Academic literature on the topic 'Grass grub'

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Journal articles on the topic "Grass grub"

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Mundy, D. C., P. A. Alspach, and J. Dufay. "Grass grub damage and mycorrhizal colonisation of grapevine rootstocks." New Zealand Plant Protection 58 (August 1, 2005): 234–38. http://dx.doi.org/10.30843/nzpp.2005.58.4279.

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Field observations and a grower survey during 2002/2003 indicated that grass grub larvae might be responsible for the death of young grape vines In November 2003 a pot trial was established to determine whether grass grab larvae feeding caused sufficient root damage to account for observed vine deaths The experiments also evaluated whether arbuscular mycorrhizal fungi (AMF) colonisation of grape vine roots was affected by grass grub feeding Grass grub damage was found on the belowground portion of the trunk and was proportional to the numbers of grubs present However root and shoot weight and shoot length were not affected by grub density when measured two months after grubs were introduced AMF colonization varied between the four rootstocks in the trial and was higher where grass grubs had been introduced Further research is required to elucidate the causes of young vine decline in Marlborough
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Townsend, R. J., J. E. Dunbar, and T. A. Jackson. "Grass grub distribution on the upper West Coast defined by soil sampling and pheromone trapping." New Zealand Plant Protection 66 (January 8, 2013): 376. http://dx.doi.org/10.30843/nzpp.2013.66.5681.

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The New Zealand grass grub (Costelytra zealandica) is distributed throughout the South Island but surprisingly has not been recorded west of Reefton In 2006 pasture damage from rootfeeding scarab larvae on the West Coast initially attributed to grass grub was found to be caused by manuka beetles Pyronota spp Winter surveys during 20082012 between Karamea and Hokitika confirmed that most damage patches were caused by manuka beetle larvae but there was a small localised population of C zealandica associated with Westport airport and golf course In 2012 a network of phenolbaited pheromone traps was established around this epicenter during the grass grub flight season with traps spaced at approximately 05 km intervals Traps within the identified zone of grass grub infestation caught 15 beetles per night Single male beetles were trapped up to 75 km from the epicenter but with no evidence of established populations from larval sampling It is likely that the localised grass grub population became established after an accidental introduction of insects with soil or plant material to the modified and drained airport and golf course environments and may act as an infestation source for other areas Pastures on the nearby newlyflipped land of Cape Foulwind may also be suitable for grass grub and should be regularly inspected to anticipate and prevent outbreaks
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Bughrara, Suleiman S., David R. Smitley, and David Cappaert. "European Chafer Grub Feeding on Warm-season and Cool-season Turfgrasses, Native Prairie Grasses, and Pennsylvania Sedge." HortTechnology 18, no. 3 (January 2008): 329–33. http://dx.doi.org/10.21273/horttech.18.3.329.

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Six grass species representing vegetative and seeded types of native, warm-season and cool-season grasses, and pennsylvania sedge (Carex pensylvanica) were evaluated in the greenhouse for resistance to root-feeding grubs of european chafer (Rhizotrogus majalis). Potted bermudagrass (Cynodon dactylon), buffalograss (Buchlöe dactyloides), zoysiagrass (Zoysia japonica), indiangrass (Sorghastrum nutans), little bluestem (Schizachyrium scoparium), tall fescue (Festuca arundinacea), and pennsylvania sedge grown in a greenhouse were infested at the root zone with 84 grubs per 0.1 m2 or 182 grubs per 0.1 m2. The effects on plant growth, root loss, survival, and weight gain of grubs were determined. Survival rates were similar for low and high grub densities. With comparable densities of grubs, root loss tended to be proportionately less in zoysiagrass and bermudagrass than in other species. European chafer grubs caused greater root loss at higher densities. Grub weight gain and percentage recovery decreased with increasing grub density, suggesting a food limitation even though root systems were not completely devoured. Bermudagrass root weight showed greater tolerance to european chafer grubs; another mechanism is likely involved for zoysiagrass. Variation in susceptibility of plant species to european chafer suggests that differences in the ability of the plants to withstand grub feeding damage may be amenable to improvement by plant selection and breeding.
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Popay, A. J., R. J. Townsend, and L. R. Fletcher. "The effect of endophyte (Neotyphodium uncinatum) in meadow fescue on grass grub larvae." New Zealand Plant Protection 56 (August 1, 2003): 123–28. http://dx.doi.org/10.30843/nzpp.2003.56.6052.

