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Relevant bibliographies by topics / Group B streptococcus

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Academic literature on the topic 'Group B streptococcus'

Author: Grafiati

Published: 4 June 2021

Last updated: 7 October 2023

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Journal articles on the topic "Group B streptococcus"

1

MacLean, A. B. "Group B streptococcus." Journal of Obstetrics and Gynaecology 31, no. 3 (March 18, 2011): 203. http://dx.doi.org/10.3109/01443615.2011.556760.

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GREENSPOON, JEFFREY S., JOHN GORDEN WILCOX, and THOMAS H. KIRSCHBAUM. "Group B Streptococcus." Obstetrical & Gynecological Survey 46, no. 8 (August 1991): 499–508. http://dx.doi.org/10.1097/00006254-199108000-00001.

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Davies, Hannah G., Clara Carreras-Abad, Kirsty Le Doare, and Paul T. Heath. "Group B Streptococcus." Pediatric Infectious Disease Journal 38 (June 2019): S72—S76. http://dx.doi.org/10.1097/inf.0000000000002328.

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Leclair, Catherine M., Ashley E. Hart, Martha F. Goetsch, Heather Carpentier, and Jeffrey T. Jensen. "Group B Streptococcus." Journal of Lower Genital Tract Disease 14, no. 3 (July 2010): 162–66. http://dx.doi.org/10.1097/lgt.0b013e3181d3d40f.

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Schuchat, Anne. "Group B streptococcus." Lancet 353, no. 9146 (January 1999): 51–56. http://dx.doi.org/10.1016/s0140-6736(98)07128-1.

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Simpson, A. J. H., S. R. Heard, and B. Keith English. "Group B streptococcus." Lancet 346, no. 8976 (September 1995): 700. http://dx.doi.org/10.1016/s0140-6736(95)92308-x.

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Dangor, Ziyaad, Sanjay G. Lala, Gaurav Kwatra, and Shabir A. Madhi. "Group B Streptococcus." Current Opinion in Infectious Diseases 29, no. 3 (June 2016): 262–67. http://dx.doi.org/10.1097/qco.0000000000000266.

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Easmon, Charles S. F. "Group B Streptococcus." Infection Control & Hospital Epidemiology 7, S2 (February 1986): 135–37. http://dx.doi.org/10.1017/s0195941700065681.

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Over the past 25 years group B streptococci have become established as one of the main bacterial pathogens of the neonate in Western Europe and the United States. The attack rate of 0.25/1,000 live births found by Mayon White in Great Britain1 appears typical of many European countries. However, in some centers in the United States attack rates can be over 10 times higher.Two types of neonatal group B streptococcus (GBS) diseases exist, “early” and “late” onset. Early onset disease usually presents within the first few days of life. Often the most serious infections are present at birth or seen within a few hours. Early onset disease presents with pneumonia, respiratory distress and shock. Bacteremia is normally present and meningitis may occur. Mortality is high (50% to 75%). The portal of entry is probably the respiratory tract. Infants normally acquire the infecting organism from their mothers. Heavy maternal and infant colonization, prolonged rupture of membranes, prematurity, and obstetric complications are all risk factors.Delayed onset disease, as its name suggests, presents after the first week of life, primarily with bacteremia and meningitis. Mortality is much lower than for the early onset form, but still appreciable for a bacterial infection (14% to 18%). Its epidemiology is uncertain.

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Woo, Patrick CY, Jade LL Teng, Kit-wah Leung, Susanna KP Lau, Herman Tse, Beatrice HL Wong, and Kwok-yung Yuen. "Streptococcus sinensis may react with Lancefield group F antiserum." Journal of Medical Microbiology 53, no. 11 (November 1, 2004): 1083–88. http://dx.doi.org/10.1099/jmm.0.45745-0.

