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Books on the topic 'Haematopoiesis'

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Published: 4 June 2021
Last updated: 25 May 2024

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1

F, Grignani, Martelli M. F, and Mason D. Y, eds. Genotypic, phenotypic, and functional aspects of haematopoiesis. New York: Raven Press, 1987.

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2

International Symposium on Molecular Biology of Haematopoiesis (1st 1989 Innsbruck, Austria). Molecular biology of haematopoiesis: Proceedings of the International Symposium on Molecular Biology of Haematopoiesis, Innsbruck, Austria, 9-12 July 1989. Edited by Sachs Leo 1924-. Andover, Hampshire: Intercept, 1990.

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3

International Symposium on Molecular Biology of Haematopoiesis (8th 1993 Basel, Switzerland). Molecular biology of haematopoiesis: Proceedings of the 8th Symposium on Molecular Biology of Haematopoiesis held at theBasel Convention Center/New York Medical College, Basel, Switzerland, 9-13 July 1993. Edited by Abraham Nader G and New York Medical College. Andover, Hants, United Kingdom: Intercept, 1994.

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4

International, Symposium on Molecular Biology of Haematopoiesis (2nd 1991 Innsbruck Austria). Molecular biology of haematopoiesis: Proceedings of the International Conference on Molecular Biology of Haematopoiesis held at the University of Innsbruck/New York Medical College, Innsbruck, Austria, 14-18 July 1991. Andover, Hants, United Kingdom: Intercept, 1992.

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5

Stocking, Carol E. Autonomous growth of haematopoietic cells. Uxbridge: Brunel University, 1989.

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6

Bertheault-Cvitkovic, F. Breast cancer: The role of haematopoietic growth factors. Macclesfield: Gardiner-Caldwell Communications, 1991.

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7

Tura, S. Multiple myeloma: The role of haematopoietic growth factors. Macclesfield: Gardiner-Caldwell Communications Ltd., 1998.

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8

Kaye, S. B. Hodgkin's disease: The role of haematopoietic growth factors. Macclesfield: Gardiner-Caldwell Communications, 1994.

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9

Hoelzer, D. Myeloid malignancies: The role of haematopoietic growth factors. Macclesfield: Gardiner-Caldwell Communications, 1991.

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10

Crowther, D. Non-Hodgkin's lymphoma: The role of haematopoietic growth factors. Macclesfield: Gardiner-Caldwell Communications, 1990.

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11

Dexter, T. Michael. Haematopoietic growth factors: Review of biology and clinical potential. Macclesfield: Gardiner-Caldwell Communications, 1990.

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12

Barge, A. Acute myeloid leukaemia: The role of haematopoietic growth factors. Macclesfield: Gardner-Caldwell Communications, 1998.

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13

Neijt, J. P. Advanced ovarian cancer: The role of haematopoietic growth factors. Macclesfield: Gardiner-CaldwellCommunications, 1990.

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14

International Agency for Research on Cancer and World Health Organization, eds. WHO classification of tumours of haematopoietic and lymphoid tissues. 4th ed. Lyon, France: International Agency for Research on Cancer, 2008.

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15

Brown, Fraser David. The role and regulation of phospholipase D in haematopoietic cells. Birmingham: University of Birmingham, 1998.

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16

Clyde, Claire M. S. The effect of forskolin on the haematopoietic cell-line U937. [S.l: The Author], 1993.

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17

Sarkin, Jaffe Elaine, World Health Organization, and International Agency for Research on Cancer., eds. Pathology and genetics of tumours of haematopoietic and lymphoid tissues. Lyon: IARC Press, 2001.

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18

Pinkerton, C. R. Treatment strategies in paediatric cancer: The role of haematopoietic growth factors. Macclesfield: Gardiner-Caldwell Communications, 1991.

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19

International, Symposium on Cytokines and HIV Disease (1st 1995 Reims France). Haematopoietic growth factors and the treatment of HIV disease: A round table held at the Fist International Symposium on Cytokines and HIV Disease, Reims, France, 15 March 1995. London: Mediscript, 1995.

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20

A, Lange J. M., and WHO Collaborating Centre on AIDS., eds. Strategies for haematopoietic support in HIV disease: A roound table held under the auspices of the World Health Organzation Collaborating Centre on AIDS, Antwerp, Belgium, 6 March 1995. London: Mediscript, 1995.

