Log in

Relevant bibliographies by topics / Guinea pig neurotransmission

Contents

Journal articles
Dissertations / Theses
Book chapters

Academic literature on the topic 'Guinea pig neurotransmission'

Author: Grafiati

Published: 4 June 2021

Last updated: 3 February 2022

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Guinea pig neurotransmission.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "Guinea pig neurotransmission"

1

Yang, Zhi-Jie, and David F. Biggs. "Muscarinic receptors and parasympathetic neurotransmission in guinea-pig trachea." European Journal of Pharmacology 193, no. 3 (February 1991): 301–8. http://dx.doi.org/10.1016/0014-2999(91)90143-e.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Park, K. H., J. P. Long, and J. G. Cannon. "Effects of serotonin1-like receptor agonists on autonomic neurotransmission." Canadian Journal of Physiology and Pharmacology 69, no. 12 (December 1, 1991): 1855–60. http://dx.doi.org/10.1139/y91-274.

Full text

Abstract:

Serotonin1A receptor agonists, 8-hydroxy-2-(di-n-propylamino)tetralin and 10-methyl-11-hydroxyaporphine, inhibited electrical stimulation-induced contraction of the guinea-pig ileum. These agonists also inhibited the pressor and tachycardiac responses to low frequency (0.25 Hz) but not to high frequency (2.0 Hz) electrical stimulation of the sympathetic nervous system in pithed rats. Serotonin1B receptor agonist RU 24969 inhibited pressor and tachycardiac responses to both low and high frequencies of stimulation in pithed rats. In the cat nictitating membrane, serotonin1A receptor agonists did not alter contractions elicited by electrical stimulation (0.1–3.0 Hz). Serotonin not only contracted the cat nicitating membrane but also facilitated contractile responses to low frequency (0.1 – 1.0 Hz) stimulation. The contractile effect of serotonin in the cat nictitating membrane was blunted by bretylium, methysergide, and ketanserin, but not by metoclopramide. The facilitatory effect of serotonin was antagonized by methysergide, but not by ketanserin, pindolol, propranolol, or metoclopramide. These results suggest that serotonin1A receptors modulate autonomic neurotransmission in the guinea-pig ileum and pithed rats, but not in the cat nictitating membrane. Serotonin contracts the cat nictitating membrane via serotonin2 subtypes, while facilitating stimulated contractile responses through the serotonin1-like receptors.Key words: guinea-pig ileum, pithed rats, nictitating membrane, serotonin receptors.

APA, Harvard, Vancouver, ISO, and other styles

3

Zhang, Lili, John C. Hancock, and Donald B. Hoover. "Tachykinin Agonists Modulate Cholinergic Neurotransmission at Guinea-Pig Intracardiac Ganglia." Journal of Pharmacological Sciences 99, no. 3 (2005): 228–38. http://dx.doi.org/10.1254/jphs.fp0050437.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Ichinose, M., C. D. Stretton, J. C. Schwartz, and P. J. Barnes. "Histamine H3-receptors inhibit cholinergic neurotransmission in guinea-pig airways." British Journal of Pharmacology 97, no. 1 (May 1989): 13–15. http://dx.doi.org/10.1111/j.1476-5381.1989.tb11917.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Belvisi, M. G., C. D. Stretton, and P. J. Barnes. "Modulation of cholinergic neurotransmission in guinea-pig airways by opioids." British Journal of Pharmacology 100, no. 1 (May 1990): 131–37. http://dx.doi.org/10.1111/j.1476-5381.1990.tb12064.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Kortezova, N. I., L. I. Shikova, E. A. Milusheva, D. E. Itzev, V. A. Bagaev, and Z. N. Mizhorkova. "Muscarinic modulation of nitrergic neurotransmission in guinea-pig gastric fundus." Neurogastroenterology and Motility 16, no. 2 (April 2004): 155–65. http://dx.doi.org/10.1111/j.1365-2982.2004.00514.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Ishikawa, Shiro, and Nicholas Sperelakis. "Cyclic Nucleotide Regulation of Neurotransmission in Guinea Pig Mesenteric Artery." Journal of Cardiovascular Pharmacology 13, no. 6 (June 1989): 836–45. http://dx.doi.org/10.1097/00005344-198906000-00005.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Xiao-Xing, Luo, Tan Yue-Hua, and Sheng Bao-Hen. "Histamine H3-receptors inhibit sympathetic neurotransmission in guinea pig myocardium." European Journal of Pharmacology 204, no. 3 (November 1991): 311–14. http://dx.doi.org/10.1016/0014-2999(91)90857-m.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Liu, Sumei, Hong-Zhen Hu, Na Gao, Chuanyun Gao, Guodu Wang, Xiyu Wang, Owen C. Peck, et al. "Neuroimmune interactions in guinea pig stomach and small intestine." American Journal of Physiology-Gastrointestinal and Liver Physiology 284, no. 1 (January 1, 2003): G154—G164. http://dx.doi.org/10.1152/ajpgi.00241.2002.

