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Journal articles on the topic 'Glucose tolerance'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Shen, Xiaoxia, Ping Zhang, Jinping Wang, et al. "Influence of improvement or worsening of glucose tolerance on risk of stroke in persons with impaired glucose tolerance." International Journal of Stroke 13, no. 9 (2018): 941–48. http://dx.doi.org/10.1177/1747493018784432.

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Background and aim We sought to determine the effect of regression to normal glucose tolerance (NGT) or progression to diabetes in early years of impaired glucose tolerance (IGT) on subsequent risk of stroke. Methods In 1986, 576 adults aged 25 years and older with impaired glucose tolerance in Da Qing, China, were randomly assigned by clinic to control, diet, exercise, or diet plus exercise intervention groups for a six-year period. Subsequently participants received medical care in their local clinics. We tracked participants for additional 17 years to ascertain stroke events and other outco

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2

Costa, ??ngels, Ignacio Conget, and Ramon Gomis. "Impaired Glucose Tolerance." Treatments in Endocrinology 1, no. 4 (2002): 205–10. http://dx.doi.org/10.2165/00024677-200201040-00001.

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3

Newman, Byron Y. "Impaired glucose tolerance." Optometry - Journal of the American Optometric Association 78, no. 12 (2007): 622–23. http://dx.doi.org/10.1016/j.optm.2007.11.001.

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4

Davies, M. J., and I. P. Gray. "Impaired glucose tolerance." BMJ 312, no. 7026 (1996): 264–65. http://dx.doi.org/10.1136/bmj.312.7026.264.

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5

Yudkin, J. S., K. G. Alberti, D. G. McLarty, and A. B. Swai. "Impaired glucose tolerance." BMJ 301, no. 6749 (1990): 397–402. http://dx.doi.org/10.1136/bmj.301.6749.397.

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6

Foley, J. E., P. Thuillez, S. Lillioja, J. Zawadzki, and C. Bogardus. "Insulin sensitivity in adipocytes from subjects with varying degrees of glucose tolerance." American Journal of Physiology-Endocrinology and Metabolism 251, no. 3 (1986): E306—E310. http://dx.doi.org/10.1152/ajpendo.1986.251.3.e306.

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Previous studies showed that the sensitivity of glucose transport to insulin is lower in adipocytes isolated from subjects with noninsulin-dependent diabetes mellitus and impaired glucose tolerance compared with subjects with normal glucose tolerance. This study analyzed the relationship between insulin sensitivity of glucose transport and glycemia in a large group of nondiabetic-nonglucose-intolerant subjects with a wide range of glycemic response to oral glucose. Seventy-four Pima Indians with 2-h postglucose load glucoses between 77 and 197 mg/100 ml, fasting plasma glucoses between 76 and

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7

Berkus, M. D., E. M. J. Xenakis, and O. Langer. "Glucose tolerance test periodicity as a descriptor of glucose tolerance abnormality." International Journal of Gynecology & Obstetrics 40, no. 2 (1993): 187. http://dx.doi.org/10.1016/0020-7292(93)90423-t.

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8

Santos, Maria Luiza dos, Flávio F. Aragon, Carlos R. Padovani, and Walkyria P. Pimenta. "Daytime variations in glucose tolerance in people with impaired glucose tolerance." Diabetes Research and Clinical Practice 74, no. 3 (2006): 257–62. http://dx.doi.org/10.1016/j.diabres.2006.04.007.

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9

Link, Karl R. J., and Jacqueline S. Rand. "Reference values for glucose tolerance and glucose tolerance status in cats." Journal of the American Veterinary Medical Association 213, no. 4 (1998): 492–96. http://dx.doi.org/10.2460/javma.1998.213.04.492.

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Objective To determine the reference range for glucose tolerance, using a simplified glucose tolerance test (GTT), and to evaluate glucose tolerance status in cats. Design Prospective study. Animals 57 clinically normal cats. Procedure 2 catheters were placed in the cephalic veins of nonsedated cats at least 3 hours before the test. Blood samples were obtained before (0 minutes) and 2, 5, 10, 15, 30, 45, 60, 90, and 120 minutes after glucose (0.5 g/kg [0.23 g/lb] of body weight) was injected IV. Blood glucose concentration was measured by a glucose meter. Glucose half-life (t½) and disappearan

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10

Diamond, Michael P., Subodhsingh Chauhan, Michael Kruger, and Marappa Subramanian. "Values of fasting glucose levels, glucose tolerance tests, and glucose-insulin ratios as predictors of glucose tolerance." Fertility and Sterility 80, no. 4 (2003): 1022–25. http://dx.doi.org/10.1016/s0015-0282(03)01016-1.

