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Books on the topic 'Haematopoietic'

Author: Grafiati
Published: 4 June 2021
Last updated: 11 March 2023

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1

Stocking, Carol E. Autonomous growth of haematopoietic cells. Uxbridge: Brunel University, 1989.

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2

Bertheault-Cvitkovic, F. Breast cancer: The role of haematopoietic growth factors. Macclesfield: Gardiner-Caldwell Communications, 1991.

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3

Tura, S. Multiple myeloma: The role of haematopoietic growth factors. Macclesfield: Gardiner-Caldwell Communications Ltd., 1998.

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4

Kaye, S. B. Hodgkin's disease: The role of haematopoietic growth factors. Macclesfield: Gardiner-Caldwell Communications, 1994.

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5

Hoelzer, D. Myeloid malignancies: The role of haematopoietic growth factors. Macclesfield: Gardiner-Caldwell Communications, 1991.

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6

Crowther, D. Non-Hodgkin's lymphoma: The role of haematopoietic growth factors. Macclesfield: Gardiner-Caldwell Communications, 1990.

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7

Dexter, T. Michael. Haematopoietic growth factors: Review of biology and clinical potential. Macclesfield: Gardiner-Caldwell Communications, 1990.

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8

Neijt, J. P. Advanced ovarian cancer: The role of haematopoietic growth factors. Macclesfield: Gardiner-CaldwellCommunications, 1990.

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9

International Agency for Research on Cancer and World Health Organization, eds. WHO classification of tumours of haematopoietic and lymphoid tissues. 4th ed. Lyon, France: International Agency for Research on Cancer, 2008.

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10

Barge, A. Acute myeloid leukaemia: The role of haematopoietic growth factors. Macclesfield: Gardner-Caldwell Communications, 1998.

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11

Brown, Fraser David. The role and regulation of phospholipase D in haematopoietic cells. Birmingham: University of Birmingham, 1998.

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12

Sarkin, Jaffe Elaine, World Health Organization, and International Agency for Research on Cancer., eds. Pathology and genetics of tumours of haematopoietic and lymphoid tissues. Lyon: IARC Press, 2001.

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13

Clyde, Claire M. S. The effect of forskolin on the haematopoietic cell-line U937. [S.l: The Author], 1993.

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14

Pinkerton, C. R. Treatment strategies in paediatric cancer: The role of haematopoietic growth factors. Macclesfield: Gardiner-Caldwell Communications, 1991.

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15

International, Symposium on Cytokines and HIV Disease (1st 1995 Reims France). Haematopoietic growth factors and the treatment of HIV disease: A round table held at the Fist International Symposium on Cytokines and HIV Disease, Reims, France, 15 March 1995. London: Mediscript, 1995.

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16

A, Lange J. M., and WHO Collaborating Centre on AIDS., eds. Strategies for haematopoietic support in HIV disease: A roound table held under the auspices of the World Health Organzation Collaborating Centre on AIDS, Antwerp, Belgium, 6 March 1995. London: Mediscript, 1995.

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17

F, Grignani, Martelli M. F, and Mason D. Y, eds. Genotypic, phenotypic, and functional aspects of haematopoiesis. New York: Raven Press, 1987.

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18

International Symposium on Molecular Biology of Haematopoiesis (1st 1989 Innsbruck, Austria). Molecular biology of haematopoiesis: Proceedings of the International Symposium on Molecular Biology of Haematopoiesis, Innsbruck, Austria, 9-12 July 1989. Edited by Sachs Leo 1924-. Andover, Hampshire: Intercept, 1990.

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19

International Symposium on Molecular Biology of Haematopoiesis (8th 1993 Basel, Switzerland). Molecular biology of haematopoiesis: Proceedings of the 8th Symposium on Molecular Biology of Haematopoiesis held at theBasel Convention Center/New York Medical College, Basel, Switzerland, 9-13 July 1993. Edited by Abraham Nader G and New York Medical College. Andover, Hants, United Kingdom: Intercept, 1994.

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20

International, Symposium on Molecular Biology of Haematopoiesis (2nd 1991 Innsbruck Austria). Molecular biology of haematopoiesis: Proceedings of the International Conference on Molecular Biology of Haematopoiesis held at the University of Innsbruck/New York Medical College, Innsbruck, Austria, 14-18 July 1991. Andover, Hants, United Kingdom: Intercept, 1992.

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21

Provan, Drew, Trevor Baglin, Inderjeet Dokal, Johannes de Vos, and Hassan Al-Sader. Haematopoietic stem cell transplantation. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199683307.003.0009.

