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Journal articles on the topic 'Guillain-Barre Syndrome'

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Published: 4 June 2021

Last updated: 29 July 2024

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1

Kutepov, D. E., and N. I. Litvinov. "Guillain-Barre syndrome." Kazan medical journal 96, no. 6 (December 15, 2015): 1027–34. http://dx.doi.org/10.17750/kmj2015-1027.

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Guillain-Barre syndrome is an acute inflammatory demyelinating polyradiculoneuropathy of autoimmune etiology, which is characterized by peripheral paralysis and protein-cell dissociation in the cerebrospinal fluid in most cases. The Guillain-Barre syndrome prevalence is 0.6-2.4 cases per 100 thousand population. In Moscow, about 200 people are taken ill with Guillain-Barre syndrome each year. Currently, four main clinical variants of Guillain-Barre syndrome are described: acute inflammatory demyelinating polyradiculoneuropathy, axonal form, acute motor axonal neuropathy, and Miller-Fisher syndrome. Disease development is preceded by contact with the viral or bacterial infections causative agent such as Campylobacter jejuni, Mycoplasma pneumonia, cytomegalovirus, Epstein-Barr virus and influenza virus. Guillain-Barre syndrome pathogenesis is «molecular mimicry» between infectious agents surfaces and the peripheral nerves structures. High titer of antibodies to the GM1, GD1a, GD1b and GQ1b gangliosides is found in patients blood serum. Diagnostic criteria for the Guillain-Barre syndrome diagnosis are the physical examination results, cerebrospinal fluid analysis and electroneuromyographic study. The North American motor deficit severity scale is used to assess the neurological status. This scale allows to evaluate the patient’s condition and movement abilities. Currently plasmapheresis and immunoglobulin G therapy are the main treatment options for patients with Guillain-Barre syndrome. The favorable prognosis in the form of disease clinical manifestations regression reaches 60-80%. Mortality in Guillain-Barre syndrome is 5% in average and may reach 20% in patients on mechanical ventilation. The most common death causes of patients with Guillain-Barre syndrome are respiratory failure, aspiration pneumonia, sepsis, and pulmonary embolism. Early treatment initiation can reduce serious complications risk, including respiratory failure, what ultimately leads to decrease in mortality and patients disablement.

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2

Bhagat, Saroj Kumar, Shrey Sidhant, Mukesh Bhatta, Ashish Ghimire, and Bhupendra Shah. "Clinical Profile, Functional Outcome, and Mortality of Guillain-Barre Syndrome: A Five-Year Tertiary Care Experience from Nepal." Neurology Research International 2019 (June 2, 2019): 1–5. http://dx.doi.org/10.1155/2019/3867946.

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Introduction. Guillain-Barre syndrome is the most common cause of acute flaccid paralysis in the adult population. It occurs at the rate of 0.34 to 4 per 100000 individuals. This study was conducted to determine the clinicoepidemiological profile and outcome of the patients with Guillain-Barre syndrome. Materials and Methods. We conducted a retrospective study of patients with Guillain-Barre syndrome, presented at B.P. Koirala Institute of Health Sciences, a tertiary care centre in eastern Nepal, from January 2013 to December 2017. All patients diagnosed with Guillain-Barre syndrome were included in this study. The handwritten case record files of the study population were retrieved from medical record section of the institute. Results. Of 31 patients with Guillain-Barre syndrome, the mean age of patients was 17±12 years. The most common presenting symptom of study population was ascending paralysis (93.5%). Respiratory failure requiring mechanical ventilation occurred in 16.1%. The common variants are AIDP and AMAN. Respiratory tract infection (29%) was the most common antecedent event. The in-hospital mortality of Guillain-Barre syndrome was 6.45%. Conclusion. Guillain-Barre syndrome is commonly seen in the young population. The most common symptom of Guillain-Barre syndrome was ascending paralysis. The in-hospital mortality rate of patients with GBS was 6.45%.