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Grass grub (Costelytra zealandica) population density mean larval weight and visible damage were lower in meadow fescue (Festuca pratensis) infected with the endophyte Neotyphodium uncinatum (E) than in uninfected meadow fescue (E) in an unreplicated field trial In two bioassays third instar grass grubs ate all meadow fescue E roots but significantly less of the E roots Larvae fed E roots lost weight at the same rate as unfed control larvae Larvae given a choice between maize and either E or E meadow fescue in a pot trial consumed 33 more of the maize in the E treatment than in the E treatment Weight gain of larvae in E treatments was significantly less than in E in both the choice and nochoice pot trials but survival was the same It was concluded that meadow fescue infected with N uncinatum deters grass grub larval feeding but has no major toxic effects
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Wright, D. A., J. Swaminathan, M. Blaser, and T. A. Jackson. "Carrot seed coating with bacteria for seedling protection from grass grub damage." New Zealand Plant Protection 58 (August 1, 2005): 229–33. http://dx.doi.org/10.30843/nzpp.2005.58.4278.

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Carrot seedlings are susceptible to damage from grass grub larvae The biological control bacterium Serratia entomophila was applied to the surface of carrot seeds via pelleting or as a biopolymer seed coating and the activity against grass grubs determined in pot trials Seedling mortality caused by grass grub larvae was significantly reduced (Plt;005) in two trials from 88 and 64 in untreated pots to 26 and 13 in pots containing pelleted seed and 7 and 16 in pots containing biopolymercoated seed Shelf life studies showed formulations were stable at 4C for at least eight weeks and for two weeks at 20C after which cell viability decreased over time Bioassay results showed little difference between the two treatments despite a higher concentration of bacteria on the biopolymercoated than the pelleted seeds The potential of seed coating as a delivery mechanism for biocontrol agents has been demonstrated and future possibilities are discussed
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Kard, Bradford M. R., and Fred P. Hain. "CHEMICAL1 CONTROL OF THREE WHITE GRUB SPECIES (COLEOPTERA: SCARABAEIDAE) ATTACKING FRASER FIR CHRISTMAS TREES IN THE SOUTHERN APPALACHIANS." Journal of Entomological Science 22, no. 1 (January 1, 1987): 84–89. http://dx.doi.org/10.18474/0749-8004-22.1.84.

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Field experiments were conducted in 1982, 1983, and 1984 to evaluate the efficacy of several insecticides for controlling white grubs infesting Fraser fir, Abies fraseri (Pursh) Poir., Christmas trees and pastureland scheduled for fir plantings, and to evaluate insecticide phytotoxicity. The white grub complex consisted primarily of three species: Pyllophaga anxia (LeConte) Glasgow, P. fusca (Froelich) Glasgow, and Polyphylla comes Casey. Mean pretreatment white grub population densities ranged from 20.8 to 77.8 grubs per m2. Isazophos, diazinon, carbofuran, carbaryl, trichlorfon, chlorpyrifos, and isofenphos demonstrated a wide range of effectiveness in reducing populations while showing no phytotoxicity to grass sod or fir. Isazophos and diazinon applications provided the highest levels of control.
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Wrenn, N. R., R. A. McGhie, and R. P. Pottinger. "Evaluation of terbufos for grass grub control." Proceedings of the New Zealand Weed and Pest Control Conference 38 (January 8, 1985): 23–26. http://dx.doi.org/10.30843/nzpp.1985.38.9463.

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Heffernan, P. M., T. A. Jackson, C. B. Dyson, and D. J. Saville. "Sequential sampling of grass grub,Costelytra zealandica." New Zealand Journal of Agricultural Research 35, no. 3 (July 1992): 299–305. http://dx.doi.org/10.1080/00288233.1992.10427507.