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Haemopoietic stem cell transplantation (SCT) - Indications for haemopoietic SCT - Allogeneic SCT - Autologous STC - Investigations for BMT/PBSCT - Pretransplant investigation of donors - Bone marrow harvesting - Peripheral blood stem cell mobilization and harvesting - Microbiological screening for stem cell cryopreservation - Stem cell transplant conditioning regimens - Infusion of cryopreserved stem cells - Infusion of fresh non-cryopreserved stem cells - Blood product support for SCT - Graft-versus-host disease (GvHD) prophylaxis - Acute GvHD - Chronic GvHD - Veno-occlusive disease (syn. sinusoidal obstruction syndrome) - Invasive fungal infections and antifungal therapy - CMV prophylaxis and treatment - Post-transplant vaccination programme and foreign travel - Longer term effect post-transplant - Treatment of relapse post-allogeneic SCT - Discharge and follow-up
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22

Batchelor, Tracy T., Oussama Abla, Zhong-ping Chen, Dennis C. Shrieve, and Samar Issa. Tumours of the haematopoietic system. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199651870.003.0013.

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‘Tumours of the haematopoietic system’ examines the epidemiology, the pathogenesis, and the clinical features of adult and childhood primary central nervous system lymphomas (PCNSLs), extranodal forms of non-Hodgkin lymphoma, as well as the histiocytoses included in the World Health Organization (WHO) classification of central nervous system (CNS) tumours. It reviews these features in the most common PCNSL, primary central nervous system diffuse large B-cell lymphoma, as well as the other rare histopathological PCNSL variants including lymphomatoid granulomatosis, T-cell lymphoma, anaplastic large T-cell lymphoma, natural killer/T-cell lymphoma, low-grade lymphoma, mucosa-associated lymphoid tissue (MALT) of the dura, and Hodgkin lymphoma. The chapter also discusses clinical and anatomical PCNSL variants including vitreoretinal lymphoma, leptomeningeal lymphoma, intramedullary spinal cord lymphoma, intravascular lymphoma, and PCNSL in the immunocompromised host. It also reviews the CNS presentations of Langerhans cell histiocytosis and the following non-Langerhans cell histiocytoses: Erdheim–Chester disease, Rosai–Dorfman disease, juvenile xanthogranuloma, and histiocytic sarcoma. It is written for specialists and non-specialists managing these various conditions.
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23

1957-, Dallman Margaret J., and Lamb Jonathan R, eds. Haematopoietic and lymphoid cell culture. Cambridge, U.K: Cambridge University Press, 2000.

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24

Polli, E. E. Haematopoietic Growth Factors: Biology and Clinical Use - Journal: ACTA Haematologica, Vol. 86, 1991 (Haematopoietic Growth Factors). S. Karger AG (Switzerland), 1992.

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25

Kaye, S. B. Hodgkin's disease: The role of haematopoietic growth factors. Gardiner-Caldwell Communications, 1990.

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26

Schultze, Wolfgang. High-Dose Therapy and Transplantation of Haematopoietic Stem Cells. 2nd ed. Blackwell Publishing Limited, 2002.

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27

(Editor), Wolfgang Schultze, and Bad Saarow (Editor), eds. High-Dose Therapy and Transplantation of Haematopoietic Stem Cells. Blackwell Publishers, 2000.

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28

WHO classification of tumours of haematopoietic and lymphoid tissues. 4th ed. 2017.

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29

WHO classification of tumours of haematopoietic and lymphoid tissues - 4. ed. International Agency for Research on Cancer (IARC), 2008.

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30

(Editor), Margaret J. Dallman, and Jonathan R. Lamb (Editor), eds. Haematopoietic and Lymphoid Cell Culture (Handbooks in Practical Animal Cell Biology). Cambridge University Press, 2000.

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31

(Editor), Margaret J. Dallman, and Jonathan R. Lamb (Editor), eds. Haematopoietic and Lymphoid Cell Culture (Handbooks in Practical Animal Cell Biology). Cambridge University Press, 2000.

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32

Jaffe, Elaine Sarkin. Pathology and Genetics: Tumours of Haematopoietic and Lymphoid Tissues (World Health Organization Classification of Tumours). Intl Agency for Research on Cancer, 2003.

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33

Bowker, Lesley K., James D. Price, Ku Shah, and Sarah C. Smith. Haematology. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198738381.003.0016.

