Relevant bibliographies by topics / Haematopoiesis

Academic literature on the topic 'Haematopoiesis'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 May 2024

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Journal articles on the topic "Haematopoiesis"

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Štefková, Kateřina, Markéta Hanáčková, Jan Kučera, Katarzyna Anna Radaszkiewicz, Barbora Ambrůžová, Lukáš Kubala, and Jiří Pacherník. "MAPK p38alpha Kinase Influences Haematopoiesis in Embryonic Stem Cells." Stem Cells International 2019 (June 2, 2019): 1–16. http://dx.doi.org/10.1155/2019/5128135.

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The activation of p38alpha kinase mediates cell response to various extracellular factors including many interleukins and growth factors important for haematopoiesis. The role of p38alpha kinase was previously analysed in particular haematopoietic cells. In this study and for the first time, the role of p38alpha kinase in haematopoiesis was studied using a model of continuous haematopoietic development in pluripotent embryonic stem cellsin vitro. The expression of transcripts associated with haematopoiesis and the potential for the formation of specific haematopoietic cell colonies were compared between wild-type and mutant p38alpha gene-depleted cells. The absence of p38alpha kinase led to the inhibition of hemangioblast formation during the first step of haematopoiesis. Later, during differentiation, due to the lack of p38alpha kinase, erythrocyte maturation was impaired. Mutant p38α−/−cells also exhibited decreased potential with respect to the expansion of granulocyte colony-forming units. This effect was reversed in the absence of erythropoietin as shown by colony-forming unit assay in media for colony-forming unit granulocytes/macrophages. p38alpha kinase thus plays an important role in the differentiation of common myeloid precursor cells into granulocyte lineages.
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Basden, K., DW Cooper, and EM Deane. "Development of the blood-forming tissues of the tammar wallaby Macropus eugenii." Reproduction, Fertility and Development 8, no. 6 (1996): 989. http://dx.doi.org/10.1071/rd9960989.

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The development of the haematopoietic tissues of the tammar wallaby Macropus eugenii follows a similar pattern to that observed in eutherian and other metatherian mammals. At birth, the liver appears to be the only site of haematopoiesis with significant numbers of neutrophils and stem cells present in the circulation. By Day 3, the spleen shows limited haematopoietic activity and by Day 12 contains areas of erythroid and myeloid cells. At two weeks after birth, the haematopoetic activity in the liver declines and small areas of haematopoiesis are apparent in the bone marrow. By the end of the first month, the bone marrow appears to be the major site of haematopoiesis.
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GUILPIN, V. O., L. NOSBISCH, R. G. TITUS, and C. J. SWARDSON-OLVER. "Infection with Leishmania major stimulates haematopoiesis in susceptible BALB/c mice and suppresses haematopoiesis in resistant CBA mice." Parasitology 126, no. 3 (March 2003): 187–94. http://dx.doi.org/10.1017/s0031182002002779.

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Cytokine responses to Leishmania infection begin very early in infection, and differ between susceptible and resistant mice. Susceptibility to chronic Leishmania infection has been associated with increased haematopoiesis. To analyse the effect that acute infection with L. major has on bone-marrow haematopoiesis in susceptible (BALB/c) and resistant (CBA) mice, we enumerated erythroid progenitors and granulocyte-monocyte progenitors 3 days after infection. We found that haematopoiesis was stimulated in BALB/c mice infected with L. major, while haematopoiesis was inhibited in CBA mice. We found that this effect could be partially explained by cytokine production: interleukin-4 was involved in stimulation of BALB/c haematopoiesis and tumour necrosis factor-α was involved in inhibition of CBA haematopoiesis. Our conclusions are that haematopoietic changes occur shortly after L. major infection, and may be related to disease outcome.
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Oliveira, Dalila Cunha, Amanda Nogueira-Pedro, Ed Wilson Santos, Araceli Hastreiter, Graziela Batista Silva, Primavera Borelli, and Ricardo Ambrósio Fock. "A review of select minerals influencing the haematopoietic process." Nutrition Research Reviews 31, no. 2 (July 9, 2018): 267–80. http://dx.doi.org/10.1017/s0954422418000112.