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Lancefield group F streptococci have been found almost exclusively as members of the ‘Streptococcus milleri’ group, although they have been reported very occasionally in some other streptococcal species. Among 302 patients with bacteraemia caused by viridans streptococci over a 6-year period, three cases were caused by Streptococcus sinensis (type strain HKU4T, HKU5 and HKU6). All three patients had infective endocarditis complicating their underlying chronic rheumatic heart diseases. Gene sequencing showed no base differences between the 16S rRNA gene sequences of HKU5 and HKU6 and that of HKU4T. All three strains were Gram-positive, non-spore-forming cocci arranged in chains. All grew on sheep blood agar as α-haemolytic, grey colonies of 0.5–1 mm in diameter after 24 h incubation at 37 °C in ambient air. Lancefield grouping revealed that HKU5 and HKU6 were Lancefield group F, but HKU4T was non-groupable with Lancefield groups A, B, C, D, F or G antisera. HKU4T was identified by the Vitek system (GPI), API system (20 STREP) and ATB system (ID32 STREP) as 99 % Streptococcus intermedius, 51.3 % S. intermedius and 99.9 % Streptococcus anginosus, respectively. Using the same tests, HKU5 was identified as 87 % Streptococcus sanguinis/Streptococcus gordonii, 59 % Streptococcus salivarius and 99.6 % S. anginosus, respectively, and HKU6 as 87 % S. sanguinis/S. gordonii, 77 % Streptococcus pneumoniae and 98.3 % S. anginosus, respectively. The present data revealed that a proportion of Lancefield group F streptococci could be S. sinensis. Lancefield group F streptococci should not be automatically reported as ‘S. milleri'.

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Verrall, Rosemary. "The Streptococcus milleri Group." Infection Control 7, no. 11 (November 1986): 558–60. http://dx.doi.org/10.1017/s0195941700065334.

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Microbiologists traditionally have relied on biochemical and serologic tests to identify only group A, group B, and group D streptococci isolated from patients with serious infections. Until recently there has been little interest in the further classification of other streptococci isolated from clinical specimens.In 1956, Guthof repeatedly isolated a particular streptococcus from dental abscesses and named the organism Streptococcus milleri in honor of the microbiologist, W.D. Miller. In 1976, Parker and Ball associated this microorganism with other pyogenic infections and later described the characteristics of 346 strains. The Streptococcus milleri group is now recognized as both virulent and invasive and some believe it is responsible for more suppurative infections than any other group of streptococci. Physicians need to consider the unique ability of these bacteria to form abscesses in a wide range of tissue. Microbiology laboratories, therefore, need to develop schemes to identify this important group of streptococci.

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Dissertations / Theses on the topic "Group B streptococcus"

1

Smith, Jennifer Marie. "Characterization of host-bacteria interactions contributing to group B streptococcus colonization." Huntington, WV : [Marshall University Libraries], 2002. http://www.marshall.edu/etd/descript.asp?ref=64.

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Yang, Qian. "Proteomic investigation of the group B streptococcus." Thesis, Northumbria University, 2011. http://nrl.northumbria.ac.uk/2119/.

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The Group B Streptococcus (GBS) is a Gram-positive opportunistic pathogen which is a leading cause of neonatal disease globally. In 2000-2001, the general incidence of neonatal GBS infection was 0.72 per 1000 live births in U.K. and the mortality rate is about 10%, because of which neonatal GBS disease is a significant burden on society. GBS is part of the commensal flora, colonising the vagina and gastrointestinal tract of women. Vertical transmission is the main cause of early onset GBS disease. During the process of GBS neonatal disease, GBS must be able to survive in several very different host environments, including the vagina, amniotic fluid, the neonate's lung and blood. The vagina is normally acidic, low oxygen and with limited nutrients while the neonate's lung and blood are neutral, high oxygen and with abundant nutrient. Proteomic investigations of GBS protein expression under conditions representing those associated with benign maternal colonisation and foetal exposure may help us understand the molecular basis of GBS virulence. GBS growth characteristics, long term survival, acid adaptation, viable but non-culturable state and biofilm formation were investigated to help us understand how GBS survives in different environments and also help us to develop an in vitro model to reflect in vivo conditions during GBS disease development. An in vitro model of GBS growth under conditions reflecting maternal vaginal carriage (low pH, low oxygen, nutrient stress) and exposure to body fluids during invasive disease (neutral pH, aeration, nutrient sufficient) was established. Proteins expressed under each growth conditions were separated by two dimensional electrophoresis. Individual proteins were subjected to in-gel trypsin digestion and identified using liquid chromatography-mass spectrometry with peptide mass fingerprinting followed with bioinformatic research. A total of 76 proteins were identified and 16 of these were expressed differentially. The putative virulence factor C protein β antigen and proteins involved in responses to oxidative stress were up-regulated under the conditions reflecting neonatal exposure. Another in vitro model of GBS growth on Todd Hewitt agar in the presence or absence of 10% human serum was established and followed by proteomic investigation of proteins differentially expressed under these two conditions, as this model reflects GBS neonatal septicaemia (exposure to serum). A total of 84 proteins were identified and 11 of which were expressed differentially. The putative virulence factor C protein β antigen, arginine deiminase, an ABC transporter substrate-binding protein and glyceraldehyde-3-phosphate dehydrogenase were up-regulated in the presence of human serum.