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3

Rosen, B. A. "Guillain-Barre Syndrome." Pediatrics in Review 33, no. 4 (April 1, 2012): 164–71. http://dx.doi.org/10.1542/pir.33-4-164.

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4

Rabinstein, Alejandro A. "Guillain-Barre Syndrome." Open General & Internal Medicine Journal 1, no. 1 (December 5, 2007): 13–22. http://dx.doi.org/10.2174/1874076600701010013.

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5

Evans, O. B., and V. Vedanarayanan. "Guillain-Barre Syndrome." Pediatrics in Review 18, no. 1 (January 1, 1997): 10–16. http://dx.doi.org/10.1542/pir.18-1-10.

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6

Raposo, Anna Julia Lacerda, Eduardo Galvão Freire, Célia Raiany Ferreira de Farias, Pinheiro Renato Serquiz e, Francisco Irochima Pinheiro, Fausto Pierdoná Guzen, Amália Cinhtia Meneses Rêgo, and Irami Araújo-Filho. "Guillain-Barre syndrome." GSC Biological and Pharmaceutical Sciences 9, no. 1 (October 30, 2019): 098–103. http://dx.doi.org/10.30574/gscbps.2019.9.1.0198.

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7

ASHRAF, SOHAIL, and ARSHALOOZ J. RAHMAN. "GUILLAIN BARRE SYNDROME;." Professional Medical Journal 20, no. 03 (March 25, 2013): 348–53. http://dx.doi.org/10.29309/tpmj/2013.20.03.708.

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Background: Guillian Barre Syndrome is a polyradiculopathy characterized by symmetric ascending paralysis andareflexia. It affects all age groups and both sexes with an unpredictable outcome. Objectives: The aims of this study were to identify thevarious diseases presenting as Acute Flaccid Paralysis and notice clinical features and outcome of cases of Guillian Barre Syndrome.Materials and Methods: Retrospective analysis of 53 patients presenting as Acute Flaccid Paralysis was done. Clinical features andlaboratory investigations of 39 patients of Guillian Barre syndrome were reviewed. Results: Among all cases of Acute Flaccid Paralysis,Guillian Barre syndrome formed the bulk of the cases (73.6%).It was more common in females and in age group of 10 years or below. Inmajority of the cases, rapid onset of weakness was the initial symptom. Mortality was 100% in patients who had respiratory muscleinvolvement and who required mechanical ventilation. Conclusions: In our study the main factor causing death in a case of Guillian Barresyndrome was bulbar involvement requiring mechanical ventilation. Early recognition of the prognostic factors may lead to decreasedmortality in case of Guillian Barre syndrome.

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8

KAMOSHITA, Hiroshi. "Guillain-Barre Syndrome." Japanese Journal of Rehabilitation Medicine 34, no. 10 (1997): 706–11. http://dx.doi.org/10.2490/jjrm1963.34.706.

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9

Malek, Elia, and Johnny Salameh. "Guillain–Barre Syndrome." Seminars in Neurology 39, no. 05 (October 2019): 589–95. http://dx.doi.org/10.1055/s-0039-1693005.

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AbstractGuillain–Barre syndrome (GBS) is the leading cause of acute paralysis that can potentially affect all of the human population. GBS is believed to be an immune-mediated disease, possibly triggered by a recent infection, and driven by an immune attack targeting the peripheral nervous system. GBS can be divided into several subtypes depending on the phenotype, pathophysiology, and neurophysiological features. Unfortunately, morbidity and mortality rates are still high despite the current understanding of the pathophysiology and available treatment options. Additional research is still needed to shed more light into the pathogenesis for a better understanding and treatment of this condition.

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10

Costigan, D. "Guillain-Barre Syndrome." Neurology 42, no. 2 (February 1, 1992): 468. http://dx.doi.org/10.1212/wnl.42.2.468.