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11

Stout, Robert W. "Glucose Tolerance and Ageing." Journal of the Royal Society of Medicine 87, no. 10 (1994): 608–9. http://dx.doi.org/10.1177/014107689408701015.

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Hyperglycaemia, impaired glucose tolerance and non-insulin dependent diabetes become progressively more common with advancing age. The mechanism is insensitivity to the actions of insulin at the postreceptor level. Inadequate secretion of insulin and decreased hepatic sensitivity to insulin's action in suppressing glucose output also occur. The age-related changes may be made worse by obesity, renal failure or the ingestion of certain drugs, or may be lessened by increased physical activity.

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12

Zetterman, Teemu, Ritva Markkula, and Eija Kalso. "Glucose tolerance in fibromyalgia." Medicine 100, no. 46 (2021): e27803. http://dx.doi.org/10.1097/md.0000000000027803.

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13

Eiks, M. L. "Oral Glucose Tolerance Tests." Diabetes Care 19, no. 3 (1996): 271. http://dx.doi.org/10.2337/diacare.19.3.271a.

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14

Fraser, RobertB, FionaA Ford, Christine Bruce, J. David Fachnie, and FredW Whitehouse. "GLUCOSE TOLERANCE IN PREGNANCY." Lancet 333, no. 8629 (1989): 109–10. http://dx.doi.org/10.1016/s0140-6736(89)91474-8.

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15

Sutherland, H. W., D. W. M. Pearson, D. M. Campbell, and R. J. Jarrett. "GLUCOSE TOLERANCE IN PREGNANCY." Lancet 332, no. 8626-8627 (1988): 1482. http://dx.doi.org/10.1016/s0140-6736(88)90948-8.

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16

Greenspoon, Jeffrey S. "Oral Glucose Tolerance Test." Mayo Clinic Proceedings 63, no. 8 (1988): 838. http://dx.doi.org/10.1016/s0025-6196(12)62371-4.

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17

Taylor, Roy. "DRUGS AND GLUCOSE TOLERANCE." Adverse Drug Reaction Bulletin 121, no. 1 (1986): 452–55. http://dx.doi.org/10.1097/00012995-198612000-00001.

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18

Hurel, Steven J., and Roy Taylor. "Drugs and glucose tolerance." Adverse Drug Reaction Bulletin 174, no. 1 (1995): 659–62. http://dx.doi.org/10.1097/00012995-199510000-00001.

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19

DYCKNER, THOMAS, and PER-OLOV WESTER. "Peroral Glucose Tolerance Test." Acta Medica Scandinavica 220, no. 4 (2009): 315–19. http://dx.doi.org/10.1111/j.0954-6820.1986.tb02771.x.

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20

Williams, D. R. R., and R. J. Jarrett. "Height and glucose tolerance." Diabetologia 35, no. 2 (1992): 191–92. http://dx.doi.org/10.1007/bf00402557.

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21

Voelker, R. "Famine and Glucose Tolerance." JAMA: The Journal of the American Medical Association 279, no. 6 (1998): 420—b—420. http://dx.doi.org/10.1001/jama.279.6.420-b.

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22

Voelker, Rebecca. "Famine and Glucose Tolerance." JAMA 279, no. 6 (1998): 420. http://dx.doi.org/10.1001/jama.279.6.420-jwm81008-3-1.

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23

Giugliano, D. "Height and glucose tolerance." Diabetologia 35, no. 7 (1992): 698–99. http://dx.doi.org/10.1007/bf00400266.

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24

Kapaya, Habiba, Lucy May, Richard Jacques, and Priya Madhuvrata. "Sensitivity of postnatal fasting plasma glucose in identifying impaired glucose tolerance in women with gestational diabetes – 25 Years’ data." Obstetric Medicine 10, no. 3 (2017): 125–31. http://dx.doi.org/10.1177/1753495x17702786.

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Objective To assess the uptake of postnatal oral glucose tolerance test and to determine the sensitivity of fasting postnatal blood sugar in predicting 2-h impaired glucose tolerance. Methods Retrospective study of 1961 women diagnosed with gestational diabetes mellitus. All women were offered oral glucose tolerance test six weeks post-delivery. Results Of 1961 women, 1090 (56%) returned for postpartum oral glucose tolerance test. A fasting plasma glucose of ≥6.1 mmol/l identified only 76 of 167 women with impaired glucose tolerance detected by a 2-h oral glucose tolerance test (sensitivity of

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25

Havalko, Anna, Ervin Asanov, Valerii Shatilo, Ivanna Antonyuk-Shcheglova, and Svitlana Naskalova. "PECULIARITIES OF THE ORGANISM'S REACTION TO DOSED HYPOXIA IN ELDERLY PEOPLE WITH IMPAIRED GLUCOSE TOLERANCE." Problems of Endocrine Pathology 79, no. 3 (2022): 7–13. http://dx.doi.org/10.21856/j-pep.2022.3.01.

