Relevant bibliographies by topics / Group B streptococcus;human ureaplasmas

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Academic literature on the topic 'Group B streptococcus;human ureaplasmas'

Author: Grafiati
Published: 4 June 2021
Last updated: 20 February 2023

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Journal articles on the topic "Group B streptococcus;human ureaplasmas"

1

Estrada-Gutierrez, Guadalupe, Nardhy Gomez-Lopez, Veronica Zaga-Clavellina, Silvia Giono-Cerezo, Aurora Espejel-Nuñez, Marco Antonio Gonzalez-Jimenez, Salvador Espino y Sosa, David M. Olson, and Felipe Vadillo-Ortega. "Interaction between Pathogenic Bacteria and Intrauterine Leukocytes Triggers Alternative Molecular Signaling Cascades Leading to Labor in Women." Infection and Immunity 78, no. 11 (August 30, 2010): 4792–99. http://dx.doi.org/10.1128/iai.00522-10.

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ABSTRACT Increased risk of preterm labor has been linked to cervicovaginal infection with Ureaplasma urealyticum and group B streptococci. Although various experimental models have been developed to study the role of amniochorion infection in preterm labor, they typically exclude the initial interaction between intrauterine leukocytes (recruited from decidual vessels into the avascular fetal membranes) and infecting bacteria. In this work, we ascertained whether inflammatory molecules secreted by bacterium-activated intrauterine leukocytes stimulate the amniochorion production of mediators involved in human labor. Using a two-step process beginning with placental circulating leukocytes as a proxy for intrauterine leukocytes, we found that coincubation of amniochorion explants with plasma from placental whole blood preincubated with group B streptococci resulted in a significant increase in tumor necrosis factor alpha (TNF-α) and matrix metalloproteinase 9 (MMP-9) levels in tissue. Extensive changes in the connective tissue arrangement and a decrease in collagen content demonstrated the degradation of the extracellular matrix following this treatment. In contrast, plasma from blood preconditioned with U. urealyticum induced a highly significant secretion of interleukin-1β (IL-1β) and prostaglandin E2 (PGE2) by the amniochorion without changes in the extracellular matrix organization or content. These data demonstrate that group B streptococci induce degradation of the amniochorion as a result of MMP-9 production, probably via TNF-α, whereas U. urealyticum stimulates the secretion of PGE2, probably via IL-1β, potentially stimulating myometrial contraction. Our study provides novel evidence that the immunological cells circulating within the uterine microenvironment respond differentially to an infectious agent, triggering alternative molecular signaling pathways leading to human labor.
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Pyrohova, V. I., S. O. Shurpyak, Yu R. Fayta, M. Y. Malachinska, and N. M. Kuz. "Comparative study of the efficacy of topical therapy of mixed vaginitis associated with cervicitis by combined medications." HEALTH OF WOMAN, no. 6(132) (July 30, 2018): 42–49. http://dx.doi.org/10.15574/hw.2018.132.42.

