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Books on the topic 'Guillain-Barré'

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Published: 4 June 2021
Last updated: 19 February 2022

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1

Hughes, Richard A. C. Guillain-Barré Syndrome. London: Springer London, 1990. http://dx.doi.org/10.1007/978-1-4471-3175-5.

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2

Meulstee, Jan. Electrodiagnostic studies in Guillain-Barré syndrome =: Elektrodiagnostisch onderzoek van het Guillain-Barré syndroom. Delft: Eburon, 1994.

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3

S, Steinberg Joel, ed. Guillain-Barré syndrome: From diagnosis to recovery. New York, N.Y: Demos, 2007.

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4

Quand une porte se ferme, une fenêtre s'ouvre: Autobiographie. [Alma, Québec]: Éditions SM, 2010.

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5

Handelsman, Harry. Apheresis in the treatment of Guillain-Barre syndrome. Rockville, MD: National Center for Health Services Research and Health Care Technology Assessment, U.S. Dept. of Health and Human Services, Public Health Service, 1985.

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6

Buzby, Jean C. Estimated annual costs of Campylobacter-associated Guillain-Barré syndrome. [Washington, DC]: U.S. Dept. of Agriculture, ERS, 1997.

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7

Wijdicks, Eelco F. M., 1954- and Truax Bradley T. 1948-, eds. Guillain-Barré syndrome. Philadelphia: F.A. Davis, 1991.

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8

Roe, Monica M. Thaw. Asheville, N.C: Front Street, 2008.

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9

Gerlach, Holly. Happily ever after: My journey with Guillain-Barré syndrome and how I got my life back. [Bloomington, Indiana]: Trafford Publishing, 2012.

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10

Guillain-Barré syndrome. London: Springer-Verlag, 1990.

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11

Ali, Wahid. Back from the brink--: A personal account of the illness Guillain-Barré syndrome. San Juan, Trinidad, West Indies: Wali Enterprises, 1997.

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12

Cārudatta, Kalpanā. Tyāñcyā siṇḍromacī kathā. Puṇe: Mehatā Pabliśiṅga Hāusa, 2009.

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13

The two kinds of decay. New York: Farrar, Straus and Giroux, 2008.

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14

Hwan-ung, Ch'oe, ed. Paengman pun ŭi 1. Sŏul-si: Pungnaep, 2015.

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15

Hughes, R. A. C. Guillain-Barré Syndrome: A guide for patients, relatives and friends. 4th ed. Sleaford: GBS Support Group, 1998.

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16

Buzby, Jean C. Estimated annual costs of Campylobacter-associated Guillain-Barré syndrome. Washington, DC: U.S. Dept. of Agriculture, ERS, 1997.

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17

Buzby, Jean C. Estimated annual costs of Campylobacter-associated Guillain-Barré syndrome. Washington, DC: U.S. Dept. of Agriculture, ERS, 1997.

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18

Dominguez, Roy. Valor: The American odyssey of Roy Dominguez. Bloomington: Indiana University Press, 2012.

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19

Masks. New York: Orchard Books, 1996.

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20

Eastman, Gil. Deaf mosaic. Washington, DC: Dept. of Television, Film, and Photography, Gallaudet University, 1993.

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21

Joseph, Heller. No laughing matter. New York: Simon & Schuster Paperbacks, 2004.

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22

Joseph, Heller. No laughing matter. London: Cape, 1986.

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23

Joseph, Heller. No laughing matter. London: Corgi, 1987.

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24

Joseph, Heller, and Heller Joseph. No laughing matter. New York: Putnam, 1986.

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25

Buzby, Jean C. Estimated annual costs of Campylobacter-associated Guillain-Barr e syndrome. Washington, DC: U.S. Dept. of Agriculture, Economic Research Service, 1997.

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26

Baier, Sue. Bed number ten. New York: Holt, Rinehart, and Winston, 1986.

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27

Zimmeth, Schomaker Mary, ed. Bed number ten. Boca Raton, Fla: CRC Press, 1995.

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28

Valor: The American odyssey of Roy Dominguez. Bloomington: Indiana University Press, 2012.

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29

Guillain-Barré syndrome. Bethesda, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 2001.

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30

National Institute of Neurological Disorders and Stroke (U.S.). Office of Communications and Public Liaison and National Institutes of Health (U.S.), eds. Guillain-Barré syndrome. Bethesda, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 2001.

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31

National Institute of Neurological Disorders and Stroke (U.S.). Office of Communications and Public Liaison and National Institutes of Health (U.S.), eds. Guillain-Barré syndrome. Bethesda, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 2001.

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32

National Institute of Neurological and Communicative Disorders and Stroke. Office of Communications and Public Liaison and National Institutes of Health (U.S.), eds. Guillain-Barré syndrome. Bethesda, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 2001.

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33

National Institute of Neurological Disorders and Stroke (U.S.). Office of Communications and Public Liaison and National Institutes of Health (U.S.), eds. Guillain-Barré syndrome. Bethesda, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 2001.

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34

Guillain-Barré Syndrome. Springer, 2011.

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35

National Institute of Neurological and Communicative Disorders and Stroke. Office of Communications and Public Liaison. and National Institutes of Health (U.S.), eds. Guillain-Barré syndrome. Bethesda, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 2001.

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36

National Institute of Neurological Disorders and Stroke (U.S.). Office of Communications and Public Liaison, ed. El síndrome de Guillain-Barré. Bethesda, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 2000.