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Jones, Nicola. "A molecular epidemiological investigation of group B streptococcus." Thesis, Open University, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.402623.

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A multilocus sequence typing (MLST) system for group B streptococcus (GBS) has been developed and validated on a global collection of human GBS strains isolated from carriage and invasive disease. A carriage study was performed, over a 3-year period, to establish the rate of carriage ofGBS in pregnant women in Oxford, UK. Invasive isolates were collected, prospectively and retrospectively over a similar time period. Twenty-one percent of women studied were asymptomatic carriers of GBS. The incidence of invasive GBS was 0.9/1000 live births in neonates and 6.11100,000 population >60 years. The population structure of GBS is best depicted. using MLST. as a network of related clusters indicating the presence of recomb inationa I events occurring in the population that interfere with a tree like branching structure of the population. A single hypervirulent clone ofGBS (ST-17 complex) is responsible for an excess of neonatal disease in Oxford (odds ratio 3.4). The possibility that a factor other than capsular type IIImay be responsible for virulence of this clonal complex in neonates is raised. Intriguingly this clonal complex was unique among human lineages in that it has emerged from bovine GBS. It was not however associated with increased invasiveness amongst adult (> 60 years). Further study ofthis hypervirulent clone of GBS is likely to contribute to the understanding of the pathogenesis of neonatal GBS disease.

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GIUSSANI, STEFANIA. "Mechanisms of Complement evasion in Group B Streptococcus." Doctoral thesis, Università degli studi di Pavia, 2019. http://hdl.handle.net/11571/1248526.

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GIUSSANI, STEFANIA. "Mechanisms of Complement evasion in Group B Streptococcus." Doctoral thesis, Università degli studi di Pavia, 2019. http://hdl.handle.net/11571/1263953.

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GIUSSANI, STEFANIA. "Mechanisms of Complement evasion in Group B Streptococcus." Doctoral thesis, Università degli studi di Pavia, 2019. http://hdl.handle.net/11571/1264013.

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GIUSSANI, STEFANIA. "Mechanisms of Complement evasion in Group B Streptococcus." Doctoral thesis, Università degli studi di Pavia, 2019. http://hdl.handle.net/11571/1263915.

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GIUSSANI, STEFANIA. "Mechanisms of Complement evasion in Group B Streptococcus." Doctoral thesis, Università degli studi di Pavia, 2019. http://hdl.handle.net/11571/1264035.

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GIUSSANI, STEFANIA. "Mechanisms of Complement evasion in Group B Streptococcus." Doctoral thesis, Università degli studi di Pavia, 2019. http://hdl.handle.net/11571/1248546.

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GIUSSANI, STEFANIA. "Mechanisms of Complement evasion in Group B Streptococcus." Doctoral thesis, Università degli studi di Pavia, 2019. http://hdl.handle.net/11571/1263975.

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Books on the topic "Group B streptococcus"

1

Verani, Jennifer R. Prevention of perinatal group B streptococcal disease: Revised guidelines from CDC, 2010. Atlanta, GA: Dept. of Health and Human Services, Centers for Disease Control and Prevention, 2010.

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2

Bai, Yu. A latent-active glycosylation approach for the synthesis of saccharides derived from the capsular polysaccharide of group B streptococcus type IA. Birmingham: University of Birmingham, 2000.

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Monoclonal antibody to the immunodominant lipopolysaccharide antigen of bacteroides fragilis cross-reacting with type II group B streptococci. Turku: Turun yliopisto, 1988.

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Le Doare, Kirsty, Christine E. Jones, and Paul T. Heath. Group B Streptococcus (Streptococcus agalactiae). Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190604813.003.0019.

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Group B Streptococcus (GBS) is a leading cause of early neonatal infection and neonatal mortality, with long-term adverse neurodevelopmental outcomes in up to 50% of survivors of GBS meningitis. GBS has a likely underappreciated role in causing preterm birth and stillbirth. GBS colonizes the vagina and gastrointestinal tract of the pregnant woman, and transmission to the infant occurs during or just before delivery. Although the majority of these infants do not develop invasive disease, maternal colonization is a prerequisite for early onset disease (0–6 days of life, most commonly associated with sepsis and respiratory distress) and a significant risk factor for late onset disease (7–89 days of life, most commonly associated with sepsis and meningitis). The introduction of intrapartum antibiotic prophylaxis has resulted in significant declines in the incidence of early onset disease but provides no protection against late onset disease.