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The objective: to increase the effectiveness of local therapy for recurrent nonspecific vaginitis associated with cervicitis in women of reproductive age on the basis of a comparative evaluation of combined drugs Terzhinan and Neo Penotran Forte. Materials and methods. A prospective, open comparative study included 56 women aged 27.5±2.8 years with recurrent nonspecific vaginitis and cervicitis who were randomized to the main group and comparison group. Patients of the main group (n=28) received the drug Terzhinan® (1 vaginal tablet in the evening, before bed, for 10 days). The comparison group included 28 patients who received Neo-Pentran Forte (1 vaginal suppository in the evening, before bedtime, for 10 days), one vaginal suppository containing 750 mg of metronidazole and 200 mg of miconazole nitrate. The complex clinical-paraclinical examination included the determination of the state of the vaginal microbiota using several methods in parallel: a bacterioscopy of vaginal smears stained by Gram, a bacteriological rapid method using AFGENITAL SYSTEM (Liofilchem®, Italy), real-time PCR (Florocenosis) with detection antigens of chlamydia, herpes simplex virus, human papillomavirus, trichomonads. Results. The main reason for the treatment of patients were abundant pathological discharge from the genital tract (73.2%), pruritus (37.5%) and burning (23.2%) in the vulva, pain during sexual intercourse (8.9%), while 33.9% of women expressed combined complaints. Attention was drawn to the significant frequency of dyshormonal pathology among women with recurrent cervico-vaginal infections. In the examined women, uterine leiomyoma was diagnosed (28.6%), genital endometriosis (19.6%), fibrocystic breast disease (37.5%), combined benign dyshormonal diseases of the genital organs (14.3%). About 21.4% of patients treatment of thyroid gland dysfunction (hypothyroidism). According to the comprehensive examination, in all patients of clinical groups, decompensated vaginal dysbiosis was diagnosed, which was manifested by a sharp decrease in the absence of Lactobacillus spp strains in 39.3% of patients and an increase in the number of isolated opportunistic and pathogenic microorganisms to 1011 CFU/ml with an increase in the number of microorganisms in microbial associations (from 2–3 to 5–6 conditionally pathogenic and pathogenic pathogens) in all the cases analyzed. When using the genital express system in vaginal contents, women of the main group identified Escherichia coli (17.9%), Pseudomonas spp. (10.7%), Gardnerella vaginalis (39.3%), Staphylococcus aureus (17 9%), Enterococcus faecalis (25.0%), Streptococcus Group B (10.7%), Candida spp. (46.4%), Mycoplasma spp./Ureaplasma ur. in the title > 105 (14.0%). In the comparison group, the spectrum of detected pathogenic and conditionally pathogenic microorganisms did not differ significantly from the data of the main group. 92.6% of patients in the main group had a pronounced positive clinical effect, and a positive microbiological effect was achieved in 96.4% of cases that persisted during the next two months of follow-up. Without additional prescription of antifungal agents, a positive effect was achieved in 84.6% of patients in the main group with mixed bacterial-candidiasis vaginitis at 54.5% in the comparison group. The independent recovery of the lactobacilli pool to a titer of 107–109 CFU/ml in 17.9% of patients with a lack of detection of lactobacilli before treatment. A similar effect was not observed in the comparison group. Сonclusion. In a comparative study of the results of the use of Terzhinan and Neo-Penotran Forte in monotherapy in patients with inflammatory diseases of the lower genitalia (nonspecific recurrent vaginitis and cervicitis), the high clinical and microbiological efficacy of Terzhinan has been demonstrated. Key words: mixed vaginitis, cervicitis, Terzhinan, Neo-Penotran Forte.
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Patras, Kathryn A., Philip A. Wescombe, Berenice Rösler, John D. Hale, John R. Tagg, and Kelly S. Doran. "Streptococcus salivarius K12 Limits Group B Streptococcus Vaginal Colonization." Infection and Immunity 83, no. 9 (June 15, 2015): 3438–44. http://dx.doi.org/10.1128/iai.00409-15.

