobin above 10 g. Two hours postoperatively she suddenly developed severe hypotension and an echocardiography done revealed a global left ventricular dysfunction with a 28% ejection fraction. She was intubated and put on inotropic support. Utrasound abdomen revealed fluids and features suggestive of intestinal perforation. So she was reopened on the 3rd postoperative day and primary closure of the intestinal perforation was done. Thereafter she became haemodynamically stable and we were able to extubate her on the fourth post operative day. Thus we conclude that goal directed fluid therapy with advanced monitoring, thorough evaluation, skeptical vigilance and preemtive thinking is required to deal with the challenges posed by CRS with HIPEC.
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