Log in

Relevant bibliographies by topics / Glucose uptake / Books

Books on the topic 'Glucose uptake'

To see the other types of publications on this topic, follow the link: Glucose uptake.

Author: Grafiati

Published: 4 June 2021

Last updated: 9 March 2023

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 15 books for your research on the topic 'Glucose uptake.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse books on a wide variety of disciplines and organise your bibliography correctly.

1

Sheard, Jonathan P. Glucose uptake by pea mesophyll protoplasts. Norwich: University of East Anglia, 1988.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

2

Sarabia, Vivian E. Calcium homeostasis and regulation of glucose uptake in human skeletal muscle cells in culture. Ottawa: National Library of Canada, 1990.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

3

Wan, Calvin Ka Nung. The effects of pioglitazone on hepatic and peripheral glucose uptake in diabetic dogs studied by a novel intraportal glucose loading-euglycemic clamp method. Ottawa: National Library of Canada, 1995.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

4

Lu, Bing. The effect of oxidative stress on vandate and insulin stimulation of glucose uptake in rat adipocytes. Ottawa: National Library of Canada, 1996.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

5

Shahi, B. Chopra. Studies on the uptake and utilization of pyruvate and glucose by isolated segmental ganglia of the leech Haemopis Sanguisuga. Salford: University of Salford, 1987.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

6

Asefaw, Senai. Stimulation of myocardial AMP-activated protein kinase by AICAR increases cardiac glucose uptake and causes GLUT4 and GLUT1 translocation in vivo. [New Haven, Conn: s.n.], 1999.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

7

Hunnisett, Douglas J. Leptin demonstrates no significant effect on basal or insulin-stimulated 2-deoxyglucose uptake and C14-labelled glucose incorporation into glycogen in L6 myotubes. Ottawa: National Library of Canada, 2000.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

8

Glucose Uptake: Regulation, Signaling Pathways and Health Implications. Nova Science Pub Inc, 2013.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

9

Somwar, Romel. GLUT4 activation: A component of the stimulation of glucose uptake by insulin. 2002.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

10

Konrad, Daniel. The role of GLUT4 activation in glucose uptake: Potential implication for insulin resistance? 2003.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

11

Fugmann, Andreas. Effects of Acute Metabolic Interventions on Hemodynamics, Endothelial Function and Forearm Glucose Uptake. Uppsala Universitet, 2001.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

12

Madl, Ulrike. Pathophysiology of glucose control. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0258.

Full text

Abstract:

Hyperglycaemia is a frequent phenomenon in critically-ill patients, associated with increased morbidity and mortality. Hyperglycaemia results in cellular glucose overload and toxic adverse effects of glycolysis and oxidative phosphorylation, especially in tissues with insulin-independent glucose uptake, and acute hyperglycaemia can exert a variety of negative effects. It is the main side effect of intensive insulin therapy. Both severe and moderate hypoglycaemia are independent risk factors of mortality in critically-ill patients. Prolonged hypoglycaemia induces neuronal damage, but may also have adverse cardiovascular effects. Several risk factors predispose critically-ill patients to hypoglycaemic events. Rapid glucose fluctuations may induce oxidative stress and lead to vascular damage. Glucose complexity is a marker of endogenous glucose regulation. Association between hyperglycaemia and outcome is weaker in diabetic critically-ill patients than in non-diabetic patients. Pre-admission glucose control in diabetic critically-ill patients plays a role in the response to glucose control and mortality.

APA, Harvard, Vancouver, ISO, and other styles

13

Cunnane, Stephen C., Alexandre Courchesne-Loyer, Valerie St-Pierre, Camille Vandenberghe, Etienne Croteau, and Christian-Alexandre Castellano. Glucose and Ketone Metabolism in the Aging Brain. Edited by Jong M. Rho. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0015.

Full text

Abstract:

Brain glucose uptake is impaired in Alzheimer’s disease (AD). A key question is whether cognitive decline could be delayed if this defect were at least partly corrected or bypassed. Ketones (or ketone bodies) such as beta-hydroxybutyrate and acetoacetate are the brain’s main alternative fuels. Several studies have shown that in mild-to-moderate AD, brain ketone uptake is similar to that of healthy age-matched controls. Published clinical trials show that increasing ketone availability to the brain via nutritional ketosis has modest benefits on cognitive outcomes in mild-to-moderate AD and in mild cognitive impairment. Nutritional ketosis can be safely achieved by a high-fat ketogenic diet or supplements providing medium chain triglycerides. Given the acute dependence of the brain on its energy supply and the ineffectiveness of current therapeutic strategies for AD consideration be given to correcting the underlying problem of deteriorating brain fuel supply during aging.

APA, Harvard, Vancouver, ISO, and other styles

14

Gerbec, Elizabeth A. Noland. The effects of maternal exposure to trichloroethylene on glucose uptake and nucleic acid and protein levels in the brains of developing rat pups. 1985.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

15

Hyde, Parker, Vincent J. Miller, and Jeff S. Volek. Keto-Adaptation in Health and Fitness. Edited by Dominic P. D’Agostino. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0038.

Full text

Abstract:

When dietary carbohydrate is restricted and protein consumed in moderation, the evolutionarily-conserved ketogenic metabolic machinery awakens. After just a few days circulating ketones increase by an order of magnitude, and over several weeks there is a profound shift away from glucose as the primary energy substrate to the preferred use of fatty acids and ketones. This metabolic process is known as keto-adaptation. The deemphasis on insulin-dependent glucose uptake into cells and concomitant increase in fat oxidation has important implications in management of insulin resistance and its secondary manifestations, which are all functionally carbohydrate-intolerant conditions. The health implications of keto-adaptation are profound. In a definitive break from traditional groupthink, athletes are now experimenting with diets low in carbohydrate in an effort to improve their health, body composition, performance, and recovery. This chapter explores the rationale for the construct of keto-adaptation as a tool for achieving general well-being and improved performance.

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!