Relevant bibliographies by topics / Guillain-Barré

Academic literature on the topic 'Guillain-Barré'

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Published: 4 June 2021
Last updated: 19 February 2022

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Journal articles on the topic "Guillain-Barré"

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Cibulčík, František. "Guillain-Barré polyradiculoneuritis." Neurologie pro praxi 17, no. 1 (February 1, 2016): 11–15. http://dx.doi.org/10.36290/neu.2016.003.

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Hledíková, Andrea, Lukáš Hruban, René Jura, Igor Sas, Ondřej Hrdý, and Petr Janků. "Guillain-Barré syndrome in pregnancy." Česká gynekologie 86, no. 3 (June 24, 2021): 189–93. http://dx.doi.org/10.48095/cccg2021189.

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Summary Objective: A case report of a 23-year-old pregnant woman diagnosed with Guillain-Barré syndrome in the 31st week of pregnancy. Case report: We present a case study of a patient in the 31st week of pregnancy hospitalized at the University Hospital in Brno for expressed bulbar syndrome, neck muscle weakness, paresthesia of the arms and medical history of diarrhea in the previous week. During hospitalization, there was a rapid progression of symptoms and respiratory failure, requiring orotracheal intubation. The diagnosis of Guillain-Barré syndrome was determined and intravenous immunoglobulin therapy was initiated. The pregnancy was terminated in the 32nd week of gestation based on the maternal indication after a completed lung maturation of the fetus. Conclusion: Guillain-Barré syndrome is a neurological disease that can rarely occur during pregnancy and puerperium. The syndrome presents a serious pregnancy complication with an uncertain prognosis and risk for both mother and fetus. If the syndrome is diagnosed in time and treated correctly, the prognosis is favorable despite the complicated course.
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Yuki, Nobuhiro, and Hans-Peter Hartung. "Guillain–Barré Syndrome." New England Journal of Medicine 366, no. 24 (June 14, 2012): 2294–304. http://dx.doi.org/10.1056/nejmra1114525.

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Wakerley, Benjamin R., and Nobuhiro Yuki. "Guillain–Barré syndrome." Expert Review of Neurotherapeutics 15, no. 8 (July 8, 2015): 847–49. http://dx.doi.org/10.1586/14737175.2015.1065732.

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Kankam, Cygethia Gayle, and Robert Sallis. "Guillain-Barré syndrome." Postgraduate Medicine 101, no. 3 (March 1997): 279–90. http://dx.doi.org/10.3810/pgm.1997.03.189.

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Chung, Aimee, and Melissa Deimling. "Guillain-Barré Syndrome." Pediatrics in Review 39, no. 1 (January 2018): 53–54. http://dx.doi.org/10.1542/pir.2017-0189.

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Crawford, Doreen, and Annette Dearmun. "Guillain-Barré syndrome." Nursing Children and Young People 29, no. 2 (March 6, 2017): 17. http://dx.doi.org/10.7748/ncyp.29.2.17.s20.

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Zia, Muhammad Ahmed, Yasser Masood, and Muhammad Kashif Salman. "GUILLAIN-BARRÉ SYNDROME;." Professional Medical Journal 25, no. 04 (April 8, 2018): 538–44. http://dx.doi.org/10.29309/tpmj/18.4320.

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Payus, Alvin O., Tan HUI Jan, and Azman A. Raymond. "Guillain–Barré syndrome." Clinical Medicine 20, no. 6 (November 2020): e281.1-e281. http://dx.doi.org/10.7861/clinmed.let.20.6.6.

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Pritchard, Jane. "Guillain-Barré syndrome." Clinical Medicine 10, no. 4 (August 2010): 399–401. http://dx.doi.org/10.7861/clinmedicine.10-4-399.

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Dissertations / Theses on the topic "Guillain-Barré"

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Phongsisay, Vongsavanh, and vongsavang@yahoo com au. "Campylobacter jejuni and the Guillain-Barré syndrome." RMIT University. Applied Sciences, 2006. http://adt.lib.rmit.edu.au/adt/public/adt-VIT20061221.100446.