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Background. The incidence of carbohydrate metabolism disorders increases with age, resulting in a significantly increased risk of cardiovascular disease. Hypoxia plays an important role in the pathogenesis of carbohydrate metabolism disorders. The aim – to establish the features of carbohydrate metabolism in hypoxia in the elderly with impaired carbohydrate tolerance. Materials and methods. 20 elderly people with impaired glucose tolerance and 23 elderly people with preserved glucose tolerance were examined. Determined the concentration of glucose and insulin in the blood when breathing

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26

Buch, Inge, Peter J. Hornnes, and Claus Kühl. "Glucose tolerance in early pregnancy." Acta Endocrinologica 112, no. 2 (1986): 263–66. http://dx.doi.org/10.1530/acta.0.1120263.

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Abstract. The effect of pregnancy on oral glucose tolerance (50 g of glucose) and plasma insulin and glucagon responses to oral glucose was studied in weeks 10 and 32 of pregnancy and again 1 year post partum in 12 normal women. Already in week 10, fasting plasma glucose was decreased and the glucose-induced insulin secretion increased as compared with post partum. However, glucose tolerance was not affected at this time. In week 32, glucose tolerance had deteriorated, although the levels of both fasting and glucose-induced insulin were higher than those found in early pregnancy and post partu

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27

Lages, Marlene, Renata Barros, Pedro Moreira, and Maria P. Guarino. "Metabolic Effects of an Oral Glucose Tolerance Test Compared to the Mixed Meal Tolerance Tests: A Narrative Review." Nutrients 14, no. 10 (2022): 2032. http://dx.doi.org/10.3390/nu14102032.

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The oral glucose tolerance test (OGTT) is recommended for assessing abnormalities in glucose homeostasis. Recognised as the gold standard test for diagnosing diabetes, the OGTT provides useful information about glucose tolerance. However, it does not replicate the process of absorption and digestion of complex foods, such as that which occurs with a mixed meal tolerance test (MMTT), an alternative that is still not well explored in the diagnosis of metabolic alterations. The MMTT could be an asset in detecting glucose homeostasis disorders, including diabetes since it has more similarities to

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28

Haffner, S. M., A. Agil, L. Mykkanen, M. P. Stern, and I. Jialal. "Plasma Oxidizability in Subjects With Normal Glucose Tolerance, Impaired Glucose Tolerance, and NIDDM." Diabetes Care 18, no. 5 (1995): 646–53. http://dx.doi.org/10.2337/diacare.18.5.646.

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29

Hung, Yi-Jen, Chang-Hsun Hsieh, Dee Pei, et al. "Rosiglitazone improves insulin sensitivity and glucose tolerance in subjects with impaired glucose tolerance." Clinical Endocrinology 62, no. 1 (2005): 85–91. http://dx.doi.org/10.1111/j.1365-2265.2004.02178.x.

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30

Cockram, C. S. "Fasting glucose and progression of impaired glucose tolerance." Diabetes Care 22, no. 2 (1999): 369–70. http://dx.doi.org/10.2337/diacare.22.2.369.

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31

Henriksen, J. E., F. Alford, A. Handberg, A. Vaag, and H. Beck-Nielsen. "Glucose processing during the intravenous glucose tolerance test." Metabolism 45, no. 5 (1996): 598–605. http://dx.doi.org/10.1016/s0026-0495(96)90030-x.

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32

Wu, Tongzhi, Christopher K. Rayner, Karen L. Jones, Cong Xie, Chinmay Marathe, and Michael Horowitz. "Role of intestinal glucose absorption in glucose tolerance." Current Opinion in Pharmacology 55 (December 2020): 116–24. http://dx.doi.org/10.1016/j.coph.2020.10.017.

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33

Cucinotta, Domenico, Filippo De Luca, Alfonso Gigante, et al. "No changes of insulin sensitivity in cystic fibrosis patients with different degrees of glucose tolerance: an epidemiological and longitudinal study." European Journal of Endocrinology 130, no. 3 (1994): 253–58. http://dx.doi.org/10.1530/eje.0.1300253.