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This chapter provides information on the ageing haematopoietic system, investigating anaemia in older people, diagnosis of iron deficiency anaemia, treatment of iron deficiency anaemia, macrocytic anaemia, anaemia of chronic disease, paraproteinaemias, multiple myeloma, myelodysplasia and myelodysplastic syndrome, and chronic lymphocytic leukaemia.
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34

Mohr, Ulrich. International Classifications of Rodent Tumours: Part 1: The Rat Fascicle No. 4: Haematopoietic System (DISCONTINUED (IARC Scient Pub)). IARC Scientific Publications, 1994.

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35

Collins, Graham, and Chris Bunch. Acute leukaemia. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0286.

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Acute leukaemias are rapidly progressive, clonal haematopoietic stem cell disorders resulting in the accumulation of immature blood cell precursors (known as blasts) in the bone marrow. There are two main types, defined by the presence of myeloid lineage or lymphoid markers on the blast cells: acute myeloid leukaemia and acute lymphoblastic leukaemia. This chapter addresses the causes, diagnosis, and management of the acute leukaemias.
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36

Collins, Graham, and Chris Bunch. Myeloproliferative disorders. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0291.

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Myeloproliferative disorders (also called myeloproliferative neoplasms) can be defined as clonal haematopoietic disorders resulting in excess production of one or more blood cell lineage. The four main conditions are primary polycythaemia, which is characterized by excess red-cell production; essential thrombocythaemia, which is characterized by excess platelet production; chronic myeloid leukaemia, which is characterized by excess granulocyte production; and myelofibrosis, which is characterized by excess megakaryocyte proliferation, which results in a reactive fibroblast proliferation causing marrow fibrosis and failure. This chapter addresses the causes, diagnosis, and management of these myeloproliferative disorders.
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37

Townsend, William M., and Emma C. Morris. ICU selection and outcome of patients with haematological malignancy. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0374.

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Patients with haematological malignancies require admission to the intensive care unit (ICU) due to the underlying disease, as a consequence of treatment with chemotherapy or after haematopoietic stem cell transplantation. With an increasing numbers of patients being diagnosed with these diseases and longer survival as treatments improve, the burden on ICU is anticipated to increase. There is compelling evidence that patients should not be denied admission to ICU based on the presence of a haematological malignancy. In this chapter the disease- and treatment-related reasons for ICU admission, outcome, and risk prediction scores for patients with haematological malignancies are discussed.
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38

Bunch, Chris. Chronic leukaemia. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0287.

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In the chronic leukaemias, leukaemogenesis occurs in two different cell types (and possibly even two different anatomical sites), leading to two very different forms of the disease: chronic myeloid leukaemia and chronic lymphocytic leukaemia. Chronic myeloid leukaemia is best thought of as a myeloproliferative disorder. It is a clonal disorder of the haematopoietic stem cell, leading to overproduction of the myeloid cells: neutrophils and their precursors, basophils and eosinophils. By contrast, chronic lymphocytic leukaemia can be viewed as a low-grade lymphoma. It is a clonal disorder of mature B-lymphocytes (possibly memory B-cells). This chapter reviews the causes, diagnosis, and management of these two forms of chronic leukaemia.
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39

Foster, Brogan, and Paul A. Brogan. Specialized therapeutic approaches. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198738756.003.0008.

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This chapter provides updated guidance on specialized therapeutic approaches relevant to paediatric rheumatology. Detailed overviews include: intra-articular injections including guidance on triamcinolone hexacetonide and dose for paediatric joint injection; current indications and recommended doses for biologic therapies, including updated summaries of regulatory approvals for the use of these treatments; an overview of medicines commonly used in paediatric rheumatology, including a commentary on paediatric pharmacokinetics and specific safety issues; dose calculation in paediatric practice; and practical advice when considering different medicine formulations in paediatric practice. An important feature of the second edition is a fully updated section on haematopoietic stem cell transplantation for autoimmune and autoinflammatory diseases, providing a comprehensive and up-to-date overview of the subject.
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40

Hodgkiss, Andrew. Further clinical issues. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198759911.003.0012.

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The clinical challenges arising when a person with a severe mental illness, such as schizophrenia or bipolar disorder, develops a cancer are surveyed. Delayed diagnosis and access to oncological treatment, factors contributing to reduced adherence, and the interruption of specialist community psychiatric care are discussed. Long-term psychotropic medication may complicate end-of-life care, and access to palliative care is usually limited for those in secure mental health inpatient units. The striking inverse relationship between neurodegenerative disorders (Alzheimer-type dementia) and proliferative disorders (cancers) is considered.Psychiatric aspects of haematopoietic stem cell transplantation (HSCT) are reviewed, including psychopathology arising from drugs used to prevent graft-versus-host disease and from infections complicating chronic immunosuppression. Cognitive impairment and suicide after HSCT are considered.
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41

Pleniceanu, Oren, and Benjamin Dekel. Kidney stem cells. Edited by Adrian Woolf. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0344.