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AbstractMicronutrients are indispensable for adequate metabolism, such as biochemical function and cell production. The production of blood cells is named haematopoiesis and this process is highly consuming due to the rapid turnover of the haematopoietic system and consequent demand for nutrients. It is well established that micronutrients are relevant to blood cell production, although some of the mechanisms of how micronutrients modulate haematopoiesis remain unknown. The aim of the present review is to summarise the effect of Fe, Mn, Ca, Mg, Na, K, Co, iodine, P, Se, Cu, Li and Zn on haematopoiesis. This review deals specifically with the physiological requirements of selected micronutrients to haematopoiesis, showing various studies related to the physiological requirements, deficiency or excess of these minerals on haematopoiesis. The literature selected includes studies in animal models and human subjects. In circumstances where these minerals have not been studied for a given condition, no information was used. All the selected minerals have an important role in haematopoiesis by influencing the quality and quantity of blood cell production. In addition, it is highly recommended that the established nutrition recommendations for these minerals be followed, because cases of excess or deficient mineral intake can affect the haematopoiesis process.
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Haznedaroglu, Ibrahim C., and Yavuz Beyazit. "Local bone marrow renin–angiotensin system in primitive, definitive and neoplastic haematopoiesis." Clinical Science 124, no. 5 (November 12, 2012): 307–23. http://dx.doi.org/10.1042/cs20120300.

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The locally active ligand peptides, mediators, receptors and signalling pathways of the haematopoietic BM (bone marrow) autocrine/paracrine RAS (renin–angiotensin system) affect the essential steps of definitive blood cell production. Haematopoiesis, erythropoiesis, myelopoiesis, formation of monocytic and lymphocytic lineages, thrombopoiesis and other stromal cellular elements are regulated by the local BM RAS. The local BM RAS is present and active even in primitive embryonic haematopoiesis. ACE (angiotensin-converting enzyme) is expressed on the surface of the first endothelial and haematopoietic cells, forming the marrow cavity in the embryo. ACE marks early haematopoietic precursor cells and long-term blood-forming CD34+ BM cells. The local autocrine tissue BM RAS may also be active in neoplastic haematopoiesis. Critical RAS mediators such as renin, ACE, AngII (angiotensin II) and angiotensinogen have been identified in leukaemic blast cells. The local tissue RAS influences tumour growth and metastases in an autocrine and paracrine fashion via the modulation of numerous carcinogenic events, such as angiogenesis, apoptosis, cellular proliferation, immune responses, cell signalling and extracellular matrix formation. The aim of the present review is to outline the known functions of the local BM RAS within the context of primitive, definitive and neoplastic haematopoiesis. Targeting the actions of local RAS molecules could represent a valuable therapeutic option for the management of neoplastic disorders.
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Kondera, Elżbieta. "Haematopoiesis and haematopoietic organs in fish." Roczniki Naukowe Polskiego Towarzystwa Zootechnicznego 15, no. 1 (March 31, 2019): 9–16. http://dx.doi.org/10.5604/01.3001.0013.4535.

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Haematopoiesis is a complex process in which haematopoietic stem cells, the most immature elements of the haematopoietic hierarchy, proliferate and differentiate into various classes of haematopoietic progenitor cells. These progenitor cells have been shown to be able to differentiate into mature blood cells: erythrocytes, lymphocytes, thrombocytes, granulocytes, and monocytes. The pronephros, or head kidney, is a basic organ forming the blood elements, and is also a reservoir of blood cells. Basic haematopoietic structures and mechanisms in fish are similar to those functioning in other vertebrates, and all haematopoietic cell types are very similar to those of mammals.
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Mak, Ka Sin, Alister P. W. Funnell, Richard C. M. Pearson, and Merlin Crossley. "PU.1 and Haematopoietic Cell Fate: Dosage Matters." International Journal of Cell Biology 2011 (2011): 1–6. http://dx.doi.org/10.1155/2011/808524.