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(Foreword), David Heymann, and I. Edward Alcamo (Editor), eds. Streptococcus (Group B) (Deadly Diseases and Epidemics). Chelsea House Publications, 2007.

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6

Food and Agriculture Organization of the United Nations. Group B Streptococcus -? Streptococcus Agalactiae Sequence Type 283 in Freshwater Fish: Risk Profile. Food & Agriculture Organization of the United Nations, 2022.

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Group B Streptococcus : Streptococcus Agalactiae Sequence Type 283 in Freshwater Fish: Risk Profile. Food & Agriculture Organization of the United Nations, 2022.

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Risk profile - Group B Streptococcus (GBS) – Streptococcus agalactiae sequence type (ST) 283 in freshwater fish. FAO, 2021. http://dx.doi.org/10.4060/cb5067en.

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Publications, ICON Health. The Official Patient's Sourcebook on Group B Streptococcus Infection: A Revised and Updated Directory for the Internet Age. ICON Health Publications, 2002.

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Fox, Grenville, Nicholas Hoque, and Timothy Watts. Infection. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198703952.003.0012.

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This chapter provides a problem-oriented approach to investigation and treatment of early and late onset neonatal bacterial infection, including group B streptococcus, meningitis, urine infection, conjunctivitis, umbilical sepsis, osteomyelitis, and septic arthritis. In addition, the prevention and management of congenital infection is covered, including hepatitis B and C, HIV, syphilis, CMV, toxoplasma, rubella, herpes simplex, and chickenpox. Other topics covered are infection control (including MRSA), fungal sepsis, TB, and an overview of immunizations in the first year of life.

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Book chapters on the topic "Group B streptococcus"

1

Hutto, Cecelia. "Group B Streptococcus." In Congenital and Perinatal Infections, 217–23. Totowa, NJ: Humana Press, 2006. http://dx.doi.org/10.1385/1-59259-965-6:217.

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Dinsmoor, Mara J. "Group B Streptococcus." In Protocols for High-Risk Pregnancies, 334–39. Oxford, UK: Wiley-Blackwell, 2010. http://dx.doi.org/10.1002/9781444323870.ch40.

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Thomas, Stephanie. "Group B Streptococcus." In Maternal-Fetal Evidence Based Guidelines, 387–92. 4th ed. Boca Raton: CRC Press, 2022. http://dx.doi.org/10.1201/9781003099062-39.

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Raabe, Vanessa N., and Andi L. Shane. "Group B Streptococcus (Streptococcus agalactiae)." In Gram-Positive Pathogens, 228–38. Washington, DC, USA: ASM Press, 2019. http://dx.doi.org/10.1128/9781683670131.ch14.

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Shabayek, Sarah. "Streptococcus agalactiae (Group B Streptococcus)." In Molecular Typing in Bacterial Infections, Volume I, 167–89. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-74018-4_8.

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Carlson, Laura, and M. Kathryn Menard. "37. Group B Streptococcus." In Maternal-Fetal Evidence Based Guidelines, 3e, 325–31. Taylor & Francis Group, 6000 Broken Sound Parkway NW, Suite 300, Boca Raton, FL 33487-2742: CRC Press, 2016. http://dx.doi.org/10.1201/9781315200910-38.

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Winn, Jessica, and Hung N. Winn. "Group B streptococcus infection." In Clinical Maternal-Fetal Medicine Online, 74.1–74.4. 2nd ed. London: CRC Press, 2021. http://dx.doi.org/10.1201/9781003222590-65.

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Nigam, Aruna, and Pragati Aggarwal. "Group B Streptococcus Infection." In Infections and Pregnancy, 355–65. Singapore: Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-7865-3_23.

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Young, Hugh, and Marie Ogilvie. "Streptococcus beta-haemolytic group B (Streptococcus agalactiae)." In Genitourinary Infections, 280–87. Dordrecht: Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-017-5080-6_11.

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Dinsmoor, Mara J. "Protocol 31: Group B Streptococcus." In Protocols for High-Risk Pregnancies, 251–59. Chichester, UK: John Wiley & Sons, Ltd, 2015. http://dx.doi.org/10.1002/9781119001256.ch31.