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End-stage renal failure is a major cause of death with currently only dialysis and transplantation available as therapeutic options, each with its own limitations and drawbacks. To allow regenerative medicine-based kidney replacement therapies and due to the fact that neither haematopoietic stem cells nor mesenchymal stem cells, the most accessible human stem cells, can be used to derive genuine nephron progenitors, much attention has been given to finding adult renal stem cells. Several candidates for this have been described, but their true identity as stem or progenitor cells and their potential use in therapy has not yet been shown. However, the analysis of embryonic renal stem cells, specifically stem/progenitor cells that are induced into the nephrogenic pathway to form nephrons until the 34th week of gestation, has been much more conclusive.
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42

Koehler, Philipp, and Oliver A. Cornely. Fungal infections in haemato-oncology. Edited by Christopher C. Kibbler, Richard Barton, Neil A. R. Gow, Susan Howell, Donna M. MacCallum, and Rohini J. Manuel. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755388.003.0032.

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Invasive fungal infections on haemato-oncology wards present a major challenge. Patients at risk for invasive fungal infection usually have a compromised immune system due to bone marrow failure caused by underlying disease, prolonged neutropenia after intensive chemotherapy, or immunosuppression after haematopoietic stem cell transplantation to avoid graft-versus-host disease. Three major entities—invasive candidiasis, invasive aspergillosis, and mucormycosis—account for the majority of fungal infections. Here, we describe specific host and therapeutic factors predisposing to invasive fungal infection in the haemato-oncology setting. Clinical presentation is highly variable and dependent on the underlying pathogen, organ involvement, and site of infection. Diagnosis is mainly based on radiographic imaging combined with microbiological and histopathological work-up. Various prophylaxis and treatment strategies have been developed, and the evidence for these is discussed.
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43

Burton, Derek, and Margaret Burton. Excretion. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198785552.003.0008.

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Excretion is the removal of metabolic wastes such as ammonia, carbon dioxide, ions and water as well as toxic xenobiotics and metals. The process involves the gills, kidney, liver and rectal gland (elasmobranchs and coelacanth). In the liver, amino acids, haemoglobin, steroids and molecules resulting from human activities are transformed to excretable products. The rectal gland excretes ions, notably Na+ and Cl−. The kidney in teleosts has a distinction between an anterior head-kidney containing haematopoietic tissue and endocrine tissue and the posterior region with nephrons (kidney tubules). Fish nephrons generally have a Malphigian corpuscle with a glomerulus but the structure varies between fish taxa and some marine teleosts lack a glomerulus. Control systems for fish excretion are unclear but it is expected that various hormones influence excretory homeostasis.
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44

McCann, Shaun R. Leukaemia. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198717607.003.0007.

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The word leukaemia still is associated with foreboding and a fear of premature death. Steady advances have been made in the treatment of childhood leukaemia but, with notable exceptions, the same is not true in adults. The so-called genetic/molecular revolution has extended the understanding of the pathogenesis of many forms of leukaemia but, as yet, has rarely facilitated cure. With the introduction of tyrosine kinase inhibitor therapy, chronic myeloid leukaemia is the obvious exception but it still needs to be seen as to whether the cytogenetic/molecular revolution can provide cures for many elderly patients with leukaemia, as such patients respond poorly to chemotherapy. Haematopoietic stem cell transplantation, although toxic, expensive, and difficult, still provides a cure for many patients. In spite of all these advances, however, most adults with acute leukaemia or myelodysplastic syndrome are destined to die from their disease, and the causes of these fatal illnesses continue to elude researchers.
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45

Tyndall, Alan, and Jacob M. van Laar. Stem cell therapies. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0085.

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Since the start of the international project in 1997, over 1500 patients have received a haematopoietic stem cell transplant (HSCT), mostly autologous, as treatment for a severe autoimmune disease, with overall 85% 5-year survival and 43% progression-free survival. Around 30% of patients in all disease subgroups had a complete response, often durable despite full immune reconstitution. In many cases, e.g. systemic sclerosis, morphological improvement such as reduction of skin collagen and normalization of microvasculature was documented, beyond any predicted known effects of intense immunosuppression alone. It is hoped that the results of the three running large prospective randomized controlled trials will allow modification of the protocols to reduce the high transplant-related mortality which relates to regimen intensity, age of patient, and comorbidity. Multipotent mesenchymal stromal cells (MSC) have been recently tested in various autoimmune diseases, exploiting their immune modulating properties and apparent low acute toxicity. MSC display immune privilege in that the patient requires no immunosuppression prior to allogeneic MSC infusion. Despite encouraging small phase I/II studies, no positive data from randomized prospective studies are as yet available in the peer-reviewed literature.
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46

Tyndall, Alan, and Jacob M. van Laar. Stem cell therapies. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642489.003.0085_update_003.

