Relevant bibliographies by topics / Growth hormone

Academic literature on the topic 'Growth hormone'

Author: Grafiati
Published: 4 June 2021
Last updated: 29 July 2024

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Journal articles on the topic "Growth hormone"

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Diamanti-Kandarakis, Evanthia, Dimitrios Tsilakis, Stefanos Lazarides, Helen Kandarakis, and Angeliki Bergele. "Hormones in sports: growth hormone abuse." HORMONES 3, no. 1 (January 15, 2004): 37–45. http://dx.doi.org/10.14310/horm.2002.11108.

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Makras, Polyzois, Dimitris Papadogias, Grigoris Kaltsas, Nikolaos Kaklas, and Georgios Piaditis. "Growth without growth hormone (GH): A case report." HORMONES 3, no. 4 (October 15, 2004): 259–65. http://dx.doi.org/10.14310/horm.2002.11135.

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Smith, P. J., and C. G. Brook. "Growth hormone releasing hormone or growth hormone treatment in growth hormone insufficiency?" Archives of Disease in Childhood 63, no. 6 (June 1, 1988): 629–34. http://dx.doi.org/10.1136/adc.63.6.629.

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Lippman, Marc E., and Robert B. Dickson. "Growth control of normal and malignant breast epithelium." Proceedings of the Royal Society of Edinburgh. Section B. Biological Sciences 95 (1989): 89–106. http://dx.doi.org/10.1017/s0269727000010587.

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SynopsisWe review information highlighting the multiple roles of both steroidal (primarily oestrogen) and polypeptide regulators of mammary epithelial cell growth, emphasising the work of our laboratory. Effects of both classes of hormones are complex and involve multiple interactions with non-tumour, host tissue. Oestrogen may induce growth regulatory polypeptide growth factors and interact with them in hormone dependent breast cancer. Progression of hormone-dependent breast cancer to hormone independence may involve multiple genetic mechanisms of oncogene activation, loss of the oestrogen receptor, or loss of hormone responsivity of other gene products. Initial carcinogenesis and progression of mammary epithelium to cancer probably also requires both proliferative stimuli (oestrogen, polypeptide growth factors) and genetic damage, leading to qualitatively different hormonal responses (hormone responsive cancer). Future therapies should be designed to block hormonal stimulation better and to interfere with necessary activated or induced components of malignant progression such as oncogenes or polypeptide growth factors receptor systems.
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Vance, M. L. "Growth-hormone-releasing hormone." Clinical Chemistry 36, no. 3 (March 1, 1990): 415–20. http://dx.doi.org/10.1093/clinchem/36.3.415.

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Abstract Growth-hormone-releasing hormone (GHRH, somatoliberin) is the hypothalamic peptide hormone that specifically stimulates synthesis and release of growth hormone (GH, somatotropin) by somatotrope cells of the anterior pituitary gland. GHRH is the last of the classically postulated hypothalamic hormones to be characterized, synthesized, and used in clinical medicine. In this review of GHRH, I discuss the discovery and characterization of the peptide, its role in the regulation of GH secretion, and its clinical use in pathological states of GH excess and GH deficiency. The two most clinically useful aspects of GHRH are to establish the etiology of GH deficiency, most commonly the result of a hypothalamic GHRH deficiency, and to treat GH-deficient children. Use of GHRH as therapy for GH deficiency currently is experimental and, to date, results encourage the idea of a therapeutic role for this peptide in promoting endogenous GH secretion with resulting acceleration of linear growth.
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Belgorosky, A., A. Martinez, J. J. Heinrich, and M. A. Rivarola. "Lack of correlation of serum estradiol with growth velocity during male pubertal growth." Acta Endocrinologica 120, no. 5 (May 1989): 579–83. http://dx.doi.org/10.1530/acta.0.1200579.

