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Relevant bibliographies by topics / Gliomas

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Academic literature on the topic 'Gliomas'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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Journal articles on the topic "Gliomas"

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Soukup, Jiri, Lucie Gerykova, Anjali Rachelkar, et al. "Diagnostic Utility of Immunohistochemical Detection of MEOX2, SOX11, INSM1 and EGFR in Gliomas." Diagnostics 13, no. 15 (2023): 2546. http://dx.doi.org/10.3390/diagnostics13152546.

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Histological identification of dispersed glioma cells in small biopsies can be challenging, especially in tumours lacking the IDH1 R132H mutation or alterations in TP53. We postulated that immunohistochemical detection of proteins expressed preferentially in gliomas (EGFR, MEOX2, CD34) or during embryonal development (SOX11, INSM1) can be used to distinguish reactive gliosis from glioma. Tissue microarrays of 46 reactive glioses, 81 glioblastomas, 34 IDH1-mutant diffuse gliomas, and 23 gliomas of other types were analysed. Glial neoplasms were significantly more often (p < 0.001, χ2) positi

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Olaofe, O. O., B. A. Adewara, C. C. Okongwu, and E. E. Ewoye. "Case report: A case report of optic nerve glioma in a 5-year-old African girl." Research Journal of Health Sciences 12, no. 1 (2024): 82–87. http://dx.doi.org/10.4314/rejhs.v12i1.10.

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Background: Optic nerve gliomas are rare tumors that constitute less than 5% of all pediatric central nervous system tumors. Case Presentation: We present the case of a 5-year-old girl with complaints of progressive protrusion of the right eye and poor vision of 3 years duration who was referred to the ophthalmologist. Ophthalmological evaluation of the right eye revealed tearing, no perception of light, inferior dystopia, severe proptosis, and a firm non-tender orbital mass palpable posterior to the eyeball. The cranial CT-scan was suggestive of a right optic nerve glioma. Histology sho

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Hewer, Ekkehard, Jaison Phour, Marielena Gutt-Will, Philippe Schucht, Matthias S. Dettmer, and Erik Vassella. "TERT Promoter Mutation Analysis to Distinguish Glioma From Gliosis." Journal of Neuropathology & Experimental Neurology 79, no. 4 (2020): 430–36. http://dx.doi.org/10.1093/jnen/nlaa004.

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Abstract Among the most challenging diagnostic issues in surgical neuropathology is the distinction between scant infiltration by diffuse gliomas and reactive gliosis. The best documented ancillary marker to establish a definitive diagnosis of glioma in this setting is the identification of hotspot mutations in the isocitrate dehydrogenase 1 and 2 (IDH1/IDH2) genes, which is limited, however, by the low prevalence of these mutations in gliomas of elderly adults. Since telomerase reverse transcriptase (TERT) promoter mutations are present in the vast majority of IDH-wildtype diffuse gliomas, we

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Park, Jong-Whi. "Metabolic Rewiring in Adult-Type Diffuse Gliomas." International Journal of Molecular Sciences 24, no. 8 (2023): 7348. http://dx.doi.org/10.3390/ijms24087348.

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Multiple metabolic pathways are utilized to maintain cellular homeostasis. Given the evidence that altered cell metabolism significantly contributes to glioma biology, the current research efforts aim to improve our understanding of metabolic rewiring between glioma’s complex genotype and tissue context. In addition, extensive molecular profiling has revealed activated oncogenes and inactivated tumor suppressors that directly or indirectly impact the cellular metabolism that is associated with the pathogenesis of gliomas. The mutation status of isocitrate dehydrogenases (IDHs) is one of the mo

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Xia, He-chun, Zhan-feng Niu, Hui Ma, et al. "Deregulated Expression of the Per1 and Per2 in Human Gliomas." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 37, no. 3 (2010): 365–70. http://dx.doi.org/10.1017/s031716710001026x.