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Cucinotta D, De Luca F, Gigante A, Arrigo T, Di Benedetto A, Tedeschi A, Lombardo F, Romano G, Sferlazzas C. No changes of insulin sensitivity in cystic fibrosis patients with different degrees of glucose tolerance: an epidemiological and longitudinal study. Eur J Endocrinol 1994;130:253–8. ISSN 0804–4643 Plasma glucose and insulin responses to oral glucose and insulin sensitivity by the euglycemic hyperinsulinemic clamp technique were investigated in 30 cystic fibrosis patients with normal fasting blood glucose levels and normal (N = 12), impaired (N = 12) or diabetic (N = 6) glucose toleranc

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34

Goldfine, Allison B., Robert W. Gerwien, Janice A. Kolberg, et al. "Biomarkers in Fasting Serum to Estimate Glucose Tolerance, Insulin Sensitivity, and Insulin Secretion." Clinical Chemistry 57, no. 2 (2011): 326–37. http://dx.doi.org/10.1373/clinchem.2010.156133.

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BACKGROUND Biomarkers for estimating reduced glucose tolerance, insulin sensitivity, or impaired insulin secretion would be clinically useful, since these physiologic measures are important in the pathogenesis of type 2 diabetes mellitus. METHODS We conducted a cross-sectional study in which 94 individuals, of whom 84 had 1 or more risk factors and 10 had no known risk factors for diabetes, underwent oral glucose tolerance testing. We measured 34 protein biomarkers associated with diabetes risk in 250-μL fasting serum samples. We applied multiple regression selection techniques to identify the

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35

Panicke, L., R. Staufenbiel, O. Burkert, E. Fischer, and F. Reinhardt. "Zusammenhang zwischen Parametern des Glukosetoleranztestes bei Jungbullen und deren Nachkommenzuchtwert." Archives Animal Breeding 43, no. 3 (2000): 231–40. http://dx.doi.org/10.5194/aab-43-231-2000.

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Abstract. Title of the paper: Relationships between parameters of the glucose tolerance test by young sires and estimated their breeding values A high milk Performance connected to a sound health regarding metabolism and a sufficient fertility in dairy cows depends on a well balanced distribution of energy in body. The terms "type of tumover" and "type of deposition" embody two extremes of food energy preferably for milk production or for body composition. Insulin plays an outstanding role based on its central position in energetic metabolism. The function of insulin may be recorded by means o

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36

Abbasi, Fahim, Paul JW Tern, and Gerald M. Reaven. "Plasma glucose concentration 60 min post oral glucose load and risk of type 2 diabetes and cardiovascular disease: Pathophysiological implications." Diabetes and Vascular Disease Research 16, no. 4 (2019): 337–43. http://dx.doi.org/10.1177/1479164119827239.

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Aim: The aim of this study was to gain insight into the pathophysiological significance of elevated plasma glucose concentrations (mmol/L) 60 min post oral glucose load in apparently healthy individuals. Methods: Comparison of resistance to insulin action and associated cardio-metabolic risk factors in 490 apparently healthy persons, subdivided into those with a plasma glucose concentration 60 min following a 75-g oral glucose challenge of <8.6 versus ⩾8.6. Results: Insulin resistance was significantly greater in persons with normal glucose tolerance whose 60-min glucose concentration was ⩾

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37

Koop, Brenda L., Alan G. Harris, and Shereen Ezzat. "Effect of octreotide on glucose tolerance in acromegaly." European Journal of Endocrinology 130, no. 6 (1994): 581–86. http://dx.doi.org/10.1530/eje.0.1300581.

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Koop BL, Harris AG. Ezzat S. Effect of octreotide on glucose tolerance in acromegaly. Eur J Endocrinol 1994;130:581–6. ISSN 0804–4643 To determine the effect of the somatostatin analog octreotide on glucose tolerance in acromegaly, we examined glucose profiles, oral glucose tolerance and the insulinogenic index in patients treated with this analog. Ninety patients participated in a long-term, prospective, open-label study. There was no significant change between mean daily blood glucose profiles at baseline or during octreotide treatment. Using glucose tolerance test criteria, 61% of 90 patien

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38

Swaminathan, Anandini, Indrė Libnickienė, Tomas Venckūnas, and Hans Degens. "Comparison of Glucose Administration Routes to Determine Glucose Tolerance in Old and Young Mice." Baltic Journal of Sport and Health Sciences 3, no. 126 (2022): 16–21. http://dx.doi.org/10.33607/bjshs.v3i126.1274.