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21

Dainiak, Nicholas. The Biology of Haematopoiesis (Progress in Clinical & Biological Research). John Wiley & Sons Inc, 1990.

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22

Fung, Iris Chin Wai. Cre-conditional expression of TEL-AML1: Haematopoiesis and leukaemia. 2004.

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23

Tyazhelova, V., and V. G. Tyazhelova. Haematopoiesis During Chronic Irradiation (Soviet Scientific Reviews Series, Section F). Routledge, 1997.

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24

Clark, Steven C., and David W. Golde. Haematopoiesis (UCLA Symposia on Molecular and Cellular Biology, New Series). John Wiley & Sons Inc, 1990.

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25

Molecular biology of haematopoiesis: Proceedings of the 8th symposium on molecular biology of haematopoiesis held at Basel convention center/New York medical college, Basel, Switzerland, 9-13 July 1993. Andover: Intercept, 1994.

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26

Cell Lineage Choice During Haematopoiesis: A Commemorative Issue in Honor of Professor Antonius Rolink. MDPI, 2018. http://dx.doi.org/10.3390/books978-3-03897-275-4.

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27

Campbell, Leslie Elspeth. The effects of chronic low level radiation, delivered at various dose rates, on haematopoiesis in mice. 1986.

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28

Lee, Christina Rebecca. Further investigation of the role of Fli-1 in haematopoiesis using a conditional transgenic mouse model. 2005.

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29

Abraham, Nader G. MOLECULAR BIOLOGY HAEMATOPOIES,. Intercept, 1993.

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30

Provan, Drew, Trevor Baglin, Inderjeet Dokal, Johannes de Vos, and Hassan Al-Sader. Haematopoietic stem cell transplantation. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199683307.003.0009.

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Haemopoietic stem cell transplantation (SCT) - Indications for haemopoietic SCT - Allogeneic SCT - Autologous STC - Investigations for BMT/PBSCT - Pretransplant investigation of donors - Bone marrow harvesting - Peripheral blood stem cell mobilization and harvesting - Microbiological screening for stem cell cryopreservation - Stem cell transplant conditioning regimens - Infusion of cryopreserved stem cells - Infusion of fresh non-cryopreserved stem cells - Blood product support for SCT - Graft-versus-host disease (GvHD) prophylaxis - Acute GvHD - Chronic GvHD - Veno-occlusive disease (syn. sinusoidal obstruction syndrome) - Invasive fungal infections and antifungal therapy - CMV prophylaxis and treatment - Post-transplant vaccination programme and foreign travel - Longer term effect post-transplant - Treatment of relapse post-allogeneic SCT - Discharge and follow-up
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31

Batchelor, Tracy T., Oussama Abla, Zhong-ping Chen, Dennis C. Shrieve, and Samar Issa. Tumours of the haematopoietic system. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199651870.003.0013.

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‘Tumours of the haematopoietic system’ examines the epidemiology, the pathogenesis, and the clinical features of adult and childhood primary central nervous system lymphomas (PCNSLs), extranodal forms of non-Hodgkin lymphoma, as well as the histiocytoses included in the World Health Organization (WHO) classification of central nervous system (CNS) tumours. It reviews these features in the most common PCNSL, primary central nervous system diffuse large B-cell lymphoma, as well as the other rare histopathological PCNSL variants including lymphomatoid granulomatosis, T-cell lymphoma, anaplastic large T-cell lymphoma, natural killer/T-cell lymphoma, low-grade lymphoma, mucosa-associated lymphoid tissue (MALT) of the dura, and Hodgkin lymphoma. The chapter also discusses clinical and anatomical PCNSL variants including vitreoretinal lymphoma, leptomeningeal lymphoma, intramedullary spinal cord lymphoma, intravascular lymphoma, and PCNSL in the immunocompromised host. It also reviews the CNS presentations of Langerhans cell histiocytosis and the following non-Langerhans cell histiocytoses: Erdheim–Chester disease, Rosai–Dorfman disease, juvenile xanthogranuloma, and histiocytic sarcoma. It is written for specialists and non-specialists managing these various conditions.
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32

1957-, Dallman Margaret J., and Lamb Jonathan R, eds. Haematopoietic and lymphoid cell culture. Cambridge, U.K: Cambridge University Press, 2000.