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Background:Growing evidence shows that the deregulation of the circadian clock plays an important role in the development of malignant tumors, including gliomas. However, the molecular mechanisms of genes controlling circadian rhythm in glioma cells have not been explored.Methods:Using reverse transcription polymerase chain reaction and immunohistochemistry techniques, we examined the expression of two important clock genes, Per1 and Per2, in 33 gliomas.Results:In this study, out of 33 gliomas, 28 were Per1-positive, and 23 were Per2-positive. The expression levels of Per1 and Per2 in glioma c

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Sánchez, Manuel Lisardo, Arturo Mangas, and Rafael Coveñas. "Glioma and Peptidergic Systems: Oncogenic and Anticancer Peptides." International Journal of Molecular Sciences 25, no. 14 (2024): 7990. http://dx.doi.org/10.3390/ijms25147990.

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Glioma cells overexpress different peptide receptors that are useful for research, diagnosis, management, and treatment of the disease. Oncogenic peptides favor the proliferation, migration, and invasion of glioma cells, as well as angiogenesis, whereas anticancer peptides exert antiproliferative, antimigration, and anti-angiogenic effects against gliomas. Other peptides exert a dual effect on gliomas, that is, both proliferative and antiproliferative actions. Peptidergic systems are therapeutic targets, as peptide receptor antagonists/peptides or peptide receptor agonists can be administered

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Kim, Junhyung, Min Woo Park, Young Joon Park, et al. "miR-542-3p Contributes to the HK2-Mediated High Glycolytic Phenotype in Human Glioma Cells." Genes 12, no. 5 (2021): 633. http://dx.doi.org/10.3390/genes12050633.

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(1) Background: The elevation of glucose metabolism is linked to high-grade gliomas such as glioblastoma multiforme (GBM). The high glycolytic phenotype is associated with cellular proliferation and resistance to treatment with chemotherapeutic agents in GBM. MicroRNA-542-3p (miR-542-3p) has been implicated in several tumors including gliomas. However, the role of miR-542-3p in glucose metabolism in human gliomas remains unclear; (2) Methods: We measured the levels of cellular proliferation in human glioma cells. We measured the glycolytic activity in miR-542-3p knockdown and over-expressed hu

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Kim, Junhyung, Min Woo Park, Ju Won Ahn, et al. "TAMI-47. MIR-542-3P CONTRIBUTES TO THE HK2-MEDIATED HIGH GLYCOLYTIC PHENOTYPE IN HUMAN GLIOMA CELLS." Neuro-Oncology 23, Supplement_6 (2021): vi208. http://dx.doi.org/10.1093/neuonc/noab196.830.

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Abstract BACKGROUND The elevation of glucose metabolism is linked to high-grade gliomas such as glioblastoma multiforme (GBM). The high glycolytic phenotype is associated with cellular proliferation and resistance to treatment with chemotherapeutic agents in GBM. MicroRNA-542-3p (miR-542-3p) has been implicated in several tumors including gliomas. However, the role of miR-542-3p in glucose metabolism in human gliomas remains unclear. METHODS We measured the levels of cellular proliferation in human glioma cells. We measured the glycolytic activity in miR-542-3p knockdown and over-expressed hum

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Amin, Samirkumar, Kevin Anderson, Beth Bourdreau, et al. "GENE-57. COMPARATIVE MOLECULAR LIFE HISTORY OF SPONTANEOUS CANINE AND HUMAN GLIOMA." Neuro-Oncology 21, Supplement_6 (2019): vi110. http://dx.doi.org/10.1093/neuonc/noz175.459.

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Abstract Diffuse gliomas are the commonest of malignant brain tumors with high-grade tumors carrying dismal prognosis. Preclinical models have proven themselves as poor predictors of clinical efficacy, attributed to the lack of a comparable tumor microenvironment. Comparative genomics of canine and human gliomas provide an attractive alternative modality to identify conserved drivers and underlying mutational processes of glioma as well as evaluate life history tradeoffs related to tissue context and aging that can shape type and relative timing of drivers in gliomagenesis. We performed a comp

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Wirth, Thomas, Farizan Ahmad, Agnieszka Pacholska, Haritha Samaranayake, and Seppo Ylä-Herttuala. "The Syngeneic BT4C Rat Malignant Glioma is a Valuable Model to study Myelomonocytic cells in Tumors." Cancer Growth and Metastasis 5 (January 2012): CGM.S9314. http://dx.doi.org/10.4137/cgm.s9314.