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Campylobacter jejuni is an enteric bacterium that causes human gastroenteritis worldwide. Some C. jejuni strains exhibiting human ganglioside-like lipooligosaccharide (LOS) structures, such as GM1 ganglioside, can induce an autoimmune neuropathy of the peripheral nervous system known as the Guillain-Barré syndrome (GBS). This GBS-inducible determinant is encoded by a gene cluster, which shows a high degree of variation among C. jejuni strains. The experiments presented in this thesis were conducted to give a better insight into the LOS synthesis genes in relation to the pathophysiology of C. jejuni. Firstly, a C. jejuni strain without GM1-like molecules was shown to be able to take up large DNA fragments, including LOS synthesis genes, from a strain expressing GM1-like molecules and consequently be transformed into a number of potential GBS-inducible transformants, which exhibited a high degree of genetic and phenotypic diversity. The ability of C. jejuni to take up and integrate foreign DNA explains the genome plasticity observed in this pathogen. Secondly, while attempting to analyse transcription of the LOS gene cluster, neither published methods nor any commercially available kits for RNA isolation could produce DNA-free RNA from C. jejuni. Combinations of these methods were trialled and only the combination of RNAzolB, TURBO DNase treatment, and acid phenol extraction was able to produce DNA-free RNA. The RNA isolated from most C. jejuni strains showed different RNA patterns to that of other bacteria. In addition the RNA from C. jejuni seemed closely associated with DNA compaired to RNA from other organisms. This might be caused by species-specific DNA conformation or chromatin structure. Thirdly, bidirectional transcription was observed in the LOS gene cluster. Both DNA strands were transcribed but transcription of the non-coding strands was at a lower rate, and both sense and antisense transcripts of each LOS gene tested were responsive to acid stress. This unusual transcription might have a potential effect on the expression of the GBS-inducing determinant. Finally, one of the LOS genes, the htrB gene, was further analysed. It was shown that expression of the htrB gene affects morphology, viability, growth ability, and sensitivity to stress environments. These results showed that the LOS molecule of C. jejuni is involved in many processes and is an important molecule for survival.
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Mori, Izumi. "Syndrome de Guillain-Barré et perturbations immunitaires." Paris 6, 2008. http://www.theses.fr/2008PA066343.

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Le syndrome de Guillain–Barré (SGB) consiste en une atteinte auto–immune aiguë du système nerveux périphérique. L’infection déclenchant l’atteinte neurologique est souvent détectée, et la date du début de la maladie est connue. Notre hypothèse était que le profil de la réponse immunitaire pourrait influencer non seulement l’évolution naturelle de la maladie, mais également la sensibilité au traitement par immunoglobulines intraveineuses (IgIv). Nous proposons ici le premier biomarqueur de l’efficacité du traitement par IgIv au cours du SGB. Une forte proportion de cellules B matures circulantes permettrait de prédire une bonne réponse aux IgIv. Par ailleurs, les traitements par IgIv entraînent une impressionnante mobilisation de plasmocytes, associée à une récupération rapide. Pour finir, nous montrons que les patients les plus sévères et les moins sensibles aux IgIv présentaient plus volontiers une expansion de cellules T cytomégalovirus–spécifiques. Il découle donc de ces observations que l’immunomonitorage au cours du SGB pourrait renseigner non seulement sur la physiopathologie de cette affection, mais également sur sa sensibilité aux IgIv.
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Sampaio, Pedro Henrique Marte de Arruda. "Assimetrias no exame neurológico de crianças com síndrome de Guillain-Barré." Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/17/17161/tde-25042018-150422/.