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The ETS family transcription factor PU.1 is a key regulator of haematopoietic differentiation. Its expression is dynamically controlled throughout haematopoiesis in order to direct appropriate lineage specification. Elucidating the biological role of PU.1 has proved challenging. This paper will discuss how a range of experiments in cell lines and mutant and transgenic mouse models have enhanced our knowledge of the mechanisms by which PU.1 drives lineage-specific differentiation during haematopoiesis.
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Dickson, M. C., J. S. Martin, F. M. Cousins, A. B. Kulkarni, S. Karlsson, and R. J. Akhurst. "Defective haematopoiesis and vasculogenesis in transforming growth factor-beta 1 knock out mice." Development 121, no. 6 (June 1, 1995): 1845–54. http://dx.doi.org/10.1242/dev.121.6.1845.

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Transforming growth factor beta 1 (TGF beta 1) is shown here to be required for yolk sac haematopoiesis and endothelial differentiation. Mice with a targeted mutation in the TGF beta 1 gene were examined to determine the cause of prenatal lethality, which occurs in 50% of homozygous TGF beta 1 null (TGF beta 1−/−) conceptions. 50% of TGF beta 1−/− and 25% of TGF beta 1-+-) conceptions. 50% of TGF beta 1−/− and 25% of TGF beta 1+/− conceptuses were found to die at around 10.5 dpc. The primary defects were restricted to extraembryonic tissues, namely the yolk sac vasculature and haematopoietic system. The embryos per se showed developmental retardation, oedema and necrosis, which were probably secondary to the extraembryonic lesions. The defect in vasculogenesis appeared to affect endothelial differentiation, rather than the initial appearance and outgrowth of endothelial cells. Initial differentiation of yolk sac mesoderm to endothelial cells occurred, but defective differentiation resulted in inadequate capillary tube formation, and weak vessels with reduced cellular adhesiveness. Defective haematopoiesis resulted in a reduced erythroid cell number within the yolk sac. Defective yolk sac vasculogenesis and haematopoiesis were present either together, or in isolation of each other. The phenotypes are consistent with the observation of abundant TGF beta 1 gene expression in both endothelial and haematopoietic precursors. The data indicate that the primary effect of loss of TGF beta 1 function in vivo is not increased haematopoietic or endothelial cell proliferation, which might have been expected by deletion of a negative growth regulator, but defective haematopoiesis and endothelial differentiation.
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Keenan, Christine R. "Heterochromatin and Polycomb as regulators of haematopoiesis." Biochemical Society Transactions 49, no. 2 (April 30, 2021): 805–14. http://dx.doi.org/10.1042/bst20200737.

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Haematopoiesis is the process by which multipotent haematopoietic stem cells are transformed into each and every type of terminally differentiated blood cell. Epigenetic silencing is critical for this process by regulating the transcription of cell-cycle genes critical for self-renewal and differentiation, as well as restricting alternative fate genes to allow lineage commitment and appropriate differentiation. There are two distinct forms of transcriptionally repressed chromatin: H3K9me3-marked heterochromatin and H3K27me3/H2AK119ub1-marked Polycomb (often referred to as facultative heterochromatin). This review will discuss the role of these distinct epigenetic silencing mechanisms in regulating normal haematopoiesis, how these contribute to age-related haematopoietic dysfunction, and the rationale for therapeutic targeting of these pathways in the treatment of haematological malignancies.
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De, Ranita, Kulkarni Uday Prakash, and Eunice S. Edison. "Complex Interactions in Regulation of Haematopoiesis—An Unexplored Iron Mine." Genes 12, no. 8 (August 20, 2021): 1270. http://dx.doi.org/10.3390/genes12081270.

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Iron is one of the most abundant metals on earth and is vital for the growth and survival of life forms. It is crucial for the functioning of plants and animals as it is an integral component of the photosynthetic apparatus and innumerable proteins and enzymes. It plays a pivotal role in haematopoiesis and affects the development and differentiation of different haematopoietic lineages, apart from its obvious necessity in erythropoiesis. A large amount of iron stores in humans is diverted towards the latter process, as iron is an indispensable component of haemoglobin. This review summarises the important players of iron metabolism and homeostasis that have been discovered in recent years and highlights the overall significance of iron in haematopoiesis. Its role in maintenance of haematopoietic stem cells, influence on differentiation of varied haematopoietic lineages and consequences of iron deficiency/overloading on development and maturation of different groups of haematopoietic cells have been discussed.
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Dissertations / Theses on the topic "Haematopoiesis"

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Robinson, Simon N. "Proliferation regulation of haematopoietic stem cells in normal and leukaemic haematopoiesis." Thesis, University of St Andrews, 1992. http://hdl.handle.net/10023/14965.

