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1

Newcombe, David S. Gout. Edited by Dwight R. Robinson. London: Springer London, 2013. http://dx.doi.org/10.1007/978-1-4471-4264-5.

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2

K, Choi Hyon, ed. Gout. Philadelphia: Saunders, 2006.

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3

Khunti, Kamlesh. Gout: [a protocol. Leicester: Eli Lilly National Clinical Audit Centre, 1995.

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4

Michel, Butor. Avant-gout II. Chavagne: Ubacs, 1987.

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5

Groddington, George. Gout. Managing Gout and Coping with Gout. Reference for Gout Sufferers Including Gout Diet. IMB Publishing, 2014.

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6

Gout and Rheumatic Gout. HardPress, 2020.

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7

Gout and Rheumatic Gout. Creative Media Partners, LLC, 2022.

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8

Gout and Rheumatic Gout. Creative Media Partners, LLC, 2022.

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9

Gout and Rheumatic Gout. BiblioLife, 2008.

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10

Carioscia, Marshall. Gout Cookbook : Gout-Friendly Recipes: Gout Friendly Cookbook. Independently Published, 2021.

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11

Dalbeth, Nicola. Gout. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0141.

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Gout is a common and treatable disorder of purine metabolism. Gout typically presents as recurrent self-limiting episodes of severe inflammatory arthritis affecting the foot. In the presence of persistent hyperuricaemia, tophi, chronic synovitis, and joint damage may develop. Diagnosis of gout is confirmed by identification of monosodium urate (MSU) crystals using polarizing light microscopy. Hyperuricaemia is the central biochemical cause of gout. Genetic variants in certain renal tubular urate transporters including SLC2A9 and ABCG2, and dietary factors including intake of high-purine meats and seafood, beer, and fructose, contribute to development of hyperuricaemia and gout. Gout treatment includes: (1) management of the acute attack using non-steroidal anti-inflammatory drugs (NSAIDs), corticosteroids, or low-dose colchicine; (2) prophylaxis against gout attacks when commencing urate-lowering therapy (ULT), with NSAIDs or colchicine; and (3) long-term ULT to achieve a target serum urate of less than 0.36 mmol/litre. Interleukin (IL)-1β‎ is a central mediator of acute gouty inflammation and anti-IL-1β‎ therapies show promise for treatment of acute attacks and prophylaxis. The mainstay of ULT remains allopurinol. However, old ULT agents such as probenecid and benzbromarone and newer agents such as febuxostat and pegloticase are also effective, and should be considered in patients in whom allopurinol is ineffective or poorly tolerated. Management of gout should be considered in the context of medical conditions that frequently coexist with gout, including type 2 diabetes, hypertension, dyslipidaemia, and chronic kidney disease. Patient education is essential to ensure that acute gout attacks are promptly and safely managed, and long-term ULT is maintained.
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12

Dalbeth, Nicola. Gout. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642489.003.0141_update_003.

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Gout is a common and treatable disorder of purine metabolism. Gout typically presents as recurrent self-limiting episodes of severe inflammatory arthritis affecting the foot. In the presence of persistent hyperuricaemia, tophi, chronic synovitis, and joint damage may develop. Diagnosis of gout is confirmed by identification of monosodium urate (MSU) crystals using polarizing light microscopy. Hyperuricaemia is the central biochemical cause of gout. Genetic variants in certain renal tubular urate transporters including SLC2A9 and ABCG2, and dietary factors including intake of high-purine meats and seafood, beer, and fructose, contribute to development of hyperuricaemia and gout. Gout treatment includes: (1) management of the acute attack using non-steroidal anti-inflammatory drugs (NSAIDs), corticosteroids, or low-dose colchicine; (2) prophylaxis against gout attacks when commencing urate-lowering therapy (ULT), with NSAIDs or colchicine; and (3) long-term ULT to achieve a target serum urate of less than 0.36 mmol/litre. Interleukin (IL)-1β‎ is a central mediator of acute gouty inflammation and anti-IL-1β‎ therapies show promise for treatment of acute attacks and prophylaxis. The mainstay of ULT remains allopurinol. However, old ULT agents such as probenecid and benzbromarone and newer agents such as febuxostat and pegloticase are also effective, and should be considered in patients in whom allopurinol is ineffective or poorly tolerated. Management of gout should be considered in the context of medical conditions that frequently coexist with gout, including type 2 diabetes, hypertension, dyslipidaemia, and chronic kidney disease. Patient education is essential to ensure that acute gout attacks are promptly and safely managed, and long-term ULT is maintained.
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13

Dalbeth, Nicola. Introduction to gout. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198748311.003.0001.