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33

Polli, E. E. Haematopoietic Growth Factors: Biology and Clinical Use - Journal: ACTA Haematologica, Vol. 86, 1991 (Haematopoietic Growth Factors). S. Karger AG (Switzerland), 1992.

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34

Kaye, S. B. Hodgkin's disease: The role of haematopoietic growth factors. Gardiner-Caldwell Communications, 1990.

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35

Albert, Tyler J., and Erik R. Swenson. The blood cells and blood count. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0265.

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Blood is a dynamic fluid consisting of cellular and plasma components undergoing constant regeneration and recycling. Like most physiological systems, the concentrations of these components are tightly regulated within narrow limits under normal conditions. In the critically-ill population, however, haematological abnormalities frequently occur and are largely due to non-haematological single- or multiple-organ pathology. Haematopoiesis originates from the pluripotent stem cell, which undergoes replication, proliferation, and differentiation, giving rise to cells of the erythroid, myeloid, and lymphoid series, as well as megakaryocytes, the precursors to platelets. The haemostatic system is responsible for maintaining blood fluidity and, at the same time, prevents blood loss by initiating rapid, localized, and appropriate blood clotting at sites of vascular damage. This system is complex, comprising both cellular and plasma elements, i.e. platelets, coagulation and fibrinolytic cascades, the natural intrinsic and extrinsic pathways of anticoagulation, and the vascular endothelium. A rapid, reliable, and inexpensive method of examining haematological disorders is the peripheral blood smear, which allows practitioners to assess the functional status of the bone marrow during cytopenic states. Red blood cells, which are primarily concerned with oxygen and carbon dioxide transport, have a normal lifespan of only 120 days and require constant erythropoiesis. White blood cells represent a summation of several circulating cell types, each deriving from the hematopoietic stem cell, together forming the critical components of both the innate and adaptive immune systems. Platelets are integral to haemostasis, and also aid our inflammatory and immune responses, help maintain vascular integrity, and contribute to wound healing.
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36

Schultze, Wolfgang. High-Dose Therapy and Transplantation of Haematopoietic Stem Cells. 2nd ed. Blackwell Publishing Limited, 2002.

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37

(Editor), Wolfgang Schultze, and Bad Saarow (Editor), eds. High-Dose Therapy and Transplantation of Haematopoietic Stem Cells. Blackwell Publishers, 2000.

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38

WHO classification of tumours of haematopoietic and lymphoid tissues. 4th ed. 2017.

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39

(Editor), Margaret J. Dallman, and Jonathan R. Lamb (Editor), eds. Haematopoietic and Lymphoid Cell Culture (Handbooks in Practical Animal Cell Biology). Cambridge University Press, 2000.

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40

(Editor), Margaret J. Dallman, and Jonathan R. Lamb (Editor), eds. Haematopoietic and Lymphoid Cell Culture (Handbooks in Practical Animal Cell Biology). Cambridge University Press, 2000.

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41

WHO classification of tumours of haematopoietic and lymphoid tissues - 4. ed. International Agency for Research on Cancer (IARC), 2008.

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42

Bowker, Lesley K., James D. Price, Ku Shah, and Sarah C. Smith. Haematology. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198738381.003.0016.

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This chapter provides information on the ageing haematopoietic system, investigating anaemia in older people, diagnosis of iron deficiency anaemia, treatment of iron deficiency anaemia, macrocytic anaemia, anaemia of chronic disease, paraproteinaemias, multiple myeloma, myelodysplasia and myelodysplastic syndrome, and chronic lymphocytic leukaemia.
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43

Jaffe, Elaine Sarkin. Pathology and Genetics: Tumours of Haematopoietic and Lymphoid Tissues (World Health Organization Classification of Tumours). Intl Agency for Research on Cancer, 2003.

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44

Collins, Graham, and Chris Bunch. Acute leukaemia. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0286.