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The cellular integrity of the blood is maintained by the cellular output of the haematopoietic stem cell population which produces the specialized precursors and differentiated cells which constitute the blood. The investigation of haematopoietic stem cell behaviour and regulation has been hampered by both the difficulty in their identification and the development of relevant assay systems. The purpose of this investigation was to study the behaviour and regulation of the haematopoietic stem cell population in normal and leukaemic haematopoiesis using an in vitro assay of a primitive haematopoietic precursor. The use of a combination of haematopoietic colony-stimulating factors [interleukin 3 (IL3)/multi-CSF and macrophage colony-stimulating factor (M-CSF/CSF-1)] in semi-solid agar culture of murine haematopoietic tissue, stimulated the proliferation of a haematopoietic colony-forming cell, defined as the "HPP-CFCIL3+CSF-1" population, which was characterized by a high proliferative potential, a multipotency and behavioural and regulatory properties consistent with its being a primitive haematopoietic precursor and possibly a component of the haematopoietic stem cell population. The proportion of the in vitro HPP-CFCIL3+csf-1 population in S-phase in normal murine marrow, was determined to be relatively low at approximately 10%, increasing to approximately 40% in sublethally X-irradiated, regenerating murine marrow and the respective presence of the haematopoietic stem cell proliferation inhibitor and stimulator was demonstrable by the induction of appropriate kinetic changes in the in vitro HPP-CFCIL3+CSF-1 population. In leukaemic haematopoiesis, leukaemic proliferation often occurs at the expense of apparently suppressed normal haematopoiesis. In vitro HPP-CFCIL3+CSF-1 assay of the haematopoietic stem cell proliferation regulators in a number of murine, myeloid leukaemic cell lines, failed to demonstrate either increased levels of the haematopoietic stem cell proliferation inhibitor, or evidence of a direct-acting, leukaemia- associated proliferation inhibitor, however, evidence of a leukaemia- associated impairment of inhibitor and stimulator production was observed and this may be a possible mechanism by which the leukaemic population develops a proliferative advantage over normal haematopoietic tissue. The identification of a possible mechanism of leukaemic progression and suppression of normal haematopoiesis may subsequently allow the development of potentially more effective disease treatment and management regimes. The endogenous haemoregulatory tetrapeptide: Acetyl-N-Ser- Asp-Lys-Pro [AcSDKP, Mr=487 amu] is reported to prevent the G0-G1 transition of haematopoietic stem cells into S-phase. The mechanism of action of AcSDKP and a number of related peptides, was investigated in relation to the stem cell proliferation stimulator and inhibitor. AcSDKP demonstrated no direct haemoregulatory role against the in vitro HPP-CFCIL3+CSF-1 population, which is consistent with reports that AcSDKP is not active against cells already in late G1, or S-phase, rather it appeared to act indirectly by impairing the capacity of the haematopoietic stem cell proliferation stimulator to increase the proportion of the in vitro HPP-CFCIL3+CSF-1 population in S-phase. An apparent impairment of stimulator action may explain the reported AcSDKP-associated 'block' of haematopoietic stem cell recruitment. A putative endogenous AcSDKP precursor and synthetic and degradative enzyme systems have been reported and the possible physiopathological role of AcSDKP in a number of myeloproliferative disorders has been implicated. The potential application of AcSDKP as a 'haemoprotective' agent administered prior to the use of S-phase- specific chemotherapy may be of clinical significance. The in vitro HPP-CFCIL3+CSF-1 assay of a primitive haematopoietic precursor cell population, which may be a component of the haematopoietic stem cell population, should play a significant role in the investigation of haematopoietic stem cell behaviour and regulation in both normal and aberrant haematopoiesis. With the characterization of the mechanism(s) of action of the haematopoietic stem cell proliferation inhibitor and stimulator and the haemoregulatory tetrapeptide AcSDKP, the manipulation of the haematopoietic system to clinical advantage can be envisaged, while the identification of the aberrant regulatory mechanism(s) in haematopoietic dysfunction may allow, the development of more effective disease treatment and management regimes.
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Bureau, Emilie Aurelie. "Investigation of the role of haematopoietic cell kinase in normal and leukaemic haematopoiesis." Thesis, University College London (University of London), 2008. http://discovery.ucl.ac.uk/1444125/.