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Streptococcus agalactiae(group B streptococcus [GBS]) colonizes the rectovaginal tract in 20% to 30% of women and during pregnancy can be transmitted to the newborn, causing severe invasive disease. Current routine screening and antibiotic prophylaxis have fallen short of complete prevention of GBS transmission, and GBS remains a leading cause of neonatal infection. We have investigated the ability ofStreptococcus salivarius, a predominant member of the native human oral microbiota, to control GBS colonization. Comparison of the antibacterial activities of multipleS. salivariusstrains by use of a deferred-antagonism test showed thatS. salivariusstrain K12 exhibited the broadest spectrum of activity against GBS. K12 effectively inhibited all GBS strains tested, including disease-implicated isolates from newborns and colonizing isolates from the vaginal tract of pregnant women. Inhibition was dependent on the presence of megaplasmid pSsal-K12, which encodes the bacteriocins salivaricin A and salivaricin B; however, in coculture experiments, GBS growth was impeded by K12 independently of the megaplasmid. We also demonstrated that K12 adheres to and invades human vaginal epithelial cells at levels comparable to GBS. Inhibitory activity of K12 was examinedin vivousing a mouse model of GBS vaginal colonization. Mice colonized with GBS were treated vaginally with K12. K12 administration significantly reduced GBS vaginal colonization in comparison to nontreated controls, and this effect was partially dependent on the K12 megaplasmid. Our results suggest that K12 may have potential as a preventative therapy to control GBS vaginal colonization and thereby prevent its transmission to the neonate during pregnancy.
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Anthony, B. F., N. F. Concepcion, and K. F. Concepcion. "Human Antibody to the Group-Specific Polysaccharide of Group B Streptococcus." Journal of Infectious Diseases 151, no. 2 (February 1, 1985): 221–26. http://dx.doi.org/10.1093/infdis/151.2.221.

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Kasper, Dennis L., Michael R. Wessels, Hilde-Kari Guttormsen, Lawrence C. Paoletti, Morven S. Edwards, and Carol J. Baker. "Measurement of Human Antibodies to Type III Group B Streptococcus." Infection and Immunity 67, no. 8 (August 1999): 4303–5. http://dx.doi.org/10.1128/iai.67.8.4303-4305.1999.

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Andreas, Nicholas J., Asmaa Al-Khalidi, Mustapha Jaiteh, Edward Clarke, Matthew J. Hyde, Neena Modi, Elaine Holmes, Beate Kampmann, and Kirsty Mehring Le Doare. "Role of human milk oligosaccharides in Group B Streptococcus colonisation." Clinical & Translational Immunology 5, no. 8 (August 26, 2016): e99. http://dx.doi.org/10.1038/cti.2016.43.

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MOYO, SYLVESTER R., JOHAN A. MAELAND, and KÅRE BERGH. "Typing of human isolates of Streptococcus agalactiae (group B streptococcus, GBS) strains from Zimbabwe." Journal of Medical Microbiology 51, no. 7 (July 1, 2002): 595–662. http://dx.doi.org/10.1099/0022-1317-51-7-595.

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Tsai, Pei-Jane, Yee-Shin Lin, Chih-Feng Kuo, Huan-Yao Lei, and Jiunn-Jong Wu. "Group A Streptococcus Induces Apoptosis in Human Epithelial Cells." Infection and Immunity 67, no. 9 (September 1, 1999): 4334–39. http://dx.doi.org/10.1128/iai.67.9.4334-4339.1999.

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ABSTRACT Internalization of group A streptococcus (GAS) by epithelial cells may have a role in causing invasive diseases. The purpose of this study was to examine the fate of GAS-infected epithelial cells. GAS has the ability to invade A-549 and HEp-2 cells. Both A-549 and HEp-2 cells were killed by infection with GAS. Epithelial cell death mediated by GAS was at least in part through apoptosis, as shown by changes in cellular morphology, DNA fragmentation laddering, and propidium iodide staining for hypodiploid cells. A total of 20% of A-549 cells and 11 to 13% of HEp-2 cells underwent apoptosis after 20 h of GAS infection, whereas only 1 to 2% of these cells exhibited spontaneous apoptosis. We further examined whether streptococcal pyrogenic exotoxin B (SPE B), a cysteine protease produced by GAS, was involved in the apoptosis of epithelial cells. The speB isogenic mutants had less ability to induce cell death than wild-type strains. When A-549 cells were cocultured with the mutant and SPE B for 2 h, the percentage of apoptotic cells did not increase although the number of intracellular bacteria increased to the level of wild-type strains. In addition, apoptosis was blocked by cytochalasin D treatment, which interfered with cytoskeleton function. The caspase inhibitors Z-VAD.FMK, Ac-YVAD.CMK, and Ac-DEVD.FMK inhibited GAS-induced apoptosis. These results demonstrate for the first time that GAS induces apoptosis of epithelial cells and internalization is required for apoptosis. The caspase pathway is involved in GAS-induced apoptosis, and the expression of SPE B in the cells enhances apoptosis.
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Liu, Yuxin, and Jinhui Liu. "Group B Streptococcus: Virulence Factors and Pathogenic Mechanism." Microorganisms 10, no. 12 (December 15, 2022): 2483. http://dx.doi.org/10.3390/microorganisms10122483.