Full text

Abstract:

Enteric neuroimmune interactions in gastrointestinal hypersensitivity responses involve antigen detection by mast cells, mast cell degranulation, release of chemical mediators, and modulatory actions of the mediators on the enteric nervous system (ENS). Electrophysiological methods were used to investigate electrical and synaptic behavior of neurons in the stomach and small intestine during exposure to β-lactoglobulin in guinea pigs sensitized to cow's milk. Application of β-lactoglobulin to sensitized preparations depolarized the membrane potential and increased neuronal excitability in small intestinal neurons but not in gastric neurons. Effects on membrane potential and excitability in the small intestine were suppressed by the mast cell stabilizing drug ketotifen, the histamine H2 receptor antagonist cimetidine, the cyclooxygenase inhibitor piroxicam, and the 5-lipoxygenase inhibitor caffeic acid. Unlike small intestinal ganglion cells, gastric myenteric neurons did not respond to histamine applied exogenously. Antigenic exposure suppressed noradrenergic inhibitory neurotransmission in the small intestinal submucosal plexus. The histamine H3receptor antagonist thioperamide and piroxicam, but not caffeic acid, prevented the allergic suppression of noradrenergic inhibitory neurotransmission. Antigenic stimulation of neuronal excitability and suppression of synaptic transmission occurred only in milk-sensitized animals. Results suggest that signaling between mast cells and the ENS underlies intestinal, but not gastric, anaphylactic responses associated with food allergies. Histamine, prostaglandins, and leukotrienes are paracrine signals in the communication pathway from mast cells to the small intestinal ENS.

APA, Harvard, Vancouver, ISO, and other styles

10

Patra, Phani B., and David P. Westfall. "Potentiation of Purinergic Neurotransmission in Guinea Pig Urinary Bladder by Histamine." Journal of Urology 151, no. 3 (March 1994): 787–90. http://dx.doi.org/10.1016/s0022-5347(17)35088-7.

Full text

APA, Harvard, Vancouver, ISO, and other styles

More sources

Dissertations / Theses on the topic "Guinea pig neurotransmission"

1

Harvey, Richard Alan. "Purinergic neurotransmission in the human and guinea pig bladder." Thesis, University of London, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.407186.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Lee, John Joseph. "Presynaptic receptors in the guinea-pig ileum : signalling pathways and interactions." Thesis, University of Hertfordshire, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.245500.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Manchanda, Rohit. "New insights into sympathetic transmission." Thesis, University of Oxford, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.238131.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Patel, Hema. "Modulation of cholinergic neurotransmission in guinea-pig and human airways." Thesis, Imperial College London, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.267164.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Leger, James P. "Adrenergic modulation of neurotransmission in the intracardiac nervous system of the guinea-pig." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp03/NQ49274.pdf.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Shah, Saloni. "Modulation of excitatory non-adrenergic non-cholinergic neurotransmission in guinea-pig airways : functional and release studies." Thesis, King's College London (University of London), 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.391715.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

PETERS, STEPHEN. "NONCHOLINERGIC NEUROTRANSMISSION IN THE GUINEA PIG INFERIOR MESENTERIC GANGLION: A SENSORY ROLE IN GASTROINTESTINAL PHYSIOLOGY (SYMPATHETIC, SLOW EPSP, PREVERTEBRAL, VASOPRESSIN, SUBSTANCE P)." Diss., The University of Arizona, 1985. http://hdl.handle.net/10150/188009.

