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Relevant bibliographies by topics / Glucose tolerance

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Academic literature on the topic 'Glucose tolerance'

Author: Grafiati

Published: 4 June 2021

Last updated: 9 March 2023

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Journal articles on the topic "Glucose tolerance"

1

Costa, ??ngels, Ignacio Conget, and Ramon Gomis. "Impaired Glucose Tolerance." Treatments in Endocrinology 1, no. 4 (2002): 205–10. http://dx.doi.org/10.2165/00024677-200201040-00001.

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Newman, Byron Y. "Impaired glucose tolerance." Optometry - Journal of the American Optometric Association 78, no. 12 (December 2007): 622–23. http://dx.doi.org/10.1016/j.optm.2007.11.001.

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Davies, M. J., and I. P. Gray. "Impaired glucose tolerance." BMJ 312, no. 7026 (February 3, 1996): 264–65. http://dx.doi.org/10.1136/bmj.312.7026.264.

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Yudkin, J. S., K. G. Alberti, D. G. McLarty, and A. B. Swai. "Impaired glucose tolerance." BMJ 301, no. 6749 (September 1, 1990): 397–402. http://dx.doi.org/10.1136/bmj.301.6749.397.

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Shen, Xiaoxia, Ping Zhang, Jinping Wang, Yali An, Edward W. Gregg, Bo Zhang, Hui Li, et al. "Influence of improvement or worsening of glucose tolerance on risk of stroke in persons with impaired glucose tolerance." International Journal of Stroke 13, no. 9 (June 29, 2018): 941–48. http://dx.doi.org/10.1177/1747493018784432.

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Background and aim We sought to determine the effect of regression to normal glucose tolerance (NGT) or progression to diabetes in early years of impaired glucose tolerance (IGT) on subsequent risk of stroke. Methods In 1986, 576 adults aged 25 years and older with impaired glucose tolerance in Da Qing, China, were randomly assigned by clinic to control, diet, exercise, or diet plus exercise intervention groups for a six-year period. Subsequently participants received medical care in their local clinics. We tracked participants for additional 17 years to ascertain stroke events and other outcomes. Results At the end of 6-year intervention trial follow-up, 272 (50.2%) had progressed to diabetes, 169 (31.2%) regressed to normal glucose tolerance, and 101 (18.6%) remained impaired glucose tolerance. During the subsequent 17-year follow-up, 173 (31.9%) developed a stroke, 26.7% of normal glucose tolerances, 30.7% of impaired glucose tolerances, and 36.1% of those with diabetes. After controlling for age, sex, baseline blood pressure, smoking, total cholesterol, previous cardiovascular disease and intervention group, those who developed diabetes in the first six years had a higher incidence of stroke than those who reverted to normal glucose tolerance (HR = 1.49, 95% CI 1.01–2.19, p = 0.04), whereas for those who remained impaired glucose tolerance compared to those who regressed to normal glucose tolerance the HR was 1.25 (95% CI 0.80–1.93; p = 0.30). A 1-mmol/L increase in both fasting and 2-h post-load plasma glucose from entry to end of the six-year trial was significantly associated with a higher risk of development of stroke in the subsequent 17 years, respectively (HR = 1.07, 95% CI 1.03–1.11, p < 0.0001 for fasting glucose, HR = 1.05, 95% CI 1.02–1.09, p = 0.007 for 2-h post-load plasma glucose). Conclusions Among Chinese adults with impaired glucose tolerance, early progression to diabetes predicted a higher risk of stroke, compared those who regressed to normal glucose tolerance.

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Berkus, M. D., E. M. J. Xenakis, and O. Langer. "Glucose tolerance test periodicity as a descriptor of glucose tolerance abnormality." International Journal of Gynecology & Obstetrics 40, no. 2 (February 1993): 187. http://dx.doi.org/10.1016/0020-7292(93)90423-t.

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Santos, Maria Luiza dos, Flávio F. Aragon, Carlos R. Padovani, and Walkyria P. Pimenta. "Daytime variations in glucose tolerance in people with impaired glucose tolerance." Diabetes Research and Clinical Practice 74, no. 3 (December 2006): 257–62. http://dx.doi.org/10.1016/j.diabres.2006.04.007.