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Abstract. The adolescent growth spurt in boys is under hormonal control. It is accepted that androgens and growth hormone contribute to male pubertal growth, but the role of estrogens is uncertain even though low-dose estradiol administration stimulates growth in prepubertal boys. In the present work, the correlation of serum testosterone and serum estradiol with growth velocity was studied in 16 pubertal normal boys. The study included correlations of growth velocity with serum nonsex hormone-binding globulin-bound testosterone and with serum nonsex hormone-binding globulin-bound estradiol, which are parameters of serum bioavailable sex hormones. A statistically significant positive correlation was found between serum testosterone and growth velocity but not between serum estradiol and growth velocity. These findings are against the hypothesis that estrogens play a growth promoting role during male puberty.
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Clapp, Carmen, Stéphanie Thebault, Michael C. Jeziorski, and Gonzalo Martínez De La Escalera. "Peptide Hormone Regulation of Angiogenesis." Physiological Reviews 89, no. 4 (October 2009): 1177–215. http://dx.doi.org/10.1152/physrev.00024.2009.

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It is now apparent that regulation of blood vessel growth contributes to the classical actions of hormones on development, growth, and reproduction. Endothelial cells are ideally positioned to respond to hormones, which act in concert with locally produced chemical mediators to regulate their growth, motility, function, and survival. Hormones affect angiogenesis either directly through actions on endothelial cells or indirectly by regulating proangiogenic factors like vascular endothelial growth factor. Importantly, the local microenvironment of endothelial cells can determine the outcome of hormone action on angiogenesis. Members of the growth hormone/prolactin/placental lactogen, the renin-angiotensin, and the kallikrein-kinin systems that exert stimulatory effects on angiogenesis can acquire antiangiogenic properties after undergoing proteolytic cleavage. In view of the opposing effects of hormonal fragments and precursor molecules, the regulation of the proteases responsible for specific protein cleavage represents an efficient mechanism for balancing angiogenesis. This review presents an overview of the actions on angiogenesis of the above-mentioned peptide hormonal families and addresses how specific proteolysis alters the final outcome of these actions in the context of health and disease.
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Avsar, O., S. Sancak, I. Koroglu, and E. Avcı. "Growth hormone, growth hormone receptor and insulin-like growth factor serum levels in patients with obesity and food addiction." Ukrainian Biochemical Journal 93, no. 6 (December 20, 2021): 70–75. http://dx.doi.org/10.15407/ubj93.06.070.

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Richalet, Jean-Paul, Murielle Letournel, and Jean-Claude Souberbielle. "Effects of high-altitude hypoxia on the hormonal response to hypothalamic factors." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 299, no. 6 (December 2010): R1685—R1692. http://dx.doi.org/10.1152/ajpregu.00484.2010.

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Acute and chronic exposure to high altitude induces various physiological changes, including activation or inhibition of various hormonal systems. In response to activation processes, a desensitization of several pathways has been described, especially in the adrenergic system. In the present study, we aimed to assess whether the hypophyseal hormones are also subjected to a hypoxia-induced decrease in their response to hypothalamic factors. Basal levels of hormones and the responses of TSH, thyroid hormones, prolactin, sex hormones, and growth hormone to the injection of TRH, gonadotropin-releasing hormone, and growth hormone-releasing hormone (GHRH) were studied in eight men in normoxia and on prolonged exposure (3–4 days) to an altitude of 4,350 m. Thyroid hormones were elevated at altitude (+16 to +21%), while TSH levels were unchanged, and follicle-stimulating hormone and prolactin decreased, while leutinizing hormone was unchanged. Norepinephrine and cortisol levels were elevated, while no change was observed in levels of epinephrine, dopamine, growth hormone (GH), IGF-1, and IGFBP-3. The mean response to hypothalamic factors was similar in both altitudes for all studied hormones, although total T4 was lower in hypoxia during 45 to 60 min after injection. The effect of hypoxia on the hypophyseal response to hypothalamic factors was similar among subjects, except for the GH response to GHRH administration. We conclude that prolonged exposure to high-altitude hypoxia induces contrasted changes in hormonal levels, but the hypophyseal response to hypothalamic factors does not appear to be blunted.
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Krukowska-Andrzejczyk, Barbara, Maria Kalina, and Barbara Kalina-Faska. "Effects of Treatment with Recombinant Growth Hormone in Children with Transient Partial Growth Hormone Deficiency – preliminary report." Pediatric Endocrinology 12, no. 1 (2013): 29–36. http://dx.doi.org/10.18544/ep-01.12.01.1438.

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Dissertations / Theses on the topic "Growth hormone"

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陳蒓 and Tzun Rachel Chan. "Growth hormone therapy for growth hormone deficiency." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2001. http://hub.hku.hk/bib/B31970308.