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11

Walk, David. "Guillain-Barre Syndrome." Surgical Neurology 47, no. 3 (March 1997): 305–7. http://dx.doi.org/10.1016/s0090-3019(96)00478-8.

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12

Winer, J. B. "Guillain-Barre syndrome." BMJ 337, jul17 1 (July 17, 2008): a671. http://dx.doi.org/10.1136/bmj.a671.

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13

Hughes, Richard A. C., and Jeremy H. Rees. "Guillain-Barre syndrome." Current Opinion in Neurology 7, no. 5 (October 1994): 386–92. http://dx.doi.org/10.1097/00019052-199410000-00004.

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14

Seneviratne, U. "Guillain-Barre syndrome." Postgraduate Medical Journal 76, no. 902 (December 1, 2000): 774–82. http://dx.doi.org/10.1136/pmj.76.902.774.

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15

Al Twaijri, Waleed Abdulaziz. "Guillain-Barre Syndrome." International Journal of Child Neuropsychiatry 13, no. 1-2 (June 2016): 7–15. http://dx.doi.org/10.12816/0036871.

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16

Bowyer, Holly R., and Melissa Glover. "Guillain-Barre Syndrome." Journal of Neuroscience Nursing 42, no. 5 (October 2010): 288–93. http://dx.doi.org/10.1097/jnn.0b013e3181ecafa9.

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17

McKhann, G. M. "Guillain-Barre Syndrome." Archives of Neurology 49, no. 11 (November 1, 1992): 1110. http://dx.doi.org/10.1001/archneur.1992.00530350020007.

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18

Farazdaghi, Mohsen, Anahita Zoghi, and Afshin Borhani Haghighi. "Guillain-Barre Syndrome after Kidney Transplantation: a Case Report." Galen Medical Journal 2, no. 1 (March 31, 2013): 35–36. http://dx.doi.org/10.31661/gmj.v2i1.33.

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Background: Guillain-Barre Syndrome is an unusual complication of hematopoietic stem cell transplantation but it is extremely rare after solid organ transplantation such as kidney or liver transplantationCase report: A 48-year-old man, a case of kidney transplantation presented with generalized weakness in an ascending pattern. History and examination were compatible with the diagnosis of Guillain-Barre Syndrome (GBS) and paraclinical studies confirmed this diagnosis. He was treated for Guillain-Barre syndrome but no significant response was observed.Conclusion: Guillain–Barre´ syndrome rarely appears after organ transplantation but it should be considered in a patient presenting with its associated symptoms after transplantation.

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19

Mungan, Semra. "Prognostic factors in Guillain-Barre syndrome." Dicle Medical Journal 41, no. 4 (December 1, 2014): 667–70. http://dx.doi.org/10.5798/diclemedj.0921.2014.04.0496.

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20

Millichap, J. Gordon. "Neonatal Guillain-Barre’ Syndrome." Pediatric Neurology Briefs 2, no. 9 (September 1, 1988): 66. http://dx.doi.org/10.15844/pedneurbriefs-2-9-2.

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21

Pfister, SM, and JB Bullas. "Acute Guillain-Barre syndrome." Critical Care Nurse 10, no. 10 (November 1, 1990): 68–73. http://dx.doi.org/10.4037/ccn1990.10.10.68.

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22

Millichap, J. Gordon. "Guillain-Barre Syndrome Outcome." Pediatric Neurology Briefs 11, no. 2 (February 1, 1997): 13. http://dx.doi.org/10.15844/pedneurbriefs-11-2-7.

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23

Vallat, J. M. "Sensory Guillain-Barre syndrome." Neurology 39, no. 6 (June 1, 1989): 879. http://dx.doi.org/10.1212/wnl.39.6.879.

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24

Lawn, N. D., and E. F. M. Wijdicks. "Fatal Guillain-Barre syndrome." Neurology 52, no. 3 (February 1, 1999): 635. http://dx.doi.org/10.1212/wnl.52.3.635.