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Diamond, Michael P., Subodhsingh Chauhan, Michael Kruger, and Marappa Subramanian. "Values of fasting glucose levels, glucose tolerance tests, and glucose-insulin ratios as predictors of glucose tolerance." Fertility and Sterility 80, no. 4 (October 2003): 1022–25. http://dx.doi.org/10.1016/s0015-0282(03)01016-1.

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Foley, J. E., P. Thuillez, S. Lillioja, J. Zawadzki, and C. Bogardus. "Insulin sensitivity in adipocytes from subjects with varying degrees of glucose tolerance." American Journal of Physiology-Endocrinology and Metabolism 251, no. 3 (September 1, 1986): E306—E310. http://dx.doi.org/10.1152/ajpendo.1986.251.3.e306.

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Previous studies showed that the sensitivity of glucose transport to insulin is lower in adipocytes isolated from subjects with noninsulin-dependent diabetes mellitus and impaired glucose tolerance compared with subjects with normal glucose tolerance. This study analyzed the relationship between insulin sensitivity of glucose transport and glycemia in a large group of nondiabetic-nonglucose-intolerant subjects with a wide range of glycemic response to oral glucose. Seventy-four Pima Indians with 2-h postglucose load glucoses between 77 and 197 mg/100 ml, fasting plasma glucoses between 76 and 108 mg/100 ml, and no postload glucoses less than 199 mg/100 ml were studied. Isolated adipocytes were prepared in vitro after an abdominal fat biopsy, ED50 of insulin for glucose transport was correlated with 2-h postload glucoses, but not between insulin binding per cell or per cell surface area or in ED50 of insulin for antilipolysis and 2-h postglucose load glucoses. Although only 17% of the variation in glucose tolerance could be explained by a change in the sensitivity of glucose transport to insulin, the data suggests that a postinsulin-binding defect in the coupling of insulin binding to glucose transport may be an early step in the development of insulin resistance in human adipocytes.

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Stout, Robert W. "Glucose Tolerance and Ageing." Journal of the Royal Society of Medicine 87, no. 10 (October 1994): 608–9. http://dx.doi.org/10.1177/014107689408701015.

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Hyperglycaemia, impaired glucose tolerance and non-insulin dependent diabetes become progressively more common with advancing age. The mechanism is insensitivity to the actions of insulin at the postreceptor level. Inadequate secretion of insulin and decreased hepatic sensitivity to insulin's action in suppressing glucose output also occur. The age-related changes may be made worse by obesity, renal failure or the ingestion of certain drugs, or may be lessened by increased physical activity.

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Dissertations / Theses on the topic "Glucose tolerance"

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Dobson, Lee. "Glucose tolerance in cystic fibrosis." Thesis, University of Exeter, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.403679.

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Legate, Nicola J. "Glucose tolerance in 3 teleost species." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp01/MQ45235.pdf.

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Berrish, Taher S. "Metabolic heterogeneity in impaired glucose tolerance." Thesis, University of Newcastle Upon Tyne, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.321310.

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Krishnaveni, Ghattu Vedamurthy. "Anthropometry, glucose tolerance and insulin concentrations in South Indian children : relationships to maternal glucose tolerance during pregnancy." Thesis, University of Southampton, 2005. https://eprints.soton.ac.uk/210920/.