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Chan, Tzun Rachel. "Growth hormone therapy for growth hormone deficiency." Hong Kong : University of Hong Kong, 2001. http://sunzi.lib.hku.hk/hkuto/record.jsp?B22926288.

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Holmes, Sarah Jane. "Growth hormone replacement in adults with growth hormone deficiency." Thesis, Imperial College London, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.297239.

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Mahajan, Tripti. "Diagnosis of growth hormone deficiency and study of the effects of growth hormone treatment in growth hormone deficient adults." Thesis, University of Bristol, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.247557.

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Ehrnborg, Christer. "Growth hormone in athletes /." Göteborg : Department of Internal Medicine, Institute of Medicine, The Sahlgrenska Academy at Göteborg University, 2007. http://hdl.handle.net/2077/4709.

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Van, Koesveld Marika J. "Potential applications of growth hormone-releasing peptide-6 as a growth promotant." Thesis, The University of Sydney, 2003. https://hdl.handle.net/2123/28158.

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Consumer demand for high quality, lean meat has driven pork producers to seek new methods to maximise lean meat production and minimise fat deposition. Manipulation of the somatotropic axis of an animal has been identified as an effective method to increase growth efficiency and decrease adipose tissue deposition. The potential of H-His-D-Trp-Ala-Trp-D-Phe—Lys-NHZ (GHRP-6) to improve grth was evaluated by two methods, as a neonatal imprinting agent, and as an orally active in—feed additive via conjugation to vitamin B12. Neonatal imprinting with once-daily injections of GHRP-6 to piglets for three days postnatally resulted in an 8% increase in slaughter weight when animals were fed a high protein specification diet, compared to control animals fed a standard commercial diet. The increase in growth rate appears to be due to stimulation of feed intake, although the exact mechanism causing this effect is unknown at this stage. Feed efficiency and carcass composition did not differ from control animals. Further refinement of this technology may make it a highly desirable and cost-effective method to enhance growth rate and allow animals to attain a marketable liveweight in a shorter period of time.
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Gleeson, Helena. "Understanding growth hormone sensitivity and responsiveness: factors affecting IGF-1 response to growth hormone." Thesis, University of Manchester, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.493445.

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In endocrinology dynamic testing forms an essential part of investigation. To assess the central capacity of the GH-lGF-1 axis there is extensive experience in paediatric and adult populations of numerous physiological and pharmacological tests. However tests to assess the peripheral capacity of the GH-IGF-1 axis beyond a simple baseline lGF-1 level are limited to the IGF-1 generation test (IGFGT).
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Betley, Stephen. "Regulation of hepatocyte function by insulin, growth hormone and thyroid hormones." Thesis, University of Newcastle Upon Tyne, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.287415.

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Johnson, Christopher Derek Martin. "Growth hormone in chick embryogenesis." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp01/MQ28952.pdf.

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Edén, Engström Britt. "Growth Hormone and Gender. Studies in Healthy Adults and in Patients with Growth Hormone Disorders." Doctoral thesis, Uppsala universitet, Institutionen för medicinska vetenskaper, 2001. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-1262.

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The use of a new, more sensitive immunoassay for growth hormone (GH) revealed that the serum levels in men were lower than expected in sera drawn ambulatory in the morning after an overnight fast and that the gender difference was more than 10 times greater than reported. These observations led to a more thorough study on the impact of gender and sex steroids on the levels of GH and other hormones in ambulatory morning samples and over a 24-hour period. Furthermore, the impact of gender was studied in GH deficient (GHD) patients and healthy young adults treated with GH, and in patients with acromegaly treated with octreotide. An 80-fold gender difference in the morning GH levels was observed in young individuals as a reaction to ambulation, with decreased levels in men and increased in women. Oral contraceptives (OCs) given to women further increased the morning GH levels. During the day, higher outputs of epinephrine and lower levels of GH were seen in the men, while no gender differences were seen at night. The gender difference in morning GH levels decreased with age due to opposite changes in men and women. Administration of 17β-estradiol (E2) via subcutaneous implants in postmenopausal women, which increased the E2-concentrations to luteal phase levels, had no effect on the morning GH levels, indicating that the different reactions to ambulation do not appear to result from a direct sex steroid effect alone. Short-term administration of GH to young, healthy adults resulted in larger effects on insulin-like growth factor I (IGF-I) and other key metabolic parameters in men than in women. The smallest response was noted in women taking OCs. The clinical studies involving long-term GH treatment of patients with GHD demonstrate a gender difference in GH responsiveness, with women being less sensitive than men, a fact which should have a therapeutic impact in patients with GH disorders. A further gender difference of therapeutic importance was observed in men and women with acromegaly. Long-term treatment with a slow-release formulation of octreotide resulted in higher IGF-I levels in the men, despite equal doses of the drug and similar levels of GH.
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Books on the topic "Growth hormone"