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37

National Institute of Neurological Disorders and Stroke (U.S.). Office of Communications and Public Liaison, ed. El síndrome de Guillain-Barré. Bethesda, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 2000.

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38

National Institute of Neurological Disorders and Stroke (U.S.). Office of Communications and Public Liaison, ed. El síndrome de Guillain-Barré. Bethesda, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 2000.

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39

National Institute of Neurological Disorders and Stroke (U.S.). Office of Communications and Public Liaison, ed. El síndrome de Guillain-Barré. Bethesda, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 2000.

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40

Miller, Aaron E., and Teresa M. DeAngelis. Acute Inflammatory Demyelinating Polyneuropathy (Guillain-Barré Syndrome). Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199732920.003.0024.

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Acute inflammatory demyelinating polyneuropathy (AIDP), also known as Guillain-Barré syndrome (GBS), is a common acute neurological presentation encountered in both the outpatient setting and hospital wards. The hallmark of the disorder is the development of ascending motor paralysis with loss of deep tendon reflexes. In this chapter, we outline the classical clinical and laboratory findings in GBS as well as critical therapeutic and supportive measures along with prognosis.
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41

Steinberg, Joel S., and Gareth John Parry. Guillain-Barre Syndrome: From Diagnosis to Recovery (American Academy of Neurology). Demos Medical Publishing, 2007.

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42

Brealey, David, and Nicholas Hirsch. Diagnosis, assessment, and management of Guillain–Barré syndrome. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0246.

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The Guillain–Barré Syndrome describes a spectrum of acute inflammatory polyneuropathies and is the commonest cause of acute flaccid paralysis within the western world. The pathophysiology is complex and poorly understood, but appears to be an immune-mediated destruction of either the myelin sheath and/or the axons, predominantly of motor nerves. The clinical presentation is classically a rapid, ascending, flaccid paralysis, with minimal sensory deficit. This may ascend to involve respiratory or bulbar muscle function. These patients need careful monitoring and, if deteriorating, should be electively intubated and ventilated. Autonomic instability and sensory disturbance, including pain, is common. Treatment of the underlying condition relies upon immunomodulation with either intravenous immunoglobulin or plasma exchange. Supportive care is aimed at maintaining a safe airway, ventilatory support, and managing the complications of autonomic dysfunction and prolonged immobility. Mortality rates range up to 20%, but are significantly better in specialist neuromedical units. Survivors are often left with significant disability.
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43

Iannello, Silvia. Guillain-Barre Syndrome: Pathological, Clincal, and Therapeutical Aspects. Nova Science Publishers, 2005.

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44

Katirji, Bashar. Case 23. Edited by Bashar Katirji. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190603434.003.0027.

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Guillain-Barré syndrome is the prototype of acute immune-mediated neuropathies. Guillain-Barré syndrome has several subtypes including acute inflammatory demyelinating polyneuropathy, acute motor axonal neuropathy, and acute motor sensory axonal neuropathy. Guillain-Barré syndrome has also several variants including Miller Fisher syndrome, ataxic form, and pharyngeal–cervical–brachial form. This case highlights the clinical findings in Guillain-Barré syndrome and discusses in details the diagnostic criteria that are essential in confirming the diagnosis and excluding mimickers of the disorder. This is followed by a detailed discussion on the electrodiagnostic findings in Guillain-Barré syndrome during the acute presentation and recovery phase. The diagnostic sensitivity and specificity of the various findings seen on nerve conduction studies are included.
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45

Thaisetthawatkul, Pariwat, and Eric Logigian. Guillain-Barré Syndrome and Chronic Inflammatory Demyelinating Polyradiculoneuropathy in Pregnancy. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190667351.003.0026.

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Guillain-Barré syndrome (GBS) and chronic inflammatory demyelinating polyradiculoneuropathy (CIDP) are both immune-mediated diseases of the peripheral nervous system that typically present with symmetric, progressive muscle weakness, areflexia, and sensory symptoms or signs. GBS evolves rapidly with a nadir at 2–4 weeks usually with an antecedent viral illness, while CIDP progresses more slowly over months to years. GBS is sometimes complicated by life-threatening respiratory failure or dysautonomia. Onset of GBS and relapse of CIDP can occur during pregnancy or postpartum. But with appropriate supportive care and immunotherapy, maternal and fetal outcome in both conditions is typically excellent. The exception is fetal outcome in GBS triggered by maternal CMV or Zika infection transmitted to the fetus. Full-term vaginal delivery and regional anesthesia are preferred in maternal GBS and CIDP, but if C-section and general anesthesia are indicated, non-depolarizing agents such as succinylcholine should be avoided.
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46

Roe, Monica. Thaw. Front Street imprint of Boyds Mills Press, 2008.

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47

Kaplan, Tamara, and Tracey Milligan. Demyelinating Diseases 2: NMO, ADEM, GBS, CIDP (DRAFT). Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190650261.003.0014.

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The video in this chapter explores demyelinating diseases, and focuses on neuromyelitis optica (NMO), acute disseminated encephalomyelitis (ADEM), Guillain-Barré syndrome (GBS) and chronic inflammatory demyelinating polyneuropathy (CIDP), including their symptoms, causes, and diagnostic tests.
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48

Hatrick, Gloria. MASKS. Julia MacRae, 1992.

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49

Vogel, Speed, and Heller Joseph. No Laughing Matter. Simon & Schuster, 2004.

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50

Joseph, Heller. No Laughing Matter. Plume, 1995.

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