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Acute myeloid leukaemia (AML) is characterised by an accumulation of immature blasts that fail to fully differentiate. Leukaemia is organised as a hierarchy, which is maintained by leukaemic stem cells (LSC). To identify molecular differences between normal haematopoietic stem cells (HSC) and LSC, we performed microarray analysis and found that haematopoietic cell kinase (HCK) is overexpressed in LSC. Thus, by knocking-down HCK in leukaemic cells or overexpressing it in stem cell enriched fractions, we should be able to evaluate its role in leukaemogenesis. Since LSC are difficult to culture in vitro, we started to validate HCK silencing in leukaemic cell lines, Mono-mac-6 (MM6), U937 and Fujioka/P31, which highly express HCK. In all cell lines studied, no more than 50% silencing could be achieved, even when a short-hairpin anti-HCK was cloned into a lentiviral backbone to follow the long-term effect of HCK silencing. Decrease in kinase activity was confirmed using kinase assay and phospho-specific antibody recognising the activated HCK kinase. We show that HCK silencing does not affect the cell cycle, proliferation, differentiation or apoptosis of the cell lines. However, using methylcellulose assay, we observe a significant change in MM6 colony morphology caused by a decrease in their migration properties. Moreover, using phospho-flow cytometry, G-CSF and GM-CSF signal transduction towards STAT5 could be proven to occur via HCK in MM6, but not in Fujioka/P31 or U937 cells. HSC enriched umbilical cord blood cells were also transduced with a lentivirus encoding p59HCK. Overexpression of p59HCK in these cells led to their enhanced erythroid differentiation at the expense of myeloid differentiation underlined by the activation of c-Raf, ERK and STATS. Overall, this study can be used as a preliminary set up for further investigation of the role of HCK in normal human stem cells and in primary AML samples in vivo.
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Lau, C. I. "Hedgehog signalling in haematopoiesis." Thesis, University College London (University of London), 2014. http://discovery.ucl.ac.uk/1428443/.

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The Hedgehog (Hh) family proteins and their signalling pathway are key mediators of, and important in, many mammalian developmental processes. Malfunction of the Hh signalling pathway contributes to developmental disorders and birth defects. This project aims to investigate the role of the Hh signalling pathway in murine haematopoiesis. In our study, we found that Dhh plays a negative regulatory role in normal erythropoiesis and under stress conditions. However, it is not required for regulating erythropoiesis in the fetal liver during embryo development. In contrast, analysis of conditional deletion of Smo in haematopoietic cells revealed that Smo controls early haematopoietic differentiation in the fetal liver but is dispensable for regulating haematopoiesis in adult bone marrow and spleen. Furthermore, pervious studies have demonstrated that Hh signalling is involved in T-cell development throughout maturation. We tested the hypothesis that Foxa2, a downstream target gene of Hh during pre-TCR signalling, is also required for late T-cell development and activation. Analysis of mice conditionally Foxa2-deficient in mature T-cells revealed that Foxa2 is important in the process of maturation in late thymocyte development. In addition, Foxa2 is also involved in regulation of T-cell activation, and the differentiation of T helper cells. Gene expression experiments confirmed that Foxa2 is also a Hh target gene in the thymus. Taken together, our findings revealed that the Hh signalling pathway and its target genes play critical roles in haematopoiesis during embryogenesis and in adult mice.
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Warren, Alan John. "The role of Rbtn2 in haematopoiesis." Thesis, University of Cambridge, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.339542.

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Lennon, Joy Elizabeth. "Studies of haematopoiesis in the mouse." Thesis, University of Edinburgh, 1985. http://hdl.handle.net/1842/12402.

