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Books on the topic 'Glioma diagnosis'

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Author: Grafiati

Published: 10 December 2022

Last updated: 28 January 2023

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1

Gliomas. Berlin: Springer, 2009.

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2

P, Rock Jack, ed. The practical management of low-grade primary brain tumors. Philadelphia: Lippincott Williams & Wilkins, 1999.

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3

Scarabino, Tommaso. Imaging Gliomas After Treatment: A Case-based Atlas. Milano: Springer Milan, 2012.

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4

Glioma. Springer Verlag, 1991.

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5

Karim, Abul Bashr Mohammed Fazlul. and Laws Edward R, eds. Glioma: Principles and practice in neuro-oncology. Berlin: Springer-Verlag, 1991.

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6

Barnett, Gene H. High-grade Gliomas: Diagnosis and Treatment. Humana P.,U.S., 2006.

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7

Barnett, Gene H. High-Grade Gliomas: Diagnosis and Treatment (Current Clinical Oncology). Humana Press, 2006.

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8

H, Barnett Gene, ed. High-grade gliomas: Diagnosis and treatment. Totowa, N.J: Humana Press, 2007.

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9

High-Grade Gliomas: Diagnosis and Treatment. Humana, 2010.

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10

Friedman, Allan H., Roger E. McLendon, Darell D. Bigner, John H. Sampson, and Henry S. Friedman. Duke Glioma Handbook: Pathology, Diagnosis, and Management. Cambridge University Press, 2016.

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11

Friedman, Allan H., Darell D. Bigner, John H. Sampson, Henry S. Friedman, and Roger McLendon. Duke Glioma Handbook: Pathology, Diagnosis, and Management. Cambridge University Press, 2016.

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12

Friedman, Allan H., Roger E. McLendon, Darell D. Bigner, John H. Sampson, and Henry S. Friedman. Duke Glioma Handbook: Pathology, Diagnosis and Management. Cambridge University Press, 2016.

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13

van den Bent, Martin J., Frederic Dhermain, and Walter Stummer. Other neuroepithelial tumours: astroblastoma, angiocentric glioma, and chordoid glioma. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199651870.003.0007.

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This chapter contains a description of three rare entities: astroblastoma, angiocentric glioma, and chordoid glioma. Because these tumours are so rare, the evidence on how to best treat them is anecdotal and essentially consists of case series, but it is the best guidance available and the presentation of larger series with new cases remain unlikely. If one is confronted with such a case, a meticulous review of the clinical, radiological, and pathological characteristics of the case is indicated, to minimize the risk of an erroneous diagnosis. If the case does not fit in with the descriptions of previous cases, alternative diagnoses should be considered.

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14

Malignant Gliomas. Springer Publishing Company, Incorporated, 2012.

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15

Chakravarti, Arnab, Martin Fuss, and Thomas Charles R. Jr. Malignant Gliomas. Springer Publishing Company, Incorporated, 2012.

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16

Chakravarti, Arnab, and Martin Fuss. Malignant Gliomas: Rmr V3 I2. Springer Publishing Company, Incorporated, 2012.

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17

Taillandier, Luc, Laurent Capelle, and Hugues Duffau. New Therapeutic Strategies in Low-grade Gliomas. Nova Science Publishers, 2006.

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18

Sanai, Nader. Low-Grade Gliomas. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190696696.003.0027.

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Low grade gliomas encompass multiple histologic diagnoses of primary brain tumors, most commonly grade 2 oligodendroglioma and grade 2 astrocytoma. This chapter presents a case of a patient with a left temporal low grade glioma who presented with seizures, which are a common presenting symptom for this tumor type. Management of patients with a newly diagnosed low grade glioma typically begins with maximal safe surgical resection for surgically accessible tumors. Surgical planning may involve functional imaging such as with fMRI. Genetic and molecular markers help distinguish subtypes of low grade gliomas, and this subtyping has implications for the type and timing of adjuvant therapy.

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19

Neuropathology. Philadelphia: Elsevier Churchill-Livingstone, 2005.

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20

Neuropathology. Elsevier - Health Sciences Division, 2011.

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21

Omerhodžić, Ibrahim, and Kenan Arnautović, eds. Glioma - Contemporary Diagnostic and Therapeutic Approaches. IntechOpen, 2019. http://dx.doi.org/10.5772/intechopen.72102.

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22

Wiranowska, Marzenna. Gliomas: Symptoms, Diagnosis and Treatment Options. Nova Science Publishers, Inc., 2013.

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23

Black, Peter M. Low Grade Gliomas: Diagnosis and Treatment. Humana Press, 2008.

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24

Hekmatpanah, J. Gliomas: Current Concepts in Biology, Diagnosis and Therapy. Springer, 2012.

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25

Gliomas: Current Concepts in Biology, Diagnosis and Therapy. Springer, 2011.

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26

Weller, Michael, Michael Brada, Tai-Tong Wong, and Michael A. Vogelbaum. Astrocytic tumours: diffuse astrocytoma, anaplastic astrocytoma, glioblastoma, and gliomatosis cerebri. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199651870.003.0003.