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Background: It has been observed that old mice have a better tolerance to glucose than young after an intraperitoneal injection of glucose. We hypothesized that an intraperitoneal injection of glucose overestimates glucose tolerance in old mice. Methods: We assessed differences in glucose tolerance outcomes in 2- (young, n=23) and 23- to 27-month-old (old, n=23) male C57BL/6J mice after an intraperitoneal (IP) or intravenous (IV) injection, or oral gavage (OG) of glucose. Results: The area under the curve (AUC) of the changes of blood glucose concentration over 2 hours after glucos

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39

Havalko, Anna, Ervin Asanov, and Valeri Shatilo. "Response of some indicators of the respiratory system to dosed hypoxia in elderly people with impaired glucose tolerance." Volume 3 Issue 1 2022, no. 1 2022 (April 18, 2022): 27–31. http://dx.doi.org/10.47855/10.47855/jal9020-2022-1-4.

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Abstract. The response of the respiratory system to dosed hypoxia (breathing with a gas mixture of 12% oxygen for 20 min) in the elderly with impaired (n = 35) and preserved glucose tolerance (n = 33) was studied. It is shown that the increase in lung ventilation occurs regardless of the state of carbohydrate metabolism in hypoxia. In people with impaired glucose tolerance, changes in lung ventilation in hypoxia are less significant than in people with persistent glucose tolerance. In persons with impaired glucose tolerance, an inverse relationship was found between the increase in pulmonary v

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40

Bajaj, Harpreet S., Chang Ye, Anthony J. Hanley, Mathew Sermer, Bernard Zinman, and Ravi Retnakaran. "Biomarkers of vascular injury and endothelial dysfunction after recent glucose intolerance in pregnancy." Diabetes and Vascular Disease Research 15, no. 5 (2018): 449–57. http://dx.doi.org/10.1177/1479164118779924.

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Objective: Women with gestational diabetes mellitus and milder gestational impaired glucose intolerance have elevated future risks of type 2 diabetes and cardiovascular disease. However, it is unclear whether they show postpartum evidence of vascular injury/dysfunction, an early event in the natural history of cardiovascular disease. Methods: In total, 337 women underwent a glucose challenge test and oral glucose tolerance test in pregnancy, yielding four gestational glucose tolerance groups: gestational diabetes mellitus, gestational impaired glucose intolerance, abnormal glucose challenge te

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41

&NA;. "Protease inhibitors impair glucose tolerance." Reactions Weekly &NA;, no. 738 (1999): 5. http://dx.doi.org/10.2165/00128415-199907380-00007.

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&NA;. "Protease inhibitors impair glucose tolerance." Inpharma Weekly &NA;, no. 1174 (1999): 21. http://dx.doi.org/10.2165/00128413-199911740-00042.

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43

Kidd, G. S., G. E. Graff, B. F. Davies, M. T. McDermott, J. L. Aeling, and F. D. Hofeldt. "Glucose Tolerance in Granuloma Annulare." Diabetes Care 8, no. 4 (1985): 380–84. http://dx.doi.org/10.2337/diacare.8.4.380.

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44

Mobin, Ahsan, Imtiaz Manzoor, Jawahar Lal, and Jawahar Kumar. "HYPERTENSIVE PATIENTS; IMPAIRED GLUCOSE TOLERANCE." Professional Medical Journal 24, no. 10 (2017): 1480–83. http://dx.doi.org/10.17957/tpmj/17.4077.

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45

Machtey, I. "Tendinitis and Abnormal Glucose Tolerance." Scandinavian Journal of Rheumatology 15, no. 2 (1986): 224. http://dx.doi.org/10.3109/03009748609102093.

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46

Reaven, P. D., E. L. Barrett-Connor, and D. K. Browner. "Abnormal Glucose Tolerance and Hypertension." Diabetes Care 13, no. 2 (1990): 119–25. http://dx.doi.org/10.2337/diacare.13.2.119.

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47

Laposata, Michael, Donna H. MacMillan, and Jirina Vasek. "Reflex Testing; Glucose Tolerance Testing." Laboratory Medicine 29, no. 10 (1998): 595–97. http://dx.doi.org/10.1093/labmed/29.10.595.

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48

Marre, M., C. Gauville, and Ph Passa. "Calcium Antagonists and Glucose Tolerance." Journal of Hypertension 3, no. 5 (1985): 540. http://dx.doi.org/10.1097/00004872-198510000-00035.

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&NA;. "CS-045 improves glucose tolerance." Inpharma Weekly &NA;, no. 815 (1991): 8. http://dx.doi.org/10.2165/00128413-199108150-00022.

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50

Prasad, M. C., and P. L. Kaul. "Glucose Tolerance Test in Goats." Zentralblatt für Veterinärmedizin Reihe A 28, no. 9-10 (2010): 732–36. http://dx.doi.org/10.1111/j.1439-0442.1981.tb01244.x.

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