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Conference papers on the topic "Group B streptococcus"

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Liz, Catarina Ferraz, Sara Soares, and Abílio Oliveira. "GP267 Prevention of group B streptococcus infection- results from a screening program." In Faculty of Paediatrics of the Royal College of Physicians of Ireland, 9th Europaediatrics Congress, 13–15 June, Dublin, Ireland 2019. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2019. http://dx.doi.org/10.1136/archdischild-2019-epa.326.

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Ojo, SK, L. U. Udewena, and C. O. Adetunji. "Bioactivity of Bryophyllum pinnatum and Rauvolfia vomitoria on Neonatal Group B Streptococcus." In GA – 70th Annual Meeting 2022. Georg Thieme Verlag KG, 2022. http://dx.doi.org/10.1055/s-0042-1759124.

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Grama, Alina, Irina Pop, Georgia Tita, Ligia Blaga, Romana Vulturar, Alina Nicolescu, Calin Deleanu, and Tudor L. Pop. "P121 Galactosemia presented as a fulminant liver failure and group b streptococcus (GBS) sepsis." In 8th Europaediatrics Congress jointly held with, The 13th National Congress of Romanian Pediatrics Society, 7–10 June 2017, Palace of Parliament, Romania, Paediatrics building bridges across Europe. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2017. http://dx.doi.org/10.1136/archdischild-2017-313273.209.

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McCormack, Siobhan, Claire Thompson, Caitriona Ní Chathasaigh, Miriam Nolan, Mendinaro Imcha, Ann Dee, Jean Saunders, and Roy K. Philip. "GP254 Maternal awareness, acceptability and willingness towards group B streptococcus (GBS) vaccination during pregnancy." In Faculty of Paediatrics of the Royal College of Physicians of Ireland, 9th Europaediatrics Congress, 13–15 June, Dublin, Ireland 2019. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2019. http://dx.doi.org/10.1136/archdischild-2019-epa.313.

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Pumpure, E., I. Morozova, L. Lapidus, M. Koka, V. Veisa, D. Rezeberga, and S. Markova. "Positive Group B Streptococcus and the use of antimicrobial therapy in early neonatal period." In 62. Kongress der Deutschen Gesellschaft für Gynäkologie und Geburtshilfe – DGGG'18. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1671439.

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Ramos, Nicolle, Beatriz Silva, Maximiano Teixeira, Nicea Silva, Marco Henrique, and Ivano Filippis. "Molecular characterization of Streptococcus agalactiae group B (SGB) isolated from pregnant women in Rio de Janeiro." In International Symposium on Immunobiological. Instituto de Tecnologia em Imunobiológicos, 2021. http://dx.doi.org/10.35259/isi.2021_46635.

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CAMILA MIYOSHI, ISABELI, HELAINE MARIA BESTETI PIRES, FERNANDA S. CAVICHIOLLI, MARINA S. POLYDORO, and ADRIANE M. DELICIO. "Group B Streptococcus colonization in HIV infected pregnant women in the Hospital Professor Doutor José Aristodemo Pinotti between 2000 and 2015." In XXIV Congresso de Iniciação Científica da UNICAMP - 2016. Campinas - SP, Brazil: Galoa, 2016. http://dx.doi.org/10.19146/pibic-2016-52153.

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Seedat, F., J. Geppert, C. Stinton, J. Patterson, CS Brown, B. Tan, K. Freeman, et al. "OP34 Universal antenatal culture-based screening for maternal group b streptococcus (gbs) carriage to prevent early-onset gbs disease: a systematic review for the uk national screening committee (nsc)." In Society for Social Medicine, 61st Annual Scientific Meeting, University of Manchester, 5–8 September 2017. BMJ Publishing Group Ltd, 2017. http://dx.doi.org/10.1136/jech-2017-ssmabstracts.34.

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Bermúdez Martínez, MA, and L. Müller. "Intrapartum detection of Group B Streptococci (GBS) by point of care (POCT) real time PCR testing." In 62. Kongress der Deutschen Gesellschaft für Gynäkologie und Geburtshilfe – DGGG'18. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1671473.

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O’Sullivan, CP. "I3 Group b streptococcal (gbs) disease in uk and irish infants younger than 90 days, 2014–2015." In Royal College of Paediatrics and Child Health, Abstracts of the Annual Conference, 13–15 March 2018, SEC, Glasgow, Children First – Ethics, Morality and Advocacy in Childhood, The Journal of the Royal College of Paediatrics and Child Health. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2018. http://dx.doi.org/10.1136/archdischild-2018-rcpch.471.

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