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Books on the topic 'Gonadotrophins'

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Author: Grafiati

Published: 4 June 2021

Last updated: 19 February 2023

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1

Wood, Anita Margaret. The control of human granulosa-lutein cell growth and function by gonadotrophins and insulin-like growth factor-1. Manchester: University of Manchester, 1993.

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2

Emperaire, Jean-Claude. Pratique de la stimulation ovulatoire par les gonadotrophines. Paris: Springer Paris, 2013. http://dx.doi.org/10.1007/978-2-8178-0398-2.

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3

Goodman, Stephanie Robin. Effects of gonadotrophin releasing hormone on growth hormone release in the rat. [New Haven, Conn: s.n.], 1993.

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4

Saade, Georges. The regulation of luteinizing hormone and prolactin gene expression by gonadotrophin-releasing hormone and gonadal steroids in mice. Birmingham: University of Birmingham, 1988.

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5

Elgendy, Manal. Minimising the dose of gonadotrophin releasing hormone agonist [GnRHa] and recombinant follicle stimulating hormone [FSH] used for controlled ovarian hyperstimulation in in-vitro fertilisation. Birmingham: University of Birmingham, 2001.

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6

The HCG diet book of secrets: Stabilizing after HCG and staying slim forever. Houston, Tex: Harmonious Clarity Group, LLC., 2011.

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7

Dohle, Gert R. Surgical treatment of male infertility. Edited by David John Ralph. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0097.

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Surgical treatment of male infertility is indicated in men with obstructive azoospermia due to epididymal and vassal blockage, in infertile men with a varicocele and oligozoospermia, and to harvest spermatozoa for future intracytoplasmic sperm injection (ICSI). Testis biopsy may be performed in men with normal testis volume and normal gonadotrophins to confirm the diagnosis of obstructive azoospermia. Furthermore, testis biopsies are indicated in men with risk factors for testis cancer, such as infertility and ultrasonograhic abnormalities.Varicocele repair seems effective in case of an infertility duration of at least 2 years, oligozoospermia, and otherwise unexplained infertility in a couple. The advantages of surgery in these couples are a fair chance of spontaneous pregnancies at relative low cost and with less obstetric problems and birth defect compared to pregnancies from IVF procedures.

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8

Yves, Combarnous, Volland-Nail Patricia, and Institut national de la recherche agronomique (France), eds. Les Gonadotropines. Paris: Institut national de la recherche agronomique, 1997.

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9

EMPERAIRE, Jean-Claude. Pratique de la Stimulation Ovulatoire Par les Gonadotrophines. Springer, 2014.

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10

Structure and Function of the Gonadotropins. Springer, 2012.

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11

McKerns, Kenneth W. Structure and Function of the Gonadotropins. Springer, 2012.

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12

McKerns, Kenneth W. Structure and Function of the Gonadotropins. Springer, 2012.

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13

The effects of prolonged submaximal exercise on androgen and gonadotrophin levels in males with insulin dependent diabetes mellitus. 1986.

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14

The effects of prolonged submaximal exercise on androgen and gonadotrophin levels in males with insulin dependent diabetes mellitus. 1986.

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15

The effects of prolonged submaximal exercise on androgen and gonadotrophin levels in males with insulin dependent diabetes mellitus. 1986.

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16

Bower, Mark, Louise Robinson, and Sarah Cox. Endocrine and metabolic complications of advanced cancer. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656097.003.0142.

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Cancer produces endocrine and metabolic complications in two ways. Firstly, the primary tumour or its metastases may interfere with the function of endocrine glands, kidneys, or liver by invasion or obstruction. Secondly, tumours may give rise to remote effects without local spread and these actions are termed paraneoplastic manifestations of malignancy. Generally, these paraneoplastic syndromes arise from secretion by tumours of hormones, cytokines, and growth factors, but also occur when normal cells secrete products in response to the presence of tumour. This chapter reviews the pathogenesis, epidemiology, and management of the commonest paraneoplastic endocrinopathies including hypercalcaemia, Cushing’s syndrome, the syndrome of inappropriate antidiuresis, non-islet cell tumour hypoglycaemia, enteropancreatic hormone syndromes, Carcinoid syndrome, phaeochromocytoma, gonadotrophin secretion syndromes, prolactin and oxytocin secretion, and paraneoplastic pyrexia. The chapter concludes with a brief discussion of the management of metabolic disease in the context of advanced malignancy including hyperglycaemia, thyroid dysfunction, metabolic bone disease, renal failure, liver failure, and lactic acidosis.

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17

Hcg Diet Made Simple Your Stepbystep Guide Beyond Pounds And Inches. Harmonious Clarity Group, LLC, 2009.

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18

Cassidy, Jim, Donald Bissett, Roy A. J. Spence OBE, Miranda Payne, Gareth Morris-Stiff, and Madhumita Bhattacharyya. Gynaecological cancers. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199689842.003.0020_update_001.

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Genitourinary cancers examines the malignancies arising in the kidney, ureter, bladder, prostate, testis, and penis. Renal cancer has high propensity for systemic spread, largely mediated by overexpression of vascular endothelial growth factor (VEGF). Treatments include surgery, immunotherapy, and targeted therapy. Wilms tumour, a childhood malignancy of the kidney, warrants specialist paediatric oncology management to provide expertise in its unique pathology, staging, and treatment, often with surgery and chemotherapy. Cancer of the bladder and ureters, another tobacco related cancer, may present as either superficial or invasive disease. The former is managed by transurethral resection and intravesical therapy. The latter may require radical surgery, preoperative chemotherapy, or radiotherapy. Prostate cancer, the commonest male cancer, is an androgen dependent malignancy. It has attracted controversy with regards to PSA screening, and potential over treatment with radical prostatectomy. Division into low, intermediate, and high risk disease according to tumour grade, stage, and PSA helps in deciding best treatment, antiandrogen therapy for metastatic disease, radiotherapy and adjuvant hormone therapy for locally advanced disease, either surgery or radiotherapy for early intermediate risk disease, and active monitoring for low risk cases. Testicular cancer divides according to pathology into seminoma, nonseminomatous germ cell tumours (NSGCT), and mixed tumours, the latter two frequently producing tumour markers, alpha-fetoprotein (AFP) and/or human chorionic gonadotrophin (HCG). Stage I disease is managed by inguinal orchidectomy and surveillance or adjuvant chemotherapy. More advanced disease is managed by chemotherapy, with high probability of cure in the majority. Penile cancer, often HPV related, can be excised when it presents early, but delay in presentation may lead to regional and systemic spread with poor prognosis.

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