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Guo, Yanping. "The mechanism of Nov (CCN3) function in haematopoiesis." Thesis, University of Oxford, 2012. http://ora.ox.ac.uk/objects/uuid:5785f3b9-3206-4bb4-b486-d90cded680f8.

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Haematopoietic stem cells (HSC) are strictly regulated by intrinsic regulators and extrinsic signals from the microenvironment. Nov (CCN3), a matricellular protein of the CCN family, has been reported as a suppressor gene in solid tumours and chronic myeloid leukaemia (CML). Recent study identified Nov as a positive regulator in human cord blood CD34+ stem cells. However, the functions of Nov in haematopoiesis and adult HSC remain largely unknown.
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Lee-Prudhoe, J. E. "The genomic structure and regulation of murine CD33 : a marker of erythromyeloid commitment." Thesis, University of Oxford, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.249473.

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Thomson, Andrew M. "Globin gene regulation during erythroid differentiation." Thesis, University of Oxford, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.339104.

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Bailie, Karen Elizabeth Margaret. "G-CSF and GM-CSF : effects on neonatal neutrophils." Thesis, Queen's University Belfast, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.482043.

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Weston, Victoria Jane. "A study of molecular mechanisms involved in the pathogenesis of paediatric B-precursor acute lymphoblastic leukaemia." Thesis, University of Birmingham, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.269214.

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Books on the topic "Haematopoiesis"

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F, Grignani, Martelli M. F, and Mason D. Y, eds. Genotypic, phenotypic, and functional aspects of haematopoiesis. New York: Raven Press, 1987.

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International Symposium on Molecular Biology of Haematopoiesis (1st 1989 Innsbruck, Austria). Molecular biology of haematopoiesis: Proceedings of the International Symposium on Molecular Biology of Haematopoiesis, Innsbruck, Austria, 9-12 July 1989. Edited by Sachs Leo 1924-. Andover, Hampshire: Intercept, 1990.

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International Symposium on Molecular Biology of Haematopoiesis (8th 1993 Basel, Switzerland). Molecular biology of haematopoiesis: Proceedings of the 8th Symposium on Molecular Biology of Haematopoiesis held at theBasel Convention Center/New York Medical College, Basel, Switzerland, 9-13 July 1993. Edited by Abraham Nader G and New York Medical College. Andover, Hants, United Kingdom: Intercept, 1994.

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International, Symposium on Molecular Biology of Haematopoiesis (2nd 1991 Innsbruck Austria). Molecular biology of haematopoiesis: Proceedings of the International Conference on Molecular Biology of Haematopoiesis held at the University of Innsbruck/New York Medical College, Innsbruck, Austria, 14-18 July 1991. Andover, Hants, United Kingdom: Intercept, 1992.

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Stocking, Carol E. Autonomous growth of haematopoietic cells. Uxbridge: Brunel University, 1989.

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Bertheault-Cvitkovic, F. Breast cancer: The role of haematopoietic growth factors. Macclesfield: Gardiner-Caldwell Communications, 1991.

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Tura, S. Multiple myeloma: The role of haematopoietic growth factors. Macclesfield: Gardiner-Caldwell Communications Ltd., 1998.

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Kaye, S. B. Hodgkin's disease: The role of haematopoietic growth factors. Macclesfield: Gardiner-Caldwell Communications, 1994.

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Hoelzer, D. Myeloid malignancies: The role of haematopoietic growth factors. Macclesfield: Gardiner-Caldwell Communications, 1991.

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Crowther, D. Non-Hodgkin's lymphoma: The role of haematopoietic growth factors. Macclesfield: Gardiner-Caldwell Communications, 1990.

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Book chapters on the topic "Haematopoiesis"

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NíChonghaile, Mairéad. "Donor Selection." In The European Blood and Marrow Transplantation Textbook for Nurses, 45–52. Cham: Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-23394-4_3.