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Background The impact of infiltrating macrophages on tumor progression in malignant gliomas has been studied extensively. However, there is a lack of animal models for studying the role of infiltrating macrophages in malignant gliomas. Material and methods: The BT4C rat malignant glioma model was characterized by immunohistochemical analysis of inflammatory cell types associated with the tumors. Results BT4C malignant gliomas are highly vascularized tumors with an infiltrative behavior. BT4C gliomas demonstrated a high infiltration rate of macrophages. Particularly, a CD68/VEGFR-1 positive sub

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Dissertations / Theses on the topic "Gliomas"

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Lilja, Åsa. "Psychoneurooncology psychological dynamics in glioma patients /." Lund : Dept. of Psychology, Lund University, 1992. http://books.google.com/books?id=SnZrAAAAMAAJ.

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Sadeghi-Meibodi, Niloufar. "Image-based biomarkers for the invivo evaluation of human brain gliomas." Doctoral thesis, Universite Libre de Bruxelles, 2010. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/209991.

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Gliomas constitute 36% of all primary brain tumors and 81% of all primary malignant brain tumors. The overall prognosis in patients with gliomas depends mainly on the location and histologic grade of the tumor.<p>The World Health Organization classification of gliomas is the primary basis for guiding therapy and assessing overall prognosis in gliomas. This classification system, based on histological features, often falls short of predicting therapeutic response of individual tumors within the same histological grade. Yet, it still remains the grading method for both research and clinical pros

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Nanda, Dharminderkoemar. "Gene therapy for gliomas." [S.l.] : Rotterdam : [The Author] ; Erasmus University [Host], 2008. http://hdl.handle.net/1765/13140.

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Noreen, Razia. "FTIR imaging of collagens in gliomas." Thesis, Bordeaux 1, 2011. http://www.theses.fr/2011BOR14316/document.

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Le gliome est le type le plus agressif et mortel de tumeur cérébrale. Ces tumeurs se caractérisent par la présence conjointe de phénotypes solides (de bas grade, moins invasif, hautement vascularisé) et diffus (haut grade, très envahissant et diffus) des glioblastomes multiformes. Les collagènes sont des composants majeurs de la MEC des cellules tumorales des gliomes, et sont également présents dans la membrane basale des vaisseaux sanguins, mais avec une composition différente entre vasculatures saine et tumorale. L'abondance et la typologie des collagènes dans la MEC des cellules tumorales e

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Alentorn, Agusti. "Caractérisation génomique et génétique des gliomes diffus de bas grade de l’adulte." Thesis, Paris 11, 2014. http://www.theses.fr/2014PA11T011.

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La caractérisation moléculaire multidimensionnelle des tumeurs et des tumeurs gliales en particulier est une étape importante pour l’identification de biomarqueurs (diagnostique, pronostique, théranostique et/ou de prédisposition), pour l’identification de cibles thérapeutiques et pour une meilleure compréhension de l’oncogénèse moléculaire.Nos travaux ont permis de confirmer et de consolider certaines données de la littérature comme par exemple : (i) la valeur pronostique favorable de la codélétion 1p/19q, (ii) la valeur pronostique favorable de la mutation IDH, (iii) le caractère mutuellemen

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Fu, Peng. "Membrane Hsp70 expression in gliomas." Diss., lmu, 2012. http://nbn-resolving.de/urn:nbn:de:bvb:19-149571.

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Horst, Maarten ter. "Gene therapy of malignant gliomas." [S.l.] : Rotterdam : [The Author] ; Erasmus University [Host], 2008. http://hdl.handle.net/1765/10864.

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Lau, Cara Jean. "Gene therapy for malignant gliomas." Thesis, McGill University, 2007. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=18478.