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Earlier studies have shown that individuals whose mothers were diabetic when they were in utero, have an increased risk of early obesity, and impaired glucose tolerance (lGT) and type 2 diabetes in adult life. This study was designed to test whether adiposity, glucose tolerance and insulin concentrations are altered in Indian children born to mothers with gestational diabetes (GDM), and are related to maternal glucose and insulin concentrations in pregnancy even in the absence of GDM. 830 pregnant women attending the antenatal clinics of the Holdsworth Memorial Hospital (HMH), Mysore, India underwent an Oral Glucose Tolerance Test (OGTT) at 30+/-2 weeks. 674 of these women delivered at HMH. Detailed anthropometry was performed on the offspring at birth, and annually thereafter. 585 mothers returned with their offspring at 5 years of age for detailed investigations including OGTT for glucose and insulin concentrations, bio-impedance for fat estimation and blood pressure measurement. OGTT was administered to mothers and fasting plasma glucose and insulin concentrations were measured in fathers. The Mysore babies were small compared to UK neonates, but the deficit varied for different body measurements. While birthweight (-1.1 SD) was considerably lower, crown-heel length (-0.3 SD) and subscapular skinfold thickness (-0.2 SD) were relatively spared. At five years, subscapular skinfold thickness was larger than the UK standards (+0.23 SD, p

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Henareh, Loghman. "Impaired glucose tolerance in ischemic heart disease /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-445-7/.

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Page, Renee C. L. "Detection and treatment of impaired glucose tolerance." Thesis, University of Manchester, 1991. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.633671.

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勞子僖 and Tzu-hsi Terence Lao. "The obstetric implications of gestational impaired glucose tolerance." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2002. http://hub.hku.hk/bib/B31981793.

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Kushnir, О. Yu. "Glucose tolerance profiles in rats with alloxan diabetes." Thesis, БДМУ, 2020. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/18366.

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Lao, Tzu-hsi Terence. "The obstetric implications of gestational impaired glucose tolerance." Hong Kong : University of Hong Kong, 2002. http://sunzi.lib.hku.hk/hkuto/record.jsp?B24463863.

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McGarry, Robert Gerard. "Modelling insulin/glucose dynamics and application to the analysis of oral glucose tolerance tests." Thesis, Queen's University Belfast, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.335562.

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Books on the topic "Glucose tolerance"

1

Hatfield, Frederick C. Training & the glucose tolerance factor. Woodland Hills, CA: Weider Health & Fitness, 1990.

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2

Pasqualina, Santaguida, United States. Agency for Healthcare Research and Quality., and McMaster University. Evidence-based Practice Center., eds. Diagnosis, prognosis, and treatment of impaired glucose tolerance and impaired fasting glucose. [Rockville, Md: Agency for Healthcare Research and Quality, 2005.

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Perus̆ic̆ová, Jindra. Glucose tolerance and secretion of insulin in chronic pancreatitis. Praha: Univerzita Karlova, 1990.

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National Diabetes Information Clearinghouse (U.S.), ed. Diagnosis of diabetes. [Bethesda, Md.]: National Institute of Diabetes and Digestive and Kidney Diseases, National Institutes of Health, U.S. Dept. of Health and Human Services, 2004.

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5

National Institutes of Health (U.S.), ed. From impaired glucose tolerance to diabetes: A two-step process. Bethesda, Md: National Institutes of Health, 1989.

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6

Nathan, David M. Beating diabetes: The first complete program clinically proven to dramatically improve your glucose tolerance. New York: McGraw-Hill, 2005.

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Hadden, Wilbur Crane. The prevalence of diagnosed diabetes, undiagnosed diabetes, and impaired glucose tolerance in adults 20-74 years of age, United States, 1976-80. Hyattsville, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Center for Health Statistics, 1987.

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Parker, Philip M., and James N. Parker. Glucose test: A medical dictionary, bibliography, and annotated research guide to internet references. San Diego, CA: ICON Health Publications, 2004.

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9

Feuer, Joshua Paul. Assessment of acculturation and its associations with type 2 diabetes, impaired glucose tolerance and obesity in an isolated Canadian Aboriginal community. Ottawa: National Library of Canada, 2001.

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10

Mehling, Christine. Comparison of low glycemic index high carbohydrate, high glycemic index high carbohydrate and monounsaturated fat enriched diets on insulin sensitivity in the treatment of impaired glucose tolerance. Ottawa: National Library of Canada, 2000.

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Book chapters on the topic "Glucose tolerance"

1

Mullan, Barbara. "Impaired Glucose Tolerance." In Encyclopedia of Behavioral Medicine, 1157–59. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-39903-0_1143.