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Group B Streptococcus (GBS) or Streptococcus agalactiae is a major cause of neonatal mortality. When colonizing the lower genital tract of pregnant women, GBS may cause premature birth and stillbirth. If transmitted to the newborn, it may result in life-threatening illnesses, including sepsis, meningitis, and pneumonia. Moreover, through continuous evolution, GBS can use its original structure and unique factors to greatly improve its survival rate in the human body. This review discusses the key virulence factors that facilitate GBS invasion and colonization and their action mechanisms. A comprehensive understanding of the role of virulence factors in GBS infection is crucial to develop better treatment options and screen potential candidate molecules for the development of the vaccine.
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Lione, Viviane de Oliveira Freitas, Michelle Hanthequeste Bittencourt dos Santos, Jessica Silva Santos de Oliveira, Ana Luiza Mattos-Guaraldi, and Prescilla Emy Nagao. "Interferon-γ inhibits group B Streptococcus survival within human endothelial cells." Memórias do Instituto Oswaldo Cruz 109, no. 7 (November 2014): 940–43. http://dx.doi.org/10.1590/0074-0276140201.

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Dissertations / Theses on the topic "Group B streptococcus;human ureaplasmas"

1

Kong, Fanrong. "Integrated study of group B streptococcus and human ureaplasmas : the paradigm shifts." Thesis, The University of Sydney, 2004. http://hdl.handle.net/2123/592.

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Group B streptococcus (GBS, S. agalactiae) and human ureaplasmas (U. parvumand U. urealyticum) are two clinically and phylogenetically related, potential perinatal pathogens. Their relationships between genotypes and pathogenesis of GBS and ureaplasma infection were still not well understood, one of the reason is that both of them are still short of a very practical genotyping system. In the study, to solve the above problem we developed genotyping systems for the organisms (the second section). For human ureaplasmas, based on four genes/gene clusters (rRNAgene clusters, the elongation factor Tu genes, urease gene complexes and multiplebanded antigen genes), we designed many primer pairs suitable for developing species identification assays for the two newly established human ureaplasma species (U. parvum and U. urealyticum). Further, based on the heterogeneity of ureaplasma multiple banded antigen gene (which contains species- and serovar-specific regions), we developed genotyping methods for each ureaplasma species.For GBS, based on three sets of molecular markers (capsular polysaccharidesynthesis gene clusters, surface protein antigen genes and mobile genetic elements),we developed a genotyping system. The primary evaluation of the genotyping systems showed that the genotyping systems were practical alternative assays for the conventional serotyping and they will be useful to further explore the relationships between genotypes and pathogenesis of GBS and ureaplasma infection. In the study, we introduced novel data and tools into GBS and ureaplasma studies especially from genomic- and bioinformatics-based molecular microbiology(the third section). For two newly established human ureaplasma species, based on the U. parvum serovar-3 genome, and using the above four important genes/geneclusters, we exposed some interesting problems in the understanding of newureaplasma taxonomy especially in the post genomic era. For GBS, we studied the two published full genomes and exposed some new problems or possible future new research fields. In particular we found the two finished and one ongoing GBS genomes were all non-typical and suggest that future genomic project had better have genetic population structure viewpoint. Finally, we suggested that integrated studies of the two potential or conditional perinatal pathogens, from the viewpoint of evolution, would provide a new understanding angle of the pathogenesis of the two organisms. Studies suggested that during coevolution, human ureaplasmas(especially U. parvum) became friendlier than their ancestors to their human host (by losing most of its virulence genes); however, GBS tried to increase its invasive abilities (by getting more virulence genes) to fight against the human host attack.
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Kong, Fanrong. "Integrated study of group B streptococcus and human ureaplasmas � the paradigm shifts." University of Sydney. Medicine, 2004. http://hdl.handle.net/2123/592.