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25

Triggs, W. J., and D. Cros. "Axonal Guillain-Barre syndrome." Neurology 43, no. 7 (July 1, 1993): 1443. http://dx.doi.org/10.1212/wnl.43.7.1443.

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26

Yuki, N., and T. Miyatake. "Axonal Guillain-Barre syndrome." Neurology 43, no. 7 (July 1, 1993): 1443. http://dx.doi.org/10.1212/wnl.43.7.1443-a.

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27

van den Berg, B., C. Fokke, J. Drenthen, P. A. van Doorn, and B. C. Jacobs. "Paraparetic Guillain-Barre syndrome." Neurology 82, no. 22 (May 7, 2014): 1984–89. http://dx.doi.org/10.1212/wnl.0000000000000481.

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28

Wilmshurst, Jo M., Keith R. E. Pohl, Robert W. Vaughan, and Richard A. C. Hughes. "Familial Guillain-Barre Syndrome." European Journal of Neurology 6, no. 4 (July 1999): 499–503. http://dx.doi.org/10.1046/j.1468-1331.1999.640499.x.

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29

Winer, J. "Guillain-Barre syndrome revisited." BMJ 304, no. 6819 (January 11, 1992): 65–66. http://dx.doi.org/10.1136/bmj.304.6819.65.

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30

Kuitwaard, K., R. van Koningsveld, L. Ruts, B. C. Jacobs, and P. A. van Doorn. "Recurrent Guillain-Barre syndrome." Journal of Neurology, Neurosurgery & Psychiatry 80, no. 1 (October 17, 2008): 56–59. http://dx.doi.org/10.1136/jnnp.2008.156463.

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31

Grishina, D. A., and N. A. Suponeva. "Specificity of recovery in acute motor axonal neuropathy with conduction blocks on the example of two clinical cases." Neuromuscular Diseases 10, no. 4 (December 29, 2020): 43–51. http://dx.doi.org/10.17650/2222-8721-2020-10-4-43-51.

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Introduction. The heterogeneity of the forms and severity of Guillain-Barre syndrome explains the variability of recovery: from rapid and complete (in most cases) to slow with the development of persistent residual deficiency (rarely). It is unclear how effective the Erasmus Guillain–Barre syndrome Outcome Scores and its modified version are for different forms of the disease.The aim of the study – to demonstrate the features of recovery in acute motor axonal neuropathy with conduction blocks on the example of 2 clinical cases; to show the possibilities of Erasmus Guillain–Barre syndrome Outcome Scores and its modified version in predicting recovery in this form of the disease.Materials and methods. Data from 2 patients with acute motor axonal neuropathy with motor conduction blocks were retrospectively analyzed. Calculation of the score and assessment of the prognosis of walking recovery by 6 months from the onset of the disease were performed using the online calculator International Guillain-Barre syndrome Outcome Study Prognosis tool in the acute period.Results. In both patients, the forecast of recovery of walking by half a year from the onset of the disease on the Erasmus Guillain–Barre syndrome Outcome Scores and modified Erasmus Guillain–Barre syndrome Outcome Scores scales in the acute period was erroneous. In the first case, the total score on the Erasmus Guillain–Barre syndrome Outcome Scores and its modification in the acute period was 5 and 10 points respectively (poor prognosis), which foreshadowed a long rehabilitation process and incomplete recovery. However, the regression of disorders was dramatic and complete, and by the second month of the disease, only minimal motor disorders remained. In the second patient, on the contrary, the total Erasmus Guillain–Barre syndrome Outcome Scores and its modification during the period of increasing symptoms was 3 and 7 points respectively (good prognosis), while recovery was delayed – only by 5 months from the onset of the disease, the ability to move with support was restored.Conclusion. The Guillain–Barre syndrome is a disease with a favorable prognosis for recovery. However, the prediction of regression of motor disorders should be approached carefully, because in some cases, generally accepted criteria and prognostic scales may not work. Acute motor axonal neuropathy with conduction blocks is a unique form of the disease that has pathophysiological and clinical-neurophysiological features, which should be taken into account when managing this category of patients.