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Gout is a chronic condition of monosodium urate crystal deposition. It is the most common form of inflammatory arthritis in adults, and leads to recurrent flares of severe joint damage and musculoskeletal disability. Although treatment targets are well defined, gout management is currently poor, with low levels of treatment targets achieved. The last decade has seen major advances in the understanding and treatment of gout. This handbook summarizes key scientific advances, including new insights into mechanisms of hyperuricaemia, acute gouty arthritis, and joint damage. Principles of gout diagnosis and management are discussed in detail, with practical information about use of well-established agents and also newer therapies. Gout-specific research tools are outlined to assist clinicians with interpretation of the latest scientific literature in gout. Future strategies for improved gout management are also discussed.
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14

Porter, Roy, Roy Porter G. S. Rousseau, and G. S. Rousseau. Gout. Yale University Press, 2000.

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15

Gout. Elsevier, 2019. http://dx.doi.org/10.1016/c2016-0-05041-6.

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16

Lipsky, Peter E., and Naomi Schlesinger. Gout. Elsevier - Health Sciences Division, 2018.

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17

Mortis, Igor. Gout. Legend Press, 2013.

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18

Luff, Arthur Pearson. Gout. Creative Media Partners, LLC, 2018.

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19

Gout. Franklin Classics, 2018.

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20

Gout. Creative Media Partners, LLC, 2022.

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21

Dalbeth, Nicola, Lisa Stamp, and Tony Merriman. Gout. Oxford University Press, 2016.

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22

Mortis, Igor. Gout. Legend Press, 2013.

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23

Luff, Arthur Pearson. Gout. Franklin Classics Trade Press, 2018.

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24

Luff, Arthur Pearson. Gout. Franklin Classics Trade Press, 2018.

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25

Llewellyn, Richard Llewellyn Jones. Gout. Franklin Classics Trade Press, 2018.

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26

Gout. Franklin Classics, 2018.

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27

Gout. Creative Media Partners, LLC, 2022.

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28

Gout. Nabu Press, 2010.

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29

Radziewicz, Blanch. Ultimate Gout Cookbook : Gout Risk Factors: Gout Cookbook Indigo. Independently Published, 2021.

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30

Skeans, Ela. Gout Hater's Cookbook : Stages of Gout: The Healing Gout Cookbook. Independently Published, 2021.

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31

Lazer, Demetria. Healthy Gout Cookbook : History of Gout: Gout Diet Recipes 2010. Independently Published, 2021.

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32

Gout: Questions and answers about gout. Bethesda, MD: National Institute of Arthritis and Musculoskeletal and Skin Diseases (NIAMS), National Institutes of Health, 2010.

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33

Khanna, Puja. Treatment of acute gout. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0045.

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Acute gout is a common inflammatory arthritis in the adult population. Epidemiological evidence suggests that the prevalence of gout is steadily on the rise due to longevity, coexisting comorbidities, and iatrogenic causes contributing to hyperuricaemia. Acute gout usually presents as a self-limiting flare of synovitis that occurs due to deposition of monosodium urate crystals. The frequency of flares generally increases over time in patients who continue to have hyperuricaemia and their risk factors for acute gout attacks have not been adequately addressed. Effective treatment of acute gouty arthritis is primary focused on pain which is the primary symptom but must target both the pain and underlying inflammation. Acute gout is frequently treated with non-steroidal anti-inflammatory agents, colchicine, and corticosteroids. This chapter reviews the available therapies for management of acute gout and ones that have shown promising results.
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34

Keenan, Robert T., Sneha Pai, and Naomi Schlesinger. Imaging of gout. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0043.