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Patchett, B. J., R. B. Chapman, L. R. Fletcher, and S. R. Gooneratne. "Root loline concentration in endophyteinfected meadow fescue (Festuca pratensis) is increased by grass grub (Costelytra zealandica) attack." New Zealand Plant Protection 61 (August 1, 2008): 210–14. http://dx.doi.org/10.30843/nzpp.2008.61.6844.

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The larvae of New Zealand grass grub are economically important subterranean pests of pastures Some endophyteinfected meadow fescues contain loline alkaloids in the roots which can protect the plant from insect attack Loline concentrations in the roots of meadow fescue ecotypes in autumn were similar to concentrations in shoots of the same line Loline concentrations in the roots of the meadow fescue ecotypes exposed to grass grub were significantly higher (P
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Zydenbos, S. M., R. J. Townsend, P. M. S. Lane, S. Mansfield, M. O?Callaghan, C. Van_Koten, and T. A. Jackson. "Effect of Serratia entomophila and diazinon applied with seed against grass grub populations on the North Island volcanic plateau." New Zealand Plant Protection 69 (January 8, 2016): 86–93. http://dx.doi.org/10.30843/nzpp.2016.69.5919.

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The bacterial biocontrol agent Serratia entomophila and the insecticide diazinon were applied as separate granular formulations with ryegrass seed and compared with a seedonly control treatment on three pastures of different ages and composition on the North Island volcanic plateau In the first 2 years diazinon and S entomophila significantly reduced healthy grass grub populations compared with the control However by the third year populations in the diazinon treatments had recovered and were significantly higher than in S entomophila or control plots Grass grub populations were reduced by disease outbreaks after S entomophila was applied which infected >40 of grass grub larvae in the treated plots in year two Bacterial extraction from soil a year after application confirmed establishment and persistence of S entomophila in treated plots Visual positive pasture growth responses were noted in both the S entomophila and diazinontreated plots
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Dissertations / Theses on the topic "Grass grub"

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Sheen, Tamsin, and n/a. "Osmotic and desiccation stress-tolerance of Serratia entomophila." University of Otago. Department of Microbiology & Immunology, 2008. http://adt.otago.ac.nz./public/adt-NZDU20081208.114925.