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Bengtsson, Bengt-Åke, ed. Growth Hormone. Boston, MA: Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-5163-8.

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G, Scanes C., and Daughaday William H. 1918-, eds. Growth hormone. Boca Raton: CRC Press, 1995.

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Bengt-Ake, Bengtsson, ed. Growth hormone. Norwell, Mass: Kluwer Academic Publishers, 1999.

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Smith, Roy G., and Michael O. Thorner. Human Growth Hormone. New Jersey: Humana Press, 2000. http://dx.doi.org/10.1385/1592590152.

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Cohen, Laurie E., ed. Growth Hormone Deficiency. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-28038-7.

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Raiti, Salvatore, and Robert A. Tolman, eds. Human Growth Hormone. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4615-7201-5.

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Bercu, Barry B., and Richard F. Walker, eds. Growth Hormone Secretagogues. New York, NY: Springer New York, 1996. http://dx.doi.org/10.1007/978-1-4612-2396-2.

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Bercu, Barry B., and Richard F. Walker, eds. Growth Hormone II. New York, NY: Springer New York, 1994. http://dx.doi.org/10.1007/978-1-4613-8372-7.

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Smith, Roy G., and Michael O. Thorner, eds. Human Growth Hormone. Totowa, NJ: Humana Press, 2000. http://dx.doi.org/10.1007/978-1-59259-015-5.

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M, Ross R. J., and Savage M. O, eds. Growth hormone resistance. London: Baillière Tindall, 1996.

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Book chapters on the topic "Growth hormone"

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Rose, Susan R. "Growth Hormone Deficiency: Growth Hormone Tests and Growth Hormone Measurements." In Growth Hormone Therapy in Pediatrics - 20 Years of KIGS, 38–46. Basel: KARGER, 2007. http://dx.doi.org/10.1159/000101521.

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Levesque, Roger J. R. "Growth Hormone." In Encyclopedia of Adolescence, 1240–41. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-1695-2_552.

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Chapman, Ian M., and Michael O. Thorner. "Growth Hormone." In Diseases of the Pituitary, 79–112. Totowa, NJ: Humana Press, 1997. http://dx.doi.org/10.1007/978-1-4612-3954-3_5.

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Kannan, C. R. "Growth Hormone." In The Pituitary Gland, 25–56. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-1849-1_2.

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Wondisford, Fredric E. "Growth Hormone." In Essentials of Endocrinology and Metabolism, 229–40. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-39572-8_26.

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Seth, John. "Growth Hormone." In The Immunoassay Kit Directory, 159–78. Dordrecht: Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-011-1414-1_25.

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Mjaaland, M., K. Unneberg, and A. Revhaug. "Growth Hormone." In Acute Catabolic State, 269–82. Berlin, Heidelberg: Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-48801-6_24.

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Weltman, Arthur. "Growth Hormone." In Hormone Use and Abuse by Athletes, 89–98. Boston, MA: Springer US, 2010. http://dx.doi.org/10.1007/978-1-4419-7014-5_10.

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Bidlingmaier, Martin, and Christian J. Strasburger. "Growth Hormone." In Handbook of Experimental Pharmacology, 187–200. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-79088-4_8.

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Levesque, Roger J. R. "Growth Hormone." In Encyclopedia of Adolescence, 1–2. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-32132-5_552-2.

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Conference papers on the topic "Growth hormone"

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PAUL, Corina, Camelia EPURE, and Iulian VELEA. "P58 Growth hormone therapy for total growth hormone deficiency after complete therapy for parameningeal rhabdomyosarcoma." In 8th Europaediatrics Congress jointly held with, The 13th National Congress of Romanian Pediatrics Society, 7–10 June 2017, Palace of Parliament, Romania, Paediatrics building bridges across Europe. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2017. http://dx.doi.org/10.1136/archdischild-2017-313273.146.