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Since the start of the international project in 1997, over 2000 patients have received a haematopoietic stem cell transplant (HSCT), mostly autologous, as treatment for a severe autoimmune disease, with overall 85% 5-year survival and 43% progression-free survival. Around 30% of patients in all disease subgroups had a complete response, often durable despite full immune reconstitution. In many cases, e.g. systemic sclerosis, morphological improvement such as reduction of skin collagen and normalization of microvasculature was documented, beyond any predicted known effects of intense immunosuppression alone. It is hoped that the results of the three running large prospective randomized controlled trials will allow modification of the protocols to reduce the high transplant-related mortality which relates to regimen intensity, age of patient, and comorbidity. Multipotent mesenchymal stromal cells (MSC) have been recently tested in various autoimmune diseases, exploiting their immune modulating properties and apparent low acute toxicity. MSC display immune privilege in that the patient requires no immunosuppression prior to allogeneic MSC infusion. Despite encouraging many small phase I/II studies, only 2 prospective controlled trials which achieved their primary endpoints have been published.
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47

Dainiak, Nicholas. The Biology of Haematopoiesis (Progress in Clinical & Biological Research). John Wiley & Sons Inc, 1990.

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48

Fung, Iris Chin Wai. Cre-conditional expression of TEL-AML1: Haematopoiesis and leukaemia. 2004.

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49

Choon, Francis Chin Kuok, and Phua Dong Haur. Management of radiation poisoning. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0331.

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In radiation poisoning, a distinction between exposure and contamination should be made. Decontamination by removing clothes, washing of skin, and removal of debris can remove up to 90% of external contaminated radiation. Treatment of acute life-threatening injuries takes priority over treatment of radiation poisoning. Triage of severely exposed patients can give an indication of dose and severity of the radiation dose absorbed. Survival is related to dose absorbed. Identification of the radiation source should be made by the radiation characteristics to determine the shielding necessary for protection of hospital staff and the antidote required. Early gastric lavage and specific antidotes for ingested radiation poisoning should be used with caution. Death is mainly due to infection and haemorrhage. Acute radiation syndrome (ARS) is a manifestation of haematopoietic, gastrointestinal, cardiovascular, central nervous system, and cutaneous syndromes. Those receiving whole body doses of 1–5 Gy may recover easily with appropriate medical management; those with doses of 6–10 Gy may survive with intensive management; and those with doses of >10 Gy seldom survive. Treatment of ARS is supportive with the use of antibiotics, colony-stimulating factors, blood products, and stem cell transplants. Protection of the staff is by reducing time exposed, increasing distance from source and proper shielding. Psychological counselling should be available to patient or staff if required.
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50

Foster, Helen E., and Paul A. Brogan, eds. Paediatric Rheumatology. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198738756.001.0001.

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Paediatric Rheumatology, second edition is an indispensable resource for the identification and management of rheumatological diseases of children and young people. As well as covering common and rare musculoskeletal problems, there are also chapters on rheumatological emergencies designed for quick reference, and essential core clinical skills of relevance to the full spectrum of paediatric rheumatological disease. This second edition is fully updated, including state-of-the-art descriptions of new autoinflammatory diseases, advances in genetics in paediatric rheumatology, up-to-date guidance on the treatment of JIA, vasculitis, SLE, JDM, and other connective tissue diseases. Highlights of the second edition include alignment of management approaches for paediatric rheumatological diseases with recent evidence-based / consensus European guidelines; a description of North American treatment approaches to JIA; updated chapters on more specialist interventions including immunization in the immunosuppressed, and haematopoietic stem cell transplantation; and sections describing approaches to the management of rheumatological diseases in developing countries. Bone diseases are also described in detail including CRMO, skeletal dysplasias, and metabolic bone diseases. The second edition also includes a colour plate depicting rashes presenting in paediatric rheumatological conditions. This second edition is fully endorsed by the British Society for Paediatric and Adolescent Rheumatology (BSPAR), and many members of BSPAR have contributed most of the chapters. This second edition thus provides fully updated clinical guidelines and supporting information needed to successfully manage children and young people with rheumatological conditions.
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