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A Síndrome de Guillain-Barré (SGB) é uma neuropatia periférica inflamatória aguda que tem sido definida pelo achado ou história de tetraparesia flácida arreflexa ascendente. Apresentações atípicas podem ser mais frequentes do que tem sido referido na literatura, particularmente na faixa etária infantil. Objetivo: Avaliar dados epidemiológicos e a prevalência de assimetria no exame neurológico em crianças com SGB. Métodos: Foram revisados 40 prontuários de crianças de 0 a 15 anos de idade com o diagnóstico de SGB, atendidas entre janeiro de 2000 e agosto de 2016. Avaliouse a presença de assimetrias no exame neurológico na admissão hospitalar, os desfechos clínicos e as características demográficas e clinico-laboratoriais. Resultados: Dois pacientes apresentaram assimetria no exame neurológico na admissão hospitalar e três pacientes admitidos com tetraparesia simétrica apresentaram um quadro motor assimétrico antes da internação. Uma criança evoluiu para assimetria após ter sido admitida com quadro simétrico. Outros oito casos tinham fraqueza segmentar. A presença de assimetria motora ou fraqueza segmentar se correlacionou com a progressão estática dos sintomas (p=0,004) e observou-se uma tendência desses pacientes serem mais jovens, mas essa diferença não foi significativa (p=0,08). Onze pacientes apresentavam reflexos miotáticos preservados e um paciente exibia hiperreflexia na admissão hospitalar. A maioria dos pacientes foi admitida sem conseguir deambular e, na alta, a maioria deambulava com ou sem apoio. Cinco crianças necessitaram de suporte ventilatório e nenhuma foi a óbito. Conclusão: Uma proporção significativa dos pacientes apresentava quadro motor assimétrico ou segmentar e reflexos miotáticos preservados. Os resultados obtidos delineiam aspectos clínicos atípicos na SGB em crianças e podem ajudar na definição diagnóstica e instituição de tratamento precoce.
Guillain-Barré syndrome (GBS) is an acute, inflammatory, peripheral neuropathy that has been being defined as an ascending flaccid tetraparesis. Atypical presentations can be frequent, particularly in children, leading to greater challenges in the diagnosis. Objectives: To analyze the epidemiological data and the prevalence of motor asymmetries in the neurological examination of children with GBS. Methods: A total of 40 medical records were analyzed, of children aged 0 to 15 years old diagnosed with GBS, admitted from January 2000 to August 2016. We evaluated the presence of motor asymmetries at the hospital admission, the clinical outcomes and the demographic and clinic-laboratorial characteristics. Results: Two patients had motor asymmetries at hospital admission and three patients admitted with symmetric tetraparesis had an initial motor asymmetry before admission. One patient progressed to asymmetric tetraparesis after being initially admitted with symmetric weakness. Eight other cases had segmental weakness at admission. Motor asymmetry and segmental weakness correlated with a static progression of symptoms (p=0.004) and these patients tended to be younger, but this difference was not significant (p=0.08). Eleven patients had preserved deep tendon reflexes and one exhibited hyperreflexia at the hospital admission. Most patients were admitted on wheel-chair or bedridden, and at discharge the majority could walk with or without help. Five children required mechanical ventilation and no patient died. Conclusion: A significant proportion of patients had asymmetric or segmental weakness and preserved deep tendon reflexes. Those results show that the so-called atypical clinical findings in children with GBS are not uncommon, and needs to be kept in mind to allow an earlier diagnosis and treatment.
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Perez, Simone. "Fator neurotrófico ciliar e interleucina-6 na síndrome de Guillain-Barré." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2011. http://hdl.handle.net/10183/30980.

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A Síndrome de Guillain-Barré é uma polirradiculoneuropatia aguda imuno-mediada, clinicamente apresentando-se com envolvimento sensitivo e motor e curso progressivo, em muitos casos determinando grande incapacidade e morbimortalidade nos pacientes que a desenvolvem. Este projeto de pesquisa teve como objetivo o seguimento de uma coorte de 22 indivíduos portadores da Síndrome de Guillain-Barré admitidos no Hospital de Clínicas de Porto Alegre durante o período de Janeiro de 2008 a Dezembro de 2009 para a correlação dos achados clínicos com os níveis liquóricos de interleucina-6 (IL-6) e do fator neurotrófico ciliar do nervo (CNTF). Na admissão, foi coletado o líquido cefalorraquidiano dos pacientes para dosagem de IL-6 e CNTF e, após seis meses, os pacientes foram estratificados em dois grupos, de acordo com a escala de Hughes: bom e mau prognóstico. Não foi identificada associação entre esses níveis e os achados clínicos. Mesmo assim, acreditamos que o estudo dessas substâncias pode ajudar a esclarecer a fisiopatologia da Síndrome de Guillain-Barré.
The Guillain-Barré syndrome (GBS) is an acute polyradiculoneuropathy, which is usually immune-mediated. It is characterized by clinical features associated with a progressive motor and sensory involvement, leading often to major disability and morbidity in affected individuals. In this work, we investigate the correlation between clinical findings and cerebrospinal fluid levels of interleukine-6 (IL-6) and of ciliary neurotrophic factor (CNTF) in a cohort of patients with GBS during the period January 2008 - December 2009 at the Hospital de Clínicas in Porto Alegre. Interleukine-6 is an immune modulator produced in the immune system with the function of B-cells’ stimulation and antibody secretion. The CNTF is produced in the CNS and plays an important role in the survival of some types of neurons. In this regard, the clinical and prognostic correlation with cerebrospinal fluid may help to elucidate the physiopathology of the Guillain-Barré syndrome.
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Maire, Olivier. "Avenir fonctionnel et rééducation du syndrome de Guillain et Barré d'évolution prolongée." Montpellier 1, 1991. http://www.theses.fr/1991MON11176.