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Lucas, Rudolf, Supruya Sridhar, Boris Gorshkov, Richard White, Nagavedi S. Umapathy, Evgeny Zemskov, Guang Yang, et al. "Growth Hormone Releasing Hormone Agonist Protects From Pneumolysin-Induced Pulmonary Permeability." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a3664.

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Burnside, D., and V. Puthi. "G232(P) Recombinant human growth hormone- should access grow?" In Royal College of Paediatrics and Child Health, Abstracts of the Annual Conference, 13–15 March 2018, SEC, Glasgow, Children First – Ethics, Morality and Advocacy in Childhood, The Journal of the Royal College of Paediatrics and Child Health. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2018. http://dx.doi.org/10.1136/archdischild-2018-rcpch.226.

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Avula, A., N. Narula, S. Toom, S. Ngu, D. Sharma, and M. N. Chalhoub. "Growth Hormone Injections Causing Mysterious Mediastinal Mass." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a3916.

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Catanuto, P., X. Xia, S. Pereira-Simon, A. Schally, S. Elliot, and M. K. Glassberg Csete. "MIA602 Downregulates Bleomycin-Induced Fibrosis Mediated by Growth Hormone-Releasing Hormone Receptor Activation." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a2225.

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Joy, R., V. Mathew, S. Jose, and S. Gupta. "G172(P) Improvement in motor function after growth hormone replacement in children with growth hormone deficiency and developmental delay." In Royal College of Paediatrics and Child Health, Abstracts of the Annual Conference, 24–26 May 2017, ICC, Birmingham. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2017. http://dx.doi.org/10.1136/archdischild-2017-313087.171.

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Đokovic, Radojica, Marko Cincovic, Vladimir Kurćubic, Milun D. Petrovic, Miloš Ži Petrovic, Ljiljana Anđušic, and Biljana Anđelic. "HOMEORETSKA REGULACUJA METABOLIČKIH FUNKCIJA KOD KRAVA U PERIPARTALNOM PERIODU." In SAVETOVANJE o biotehnologiji sa međunarodnim učešćem. University of Kragujevac, Faculty of Agronomy, 2021. http://dx.doi.org/10.46793/sbt26.235dj.

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The aim of this paper is to describe complex homeoretic and homeostatic mechanisms in dairy cows during the peripartum period. The endocrine system has a key function in regulating the adaptation of metabolism during the peripartum period. Homeoresis represents the functioning of the endocrine system and metabolism in conditions when the organism must primarily provide certain physiological processes, such as fetal growth or lactation. Then the function of all tissues is adjusted to the new situation. Homeoretic hormones (growth hormone, prolactin, glucocorticosteroids, thyroid hormones, insulin, glucagon and leptin) in dairy cows in the peripartum period play a key role in maintaining high lactation and maintaining cow health.
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Jackson, R. M., T. Cui, M. Wangpaichitr, W. Sha, and A. V. Schally. "Growth Hormone Releasing Hormone Receptor (GHRH-R) Mediates Alveolar Epithelial Type 2 Cell Inflammation." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a4379.

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Milton, Camille K., Panayiotis E. Pelargos, and Ian F. Dunn. "Cavernous Sinus invasion by Growth-Hormone Secreting Pituitary Adenomas." In Special Virtual Symposium of the North American Skull Base Society. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1725512.

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Magfirah, Irma, Soebagijo Adi Soelistijo, Hermina Novida, and Deasy Ardiany. "Effect of Growth Hormone Deficiency on the Cardiovascular System." In Surabaya International Physiology Seminar. SCITEPRESS - Science and Technology Publications, 2017. http://dx.doi.org/10.5220/0007338903420348.

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Reports on the topic "Growth hormone"

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Zeitler, Philip S. The Physiology of Growth Hormone-Releasing Hormone (GHRH) in Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, June 2001. http://dx.doi.org/10.21236/ada396677.

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Zeitler, Philip S. The Physiology of Growth Hormone-Releasing Hormone (GHRH) in Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, June 2002. http://dx.doi.org/10.21236/ada407126.

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Zeitler, Philip S. The Physiology of Growth Hormone-Releasing Hormone (GHRH) in Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, June 2003. http://dx.doi.org/10.21236/ada418139.