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32

Lowery, Megan M., Muhammad Taimur Malik, Joseph Seemiller, and Cynthia S. Tsai. "Atypical Variant of Guillain Barre Syndrome in a Patient with COVID-19." Journal of Critical Care Medicine 6, no. 4 (November 7, 2020): 231–36. http://dx.doi.org/10.2478/jccm-2020-0038.

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AbstractObjectiveA rare variant Miller Fisher Syndrome overlap with Guillain Barre Syndrome is described in an adult patient with SARS-COV-2 infection.Case PresentationThe clinical course of a 45-year-old immunosuppressed man is summarized as a patient who developed ataxia, ophthalmoplegia, and areflexia after upper respiratory infection symptoms began. A nasopharyngeal swab was positive for COVID-19 polymerase chain reaction. He progressed to acute hypoxemic and hypercapnic respiratory failure requiring intubation and rapidly developed tetraparesis. Magnetic resonance imaging of the spine was consistent with Guillain Barre Syndrome. However, the clinical symptoms, along with positive anti-GQ1B antibodies, were consistent with Miller Fisher Syndrome and Guillain Barre Syndrome overlap. The patient required tracheostomy and had limited improvement in his significant neurological symptoms after several months.ConclusionsThe case demonstrates the severe neurological implications, prolonged recovery and implications in the concomitant respiratory failure of COVID-19 patients with neurological symptoms on the spectrum of disorders of Guillain Barre Syndrome.

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33

Suroshe M., Boinwad V., and Gogate V. "MANAGEMENT OF GUILLAIN BARRE SYNDROME THROUGH AYURVEDA – A CASE STUDY." International Journal Of Indian Medicine 04, no. 06 (2023): 09–18. http://dx.doi.org/10.55552/ijim.2023.4602.

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Guillain Barre Syndrome is acute paralysis neuropathy. It is evolving reflexes motor paralysis with or without sensory disturbances. Characterised by rapidly developing motor weakness. It is autoimmune in nature and trigger by preceding infections. A 47year female patient was came in OPD of kayachikitsa in our institute with complaining of generalised body weakness, tingling sensation in both hands and legs with bilateral exaggerated reflexes and decrease muscle tone and power. Patient had history of Guillain Barre Syndrome 4 years ago. For this she had taken allopathic medicine like Intra Venous Immunoglobulin in government hospital. Patient started complaint about same symptoms, for this she started Ayurvedic treatment. In Ayurveda Guillain Barre Syndrome is not explain by name hence it is taken under the name of vata vyadhi like sarvangagat vata. While thoroughly understanding Guillain Barre Syndrome through modern as well as Ayurvedic aspect Guillain Barre Syndrome considered as sarvangagata vata vyadhi and managed by Ayurvedic treatment on the basis of basic principles, and found better improvement in this case. Due to Ayurvedic treatment and internal medicine with 15 days with follows up treatment for 1 months gives significant relief in all motor and sensory reflex with increase muscle tone and power

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34

Archit, Dahiya, and Nandy Parvati. "A Case of Recurrent Guillain-Barre Syndrome." Indian Journal of Emergency Medicine 2, no. 1 (2016): 45–46. http://dx.doi.org/10.21088/ijem.2395.311x.2116.9.

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35

Khanal, Sushmita, and Sunil Babu Khanal. "Guillain-Barre Syndrome Following Ad26.Cov2.S Covid-19 Vaccine: A Case Report." Journal of Nobel Medical College 10, no. 2 (December 31, 2021): 63–64. http://dx.doi.org/10.3126/jonmc.v10i2.41790.

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Guillain-Barre Syndrome has been reported as a rare side effect in the recipients of Johnson & Johnson®'s Janssen Ad26.COV2.S COVID-19 vaccine. We report a case of Guillain-Barre Syndrome in a 50 years old female following the administration of the aforementioned vaccine.