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Gliomas are the most common primary brain tumours found in adults. The median survival of patients diagnosed with the most malignant form, glioblastoma multiforme (GBM), is 9-12 months and has changed little over the years despite advances in medical technology. Gene therapy may offer new solutions to treat this resistant disease. Hence, we tested three different gene therapy strategies. In our first study, we tested the efficacy of targeted therapy to correct common aberrations found in gliomas including amplification/mutation of receptor tyrosine kinases (RTK) and loss of PTEN, which result

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Bergthold, Guillaume. "Genomic Profiling of Pediatric Low-Grade Gliomas." Thesis, Paris 11, 2015. http://www.theses.fr/2015PA11T053/document.

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Les gliomes de bas-grade représentent la tumeur cérébrale la plus fréquente chez l’enfant. Elles sont caractérisées par un large spectre de sous-types tumoraux, très hétérogènes. Leur définition actuelle est principalement basée sur des critères histologiques ce qui représente une limite importante car ces classifications souffrent d’un manque de précision. Les progrès récents de la génomique nous permettent d’approfondir considérablement les connaissances sur la biologie de ces tumeurs afin d’enrichir leur classification actuelle. Ce travail présente une analyse approfondie des altérations gé

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Gulati, Sasha. "Surgical Resection of High-Grade Gliomas." Doctoral thesis, Norges teknisk-naturvitenskapelige universitet, Institutt for laboratoriemedisin, barne- og kvinnesykdommer, 2012. http://urn.kb.se/resolve?urn=urn:nbn:no:ntnu:diva-16444.

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Reseksjon av høygradige gliomer Høygradige gliomer er den vanligste formen for primær hjernesvulst. Glioblastomer (Verdens Helseorganisasjon grad IV) og anaplastiske astrocytomer (Verdens Helseorganisasjon grad III) utgjør mellom 70 og 85 % av høygradige gliomer. Høygradige gliomer er assosiert med både høy morbiditet og mortalitet. Nærmest alle pasienter med høygradige gliomer opplever tilbakefall og dør som følge av sykdommen. Til tross for kirurgi, strålebehandling og cellegift, er median overlevelse for pasienter med glioblastom fremdeles under 12 måneder. For pasienter med anaplastisk ast

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Books on the topic "Gliomas"

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von Deimling, Andreas, ed. Gliomas. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-31206-2.

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A, Yung W. K., ed. Cerebral gliomas. Baillière Tindall, 1996.

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S, Berger Mitchel, and Wilson Charles B. 1929-, eds. The gliomas. W.B. Saunders, 1999.

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Schramm, J., ed. Low-Grade Gliomas. Springer Vienna, 2010. http://dx.doi.org/10.1007/978-3-211-99481-8.

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Barnett, Gene H., ed. High-Grade Gliomas. Humana Press, 2007. http://dx.doi.org/10.1007/978-1-59745-185-7.

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Schramm, J. Low-Grade Gliomas. Springer Vienna, 2010.

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1924-, Suzuki Jirō, ed. Treatment of glioma: With 137 figures. Springer-Verlag, 1988.

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Scarabino, Tommaso, and Saverio Pollice, eds. Imaging Gliomas After Treatment. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-31210-7.

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Scarabino, Tommaso, ed. Imaging Gliomas After Treatment. Springer Milan, 2012. http://dx.doi.org/10.1007/978-88-470-2370-3.

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Kurt, Piscol, Klinger M. 1943-, Brock M. 1938-, and Deutsche Gesellschaft für Neurochirurgie. Tagung, eds. Neurosurgical standards, cerebral aneurysms, malignant gliomas. Springer-Verlag, 1992.

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Book chapters on the topic "Gliomas"

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Herholz, Karl. "Gliomas." In PET and SPECT in Neurology. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-54307-4_41.

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Jenkins, Robert. "Gliomas." In Molecular Pathology in Clinical Practice: Oncology. Springer US, 2009. http://dx.doi.org/10.1007/978-0-387-87366-4_13.

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Herholz, Karl. "Gliomas." In PET and SPECT in Neurology. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-53168-3_39.

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Jenkins, Robert. "Gliomas." In Molecular Pathology in Clinical Practice. Springer New York, 2007. http://dx.doi.org/10.1007/978-0-387-33227-7_27.