Full text

Abstract:

Noncholinergic neurotransmission was studied in vitro in the guinea pig inferior mesenteric ganglion (IMG) using the technique of intracellular recording. First, the role of substance P (SP) in noncholinergic transmission was examined. Forty-four percent of IMG neurons depolarized upon superfusion of SP (1-10 x 10⁻⁷M); however, some neurons that were insensitive to SP still exhibited slow excitatory potentials (EPSPs) in response to nerve stimulation. During exposure to SP, slow EPSPs were depressed by 52% compared to paired control EPSPs. In animals treated with systemic doses of capsaicin (50-350 mg/kg), mean slow EPSP amplitude was 3.5 mV compared to 6.8 mV in untreated animals. Arginine-vasopressin (AVP) was tested for its electrophysiological effects on IMG neurons and synaptic transmission. AVP (0.5-10 x 10⁻⁷M) produced a depolarization in 67% of neurons, accompanied by an increase in membrane resistance of 44%. The depolarizations and increase in resistance were blocked by a specific V₁ receptor antagonist. During AVP-induced depolarizations, slow EPSPs were reversibly depressed in the majority of neurons by a mean of 71% relative to paired control EPSPs. The V₁ antagonist blocked slow EPSPs in only 10% of neurons tested. A group of neurons exhibiting slow EPSPs was exposed separately to both AVP and SP. Some neurons were exclusively sensitive to either SP or AVP, others were sensitive to both peptides, and still others were sensitive to neither peptide. The physiologic role of noncholinergic transmission was examined using a preparation consisting of a segment of distal colon attached to the IMG. Distension of the colon segment produced a slow depolarization resistant to cholinergic antagonists in 44% of IMG neurons. Distension-induced noncholinergic depolarizations increased in amplitude with colonic intraluminal pressure and with membrane hyperpolarization, and were accompanied by an increase in membrane input resistance of 21%. Capsaicin in vivo reduced the number of neurons exhibiting the noncholinergic mechanosensory depolarization, and in vitro capsaicin and SP desensitization reduced the amplitude of the depolarization. These results suggest that (1) both SP and AVP may be transmitters of noncholinergic potentials in the IMG, (2) some IMG neurons receive heterogeneous peptidergic innervation, and (3) noncholinergic transmission in the IMG is involved in sensory regulation of visceral autonomic function.

APA, Harvard, Vancouver, ISO, and other styles

8

Chih-Hsien, Shih, and 施志賢. "Calcium channel subtypes for cholinergic and non-adrenergic non-cholinergic neurotransmission in the isolated guinea-pig trachea." Thesis, 1998. http://ndltd.ncl.edu.tw/handle/15691219016239188311.

Full text

Abstract:

碩士
台北醫學院
醫學研究所
86
1. The Ca2+ channel subtypes of the neurotransmission of isolated guinea-pig trachea were elucidated by monitoring the effects of specific Ca2+ channel blo ckers on the cholinergic contraction and non-adrenergic non-cholinergic (NANC) relaxation elicited by electrical field stimulation (EFS).2. In the isolated guinea-pig trachea, the cholinergic contractile responses to low and high fre quency of EFS were inhibited by both N-type calcium channel blockers, ω-conot oxin GVIA and ω-conotoxin MVIIA. ω-Agatoxin IVA (a P-type blocker), ω-conot oxin MVIIC (a N-, O-, P- and Q-type blocker) and nifedipine (a L-type blocker) were ineffective. Whereas, Ni2+ (a T- and R-type blocker), facilitated the ch olinergic contraction and produced a late contractile phase when its concentra tion was higher than 30 mM. The more concentration of Ni2+ increased, the more incidence and its contractile area of the late contraction occurred. The late contraction seems not due to the effects of acetylcholine, tachykinins and ot her polypeptides.3. The NANC relaxant response elicited by low and high frequ ency of EFS was inhibited by ω-conotoxin GVIA, ω-conotoxin MVIIA and Ni2+. ω-Agatoxin IVA, ω-conotoxin MVIIC and nifedipine were ineffective.4. These results suggested that, in the isolated guinea-pig trachea, the cholinergic co ntraction is regulated by N-type calcium channel while that of NANC relaxation is controlled by N-, T- and R-type calcium channels.

APA, Harvard, Vancouver, ISO, and other styles

Book chapters on the topic "Guinea pig neurotransmission"

1

Kamikawa, Y., and Y. Shimo. "Modulating Effects of Antiasthmatic and Antitussive Drugs on Autonomic Neurotransmission in Guinea-Pig Bronchial Muscle in Vitro." In Mediators in Airway Hyperreactivity, 67–70. Basel: Birkhäuser Basel, 1990. http://dx.doi.org/10.1007/978-3-0348-7379-6_7.

Full text

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!