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Astrocytic gliomas are primary brain tumours thought to originate from neural stem or progenitor cells. They are assigned grades II, III, or IV by the World Health Organization according to degree of malignancy as defined by histology. The following molecular markers are increasingly used for diagnostic subclassification or clinical decision-making: 1p/19q co-deletion status, O6-methylguanine-DNA methyltransferase (MGMT) promoter methylation status, and isocitrate dehydrogenase 1 and 2 mutation status. Extent of resection is a favourable prognostic factor, but surgery is never curative. Radiotherapy prolongs progression-free survival across all astrocytic glioma entities. Alkylating agent chemotherapy is an active treatment in particular for patients with MGMT promoter-methylated tumours. Anti-angiogenic therapies have failed to improve survival, and the current focus of major clinical trials is on novel targeted agents or on immunotherapy.

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27

Huntoon, Kristin, and J. Bradley Elder. High-Grade Gliomas. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190696696.003.0001.

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Glioblastoma is the most common primary malignant brain tumor. This chapter discusses the clinical presentation and initial workup for a patient with a suspected glioblastoma, as well as the optimal treatment strategy and prognosis. Diagnosis is typically made using magnetic resonance imaging. Optimal treatment involves maximal safe surgical resection followed by adjuvant chemotherapy and radiation therapy. Surgical adjuncts including intraoperative imaging modalities and brain mapping techniques help improve neurologic morbidity associated with surgery. Despite maximal treatment, virtually all patients with glioblastoma will experience recurrence of their tumor and may be considered for clinical trials or second-line therapy. This chapter highlights important pearls associated with management of patients with glioblastoma and written for those who are interested in neuro-oncology, neurosurgery, and the field of brain tumors.

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28

Haranhalli, Neil, and Jerome J. Graber. Pineal Region Neoplasms. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0131.

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Pineal region tumors include a diverse array of neoplasms arising from various components of the pineal gland, including germ cell tumors, germinomas, teratomas, pineocytomas, pineoblastomas, and tumors derived from glial tissues including gliomas, astrocytomas, oligodendrogliomas, and ependymomas. Benign lesions of the pineal gland can include pineal cysts, calcifications and meningiomas. Metastatic tumors can also be found in the pineal region. Numerous infectious and inflammatory conditions can mimic pineal tumors. Most patients present with symptoms of hydrocephalus or Parinaud’s syndrome. Diagnosis often requires biopsy, though some germinomas may be diagnosed based solely on serum and cerebrospinal fluid biomarkers.

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29

Scarabino, Tommaso. Imaging Gliomas After Treatment: A Case-based Atlas. Springer, 2012.

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30

Scarabino, Tommaso, and Saverio Pollice. Imaging Gliomas after Treatment: A Case-Based Atlas. Springer International Publishing AG, 2021.

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31

Scarabino, Tommaso, and Saverio Pollice. Imaging Gliomas After Treatment: A Case-based Atlas. Springer, 2019.

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32

Scarabino, Tommaso. Imaging Gliomas after Treatment: A Case-Based Atlas. Springer Milan, 2016.

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33

Amirian, E. Susan, Quinn T. Ostrom, Yanhong Liu, Jill Barnholtz-Sloan, and Melissa L. Bondy. Nervous System. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0056.

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Although brain and other nervous system tumors are relatively rare, constituting up to 4% of incident primary cancer diagnoses, they are often associated with high morbidity and mortality. Several etiologic factors have been examined in relation to nervous system tumors, with the majority of studies focusing on central nervous system tumors. Despite decades of research, the only established risk factors for brain tumors are family history and moderate to high levels of ionizing radiation exposure. Differences in study designs, case ascertainment, control selection, and accuracy of exposure assessment are challenges associated with studying risk factors for nervous system tumors, and may partly explain why the majority of risk for these tumors remains unexplained. There is now substantial evidence that gliomas are inversely associated with allergies and atopy and positively associated with taller height.

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34

Pugliese, Roberto. Possibilità Di Trattamento Del Glioblastoma Multiforme e Di Altri Gliomi Di Alto Grado: Ben A. Williams Sopravvissuto Alla Diagnosi Di Glioblastoma Dal 30 Marzo 1995. Lulu Press, Inc., 2021.

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35

Letendre, Scott, Jennifer Iudicello, Beau Ances, Thomas D. Marcotte, Serena Spudich, and Mary Ann Cohen. HIV-Associated Neurocognitive Disorders. Edited by Mary Ann Cohen, Jack M. Gorman, Jeffrey M. Jacobson, Paul Volberding, and Scott Letendre. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199392742.003.0016.

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The human immunodeficiency virus (HIV) enters the central nervous system soon after infection; can infect glia and tissue macrophages in the brain; and can injure neurons, resulting in loss of dendrites. These and other processes underpin a syndrome of cognitive and motor impairment termed HIV-associated neurocognitive disorder (HAND). This chapter principally focuses on HAND, although delirium and other neurocognitive disorders are also discussed and should remain in the differential diagnosis of cognitive impairment in persons with HIV. A differential diagnosis of cognitive impairment in HIV also includes multimorbid conditions that can influence neurocognitive performance, such as metabolic syndrome, vascular disease, medication toxicity, and substance use disorders. When developing treatment recommendations for HAND, initiation of ART and treatment of multimorbid conditions and other neurocognitive disorders should be prioritized. It is important for clinicians to regularly monitor HIV patients for HAND and other neurocognitive disorders since cognitive impairment can affect activities of daily living; quality of life; adherence to risk reduction, medical care, and medication; and survival.

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