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Acute leukaemias are rapidly progressive, clonal haematopoietic stem cell disorders resulting in the accumulation of immature blood cell precursors (known as blasts) in the bone marrow. There are two main types, defined by the presence of myeloid lineage or lymphoid markers on the blast cells: acute myeloid leukaemia and acute lymphoblastic leukaemia. This chapter addresses the causes, diagnosis, and management of the acute leukaemias.
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45

Mohr, Ulrich. International Classifications of Rodent Tumours: Part 1: The Rat Fascicle No. 4: Haematopoietic System (DISCONTINUED (IARC Scient Pub)). IARC Scientific Publications, 1994.

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46

Collins, Graham, and Chris Bunch. Myeloproliferative disorders. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0291.

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Myeloproliferative disorders (also called myeloproliferative neoplasms) can be defined as clonal haematopoietic disorders resulting in excess production of one or more blood cell lineage. The four main conditions are primary polycythaemia, which is characterized by excess red-cell production; essential thrombocythaemia, which is characterized by excess platelet production; chronic myeloid leukaemia, which is characterized by excess granulocyte production; and myelofibrosis, which is characterized by excess megakaryocyte proliferation, which results in a reactive fibroblast proliferation causing marrow fibrosis and failure. This chapter addresses the causes, diagnosis, and management of these myeloproliferative disorders.
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47

Townsend, William M., and Emma C. Morris. ICU selection and outcome of patients with haematological malignancy. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0374.

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Patients with haematological malignancies require admission to the intensive care unit (ICU) due to the underlying disease, as a consequence of treatment with chemotherapy or after haematopoietic stem cell transplantation. With an increasing numbers of patients being diagnosed with these diseases and longer survival as treatments improve, the burden on ICU is anticipated to increase. There is compelling evidence that patients should not be denied admission to ICU based on the presence of a haematological malignancy. In this chapter the disease- and treatment-related reasons for ICU admission, outcome, and risk prediction scores for patients with haematological malignancies are discussed.
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48

Bunch, Chris. Chronic leukaemia. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0287.

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In the chronic leukaemias, leukaemogenesis occurs in two different cell types (and possibly even two different anatomical sites), leading to two very different forms of the disease: chronic myeloid leukaemia and chronic lymphocytic leukaemia. Chronic myeloid leukaemia is best thought of as a myeloproliferative disorder. It is a clonal disorder of the haematopoietic stem cell, leading to overproduction of the myeloid cells: neutrophils and their precursors, basophils and eosinophils. By contrast, chronic lymphocytic leukaemia can be viewed as a low-grade lymphoma. It is a clonal disorder of mature B-lymphocytes (possibly memory B-cells). This chapter reviews the causes, diagnosis, and management of these two forms of chronic leukaemia.
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49

Foster, Brogan, and Paul A. Brogan. Specialized therapeutic approaches. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198738756.003.0008.

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This chapter provides updated guidance on specialized therapeutic approaches relevant to paediatric rheumatology. Detailed overviews include: intra-articular injections including guidance on triamcinolone hexacetonide and dose for paediatric joint injection; current indications and recommended doses for biologic therapies, including updated summaries of regulatory approvals for the use of these treatments; an overview of medicines commonly used in paediatric rheumatology, including a commentary on paediatric pharmacokinetics and specific safety issues; dose calculation in paediatric practice; and practical advice when considering different medicine formulations in paediatric practice. An important feature of the second edition is a fully updated section on haematopoietic stem cell transplantation for autoimmune and autoinflammatory diseases, providing a comprehensive and up-to-date overview of the subject.
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50

Hodgkiss, Andrew. Further clinical issues. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198759911.003.0012.

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The clinical challenges arising when a person with a severe mental illness, such as schizophrenia or bipolar disorder, develops a cancer are surveyed. Delayed diagnosis and access to oncological treatment, factors contributing to reduced adherence, and the interruption of specialist community psychiatric care are discussed. Long-term psychotropic medication may complicate end-of-life care, and access to palliative care is usually limited for those in secure mental health inpatient units. The striking inverse relationship between neurodegenerative disorders (Alzheimer-type dementia) and proliferative disorders (cancers) is considered.Psychiatric aspects of haematopoietic stem cell transplantation (HSCT) are reviewed, including psychopathology arising from drugs used to prevent graft-versus-host disease and from infections complicating chronic immunosuppression. Cognitive impairment and suicide after HSCT are considered.
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