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Gout is a systemic metabolic disease. The enzyme urate oxidase (uricase) that catalyses the oxidation of uric acid to the more soluble compound allantoin is inactive in humans. This may lead to hyperuricaemia. Hyperuricaemia is often present for many years prior to clinical signs of gout. Acute attacks occur as a result of an inflammatory response to monosodium urate (MSU) crystal deposition leading to intense pain and inflammation in the affected joints. Uncontrolled hyperuricaemia and resultant gout can evolve into a destructive arthritis. Imaging may be helpful in the diagnosis of gout as well as in monitoring the response to gout treatment. Plain X-rays are widely used for joint imaging in patients with gout. However, plain X-rays of joints affected by gout are frequently normal, especially early in the disease. In these cases, advanced imaging modalities may be useful. Advanced imaging can help evaluate inflammation, structural joint changes, and magnitude of tophaceous deposits. Advanced imaging modalities include computed tomography (CT), dual-energy CT (DECT), magnetic resonance imaging (MRI), and ultrasound (US). CT may be most suitable to evaluate bone changes in gouty joints and DECT to evaluate tophaceous deposits. MRI may best evaluate soft tissues and Inflammation. US is useful during patients’ visits to the rheumatologist and allows evaluation of cartilage, soft tissues, synovium, and tophaceous deposits. This chapter reviews imaging modalities used in gout patients and discusses their application in the diagnosis and management of gout.
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35

Harpole, Florene. Living with Gout Tips : Understand Gout: Should I Walk with Gout. Independently Published, 2021.

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36

Happe, Roxane. Living with Gout Tips : Understand Gout: Living with Someone with Gout. Independently Published, 2021.

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37

Dalbeth, Nicola. Laboratory testing in gout diagnosis and management. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198748311.003.0006.

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Gout typically presents as recurrent flares of acute self-limiting arthritis. The acute gout flare is characterized by severe joint inflammation. In the presence of prolonged untreated hyperuricaemia, some people with gout may develop gouty tophi, which cause retracted joint movement, ulceration, and joint damage. The differential diagnosis for gout includes septic arthritis (which may co-exist with gout), joint injury, calcium pyrophosphate deposition, basic calcium phosphate arthritis or tendinitis, reactive arthritis, and psoriatic arthritis.
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38

Understanding Gout. ReadHowYouWant.com, Limited, 2015.

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39

Jefferson, Warren. Understanding GOUT. Book Publishing Company, 2014.

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40

Pestana. QRG Gout. Built by Doctors World Ltd, 2021.

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41

Haut Gout. Broadway Play Publishing Inc, 2013.

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42

Garrod, Alfred Baring. A Treatise On Gout and Rheumatic Gout. Arkose Press, 2015.

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43

DORIS, CARA. 24 Foods Gout Prevention: Gout Diet Cookbook. Independently Published, 2019.

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44

Scott, Rose. Goodbye to Gout: A New Gout Diet. Lulu Press, Inc., 2014.

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45

GREGORY, Lisa H. Gout Diet Cookbook: The Healing Gout Cookbook. Independently Published, 2020.

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46

Haisley, Danika. Living with Gout Arthritis : Master Know about Gout: Living with Gout Diet. Independently Published, 2021.

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47

Gout Be Gone - The Ultimate Gout Cookbook - 50+ Gout Recipes for Inflammatory Relief -: Gout Remedies are Through Diet - Live Life Gout Free! CreateSpace Independent Publishing Platform, 2014.

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48

Sugerak, Terry. Gout Diet Guide : Foods That Can Help You Control Gout: Gout Diet Book. Independently Published, 2021.

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49

Coppage, Noble. Healthy Gout Cookbook : What Is a Good Breakfast for Gout?: Gout Hater's Cookbook. Independently Published, 2021.

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50

Suggs, Emilio. How to Defeat Gout? : Guides and Tips to Banish Gout: Youtube Gout Remedies. Independently Published, 2021.

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