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Dolgushin, Mikhail, Valery Kornienko, and Igor Pronin. "Gliomas." In Brain Metastases. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-57760-9_28.

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Lavi, Ehud. "Gliomas." In Molecular Pathology in Clinical Practice. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-19674-9_38.

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Timson, David J., Richard J. Reece, James B. Thoden, et al. "Gliomas." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_3134.

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Kim, Jonathan, Sumayya J. Almarzouqi, Michael L. Morgan, and Andrew G. Lee. "Optic Gliomas." In Encyclopedia of Ophthalmology. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-35951-4_1216-1.

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Lin, Tiffany F., and Michael Prados. "Brainstem Gliomas." In Pediatric Oncology. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-30789-3_3.

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Gullotta, F. "Cryptic Gliomas." In Minimal Neoplasia. Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-83245-1_18.

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Conference papers on the topic "Gliomas"

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Aleksandrova, P. V., K. I. Zaytsev, P. V. Nikitin, et al. "OCT-based feature analysis of brain tissue ex vivo: rat gliomas 101.8, C6 and human gliomas of different grades." In 2024 International Conference Laser Optics (ICLO). IEEE, 2024. http://dx.doi.org/10.1109/iclo59702.2024.10624409.

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Ravikumar, H. J. Ananya, N. R. Keerthana, Deepak S. Gowda, M. G. Jeevan, and S. Harisha. "Gliomas Disease Prediction Using CNN and Image Processing Techniques." In 2025 International Conference on Knowledge Engineering and Communication Systems (ICKECS). IEEE, 2025. https://doi.org/10.1109/ickecs65700.2025.11034911.

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Dutra, Milla Giancristofaro, Bernardo Valle Zanetti, Ana Luiza Badini Tubenchlak, et al. "Management of low-grade gliomas." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.052.

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Background: Gliomas are the most aggressive and prevalent primary malignant tumors of the central nervous system. For better mapping, they are subclassified into degrees in proportion to their malignancy. Although low-grade patients have a better prognosis, they are extremely heterogeneous. Since the high variability in the outcomes of the condition, it is essential to investigate the current therapeutic strategies available. Objective: Analyze the management of low-grade gliomas. Methods: In April 2021, a literature review was conducted on MEDLINE using the descriptors: “Glioma”, “Low Grade”;

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FELIN, CAROLLINA DANEZI, GIULLIANO DANEZI FELIN, GIANCARLLO DANEZI FELIN, FELLIPE DANEZI FELIN, and IZABELLA PAZ DANEZI FELIN. "GENÉTICA MOLECULAR DOS GLIOMAS CEREBRAIS: IMPORTÂNCIA NA CLASSIFICAÇÃO, SOBREVIDA E TRATAMENTO." In I Congresso Nacional de Pesquisas e Estudos Genéticos On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/geneticon/9009.

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Introdução: No Brasil os gliomas representam 42% dos tumores cerebrais. Originam-se do parênquima cerebral e tem comportamento variado. Os de alto grau são agressivos e tem sobrevida média global de 15 meses. Os de baixo grau tem sobrevida de 2-5 anos, podem recidivar ou progredir para piores graus. Objetivos: Identificar a genética molecular dos gliomas e sua importância na classificação, sobrevida e tratamento desses tumores. Metodologia: Revisão de literatura através de pesquisa na base de dados MEDLINE, via PubMed, utilizando os termos: “glioma” [and] “molecular genetics” [and] “therapy”.

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Pereira, Edmundo. "Nanotecnologia em gliomas malignos." In XXXII Congresso Brasileiro de Neurocirurgia. Thieme Revinter Publicações Ltda, 2018. http://dx.doi.org/10.1055/s-0038-1672796.

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Ul Ain, Qurat, Iqra Duaa, Komal Haroon, Faisal Amin, and Muhammad Zia ur Rehman. "MRI Based Glioma Detection and Classification into Low-grade and High-Grade Gliomas." In 2021 15th International Conference on Open Source Systems and Technologies (ICOSST). IEEE, 2021. http://dx.doi.org/10.1109/icosst53930.2021.9683838.