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Boltz, Marie, Holly Rau, Paula Williams, Holly Rau, Paula Williams, Jane Upton, Jos A. Bosch, et al. "Impaired Glucose Tolerance." In Encyclopedia of Behavioral Medicine, 1041–42. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1005-9_1143.

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Lascelles, P. T., and D. Donaldson. "Glucose Tolerance Test (GTT)." In Diagnostic Function Tests in Chemical Pathology, 74–76. Dordrecht: Springer Netherlands, 1989. http://dx.doi.org/10.1007/978-94-009-1846-7_39.

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Jensen, Chad D., Amy F. Sato, Elissa Jelalian, Elizabeth R. Pulgaron, Alan M. Delamater, Chad D. Jensen, Amy F. Sato, et al. "Oral Glucose Tolerance Test (OGTT)." In Encyclopedia of Behavioral Medicine, 1389. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1005-9_769.

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Lascelles, P. T., and D. Donaldson. "Glucose Tolerance Test (GTT)6." In Diagnostic Function Tests in Chemical Pathology, 72–73. Dordrecht: Springer Netherlands, 1989. http://dx.doi.org/10.1007/978-94-009-1846-7_38.

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Vincent, John B. "Chromium and Glucose Tolerance Factor." In Encyclopedia of Metalloproteins, 603–7. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-1533-6_19.

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Carrillo, Adriana. "Oral Glucose Tolerance Test (OGTT)." In Encyclopedia of Behavioral Medicine, 1567. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-39903-0_769.

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Kumar, Vijay, and Kiran Dip Gill. "To Perform Glucose Tolerance Test." In Basic Concepts in Clinical Biochemistry: A Practical Guide, 63–66. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-8186-6_15.

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Elahi, Dariush, Denis C. Muller, Josephine M. Egan, Reubin Andres, Johannes D. Veldhuis, and Graydon S. Meneilly. "Glucose Tolerance, Glucose Utilization and Insulin Secretion in Ageing." In Endocrine Facets of Ageing, 222–46. Chichester, UK: John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/0470846542.ch14.

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Jain, Aakanchha, Richa Jain, and Sourabh Jain. "To Perform Oral Glucose Tolerance Test." In Basic Techniques in Biochemistry, Microbiology and Molecular Biology, 211–12. New York, NY: Springer US, 2020. http://dx.doi.org/10.1007/978-1-4939-9861-6_48.

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Conference papers on the topic "Glucose tolerance"

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Wahyu Asrizal, Cynthia, and Bambang Purwanto. "Does Sequential Diabetes Dance Improve on Glucose Level and Glucose Tolerance?" In Surabaya International Physiology Seminar. SCITEPRESS - Science and Technology Publications, 2017. http://dx.doi.org/10.5220/0007332200330036.

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Dritsas, Elias, Sotiris Alexiou, Ioannis Konstantoulas, and Konstantinos Moustakas. "Short-term Glucose Prediction based on Oral Glucose Tolerance Test Values." In 15th International Conference on Health Informatics. SCITEPRESS - Science and Technology Publications, 2022. http://dx.doi.org/10.5220/0010974200003123.

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Ke, H., T. Yang, and G. Li. "Effect of Blood Glucose Tolerance on Pulmonary Function." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a3247.

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Hassoun, Shaimaa, Meis Alkasem, Zainab Dabbous, Amin Jayyousi, Abdul-bari Bener, Abdul-badi Abou Samra, Mahmoud Zirie, and Muhammad Abdul-ghani. "Pathophysiological Features Of Impaired Fasting Glucose (IFG) And Impaired Glucose Tolerance (IGT) In Arab Individuals." In Qatar Foundation Annual Research Conference Proceedings. Hamad bin Khalifa University Press (HBKU Press), 2014. http://dx.doi.org/10.5339/qfarc.2014.hbpp0435.

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Chierici, Marco, Gianluigi Pillonetto, Gianna Toffolo, and Claudio Cobelli. "Glucose Production by Deconvolution in Intravenous and Oral Glucose Tolerance Tests: Role of Output Variable." In Conference Proceedings. Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2006. http://dx.doi.org/10.1109/iembs.2006.259961.