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Serratia entomophila, the causative agent of amber disease, is an endemic bacterium used for the biocontrol of New Zealand grass grub larvae. Although the available biopesticide is effective, its use is limited to areas where sub-surface application is feasible, and is also impacted by soil conditions such as moisture levels and osmolarity. The aim of this study was to elucidate the responses of S. entomophila to osmotic and desiccation stresses in relation to challenges encountered during production, storage and soil application, with the goal of developing a more robust and versatile biocontrol agent. RpoS is a key factor in the stress response of many enteric bacteria. In order to dissociate the effect of RpoS from subsequent cellular stress studies, an rpoS mutant was constructed by site-directed mutagenesis. Assessment of the rpoS mutant showed that RpoS was not implicated in NaC1 or desiccation tolerance of S. entomophila. The rpoS mutant was instead found to have enhanced salt tolerance and could be distinguished from the wild-type by the ability to ferment arabinose, a phenotype that was confirmed through complementation. Complete abolition of the amber disease process was observed using an rpoS strain also missing the Sep virulence genes, suggesting that RpoS is a regulator of the S. entomophila anti-feeding prophage (Afp). These findings indicate a subtle interplay between NaC1 tolerance, virulence and RpoS-mediated regulation of amber disease in S. entomophila. A transposon mutagenesis screen was carried out to identify genes associated with NaC1 tolerance in S. entomophila. Fourteen mutants displaying NaC1 sensitivity were identified, two of which had mutations in genes with potential implications for the formulation of the bacterium as a biocontrol agent. The gene leuO that encodes a LysR-family transcriptional regulator was found to be essential for S. entomophila NaC1 tolerance. The toxicity of increased cellular LeuO from an over-expression vector led to the investigation of the effects of leuO mutation on the proteome. Multiple protein changes observed by two-dimensional gel analysis suggested that LeuO may be a global regulator in S. entomophila, as has been hypothesised for Salmonella species. A second NaC1-sensitive mutant contained an insertion in afp15, the product of which is thought to be involved in assembly of the Afp. As well as being sensitive to NaC1, the afp15 mutant was unable to induce the anti-feeding component of amber disease, again highlighting the link between stress tolerance and virulence in S. entomophila. This study also determined that pre-exposure to NaC1 in conjunction with the provision of exogenous glycine betaine significantly enhanced the survival of S. entomophila either in a desiccated state or after application to soil, regardless of the soil moisture content. The implication of this finding on the future formulation of S. entomophila led to investigation of the underlying genetic mechanisms involved in glycine betaine synthesis and NaC1 tolerance. The genes involved in glycine betaine biosynthesis from choline were identified through genomic comparison, degenerate PCR and primer walking. A 6.5 kb region was sequenced and found to contain four genes with homology and similar chromosomal arrangement to the E. coli bet genes (betTIBA). The S. entomophila betIBA genes comprised an operon, flanked by the divergently-transcribed betT gene whose product is responsible for choline transport. To ascertain the relative transcription levels of components of the bet operon, quantitative RT-PCR was performed. Results of qRT-PCR showed that choline in conjunction with NaC1 induced the greatest levels of bet gene transcription, and that levels of the betA transcript were significantly lower than those of the other bet genes. Examination of the betA 5� non-coding region identified a previously undetected hairpin region, possibly accounting for the observed decrease in betA transcript levels. The findings of this study have significantly advanced our understanding of how S. entomophiia responds to stress, and will contribute to the development of formulation strategies for the production of a robust product capable of application to pasture by a range of teclmiques. In addition, there is significant potential to utilise these findings in the development of other bacterial inocula for a range of biotechnological applications.
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Nunez-Valdez, Maria Eugenia. "Identification and analysis of the virulence factors in Serratia entomophila causing amber disease to the grass grub Costelytra zealandica : A molecular genetics approach." Thesis, University of Canterbury. Molecular Genetics, 1994. http://hdl.handle.net/10092/6807.

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Amber disease caused by Serratia entomophila to larvae of Costelytra zealandica (Coleoptera: Scarabaeidae), is characterized by the production of two symptoms: anti-feeding effect (AFE) and amber coloration (AC). This study was aimed to identify and characterize the virulence factors involved in the disease. Three factors were identified: i) MRE-HA fimbriae; ii) an extracellular protease and, iii) an anti-feeding toxin. i) Fimbriae type 1, 3 and MRE-HA were identified and characterized in S. entomophila by haemagglutination tests and electron microscopy. Analysis of nonpathogenic mutants suggested that the MRE-HA fimbriae were associated with pathogenicity. ii) The locus coding for the extracellular protease of S. entomophila was identified and cloned. Examination and complementation assays of pathogenic and nonpathogenic strains showed that the protease is not directly involved, but it might potentiate the disease. It was suggested that the protease might be linked with pathogenicity by a common regulator factor. iii) A locus named amb2 was identified, isolated and cloned. Genetic evidence and complementation assays with nonpathogenic mutants demonstrated that amb2 is responsible for the AFE. SDS-PAGE analysis of the amb2 gene products expressed in minicells showed the synthesis of two proteins of 21 and 25 kDa, named AnfA and AnfB. The genes encoding these proteins were mapped by deletion analysis and lacZ-gene fusions. DNA sequencing of the anfA gene revealed that another protein of ~12 kDa (AnfA2) was also encoded by amb2. Consensus sequences with homology to the binding sites of the bacterial regulators CAP, Fur and ToxR were identified in the promoter regions. Homology of 50% was found between a hydrophobic motif of the δ-endotoxin of Bacillus thuringiensis and AnfA1, The results suggest that AnfA1 , AnfA2 and AnfB might be subunits of a toxin causing the AFE. It was concluded that virulence determinants in S. entomophila including the MRE-HA fimbriae, the extracellular protease and the anti-feeding toxin act in collaboration to produce amber disease.
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Books on the topic "Grass grub"

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Porter, Cheryl. Gross grub: Wretched recipes that look yucky but taste yummy. New York: Random House, 1995.