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Fok, Nga-yin Angel, and 霍雅妍. "Influenza vaccination and its association with Guillain-barréSyndrome." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B45172043.

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Pontes, Tainá Madeira Barros. "Identificação etiológica de dengue em indivíduos com suspeita clínica da síndrome de Guillain Barré." Universidade de Fortaleza, 2017. http://dspace.unifor.br/handle/tede/108656.

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Made available in DSpace on 2019-03-30T00:18:21Z (GMT). No. of bitstreams: 0 Previous issue date: 2017-09-27
Guillain-Barré Syndrome is a polyneuropathy characterized by weakness, often started in an acute way, which bilaterally affects the limbs in a symmetrical manner, although there are some cases with asymmetry. In two-thirds of the patients, the start of the symptoms is preceded by an infectious disease of the upper respiratory or gastrointestinal tract. In literature, there are reports showing that the dengue virus is the causal agent of this syndrome; that way, this research was to identify a presence of the dengue virus as an etiological agent in participants suspected of Guillain-Barré Syndrome in the State of Ceará. The study was performed in a tertiary hospital in the period from April 2016 to March 2017 by means of an active search for participants with clinical suspicion of Guillain-Barré Syndrome. Data collection took place during the hospital stay of the individuals through structured interviews, analysis of medical charts and collection of serum and/or cerebrospinal fluid samples from the participants. In this study, serological diagnoses for dengue (IgG and IgM) and molecular diagnoses were accomplished by using the RT-PCR technique. Of the 23 individuals with clinical suspicion of Guillain-Barré Syndrome, 22 were analyzed according to the inclusion criteria of the research. Of these 22 participants, 11 were diagnosed with Guillain-Barré Syndrome. After performing an IgM and RT-PCR serological test for dengue, four individuals showed positive IgM for dengue, two of them with diagnosis of Guillain-Barré Syndrome and one of them showed positive RT-PCR. The individual who showed positivity for dengue through the RT-PCR method during hospital stay concomitantly with neurological symptoms evolved with severity of the clinical picture, and then died. Therefore, we conclude that, probably, the dengue virus infection seems to be present in a higher proportion than expected, thereby suggesting a possible neglect of this etiology in the investigation of the cases of Guillain-Barré Syndrome in the State of Ceará. KEYWORDS: Guillain-Barré Syndrome; Dengue fever.
A Síndrome de Guillain-Barré é uma polineuropatia que se caracteriza por fraqueza, geralmente, de início agudo, acometendo bilateralmente os membros de forma simétrica, embora alguns casos apresentem assimetria. Em dois terços dos pacientes, o início dos sintomas é precedido por uma doença infecciosa do trato respiratório superior ou gastrointestinal. Há relatos na literatura do vírus dengue ser o agente causador da síndrome, dessa forma, o objetivo desta pesquisa foi identificar a presença do vírus dengue como agente etiológico em participantes suspeitos da Síndrome de Guillain-Barré no Estado do Ceará. O estudo foi realizado em um hospital terciário, durante os meses de abril de 2016 a março de 2017, através de busca ativa de participantes com suspeita clínica da Síndrome de Guillain-Barré. A coleta de dados ocorreu durante a internação do indivíduo, através de entrevista estruturada, por meio de análise de prontuários e coleta de amostras de soro e/ou líquido cefalorraquidiano dos participantes. Neste estudo, foram realizados diagnóstico sorológicos para dengue (IgG e IgM) e diagnóstico molecular através da técnica de RT-PCR. Dos 23 indivíduos com suspeita clínica da Síndrome de Guillain-Barré, 22 foram analisados conforme os critérios de inclusão da pesquisa. Desses 22 participantes, 11 foram diagnosticados com a Síndrome de Guillain-Barré. Após realizar teste sorológico para dengue IgM e RT-PCR, 4 indivíduos apresentaram IgM positivo para dengue, 2 desses com diagnóstico da Síndrome de Guillain-Barré e um deles apresentou RT-PCR positivo. O indivíduo que apresentou positividade para dengue, através do método RT-PCR, durante o internamento hospitalar concomitante aos sintomas neurológicos, evoluiu com gravidade do quadro e óbito. Portanto, concluímos que, provavelmente a infecção pelo vírus dengue parece estar presente em uma proporção maior que esperada, sugerindo possível negligência desta etiologia na investigação dos casos de Síndrome de Guillain-Barré em nosso Estado. PALAVRAS-CHAVE: Síndrome de Guillain-Barré; Dengue.
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Torres, Vitor Félix. "Níveis séricos e liquóricos da proteína S100B, enolase específica do neurônio e neurotrofinas na síndrome de Guillain-Barré." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2010. http://hdl.handle.net/10183/26920.