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Wu, Wei, Ningyi Song, Yue Zhao, Caiqi Du, Yuning Zhao, You Wu, and Xiaoping Luo. Evidence mapping of metabolic changes in children with growth hormone deficiency (GHD) treated with growth hormone. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2024. http://dx.doi.org/10.37766/inplasy2024.5.0064.

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Swanson, Steven M. Role of Growth Hormone in Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, February 2007. http://dx.doi.org/10.21236/ada467973.

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Ecker, Joseph Robert. Epigenetic Regulation of Hormone-dependent Plant Growth Processes. Office of Scientific and Technical Information (OSTI), November 2016. http://dx.doi.org/10.2172/1332760.

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Morrison, Tiffany. The Regulation of Insulin-like Growth Factor 1 by Growth Hormone via Stat5b. Portland State University Library, January 2012. http://dx.doi.org/10.15760/honors.14.

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Savaldi-Goldstein, Sigal, and Todd C. Mockler. Precise Mapping of Growth Hormone Effects by Cell-Specific Gene Activation Response. United States Department of Agriculture, December 2012. http://dx.doi.org/10.32747/2012.7699849.bard.

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Plant yield largely depends on a complex interplay and feedback mechanisms of distinct hormonal pathways. Over the past decade great progress has been made in elucidating the global molecular mechanisms by which each hormone is produced and perceived. However, our knowledge of how interactions between hormonal pathways are spatially and temporally regulated remains rudimentary. For example, we have demonstrated that although the BR receptor BRI1 is widely expressed, the perception of BRs in epidermal cells is sufficient to control whole-organ growth. Supported by additional recent works, it is apparent that hormones are acting in selected cells of the plant body to regulate organ growth, and furthermore, that local cell-cell communication is an important mechanism. In this proposal our goals were to identify the global profile of translated genes in response to BR stimulation and depletion in specific tissues in Arabidopsis; determine the spatio-temporal dependency of BR response on auxin transport and signaling and construct an interactive public website that will provide an integrated analysis of the data set. Our technology incorporated cell-specific polysome isolation and sequencing using the Solexa technology. In the first aim, we generated and confirmed the specificity of novel transgenic lines expressing tagged ribosomal protein in various cell types in the Arabidopsis primary root. We next crossed these lines to lines with targeted expression of BRI1 in the bri1 background. All lines were treated with BRs for two time points. The RNA-seq of their corresponding immunopurified polysomal RNA is nearly completed and the bioinformatic analysis of the data set will be completed this year. Followed, we will construct an interactive public website (our third aim). In the second aim we started revealing how spatio-temporalBR activity impinges on auxin transport in the Arabidopsis primary root. We discovered the unexpected role of BRs in controlling the expression of specific auxin efflux carriers, post-transcriptionally (Hacham et al, 2012). We also showed that this regulation depends on the specific expression of BRI1 in the epidermis. This complex and long term effect of BRs on auxin transport led us to focus on high resolution analysis of the BR signaling per se. Taking together, our ongoing collaboration and synergistic expertise (hormone action and plant development (IL) and whole-genome scale data analysis (US)) enabled the establishment of a powerful system that will tell us how distinct cell types respond to local and systemic BR signal. BR research is of special agriculture importance since BR application and BR genetic modification have been shown to significantly increase crop yield and to play an important role in plant thermotolerance. Hence, our integrated dataset is valuable for improving crop traits without unwanted impairment of unrelated pathways, for example, establishing semi-dwarf stature to allow increased yield in high planting density, inducing erect leaves for better light capture and consequent biomass increase and plant resistance to abiotic stresses.
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9

Cho, Sang-Joon, Jin-Sook Lee, Eric D. Mathias, Sender Lkhagvadorj, Lloyd L. Anderson, Ching Chang, and Gerard J. Hickey. Central and Peripheral Administration of Growth Hormone Secretagogue L-692-585, Somatostatin, Neuropeptide Y and Galanin in Pig: Dose-dependent Effects on Growth Hormone Secretion. Ames (Iowa): Iowa State University, January 2011. http://dx.doi.org/10.31274/ans_air-180814-840.

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10

Quinn, Dana. The effect of developmental temperature on morphology, energy metabolism, growth hormone and thyroid stimulating hormone in Long-Evans rats. Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.2830.

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