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Group B streptococcus (GBS, S. agalactiae) and human ureaplasmas (U. parvumand U. urealyticum) are two clinically and phylogenetically related, potential perinatal pathogens. Their relationships between genotypes and pathogenesis of GBS and ureaplasma infection were still not well understood, one of the reason is that both of them are still short of a very practical genotyping system. In the study, to solve the above problem we developed genotyping systems for the organisms (the second section). For human ureaplasmas, based on four genes/gene clusters (rRNAgene clusters, the elongation factor Tu genes, urease gene complexes and multiplebanded antigen genes), we designed many primer pairs suitable for developing species identification assays for the two newly established human ureaplasma species (U. parvum and U. urealyticum). Further, based on the heterogeneity of ureaplasma multiple banded antigen gene (which contains species- and serovar-specific regions), we developed genotyping methods for each ureaplasma species.For GBS, based on three sets of molecular markers (capsular polysaccharidesynthesis gene clusters, surface protein antigen genes and mobile genetic elements),we developed a genotyping system. The primary evaluation of the genotyping systems showed that the genotyping systems were practical alternative assays for the conventional serotyping and they will be useful to further explore the relationships between genotypes and pathogenesis of GBS and ureaplasma infection. In the study, we introduced novel data and tools into GBS and ureaplasma studies especially from genomic- and bioinformatics-based molecular microbiology(the third section). For two newly established human ureaplasma species, based on the U. parvum serovar-3 genome, and using the above four important genes/geneclusters, we exposed some interesting problems in the understanding of newureaplasma taxonomy especially in the post genomic era. For GBS, we studied the two published full genomes and exposed some new problems or possible future new research fields. In particular we found the two finished and one ongoing GBS genomes were all non-typical and suggest that future genomic project had better have genetic population structure viewpoint. Finally, we suggested that integrated studies of the two potential or conditional perinatal pathogens, from the viewpoint of evolution, would provide a new understanding angle of the pathogenesis of the two organisms. Studies suggested that during coevolution, human ureaplasmas(especially U. parvum) became friendlier than their ancestors to their human host (by losing most of its virulence genes); however, GBS tried to increase its invasive abilities (by getting more virulence genes) to fight against the human host attack.
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Hull, James Richard. "The interactions of group B Streptococci with human fibronectin /." Thesis, Connect to this title online; UW restricted, 2007. http://hdl.handle.net/1773/9917.

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Ipe, Deepak Samuel. "Molecular Mechanisms of Group B Streptococcus Urinary Tract Infection and Adaptability to Growth in Human Urine." Thesis, Griffith University, 2015. http://hdl.handle.net/10072/366019.

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Bacteriuria, or the presence of bacteria in urine, is associated with both asymptomatic, as well as symptomatic urinary tract infection (UTI) and underpins much of the dynamic of microbial colonization of the urinary tract. The prevalence of bacteriuria in dissimilar patient groups such as healthy adults, institutionalized elderly, pregnant women, and immune-compromised patients varies widely. In addition, assessing the importance of ‘significant bacteriuria’ in infected individuals represents a diagnostic challenge, partly due to various causal microbes, and requires careful consideration of the distinct etiologies of bacteriuria in different populations and circumstances. Recent molecular discoveries have revealed how some bacterial traits can enable organisms to grow in human urine, which, as a fitness adaptation, is likely to influence the progression of bacteriuria in some individuals. This study was designed as a comprehensive analysis of asymptomatic bacteriuria (ABU) causal organisms in dissimilar populations, and an in-depth microbiological analysis of the mechanisms used by one such causal organism, Streptococcus agalactiae. This organism causes UTI including ABU; however, growth of S. agalactiae in human urine has not been reported. In the first part of this study, we evaluate the prevalence and etiology of bacteruria, and discuss recent advances in the molecular detection of bacteriuria from a diagnostic viewpoint.
Thesis (PhD Doctorate)
Doctor of Philosophy (PhD)
School of Medical Science
Griffith Health
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Alves, Joana Emanuela Vieira. "Immunobiological studies on two human pathogens: Group B - Streptococcus and Escherichia coli." Doctoral thesis, 2016. https://repositorio-aberto.up.pt/handle/10216/101800.