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Muller, Paul J., and Brian C. Wilson. "Photodynamic therapy of supratentorial gliomas." In BiOS '98 International Biomedical Optics Symposium, edited by Thomas J. Dougherty. SPIE, 1998. http://dx.doi.org/10.1117/12.308129.

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Modenese, Alberto, Leena Korpinen, and Fabriziomaria Gobba. "934 Gliomas incidence in italy." In 32nd Triennial Congress of the International Commission on Occupational Health (ICOH), Dublin, Ireland, 29th April to 4th May 2018. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/oemed-2018-icohabstracts.1230.

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Muller, Paul J., and Brian C. Wilson. "Photodynamic therapy of supratentorial gliomas." In BiOS '97, Part of Photonics West, edited by Thomas J. Dougherty. SPIE, 1997. http://dx.doi.org/10.1117/12.273505.

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Pinho, Rafaela Seixas, Afonso Moraes Melo Junior, Rafael Silva Lemos, Amanda da Silva Furtado, and Luís Eduardo Werneck de Carvalho. "Gliomas: tumor markers and prognosis." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.538.

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Background: Gliomas are classified based from the molecular parameters involved in their pathogenesis, which influence their prognosis. The parameters are based on the mutation of the genes encoding the enzyme isocitrate dehydrogenase 1 and 2 (IDH1 and IDH2), on the codelection of the arms of chromosome 1p/19q and the promoting hypermethylation of the MGMT gene. Objectives: identify tumor markers related to gliomas and their prognostic values. Methods: integrative review of the literature based on pubmed, lilacs and scielo platforms. Articles published in English, Portuguese and Spanish betwee

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Reports on the topic "Gliomas"

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Keating, Amy. Targeting Pediatric Glioma with Apoptosis and Autophagy Manipulation. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada567861.

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Keating, Amy. Targeting Pediatric Glioma with Apoptosis and Autophagy Manipulation. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada614915.

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Zhang, Hui-Mei, Xiao-Bing Huo, Hua-Long Wang, and Chen Wang. Recurrent glioma and radiation necrosis: a meta-analysis of MRI diagnosis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.12.0028.

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Becher, Oren, and Alex Chung. Genetically Engineered Mouse Model of Diffuse Intrinsic Pontine Glioma as a Preclinical Tool. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada569511.

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Becher, Oren. Genetically Engineered Mouse Model of Diffuse Intrinsic Pontine Glioma as a Preclinical Tool. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada620002.

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Chen, Qian, Yuhua Hu, Haihua Zhan, and Yawei He. The diagnostic value of miR-221/222 in glioma : a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.3.0064.

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Jin, Yin, and Xingchen Ye. Prognostic and clinicopathological role of C-reactive protein in glioma: a meta-analysis of 2,064 patients. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.10.0010.

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Yang, Chao, Tao-junjin Lu, Jie Wang, Qing Zhang, Ze-Fen Wang, and Zhi-Qiang Li. Association of systemic immune-inflammation index with grade and prognosis in glioma patients: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.4.0072.

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Gu, Xindong, Xining He, Hualong Wang, et al. Dynamic susceptibility contrast-enhanced perfusion-weighted imaging in differentiation between recurrence and pseudoprogression in high-grade glioma: a meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.2.0056.

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Mahmoudi, Farhad, Mahtab Mokarram, Sadegh Sabouhi, Sara Hashemi, Parastoo Saberi, and Hadi Zamanian. Application of digital health for improving medication adherence in MS patients. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.10.0058.

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Abstract:

Review question / Objective: The aim of this study is to evaluate the efficacy of digital health interventions in monitoring and improving medication adherence in Multiple Sclerosis patients. Condition being studied: Multiple sclerosis (MS) is the most prevalent chronic inflammatory disease of the central nervous system (CNS), which leads to focal lesions in the white matter, characterized by selective primary demyelination with partial preservation of axons and reactive astrocytic gliosis. The disease is thought to be due to a complex interaction between different genetic and environmental fa

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