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A Síndrome de Guillain-Barré é caracterizada por uma polirradiculoneuropatia de instalação aguda, usualmente imuno-mediada, com características clínicas associado a um envolvimento sensitivo e motor progressivo, muitas vezes determinando grande incapacidade e morbimortalidade nos indivíduos acometidos pela doença. Este projeto de pesquisa teve como objetivo o seguimento de uma coorte de indivíduos portadores da Síndrome de Guillain-Barré admitidos no Hospital de Clínicas de Porto Alegre durante o período de Janeiro de 2008 à Dezembro de 2009 para a correlação dos achados clínicos com os níveis plasmáticos e liquóricos da proteína S100B. A proteína S100B é basicamente encontrada nos astrócitos e tem tido grande importância para o melhor entendimento dos processos fisiopatológicos na injúria do Sistema nervoso central. Alguns autores relataram a correlação entre a Síndrome de Guillain-Barré e os níveis liquóricos da proteína S100B, baseado nestas descrições acreditamos que o melhor entendimento fisiopatológico da Síndrome de Guillain-Barré associado a determinação dos níveis plasmáticos e liquóricos da proteína S100B possam auxiliar e estabelecer uma importante ferramenta prognóstica precoce na determinação da gravidade dos portadores da Síndrome de Guillain-Barré.
The Guillain-Barre syndrome is characterized by a polyradiculoneuropathy of acute onset, usually immune-mediated, with clinical features associated with a progressive motor and sensory involvement, often determining major disability and morbidity in individuals affected by this disease. This research Project was aimed at following a cohort of patients with Guillain-Barre syndrome admitted to the Hospital de Clinicas de Porto Alegre during the period of January 2008 to December 2009. We measured the sera and cerebrospinal fluid (CSF) concentrations of S100B protein, neuron-specific enolase, neurotrophins and interleukin 6 using enzyme immunoassay methods in 22 patients with Guillain- Barre syndrome and 32 controls. The clinical and laboratory findings observed in the cases were important for better understanding of the pathophysiology of this disease coupled with the outcome of disabilities among the cases.
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Gries, Manuela [Verfasser], and Tobias [Akademischer Betreuer] Hartmann. "Die Bedeutung angeborener Immunrezeptoren beim experimentellen Guillain-Barré-Syndrom / Manuela Gries. Betreuer: Tobias Hartmann." Saarbrücken : Saarländische Universitäts- und Landesbibliothek, 2013. http://d-nb.info/1052904904/34.

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Greenshields, Kay. "Pathogenic potential of anti-ganglioside antibodies in a murine model of axonal Guillain-Barré syndrome." Thesis, University of Glasgow, 2007. http://theses.gla.ac.uk/3760/.