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36

Bhattarai, Rupak, Sabina Lamichhane, and Chitta Ranjan Das. "Lower Segment Cesarean Section for Guillain- Barre Syndrome - A Novel Anaesthetic Technique." Birat Journal of Health Sciences 6, no. 1 (June 13, 2021): 1346–51. http://dx.doi.org/10.3126/bjhs.v6i1.37628.

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Introduction: Guillain Barre Syndrome in pregnant ladies, undergoing cesarean section, has not yet reported in Nepal. Anaesthetic management of 15 patients with Guillain Barre Syndrome who underwent lower segment cesarean section at Nobel Medical College Teaching Hospital is reported here. Objective: The purpose of this study is to assess the benefits of Rectus sheath nerve block along with infiltration of retropubic space of Retzius in Guillain Barre syndrome patients planned for lower segment cesarean section (LSCS). Methodology: During the period from 1st August 2015 to 31st April 2020 at NMCTH, a retrospective descriptive analysis of 15 pregnant ladies with Guillain Barre Syndrome who underwent lower segment cesarean section under Rectus sheath nerve block along with Retro pubic space of Retzius, infiltration and visceral peritoneum infiltration is discussed. Result: Considering Surgeons opinion about the operating conditions like Relaxation, Straining, Coughing, Bucking, Satisfactory to good operative conditions were reported. Excellent to good satisfaction was expressed by 39.9% of patients, 60% patients reported satisfactory. Hypotension and Arrhythmias was seen in 2 patients. Diaphoresis was seen in 1 patient. Fetomaternal outcome was good. There was no mortality. Conclusion: Rectus sheath block along with infiltration of retro pubic space of Retzius block can be considered as a good alternative to general anaesthesia or neuraxial block incase where general anesthesia & neuraxial block is risky or contraindicated for lower segment cesarean section in patients with Guillain Barre Syndrome.

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37

Sabaruddin, Hermin, Pribakti Budinurdjaja, and Fakhrurrazy Fakhrurrazy. "Guillain Barre Syndrome pada Kehamilan." Jurnal Ilmiah Kedokteran Wijaya Kusuma 9, no. 2 (October 1, 2020): 256. http://dx.doi.org/10.30742/jikw.v9i2.702.

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Guillain-Barre Syndrome (GBS) is a clinical syndrome characterized by the presence of the complete flaksid that occurs in acute. GBS associated with autoimmune reaction that affect peripheral nerve, radix, and cranial nerve. The incidence of GBS is 1 – 2 per 100,000 people/year. The incident was followed by increased age and the increasing population of obstetrics. GBS in pregnancy ranged from 13% in the first trimester, 47% in the second trimester, and 40% in the third trimester. In this case report reported Mrs. M 27 years old with a diagnosis of G2P1A0 h. 39-40 weeks + insimanation + living single fetal Presentation Head + Inpartu kala II + GBS + Failed + Vacuum Severily Underweight (BMI = 17) + TBJ 3000 Gr. Diagnosis of GBS are enforced based on anamnesis, physical examination and complementary examinations. From a previous illness history found anamnesis the weakness of limbs beginning in 2016. A history of the use of breathing apparatus and admitted tot the ICU in the first pregnancy. Mrs. M had a history of infections before being diagnosed with GBS. On this second pregnancy patients cannot move lower extremity but upper extermity is still functioning. Physical examination result of mothers and babies in the normal range even though found in conditions of severily BMI underweight. The patient finally decided to SC (section caesaria) and applied the IUD intracaesarean GBS in pregnancy is a coincidental. GBS is rarely aggravate pregnancy, but if not quickly identified and handled can enhance the high morbidity in both mother and fetus. In acute attacks (AIDP) in pregnant women with GBS increase stress on the mother or the fetus. The stress that occurs can also stimulate the immune system to produce prostaglandins, resulting in premature birth. Patients can give birth when the gestational age is still 7 months. It was different in the second pregnancy in this case where the patient was diagnosed with chronic inflammatory demyelinating polyradiculopathy (CIDP) so that GBS did not affect the mother and the fetus.