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AbstractAllogeneic haematopoietic stem cell transplant (HSCT) is the treatment of choice for a variety of malignant and non-malignant disorders. The aim of HSCT is to replace the patient’s haematopoiesis with that taken from a donor, and a prerequisite is the identification of a suitable donor. It is an intense and demanding process and puts considerable strain on both recipients and donors. The choice of donor has an impact on the transplantation process from scheduling to outcome. There are several common donor issues whether the donor is related or unrelated including eligibility, confidentiality, informed consent and right to refuse consent.
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Ribatti, Domenico, Angelo Vacca, Beatrice Nico, Enrico Crivellato, Giuseppe De Falco, and Marco Presta. "Cross Talk between Haematopoiesis and Angiogenesis." In Advances in Experimental Medicine and Biology, 25–36. Boston, MA: Springer US, 2003. http://dx.doi.org/10.1007/978-1-4615-0169-5_4.

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Dame, Chr. "Regulation of Developmental Haematopoiesis By Gata Transcription Factors." In Neonatology and Blood Transfusion, 3–23. Boston, MA: Springer US, 2005. http://dx.doi.org/10.1007/978-0-387-23600-1_1.

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Lynch, Stephen, and Jon Borresen. "Oscillations, Feedback and Bifurcations in Mathematical Models of Angiogenesis and Haematopoiesis." In Handbook of Vascular Biology Techniques, 373–90. Dordrecht: Springer Netherlands, 2015. http://dx.doi.org/10.1007/978-94-017-9716-0_29.

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Tsuzuki, S., J. Brown, K. Gale, C. Heyworth, G. May, and T. Enver. "Biology of Transcription Factor GATA-2 in Normal and Leukaemic Haematopoiesis." In Haematology and Blood Transfusion Hämatologie und Bluttransfusion, 44–51. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-59358-1_11.

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Günthert, Ursula, C. Schwärzler, B. Wittig, J. Laman, P. Ruiz, R. Stauder, A. Bloem, F. Smadja-Joffe, M. Zöller, and A. Rolink. "Functional Involvement of CD44, a Family of Cell Adhesion Molecules, in Immune Responses, Tumour Progression and Haematopoiesis." In Advances in Experimental Medicine and Biology, 43–49. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-5357-1_7.

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Taylor, Alice M., and Barbara J. Bain. "The haematopoietic system." In The Biology of Ageing and Its Clinical Implication, 50–64. London: CRC Press, 2022. http://dx.doi.org/10.1201/9781846197925-7.

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Zschunke, F., J. Barth, H. Kreipe, and H. J. Radzun. "Human Monocyte/Macrophage Serine Esterase-1: Expression in Haematopoiesis and Transcript Deficiency in Alveolar Macrophages Derived from Patients Affected by Pulmonary Fibrosis." In Esterases, Lipases, and Phospholipases, 85–89. Boston, MA: Springer US, 1994. http://dx.doi.org/10.1007/978-1-4899-0993-0_9.

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Taylor, Ian. "Lymphoid and haematopoietic system." In A Practical Guide to the Histology of the Mouse, 149–68. Chichester, UK: John Wiley & Sons, Ltd, 2013. http://dx.doi.org/10.1002/9781118789568.ch9.

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Baranov, Alexander, Dirk Densow, T. M. Fliedner, and Hauke Kindler. "Haematopoietic Stem Cell Transplantation." In Clinical Pre Computer Proforma for the International Computer Database for Radiation Exposure Case Histories, 93–97. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-78740-9_7.

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Conference papers on the topic "Haematopoiesis"

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TRONCALE, SYLVIE, FARIZA TAHI, DAVID CAMPARD, JEAN-PIERRE VANNIER, and JANINE GUESPIN. "MODELING AND SIMULATION WITH HYBRID FUNCTIONAL PETRI NETS OF THE ROLE OF INTERLEUKIN-6 IN HUMAN EARLY HAEMATOPOIESIS." In Proceedings of the Pacific Symposium. WORLD SCIENTIFIC, 2005. http://dx.doi.org/10.1142/9789812701626_0039.

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Tan, K. K., K. Z. Tang, S. Huang, A. S. Putra, T. H. Lee, S. C. Ng, Jerry K. Y. Chan, L. G. Tan, and Mark S. K. Chong. "Automated haematopoietic stem cells harvesting machine." In 2009 International Conference on Mechatronics and Automation (ICMA). IEEE, 2009. http://dx.doi.org/10.1109/icma.2009.5246304.