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Porter, Cheryl. Gross grub: Wretched recipes that look yucky but taste yummy. New York: Random House, 1995.

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Dpal gsaṅ chen grub paʼi rnal ʼbyor ba chen po dge sloṅ Chos-grags-rgya-mtshoʼi gso thabs ṅo mtshar rdo rje lhun po. [Qinghai: Rñe dben gnas gsaṅ chen rol baʼi dgaʼ tshal], 2005.

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Źabs-druṅ, ʼJam-dpal-dge-ʼdun-rgya-mtsho Stoṅ-ʼkhor, ed. Rje-btsun Grags-pa-bśad-sgrub kyi mdzad paʼi grub mthaʼ, sa lam daṅ Stoṅ-ʼkhor Źabs-druṅ gi mdzad paʼi don bdun cu bcas. 2nd ed. Bylakuppe, Mysore District, Karnataka State, India: Ser-smad dpe mdzod khaṅ, 1995.

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Nam-mkhaʼ-rin-chen and Nam-mkhaʼ-rin-chen. Mgon po Tshogs bdag gi gsaṅ sgrub dṅos grub ʼbyuṅ gter: The guhyasādhana of Mahākāla in the form of Gaṇapati according to the revelations of Ldi-ri Chos-grags. Bylakuppe, Mysore, India: Pema Norbu Rinpoche Nyingmapa Monastery, 1985.

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Dgaʼ-ldan Śar-rste Thos-bsam-nor-gliṅ Grwa-tshaṅ. Dgaʼ-śar Dpe-mdzod-khaṅ., ed. Rtsod dus kyi mahā Paṇḍi-ta mkhas śiṅ grub paʼi dbaṅ phyug mdo sṅags rab byams smra ba Paṇ-chen Bsod-nams-grags-paʼi dpal rnam dpyod mchog gi sdeʼi gsuṅ ʼbum (Ga pa). Mundgod, Distt. Karwar, Karnataka State: Dgaʼ-śar Dpe-mdzod-khaṅ, 2003.

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ʼJam-dbyaṅs-mkhyen-rab-rgya-mtsho. Dpal ldan bla ma dam pa rigs daṅ dkyil ʼkhor rgya mtshoʼi mṅaʼ bdag bkaʼ drin mtshuṅs med Gʼyuṅ-druṅ-lhun-grub dbaṅ gyi rgyal po Tshul-khrims-bstan-pa-ʼbrug-grags dpal bzaṅ poʼi sku gsuṅ thugs kyi rtogs pa brjod pa skal ldan padmo ʼbyed pai ñin byed bźugs so. Tibet: s.n., 2005.

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Dgaʼ-ldan Śar-rtse Thos-bsam-nor-gliṅ-grwa-tshaṅ. Dgaʼ-śar dpe mdzod khaṅ., ed. Rtsod dus kyi Mahā Paṇḍi-ta mkhas śiṅ grub paʼi dbaṅ phyug mdo sṅags rab ʼbyams pa smra ba Paṅ-chen Bsod-nams-grags-paʼi dpal rnam dpyod mchod gi sdeʼi gsuṅ ʼbum dbu maʼi spyi don zab don gsal baʼi sgron me daṅ dbu ma ʼjug paʼi brgal lan zab don yaṅ gsal sgron me źes bya ba bźugs so. Mundgod, Distt. Karwar, North Kanara, Karnataka State: Dgaʼ-śar dpe mdzod khaṅ, 2000.

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Gross Grub. Random House, 1995.

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Grab them by the...: Grasp the power of social media. aura free press, 2017.