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Guillian-Barré Syndrome (GBS) is the world’s leading cause of neuromuscular paralysis occurring in serologically and pathogenically distinct forms. GBS is believed to have an autoimmune basis, where antibodies raised during antecedent infections (eg Campylobacter jejuni) cross-react with self antigens, exemplifying the process of molecular mimicry. These self-antigens are gangliosides, which are glycolipid structures enriched in peripheral nerve in specific membrane compartments termed lipid rafts. To date, successful murine models of anti-GD1a and anti-Gq1b ganglioside mediated neuropathy exist. Clinical evidence supports the involvement of anti-GM1 antibodies in nerve injury, however generation of anti-GM1 antibody mediated neuropathy models remain an enigma, and to date, the only successful model is based in Japanese rabbits. This thesis aims to address the controversies surrounding anti-GM1 antibody mediated neuropathy by utilising a panel of anti-GM1 antibodies of differing specificity, and explores how the stereometric interactions of GM1 with lipid raft species underpin the pathogenic potential of these antibodies.
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Books on the topic "Guillain-Barré"

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Hughes, Richard A. C. Guillain-Barré Syndrome. London: Springer London, 1990. http://dx.doi.org/10.1007/978-1-4471-3175-5.

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Meulstee, Jan. Electrodiagnostic studies in Guillain-Barré syndrome =: Elektrodiagnostisch onderzoek van het Guillain-Barré syndroom. Delft: Eburon, 1994.

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S, Steinberg Joel, ed. Guillain-Barré syndrome: From diagnosis to recovery. New York, N.Y: Demos, 2007.

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Quand une porte se ferme, une fenêtre s'ouvre: Autobiographie. [Alma, Québec]: Éditions SM, 2010.

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Handelsman, Harry. Apheresis in the treatment of Guillain-Barre syndrome. Rockville, MD: National Center for Health Services Research and Health Care Technology Assessment, U.S. Dept. of Health and Human Services, Public Health Service, 1985.

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Buzby, Jean C. Estimated annual costs of Campylobacter-associated Guillain-Barré syndrome. [Washington, DC]: U.S. Dept. of Agriculture, ERS, 1997.

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Wijdicks, Eelco F. M., 1954- and Truax Bradley T. 1948-, eds. Guillain-Barré syndrome. Philadelphia: F.A. Davis, 1991.

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Roe, Monica M. Thaw. Asheville, N.C: Front Street, 2008.

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Gerlach, Holly. Happily ever after: My journey with Guillain-Barré syndrome and how I got my life back. [Bloomington, Indiana]: Trafford Publishing, 2012.

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Guillain-Barré syndrome. London: Springer-Verlag, 1990.

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Book chapters on the topic "Guillain-Barré"

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Fary, Khan, and Louisa Ng. "Guillain-Barré Syndrome." In Encyclopedia of Clinical Neuropsychology, 1617–24. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_554.

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Bowes, Denise. "Guillain-Barré Syndrome." In When Doctors Get Sick, 53–58. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4899-2001-0_7.

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Mellits, E. David, and Guy M. McKhann. "Guillain-Barré: Plasmapheresis." In Foundations of Neurology, 225–47. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4613-1495-0_17.

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Kaida, Kenichi. "Guillain–Barré Syndrome." In Advances in Experimental Medicine and Biology, 323–31. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-32-9636-7_20.

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Vagts, Dierk A., Heike Kaltofen, Uta Emmig, and Peter Biro. "Guillain-Barré-Syndrom." In Anästhesie bei seltenen Erkrankungen, 1–3. Berlin, Heidelberg: Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-662-44368-2_107-1.

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Kotterba, Sylvia. "Guillain-Barré-Syndrom." In Springer Reference Medizin, 1–3. Berlin, Heidelberg: Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-642-54672-3_77-1.

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Rana, Abdul Qayyum, and John Anthony Morren. "Guillain-Barré Syndrome." In Neurological Emergencies in Clinical Practice, 47–53. London: Springer London, 2013. http://dx.doi.org/10.1007/978-1-4471-5191-3_6.

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Rinaldi, Simon. "Guillain-Barré syndrome." In Acute Medicine - A Practical Guide to the Management of Medical Emergencies, 5th Edition, 436–41. Chichester, UK: John Wiley & Sons, Ltd, 2017. http://dx.doi.org/10.1002/9781119389613.ch71.

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Scadding, John W. "Guillain-Barré Syndrome." In Encyclopedia of Pain, 1393–97. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-28753-4_1688.

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Khan, Fary, and N. G. Louisa. "Guillain–Barré Syndrome." In Encyclopedia of Clinical Neuropsychology, 1181–86. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-79948-3_554.

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Conference papers on the topic "Guillain-Barré"

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Rodriguez, W., and S. M. Alegre-Boschetti. "One in a Million: Guillain-Barré Syndrome." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a1775.