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Thieffry, Denis. "Logical modelling of haematopoietic cell fate reprogramming." In the 9th International Conference. New York, New York, USA: ACM Press, 2011. http://dx.doi.org/10.1145/2037509.2037512.

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Tent, Michiel. "Autologous haematopoietic stem cell transplantation versus DMTs." In ECTRIMS Congress 2022, edited by Hans-Peter Hartung. Baarn, the Netherlands: Medicom Medical Publishers, 2022. http://dx.doi.org/10.55788/320f3185.

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Lennon, Jonathon, Ian Bilmon, Megan Hogg, David Gottlieb, and Peter Middleton. "Pulmonary function changes following allogeneic haematopoietic cell transplantation." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa4190.

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van den Heuvel, Robert. "Mapping healthy HPSC variations to diagnose haematopoietic abnormalities." In EHA2023 Hybrid Congress, edited by Gert Ossenkoppele. Baarn, the Netherlands: Medicom Medical Publishers, 2023. http://dx.doi.org/10.55788/2b4af45f.

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Tang, K. Z., M. Rauff, H. C. Tan, and Y. Zhou. "Precession-based control methodology for haematopoietic stem cells harvesting." In 2013 10th IEEE International Conference on Control and Automation (ICCA). IEEE, 2013. http://dx.doi.org/10.1109/icca.2013.6565204.

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Falade, Funmi, Gulrukh Ahsan, Kimberly C. Gilmour, Fariba Tahami, and Jesmina James. "83 Immune reconstitution of patients with unrelated allogeneic haematopoietic transplants." In GOSH Conference 2019, Care of the Complex Child. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2019. http://dx.doi.org/10.1136/archdischild-2019-gosh.83.

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Yan, W. W., Y. Liu, and B. M. Fu. "Mechanical Mechanism of Circadian Fluctuations Regulated Haematopoietic Stems Cell Release." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53377.

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Abstract:
Haematopoietic stem cells (HSCs) circulate in the bloodstream under flow conditions, but the mechanical mechanisms of governing their physiological trafficking in mammals are still not yet clearly understood. The mobilization of HSCs and their progenitors into the circulation represents the basis for modern bone marrow transplantation procedures [1]. Recently, Mendez-Ferrer et al. performed experimental investigations on mice [2]. They demonstrated that the circulating HSCs and their progenitors exhibit robust circadian oscillations, and the circadian fluctuations could also be significantly altered when the HSCs were subjected to different time of lighting. These results indicated that the photic cues could affect the trafficking of HSCs in healthy animals. This implies that the light is the stimulus of HSCs release. When the HSCs are exposed to light, the HSCs release would markedly increase; when the HSCs are in darkness, the HSCs release keeps low efficiency. In this study, we numerically simulate this phenomenon to study the mechanical mechanism of circadian fluctuations regulated HSCs release under the influence of periodic lighting time.
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Cantini, Marco, Gianfranco B. Fiore, Alberto Redaelli, and Monica Soncini. "CFD-Aided Design of a Dynamic Culture System for the Co-Culture of Adherent and Non-Adherent Cells." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206431.

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Haematopoietic stem cell (HSC) transplantation has been widely used to treat patients that have undergone high-dose chemotherapy or radiotherapy for haematological or non-haematological malignancies, and is currently investigated for the treatment of several other pathologic conditions. Nevertheless, present and expected clinical applications are hindered by the shortage of cells available for transplantation. Hence, many researchers have attempted to achieve an in vitro expansion of HSCs, using different experimental set-ups and approaches, which range from traditional static monolayer cultures to three-dimensional (3D) dynamic systems. Specifically, several bioreactor systems have been proposed, including perfusion chambers, stirred, rotating, hollow fiber, and packed bed reactors [1]. Taken together, literature studies suggest that a dynamic 3D culture system may provide superior expansion of HSCs.
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Reports on the topic "Haematopoiesis"

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Hristova, Marina, Plamen Todorov, Nadya Petrova, Diana Gulenova, Ibryam Ibryam, and Elena Hristova. Clonogenic Activity of Human Haematopoietic Stem Cells Cultured under Micro-vibrations. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, May 2018. http://dx.doi.org/10.7546/crabs.2018.04.08.

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