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Book chapters on the topic "Grass grub"

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Oliveira, Gustavo de L. T., Ben M. McKay, and Juan Liu. "Beyond land grabs: new insights on land struggles and global agrarian change." In Beyond the Global Land Grab, 1–18. London: Routledge, 2021. http://dx.doi.org/10.4324/9781003218906-1.

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Gyapong, Adwoa Yeboah. "Open Access: Land grabs, farmworkers, and rural livelihoods in West Africa: some silences in the food sovereignty discourse." In Beyond the Global Land Grab, 19–34. London: Routledge, 2021. http://dx.doi.org/10.4324/9781003218906-2.

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Orchiston, Wayne. "A Gain in Light Grasp: The Legacy of the Grubb Telescope." In John Tebbutt, 319–92. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-44521-2_11.

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Liu, Xiaoyu, Heidi H. Shi, and Jing-Schmidt Zhuo. "Manual Action Metaphors in Chinese A Usage-Based Constructionist Study." In Sinica venetiana. Venice: Fondazione Università Ca’ Foscari, 2020. http://dx.doi.org/10.30687/978-88-6969-406-6/005.

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This article examines Chinese manual motor metaphors involving manual object manipulation as the source domain. Specifically, we use corpus data to investigate two transitive constructions [抓紧zhuājĭn ‘grab tightly, clutch’ NP] and [把住 băzhù ‘grasp firmly’ NP] and a causative construction [把bă NP捧pĕng COMPL] ‘lift NP with deliberation’ where the referent of the NP does not lend itself to manual manipulation in the literal sense and must be interpreted as metaphoric in the unity of semantic domains. Results from both quantitative and qualitative analyses show that the two transitive grasping actions are systematically used to abstract actions requiring a keen sense of urgency and/or importance and that the causative action of lifting systematically conceptualizes over-promotion of an undeserving entity. The findings point to the bodily origin of social cognition and the embodiment of conceptualization.
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Berent, Iris. "Insane About the Brain." In The Blind Storyteller, 177–90. Oxford University Press, 2020. http://dx.doi.org/10.1093/oso/9780190061920.003.0011.

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As the discoveries from neuroscience pour in, the newspaper headlines capture our imagination. They announce that “Musicians’ Brains Really Work Differently” and promise to tell us “How Learning a New Language Actually Rewires the Brain.” What is this hype all about? Do you really need a brain scan to tell that a musician’s brain is different than a nonmusician’s? Cann’t you hear the difference right from the first sound? And don’t we all know that learning a language happens “up north”? It is not the science or technology that attracts our attention, nor is it their aesthetics. Images of genes, proteins, and viruses have no less scientific significance and visual appeal, but they don’t mesmerize us in the same way. Yet we incorrectly believe that evidence from neuroscience is stronger than the results from cognitive experiments. In fact, people fall for “brain stories” even when these accounts are clearly fallacious. Why do we blindly follow the brain? This chapter traces the allure of neuroscience to Dualism. The Dualist view condemns us to an eternal state of wonder because we fail to grasp how “thinking” (e.g., of a cup) can command changes in our material body (e.g., I move my hand to grab it)—how the immaterial mind can effect change in matter. Brain scans alleviate the dissonance by presenting us with tangible proof that thinking is material. The mind–body dissonance that plagues our reasoning about our healthy selves also derails our reasoning about a number of psychiatric conditions, discussed in the next chapters.
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Conference papers on the topic "Grass grub"

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Jude, Alvin, G. Michael Poor, and Darren Guinness. "Grasp, Grab or Pinch? Identifying User Preference for In-Air Gestural Manipulation." In SUI '16: Symposium on Spatial User Interaction. New York, NY, USA: ACM, 2016. http://dx.doi.org/10.1145/2983310.2989209.

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Muller, Judith, Udo Frese, and Thomas Rofer. "Grab a mug - Object detection and grasp motion planning with the Nao robot." In 2012 12th IEEE-RAS International Conference on Humanoid Robots (Humanoids 2012). IEEE, 2012. http://dx.doi.org/10.1109/humanoids.2012.6651543.

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