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Alves, Joana Emanuela Vieira. "Immunobiological studies on two human pathogens: Group B - Streptococcus and Escherichia coli." Tese, 2016. https://repositorio-aberto.up.pt/handle/10216/101800.

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Lopes, Inês Rodrigues. "Exploring the immunobiological basis of prospective strategies towards two human neonatal pathogens: Group B Streptococcus and Escherichia coli." Master's thesis, 2016. https://hdl.handle.net/10216/90975.

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Lopes, Inês Rodrigues. "Exploring the immunobiological basis of prospective strategies towards two human neonatal pathogens: Group B Streptococcus and Escherichia coli." Dissertação, 2016. https://hdl.handle.net/10216/90975.

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Yang, Q., M. Zhang, Dean J. Harrington, G. W. Black, and I. C. Sutcliffe. "A proteomic investigation of Streptococcus agalactiae reveals that human serum induces the C protein β antigen and arginine deiminase." 2011. http://hdl.handle.net/10454/11568.

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Streptococcus agalactiae is a major neonatal pathogen. Disease progression is characterised by bacterial adaptation from commensal maternal vaginal colonisation to environments associated with neonatal disease, including exposure to blood. To explore this adaptation in vitro, we have used proteomics to identify proteins differentially expressed following growth on Todd Hewitt agar in the presence or absence of 10% v/v human serum. Twelve differentially expressed proteins were identified. Notably, the C protein β antigen and arginine deiminase proteins were upregulated following growth in the presence of human serum, consistent with previous studies implicating these two proteins in the pathogenesis of S. agalactiae disease.
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Book chapters on the topic "Group B streptococcus;human ureaplasmas"

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Paoletti, Lawrence C., and Dennis L. Kasper. "Surface Structures of Group B Streptococcus Important in Human Immunity." In Gram-Positive Pathogens, 204–27. Washington, DC, USA: ASM Press, 2019. http://dx.doi.org/10.1128/9781683670131.ch13.

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Rapose, Alwyn. "Streptococcal Skin and Skin-Structure Infections." In Streptococcal Infections [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.102894.

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Infections attributable to Streptococcus are protean. These range from mild skin and soft tissue infections to life-threatening conditions like meningitis, endocarditis and toxic shock syndrome. In addition, streptococcal infection can be associated with noninfectious sequelae like rheumatic fever and post-streptococcal glomerulonephritis. There is a wide range of Streptococcus spp. causing human infections and different classifications of these organisms have been described, the most quoted being the Lancefield classification based on cell-wall antigens. Streptococci can be studied based on their species: S. pyogenes, S. pneumoniae, S. anginosus etc. or by the Lancefield classification group A, B, C, D etc. or by the clinical syndromes associated with these bacteria. This chapter will describe clinical syndromes associated with streptococcal skin and soft tissue infections ranging from mild: cellulitis and lymphangitis which can be treated in the out-patient setting, to more aggressive manifestations that require hospitalization (sepsis and toxic shock syndrome) and even surgery (necrotizing fasciitis, myositis and gangrene), It will also provide clues to clinical diagnosis as well as suggest recommendations for optimized management of these conditions.
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