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Chierici, Marco, Gianluigi Pillonetto, Gianna Toffolo, and Claudio Cobelli. "Glucose Production by Deconvolution in Intravenous and Oral Glucose Tolerance Tests: Role of Output Variable." In Conference Proceedings. Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2006. http://dx.doi.org/10.1109/iembs.2006.4398587.

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Severeyn, Erika, Sara Wong, Jesus Velasquez, Hector Herrera, Gilberto Perpinan, and Monica Huerta. "Analysis of Insulin Receiver Operating Characteristic Curve for Impaired Glucose Tolerance and Impaired Fasting Glucose Diagnosis." In 2019 IEEE Fourth Ecuador Technical Chapters Meeting (ETCM). IEEE, 2019. http://dx.doi.org/10.1109/etcm48019.2019.9014901.

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Huang, Ke, Ting Yang, and Guangwei Li. "Decreased pulmonary function tests are affected by blood glucose tolerance." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.132.

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Sekizuka, Tomomi, Tomotaka Kawayama, Hidenobu Ishii, Kosuke Ito, Kazuko Matsunaga, Tomoaki Hoshino, and Hisamichi Aizawa. "Impairment Of Glucose Tolerance In Subjects With COPD In Japan." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a5937.

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Perpi�an, Gilberto, Erika Severeyn, Sara Wong, and Miguel Altuve. "Nonlinear Heart Rate Variability Measures During the Oral Glucose Tolerance Test." In 2017 Computing in Cardiology Conference. Computing in Cardiology, 2017. http://dx.doi.org/10.22489/cinc.2017.148-302.

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Reports on the topic "Glucose tolerance"

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Browdy, Craig, and Esther Lubzens. Cryopreservation of Penaeid Shrimp Embryos: Development of a Germplasm Cryo-Bank for Preservation of High Health and Genetically Improved Stocks. United States Department of Agriculture, August 2002. http://dx.doi.org/10.32747/2002.7695849.bard.

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The objectives of the project were to develop a successful protocol for cryopreservation of penaeid germ plasm in order to preserve a pathogen-free broodstock nucleus for commercial exploitation of marine shrimp in aquaculture. The critical parameters to be characterized in the project were: 1. Determination of chill sensitivity and chill tolerant embryonic stages, including a full description and time course study of embryonic developmental stages. 2. Development of protocols for loading and removal of cryoprotectant agents (CPAs) from embryos; determination of optimal concentrations and duration of loading. 3. Characterization of the toxicity of the selected CP As and 4. Establishing optimal cooling and thawing procedures. Studies were performed on two penaeid species: Litopenaeus vannamei (in the USA) and P. semisulcatus (in Israel). The effect of incubation temperature on embryonic development rate and hatching success was studied in L. vannamei, showing that spawns maybe maintained at temperatures ranging from 24°C to 30°C, without compromising hatchability. Embryonic development extends from 12 hr to 19 hr at 30°C and 24°C, respectively. Studies showed that advanced embryonic developmental stages were chill tolerant in the two studied species, but P. semisulcatus could better endure lower temperatures than L. vannamei. A large number of experiments were performed to determine the optimal CP As, their concentration and duration of loading. Permeating (e.g. glycerol, methanol, DMSO, 1,2- propanediol, ethylene glycol, glucose) and non-permeating CPAs (sucrose, PVP, polyethylene glycol) were tested and several combinations of permeating and non-permeating CP As, on fertilized eggs (embryos), nauplii and protozoeae. In general, nauplii tolerated higher CPA concentrations than eggs and nauplii were also more permeable to radiolabeled methanol. Chlorine treatment intended to remove the chitinous envelop from eggs, did not increase dramatically the permeation of radiolabled methanol into eggs. Cooling eggs, nauplii or protozoeae to cryogenic temperatures, by either vitrification or slow cooling protocols, did not result in full survival of thawed samples, despite exhaustive attempts testing various protocols and CP As. Results seemed more encouraging in freezing of nauplii in comparison to eggs or protozoeae. Successful preliminary results in cryopreservation of spermatozoa of P. vannamei, will facilitate preservation of genetic specific to some extent.