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de Quadros Martins, Vanessa, Jean Paulo Veronese de Souza, Guilherme Stüker, Roberta Casanova Wilhelms, Camila Schafer, Deise Marcela Piovasan, Bárbara Mendes da Silva, Marcelo Maltchik, Iloite Maria Scheibel, and Markus Bredemeier. "EOSINOPHILIC GRANULOMATOSIS WITH POLYANGIITIS MIMICKING GUILLAIN-BARRÉ SYNDROME." In Congresso Brasileiro de Reumatologia 2020. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2020.17201.

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Allgaier, J., M. A. Tidswell, and M. Ali. "Neurally Adjusted Ventilatory Assist: Exciting News for Guillain-Barré Syndrome." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a7527.

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Lourenço, Gustavo Roberto, Danielly Dantas Pimentel, Gizelle Gouvea Rezende, Iane Tamara Dondé, Taisa Morete da Silva, Viviane Queiroz de Oliveira Maia, Viviane Alves Costa, Juliana de Jesus Boscolo, and Luciana Akita. "GUILLAIN-BARRÉ SYNDROME AS A MANIFESTATION OF SYSTEMIC LUPUS ERYTHEMATOSUS." In Congresso Brasileiro de Reumatologia 2020. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2020.17397.

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Daspal, Ujjwal, Rekha Gupta, Mudit Garg, and Deepak Goel. "A007 An Unusual Presentation of Guillain-Barré Syndrome with Cervicobulbar Palsy." In 20th Annual Conference of Indian Society of Neuroanaesthesiology and Critical Care (ISNACC). Thieme Medical and Scientific Publishers Private Ltd., 2019. http://dx.doi.org/10.1055/s-0039-1684114.

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SANDRI, RAQUEL MARQUES, KETLIN BATISTA MORAIS MENDES, BÁRBARA SILVESTRE VICENTIM, THAÍS ROCKENBACH GAONA LENZI, GABRIELA MOREIRA FERLE, VANESSA ALMEIDA SANTOS, WALTER ALMEIDA AGUIAR JUNIOR, et al. "GUILLAIN-BARRÉ SYNDROME PRECEDING THE DIAGNOSIS OF SJÖGREN'S SYNDROME: A CASE REPORT." In 36º Congresso Brasileiro de Reumatologia. São Paulo: Editora Blucher, 2019. http://dx.doi.org/10.5151/sbr2019-114.

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Cathcart, Alexandra E., Sarah Harney, and Robin McKinney. "Hiding Behind a Cough: Guillain-Barré Syndrome Masquerading as Community-Acquired Pneumonia." In AAP National Conference & Exhibition Meeting Abstracts. American Academy of Pediatrics, 2021. http://dx.doi.org/10.1542/peds.147.3_meetingabstract.868.

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Barbisan, C. W., P. L. Dutra, S. S. Kretzer, R. K. Dal Molin, J. A. Victorino, and E. C. Gomes. "Síndrome De Guillain-Barré Abrindo Quadro De Lúpus Eritematoso Sistêmico: Relato De Caso." In II Congresso Brasileiro de Medicina Hospitalar. São Paulo: Editora Edgard Blücher, 2014. http://dx.doi.org/10.5151/medpro-ii-cbmh-044.

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Sergio, Albiol-Pérez, Forcano-García Mercedes, Muñoz-Tomás Mª Teresa, Manzano-Hernández Pilar, Solsona-Hernández Sofía, and Gil-Gómez José-Antonio. "Development of a novel therapy in virtual motor rehabilitation after severe Guillain-Barré symptomatology." In ICTs for improving Patients Rehabilitation Research Techniques. IEEE, 2013. http://dx.doi.org/10.4108/icst.pervasivehealth.2013.252190.

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Ríos, F. J., and B. V. Rivero. "Relationship Between Diaphragmatic Excursion and Maximum Inspiratory Pressure (MIP) in Adults Patients with Guillain Barré Syndrome." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a5792.

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Reports on the topic "Guillain-Barré"

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Khalfaoui, S. Guillain Barré syndrome complicated by hip osteoarthropathy. Science Repository OU, April 2019. http://dx.doi.org/10.31487/j.ssg.2018.01.02.

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