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38

Evans, O. B. "Guillain-Barre Syndrome in Children." Pediatrics in Review 8, no. 3 (September 1, 1986): 69–74. http://dx.doi.org/10.1542/pir.8-3-69.

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39

김동휘. "Electrodiagnosis of Guillain-Barre Syndrome." Jouranl of Korean Association of EMG Electrodiagnostic Medicine 16, no. 1 (June 2014): 14–19. http://dx.doi.org/10.18214/jkaem.2014.16.1.14.

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40

Millichap, J. Gordon. "Guillain-Barre Syndrome: Age Variations." Pediatric Neurology Briefs 5, no. 8 (August 1, 1991): 59. http://dx.doi.org/10.15844/pedneurbriefs-5-8-3.

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41

Duncan, S., R. G. Will, and J. Catnach. "Mumps and Guillain-Barre syndrome." Journal of Neurology, Neurosurgery & Psychiatry 53, no. 8 (August 1, 1990): 709. http://dx.doi.org/10.1136/jnnp.53.8.709.

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42

Miralles, F., J. Montero, R. Rene, and J. A. Martinez Matos. "Pure sensory Guillain-Barre syndrome." Journal of Neurology, Neurosurgery & Psychiatry 55, no. 5 (May 1, 1992): 411–12. http://dx.doi.org/10.1136/jnnp.55.5.411-a.

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43

Warner, T. T., S. Mossman, and N. M. Murray. "Hypokalaemia mimicking Guillain-Barre syndrome." Journal of Neurology, Neurosurgery & Psychiatry 56, no. 10 (October 1, 1993): 1134–35. http://dx.doi.org/10.1136/jnnp.56.10.1134.

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44

Hughes, R. "BOOK REVIEWS: Guillain-Barre Syndrome." Journal of Neurology, Neurosurgery & Psychiatry 57, no. 1 (January 1, 1994): 127. http://dx.doi.org/10.1136/jnnp.57.1.127-a.

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45

Preston, D. C., and J. J. Kelly. ""Pseudospasticity" in Guillain-Barre syndrome." Neurology 41, no. 1 (January 1, 1991): 131. http://dx.doi.org/10.1212/wnl.41.1.131.

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46

Perry, J. R. "MRI in Guillain-Barre syndrome." Neurology 45, no. 5 (May 1, 1995): 1024. http://dx.doi.org/10.1212/wnl.45.5.1024-b.

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47

Briscoe, D. M., J. B. McMenamin, and N. V. O'Donohoe. "Prognosis in Guillain-Barre syndrome." Archives of Disease in Childhood 62, no. 7 (July 1, 1987): 733–35. http://dx.doi.org/10.1136/adc.62.7.733.

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48

Bae, J. S., N. Yuki, S. Kuwabara, J. K. Kim, S. Vucic, C. S. Lin, and M. C. Kiernan. "Guillain-Barre syndrome in Asia." Journal of Neurology, Neurosurgery & Psychiatry 85, no. 8 (December 19, 2013): 907–13. http://dx.doi.org/10.1136/jnnp-2013-306212.

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49

Winer, J. B. "PROGNOSIS IN GUILLAIN-BARRE SYNDROME." Pediatric Infectious Disease Journal 4, no. 6 (November 1985): 709. http://dx.doi.org/10.1097/00006454-198511000-00047.

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50

Abdulaziz, Ammar Taha Abdullah, Dong Zhou, and Jin Mei Li. "Hydrocephalus in Guillain barre syndrome." Medicine 99, no. 16 (April 2020): e18638. http://dx.doi.org/10.1097/md.0000000000018638.

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