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Hochman, Ayala, Thomas Nash III, and Pamela Padgett. Physiological and Biochemical Characterization of the Effects of Oxidant Air Pollutants, Ozone and Gas-phase Nitric Acid, on Plants and Lichens for their Use as Early Warning Biomonitors of these Air Pollutants. United States Department of Agriculture, January 2011. http://dx.doi.org/10.32747/2011.7697115.bard.

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Introduction. Ozone and related oxidants are regarded as the most important phytotoxic air pollutant in many parts of the western world. A previously unrecognized component of smog, nitric acid, may have even greater deleterious effects on plants either by itself or by augmenting ozone injury. The effects of ozone on plants are well characterized with respect to structural and physiological changes, but very little is known about the biochemical changes in plants and lichens exposed to ozone and/or HNO3. Objectives.To compare and contrast the responses of crop plants and lichens to dry deposition of HNO3 and O3., separately, and combined in order to assess our working hypothesis that lichens respond to air pollution faster than plants. Lichens are most suitable for use as biomonitors because they offer a live-organism-based system that does not require maintenance and can be attached to any site, without the need for man-made technical support systems. Original Immediate aims To expose the tobacco (Nicotiana tabacum L.) cultivar Bel-W3 that is ozone supersensitive and the ozone sensitive red kidney bean (Phaseolusvulgaris) and the lichen Ramalinamenziesii to controlled HNO3 and O3 fumigations and combined and to follow the resulting structural, physiological and biochemical changes, with special reference to reactive oxygen species related parameters. Revised. Due to technical problems and time limitations we studied the lichen Ramalinamenziesii and two cultivar of tobacco: Bel-W3 that is ozone supersensitive and a resistant cultivar, which were exposed to HNO3 and O3 alone (not combined). Methodology. Plants and lichens were exposed in fumigation experiments to HNO3 and O3, in constantly stirred tank reactors and the resulting structural, physiological and biochemical changes were analyzed. Results. Lichens. Exposure of Ramalinamenziesiito HNO3 resulted in cell membrane damage that was evident by 14 days and continues to worsen by 28 days. Chlorophyll, photosynthesis and respiration all declined significantly in HNO3 treatments, with the toxic effects increasing with dosage. In contrast, O3 fumigations of R. menziesii showed no significant negative effects with no differences in the above response variables between high, moderate and low levels of fumigations. There was a gradual decrease in catalase activity with increased levels of HNO3. The activity of glutathione reductase dropped to 20% in thalli exposed to low HNO3 but increased with its increase. Glucose 6-phosphate dehydrogenase activity increase by 20% with low levels of the pollutants but decreased with its increase. Tobacco. After 3 weeks of exposure of the sensitive tobacco cultivar to ozone there were visible symptoms of toxicity, but no danmage was evident in the tolerant cultivar. Neither cultivar showed any visible symptoms after exposure to HNO3.In tobacco fumigated with O3, there was a significant decrease in maximum photosynthetic CO2 assimilation and stomatal conductance at high levels of the pollutant, while changes in mesophyll conductance were not significant. However, under HNO3 fumigation there was a significant increase in mesophyll conductance at low and high HNO3 levels while changes in maximum photosynthetic CO2 assimilation and stomatal conductance were not significant. We could not detect any activity of the antioxidant enzymes in the fumigated tobacco leaves. This is in spite of the fact that we were able to assay the enzymes in tobacco leaves grown in Israel. Conclusions. This project generated novel data, and potentially applicable to agriculture, on the differential response of lichens and tobacco to HNO3 and O3 pollutants. However, due to experimental problems and time limitation discussed in the body of the report, our data do not justify yet application for a full, 4-year grant. We hope that in the future we shall conduct more experiments related to our objectives, which will serve as a basis for a larger scale project to explore the possibility of using lichens and/or plants for biomonitoring of ozone and nitric acid air pollution.

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